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Artigo de periodico
Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats (2020)
Passaglia, Patrícia; Faim, Felipe de Lima; Batalhão, Marcelo Eduardo; Bendhack, Lusiane Maria ; Antunes-Rodrigues, José; Ulloa, Luis; Kanashiro, Alexandre; Cárnio, Evelin Capellari
Source: Brain, Behavior, and Immunity. Unidades: EERP, FCFRP, FMRP
Subjects: ANGIOTENSINAS, LIPOPOLISSACARÍDEOS, SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, HIPOTENSÃO
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PASSAGLIA, Patrícia et al. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats. Brain, Behavior, and Immunity, v. 88, p. 606-618, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2020.04.059. Acesso em: 10 nov. 2024.
APA
Passaglia, P., Faim, F. de L., Batalhão, M. E., Bendhack, L. M., Antunes-Rodrigues, J., Ulloa, L., et al. (2020). Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats. Brain, Behavior, and Immunity, 88, 606-618. doi:10.1016/j.bbi.2020.04.059
NLM
Passaglia P, Faim F de L, Batalhão ME, Bendhack LM, Antunes-Rodrigues J, Ulloa L, Kanashiro A, Cárnio EC. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats [Internet]. Brain, Behavior, and Immunity. 2020 ; 88 606-618.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbi.2020.04.059
Vancouver
Passaglia P, Faim F de L, Batalhão ME, Bendhack LM, Antunes-Rodrigues J, Ulloa L, Kanashiro A, Cárnio EC. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats [Internet]. Brain, Behavior, and Immunity. 2020 ; 88 606-618.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbi.2020.04.059
Artigo de periodico
Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation (2020)
Amorim, Mateus Ramos; Deus, Júnia Lara de; Pereira, Camila André; Silva, Luiz Eduardo Virgilio da ; Borges, Gabriela Silva; Ferreira, Nathanne dos Santos; Batalhão, Marcelo Eduardo; Antunes-Rodrigues, José; Cárnio, Evelin Capellari ; Tostes, Rita de Cássia Aleixo ; Branco, Luiz Guilherme de Siqueira
Source: Scientific Reports. Unidades: EERP, FMRP, FORP, EEFE
Subjects: BARORREFLEXO, IMUNOLOGIA, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, HEMODINÂMICA, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS
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AMORIM, Mateus Ramos et al. Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation. Scientific Reports, v. 10, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-63949-x. Acesso em: 10 nov. 2024.
APA
Amorim, M. R., Deus, J. L. de, Pereira, C. A., Silva, L. E. V. da, Borges, G. S., Ferreira, N. dos S., et al. (2020). Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation. Scientific Reports, 10( 1). doi:10.1038/s41598-020-63949-x
NLM
Amorim MR, Deus JL de, Pereira CA, Silva LEV da, Borges GS, Ferreira N dos S, Batalhão ME, Antunes-Rodrigues J, Cárnio EC, Tostes R de CA, Branco LG de S. Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-020-63949-x
Vancouver
Amorim MR, Deus JL de, Pereira CA, Silva LEV da, Borges GS, Ferreira N dos S, Batalhão ME, Antunes-Rodrigues J, Cárnio EC, Tostes R de CA, Branco LG de S. Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-020-63949-x
Artigo de periodico
Increased hypothalamic hydrogen sulphide contributes to endotoxin tolerance by down-modulating PGE2 production (2020)
Santos, Bruna Maitan ; Francescato, Heloisa Della Coletta; Turcato, Flávia Corrêa ; Antunes-Rodrigues, José ; Coimbra, Terezila Machado ; Branco, Luiz Guilherme de Siqueira
Source: Acta Physiologica. Unidades: FMRP, FORP
Subjects: HORMÔNIOS DO CÓRTEX ADRENAL, CITOCINAS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS
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SANTOS, Bruna Maitan et al. Increased hypothalamic hydrogen sulphide contributes to endotoxin tolerance by down-modulating PGE2 production. Acta Physiologica, v. 228, n. 3, p. [12] , 2020Tradução . . Disponível em: https://doi.org/10.1111/apha.13373. Acesso em: 10 nov. 2024.
