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Artigo de periodico
Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity (2022)
Monteiro, Lauar de Brito ; Prodonoff, Juliana Silveira ; Aguiar, Cristhiane Favero de ; Silva, Felipe Correa da; Castoldi, Angela ; Bakker, Nikki van Teijlingen ; Davanzo, Gustavo Gastão ; Donato Junior, José ; Câmara, Niels Olsen Saraiva
Source: Diabetes. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO, MACRÓFAGOS, RESISTÊNCIA À INSULINA, CITOCINAS, TECIDO ADIPOSO, INFLAMAÇÃO, OBESIDADE, CAMUNDONGOS
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ABNT
MONTEIRO, Lauar de Brito et al. Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity. Diabetes, v. 71, n. 7, p. 1546–1561, 2022Tradução . . Disponível em: https://doi.org/10.2337/db21-0842. Acesso em: 18 nov. 2024.
APA
Monteiro, L. de B., Prodonoff, J. S., Aguiar, C. F. de, Silva, F. C. da, Castoldi, A., Bakker, N. van T., et al. (2022). Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity. Diabetes, 71( 7), 1546–1561. doi:10.2337/db21-0842
NLM
Monteiro L de B, Prodonoff JS, Aguiar CF de, Silva FC da, Castoldi A, Bakker N van T, Davanzo GG, Donato Junior J, Câmara NOS. Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity [Internet]. Diabetes. 2022 ; 71( 7): 1546–1561.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db21-0842
Vancouver
Monteiro L de B, Prodonoff JS, Aguiar CF de, Silva FC da, Castoldi A, Bakker N van T, Davanzo GG, Donato Junior J, Câmara NOS. Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity [Internet]. Diabetes. 2022 ; 71( 7): 1546–1561.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db21-0842
Artigo de periodico
Melatonin increases brown adipose tissue volume and activity in patients with melatonin deficiency: a proof-of-concept study (2019)
Halpern, Bruno; Mancini, Marcio Côrrea; Bueno, Clarissa; Barcelos, Isabella Peixoto de; Melo, Maria Edna de; Lima, Marcos Santos; Carneiro, Camila de Godoi; Sapienza, Marcelo Tatit; Buchpiguel, Carlos Alberto ; Amaral, Fernanda Gaspar do; Cipolla Neto, José
Source: Diabetes. Unidades: FM, ICB
Subjects: MELATONINA, METABOLISMO ENERGÉTICO, TECIDO ADIPOSO, AVALIAÇÃO TERAPÊUTICA, HORMÔNIO DA GLÂNDULA PINEAL, RESISTÊNCIA À INSULINA, BETA-BLOQUEADORES
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ABNT
HALPERN, Bruno et al. Melatonin increases brown adipose tissue volume and activity in patients with melatonin deficiency: a proof-of-concept study. Diabetes, v. 68, n. 5, p. 947-952, 2019Tradução . . Disponível em: https://doi.org/10.2337/db18-0956. Acesso em: 18 nov. 2024.
APA
Halpern, B., Mancini, M. C., Bueno, C., Barcelos, I. P. de, Melo, M. E. de, Lima, M. S., et al. (2019). Melatonin increases brown adipose tissue volume and activity in patients with melatonin deficiency: a proof-of-concept study. Diabetes, 68( 5), 947-952. doi:10.2337/db18-0956
NLM
Halpern B, Mancini MC, Bueno C, Barcelos IP de, Melo ME de, Lima MS, Carneiro C de G, Sapienza MT, Buchpiguel CA, Amaral FG do, Cipolla Neto J. Melatonin increases brown adipose tissue volume and activity in patients with melatonin deficiency: a proof-of-concept study [Internet]. Diabetes. 2019 ; 68( 5): 947-952.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db18-0956
Vancouver
Halpern B, Mancini MC, Bueno C, Barcelos IP de, Melo ME de, Lima MS, Carneiro C de G, Sapienza MT, Buchpiguel CA, Amaral FG do, Cipolla Neto J. Melatonin increases brown adipose tissue volume and activity in patients with melatonin deficiency: a proof-of-concept study [Internet]. Diabetes. 2019 ; 68( 5): 947-952.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db18-0956
Artigo de periodico
Altered response of skeletal muscle to IL-6 in type 2 diabetic patients (2013)
Jiang, Lake Q.; Duque-Guimaraes, Daniella Esteves; Machado, Ubiratan Fabres ; Zierath, Juleen R.; Krook, Anna
Source: Diabetes. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
JIANG, Lake Q. et al. Altered response of skeletal muscle to IL-6 in type 2 diabetic patients. Diabetes, v. 62, n. 2, p. 355-361, 2013Tradução . . Disponível em: https://doi.org/10.2337/db11-1790. Acesso em: 18 nov. 2024.
