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Artigo de periodico-dep/entr
Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia (2020)
Amorim, Barbara Casella ; Latini, Ana Carla Pereira ; Golim, Marjorie de Assis ; Ruiz Júnior, Raul Lopes; Yoo, Hugo Hyung Bok ; Arruda, Maria Sueli Pereira de; Tavares, Aldo Henrique Fonseca Pacheco ; Cavalcante, Ricardo de Souza ; Mendes, Rinaldo Poncio ; Pontillo, Alessandra ; Venturini, James
Source: Microbes and Infection. Unidade: ICB
Subjects: IMUNOLOGIA, MONÓCITOS, PARACOCCIDIOIDES, TABAGISMO, MICOSES, PENICILLIUM, FIBROSE PULMONAR, FUMO, INFLAMAÇÃO, CITOCINAS
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ABNT
AMORIM, Barbara Casella et al. Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia. Microbes and Infection. Issy les Moulineaux: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.micinf.2019.11.001. Acesso em: 08 nov. 2024. , 2020
APA
Amorim, B. C., Latini, A. C. P., Golim, M. de A., Ruiz Júnior, R. L., Yoo, H. H. B., Arruda, M. S. P. de, et al. (2020). Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia. Microbes and Infection. Issy les Moulineaux: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.micinf.2019.11.001
NLM
Amorim BC, Latini ACP, Golim M de A, Ruiz Júnior RL, Yoo HHB, Arruda MSP de, Tavares AHFP, Cavalcante R de S, Mendes RP, Pontillo A, Venturini J. Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia [Internet]. Microbes and Infection. 2020 ; 22( 3): 137-143.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2019.11.001
Vancouver
Amorim BC, Latini ACP, Golim M de A, Ruiz Júnior RL, Yoo HHB, Arruda MSP de, Tavares AHFP, Cavalcante R de S, Mendes RP, Pontillo A, Venturini J. Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia [Internet]. Microbes and Infection. 2020 ; 22( 3): 137-143.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2019.11.001
Artigo de periodico
Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins (2019)
Torres, Azaf Moreno ; Jiménez, Irving R. Malvido; Moctezuma, Alejandro de la Peña ; Sánchez, Luz O. Castillo ; Fraga, Tatiana Rodrigues ; Barbosa, Angela Silva ; Isaac, Lourdes ; Ruiz, Alfredo Sahagún
Source: Microbes and Infection. Unidade: ICB
Subjects: IMUNOLOGIA, LEPTOSPIROSE, BACTÉRIAS GRAM-NEGATIVAS, PROTEÍNAS
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TORRES, Azaf Moreno et al. Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins. Microbes and Infection, v. 21, n. 8–9, p. 377-385, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2019.03.001. Acesso em: 08 nov. 2024.
APA
Torres, A. M., Jiménez, I. R. M., Moctezuma, A. de la P., Sánchez, L. O. C., Fraga, T. R., Barbosa, A. S., et al. (2019). Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins. Microbes and Infection, 21( 8–9), 377-385. doi:10.1016/j.micinf.2019.03.001
NLM
Torres AM, Jiménez IRM, Moctezuma A de la P, Sánchez LOC, Fraga TR, Barbosa AS, Isaac L, Ruiz AS. Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins [Internet]. Microbes and Infection. 2019 ; 21( 8–9): 377-385.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2019.03.001
Vancouver
Torres AM, Jiménez IRM, Moctezuma A de la P, Sánchez LOC, Fraga TR, Barbosa AS, Isaac L, Ruiz AS. Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins [Internet]. Microbes and Infection. 2019 ; 21( 8–9): 377-385.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2019.03.001
Artigo de periodico
Evaluation of cytotoxic effect of the combination of a pyridinyl carboxamide derivative and oxaliplatin on NCI-H1299 human non-small cell lung carcinoma cells (2016)
Teixeira, Sarah Fernandes; Azevedo, Ricardo Alexandre; Silva, Arthur Carvalho; Braga, Rodolpho Campos; Jorge, Salomão Dória; Barbuto, José Alexandre Marzagão ; Andrade, Carolina Horta; Ferreira, Adilson Kleber
Source: Biomedicine & Pharmacotherapy. Unidade: ICB
Assunto: IMUNOLOGIA
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TEIXEIRA, Sarah Fernandes et al. Evaluation of cytotoxic effect of the combination of a pyridinyl carboxamide derivative and oxaliplatin on NCI-H1299 human non-small cell lung carcinoma cells. Biomedicine & Pharmacotherapy, v. 84, p. 1019-1028, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2016.10.025. Acesso em: 08 nov. 2024.