APA
Santos, B. M., Francescato, H. D. C., Turcato, F. C., Antunes-Rodrigues, J., Coimbra, T. M., & Branco, L. G. de S. (2020). Increased hypothalamic hydrogen sulphide contributes to endotoxin tolerance by down-modulating PGE2 production. Acta Physiologica, 228( 3), [12] . doi:10.1111/apha.13373
NLM
Santos BM, Francescato HDC, Turcato FC, Antunes-Rodrigues J, Coimbra TM, Branco LG de S. Increased hypothalamic hydrogen sulphide contributes to endotoxin tolerance by down-modulating PGE2 production [Internet]. Acta Physiologica. 2020 ; 228( 3): [12] .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/apha.13373
Vancouver
Santos BM, Francescato HDC, Turcato FC, Antunes-Rodrigues J, Coimbra TM, Branco LG de S. Increased hypothalamic hydrogen sulphide contributes to endotoxin tolerance by down-modulating PGE2 production [Internet]. Acta Physiologica. 2020 ; 228( 3): [12] .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/apha.13373
Artigo de periodico
Increase in hypothalamic AMPK phosphorylation induced by prolonged exposure to LPS involves ghrelin and CB1R signaling (2017)
Rivas, Priscila M. S.; Vechiato, Fernanda M. V.; Borges, Beatriz C.; Rorato, Rodrigo; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Hormones and Behavior. Unidade: FMRP
Subjects: CANABINOIDES, GRELINA, HIPOTÁLAMO, LIPOPOLISSACARÍDEOS, RECEPTORES, TRANSDUÇÃO DE SINAL CELULAR
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RIVAS, Priscila M. S. et al. Increase in hypothalamic AMPK phosphorylation induced by prolonged exposure to LPS involves ghrelin and CB1R signaling. Hormones and Behavior, v. 93, p. 166-174, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2017.05.018. Acesso em: 10 nov. 2024.
APA
Rivas, P. M. S., Vechiato, F. M. V., Borges, B. C., Rorato, R., Antunes-Rodrigues, J., & Elias, L. L. K. (2017). Increase in hypothalamic AMPK phosphorylation induced by prolonged exposure to LPS involves ghrelin and CB1R signaling. Hormones and Behavior, 93, 166-174. doi:10.1016/j.yhbeh.2017.05.018
NLM
Rivas PMS, Vechiato FMV, Borges BC, Rorato R, Antunes-Rodrigues J, Elias LLK. Increase in hypothalamic AMPK phosphorylation induced by prolonged exposure to LPS involves ghrelin and CB1R signaling [Internet]. Hormones and Behavior. 2017 ; 93 166-174.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.yhbeh.2017.05.018
Vancouver
Rivas PMS, Vechiato FMV, Borges BC, Rorato R, Antunes-Rodrigues J, Elias LLK. Increase in hypothalamic AMPK phosphorylation induced by prolonged exposure to LPS involves ghrelin and CB1R signaling [Internet]. Hormones and Behavior. 2017 ; 93 166-174.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.yhbeh.2017.05.018
Artigo de periodico
Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages (2017)
Flatow, Elizabeth A.; Komegae, Evilin N.; Fonseca, Monique T.; Brito, Camila F.; Musteata, Florin M.; Antunes-Rodrigues, José; Steiner, Alexandre Alarcon
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. Unidade: FMRP
Subjects: CITOCINAS, FEBRE, PRIVAÇÃO DE ALIMENTOS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, MACRÓFAGOS
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ABNT
FLATOW, Elizabeth A. et al. Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, v. 313, p. R572-582, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00171.2017. Acesso em: 10 nov. 2024.