APA
Jiang, L. Q., Duque-Guimaraes, D. E., Machado, U. F., Zierath, J. R., & Krook, A. (2013). Altered response of skeletal muscle to IL-6 in type 2 diabetic patients. Diabetes, 62( 2), 355-361. doi:10.2337/db11-1790
NLM
Jiang LQ, Duque-Guimaraes DE, Machado UF, Zierath JR, Krook A. Altered response of skeletal muscle to IL-6 in type 2 diabetic patients [Internet]. Diabetes. 2013 ; 62( 2): 355-361.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db11-1790
Vancouver
Jiang LQ, Duque-Guimaraes DE, Machado UF, Zierath JR, Krook A. Altered response of skeletal muscle to IL-6 in type 2 diabetic patients [Internet]. Diabetes. 2013 ; 62( 2): 355-361.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db11-1790
Artigo de periodico
Targeted disruption of inducible nitric oxide synthase protects against aging, S-nitrosation, and insulin resistance in muscle of male mice (2013)
Ropelle, Eduardo Rochete; Pauli, José Rodrigo; Cintra, Dennys Esper; Silva, Adelino Sanchez Ramos da ; Souza, Cláudio Teodoro de; Guadagnini, Dioze; Carvalho, Bruno M.; Caricilli, Andréa M.; Katashima, Carlos Kiyoshi; Carvalho-Filho, Marco A.; Hirabara, Sandro; Curi, Rui; Velloso, Lício Augusto; Saad, Mario José Abdalla; Carvalheira, José B. C.
Source: Diabetes. Unidades: EEFERP, ICB
Subjects: ENVELHECIMENTO, INSULINA (RESISTÊNCIA), ÓXIDO NÍTRICO, CONDICIONAMENTO FÍSICO (EXPERIMENTAÇÃO)
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ABNT
ROPELLE, Eduardo Rochete et al. Targeted disruption of inducible nitric oxide synthase protects against aging, S-nitrosation, and insulin resistance in muscle of male mice. Diabetes, v. 62, n. 2, p. 466-470, 2013Tradução . . Disponível em: https://doi.org/10.2337/db12-0339. Acesso em: 18 nov. 2024.
APA
Ropelle, E. R., Pauli, J. R., Cintra, D. E., Silva, A. S. R. da, Souza, C. T. de, Guadagnini, D., et al. (2013). Targeted disruption of inducible nitric oxide synthase protects against aging, S-nitrosation, and insulin resistance in muscle of male mice. Diabetes, 62( 2), 466-470. doi:10.2337/db12-0339
NLM
Ropelle ER, Pauli JR, Cintra DE, Silva ASR da, Souza CT de, Guadagnini D, Carvalho BM, Caricilli AM, Katashima CK, Carvalho-Filho MA, Hirabara S, Curi R, Velloso LA, Saad MJA, Carvalheira JBC. Targeted disruption of inducible nitric oxide synthase protects against aging, S-nitrosation, and insulin resistance in muscle of male mice [Internet]. Diabetes. 2013 ; 62( 2): 466-470.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db12-0339
Vancouver
Ropelle ER, Pauli JR, Cintra DE, Silva ASR da, Souza CT de, Guadagnini D, Carvalho BM, Caricilli AM, Katashima CK, Carvalho-Filho MA, Hirabara S, Curi R, Velloso LA, Saad MJA, Carvalheira JBC. Targeted disruption of inducible nitric oxide synthase protects against aging, S-nitrosation, and insulin resistance in muscle of male mice [Internet]. Diabetes. 2013 ; 62( 2): 466-470.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2337/db12-0339
Artigo de periodico
Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles (2010)
Collins, Stephan C.; Hoppa, Michael B.; Walker, Jonathan N.; Amisten, Stefan; Abdulkader, Fernando Rodrigues de Moraes ; Bengtsson, Martin; Fearnside, Jane; Ramracheya, Reshma; Toye, Ayo A.; Zhang, Quan; Clark, Anne; Gauguier, Dominique; Rorsman, Patrik
Source: Diabetes. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
COLLINS, Stephan C. et al. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, v. 59, n. 5, p. 1192-1201, 2010Tradução . . Acesso em: 18 nov. 2024.
