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Artigo de periodico
The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages (2024)
Assis, Sayonara Ivana Santos de ; Amendola, Leonardo Szalo ; Ferreira, Guilherme da Silva ; Santos, Danielle Ribeiro ; Santana, Monique de Fátima Mello ; Santana, Kelly Gomes; Okamoto, Maristela Mitiko ; Iborra, Rodrigo Tallada ; Gianella, Maria Lúcia Cardillo Corrêa ; Barbeiro, Denise Frediani ; Soriano, Francisco Garcia ; Passarelli, Marisa ; Machado, Ubiratan Fabres
Source: International Journal of Molecular Sciences. Unidades: ICB, FM
Subjects: FISIOLOGIA, FATORES DE TRANSCRIÇÃO, PROTEÍNAS NUCLEARES, REGULAÇÃO GÊNICA, EXPRESSÃO GÊNICA, MACRÓFAGOS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, ALBUMINAS, INTERLEUCINA 6
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ASSIS, Sayonara Ivana Santos de et al. The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages. International Journal of Molecular Sciences, v. 25, n. 5, p. 16 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25052713. Acesso em: 15 out. 2024.
APA
Assis, S. I. S. de, Amendola, L. S., Ferreira, G. da S., Santos, D. R., Santana, M. de F. M., Santana, K. G., et al. (2024). The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages. International Journal of Molecular Sciences, 25( 5), 16 . doi:10.3390/ijms25052713
NLM
Assis SIS de, Amendola LS, Ferreira G da S, Santos DR, Santana M de FM, Santana KG, Okamoto MM, Iborra RT, Gianella MLCC, Barbeiro DF, Soriano FG, Passarelli M, Machado UF. The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 5): 16 .[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/ijms25052713
Vancouver
Assis SIS de, Amendola LS, Ferreira G da S, Santos DR, Santana M de FM, Santana KG, Okamoto MM, Iborra RT, Gianella MLCC, Barbeiro DF, Soriano FG, Passarelli M, Machado UF. The prolonged activation of the p65 subunit of the NF-Kappa-B nuclear factor sustains the persistent effect of advanced Glycation end products on inflammatory sensitization in macrophages [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 5): 16 .[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/ijms25052713
Artigo de periodico
Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice (2024)
Franco, Thaina Maquedo ; Tavares, Mariana Rosolen ; Novaes, Leonardo Santana ; Peixoto-Santos, Jose Eduardo ; Araujo, Ronaldo Carvalho ; Bader, Michael ; Wasinski, Frederick ; Munhoz, Carolina Demarchi ; Donato Junior, José
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, BRADICININA, INFLAMAÇÃO, SISTEMA NERVOSO CENTRAL, PRESSÃO SANGUÍNEA, PROTEÍNAS G, RECEPTORES DE CATECOLAMINA, RECEPTORES DE DOPAMINA, CAMUNDONGOS, PESOS E MEDIDAS CORPORAIS, AMINOÁCIDOS
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FRANCO, Thaina Maquedo et al. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, v. 25, n. 3, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25031490. Acesso em: 15 out. 2024.
APA
Franco, T. M., Tavares, M. R., Novaes, L. S., Peixoto-Santos, J. E., Araujo, R. C., Bader, M., et al. (2024). Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, 25( 3), 14 . doi:10.3390/ijms25031490
NLM
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/ijms25031490
Vancouver
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/ijms25031490
Artigo de periodico
TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease (2024)
Cabral, Laís M. ; Oliveira, Luis M. ; Miranda, Nicole C.; Kawamoto, Elisa Mitiko ; Costa, Soraia Katia Pereira ; Moreira, Thiago S. ; Takakura, Ana Carolina
Source: Brain Research. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, INFLAMAÇÃO, CITOCINAS, NECROSE
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CABRAL, Laís M. et al. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, v. 1822, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2023.148586. Acesso em: 15 out. 2024.
APA
Cabral, L. M., Oliveira, L. M., Miranda, N. C., Kawamoto, E. M., Costa, S. K. P., Moreira, T. S., & Takakura, A. C. (2024). TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, 1822. doi:10.1016/j.brainres.2023.148586
NLM
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586
Vancouver
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586
Artigo de periodico
Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells (2024)
Garcia, Bruna Ruschel Ewald Vega ; Makiyama, Edson Naoto ; Sampaio, Geni Rodrigues ; Freitas, Rosana Aparecida Manólio Soares ; Bovini, Andrea ; Amaral, Andressa Godoy ; Fock, Ricardo Ambrósio ; Rogero, Marcelo Macedo ; Bordin, Silvana
Source: Metabolites. Unidades: FCF, FSP, ICB
Subjects: FISIOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS EPITELIAIS E GLANDULARES, ADENOCARCINOMA, AMINOÁCIDOS, LIPOPOLISSACARÍDEOS, INFLAMAÇÃO, ANTI-INFLAMATÓRIOS, MUCOSA INTESTINAL, GLUTATIONA TRANSFERASE
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GARCIA, Bruna Ruschel Ewald Vega et al. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells. Metabolites, v. 14, n. 1, p. 13 , 2024Tradução . . Disponível em: https://doi.org/10.3390/metabo14010076. Acesso em: 15 out. 2024.
