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Artigo de periodico
Interaction between Kölliker-Fuse/A7 and the parafacial respiratory region on the control of respiratory regulation (2024)
Oliveira, Luiz M. ; Moreira, Thiago dos Santos ; Moreira, Ana Carolina Takakura
Source: Respiratory Physiology & Neurobiology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, FISIOLOGIA RESPIRATÓRIA E CIRCULATÓRIA, RATOS WISTAR, NORADRENALINA, HORMÔNIOS DA MEDULA ADRENAL, RECEPTORES ADRENÉRGICOS, RESPIRAÇÃO ANIMAL
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OLIVEIRA, Luiz M. e MOREIRA, Thiago dos Santos e MOREIRA, Ana Carolina Takakura. Interaction between Kölliker-Fuse/A7 and the parafacial respiratory region on the control of respiratory regulation. Respiratory Physiology & Neurobiology, v. 320, p. 9 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.resp.2023.104201. Acesso em: 16 jun. 2024.
APA
Oliveira, L. M., Moreira, T. dos S., & Moreira, A. C. T. (2024). Interaction between Kölliker-Fuse/A7 and the parafacial respiratory region on the control of respiratory regulation. Respiratory Physiology & Neurobiology, 320, 9 . doi:10.1016/j.resp.2023.104201
NLM
Oliveira LM, Moreira T dos S, Moreira ACT. Interaction between Kölliker-Fuse/A7 and the parafacial respiratory region on the control of respiratory regulation [Internet]. Respiratory Physiology & Neurobiology. 2024 ; 320 9 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.resp.2023.104201
Vancouver
Oliveira LM, Moreira T dos S, Moreira ACT. Interaction between Kölliker-Fuse/A7 and the parafacial respiratory region on the control of respiratory regulation [Internet]. Respiratory Physiology & Neurobiology. 2024 ; 320 9 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.resp.2023.104201
Artigo de periodico
TREK-1 inhibition promotes synaptic plasticity in the prelimbic cortex (2024)
Francis-Oliveira, José Francis de ; Higa, Guilherme Shigueto Vilar ; Viana, Felipe José Costa ; Cruvinel, Emily ; Carlos-Lima, Estevão ; Borges, Fernando da Silva ; Zampieri, Thais Tessari ; Rebello, Fernanda Pereira ; Ulrich, Henning ; De Pasquale, Roberto
Source: Experimental Neurology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, PLASTICIDADE NEURONAL, DEPRESSÃO, SISTEMA LÍMBICO, CÓRTEX PRÉ-FRONTAL, SINAPSE, POTÁSSIO
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FRANCIS-OLIVEIRA, José Francis de et al. TREK-1 inhibition promotes synaptic plasticity in the prelimbic cortex. Experimental Neurology, v. 373, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2023.114652. Acesso em: 16 jun. 2024.
APA
Francis-Oliveira, J. F. de, Higa, G. S. V., Viana, F. J. C., Cruvinel, E., Carlos-Lima, E., Borges, F. da S., et al. (2024). TREK-1 inhibition promotes synaptic plasticity in the prelimbic cortex. Experimental Neurology, 373, 20 . doi:10.1016/j.expneurol.2023.114652
NLM
Francis-Oliveira JF de, Higa GSV, Viana FJC, Cruvinel E, Carlos-Lima E, Borges F da S, Zampieri TT, Rebello FP, Ulrich H, De Pasquale R. TREK-1 inhibition promotes synaptic plasticity in the prelimbic cortex [Internet]. Experimental Neurology. 2024 ; 373 20 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2023.114652
Vancouver
Francis-Oliveira JF de, Higa GSV, Viana FJC, Cruvinel E, Carlos-Lima E, Borges F da S, Zampieri TT, Rebello FP, Ulrich H, De Pasquale R. TREK-1 inhibition promotes synaptic plasticity in the prelimbic cortex [Internet]. Experimental Neurology. 2024 ; 373 20 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2023.114652
Artigo de periodico
The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages (2024)
Assis, Sayonara Ivana Santos de ; Amendola, Leonardo Szalo ; Ferreira, Guilherme da Silva ; Santos, Danielle Ribeiro ; Santana, Monique de Fátima Mello ; Santana, Kelly Gomes; Okamoto, Maristela Mitiko ; Iborra, Rodrigo Tallada ; Gianella, Maria Lúcia Cardillo Corrêa ; Barbeiro, Denise Frediani ; Soriano, Francisco Garcia ; Passarelli, Marisa ; Machado, Ubiratan Fabres
Source: International Journal of Molecular Sciences. Unidades: ICB, FM
Subjects: FISIOLOGIA, FATORES DE TRANSCRIÇÃO, PROTEÍNAS NUCLEARES, REGULAÇÃO GÊNICA, EXPRESSÃO GÊNICA, MACRÓFAGOS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, ALBUMINAS, INTERLEUCINA 6
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ASSIS, Sayonara Ivana Santos de et al. The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages. International Journal of Molecular Sciences, v. 25, n. 5, p. 16 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25052713. Acesso em: 16 jun. 2024.