APA
Collins, S. C., Hoppa, M. B., Walker, J. N., Amisten, S., Abdulkader, F. R. de M., Bengtsson, M., et al. (2010). Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, 59( 5), 1192-1201.
NLM
Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 nov. 18 ]
Vancouver
Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
The Improvement of Pancreatic Islets Function by Dehidroepiandrosterone (DHEA) Administration in Aging Rats (2010)
Almeida, Felipe N.; Amaral, Anderson C.; Camporez, João Paulo; Veras, Katherine M. A.; Campana, Amanda B.; Siqueira Filho, Mario A.; Ribeiro, Luciene; Carvalho, Carla Roberta de Oliveira
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
ALMEIDA, Felipe N. et al. The Improvement of Pancreatic Islets Function by Dehidroepiandrosterone (DHEA) Administration in Aging Rats. Diabetes. Estados Unidos: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2010
APA
Almeida, F. N., Amaral, A. C., Camporez, J. P., Veras, K. M. A., Campana, A. B., Siqueira Filho, M. A., et al. (2010). The Improvement of Pancreatic Islets Function by Dehidroepiandrosterone (DHEA) Administration in Aging Rats. Diabetes. Estados Unidos: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Almeida FN, Amaral AC, Camporez JP, Veras KMA, Campana AB, Siqueira Filho MA, Ribeiro L, Carvalho CR de O. The Improvement of Pancreatic Islets Function by Dehidroepiandrosterone (DHEA) Administration in Aging Rats. Diabetes. 2010 ; 59[citado 2024 nov. 18 ]
Vancouver
Almeida FN, Amaral AC, Camporez JP, Veras KMA, Campana AB, Siqueira Filho MA, Ribeiro L, Carvalho CR de O. The Improvement of Pancreatic Islets Function by Dehidroepiandrosterone (DHEA) Administration in Aging Rats. Diabetes. 2010 ; 59[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Pioglitazone promotes recovery of adipose mass in streptozotocin-induced diabetic rats by reducing lipolysis and stimulating lipogenic enzymes acitivity and their gene expression (2008)
Takada, Julie; Campos, Tarcila B. Ferraz de; Fonseca-Alaniz, Miriam H.; Andreotti, Sandra ; Campana, Amanda B.; Lima, Fábio Bessa
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
TAKADA, Julie et al. Pioglitazone promotes recovery of adipose mass in streptozotocin-induced diabetic rats by reducing lipolysis and stimulating lipogenic enzymes acitivity and their gene expression. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2008
APA
Takada, J., Campos, T. B. F. de, Fonseca-Alaniz, M. H., Andreotti, S., Campana, A. B., & Lima, F. B. (2008). Pioglitazone promotes recovery of adipose mass in streptozotocin-induced diabetic rats by reducing lipolysis and stimulating lipogenic enzymes acitivity and their gene expression. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Takada J, Campos TBF de, Fonseca-Alaniz MH, Andreotti S, Campana AB, Lima FB. Pioglitazone promotes recovery of adipose mass in streptozotocin-induced diabetic rats by reducing lipolysis and stimulating lipogenic enzymes acitivity and their gene expression. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Vancouver
Takada J, Campos TBF de, Fonseca-Alaniz MH, Andreotti S, Campana AB, Lima FB. Pioglitazone promotes recovery of adipose mass in streptozotocin-induced diabetic rats by reducing lipolysis and stimulating lipogenic enzymes acitivity and their gene expression. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Chronic supplementation with conjugated linoleic acid reduces adipose mass and impairs insulin sensitivity in periepidydimal adipose tissue (2008)
Campos, Tarcila B. Ferraz de; Takada, Julie; Fonseca-Alaniz, Miriam H.; Andreotti, Sandra ; Campana, Amanda B.; Lima, Fábio Bessa
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CAMPOS, Tarcila B. Ferraz de et al. Chronic supplementation with conjugated linoleic acid reduces adipose mass and impairs insulin sensitivity in periepidydimal adipose tissue. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2008