APA
Garcia, B. R. E. V., Makiyama, E. N., Sampaio, G. R., Freitas, R. A. M. S., Bovini, A., Amaral, A. G., et al. (2024). Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells. Metabolites, 14( 1), 13 . doi:10.3390/metabo14010076
NLM
Garcia BREV, Makiyama EN, Sampaio GR, Freitas RAMS, Bovini A, Amaral AG, Fock RA, Rogero MM, Bordin S. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells [Internet]. Metabolites. 2024 ; 14( 1): 13 .[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/metabo14010076
Vancouver
Garcia BREV, Makiyama EN, Sampaio GR, Freitas RAMS, Bovini A, Amaral AG, Fock RA, Rogero MM, Bordin S. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells [Internet]. Metabolites. 2024 ; 14( 1): 13 .[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/metabo14010076
Artigo de periodico
Autoregulation of blood flow drives early hypotension in a rat model of systemic inflammation induced by bacterial lipopolysaccharide (2023)
Moretti, Eduardo Hermógenes ; Rodrigues, Abner Cardoso ; Marques, Bruno Vinicius Duarte ; Lopes, Luciana Biagini ; Moreira, Thiago dos Santos ; Akamine, Eliana Hiromi ; Baccala, Luiz Antonio ; Fujita, André ; Steiner, Alexandre Alarcon
Source: PNAS Nexus. Unidades: ICB, EP, IME
Subjects: FISIOLOGIA, FARMACOLOGIA, IMUNOLOGIA, PRESSÃO SANGUÍNEA, CHOQUE SÉPTICO, VASODILATAÇÃO, FLUXO SANGUÍNEO, HIPOTENSÃO ANIMAL, LIPOPOLISSACARÍDEOS, MODELOS ANIMAIS DE DOENÇAS, RATOS, INFLAMAÇÃO
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MORETTI, Eduardo Hermógenes et al. Autoregulation of blood flow drives early hypotension in a rat model of systemic inflammation induced by bacterial lipopolysaccharide. PNAS Nexus, v. 2, n. 2, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1093/pnasnexus/pgad014. Acesso em: 15 out. 2024.
APA
Moretti, E. H., Rodrigues, A. C., Marques, B. V. D., Lopes, L. B., Moreira, T. dos S., Akamine, E. H., et al. (2023). Autoregulation of blood flow drives early hypotension in a rat model of systemic inflammation induced by bacterial lipopolysaccharide. PNAS Nexus, 2( 2), 1-13. doi:10.1093/pnasnexus/pgad014
NLM
Moretti EH, Rodrigues AC, Marques BVD, Lopes LB, Moreira T dos S, Akamine EH, Baccala LA, Fujita A, Steiner AA. Autoregulation of blood flow drives early hypotension in a rat model of systemic inflammation induced by bacterial lipopolysaccharide [Internet]. PNAS Nexus. 2023 ; 2( 2): 1-13.[citado 2024 out. 15 ] Available from: https://doi.org/10.1093/pnasnexus/pgad014
Vancouver
Moretti EH, Rodrigues AC, Marques BVD, Lopes LB, Moreira T dos S, Akamine EH, Baccala LA, Fujita A, Steiner AA. Autoregulation of blood flow drives early hypotension in a rat model of systemic inflammation induced by bacterial lipopolysaccharide [Internet]. PNAS Nexus. 2023 ; 2( 2): 1-13.[citado 2024 out. 15 ] Available from: https://doi.org/10.1093/pnasnexus/pgad014
Artigo de periodico
Hepatocellular carcinoma induced by hepatocyte Pten deletion reduces BAT UCP-1 and thermogenic capacity in mice, despite increasing serum FGF-21 and iWAT browning (2023)
Peixoto, Álbert S. ; Moreno, Mayara F. ; Castro, Érique de ; Perandini, Luiz A. ; Belchior, Thiago ; Oliveira, Tiago E. ; Vieira, Thayna S. ; Gilio, Gustavo R. ; Tomazelli, Caroline A. ; Leonardi, Bianca F. ; Milene Ortiz-Silva ; Silva Junior, Luciano P. ; Moretti, Eduardo H. ; Steiner, Alexandre Alarcon ; Festuccia, William Tadeu Lara
Source: Journal of Physiology and Biochemistry. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, CARCINOMA HEPATOCELULAR, HEPATOPATIAS, FÍGADO GORDUROSO, TERMOGÊNESE, INFLAMAÇÃO, CITOCINAS, CAMUNDONGOS, FIBROBLASTOS
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PEIXOTO, Álbert S. et al. Hepatocellular carcinoma induced by hepatocyte Pten deletion reduces BAT UCP-1 and thermogenic capacity in mice, despite increasing serum FGF-21 and iWAT browning. Journal of Physiology and Biochemistry, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1007/s13105-023-00970-4. Acesso em: 15 out. 2024.