APA
Teixeira, S. F., Azevedo, R. A., Silva, A. C., Braga, R. C., Jorge, S. D., Barbuto, J. A. M., et al. (2016). Evaluation of cytotoxic effect of the combination of a pyridinyl carboxamide derivative and oxaliplatin on NCI-H1299 human non-small cell lung carcinoma cells. Biomedicine & Pharmacotherapy, 84, 1019-1028. doi:10.1016/j.biopha.2016.10.025
NLM
Teixeira SF, Azevedo RA, Silva AC, Braga RC, Jorge SD, Barbuto JAM, Andrade CH, Ferreira AK. Evaluation of cytotoxic effect of the combination of a pyridinyl carboxamide derivative and oxaliplatin on NCI-H1299 human non-small cell lung carcinoma cells [Internet]. Biomedicine & Pharmacotherapy. 2016 ; 84 1019-1028.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.biopha.2016.10.025
Vancouver
Teixeira SF, Azevedo RA, Silva AC, Braga RC, Jorge SD, Barbuto JAM, Andrade CH, Ferreira AK. Evaluation of cytotoxic effect of the combination of a pyridinyl carboxamide derivative and oxaliplatin on NCI-H1299 human non-small cell lung carcinoma cells [Internet]. Biomedicine & Pharmacotherapy. 2016 ; 84 1019-1028.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.biopha.2016.10.025
Artigo de periodico
Endostatin gene therapy inhibits intratumoral macrophage M2 polarization (2016)
Foguer, Karen; Braga, Marina de Souza; Peron, Jean Pierre Schatzmann ; Bortoluci, Karina Ramalho; Bellini, Maria Helena
Source: Biomedicine & Pharmacotherapy. Unidade: ICB
Assunto: IMUNOLOGIA
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FOGUER, Karen et al. Endostatin gene therapy inhibits intratumoral macrophage M2 polarization. Biomedicine & Pharmacotherapy, v. 79, p. 102-111, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2016.01.035. Acesso em: 08 nov. 2024.
APA
Foguer, K., Braga, M. de S., Peron, J. P. S., Bortoluci, K. R., & Bellini, M. H. (2016). Endostatin gene therapy inhibits intratumoral macrophage M2 polarization. Biomedicine & Pharmacotherapy, 79, 102-111. doi:10.1016/j.biopha.2016.01.035
NLM
Foguer K, Braga M de S, Peron JPS, Bortoluci KR, Bellini MH. Endostatin gene therapy inhibits intratumoral macrophage M2 polarization [Internet]. Biomedicine & Pharmacotherapy. 2016 ; 79 102-111.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.biopha.2016.01.035
Vancouver
Foguer K, Braga M de S, Peron JPS, Bortoluci KR, Bellini MH. Endostatin gene therapy inhibits intratumoral macrophage M2 polarization [Internet]. Biomedicine & Pharmacotherapy. 2016 ; 79 102-111.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.biopha.2016.01.035
Artigo de periodico
Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death (2016)
Amaral, Eduardo P.; Lasunskaia, Elena B.; Lima, Maria Regina D'Império
Source: Microbes and Infection. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
AMARAL, Eduardo P. e LASUNSKAIA, Elena B. e LIMA, Maria Regina D'Império. Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death. Microbes and Infection, v. 18, n. 1, p. 11-20, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2015.09.005. Acesso em: 08 nov. 2024.
