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Artigo de periodico
Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation (2021)
Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner Souza; Veras, Flávio Protásio ; Sônego, Fabiane; Nascimento, Daniele C.; Gonçalves, Augusto Velozo ; Martins, Timna Varela; Morelo, David Fernando Colon ; Borges, Vanessa F.; Brauer, Verônica Soares; Damasceno, Luis Eduardo Alves ; Silva, Katiussia P.; Toller-kawahisa, Juliana Escher; Batah, Sabrina Setembre ; Souza, Ana Letícia Júlio de; Monteiro, Valter Vinícius Silva; Oliveira, Antonio Edson Rocha; Donate, Paula Barbim; Zoppi, Daniel; Borges, Marcos de Carvalho ; Almeida, Fausto Bruno dos Reis ; Nakaya, Helder Takashi Imoto ; Fabro, Alexandre Todorovic ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Zamboni, Dario Simões ; Cunha, Fernando de Queiroz
Source: Blood. Unidades: FCFRP, FMRP, FCF
Subjects: FAGÓCITOS, GRANULÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Camila Meirelles de Souza et al. Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation. Blood, v. 138, n. 25, p. 2702-2713, 2021Tradução . . Disponível em: https://doi.org/10.1182/blood.2021011525. Acesso em: 02 nov. 2024.
APA
Silva, C. M. de S., Wanderley, C. W. S., Veras, F. P., Sônego, F., Nascimento, D. C., Gonçalves, A. V., et al. (2021). Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation. Blood, 138( 25), 2702-2713. doi:10.1182/blood.2021011525
NLM
Silva CM de S, Wanderley CWS, Veras FP, Sônego F, Nascimento DC, Gonçalves AV, Martins TV, Morelo DFC, Borges VF, Brauer VS, Damasceno LEA, Silva KP, Toller-kawahisa JE, Batah SS, Souza ALJ de, Monteiro VVS, Oliveira AER, Donate PB, Zoppi D, Borges M de C, Almeida FB dos R, Nakaya HTI, Fabro AT, Cunha TM, Alves Filho JCF, Zamboni DS, Cunha F de Q. Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation [Internet]. Blood. 2021 ; 138( 25): 2702-2713.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1182/blood.2021011525
Vancouver
Silva CM de S, Wanderley CWS, Veras FP, Sônego F, Nascimento DC, Gonçalves AV, Martins TV, Morelo DFC, Borges VF, Brauer VS, Damasceno LEA, Silva KP, Toller-kawahisa JE, Batah SS, Souza ALJ de, Monteiro VVS, Oliveira AER, Donate PB, Zoppi D, Borges M de C, Almeida FB dos R, Nakaya HTI, Fabro AT, Cunha TM, Alves Filho JCF, Zamboni DS, Cunha F de Q. Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation [Internet]. Blood. 2021 ; 138( 25): 2702-2713.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1182/blood.2021011525
Artigo de periodico
SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 (2019)
Wong, Deysi Viviana T.; Ribeiro Filho, Helder Veras; Wanderley, Carlos Wagner Souza; Leite, Caio Abner Vitorino Gonçalves; Lima, Jonilson Berlink; Assef, Alexia Nathália Brígido; Cajado, Aurilene Gomes; Batista, Gabriela Loiola Ponte; González, Rafael Holanda; Silva, Karla Oliveira; Borges, Luis Philipi Carvalho; Alencar, Nylane Maria Nunes de; Wilke, Diego Veras; Cunha, Thiago Mattar ; Figueira, Ana Carolina Migliorini; Cunha, Fernando de Queiroz ; Lima Júnior, Roberto César P.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: INFLAMAÇÃO, FÁRMACOS, RECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WONG, Deysi Viviana T. et al. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4. Cancer Chemotherapy and Pharmacology, v. 84, n. 2, p. 287-298, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00280-019-03844-z. Acesso em: 02 nov. 2024.
APA
Wong, D. V. T., Ribeiro Filho, H. V., Wanderley, C. W. S., Leite, C. A. V. G., Lima, J. B., Assef, A. N. B., et al. (2019). SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4. Cancer Chemotherapy and Pharmacology, 84( 2), 287-298. doi:10.1007/s00280-019-03844-z
NLM
Wong DVT, Ribeiro Filho HV, Wanderley CWS, Leite CAVG, Lima JB, Assef ANB, Cajado AG, Batista GLP, González RH, Silva KO, Borges LPC, Alencar NMN de, Wilke DV, Cunha TM, Figueira ACM, Cunha F de Q, Lima Júnior RCP. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 [Internet]. Cancer Chemotherapy and Pharmacology. 2019 ; 84( 2): 287-298.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00280-019-03844-z
Vancouver
Wong DVT, Ribeiro Filho HV, Wanderley CWS, Leite CAVG, Lima JB, Assef ANB, Cajado AG, Batista GLP, González RH, Silva KO, Borges LPC, Alencar NMN de, Wilke DV, Cunha TM, Figueira ACM, Cunha F de Q, Lima Júnior RCP. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 [Internet]. Cancer Chemotherapy and Pharmacology. 2019 ; 84( 2): 287-298.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00280-019-03844-z
Artigo de periodico
Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide (2018)
Dornelas-Filho, Amílcar Figueiredo; Pereira, Venúcia Bruna Magalhães; Wong, Deysi Viviana T.; Nobre, Lívia Maria Soares; Melo, Anielle Torres; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner Souza; Nour, Mariana Lima; Araújo, Lis Caetano Nobrega Costa; Silva, Renan Oliveira; Pinto, Francisco Maxwell Martins; Bingana, Rudy Diavila; Souza, Marcellus Henrique Loiola Ponte de; Alencar, Nylane Maria Nunes; Silva, Paulo Goberlânio B.; Alves, Ana Paula Negreiros Nunes; Almeida, Paulo Roberto Carvalho; Cunha, Fernando de Queiroz; Lima Júnior, Roberto César P.
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS, CISTITE, HEMORRAGIA, NEUTRÓFILOS, POLISSACARÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DORNELAS-FILHO, Amílcar Figueiredo et al. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide. International Immunopharmacology, v. 62, p. 96-108, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.06.031. Acesso em: 02 nov. 2024.
APA
Dornelas-Filho, A. F., Pereira, V. B. M., Wong, D. V. T., Nobre, L. M. S., Melo, A. T., Silva, C. M. de S., et al. (2018). Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide. International Immunopharmacology, 62, 96-108. doi:10.1016/j.intimp.2018.06.031
NLM
Dornelas-Filho AF, Pereira VBM, Wong DVT, Nobre LMS, Melo AT, Silva CM de S, Wanderley CWS, Nour ML, Araújo LCNC, Silva RO, Pinto FMM, Bingana RD, Souza MHLP de, Alencar NMN, Silva PGB, Alves APNN, Almeida PRC, Cunha F de Q, Lima Júnior RCP. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide [Internet]. International Immunopharmacology. 2018 ; 62 96-108.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.intimp.2018.06.031
Vancouver
Dornelas-Filho AF, Pereira VBM, Wong DVT, Nobre LMS, Melo AT, Silva CM de S, Wanderley CWS, Nour ML, Araújo LCNC, Silva RO, Pinto FMM, Bingana RD, Souza MHLP de, Alencar NMN, Silva PGB, Alves APNN, Almeida PRC, Cunha F de Q, Lima Júnior RCP. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide [Internet]. International Immunopharmacology. 2018 ; 62 96-108.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.intimp.2018.06.031

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