APA
Campos, T. B. F. de, Takada, J., Fonseca-Alaniz, M. H., Andreotti, S., Campana, A. B., & Lima, F. B. (2008). Chronic supplementation with conjugated linoleic acid reduces adipose mass and impairs insulin sensitivity in periepidydimal adipose tissue. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Campos TBF de, Takada J, Fonseca-Alaniz MH, Andreotti S, Campana AB, Lima FB. Chronic supplementation with conjugated linoleic acid reduces adipose mass and impairs insulin sensitivity in periepidydimal adipose tissue. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Vancouver
Campos TBF de, Takada J, Fonseca-Alaniz MH, Andreotti S, Campana AB, Lima FB. Chronic supplementation with conjugated linoleic acid reduces adipose mass and impairs insulin sensitivity in periepidydimal adipose tissue. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Regulation of BMP-9 expression in the perfused liver of insulin-resistance models (2008)
Caperuto, Luciana C.; Anhê, Gabriel Forato; Cambiaghi, Tavane D.; Akamine, Eliana Hiromi ; Ribeiro, Luciene M.; Cipolla Neto, José; Carvalho, Carla Roberta de Oliveira ; Curi, Rui; Bordin, Silvana
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CAPERUTO, Luciana C. et al. Regulation of BMP-9 expression in the perfused liver of insulin-resistance models. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2008
APA
Caperuto, L. C., Anhê, G. F., Cambiaghi, T. D., Akamine, E. H., Ribeiro, L. M., Cipolla Neto, J., et al. (2008). Regulation of BMP-9 expression in the perfused liver of insulin-resistance models. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Caperuto LC, Anhê GF, Cambiaghi TD, Akamine EH, Ribeiro LM, Cipolla Neto J, Carvalho CR de O, Curi R, Bordin S. Regulation of BMP-9 expression in the perfused liver of insulin-resistance models. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Vancouver
Caperuto LC, Anhê GF, Cambiaghi TD, Akamine EH, Ribeiro LM, Cipolla Neto J, Carvalho CR de O, Curi R, Bordin S. Regulation of BMP-9 expression in the perfused liver of insulin-resistance models. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Cardiovascular and metabolic effects of training and detraining in hypertensive insulin- resistant rats (2008)
Lehnen, Alexandre; Leguisamo, Natália; Machado, Ubiratan Fabres ; Schaan, Beatriz D.
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
LEHNEN, Alexandre et al. Cardiovascular and metabolic effects of training and detraining in hypertensive insulin- resistant rats. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2008
APA
Lehnen, A., Leguisamo, N., Machado, U. F., & Schaan, B. D. (2008). Cardiovascular and metabolic effects of training and detraining in hypertensive insulin- resistant rats. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Lehnen A, Leguisamo N, Machado UF, Schaan BD. Cardiovascular and metabolic effects of training and detraining in hypertensive insulin- resistant rats. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Vancouver
Lehnen A, Leguisamo N, Machado UF, Schaan BD. Cardiovascular and metabolic effects of training and detraining in hypertensive insulin- resistant rats. Diabetes. 2008 ; 57[citado 2024 nov. 18 ]
Artigo de periodico
Loss-of-function mutation in toll-like receptor 4 prevents diet-induced obesity and insulin resistance (2007)
Tsukumo, Daniela M. L.; Carvalho-Filho, Marco A.; Carvalheira, José B. C.; Prada, Patrícia O.; Hirabara, Sandro M.; Schenka, André A.; Araújo, Eliana P.; Vassallo, José; Curi, Rui; Velloso, Lício Augusto; Saad, Mario J. A.
Source: Diabetes. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
TSUKUMO, Daniela M. L. et al. Loss-of-function mutation in toll-like receptor 4 prevents diet-induced obesity and insulin resistance. Diabetes, v. 56, n. 8, p. 1986-1998, 2007Tradução . . Acesso em: 18 nov. 2024.