APA
Amaral, E. P., Lasunskaia, E. B., & Lima, M. R. D. 'I. (2016). Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death. Microbes and Infection, 18( 1), 11-20. doi:10.1016/j.micinf.2015.09.005
NLM
Amaral EP, Lasunskaia EB, Lima MRD'I. Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death [Internet]. Microbes and Infection. 2016 ; 18( 1): 11-20.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2015.09.005
Vancouver
Amaral EP, Lasunskaia EB, Lima MRD'I. Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death [Internet]. Microbes and Infection. 2016 ; 18( 1): 11-20.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2015.09.005
Artigo de periodico
Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages (2014)
Morato, Camila I.; Silva Jr, Ildefonso A. da; Borges, Arissa F.; Dorta, Miriam L.; Oliveira, Milton A. P.; Jancar, Sonia ; Serezani, Carlos H.; Ribeiro-Dias, Fátima
Source: Microbes and Infection. Unidade: ICB
Subjects: IMUNOLOGIA, LEISHMANIA BRASILIENSIS, MACRÓFAGOS, LEUCOTRIENOS
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ABNT
MORATO, Camila I. et al. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, v. 16, n. 11, p. 945-953, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.08.015. Acesso em: 08 nov. 2024.
APA
Morato, C. I., Silva Jr, I. A. da, Borges, A. F., Dorta, M. L., Oliveira, M. A. P., Jancar, S., et al. (2014). Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, 16( 11), 945-953. doi:10.1016/j.micinf.2014.08.015
NLM
Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2014.08.015
Vancouver
Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.micinf.2014.08.015
Trabalho de evento-resumo periodico
Expression of inflammatory mediators for transforming growth factor-Beta induced endothelial to mesenchymal transition in human umbiliocal vein endothelial cells (2011)
Origassa, C. S. T.; Salvador, F. A.; Nogueira, E.; Silva, A. P.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
ORIGASSA, C. S. T. et al. Expression of inflammatory mediators for transforming growth factor-Beta induced endothelial to mesenchymal transition in human umbiliocal vein endothelial cells. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Origassa, C. S. T., Salvador, F. A., Nogueira, E., Silva, A. P., & Câmara, N. O. S. (2011). Expression of inflammatory mediators for transforming growth factor-Beta induced endothelial to mesenchymal transition in human umbiliocal vein endothelial cells. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Origassa CST, Salvador FA, Nogueira E, Silva AP, Câmara NOS. Expression of inflammatory mediators for transforming growth factor-Beta induced endothelial to mesenchymal transition in human umbiliocal vein endothelial cells. Inflammation Research. 2011 ; 60 S274 res. P-574.[citado 2024 nov. 08 ]
Vancouver
Origassa CST, Salvador FA, Nogueira E, Silva AP, Câmara NOS. Expression of inflammatory mediators for transforming growth factor-Beta induced endothelial to mesenchymal transition in human umbiliocal vein endothelial cells. Inflammation Research. 2011 ; 60 S274 res. P-574.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Mesenchymal stem cell role in murine folic acid-induced acute kidney injury model (2011)
Burgos-Silva, M.; Donizetti-Oliveira, C.; Cenedeze, M. A.; Malheiros, D. M. A. C.; Reis, M. A.; Pacheco-Silva, A.; Semedo, P.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
BURGOS-SILVA, M. et al. Mesenchymal stem cell role in murine folic acid-induced acute kidney injury model. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Burgos-Silva, M., Donizetti-Oliveira, C., Cenedeze, M. A., Malheiros, D. M. A. C., Reis, M. A., Pacheco-Silva, A., et al. (2011). Mesenchymal stem cell role in murine folic acid-induced acute kidney injury model. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Burgos-Silva M, Donizetti-Oliveira C, Cenedeze MA, Malheiros DMAC, Reis MA, Pacheco-Silva A, Semedo P, Câmara NOS. Mesenchymal stem cell role in murine folic acid-induced acute kidney injury model. Inflammation Research. 2011 ; 60 S277 res. P-577.[citado 2024 nov. 08 ]