APA
Assis, S. I. S. de, Amendola, L. S., Ferreira, G. da S., Santos, D. R., Santana, M. de F. M., Santana, K. G., et al. (2024). The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages. International Journal of Molecular Sciences, 25( 5), 16 . doi:https://doi.org/10.3390/ijms25052713
NLM
Assis SIS de, Amendola LS, Ferreira G da S, Santos DR, Santana M de FM, Santana KG, Okamoto MM, Iborra RT, Gianella MLCC, Barbeiro DF, Soriano FG, Passarelli M, Machado UF. The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 5): 16 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.3390/ijms25052713
Vancouver
Assis SIS de, Amendola LS, Ferreira G da S, Santos DR, Santana M de FM, Santana KG, Okamoto MM, Iborra RT, Gianella MLCC, Barbeiro DF, Soriano FG, Passarelli M, Machado UF. The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 5): 16 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.3390/ijms25052713
Artigo de periodico
Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice (2024)
Franco, Thaina Maquedo ; Tavares, Mariana Rosolen ; Novaes, Leonardo Santana ; Peixoto-Santos, Jose Eduardo ; Araujo, Ronaldo Carvalho ; Bader, Michael ; Wasinski, Frederick ; Munhoz, Carolina Demarchi ; Donato Junior, José
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, BRADICININA, INFLAMAÇÃO, SISTEMA NERVOSO CENTRAL, PRESSÃO SANGUÍNEA, PROTEÍNAS G, RECEPTORES DE CATECOLAMINA, RECEPTORES DE DOPAMINA, CAMUNDONGOS, PESOS E MEDIDAS CORPORAIS, AMINOÁCIDOS
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FRANCO, Thaina Maquedo et al. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, v. 25, n. 3, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25031490. Acesso em: 16 jun. 2024.
APA
Franco, T. M., Tavares, M. R., Novaes, L. S., Peixoto-Santos, J. E., Araujo, R. C., Bader, M., et al. (2024). Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, 25( 3), 14 . doi:10.3390/ijms25031490
NLM
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.3390/ijms25031490
Vancouver
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.3390/ijms25031490
Artigo de periodico
The course of attention-deficit/hyperactivity disorder through midlife (2024)
Grevet, Eugenio Horacio ; Bandeira, Cibele Edom ; Vitola, Eduardo Schneider ; Tavares, Maria Eduarda de Araujo ; Breda, Vitor ; Zeni, Gregory David ; Teche, Stefania Pigatto ; Picon, Felipe Almeida ; Salgado, Carlos Alberto Iglesias ; Karam, Rafael Gomes ; Silva, Bruna Santos da ; Sibley, Margaret H.; Rohde, Luis Augusto ; Cupertino, Renata Basso ; Bau, Claiton Henrique Dotto ; Rovaris, Diego Luiz
Source: European Archives of Psychiatry and Clinical Neuroscience. Unidade: ICB
Subjects: FISIOLOGIA, TRANSTORNO DO DEFICIT DE ATENÇÃO COM HIPERATIVIDADE, COMORBIDADE, ATENÇÃO
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GREVET, Eugenio Horacio et al. The course of attention-deficit/hyperactivity disorder through midlife. European Archives of Psychiatry and Clinical Neuroscience, v. 274, n. 1, p. 59-70, 2024Tradução . . Disponível em: https://doi.org/10.1007/s00406-022-01531-4. Acesso em: 16 jun. 2024.
