Collins, Stephan C.; Hoppa, Michael B.; Walker, Jonathan N.; Amisten, Stefan; Abdulkader, Fernando Rodrigues de Moraes 
; Bengtsson, Martin; Fearnside, Jane; Ramracheya, Reshma; Toye, Ayo A.; Zhang, Quan; Clark, Anne; Gauguier, Dominique; Rorsman, Patrik
Source: Diabetes. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
COLLINS, Stephan C. et al. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, v. 59, n. 5, p. 1192-1201, 2010Tradução . . Acesso em: 13 nov. 2024.APA
Collins, S. C., Hoppa, M. B., Walker, J. N., Amisten, S., Abdulkader, F. R. de M., Bengtsson, M., et al. (2010). Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, 59( 5), 1192-1201.NLM
Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 nov. 13 ]Vancouver
Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 nov. 13 ]
