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Artigo de periodico
The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress (2021)
Ferreira Junior, Nilson Carlos; Pereira, Maurício dos Santos ; Francis, Nour; Ramirez, Paola; Martorell, Paula; González-Lizarraga, Florencia; Figadère, Bruno; Chehin, Rosana; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick Pierre
Source: Cells. Unidade: FORP
Subjects: ESTRESSE OXIDATIVO, GLICOSE, METABOLISMO, ANTIBIÓTICOS, AGENTES ANTIMICROBIANOS, FARMACOLOGIA EXPERIMENTAL
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ABNT
FERREIRA JUNIOR, Nilson Carlos et al. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, v. 10, n. 8, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10082163. Acesso em: 07 nov. 2024.
APA
Ferreira Junior, N. C., Pereira, M. dos S., Francis, N., Ramirez, P., Martorell, P., González-Lizarraga, F., et al. (2021). The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, 10( 8), 1-16. doi:10.3390/cells10082163
NLM
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells10082163
Vancouver
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells10082163
Artigo de periodico
Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease (2021)
Pereira, Maurício dos Santos ; Abreu, Gabriel Henrique Dias; Rocca, Jeremy; Hamadat, Sabah; Raisman-Vozari, Rita; Michel, Patrick Pierre; Del Bel, Elaine Aparecida
Source: Frontiers in Pharmacology. Unidade: FORP
Subjects: ASTROCITOMA, CANABINOIDES, GLUTAMATOS
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ABNT
PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 07 nov. 2024.
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Del Bel, E. A. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Artigo de periodico
Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption (2020)
Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick P.
Source: Glia. Unidades: FMRP, FORP
Subjects: CANABINOIDES, GLICOSE, ESTRESSE OXIDATIVO
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ABNT
PEREIRA, Maurício dos Santos et al. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, v. 68, n. 3, p. 561-573, 2020Tradução . . Disponível em: https://doi.org/10.1002/glia.23738. Acesso em: 07 nov. 2024.
APA
Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., Raisman-Vozari, R., & Michel, P. P. (2020). Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, 68( 3), 561-573. doi:10.1002/glia.23738
NLM
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1002/glia.23738
Vancouver
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1002/glia.23738
Artigo de periodico
CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects (2020)
González-Lizárraga, Florencia; Ploper, Diego; Ávila, César L.; Socías, Sergio B.; Pereira, Mauricio dos Santos; Machín, Belén; Del Bel, Elaine Aparecida ; Michel, Patrick Pierre; Pietrasanta, Lía I.; Raisman-Vozari, Rita; Chehín, Rosana
Source: Scientific Reports. Unidade: FORP
Subjects: REPOSICIONAMENTO DE FÁRMACOS, INFLAMAÇÃO, DOENÇA DE PARKINSON, FARMACOTERAPIA
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ABNT
GONZÁLEZ-LIZÁRRAGA, Florencia et al. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects. Scientific Reports, v. 10, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-76927-0. Acesso em: 07 nov. 2024.
APA
González-Lizárraga, F., Ploper, D., Ávila, C. L., Socías, S. B., Pereira, M. dos S., Machín, B., et al. (2020). CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects. Scientific Reports, 10( 1). doi:10.1038/s41598-020-76927-0
NLM
González-Lizárraga F, Ploper D, Ávila CL, Socías SB, Pereira M dos S, Machín B, Del Bel EA, Michel PP, Pietrasanta LI, Raisman-Vozari R, Chehín R. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/s41598-020-76927-0
Vancouver
González-Lizárraga F, Ploper D, Ávila CL, Socías SB, Pereira M dos S, Machín B, Del Bel EA, Michel PP, Pietrasanta LI, Raisman-Vozari R, Chehín R. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/s41598-020-76927-0
Artigo de periodico
Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons (2016)
Salum, Cristiane; Schmidt, Fanny; Michel, Patrick P.; Del Bel, Elaine Aparecida; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidade: FORP
Subjects: ÓXIDO NÍTRICO, NEUROENDOCRINOLOGIA
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ABNT
SALUM, Cristiane et al. Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons. Neurotoxicity Research, v. 29, n. 1, p. 92–104, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9562-8. Acesso em: 07 nov. 2024.