APA
Peixoto, Á. S., Moreno, M. F., Castro, É. de, Perandini, L. A., Belchior, T., Oliveira, T. E., et al. (2023). Hepatocellular carcinoma induced by hepatocyte Pten deletion reduces BAT UCP-1 and thermogenic capacity in mice, despite increasing serum FGF-21 and iWAT browning. Journal of Physiology and Biochemistry, 1-13. doi:10.1007/s13105-023-00970-4
NLM
Peixoto ÁS, Moreno MF, Castro É de, Perandini LA, Belchior T, Oliveira TE, Vieira TS, Gilio GR, Tomazelli CA, Leonardi BF, Milene Ortiz-Silva, Silva Junior LP, Moretti EH, Steiner AA, Festuccia WTL. Hepatocellular carcinoma induced by hepatocyte Pten deletion reduces BAT UCP-1 and thermogenic capacity in mice, despite increasing serum FGF-21 and iWAT browning [Internet]. Journal of Physiology and Biochemistry. 2023 ; 1-13.[citado 2024 out. 15 ] Available from: https://doi.org/10.1007/s13105-023-00970-4
Vancouver
Peixoto ÁS, Moreno MF, Castro É de, Perandini LA, Belchior T, Oliveira TE, Vieira TS, Gilio GR, Tomazelli CA, Leonardi BF, Milene Ortiz-Silva, Silva Junior LP, Moretti EH, Steiner AA, Festuccia WTL. Hepatocellular carcinoma induced by hepatocyte Pten deletion reduces BAT UCP-1 and thermogenic capacity in mice, despite increasing serum FGF-21 and iWAT browning [Internet]. Journal of Physiology and Biochemistry. 2023 ; 1-13.[citado 2024 out. 15 ] Available from: https://doi.org/10.1007/s13105-023-00970-4
Artigo de periodico
Reduced intestinal butyrate availability is associated with the vascular remodeling in resistance arteries of hypertensive rats (2022)
Dardi, Patrizia ; Eichler, Rosangela Aparecida dos Santos ; Oliveira, Sarah de ; Vinolo, Marco Aurélio Ramirez Vinolo ; Câmara, Niels Olsen Saraiva ; Rossoni, Luciana Venturini
Source: Frontiers in Physiology. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, RATOS WISTAR, REAÇÃO EM CADEIA POR POLIMERASE, CROMATOGRAFIA LÍQUIDA, REMODELAÇÃO DAS VIAS AÉREAS, ÁCIDOS GRAXOS, ARTÉRIAS, PRESSÃO SANGUÍNEA, HIPERTENSÃO, MICRORNAS, INFLAMAÇÃO
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DARDI, Patrizia et al. Reduced intestinal butyrate availability is associated with the vascular remodeling in resistance arteries of hypertensive rats. Frontiers in Physiology, v. 13, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.998362. Acesso em: 15 out. 2024.
APA
Dardi, P., Eichler, R. A. dos S., Oliveira, S. de, Vinolo, M. A. R. V., Câmara, N. O. S., & Rossoni, L. V. (2022). Reduced intestinal butyrate availability is associated with the vascular remodeling in resistance arteries of hypertensive rats. Frontiers in Physiology, 13, 1-12. doi:10.3389/fphys.2022.998362
NLM
Dardi P, Eichler RA dos S, Oliveira S de, Vinolo MARV, Câmara NOS, Rossoni LV. Reduced intestinal butyrate availability is associated with the vascular remodeling in resistance arteries of hypertensive rats [Internet]. Frontiers in Physiology. 2022 ; 13 1-12.[citado 2024 out. 15 ] Available from: https://doi.org/10.3389/fphys.2022.998362
Vancouver
Dardi P, Eichler RA dos S, Oliveira S de, Vinolo MARV, Câmara NOS, Rossoni LV. Reduced intestinal butyrate availability is associated with the vascular remodeling in resistance arteries of hypertensive rats [Internet]. Frontiers in Physiology. 2022 ; 13 1-12.[citado 2024 out. 15 ] Available from: https://doi.org/10.3389/fphys.2022.998362
Artigo de periodico
ROS suppression by egg white hydrolysate in DOCA-Salt rats—an alternative tool against vascular dysfunction in severe hypertension (2022)
Abreu, Edina da Luz ; Moro, Camila Rodrigues ; Kanaan, Samia Hassan Husein ; Paula, Ricardo Bernardino de ; Herrera, Camila Teixeira ; Costa, Pedro Henrique Dorneles; Peçanha, Franck Maciel ; Vassallo, Dalton Valentim ; Rossoni, Luciana Venturini ; Castro, Marta Miguel ; Wiggers, Giulia Alessandra
Source: Antioxidants. Unidade: ICB
Subjects: FISIOLOGIA, PRESSÃO SANGUÍNEA, ENDOTÉLIO VASCULAR, INFLAMAÇÃO, MITOCÔNDRIAS, HIPERTENSÃO, ACETILCOLINA, VASODILATAÇÃO
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ABREU, Edina da Luz et al. ROS suppression by egg white hydrolysate in DOCA-Salt rats—an alternative tool against vascular dysfunction in severe hypertension. Antioxidants, v. 11, n. 9, p. 1-21, 2022Tradução . . Disponível em: https://doi.org/10.3390/antiox11091713. Acesso em: 15 out. 2024.
