Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats (2002)
- Authors:
- Autor USP: PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - ICB
- Unidade: ICB
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2002
- Source:
- Título do periódico: Journal of Cardiovascular Pharmacology
- ISSN: 0160-2446
- Volume/Número/Paginação/Ano: v. 39, n. 6, p. 892-900, 2002
-
ABNT
PASSAGLIA, Rita de Cassia Aleixo Tostes et al. Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology, v. 39, n. 6, p. 892-900, 2002Tradução . . Acesso em: 19 abr. 2024. -
APA
Passaglia, R. de C. A. T., Touyz, R. M., He, G., Ammarguellat, F., & Schiffrin, E. L. (2002). Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology, 39( 6), 892-900. -
NLM
Passaglia R de CAT, Touyz RM, He G, Ammarguellat F, Schiffrin EL. Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology. 2002 ; 39( 6): 892-900.[citado 2024 abr. 19 ] -
Vancouver
Passaglia R de CAT, Touyz RM, He G, Ammarguellat F, Schiffrin EL. Endothelin A receptor blockade decreases expression of growth factors and collagen and improves matrix metalloproteinase-2 activity in kidneys from stroke-prone spontaneously hypertensive rats. Journal of Cardiovascular Pharmacology. 2002 ; 39( 6): 892-900.[citado 2024 abr. 19 ] - ET-1-induced oxidative stress in doca-salt hypertension involves NAD(P)H oxidase-independent mechanisms
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