APA
Salum, C., Schmidt, F., Michel, P. P., Del Bel, E. A., & Raisman-Vozari, R. (2016). Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons. Neurotoxicity Research, 29( 1), 92–104. doi:10.1007/s12640-015-9562-8
NLM
Salum C, Schmidt F, Michel PP, Del Bel EA, Raisman-Vozari R. Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons [Internet]. Neurotoxicity Research. 2016 ; 29( 1): 92–104.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9562-8
Vancouver
Salum C, Schmidt F, Michel PP, Del Bel EA, Raisman-Vozari R. Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons [Internet]. Neurotoxicity Research. 2016 ; 29( 1): 92–104.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9562-8
Artigo de periodico
CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration (2016)
Tristão, Fabrine Sales Massafera; Lazzarini, Márcio; Martin, Sabine; Amar, Majid; Stühmer, Walter; Kirchhoff, Frank; Gomes, Lucas Araújo Caldi; Lanfumey, Laurance; Prediger, Rui D.; Sepulveda, Julia E.; Del Bel, Elaine Aparecida; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, INFLAMAÇÃO, DEGENERAÇÃO NEURAL
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ABNT
TRISTÃO, Fabrine Sales Massafera et al. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, v. 29, n. 3, p. 364-380, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9557-5. Acesso em: 07 nov. 2024.
APA
Tristão, F. S. M., Lazzarini, M., Martin, S., Amar, M., Stühmer, W., Kirchhoff, F., et al. (2016). CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, 29( 3), 364-380. doi:10.1007/s12640-015-9557-5
NLM
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Vancouver
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Artigo de periodico
Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways (2016)
Santa-Cecília, Flávia V.; Socias, Benjamin; Ouidja, Mohand O.; Sepulveda-Diaz, Julia E.; Acuña, Leonardo; Silva, Rangel L.; Michel, Patrick P.; Del Bel, Elaine Aparecida; Cunha, Thiago Mattar; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidades: FORP, FMRP
Subjects: DOENÇAS NEURODEGENERATIVAS, CITOCINAS, DOENÇA DE PARKINSON, FÁRMACOS
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ABNT
SANTA-CECÍLIA, Flávia V. et al. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, v. 29, n. 4, p. 447-459, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9592-2. Acesso em: 07 nov. 2024.
APA
Santa-Cecília, F. V., Socias, B., Ouidja, M. O., Sepulveda-Diaz, J. E., Acuña, L., Silva, R. L., et al. (2016). Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, 29( 4), 447-459. doi:10.1007/s12640-015-9592-2
NLM
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Vancouver
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Artigo de periodico
Glial activation is associated with l-DOPA induced dyskinesia and blocked by a nitric oxide synthase inhibitor in a rat model of Parkinson's disease (2015)
Bortolanza, Mariza; Cavalcanti-Kwiatkoski, Roberta; Padovan Neto, Fernando Eduardo; Silva, Célia Aparecida da; Mitkovski, Miso; Raisman-Vozari, Rita; Del Bel, Elaine Aparecida
Source: Neurobiology of Disease. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, DISCINESIAS, ÓXIDO NÍTRICO
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ABNT
BORTOLANZA, Mariza et al. Glial activation is associated with l-DOPA induced dyskinesia and blocked by a nitric oxide synthase inhibitor in a rat model of Parkinson's disease. Neurobiology of Disease, v. 73, p. 377–387, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.nbd.2014.10.017. Acesso em: 07 nov. 2024.
