TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease (2024)
- Authors:
- USP affiliated authors: IWASHE, ELISA MITIKO KAWAMOTO - ICB ; COSTA, SORAIA KÁTIA PEREIRA - ICB ; MOREIRA, THIAGO DOS SANTOS - ICB ; MOREIRA, ANA CAROLINA TAKAKURA - ICB ; CABRAL, LAÍS MARIA CARDOSO - ICB ; MIRANDA, NICOLE CASTRO DE SOUZA - ICB
- Unidade: ICB
- DOI: 10.1016/j.brainres.2023.148586
- Subjects: FISIOLOGIA; FARMACOLOGIA; CAMUNDONGOS; MODELOS ANIMAIS DE DOENÇAS; DOENÇA DE PARKINSON; INFLAMAÇÃO; CITOCINAS; NECROSE
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher: Elsevier BV
- Publisher place: Amsterdam
- Date published: 2024
- Source:
- Título: Brain Research
- ISSN: 1872-6240
- Volume/Número/Paginação/Ano: v. 1822, art. 148586, 2024
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
- Licença: publisher-specific-oa
-
ABNT
CABRAL, Laís M. et al. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, v. 1822, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2023.148586. Acesso em: 28 dez. 2025. -
APA
Cabral, L. M., Oliveira, L. M., Miranda, N. C., Kawamoto, E. M., Costa, S. K. P., Moreira, T. S., & Takakura, A. C. (2024). TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, 1822. doi:10.1016/j.brainres.2023.148586 -
NLM
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586 -
Vancouver
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586 - Sleep-related respiratory disruptions and laterodorsal tegmental nucleus in a mouse model of Parkinson’s disease
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- Regulation of blood vessels by ATP in the ventral medullary surface in a rat model of Parkinson’s disease
- Inhibition of anandamide hydrolysis does not rescue respiratory abnormalities observed in an animal model of Parkinson's disease
- Respiratory deficits in a female rat model of Parkinson's disease
- Attenuated baroreflex in a Parkinson's disease animal model coincides with impaired activation of non-C1 neurons
- Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats
- Regulation of the chemosensory control of breathing by Kölliker-Fuse neurons
- Independent purinergic mechanisms of central and peripheral chemoreception in the rostral ventrolateral medulla
- Molecular underpinnings of ventral surface chemoreceptor function: focus on KCNQ channels
Informações sobre o DOI: 10.1016/j.brainres.2023.148586 (Fonte: oaDOI API)
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