APA
Grevet, E. H., Bandeira, C. E., Vitola, E. S., Tavares, M. E. de A., Breda, V., Zeni, G. D., et al. (2024). The course of attention-deficit/hyperactivity disorder through midlife. European Archives of Psychiatry and Clinical Neuroscience, 274( 1), 59-70. doi:10.1007/s00406-022-01531-4
NLM
Grevet EH, Bandeira CE, Vitola ES, Tavares ME de A, Breda V, Zeni GD, Teche SP, Picon FA, Salgado CAI, Karam RG, Silva BS da, Sibley MH, Rohde LA, Cupertino RB, Bau CHD, Rovaris DL. The course of attention-deficit/hyperactivity disorder through midlife [Internet]. European Archives of Psychiatry and Clinical Neuroscience. 2024 ; 274( 1): 59-70.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1007/s00406-022-01531-4
Vancouver
Grevet EH, Bandeira CE, Vitola ES, Tavares ME de A, Breda V, Zeni GD, Teche SP, Picon FA, Salgado CAI, Karam RG, Silva BS da, Sibley MH, Rohde LA, Cupertino RB, Bau CHD, Rovaris DL. The course of attention-deficit/hyperactivity disorder through midlife [Internet]. European Archives of Psychiatry and Clinical Neuroscience. 2024 ; 274( 1): 59-70.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1007/s00406-022-01531-4
Artigo de periodico
Biofunctionalization of surfaces to minimize undesirable effects in cardiovascular assistance devices (2024)
Sá, Rosa Correa Leoncio de ; Aron, José Pazin de Andrade ; Salvadori, Maria Cecília Barbosa da Silveira ; Teixeira, Fernanda de Sá ; Corat, Evaldo José ; Moro, José Roberto ; Bock, Eduardo Guy Perpetuo ; Arioldi, Vladimir Jesus Trava ; Antunes, Vagner Roberto
Source: Artificial Organs. Unidades: IF, ICB
Subjects: FISIOLOGIA, AORTA DE ANIMAL, VENTRÍCULO CARDÍACO DE ANIMAL, BIOMATERIAIS, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS, AORTA ABDOMINAL, MICROSCOPIA ELETRÔNICA DE VARREDURA ANIMAL, TITÂNIO, CARBONO
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SÁ, Rosa Correa Leoncio de et al. Biofunctionalization of surfaces to minimize undesirable effects in cardiovascular assistance devices. Artificial Organs, v. 48, n. 2, p. 141-149, 2024Tradução . . Disponível em: https://doi.org/10.1111/aor.14683. Acesso em: 16 jun. 2024.
APA
Sá, R. C. L. de, Aron, J. P. de A., Salvadori, M. C. B. da S., Teixeira, F. de S., Corat, E. J., Moro, J. R., et al. (2024). Biofunctionalization of surfaces to minimize undesirable effects in cardiovascular assistance devices. Artificial Organs, 48( 2), 141-149. doi:10.1111/aor.14683
NLM
Sá RCL de, Aron JP de A, Salvadori MCB da S, Teixeira F de S, Corat EJ, Moro JR, Bock EGP, Arioldi VJT, Antunes VR. Biofunctionalization of surfaces to minimize undesirable effects in cardiovascular assistance devices [Internet]. Artificial Organs. 2024 ; 48( 2): 141-149.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1111/aor.14683
Vancouver
Sá RCL de, Aron JP de A, Salvadori MCB da S, Teixeira F de S, Corat EJ, Moro JR, Bock EGP, Arioldi VJT, Antunes VR. Biofunctionalization of surfaces to minimize undesirable effects in cardiovascular assistance devices [Internet]. Artificial Organs. 2024 ; 48( 2): 141-149.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1111/aor.14683
Artigo de periodico
TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease (2024)
Cabral, Laís M. ; Oliveira, Luis M. ; Miranda, Nicole C.; Kawamoto, Elisa Mitiko ; Costa, Soraia Katia Pereira ; Moreira, Thiago S. ; Takakura, Ana C.