APA
Flatow, E. A., Komegae, E. N., Fonseca, M. T., Brito, C. F., Musteata, F. M., Antunes-Rodrigues, J., & Steiner, A. A. (2017). Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 313, R572-582. doi:10.1152/ajpregu.00171.2017
NLM
Flatow EA, Komegae EN, Fonseca MT, Brito CF, Musteata FM, Antunes-Rodrigues J, Steiner AA. Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2017 ; 313 R572-582.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1152/ajpregu.00171.2017
Vancouver
Flatow EA, Komegae EN, Fonseca MT, Brito CF, Musteata FM, Antunes-Rodrigues J, Steiner AA. Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2017 ; 313 R572-582.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1152/ajpregu.00171.2017
Artigo de periodico
Central serotonin attenuates LPS-induced systemic inflammation (2017)
Mota, Clarissa M. D.; Santos, Caroline Rodrigues; Fernández, Rodrigo A. R.; Carolino, Ruither O. G.; Antunes-Rodrigues, José; Anselmo-Franci, Janete Aparecida; Branco, Luiz Guilherme de Siqueira
Source: Brain, Behavior, and Immunity. Unidades: FORP, FMRP
Subjects: ENDOTOXINAS, FEBRE, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, SEROTONINA
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ABNT
MOTA, Clarissa M. D. et al. Central serotonin attenuates LPS-induced systemic inflammation. Brain, Behavior, and Immunity, v. 66, p. 372-381, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2017.07.010. Acesso em: 10 nov. 2024.
APA
Mota, C. M. D., Santos, C. R., Fernández, R. A. R., Carolino, R. O. G., Antunes-Rodrigues, J., Anselmo-Franci, J. A., & Branco, L. G. de S. (2017). Central serotonin attenuates LPS-induced systemic inflammation. Brain, Behavior, and Immunity, 66, 372-381. doi:10.1016/j.bbi.2017.07.010
NLM
Mota CMD, Santos CR, Fernández RAR, Carolino ROG, Antunes-Rodrigues J, Anselmo-Franci JA, Branco LG de S. Central serotonin attenuates LPS-induced systemic inflammation [Internet]. Brain, Behavior, and Immunity. 2017 ; 66 372-381.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbi.2017.07.010
Vancouver
Mota CMD, Santos CR, Fernández RAR, Carolino ROG, Antunes-Rodrigues J, Anselmo-Franci JA, Branco LG de S. Central serotonin attenuates LPS-induced systemic inflammation [Internet]. Brain, Behavior, and Immunity. 2017 ; 66 372-381.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbi.2017.07.010
Artigo de periodico
LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes (2017)
Rorato, Rodrigo; Borges, Beatriz de Carvalho; Uchoa, Ernane Torres; Antunes-Rodrigues, José; Elias, Carol Fuzeti; Elias, Lucila Leico Kagohara
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: MODELOS ANIMAIS DE DOENÇAS, HIPOTÁLAMO, INFLAMAÇÃO, RESISTÊNCIA À INSULINA, LIPOPOLISSACARÍDEOS, PESOS E MEDIDAS CORPORAIS
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ABNT
RORATO, Rodrigo et al. LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes. International Journal of Molecular Sciences, v. 18, n. 7, 2017Tradução . . Disponível em: https://doi.org/10.3390/ijms18071431. Acesso em: 10 nov. 2024.
APA
Rorato, R., Borges, B. de C., Uchoa, E. T., Antunes-Rodrigues, J., Elias, C. F., & Elias, L. L. K. (2017). LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes. International Journal of Molecular Sciences, 18( 7). doi:10.3390/ijms18071431
NLM
Rorato R, Borges B de C, Uchoa ET, Antunes-Rodrigues J, Elias CF, Elias LLK. LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 7):[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/ijms18071431
Vancouver
Rorato R, Borges B de C, Uchoa ET, Antunes-Rodrigues J, Elias CF, Elias LLK. LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 7):[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/ijms18071431
Artigo de periodico
High-fat diet induces site-specific unresponsiveness to LPS-stimulated STAT3 activation in the hypothalamus (2014)
Borges, Beatriz de Carvalho; Rorato, Rodrigo; Uchôa, Ernane Torres; Marangon, Paula; Silva, Glauber S. F. da; Paula, Francisco Jose Albuquerque de; Branco, Luiz Guilherme de Siqueira; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidades: FORP, FMRP
Subjects: TEMPERATURA CORPORAL, GORDURAS NA DIETA, LIPOPOLISSACARÍDEOS, CALORÍMETROS
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ABNT
BORGES, Beatriz de Carvalho et al. High-fat diet induces site-specific unresponsiveness to LPS-stimulated STAT3 activation in the hypothalamus. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 306, n. 1, p. R34-R44, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00147.2013. Acesso em: 10 nov. 2024.