APA
Tsukumo, D. M. L., Carvalho-Filho, M. A., Carvalheira, J. B. C., Prada, P. O., Hirabara, S. M., Schenka, A. A., et al. (2007). Loss-of-function mutation in toll-like receptor 4 prevents diet-induced obesity and insulin resistance. Diabetes, 56( 8), 1986-1998.
NLM
Tsukumo DML, Carvalho-Filho MA, Carvalheira JBC, Prada PO, Hirabara SM, Schenka AA, Araújo EP, Vassallo J, Curi R, Velloso LA, Saad MJA. Loss-of-function mutation in toll-like receptor 4 prevents diet-induced obesity and insulin resistance. Diabetes. 2007 ; 56( 8): 1986-1998.[citado 2024 nov. 18 ]
Vancouver
Tsukumo DML, Carvalho-Filho MA, Carvalheira JBC, Prada PO, Hirabara SM, Schenka AA, Araújo EP, Vassallo J, Curi R, Velloso LA, Saad MJA. Loss-of-function mutation in toll-like receptor 4 prevents diet-induced obesity and insulin resistance. Diabetes. 2007 ; 56( 8): 1986-1998.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Daily melatonin administration improves the insulin sensitivity and some of the early events in the insulin signaling pathway into hypothalamus, liver, skeletal muscle, and adipose tissue of 15 month-old obese rats (2007)
Zanuto, Ricardo; Hirata, Emiko A.; Marçal, Anderson C.; Siqueira-Filho, Mario A.; Camporez, João P.; Caperuto, Luciana C; Ribeiro, Luciene M.; Bordin, Silvana ; Cipolla Neto, José; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ZANUTO, Ricardo et al. Daily melatonin administration improves the insulin sensitivity and some of the early events in the insulin signaling pathway into hypothalamus, liver, skeletal muscle, and adipose tissue of 15 month-old obese rats. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2007
APA
Zanuto, R., Hirata, E. A., Marçal, A. C., Siqueira-Filho, M. A., Camporez, J. P., Caperuto, L. C., et al. (2007). Daily melatonin administration improves the insulin sensitivity and some of the early events in the insulin signaling pathway into hypothalamus, liver, skeletal muscle, and adipose tissue of 15 month-old obese rats. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Zanuto R, Hirata EA, Marçal AC, Siqueira-Filho MA, Camporez JP, Caperuto LC, Ribeiro LM, Bordin S, Cipolla Neto J, Carpinelli AR, Carvalho CR de O. Daily melatonin administration improves the insulin sensitivity and some of the early events in the insulin signaling pathway into hypothalamus, liver, skeletal muscle, and adipose tissue of 15 month-old obese rats. Diabetes. 2007 ; 56[citado 2024 nov. 18 ]
Vancouver
Zanuto R, Hirata EA, Marçal AC, Siqueira-Filho MA, Camporez JP, Caperuto LC, Ribeiro LM, Bordin S, Cipolla Neto J, Carpinelli AR, Carvalho CR de O. Daily melatonin administration improves the insulin sensitivity and some of the early events in the insulin signaling pathway into hypothalamus, liver, skeletal muscle, and adipose tissue of 15 month-old obese rats. Diabetes. 2007 ; 56[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Pioglitazone treatment ameliorates insulin resistance in adult streptozotocin-induced diabetes rats in neonatal period by improving metabolic recovery of adipose tissue (2007)
Takada, Julie; Fonseca-Alaniz, Miriam H.; Campos, Tarcila B. Ferraz; Andreotti, Sandra ; Campana, Amanda B.; Lima, Fábio Bessa
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAKADA, Julie et al. Pioglitazone treatment ameliorates insulin resistance in adult streptozotocin-induced diabetes rats in neonatal period by improving metabolic recovery of adipose tissue. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2007
APA
Takada, J., Fonseca-Alaniz, M. H., Campos, T. B. F., Andreotti, S., Campana, A. B., & Lima, F. B. (2007). Pioglitazone treatment ameliorates insulin resistance in adult streptozotocin-induced diabetes rats in neonatal period by improving metabolic recovery of adipose tissue. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Takada J, Fonseca-Alaniz MH, Campos TBF, Andreotti S, Campana AB, Lima FB. Pioglitazone treatment ameliorates insulin resistance in adult streptozotocin-induced diabetes rats in neonatal period by improving metabolic recovery of adipose tissue. Diabetes. 2007 ; 56[citado 2024 nov. 18 ]
Vancouver
Takada J, Fonseca-Alaniz MH, Campos TBF, Andreotti S, Campana AB, Lima FB. Pioglitazone treatment ameliorates insulin resistance in adult streptozotocin-induced diabetes rats in neonatal period by improving metabolic recovery of adipose tissue. Diabetes. 2007 ; 56[citado 2024 nov. 18 ]
Artigo de periodico
S-Nitrosation of the insulin receptor, insulin receptor substrate 1, and protein Kinase B/Akt: A novel mechanism of insulin resistance (2005)
Carvalho-Filho, Marco A.; Ueno, Mirian; Hirabara, Sandro M.; Seabra, Amedea B.; Carvalheira, José B. C.; Oliveira, Marcelo G. de; Velloso, Lício Augusto; Curi, Rui; Saad, Mario J. A.