APA
Abreu, E. da L., Moro, C. R., Kanaan, S. H. H., Paula, R. B. de, Herrera, C. T., Costa, P. H. D., et al. (2022). ROS suppression by egg white hydrolysate in DOCA-Salt rats—an alternative tool against vascular dysfunction in severe hypertension. Antioxidants, 11( 9), 1-21. doi:10.3390/antiox11091713
NLM
Abreu E da L, Moro CR, Kanaan SHH, Paula RB de, Herrera CT, Costa PHD, Peçanha FM, Vassallo DV, Rossoni LV, Castro MM, Wiggers GA. ROS suppression by egg white hydrolysate in DOCA-Salt rats—an alternative tool against vascular dysfunction in severe hypertension [Internet]. Antioxidants. 2022 ; 11( 9): 1-21.[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/antiox11091713
Vancouver
Abreu E da L, Moro CR, Kanaan SHH, Paula RB de, Herrera CT, Costa PHD, Peçanha FM, Vassallo DV, Rossoni LV, Castro MM, Wiggers GA. ROS suppression by egg white hydrolysate in DOCA-Salt rats—an alternative tool against vascular dysfunction in severe hypertension [Internet]. Antioxidants. 2022 ; 11( 9): 1-21.[citado 2024 out. 15 ] Available from: https://doi.org/10.3390/antiox11091713
Artigo de periodico
The gut-brain axis and sodium appetite: can inflammation-related signaling influence the control of sodium intake? (2022)
Freitas, Flávio Eduardo Dias Araújo ; Batista, Marcos Adriano Carlos ; Braga, Daiane Cristina de Assis ; Oliveira, Lisandra Brandino de ; Antunes, Vagner Roberto ; Cardoso, Leonardo Máximo
Source: Appetite. Unidade: ICB
Subjects: FISIOLOGIA, SÓDIO, CITOCINAS, INFLAMAÇÃO, INGESTÃO, ALDOSTERONA, APETITE
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FREITAS, Flávio Eduardo Dias Araújo et al. The gut-brain axis and sodium appetite: can inflammation-related signaling influence the control of sodium intake?. Appetite, v. 175, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.appet.2022.106050. Acesso em: 15 out. 2024.
APA
Freitas, F. E. D. A., Batista, M. A. C., Braga, D. C. de A., Oliveira, L. B. de, Antunes, V. R., & Cardoso, L. M. (2022). The gut-brain axis and sodium appetite: can inflammation-related signaling influence the control of sodium intake? Appetite, 175, 1-8. doi:10.1016/j.appet.2022.106050
NLM
Freitas FEDA, Batista MAC, Braga DC de A, Oliveira LB de, Antunes VR, Cardoso LM. The gut-brain axis and sodium appetite: can inflammation-related signaling influence the control of sodium intake? [Internet]. Appetite. 2022 ; 175 1-8.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.appet.2022.106050
Vancouver
Freitas FEDA, Batista MAC, Braga DC de A, Oliveira LB de, Antunes VR, Cardoso LM. The gut-brain axis and sodium appetite: can inflammation-related signaling influence the control of sodium intake? [Internet]. Appetite. 2022 ; 175 1-8.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.appet.2022.106050
Artigo de periodico
Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction (2022)
Peres, Emília Cristina ; Victorio, Jamaira A. ; Souza, Valéria Nunes ; Faloppa, Ana Cristina Breithaupt ; Rabelo, Luiza A.; Lima, Wothan Tavares de ; Davel, Ana Paula ; Rossoni, Luciana Venturini
Source: Life Sciences. Unidades: HU, FM, ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, ARTÉRIA PULMONAR, REPERFUSÃO, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS, SUPERÓXIDO DISMUTASE, ENDOTÉLIO VASCULAR, ISQUEMIA, INFLAMAÇÃO, ÓXIDO NÍTRICO, INTERLEUCINAS
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PERES, Emília Cristina et al. Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction. Life Sciences, v. 306, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120851. Acesso em: 15 out. 2024.
APA
Peres, E. C., Victorio, J. A., Souza, V. N., Faloppa, A. C. B., Rabelo, L. A., Lima, W. T. de, et al. (2022). Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction. Life Sciences, 306, 1-10. doi:10.1016/j.lfs.2022.120851
NLM
Peres EC, Victorio JA, Souza VN, Faloppa ACB, Rabelo LA, Lima WT de, Davel AP, Rossoni LV. Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction [Internet]. Life Sciences. 2022 ; 306 1-10.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.lfs.2022.120851
Vancouver
Peres EC, Victorio JA, Souza VN, Faloppa ACB, Rabelo LA, Lima WT de, Davel AP, Rossoni LV. Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction [Internet]. Life Sciences. 2022 ; 306 1-10.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.lfs.2022.120851
Artigo de periodico
The cholinergic drug galantamine alleviates oxidative stress alongside anti-inflammatory and cardio-metabolic effects in subjects with the metabolic syndrome in a randomized trial (2021)
Miyahara, Carine Teles Sangleti; Katayama, Keyla Yukari; Angelis, Kátia De ; Moraes, Tércio Lemos de ; Araújo, Amanda Aparecida ; Lopes, Heno Ferreira ; Camacho, Cleber ; Bortolotto, Luiz Aparecido ; Michelini, Lisete Compagno ; Irigoyen, Maria Claudia Costa ; Olofsson, Peder S. ; Barnaby, Douglas P. ; Tracey, Kevin J. ; Pavlov, Valentin A. ; Colombo, Fernanda Marciano Consolim
Source: Frontiers in Immunology. Unidades: FM, ICB
Subjects: FISIOLOGIA, ESTRESSE OXIDATIVO, INFLAMAÇÃO, COLINÉRGICOS, ANTI-INFLAMATÓRIOS, CARDIOPATIAS, ENSAIO CLÍNICO CONTROLADO RANDOMIZADO, OBESIDADE, ENZIMAS OXIRREDUTORAS, CITOCINAS, FREQUÊNCIA CARDÍACA
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MIYAHARA, Carine Teles Sangleti et al. The cholinergic drug galantamine alleviates oxidative stress alongside anti-inflammatory and cardio-metabolic effects in subjects with the metabolic syndrome in a randomized trial. Frontiers in Immunology, v. 12, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.613979. Acesso em: 15 out. 2024.