APA
Borges, B. de C., Rorato, R., Uchôa, E. T., Marangon, P., Silva, G. S. F. da, Paula, F. J. A. de, et al. (2014). High-fat diet induces site-specific unresponsiveness to LPS-stimulated STAT3 activation in the hypothalamus. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 306( 1), R34-R44. doi:10.1152/ajpregu.00147.2013
NLM
Borges B de C, Rorato R, Uchôa ET, Marangon P, Silva GSF da, Paula FJA de, Branco LG de S, Antunes-Rodrigues J, Elias LLK. High-fat diet induces site-specific unresponsiveness to LPS-stimulated STAT3 activation in the hypothalamus [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2014 ; 306( 1): R34-R44.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1152/ajpregu.00147.2013
Vancouver
Borges B de C, Rorato R, Uchôa ET, Marangon P, Silva GSF da, Paula FJA de, Branco LG de S, Antunes-Rodrigues J, Elias LLK. High-fat diet induces site-specific unresponsiveness to LPS-stimulated STAT3 activation in the hypothalamus [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2014 ; 306( 1): R34-R44.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1152/ajpregu.00147.2013
Trabalho de evento-resumo
Cocaine and amphetamine transcript (CART) and NPY mRNA expression in the arcuate nucleus and food intake after repeated exposure to endotoxin (2007)
Borges, Beatriz C.; Castro, Margaret; Antunes-Rodrigues, José; Elias, Carol Fuzeti; Elias, Lucila Leico Kagohara
Source: Program and Abstracts. Conference titles: The Endocrine Society's Annual Meeting (ENDO). Unidades: FMRP, ICB
Subjects: ENDOCRINOLOGIA, LIPOPOLISSACARÍDEOS, ENDOTOXINAS
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ABNT
BORGES, Beatriz C. et al. Cocaine and amphetamine transcript (CART) and NPY mRNA expression in the arcuate nucleus and food intake after repeated exposure to endotoxin. 2007, Anais.. Toronto: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2007. . Acesso em: 10 nov. 2024.
APA
Borges, B. C., Castro, M., Antunes-Rodrigues, J., Elias, C. F., & Elias, L. L. K. (2007). Cocaine and amphetamine transcript (CART) and NPY mRNA expression in the arcuate nucleus and food intake after repeated exposure to endotoxin. In Program and Abstracts. Toronto: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Borges BC, Castro M, Antunes-Rodrigues J, Elias CF, Elias LLK. Cocaine and amphetamine transcript (CART) and NPY mRNA expression in the arcuate nucleus and food intake after repeated exposure to endotoxin. Program and Abstracts. 2007 ;[citado 2024 nov. 10 ]
Vancouver
Borges BC, Castro M, Antunes-Rodrigues J, Elias CF, Elias LLK. Cocaine and amphetamine transcript (CART) and NPY mRNA expression in the arcuate nucleus and food intake after repeated exposure to endotoxin. Program and Abstracts. 2007 ;[citado 2024 nov. 10 ]
Artigo de periodico
Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats (2004)
Giusti-Paiva, Alexandre; Martinez, Maria Regina; Felix, Jorge Vinícius Cestari; Rocha, Maria José Alves da; Cárnio, Evelin Capellari ; Elias, Lucila Leico Kagohara; Antunes-Rodrigues, José
Source: SHOCK. Unidades: FORP, EERP, FMRP
Subjects: SEPSE, PRESSÃO SANGUÍNEA, LIPOPOLISSACARÍDEOS, ÓXIDO NÍTRICO (SÍNTESE)
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ABNT
GIUSTI-PAIVA, Alexandre et al. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK, v. 21, n. 3, p. 271-275, 2004Tradução . . Acesso em: 10 nov. 2024.