Source: Diabetes. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO-FILHO, Marco A. et al. S-Nitrosation of the insulin receptor, insulin receptor substrate 1, and protein Kinase B/Akt: A novel mechanism of insulin resistance. Diabetes, v. 54, p. 959-967, 2005Tradução . . Acesso em: 18 nov. 2024.
APA
Carvalho-Filho, M. A., Ueno, M., Hirabara, S. M., Seabra, A. B., Carvalheira, J. B. C., Oliveira, M. G. de, et al. (2005). S-Nitrosation of the insulin receptor, insulin receptor substrate 1, and protein Kinase B/Akt: A novel mechanism of insulin resistance. Diabetes, 54, 959-967.
NLM
Carvalho-Filho MA, Ueno M, Hirabara SM, Seabra AB, Carvalheira JBC, Oliveira MG de, Velloso LA, Curi R, Saad MJA. S-Nitrosation of the insulin receptor, insulin receptor substrate 1, and protein Kinase B/Akt: A novel mechanism of insulin resistance. Diabetes. 2005 ; 54 959-967.[citado 2024 nov. 18 ]
Vancouver
Carvalho-Filho MA, Ueno M, Hirabara SM, Seabra AB, Carvalheira JBC, Oliveira MG de, Velloso LA, Curi R, Saad MJA. S-Nitrosation of the insulin receptor, insulin receptor substrate 1, and protein Kinase B/Akt: A novel mechanism of insulin resistance. Diabetes. 2005 ; 54 959-967.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
LPS induces S-nitrosation of IR'beta'/IRS-1 and Akt and insulin resistance in muscle of mice (2005)
Carvalho-Filho, Marco A.; Ueno, Mirian; Hirabara, Sandro M.; Seabra, Amadea B.; Carvalheira, José B. C.; Oliveira, Marcelo G.; Velloso, Lício Augusto; Curi, Rui; Saad, Mario J. A.
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO-FILHO, Marco A. et al. LPS induces S-nitrosation of IR'beta'/IRS-1 and Akt and insulin resistance in muscle of mice. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2005
APA
Carvalho-Filho, M. A., Ueno, M., Hirabara, S. M., Seabra, A. B., Carvalheira, J. B. C., Oliveira, M. G., et al. (2005). LPS induces S-nitrosation of IR'beta'/IRS-1 and Akt and insulin resistance in muscle of mice. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Carvalho-Filho MA, Ueno M, Hirabara SM, Seabra AB, Carvalheira JBC, Oliveira MG, Velloso LA, Curi R, Saad MJA. LPS induces S-nitrosation of IR'beta'/IRS-1 and Akt and insulin resistance in muscle of mice. Diabetes. 2005 ; 54 A59 res. 238-OR.[citado 2024 nov. 18 ]
Vancouver
Carvalho-Filho MA, Ueno M, Hirabara SM, Seabra AB, Carvalheira JBC, Oliveira MG, Velloso LA, Curi R, Saad MJA. LPS induces S-nitrosation of IR'beta'/IRS-1 and Akt and insulin resistance in muscle of mice. Diabetes. 2005 ; 54 A59 res. 238-OR.[citado 2024 nov. 18 ]
Artigo de periodico
Mice with targeted disruption of the dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipose tissue lipogenesis and adaptive thermogenesis (2004)
Christoffolete, Marcelo A.; Linardi, Camila C. G.; Jesus, Lucia de; Ebina, Katia Naomi; Carvalho, Suzy D.; Ribeiro, Miriam O.; Rabelo, Rogerio; Curcio, Cyntia; Martins, Luciane; Kimura, Edna Teruko ; Bianco, Antonio Carlos
Source: Diabetes. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHRISTOFFOLETE, Marcelo A. et al. Mice with targeted disruption of the dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipose tissue lipogenesis and adaptive thermogenesis. Diabetes, v. 53, n. 3, p. 577-584, 2004Tradução . . Acesso em: 18 nov. 2024.