APA
Miyahara, C. T. S., Katayama, K. Y., Angelis, K. D., Moraes, T. L. de, Araújo, A. A., Lopes, H. F., et al. (2021). The cholinergic drug galantamine alleviates oxidative stress alongside anti-inflammatory and cardio-metabolic effects in subjects with the metabolic syndrome in a randomized trial. Frontiers in Immunology, 12, 1-12. doi:10.3389/fimmu.2021.613979
NLM
Miyahara CTS, Katayama KY, Angelis KD, Moraes TL de, Araújo AA, Lopes HF, Camacho C, Bortolotto LA, Michelini LC, Irigoyen MCC, Olofsson PS, Barnaby DP, Tracey KJ, Pavlov VA, Colombo FMC. The cholinergic drug galantamine alleviates oxidative stress alongside anti-inflammatory and cardio-metabolic effects in subjects with the metabolic syndrome in a randomized trial [Internet]. Frontiers in Immunology. 2021 ; 12 1-12.[citado 2024 out. 15 ] Available from: https://doi.org/10.3389/fimmu.2021.613979
Vancouver
Miyahara CTS, Katayama KY, Angelis KD, Moraes TL de, Araújo AA, Lopes HF, Camacho C, Bortolotto LA, Michelini LC, Irigoyen MCC, Olofsson PS, Barnaby DP, Tracey KJ, Pavlov VA, Colombo FMC. The cholinergic drug galantamine alleviates oxidative stress alongside anti-inflammatory and cardio-metabolic effects in subjects with the metabolic syndrome in a randomized trial [Internet]. Frontiers in Immunology. 2021 ; 12 1-12.[citado 2024 out. 15 ] Available from: https://doi.org/10.3389/fimmu.2021.613979
Artigo de periodico
Small intestine remodeling in male Goto–Kakizaki rats (2021)
Pereira, Joice Naiara Bertaglia ; Murata, Gilson Masahiro ; Sato, Fabio Takeo ; Marosti, Aline Rosa ; Carvalho, Carla Roberta de Oliveira ; Curi, Rui
Source: Physiological Reports. Unidades: FM, ICB
Subjects: FISIOLOGIA, DIABETES MELLITUS, SISTEMA NERVOSO ENTÉRICO, INTESTINO DELGADO, INFLAMAÇÃO, RATOS WISTAR, JEJUNO DE ANIMAL, MORFOMETRIA, OBESIDADE, RESISTÊNCIA À INSULINA
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ABNT
PEREIRA, Joice Naiara Bertaglia et al. Small intestine remodeling in male Goto–Kakizaki rats. Physiological Reports, v. 9, n. 3, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.14814/phy2.14755. Acesso em: 15 out. 2024.
APA
Pereira, J. N. B., Murata, G. M., Sato, F. T., Marosti, A. R., Carvalho, C. R. de O., & Curi, R. (2021). Small intestine remodeling in male Goto–Kakizaki rats. Physiological Reports, 9( 3), 1-16. doi:10.14814/phy2.14755
NLM
Pereira JNB, Murata GM, Sato FT, Marosti AR, Carvalho CR de O, Curi R. Small intestine remodeling in male Goto–Kakizaki rats [Internet]. Physiological Reports. 2021 ; 9( 3): 1-16.[citado 2024 out. 15 ] Available from: https://doi.org/10.14814/phy2.14755
Vancouver
Pereira JNB, Murata GM, Sato FT, Marosti AR, Carvalho CR de O, Curi R. Small intestine remodeling in male Goto–Kakizaki rats [Internet]. Physiological Reports. 2021 ; 9( 3): 1-16.[citado 2024 out. 15 ] Available from: https://doi.org/10.14814/phy2.14755
Artigo de periodico
Polyphenols of cambuci (Campomanesia phaea (O. Berg.)) fruit ameliorate insulin resistance and hepatic steatosis in obese mice (2021)
Donado-Pestana, Carlos Mario ; Pessoa, Érika Vicencia Monteiro ; Rodrigues, Larissa ; Rossi, Rafaela Marteli; Moura, Márcio Hercules Caldas; Santos-Donado, Priscila Robertina ; Castro, Érique de ; Festuccia, William Tadeu Lara ; Genovese, Maria Inês
Source: Food Chemistry. Unidades: ICB, FCF, FDRP, ESALQ
Subjects: CAMBUCI, CAMUNDONGOS, COMPOSTOS FENÓLICOS, DIETA ANIMAL, FÍGADO GORDUROSO, INFLAMAÇÃO, OBESIDADE, RESISTÊNCIA À INSULINA
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ABNT
DONADO-PESTANA, Carlos Mario et al. Polyphenols of cambuci (Campomanesia phaea (O. Berg.)) fruit ameliorate insulin resistance and hepatic steatosis in obese mice. Food Chemistry, v. 340, p. 1-9, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.foodchem.2020.128169. Acesso em: 15 out. 2024.