Source: Brain Research. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, INFLAMAÇÃO, CITOCINAS, NECROSE
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CABRAL, Laís M. et al. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, v. 1822, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2023.148586. Acesso em: 16 jun. 2024.
APA
Cabral, L. M., Oliveira, L. M., Miranda, N. C., Kawamoto, E. M., Costa, S. K. P., Moreira, T. S., & Takakura, A. C. (2024). TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, 1822. doi:10.1016/j.brainres.2023.148586
NLM
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586
Vancouver
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning Alexander ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Experimental Neurology. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
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FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 16 jun. 2024.
APA
Ferreira, A. F. F., Ulrich, H. A., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich HA, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich HA, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
Long-term glucocorticoid infusion impairs epididymal adipocyte metabolism and maturation and affects miR-150–5p actions (2024)
Salgueiro, Rafael Barrera ; Bolin, Anaysa Paola ; Andreotti, Sandra ; Komino, Ayumi Cristina Medeiros ; Sousa, Érica de; Silva, Flaviane de Fatima ; Proença, André Ricardo Gomes de ; Sertie, Rogério Antonio Laurato ; Rodrigues, Alice Cristina ; Lima, Fabio Bessa
Source: Molecular and Cellular Endocrinology. Unidades: ICB, FCF
Subjects: FISIOLOGIA, FARMACOLOGIA, HORMÔNIOS GLICOCORTICOIDES, TECIDO ADIPOSO, MICRORNAS, APOPTOSE, DOENÇAS METABÓLICAS, LIPÍDEOS, GORDURAS
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SALGUEIRO, Rafael Barrera et al. Long-term glucocorticoid infusion impairs epididymal adipocyte metabolism and maturation and affects miR-150–5p actions. Molecular and Cellular Endocrinology, v. 589, p. 12 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2024.112250. Acesso em: 16 jun. 2024.
APA
Salgueiro, R. B., Bolin, A. P., Andreotti, S., Komino, A. C. M., Sousa, É. de, Silva, F. de F., et al. (2024). Long-term glucocorticoid infusion impairs epididymal adipocyte metabolism and maturation and affects miR-150–5p actions. Molecular and Cellular Endocrinology, 589, 12 . doi:10.1016/j.mce.2024.112250
NLM
Salgueiro RB, Bolin AP, Andreotti S, Komino ACM, Sousa É de, Silva F de F, Proença ARG de, Sertie RAL, Rodrigues AC, Lima FB. Long-term glucocorticoid infusion impairs epididymal adipocyte metabolism and maturation and affects miR-150–5p actions [Internet]. Molecular and Cellular Endocrinology. 2024 ; 589 12 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.mce.2024.112250
Vancouver
Salgueiro RB, Bolin AP, Andreotti S, Komino ACM, Sousa É de, Silva F de F, Proença ARG de, Sertie RAL, Rodrigues AC, Lima FB. Long-term glucocorticoid infusion impairs epididymal adipocyte metabolism and maturation and affects miR-150–5p actions [Internet]. Molecular and Cellular Endocrinology. 2024 ; 589 12 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.mce.2024.112250
Artigo de periodico
Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells (2024)
Garcia, Bruna Ruschel Ewald Vega ; Makiyama, Edson Naoto ; Sampaio, Geni Rodrigues ; Freitas, Rosana Aparecida Manólio Soares ; Bovini, Andrea ; Amaral, Andressa Godoy ; Fock, Ricardo Ambrósio ; Rogero, Marcelo Macedo ; Bordin, Silvana
Source: Metabolites. Unidades: FCF, FSP, ICB
Subjects: FISIOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS EPITELIAIS E GLANDULARES, ADENOCARCINOMA, AMINOÁCIDOS, LIPOPOLISSACARÍDEOS, INFLAMAÇÃO, ANTI-INFLAMATÓRIOS, MUCOSA INTESTINAL, GLUTATIONA TRANSFERASE
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GARCIA, Bruna Ruschel Ewald Vega et al. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells. Metabolites, v. 14, n. 1, p. 13 , 2024Tradução . . Disponível em: https://doi.org/10.3390/metabo14010076. Acesso em: 16 jun. 2024.