Vancouver
Burgos-Silva M, Donizetti-Oliveira C, Cenedeze MA, Malheiros DMAC, Reis MA, Pacheco-Silva A, Semedo P, Câmara NOS. Mesenchymal stem cell role in murine folic acid-induced acute kidney injury model. Inflammation Research. 2011 ; 60 S277 res. P-577.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Renal ischemia and reperfusion injury modulate lung allergic inflammation probably altering TH1/TH2 balance by changing cellular signal transduction (2011)
Campanholle, G.; Campanholle, G.; Silva, R. C.; Martins, J. O.; Landgraf, M. A.; Paiva, V. N.; Pacheco-Silva, A.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
CAMPANHOLLE, G. et al. Renal ischemia and reperfusion injury modulate lung allergic inflammation probably altering TH1/TH2 balance by changing cellular signal transduction. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Campanholle, G., Campanholle, G., Silva, R. C., Martins, J. O., Landgraf, M. A., Paiva, V. N., et al. (2011). Renal ischemia and reperfusion injury modulate lung allergic inflammation probably altering TH1/TH2 balance by changing cellular signal transduction. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Campanholle G, Campanholle G, Silva RC, Martins JO, Landgraf MA, Paiva VN, Pacheco-Silva A, Câmara NOS. Renal ischemia and reperfusion injury modulate lung allergic inflammation probably altering TH1/TH2 balance by changing cellular signal transduction. Inflammation Research. 2011 ; 60 S140 res. P-172.[citado 2024 nov. 08 ]
Vancouver
Campanholle G, Campanholle G, Silva RC, Martins JO, Landgraf MA, Paiva VN, Pacheco-Silva A, Câmara NOS. Renal ischemia and reperfusion injury modulate lung allergic inflammation probably altering TH1/TH2 balance by changing cellular signal transduction. Inflammation Research. 2011 ; 60 S140 res. P-172.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
NADPH-OXIDASE inhibition though apocynin modulates inflammatory process in renal ischemia and reperfusion injury (2011)
Hayashida, C. Y.; Braga, T. T.; Silva, R. C.; Amano, M. T.; Cunha, C. S.; Hiyane, Meire Ioshie; Correa-Costa, M.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
HAYASHIDA, C. Y. et al. NADPH-OXIDASE inhibition though apocynin modulates inflammatory process in renal ischemia and reperfusion injury. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Hayashida, C. Y., Braga, T. T., Silva, R. C., Amano, M. T., Cunha, C. S., Hiyane, M. I., et al. (2011). NADPH-OXIDASE inhibition though apocynin modulates inflammatory process in renal ischemia and reperfusion injury. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Hayashida CY, Braga TT, Silva RC, Amano MT, Cunha CS, Hiyane MI, Correa-Costa M, Câmara NOS. NADPH-OXIDASE inhibition though apocynin modulates inflammatory process in renal ischemia and reperfusion injury. Inflammation Research. 2011 ; 60 S124 res. P-127.[citado 2024 nov. 08 ]
Vancouver
Hayashida CY, Braga TT, Silva RC, Amano MT, Cunha CS, Hiyane MI, Correa-Costa M, Câmara NOS. NADPH-OXIDASE inhibition though apocynin modulates inflammatory process in renal ischemia and reperfusion injury. Inflammation Research. 2011 ; 60 S124 res. P-127.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Role of toll like receptors 2 and 4, and MYD88 molecule in an experimental model of tubulointerstitial nephritis (2011)
Correa-Costa, M.; Braga, T. T.; Semedo, P.; Hayashida, C. Y.; Elias, R. M.; Silva-Cunha, C.; Hiyane, Meire Ioshie; Fujihara, C.; Zatz, R.; Pacheco-Silva, A.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