APA
Donado-Pestana, C. M., Pessoa, É. V. M., Rodrigues, L., Rossi, R. M., Moura, M. H. C., Santos-Donado, P. R., et al. (2021). Polyphenols of cambuci (Campomanesia phaea (O. Berg.)) fruit ameliorate insulin resistance and hepatic steatosis in obese mice. Food Chemistry, 340, 1-9. doi:10.1016/j.foodchem.2020.128169
NLM
Donado-Pestana CM, Pessoa ÉVM, Rodrigues L, Rossi RM, Moura MHC, Santos-Donado PR, Castro É de, Festuccia WTL, Genovese MI. Polyphenols of cambuci (Campomanesia phaea (O. Berg.)) fruit ameliorate insulin resistance and hepatic steatosis in obese mice [Internet]. Food Chemistry. 2021 ; 340 1-9.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.foodchem.2020.128169
Vancouver
Donado-Pestana CM, Pessoa ÉVM, Rodrigues L, Rossi RM, Moura MHC, Santos-Donado PR, Castro É de, Festuccia WTL, Genovese MI. Polyphenols of cambuci (Campomanesia phaea (O. Berg.)) fruit ameliorate insulin resistance and hepatic steatosis in obese mice [Internet]. Food Chemistry. 2021 ; 340 1-9.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.foodchem.2020.128169
Trabalho de evento-resumo periodico
A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells (2021)
Vilas-Boas, Eloisa Aparecida ; Ortis, Fernanda ; Roma, Leticia Prates ; Carpinelli, Angelo Rafael
Source: Free Radical Biology & Medicine. Conference titles: Annual Meeting Abstracts "Redox Biology in the 21. Century". Unidades: ICB, IQ
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, ILHOTAS DE LANGERHANS, ATIVAÇÃO ENZIMÁTICA, OXIDAÇÃO, INFLAMAÇÃO, ENZIMAS OXIRREDUTORAS, RADICAIS LIVRES
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells. Free Radical Biology & Medicine. Philadelphia: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.freeradbiomed.2021.08.173. Acesso em: 15 out. 2024. , 2021
APA
Vilas-Boas, E. A., Ortis, F., Roma, L. P., & Carpinelli, A. R. (2021). A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells. Free Radical Biology & Medicine. Philadelphia: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.freeradbiomed.2021.08.173
NLM
Vilas-Boas EA, Ortis F, Roma LP, Carpinelli AR. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells [Internet]. Free Radical Biology & Medicine. 2021 ; 177 S112.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2021.08.173
Vancouver
Vilas-Boas EA, Ortis F, Roma LP, Carpinelli AR. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells [Internet]. Free Radical Biology & Medicine. 2021 ; 177 S112.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2021.08.173
Artigo de periodico
Long-term angiotensin II infusion induces oxidative and endoplasmic reticulum stress and modulates Na+ transporters through the nephron (2021)
Lins, Bruna Bezerra ; Casare, Fernando Augusto Malavazzi ; Fontenele, Flávia Ferreira ; Gonçalves, Guilherme Lopes ; Souza, Maria Oliveira de
Source: Frontiers in Physiology. Unidade: ICB
Subjects: FISIOLOGIA, ANGIOTENSINA II, ESTRESSE OXIDATIVO, TRANSPORTE DE SÓDIO, INFLAMAÇÃO, RETÍCULO ENDOPLASMÁTICO, NEFROPATIAS, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS
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ABNT
LINS, Bruna Bezerra et al. Long-term angiotensin II infusion induces oxidative and endoplasmic reticulum stress and modulates Na+ transporters through the nephron. Frontiers in Physiology, v. 12, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphys.2021.642752. Acesso em: 15 out. 2024.
APA
Lins, B. B., Casare, F. A. M., Fontenele, F. F., Gonçalves, G. L., & Souza, M. O. de. (2021). Long-term angiotensin II infusion induces oxidative and endoplasmic reticulum stress and modulates Na+ transporters through the nephron. Frontiers in Physiology, 12, 1-15. doi:10.3389/fphys.2021.642752
NLM
Lins BB, Casare FAM, Fontenele FF, Gonçalves GL, Souza MO de. Long-term angiotensin II infusion induces oxidative and endoplasmic reticulum stress and modulates Na+ transporters through the nephron [Internet]. Frontiers in Physiology. 2021 ; 12 1-15.[citado 2024 out. 15 ] Available from: https://doi.org/10.3389/fphys.2021.642752
Vancouver
Lins BB, Casare FAM, Fontenele FF, Gonçalves GL, Souza MO de. Long-term angiotensin II infusion induces oxidative and endoplasmic reticulum stress and modulates Na+ transporters through the nephron [Internet]. Frontiers in Physiology. 2021 ; 12 1-15.[citado 2024 out. 15 ] Available from: https://doi.org/10.3389/fphys.2021.642752
Artigo de periodico
Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model (2021)
Villaça, Catharina de Barros Pimentel ; Paula, Carolina Cavalcante de ; Oliveira, Caroline Cruz de ; Vilas-Boas, Eloisa Aparecida ; Silva, Junia Carolina dos Santos ; Oliveira, Sérgio Ferreira de; Abdulkader, Fernando Rodrigues de Moraes ; Ferreira, Sandra Mara; Ortis, Fernanda
Source: Experimental Physiology. Unidade: ICB
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, EXERCÍCIO FÍSICO, DIABETES MELLITUS, MODELOS ANIMAIS DE DOENÇAS, ILHOTAS DE LANGERHANS, INFLAMAÇÃO, CAMUNDONGOS, ANTINEOPLÁSICOS, ÍNSULA DO PÂNCREAS ANIMAL, GLUCAGON
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ABNT
VILLAÇA, Catharina de Barros Pimentel et al. Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model. Experimental Physiology, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1113/EP088872. Acesso em: 15 out. 2024.