APA
Garcia, B. R. E. V., Makiyama, E. N., Sampaio, G. R., Freitas, R. A. M. S., Bovini, A., Amaral, A. G., et al. (2024). Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells. Metabolites, 14( 1), 13 . doi:10.3390/metabo14010076
NLM
Garcia BREV, Makiyama EN, Sampaio GR, Freitas RAMS, Bovini A, Amaral AG, Fock RA, Rogero MM, Bordin S. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells [Internet]. Metabolites. 2024 ; 14( 1): 13 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.3390/metabo14010076
Vancouver
Garcia BREV, Makiyama EN, Sampaio GR, Freitas RAMS, Bovini A, Amaral AG, Fock RA, Rogero MM, Bordin S. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells [Internet]. Metabolites. 2024 ; 14( 1): 13 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.3390/metabo14010076
Artigo de periodico
Egg white hydrolysate protects white adipose tissue against metabolic insult in DOCA-Salt rats (2024)
Moro, Camila Rodrigues ; Abreu, Edina da Luz ; Kanaan, Samia Hassan Husein ; Márquez, Antonio; Uranga-Ocio, Jose Antonio; Vassalo, Dalton Valentim ; Castro, Marta Miguel ; Wiggers, Giulia Alessandra ; Rossoni, Luciana Venturini
Source: The British Journal of Nutrition. Unidade: ICB
Subjects: FISIOLOGIA, TECIDO ADIPOSO, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS, ESTRESSE OXIDATIVO, IMUNOFLUORESCÊNCIA EM ANIMAL, HIPERTENSÃO, CITOCINAS, DOENÇAS CARDIOVASCULARES EM ANIMAL
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MORO, Camila Rodrigues et al. Egg white hydrolysate protects white adipose tissue against metabolic insult in DOCA-Salt rats. The British Journal of Nutrition, p. 30 , 2024Tradução . . Disponível em: https://doi.org/10.1017/S0007114524000552. Acesso em: 16 jun. 2024.
APA
Moro, C. R., Abreu, E. da L., Kanaan, S. H. H., Márquez, A., Uranga-Ocio, J. A., Vassalo, D. V., et al. (2024). Egg white hydrolysate protects white adipose tissue against metabolic insult in DOCA-Salt rats. The British Journal of Nutrition, 30 . doi:10.1017/S0007114524000552
NLM
Moro CR, Abreu E da L, Kanaan SHH, Márquez A, Uranga-Ocio JA, Vassalo DV, Castro MM, Wiggers GA, Rossoni LV. Egg white hydrolysate protects white adipose tissue against metabolic insult in DOCA-Salt rats [Internet]. The British Journal of Nutrition. 2024 ;30 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1017/S0007114524000552
Vancouver
Moro CR, Abreu E da L, Kanaan SHH, Márquez A, Uranga-Ocio JA, Vassalo DV, Castro MM, Wiggers GA, Rossoni LV. Egg white hydrolysate protects white adipose tissue against metabolic insult in DOCA-Salt rats [Internet]. The British Journal of Nutrition. 2024 ;30 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1017/S0007114524000552
Artigo de periodico
Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats (2024)
Dellacqua, Lais Oliveira; Gomes, Paula Magalhães ; Batista, Julia Santos; Michelini, Lisete Compagno ; Antunes, Vagner Roberto
Source: Journal of Applied Physiology. Unidade: ICB
Subjects: FISIOLOGIA, BARORREFLEXO, FISIOLOGIA CARDIOVASCULAR, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, HIPERTENSÃO, ENVELHECIMENTO, TREINAMENTO AERÓBIO, PLASTICIDADE NEURONAL, RATOS WISTAR
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DELLACQUA, Lais Oliveira et al. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats. Journal of Applied Physiology, v. 136, n. 1, p. 189-198, 2024Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.00433.2023. Acesso em: 16 jun. 2024.