APA
Christoffolete, M. A., Linardi, C. C. G., Jesus, L. de, Ebina, K. N., Carvalho, S. D., Ribeiro, M. O., et al. (2004). Mice with targeted disruption of the dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipose tissue lipogenesis and adaptive thermogenesis. Diabetes, 53( 3), 577-584.
NLM
Christoffolete MA, Linardi CCG, Jesus L de, Ebina KN, Carvalho SD, Ribeiro MO, Rabelo R, Curcio C, Martins L, Kimura ET, Bianco AC. Mice with targeted disruption of the dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipose tissue lipogenesis and adaptive thermogenesis. Diabetes. 2004 ; 53( 3): 577-584.[citado 2024 nov. 18 ]
Vancouver
Christoffolete MA, Linardi CCG, Jesus L de, Ebina KN, Carvalho SD, Ribeiro MO, Rabelo R, Curcio C, Martins L, Kimura ET, Bianco AC. Mice with targeted disruption of the dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipose tissue lipogenesis and adaptive thermogenesis. Diabetes. 2004 ; 53( 3): 577-584.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
The insulin-induced leptin biosynthesis in isolated adipocytes aredependent on melatonin through specific 'MT IND.1' receptors (2004)
Alonso-Vale, Maria Isabel C.; Andreotti, Sandra ; Cipolla Neto, José; Lima, Fábio Bessa
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALONSO-VALE, Maria Isabel C. et al. The insulin-induced leptin biosynthesis in isolated adipocytes aredependent on melatonin through specific 'MT IND.1' receptors. Diabetes. Bethesda: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2004
APA
Alonso-Vale, M. I. C., Andreotti, S., Cipolla Neto, J., & Lima, F. B. (2004). The insulin-induced leptin biosynthesis in isolated adipocytes aredependent on melatonin through specific 'MT IND.1' receptors. Diabetes. Bethesda: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Alonso-Vale MIC, Andreotti S, Cipolla Neto J, Lima FB. The insulin-induced leptin biosynthesis in isolated adipocytes aredependent on melatonin through specific 'MT IND.1' receptors. Diabetes. 2004 ; 53 A95 res. 401-P.[citado 2024 nov. 18 ]
Vancouver
Alonso-Vale MIC, Andreotti S, Cipolla Neto J, Lima FB. The insulin-induced leptin biosynthesis in isolated adipocytes aredependent on melatonin through specific 'MT IND.1' receptors. Diabetes. 2004 ; 53 A95 res. 401-P.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Effect of food restriction schedule training on the insulin signalling transduction and GLUT4 gene expression (2004)
Sumida, Doris H.; Monteiro, Alexandre O.; Onishi, Mary E.; Brogin, Fabiana F.; Carvalho, Carla Roberta de Oliveira ; Okamoto, Maristela M.; Schaan, Beatriz D.; Machado, Ubiratan Fabres
Source: Diabetes. Conference titles: Scientific Sessions of American Diabetes Association. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SUMIDA, Doris H. et al. Effect of food restriction schedule training on the insulin signalling transduction and GLUT4 gene expression. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2004
APA
Sumida, D. H., Monteiro, A. O., Onishi, M. E., Brogin, F. F., Carvalho, C. R. de O., Okamoto, M. M., et al. (2004). Effect of food restriction schedule training on the insulin signalling transduction and GLUT4 gene expression. Diabetes. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Sumida DH, Monteiro AO, Onishi ME, Brogin FF, Carvalho CR de O, Okamoto MM, Schaan BD, Machado UF. Effect of food restriction schedule training on the insulin signalling transduction and GLUT4 gene expression. Diabetes. 2004 ; 53 A305 res. 1258-P.[citado 2024 nov. 18 ]