APA
Villaça, C. de B. P., Paula, C. C. de, Oliveira, C. C. de, Vilas-Boas, E. A., Silva, J. C. dos S., Oliveira, S. F. de, et al. (2021). Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model. Experimental Physiology, 1-16. doi:10.1113/EP088872
NLM
Villaça C de BP, Paula CC de, Oliveira CC de, Vilas-Boas EA, Silva JC dos S, Oliveira SF de, Abdulkader FR de M, Ferreira SM, Ortis F. Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model [Internet]. Experimental Physiology. 2021 ; 1-16.[citado 2024 out. 15 ] Available from: https://doi.org/10.1113/EP088872
Vancouver
Villaça C de BP, Paula CC de, Oliveira CC de, Vilas-Boas EA, Silva JC dos S, Oliveira SF de, Abdulkader FR de M, Ferreira SM, Ortis F. Beneficial effects of physical exercise for β-cell maintenance in a type 1 diabetes mellitus animal model [Internet]. Experimental Physiology. 2021 ; 1-16.[citado 2024 out. 15 ] Available from: https://doi.org/10.1113/EP088872
Trabalho de evento-resumo
A novel diselenide attenuates the carrageenan-induced inflammation in mice by inhibiting neutrophil chemotaxis (2021)
Lessa, Tássia; Correia, Thiago Macêdo; Santos, Talita Costa; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da ; Peixoto, Albert Souza ; Festuccia, William Tadeu Lara ; Yatsuda, Regiane ; Gusmão, Amélia Cristina; Santos, Alcindo Aparecido dos ; Meotti, Flavia Carla ; Queiroz, Raphael Ferreira
Source: Program and Abstract Book. Conference titles: Congress of the International Union for Pure Applied Biophysics/IUPAB. Unidades: ICB, IQ
Subjects: SELÊNIO, INFLAMAÇÃO, PEROXIDASE
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ABNT
LESSA, Tássia et al. A novel diselenide attenuates the carrageenan-induced inflammation in mice by inhibiting neutrophil chemotaxis. 2021, Anais.. São Paulo: IUPAB-SBBf-SBBq, 2021. Disponível em: http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf. Acesso em: 15 out. 2024.
APA
Lessa, T., Correia, T. M., Santos, T. C., Silva, R. P. da, Silva, B. P. da, Peixoto, A. S., et al. (2021). A novel diselenide attenuates the carrageenan-induced inflammation in mice by inhibiting neutrophil chemotaxis. In Program and Abstract Book. São Paulo: IUPAB-SBBf-SBBq. Recuperado de http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf
NLM
Lessa T, Correia TM, Santos TC, Silva RP da, Silva BP da, Peixoto AS, Festuccia WTL, Yatsuda R, Gusmão AC, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan-induced inflammation in mice by inhibiting neutrophil chemotaxis [Internet]. Program and Abstract Book. 2021 ;[citado 2024 out. 15 ] Available from: http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf
Vancouver
Lessa T, Correia TM, Santos TC, Silva RP da, Silva BP da, Peixoto AS, Festuccia WTL, Yatsuda R, Gusmão AC, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan-induced inflammation in mice by inhibiting neutrophil chemotaxis [Internet]. Program and Abstract Book. 2021 ;[citado 2024 out. 15 ] Available from: http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf
Artigo de periodico
Regulation of adipocyte and macrophage functions by mTORC1 and 2 in metabolic diseases (2020)
Festuccia, William Tadeu Lara
Source: Molecular Nutrition & Food Research. Unidade: ICB
Subjects: FISIOLOGIA, TECIDO ADIPOSO, INFLAMAÇÃO, DOENÇAS METABÓLICAS, MACRÓFAGOS, RESISTÊNCIA À INSULINA, FATORES DE CRESCIMENTO, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
FESTUCCIA, William Tadeu Lara. Regulation of adipocyte and macrophage functions by mTORC1 and 2 in metabolic diseases. Molecular Nutrition & Food Research, p. 9 , 2020Tradução . . Disponível em: https://doi.org/10.1002/mnfr.201900768. Acesso em: 15 out. 2024.