APA
Dellacqua, L. O., Gomes, P. M., Batista, J. S., Michelini, L. C., & Antunes, V. R. (2024). Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats. Journal of Applied Physiology, 136( 1), 189-198. doi:10.1152/japplphysiol.00433.2023
NLM
Dellacqua LO, Gomes PM, Batista JS, Michelini LC, Antunes VR. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats [Internet]. Journal of Applied Physiology. 2024 ; 136( 1): 189-198.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1152/japplphysiol.00433.2023
Vancouver
Dellacqua LO, Gomes PM, Batista JS, Michelini LC, Antunes VR. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats [Internet]. Journal of Applied Physiology. 2024 ; 136( 1): 189-198.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1152/japplphysiol.00433.2023
Artigo de periodico
A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice (2024)
Lessa, Tássia Liz Araújo dos Santos; Correia, Thiago Macêdo Lopes ; Santos, Talita Costa dos; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da ; Cavallini, Maria Cláudia Magalhães; Rocha, Leonardo Silva; Peixoto, Álbert Souza; Cugnasca, Beatriz dos Santos ; Cervi, Gustavo ; Correra, Thiago Carita ; Gonçalves, Augusto Cesar; Festuccia, William Tadeu Lara ; Cunha, Thiago Mattar ; Yatsuda, Regiane ; Magalhães, Amélia Cristina Mendes de ; Santos, Alcindo Aparecido dos ; Meotti, Flavia Carla ; Queiroz, Raphael Ferreira
Source: Free Radical Research. Unidades: IQ, ICB, FMRP
Subjects: NEUTRÓFILOS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LESSA, Tássia Liz Araújo dos Santos et al. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research, 2024Tradução . . Disponível em: https://dx.doi.org/10.1080/10715762.2024.2336566. Acesso em: 16 jun. 2024.
APA
Lessa, T. L. A. dos S., Correia, T. M. L., Santos, T. C. dos, Silva, R. P. da, Silva, B. P. da, Cavallini, M. C. M., et al. (2024). A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research. doi:10.1080/10715762.2024.2336566
NLM
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto ÁS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Festuccia WTL, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ;[citado 2024 jun. 16 ] Available from: https://dx.doi.org/10.1080/10715762.2024.2336566
Vancouver
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto ÁS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Festuccia WTL, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ;[citado 2024 jun. 16 ] Available from: https://dx.doi.org/10.1080/10715762.2024.2336566
Artigo de periodico
Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease (2024)
Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Figueroa, Paulina Belén Sepulveda ; Cuyul-Vásquez, Iván; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de
Source: Ageing Research Reviews. Unidades: ICB, FM
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇA DE PARKINSON, LÍTIO, DOENÇAS NEURODEGENERATIVAS, REVISÃO SISTEMÁTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SINGULANI, Monique Patricio et al. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, v. 95, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2024.102231. Acesso em: 16 jun. 2024.
APA
Singulani, M. P., Ferreira, A. F. F., Figueroa, P. B. S., Cuyul-Vásquez, I., Talib, L. L., Forlenza, O. V., & Britto, L. R. G. de. (2024). Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, 95, 20 . doi:10.1016/j.arr.2024.102231
NLM
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Vancouver
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Artigo de periodico
Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease (2023)
Pereira, Carolina Parga Martins; Oliveira, José Francis ; Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Journal of Neuroimmunology. Unidades: ICB, FM
Subjects: FISIOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, NEURÓGLIA, TOXINAS, COORDENAÇÃO MOTORA, RECEPTORES DOPAMINÉRGICOS, NEURÔNIOS MOTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Carolina Parga Martins et al. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease. Journal of Neuroimmunology, v. 375, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.jneuroim.2023.578019. Acesso em: 16 jun. 2024.