CORREA-COSTA, M. et al. Role of toll like receptors 2 and 4, and MYD88 molecule in an experimental model of tubulointerstitial nephritis. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Correa-Costa, M., Braga, T. T., Semedo, P., Hayashida, C. Y., Elias, R. M., Silva-Cunha, C., et al. (2011). Role of toll like receptors 2 and 4, and MYD88 molecule in an experimental model of tubulointerstitial nephritis. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Correa-Costa M, Braga TT, Semedo P, Hayashida CY, Elias RM, Silva-Cunha C, Hiyane MI, Fujihara C, Zatz R, Pacheco-Silva A, Câmara NOS. Role of toll like receptors 2 and 4, and MYD88 molecule in an experimental model of tubulointerstitial nephritis. Inflammation Research. 2011 ; 60 S248 res. P-497.[citado 2024 nov. 08 ]
Vancouver
Correa-Costa M, Braga TT, Semedo P, Hayashida CY, Elias RM, Silva-Cunha C, Hiyane MI, Fujihara C, Zatz R, Pacheco-Silva A, Câmara NOS. Role of toll like receptors 2 and 4, and MYD88 molecule in an experimental model of tubulointerstitial nephritis. Inflammation Research. 2011 ; 60 S248 res. P-497.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Using Transcriptional profiling to investigate proximal tubular dysfunction in kidney transplant patients with higher risk for development of chronic allograft dysfunction (2011)
Renesto, P. G.; Câmara, Niels Olsen Saraiva ; Ramos, P. L.; Azevedo, H.; Chinen, R.; Moreira-Filho, C. A.; Pacheco-Silva, A.
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
RENESTO, P. G. et al. Using Transcriptional profiling to investigate proximal tubular dysfunction in kidney transplant patients with higher risk for development of chronic allograft dysfunction. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Renesto, P. G., Câmara, N. O. S., Ramos, P. L., Azevedo, H., Chinen, R., Moreira-Filho, C. A., & Pacheco-Silva, A. (2011). Using Transcriptional profiling to investigate proximal tubular dysfunction in kidney transplant patients with higher risk for development of chronic allograft dysfunction. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Renesto PG, Câmara NOS, Ramos PL, Azevedo H, Chinen R, Moreira-Filho CA, Pacheco-Silva A. Using Transcriptional profiling to investigate proximal tubular dysfunction in kidney transplant patients with higher risk for development of chronic allograft dysfunction. Inflammation Research. 2011 ; 60 S250 res. P-503.[citado 2024 nov. 08 ]
Vancouver
Renesto PG, Câmara NOS, Ramos PL, Azevedo H, Chinen R, Moreira-Filho CA, Pacheco-Silva A. Using Transcriptional profiling to investigate proximal tubular dysfunction in kidney transplant patients with higher risk for development of chronic allograft dysfunction. Inflammation Research. 2011 ; 60 S250 res. P-503.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Immune modulation by human papillomavirus associated tumors: from the inflammatory infiltrate to secondary lynphoid organs (2011)
Lepique, Ana Paula
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
LEPIQUE, Ana Paula. Immune modulation by human papillomavirus associated tumors: from the inflammatory infiltrate to secondary lynphoid organs. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Lepique, A. P. (2011). Immune modulation by human papillomavirus associated tumors: from the inflammatory infiltrate to secondary lynphoid organs. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Lepique AP. Immune modulation by human papillomavirus associated tumors: from the inflammatory infiltrate to secondary lynphoid organs. Inflammation Research. 2011 ; 60 S193 res. P-330.[citado 2024 nov. 08 ]
Vancouver
Lepique AP. Immune modulation by human papillomavirus associated tumors: from the inflammatory infiltrate to secondary lynphoid organs. Inflammation Research. 2011 ; 60 S193 res. P-330.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis (2011)
Braga, T. T.; Correa-Costa, M.; Castoldi, A.; Oliveira, C. D. O.; Hiyane, Meire Ioshie; Silva-Cunha, C.; Albe, P.; Reis, M. A.; Faleiros, A. C. G.; Gonçalves, G. M.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
