Filtros : "He, G." "PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES" Removidos: "Zatz, Mayana" "Inglaterra" "Financiamento CNPq" Limpar

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  • Source: Arteriosclerosis, Thrombosis, and Vascular Biology. Unidade: ICB

    Assunto: FARMACOLOGIA

    How to cite
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    • ABNT

      MONTEZANO, A. C. et al. Aldosterone and angiotensin II synergistically stimulate migration in vascular smooth muscle cells through c-Src-regulated redox-sensitive RhoA pathways. Arteriosclerosis, Thrombosis, and Vascular Biology, v. 28, n. 8, p. 1511-1518, 2008Tradução . . Acesso em: 10 ago. 2024.
    • APA

      Montezano, A. C., Callera, G. E., Yogi, A., He, Y., Passaglia, R. de C. A. T., He, G., et al. (2008). Aldosterone and angiotensin II synergistically stimulate migration in vascular smooth muscle cells through c-Src-regulated redox-sensitive RhoA pathways. Arteriosclerosis, Thrombosis, and Vascular Biology, 28( 8), 1511-1518.
    • NLM

      Montezano AC, Callera GE, Yogi A, He Y, Passaglia R de CAT, He G, Schiffrin EL, Touyz RM. Aldosterone and angiotensin II synergistically stimulate migration in vascular smooth muscle cells through c-Src-regulated redox-sensitive RhoA pathways. Arteriosclerosis, Thrombosis, and Vascular Biology. 2008 ; 28( 8): 1511-1518.[citado 2024 ago. 10 ]
    • Vancouver

      Montezano AC, Callera GE, Yogi A, He Y, Passaglia R de CAT, He G, Schiffrin EL, Touyz RM. Aldosterone and angiotensin II synergistically stimulate migration in vascular smooth muscle cells through c-Src-regulated redox-sensitive RhoA pathways. Arteriosclerosis, Thrombosis, and Vascular Biology. 2008 ; 28( 8): 1511-1518.[citado 2024 ago. 10 ]
  • Source: Journal of Cardiovascular Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

    How to cite
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    • ABNT

      PASSAGLIA, Rita de Cassia Aleixo Tostes et al. Endotheli a receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive. Journal of Cardiovascular Pharmacology, v. 39, p. 892-900, 2002Tradução . . Acesso em: 10 ago. 2024.
    • APA

      Passaglia, R. de C. A. T., Touyz, R. M., He, G., Ammarguellat, F., & Schiffrin, E. L. (2002). Endotheli a receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive. Journal of Cardiovascular Pharmacology, 39, 892-900.
    • NLM

      Passaglia R de CAT, Touyz RM, He G, Ammarguellat F, Schiffrin EL. Endotheli a receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive. Journal of Cardiovascular Pharmacology. 2002 ; 39 892-900.[citado 2024 ago. 10 ]
    • Vancouver

      Passaglia R de CAT, Touyz RM, He G, Ammarguellat F, Schiffrin EL. Endotheli a receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive. Journal of Cardiovascular Pharmacology. 2002 ; 39 892-900.[citado 2024 ago. 10 ]
  • Source: Clinical Science. Unidade: ICB

    Assunto: FARMACOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      PASSAGLIA, Rita de Cassia Aleixo Tostes et al. Contribution of endothelin-I to renal activator protein-I activation and macrophage infiltration in aldosterone-induced hypertension. Clinical Science, v. 103, p. 25S-30S, 2002Tradução . . Disponível em: https://doi.org/10.1042/cs103s025s. Acesso em: 10 ago. 2024.
    • APA

      Passaglia, R. de C. A. T., Touyz, R. M., He, G., Chen, X., & Schiffrin, E. L. (2002). Contribution of endothelin-I to renal activator protein-I activation and macrophage infiltration in aldosterone-induced hypertension. Clinical Science, 103, 25S-30S. doi:10.1042/cs103s025s
    • NLM

      Passaglia R de CAT, Touyz RM, He G, Chen X, Schiffrin EL. Contribution of endothelin-I to renal activator protein-I activation and macrophage infiltration in aldosterone-induced hypertension [Internet]. Clinical Science. 2002 ; 103 25S-30S.[citado 2024 ago. 10 ] Available from: https://doi.org/10.1042/cs103s025s
    • Vancouver

      Passaglia R de CAT, Touyz RM, He G, Chen X, Schiffrin EL. Contribution of endothelin-I to renal activator protein-I activation and macrophage infiltration in aldosterone-induced hypertension [Internet]. Clinical Science. 2002 ; 103 25S-30S.[citado 2024 ago. 10 ] Available from: https://doi.org/10.1042/cs103s025s
  • Source: Journal of Cardiovascular Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      PASSAGLIA, Rita de Cassia Aleixo Tostes et al. Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology, v. 39, n. 6, p. 892-900, 2002Tradução . . Acesso em: 10 ago. 2024.
    • APA

      Passaglia, R. de C. A. T., Touyz, R. M., He, G., Ammarguellat, F., & Schiffrin, E. L. (2002). Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology, 39( 6), 892-900.
    • NLM

      Passaglia R de CAT, Touyz RM, He G, Ammarguellat F, Schiffrin EL. Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology. 2002 ; 39( 6): 892-900.[citado 2024 ago. 10 ]
    • Vancouver

      Passaglia R de CAT, Touyz RM, He G, Ammarguellat F, Schiffrin EL. Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology. 2002 ; 39( 6): 892-900.[citado 2024 ago. 10 ]

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