APA
Festuccia, W. T. L. (2020). Regulation of adipocyte and macrophage functions by mTORC1 and 2 in metabolic diseases. Molecular Nutrition & Food Research, 9 . doi:10.1002/mnfr.201900768
NLM
Festuccia WTL. Regulation of adipocyte and macrophage functions by mTORC1 and 2 in metabolic diseases [Internet]. Molecular Nutrition & Food Research. 2020 ; 9 .[citado 2024 out. 15 ] Available from: https://doi.org/10.1002/mnfr.201900768
Vancouver
Festuccia WTL. Regulation of adipocyte and macrophage functions by mTORC1 and 2 in metabolic diseases [Internet]. Molecular Nutrition & Food Research. 2020 ; 9 .[citado 2024 out. 15 ] Available from: https://doi.org/10.1002/mnfr.201900768
Artigo de periodico
The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway (2020)
Costa, Ana Carolina Panveloski ; Kuwabara, Wilson Mitsuo Tatagiba ; Bonassa, Ana Cláudia Munhoz ; Monaco, Camila Ferraz Lucena ; Curi, Rui ; Carpinelli, Angelo Rafael ; Nunes, Maria Tereza
Source: Endocrine. Unidades: ICB, IQ
Subjects: FISIOLOGIA, BIOFÍSICA, HORMÔNIOS TIREOIDIANOS, RESISTÊNCIA À INSULINA, RATOS, CITOCININAS, DIABETES MELLITUS, INFLAMAÇÃO, GLICOSE, TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Ana Carolina Panveloski et al. The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway. Endocrine, p. 9 , 2020Tradução . . Disponível em: https://doi.org/10.1007/s12020-020-02208-5. Acesso em: 15 out. 2024.
APA
Costa, A. C. P., Kuwabara, W. M. T., Bonassa, A. C. M., Monaco, C. F. L., Curi, R., Carpinelli, A. R., & Nunes, M. T. (2020). The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway. Endocrine, 9 . doi:10.1007/s12020-020-02208-5
NLM
Costa ACP, Kuwabara WMT, Bonassa ACM, Monaco CFL, Curi R, Carpinelli AR, Nunes MT. The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway [Internet]. Endocrine. 2020 ;9 .[citado 2024 out. 15 ] Available from: https://doi.org/10.1007/s12020-020-02208-5
Vancouver
Costa ACP, Kuwabara WMT, Bonassa ACM, Monaco CFL, Curi R, Carpinelli AR, Nunes MT. The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway [Internet]. Endocrine. 2020 ;9 .[citado 2024 out. 15 ] Available from: https://doi.org/10.1007/s12020-020-02208-5
Artigo de periodico
Leukotriene pathway activation associates with poor glycemic control and with cardiovascular autonomic neuropathy in type 1 diabetes (2020)
Bezerra, Daniele Perreira dos Santos ; Ribeiro Junior, Luciano Filgueiras; Monteiro, Maria Beatriz ; Admoni, Sharon Nina ; Perez, Ricardo Vessoni ; Cavaleiro, Ana Mercedes de Sousa; Machado, Cleide Guimarães; Machado, Ubiratan Fabres ; Passarelli, Marisa ; Jancar, Sonia ; Giannella, Maria Lucia Correa
Source: Mediators of Inflammation. Unidades: FM, ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, GLICEMIA, LEUCOTRIENOS, SISTEMA NERVOSO AUTÔNOMO, NEUROPATIAS DIABÉTICAS, DIABETES MELLITUS, EXPRESSÃO GÊNICA, NEFROPATIAS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BEZERRA, Daniele Perreira dos Santos et al. Leukotriene pathway activation associates with poor glycemic control and with cardiovascular autonomic neuropathy in type 1 diabetes. Mediators of Inflammation, p. 9 , 2020Tradução . . Disponível em: https://doi.org/10.1155/2020/5704713. Acesso em: 15 out. 2024.
APA
Bezerra, D. P. dos S., Ribeiro Junior, L. F., Monteiro, M. B., Admoni, S. N., Perez, R. V., Cavaleiro, A. M. de S., et al. (2020). Leukotriene pathway activation associates with poor glycemic control and with cardiovascular autonomic neuropathy in type 1 diabetes. Mediators of Inflammation, 9 . doi:10.1155/2020/5704713
NLM
Bezerra DP dos S, Ribeiro Junior LF, Monteiro MB, Admoni SN, Perez RV, Cavaleiro AM de S, Machado CG, Machado UF, Passarelli M, Jancar S, Giannella MLC. Leukotriene pathway activation associates with poor glycemic control and with cardiovascular autonomic neuropathy in type 1 diabetes [Internet]. Mediators of Inflammation. 2020 ; 9 .[citado 2024 out. 15 ] Available from: https://doi.org/10.1155/2020/5704713
Vancouver
Bezerra DP dos S, Ribeiro Junior LF, Monteiro MB, Admoni SN, Perez RV, Cavaleiro AM de S, Machado CG, Machado UF, Passarelli M, Jancar S, Giannella MLC. Leukotriene pathway activation associates with poor glycemic control and with cardiovascular autonomic neuropathy in type 1 diabetes [Internet]. Mediators of Inflammation. 2020 ; 9 .[citado 2024 out. 15 ] Available from: https://doi.org/10.1155/2020/5704713

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