APA
Bortolanza, M., Cavalcanti-Kwiatkoski, R., Padovan Neto, F. E., Silva, C. A. da, Mitkovski, M., Raisman-Vozari, R., & Del Bel, E. A. (2015). Glial activation is associated with l-DOPA induced dyskinesia and blocked by a nitric oxide synthase inhibitor in a rat model of Parkinson's disease. Neurobiology of Disease, 73, 377–387. doi:10.1016/j.nbd.2014.10.017
NLM
Bortolanza M, Cavalcanti-Kwiatkoski R, Padovan Neto FE, Silva CA da, Mitkovski M, Raisman-Vozari R, Del Bel EA. Glial activation is associated with l-DOPA induced dyskinesia and blocked by a nitric oxide synthase inhibitor in a rat model of Parkinson's disease [Internet]. Neurobiology of Disease. 2015 ; 73 377–387.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.nbd.2014.10.017
Vancouver
Bortolanza M, Cavalcanti-Kwiatkoski R, Padovan Neto FE, Silva CA da, Mitkovski M, Raisman-Vozari R, Del Bel EA. Glial activation is associated with l-DOPA induced dyskinesia and blocked by a nitric oxide synthase inhibitor in a rat model of Parkinson's disease [Internet]. Neurobiology of Disease. 2015 ; 73 377–387.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.nbd.2014.10.017
Artigo de periodico
Are cyclooxygenase-2 and nitric oxide involved in the dyskinesia of Parkinson's disease induced by L-DOPA ? (2015)
Bortolanza, Mariza; Padovan-Neto, Fernando E.; Cavalcanti-Kiwiatkoski, Roberta; Santos-Pereira, Maurício dos; Mitkovski, Miso; Raisman-Vozari, Rita; Del Bel, Elaine Aparecida
Source: Royal Society of London. Philosophical Transactions B. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, DISCINESIAS, ÓXIDO NÍTRICO
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ABNT
BORTOLANZA, Mariza et al. Are cyclooxygenase-2 and nitric oxide involved in the dyskinesia of Parkinson's disease induced by L-DOPA ?. Royal Society of London. Philosophical Transactions B, v. 370, n. 1672, 2015Tradução . . Disponível em: https://doi.org/10.1098/rstb.2014.0190. Acesso em: 07 nov. 2024.
APA
Bortolanza, M., Padovan-Neto, F. E., Cavalcanti-Kiwiatkoski, R., Santos-Pereira, M. dos, Mitkovski, M., Raisman-Vozari, R., & Del Bel, E. A. (2015). Are cyclooxygenase-2 and nitric oxide involved in the dyskinesia of Parkinson's disease induced by L-DOPA ? Royal Society of London. Philosophical Transactions B, 370( 1672). doi:10.1098/rstb.2014.0190
NLM
Bortolanza M, Padovan-Neto FE, Cavalcanti-Kiwiatkoski R, Santos-Pereira M dos, Mitkovski M, Raisman-Vozari R, Del Bel EA. Are cyclooxygenase-2 and nitric oxide involved in the dyskinesia of Parkinson's disease induced by L-DOPA ? [Internet]. Royal Society of London. Philosophical Transactions B. 2015 ; 370( 1672):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1098/rstb.2014.0190
Vancouver
Bortolanza M, Padovan-Neto FE, Cavalcanti-Kiwiatkoski R, Santos-Pereira M dos, Mitkovski M, Raisman-Vozari R, Del Bel EA. Are cyclooxygenase-2 and nitric oxide involved in the dyskinesia of Parkinson's disease induced by L-DOPA ? [Internet]. Royal Society of London. Philosophical Transactions B. 2015 ; 370( 1672):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1098/rstb.2014.0190
Artigo de periodico
Nitric oxide, a new player in l-dopa-induced dyskinesia? (2015)
Del Bel, Elaine Aparecida; Padovan Neto, Fernando Eduardo; Bortolanza, Mariza; Tumas, Vitor; Aguiar Júnior, Aderbal S.; Raisman-Vozari, Rita; Prediger, Rui D.
Source: Frontiers in Bioscience. Unidades: FORP, FMRP
Subjects: ÓXIDO NÍTRICO (METABOLISMO), DISCINESIAS, DOENÇA DE PARKINSON
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ABNT
DEL BEL, Elaine Aparecida et al. Nitric oxide, a new player in l-dopa-induced dyskinesia?. Frontiers in Bioscience, v. 7, p. 193-221, 2015Tradução . . Disponível em: https://doi.org/10.2741/E726. Acesso em: 07 nov. 2024.