Vancouver
Sumida DH, Monteiro AO, Onishi ME, Brogin FF, Carvalho CR de O, Okamoto MM, Schaan BD, Machado UF. Effect of food restriction schedule training on the insulin signalling transduction and GLUT4 gene expression. Diabetes. 2004 ; 53 A305 res. 1258-P.[citado 2024 nov. 18 ]
Artigo de periodico
Improvement of the mitochondrial antioxidant defense status prevents cytokine-induced nuclear factor-kB activation in insulin-producing cells (2003)
Azevedo-Martins, Anna Karenina; Lortz, Stephan; Lenzen, Sigurd; Curi, Rui; Eizirik, Decio L.; Tiedge, Markus
Source: Diabetes. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AZEVEDO-MARTINS, Anna Karenina et al. Improvement of the mitochondrial antioxidant defense status prevents cytokine-induced nuclear factor-kB activation in insulin-producing cells. Diabetes, v. 52, p. 93-101, 2003Tradução . . Acesso em: 18 nov. 2024.
APA
Azevedo-Martins, A. K., Lortz, S., Lenzen, S., Curi, R., Eizirik, D. L., & Tiedge, M. (2003). Improvement of the mitochondrial antioxidant defense status prevents cytokine-induced nuclear factor-kB activation in insulin-producing cells. Diabetes, 52, 93-101.
NLM
Azevedo-Martins AK, Lortz S, Lenzen S, Curi R, Eizirik DL, Tiedge M. Improvement of the mitochondrial antioxidant defense status prevents cytokine-induced nuclear factor-kB activation in insulin-producing cells. Diabetes. 2003 ; 52 93-101.[citado 2024 nov. 18 ]
Vancouver
Azevedo-Martins AK, Lortz S, Lenzen S, Curi R, Eizirik DL, Tiedge M. Improvement of the mitochondrial antioxidant defense status prevents cytokine-induced nuclear factor-kB activation in insulin-producing cells. Diabetes. 2003 ; 52 93-101.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Rebaudioside A, a glicoside of the Stevia Rebaudiana, stimulates insulin secretion in rat isolated pancreatic islets (2003)
Costa, Cecilia; Costa, Silvio; Peres, Sidney; Rocha, Marlene; Dacomi, Antonio; Piccinato, Cecilia; Carpinelli, Angelo Rafael ; Lima, Fabio Bessa
Source: Diabetes. Conference titles: Scientific Sessions. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Cecilia et al. Rebaudioside A, a glicoside of the Stevia Rebaudiana, stimulates insulin secretion in rat isolated pancreatic islets. Diabetes. Alexandria: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2003
APA
Costa, C., Costa, S., Peres, S., Rocha, M., Dacomi, A., Piccinato, C., et al. (2003). Rebaudioside A, a glicoside of the Stevia Rebaudiana, stimulates insulin secretion in rat isolated pancreatic islets. Diabetes. Alexandria: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Costa C, Costa S, Peres S, Rocha M, Dacomi A, Piccinato C, Carpinelli AR, Lima FB. Rebaudioside A, a glicoside of the Stevia Rebaudiana, stimulates insulin secretion in rat isolated pancreatic islets. Diabetes. 2003 ; 52 A370.[citado 2024 nov. 18 ]
Vancouver
Costa C, Costa S, Peres S, Rocha M, Dacomi A, Piccinato C, Carpinelli AR, Lima FB. Rebaudioside A, a glicoside of the Stevia Rebaudiana, stimulates insulin secretion in rat isolated pancreatic islets. Diabetes. 2003 ; 52 A370.[citado 2024 nov. 18 ]

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