BRAGA, T. T. et al. Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Braga, T. T., Correa-Costa, M., Castoldi, A., Oliveira, C. D. O., Hiyane, M. I., Silva-Cunha, C., et al. (2011). Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Braga TT, Correa-Costa M, Castoldi A, Oliveira CDO, Hiyane MI, Silva-Cunha C, Albe P, Reis MA, Faleiros ACG, Gonçalves GM, Câmara NOS. Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. 2011 ; 60 S63 res. OC-083.[citado 2024 nov. 08 ]
Vancouver
Braga TT, Correa-Costa M, Castoldi A, Oliveira CDO, Hiyane MI, Silva-Cunha C, Albe P, Reis MA, Faleiros ACG, Gonçalves GM, Câmara NOS. Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. 2011 ; 60 S63 res. OC-083.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Adipose tissue-derived stem cell treatment prevents renal disease progression (2011)
Donizetti-Oliveira, C.; Semedo, P.; Burgos-Silva, M.; Canedeze, M. A.; Costa-Malheiros, D.M.A.; Reis, M.; Pacheco-Silva, A.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DONIZETTI-OLIVEIRA, C. et al. Adipose tissue-derived stem cell treatment prevents renal disease progression. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Donizetti-Oliveira, C., Semedo, P., Burgos-Silva, M., Canedeze, M. A., Costa-Malheiros, D. M. A., Reis, M., et al. (2011). Adipose tissue-derived stem cell treatment prevents renal disease progression. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Donizetti-Oliveira C, Semedo P, Burgos-Silva M, Canedeze MA, Costa-Malheiros DMA, Reis M, Pacheco-Silva A, Câmara NOS. Adipose tissue-derived stem cell treatment prevents renal disease progression. Inflammation Research. 2011 ; 60 S75 res. OC-114.[citado 2024 nov. 08 ]
Vancouver
Donizetti-Oliveira C, Semedo P, Burgos-Silva M, Canedeze MA, Costa-Malheiros DMA, Reis M, Pacheco-Silva A, Câmara NOS. Adipose tissue-derived stem cell treatment prevents renal disease progression. Inflammation Research. 2011 ; 60 S75 res. OC-114.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Signaling through TOLL-LIKE receptors enhances susceptibility to asthma in mice exposed to enviromental pollutant during neonatal period (2011)
Santos, K. T.; Florenzano, J.; Peron, Jean Pierre Schatzmann ; Baptista, E. E. A.; Câmara, Niels Olsen Saraiva ; Muscará, Marcelo Nicolás ; Costa, Soraia Katia Pereira
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Subjects: IMUNOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, K. T. et al. Signaling through TOLL-LIKE receptors enhances susceptibility to asthma in mice exposed to enviromental pollutant during neonatal period. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Santos, K. T., Florenzano, J., Peron, J. P. S., Baptista, E. E. A., Câmara, N. O. S., Muscará, M. N., & Costa, S. K. P. (2011). Signaling through TOLL-LIKE receptors enhances susceptibility to asthma in mice exposed to enviromental pollutant during neonatal period. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Santos KT, Florenzano J, Peron JPS, Baptista EEA, Câmara NOS, Muscará MN, Costa SKP. Signaling through TOLL-LIKE receptors enhances susceptibility to asthma in mice exposed to enviromental pollutant during neonatal period. Inflammation Research. 2011 ; 60 S174 res. P-276.[citado 2024 nov. 08 ]
Vancouver
Santos KT, Florenzano J, Peron JPS, Baptista EEA, Câmara NOS, Muscará MN, Costa SKP. Signaling through TOLL-LIKE receptors enhances susceptibility to asthma in mice exposed to enviromental pollutant during neonatal period. Inflammation Research. 2011 ; 60 S174 res. P-276.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Modulation by estradiol of the acute lung inflammation after intestinal ischaemiareperfusion (I/R) in female and male rats (2011)
Breithaupt-Faloppa, A. C.; Vitoretti, L. B.; Romero, D. C.; Câmara, Niels Olsen Saraiva ; Bertoni, J. d. A.; Oliveira-Filho, Ricardo Moreira; Vargaftig, Bernardo Boris Jorge; Lima, Wothan Tavares de