APA
Pereira, C. P. M., Oliveira, J. F., Singulani, M. P., Ferreira, A. F. F., & Britto, L. R. G. de. (2023). Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease. Journal of Neuroimmunology, 375, 1-11. doi:10.1016/j.jneuroim.2023.578019
NLM
Pereira CPM, Oliveira JF, Singulani MP, Ferreira AFF, Britto LRG de. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease [Internet]. Journal of Neuroimmunology. 2023 ; 375 1-11.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.jneuroim.2023.578019
Vancouver
Pereira CPM, Oliveira JF, Singulani MP, Ferreira AFF, Britto LRG de. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease [Internet]. Journal of Neuroimmunology. 2023 ; 375 1-11.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.jneuroim.2023.578019
Artigo de periodico
"Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " (2023)
Raquel, Hiviny de Ataides ; Perego, Sany M. ; Masson, Gustavo S.; Jensen, Leonardo ; Colquhoun, Alison ; Michelini, Lisete Compagno
Source: Clinical Science. Unidade: ICB
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, INSUFICIÊNCIA CARDÍACA ANIMAL, BARREIRA HEMATO-ENCEFÁLICA, RATOS WISTAR, SISTEMA NERVOSO AUTÔNOMO, CIRCULAÇÃO ANIMAL, EXERCÍCIO FÍSICO, PROTEÍNAS DA MEMBRANA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAQUEL, Hiviny de Ataides et al. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training ". Clinical Science, v. 137, n. 15, p. 1049-1066, 2023Tradução . . Disponível em: https://doi.org/10.1042/CS20230489. Acesso em: 16 jun. 2024.
APA
Raquel, H. de A., Perego, S. M., Masson, G. S., Jensen, L., Colquhoun, A., & Michelini, L. C. (2023). "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training ". Clinical Science, 137( 15), 1049-1066. doi:10.1042/CS20230489
NLM
Raquel H de A, Perego SM, Masson GS, Jensen L, Colquhoun A, Michelini LC. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " [Internet]. Clinical Science. 2023 ; 137( 15): 1049-1066.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1042/CS20230489
Vancouver
Raquel H de A, Perego SM, Masson GS, Jensen L, Colquhoun A, Michelini LC. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " [Internet]. Clinical Science. 2023 ; 137( 15): 1049-1066.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1042/CS20230489
Artigo de periodico
Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions (2023)
Hamasaki, Mike Yoshio ; Mendes, Caroline; Batagello, Daniella Sabino ; Hirata, Mario Hiroyuki ; Britto, Luiz Roberto Giorgetti de ; Nogueira, Maria Inês
Source: NeuroReport. Unidades: FCF, ICB
Subjects: ANÓXIA, CÓRTEX CEREBRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HAMASAKI, Mike Yoshio et al. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, v. 34, n. 11, p. 575-582, 2023Tradução . . Disponível em: https://doi.org/10.1097/WNR.0000000000001927. Acesso em: 16 jun. 2024.
APA
Hamasaki, M. Y., Mendes, C., Batagello, D. S., Hirata, M. H., Britto, L. R. G. de, & Nogueira, M. I. (2023). Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, 34( 11), 575-582. doi:10.1097/WNR.0000000000001927
NLM
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Vancouver
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Artigo de periodico
Programming of metabolism by adipokines during development (2023)
Donato Junior, José
Source: Nature Reviews Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, TECIDO ADIPOSO, METABOLISMO, DOENÇAS METABÓLICAS, HOMEOSTASE, GLICOSE, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DONATO JUNIOR, José. Programming of metabolism by adipokines during development. Nature Reviews Endocrinology, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41574-023-00828-1. Acesso em: 16 jun. 2024.