APA
Del Bel, E. A., Padovan Neto, F. E., Bortolanza, M., Tumas, V., Aguiar Júnior, A. S., Raisman-Vozari, R., & Prediger, R. D. (2015). Nitric oxide, a new player in l-dopa-induced dyskinesia? Frontiers in Bioscience, 7, 193-221. doi:10.2741/E726
NLM
Del Bel EA, Padovan Neto FE, Bortolanza M, Tumas V, Aguiar Júnior AS, Raisman-Vozari R, Prediger RD. Nitric oxide, a new player in l-dopa-induced dyskinesia? [Internet]. Frontiers in Bioscience. 2015 ; 7 193-221.[citado 2024 nov. 07 ] Available from: https://doi.org/10.2741/E726
Vancouver
Del Bel EA, Padovan Neto FE, Bortolanza M, Tumas V, Aguiar Júnior AS, Raisman-Vozari R, Prediger RD. Nitric oxide, a new player in l-dopa-induced dyskinesia? [Internet]. Frontiers in Bioscience. 2015 ; 7 193-221.[citado 2024 nov. 07 ] Available from: https://doi.org/10.2741/E726
Artigo de periodico
Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease (2014)
Tristão, Fabrine S. M.; Amar, Majid; Latrous, Ines; Del Bel, Elaine Aparecida; Prediger, Rui D.; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidade: FORP
Subjects: MODELOS ANIMAIS DE DOENÇAS, EXPOSIÇÃO AMBIENTAL, DOENÇAS NEURODEGENERATIVAS (PATOLOGIA)
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ABNT
TRISTÃO, Fabrine S. M. et al. Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease. Neurotoxicity Research, v. 25, n. 1, p. 24-32, 2014Tradução . . Disponível em: https://doi.org/10.1007/s12640-013-9401-8. Acesso em: 07 nov. 2024.
APA
Tristão, F. S. M., Amar, M., Latrous, I., Del Bel, E. A., Prediger, R. D., & Raisman-Vozari, R. (2014). Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease. Neurotoxicity Research, 25( 1), 24-32. doi:10.1007/s12640-013-9401-8
NLM
Tristão FSM, Amar M, Latrous I, Del Bel EA, Prediger RD, Raisman-Vozari R. Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease [Internet]. Neurotoxicity Research. 2014 ; 25( 1): 24-32.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-013-9401-8
Vancouver
Tristão FSM, Amar M, Latrous I, Del Bel EA, Prediger RD, Raisman-Vozari R. Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease [Internet]. Neurotoxicity Research. 2014 ; 25( 1): 24-32.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-013-9401-8
Artigo de periodico
Counteraction by nitric oxide synthase inhibitor of neurochemical alterations of dopaminergic system in 6-OHDA-lesioned rats under L-DOPA treatment (2014)
Del Bel, Elaine Aparecida; Padovan Neto, Fernando Eduardo; Szawka, Raphael Escorsim; Silva, Célia Aparecida da; Raisman-Vozari, Rita; Anselmo-Franci, Janete Aparecida; Romano Dutra, Angélica Caroline; Guimarães, Francisco Silveira
Source: Neurotoxicity Research. Unidades: FORP, FMRP
Subjects: ÓXIDO NÍTRICO, NEUROQUÍMICA (ALTERAÇÃO)
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ABNT
DEL BEL, Elaine Aparecida et al. Counteraction by nitric oxide synthase inhibitor of neurochemical alterations of dopaminergic system in 6-OHDA-lesioned rats under L-DOPA treatment. Neurotoxicity Research, v. 25, n. 1, p. 33-44, 2014Tradução . . Disponível em: https://doi.org/10.1007/s12640-013-9406-3. Acesso em: 07 nov. 2024.
APA
Del Bel, E. A., Padovan Neto, F. E., Szawka, R. E., Silva, C. A. da, Raisman-Vozari, R., Anselmo-Franci, J. A., et al. (2014). Counteraction by nitric oxide synthase inhibitor of neurochemical alterations of dopaminergic system in 6-OHDA-lesioned rats under L-DOPA treatment. Neurotoxicity Research, 25( 1), 33-44. doi:10.1007/s12640-013-9406-3
NLM
Del Bel EA, Padovan Neto FE, Szawka RE, Silva CA da, Raisman-Vozari R, Anselmo-Franci JA, Romano Dutra AC, Guimarães FS. Counteraction by nitric oxide synthase inhibitor of neurochemical alterations of dopaminergic system in 6-OHDA-lesioned rats under L-DOPA treatment [Internet]. Neurotoxicity Research. 2014 ; 25( 1): 33-44.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-013-9406-3
Vancouver
Del Bel EA, Padovan Neto FE, Szawka RE, Silva CA da, Raisman-Vozari R, Anselmo-Franci JA, Romano Dutra AC, Guimarães FS. Counteraction by nitric oxide synthase inhibitor of neurochemical alterations of dopaminergic system in 6-OHDA-lesioned rats under L-DOPA treatment [Internet]. Neurotoxicity Research. 2014 ; 25( 1): 33-44.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-013-9406-3

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