APA
Giusti-Paiva, A., Martinez, M. R., Felix, J. V. C., Rocha, M. J. A. da, Cárnio, E. C., Elias, L. L. K., & Antunes-Rodrigues, J. (2004). Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK, 21( 3), 271-275.
NLM
Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Cárnio EC, Elias LLK, Antunes-Rodrigues J. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK. 2004 ; 21( 3): 271-275.[citado 2024 nov. 10 ]
Vancouver
Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Cárnio EC, Elias LLK, Antunes-Rodrigues J. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK. 2004 ; 21( 3): 271-275.[citado 2024 nov. 10 ]
Artigo de periodico
Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats (2003)
Rodriguez, T. T.; Albuquerque-Araújo, W I C; Reis, L C; Antunes-Rodrigues, José; Ramalho, M. J.
Source: Experimental Physiology. Unidade: FMRP
Subjects: SEPSE, LIPOPOLISSACARÍDEOS, ESTRESSE, RATOS
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ABNT
RODRIGUEZ, T. T. et al. Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats. Experimental Physiology, v. 88.6, p. 755-760, 2003Tradução . . Acesso em: 10 nov. 2024.
APA
Rodriguez, T. T., Albuquerque-Araújo, W. I. C., Reis, L. C., Antunes-Rodrigues, J., & Ramalho, M. J. (2003). Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats. Experimental Physiology, 88.6, 755-760.
NLM
Rodriguez TT, Albuquerque-Araújo WIC, Reis LC, Antunes-Rodrigues J, Ramalho MJ. Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats. Experimental Physiology. 2003 ; 88.6 755-760.[citado 2024 nov. 10 ]
Vancouver
Rodriguez TT, Albuquerque-Araújo WIC, Reis LC, Antunes-Rodrigues J, Ramalho MJ. Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats. Experimental Physiology. 2003 ; 88.6 755-760.[citado 2024 nov. 10 ]
Artigo de periodico
Role of nitric oxide in lipopolysaccharide-induced release of vasopressin in rats (2003)
Giusti-Paiva, Alexandre; Ruginsk, Silvia Graciela; Castro, Margaret de; Elias, Lucila Leico Kagohara; Cárnio, Evelin Capellari ; Antunes-Rodrigues, José
Source: Neuroscience Letters. Unidades: FMRP, EERP
Subjects: SEPSE (TERAPIA), VASOPRESSINAS, ÓXIDO NÍTRICO, LIPOPOLISSACARÍDEOS
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ABNT
GIUSTI-PAIVA, Alexandre et al. Role of nitric oxide in lipopolysaccharide-induced release of vasopressin in rats. Neuroscience Letters, v. 346, p. 21-24, 2003Tradução . . Disponível em: https://doi.org/10.1016/s0304-3940(03)00543-3. Acesso em: 10 nov. 2024.
APA
Giusti-Paiva, A., Ruginsk, S. G., Castro, M. de, Elias, L. L. K., Cárnio, E. C., & Antunes-Rodrigues, J. (2003). Role of nitric oxide in lipopolysaccharide-induced release of vasopressin in rats. Neuroscience Letters, 346, 21-24. doi:10.1016/s0304-3940(03)00543-3
NLM
Giusti-Paiva A, Ruginsk SG, Castro M de, Elias LLK, Cárnio EC, Antunes-Rodrigues J. Role of nitric oxide in lipopolysaccharide-induced release of vasopressin in rats [Internet]. Neuroscience Letters. 2003 ; 346 21-24.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/s0304-3940(03)00543-3
Vancouver
Giusti-Paiva A, Ruginsk SG, Castro M de, Elias LLK, Cárnio EC, Antunes-Rodrigues J. Role of nitric oxide in lipopolysaccharide-induced release of vasopressin in rats [Internet]. Neuroscience Letters. 2003 ; 346 21-24.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/s0304-3940(03)00543-3

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