APA
Donato Junior, J. (2023). Programming of metabolism by adipokines during development. Nature Reviews Endocrinology, 1-13. doi:10.1038/s41574-023-00828-1
NLM
Donato Junior J. Programming of metabolism by adipokines during development [Internet]. Nature Reviews Endocrinology. 2023 ; 1-13.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1038/s41574-023-00828-1
Vancouver
Donato Junior J. Programming of metabolism by adipokines during development [Internet]. Nature Reviews Endocrinology. 2023 ; 1-13.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1038/s41574-023-00828-1
Artigo de periodico
Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure (2023)
Fontes, Milene T.; Arruda Junior, Daniel F.; Santos, Danúbia Silva dos ; Silva, João Carlos Ribeiro ; Antônio, Edinei L.; Tucci, Paulo F. J. ; Rossoni, Luciana Venturini ; Girardi, Adriana C. C.
Source: Life Sciences. Unidades: ICB, FM
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FONTES, Milene T. et al. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, v. 323, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121648. Acesso em: 16 jun. 2024.
APA
Fontes, M. T., Arruda Junior, D. F., Santos, D. S. dos, Silva, J. C. R., Antônio, E. L., Tucci, P. F. J., et al. (2023). Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, 323, 1-10. doi:10.1016/j.lfs.2023.121648
NLM
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Vancouver
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 jun. 16 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Artigo de periodico
Effect of Uncaria tomentosa aqueous extract on the response to palmitate-induced lipotoxicity in cultured skeletal muscle cells (2023)
Marchi, Bruna Leticia de Freitas ; Santos, Jeniffer Farias dos ; Reigado, Gustavo Roncoli ; Fernandes, Myrian Thiago Pruschinski ; Alcalde, Felipe Santiago Chambergo ; Carvalho, Carla Roberta de Oliveira ; Dantas, Viviane Abreu Nunes Cerqueira
Source: BMC Complementary Medicine and Therapies. Unidades: EACH, ICB, BIOTECNOLOGIA
Subjects: DIABETES MELLITUS, CÉLULAS MUSCULARES, RESISTÊNCIA À INSULINA, ESTRESSE OXIDATIVO, EXTRATOS (FORMAS FARMACÊUTICAS), ESPECTROSCOPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARCHI, Bruna Leticia de Freitas et al. Effect of Uncaria tomentosa aqueous extract on the response to palmitate-induced lipotoxicity in cultured skeletal muscle cells. BMC Complementary Medicine and Therapies, v. 23, n. 412, p. 01-11, 2023Tradução . . Disponível em: http://dx.doi.org/10.1186/s12906-023-04204-4. Acesso em: 16 jun. 2024.
APA
Marchi, B. L. de F., Santos, J. F. dos, Reigado, G. R., Fernandes, M. T. P., Alcalde, F. S. C., Carvalho, C. R. de O., & Dantas, V. A. N. C. (2023). Effect of Uncaria tomentosa aqueous extract on the response to palmitate-induced lipotoxicity in cultured skeletal muscle cells. BMC Complementary Medicine and Therapies, 23( 412), 01-11. doi:10.1186/s12906-023-04204-4
NLM
Marchi BL de F, Santos JF dos, Reigado GR, Fernandes MTP, Alcalde FSC, Carvalho CR de O, Dantas VANC. Effect of Uncaria tomentosa aqueous extract on the response to palmitate-induced lipotoxicity in cultured skeletal muscle cells [Internet]. BMC Complementary Medicine and Therapies. 2023 ; 23( 412): 01-11.[citado 2024 jun. 16 ] Available from: http://dx.doi.org/10.1186/s12906-023-04204-4
Vancouver
Marchi BL de F, Santos JF dos, Reigado GR, Fernandes MTP, Alcalde FSC, Carvalho CR de O, Dantas VANC. Effect of Uncaria tomentosa aqueous extract on the response to palmitate-induced lipotoxicity in cultured skeletal muscle cells [Internet]. BMC Complementary Medicine and Therapies. 2023 ; 23( 412): 01-11.[citado 2024 jun. 16 ] Available from: http://dx.doi.org/10.1186/s12906-023-04204-4

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