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Subjects: FARMACOLOGIA, IMUNOLOGIA, PULMÃO, ISQUEMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BREITHAUPT-FALOPPA, A. C. et al. Modulation by estradiol of the acute lung inflammation after intestinal ischaemiareperfusion (I/R) in female and male rats. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Breithaupt-Faloppa, A. C., Vitoretti, L. B., Romero, D. C., Câmara, N. O. S., Bertoni, J. d. A., Oliveira-Filho, R. M., et al. (2011). Modulation by estradiol of the acute lung inflammation after intestinal ischaemiareperfusion (I/R) in female and male rats. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Breithaupt-Faloppa AC, Vitoretti LB, Romero DC, Câmara NOS, Bertoni J d. A, Oliveira-Filho RM, Vargaftig BBJ, Lima WT de. Modulation by estradiol of the acute lung inflammation after intestinal ischaemiareperfusion (I/R) in female and male rats. Inflammation Research. 2011 ; 60 S29 res. SP-25.[citado 2024 nov. 08 ]
Vancouver
Breithaupt-Faloppa AC, Vitoretti LB, Romero DC, Câmara NOS, Bertoni J d. A, Oliveira-Filho RM, Vargaftig BBJ, Lima WT de. Modulation by estradiol of the acute lung inflammation after intestinal ischaemiareperfusion (I/R) in female and male rats. Inflammation Research. 2011 ; 60 S29 res. SP-25.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Oxidative stress and modification of renal vascular permeability are associated with acute kidney injury during P. Berghei Anka infection (2011)
Elias, R. M.; Barreto, C.; Costa, M. C.; Silva, R. C.; Hayashida, C. Y.; Hiyane, Meire Ioshie; Seguro, A. C.; Pacheco-Silva, A.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ELIAS, R. M. et al. Oxidative stress and modification of renal vascular permeability are associated with acute kidney injury during P. Berghei Anka infection. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Elias, R. M., Barreto, C., Costa, M. C., Silva, R. C., Hayashida, C. Y., Hiyane, M. I., et al. (2011). Oxidative stress and modification of renal vascular permeability are associated with acute kidney injury during P. Berghei Anka infection. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Elias RM, Barreto C, Costa MC, Silva RC, Hayashida CY, Hiyane MI, Seguro AC, Pacheco-Silva A, Câmara NOS. Oxidative stress and modification of renal vascular permeability are associated with acute kidney injury during P. Berghei Anka infection. Inflammation Research. 2011 ; 60 S123 res. P-122.[citado 2024 nov. 08 ]
Vancouver
Elias RM, Barreto C, Costa MC, Silva RC, Hayashida CY, Hiyane MI, Seguro AC, Pacheco-Silva A, Câmara NOS. Oxidative stress and modification of renal vascular permeability are associated with acute kidney injury during P. Berghei Anka infection. Inflammation Research. 2011 ; 60 S123 res. P-122.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
The role of dendritic cells in CD4+ T cells modulation in kidney ischemia and reperfusion injury (2011)
Amano, M.; Correa-Costa, M.; Miyagi, M.; Hiyane, Meire Ioshie; Silva Filho, A. P.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMANO, M. et al. The role of dendritic cells in CD4+ T cells modulation in kidney ischemia and reperfusion injury. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Amano, M., Correa-Costa, M., Miyagi, M., Hiyane, M. I., Silva Filho, A. P., & Câmara, N. O. S. (2011). The role of dendritic cells in CD4+ T cells modulation in kidney ischemia and reperfusion injury. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Amano M, Correa-Costa M, Miyagi M, Hiyane MI, Silva Filho AP, Câmara NOS. The role of dendritic cells in CD4+ T cells modulation in kidney ischemia and reperfusion injury. Inflammation Research. 2011 ; 60 S251 res. P-506.[citado 2024 nov. 08 ]
Vancouver
Amano M, Correa-Costa M, Miyagi M, Hiyane MI, Silva Filho AP, Câmara NOS. The role of dendritic cells in CD4+ T cells modulation in kidney ischemia and reperfusion injury. Inflammation Research. 2011 ; 60 S251 res. P-506.[citado 2024 nov. 08 ]
Trabalho de evento-resumo periodico
Bradykinin receptors role on inflammatory reaction induced during adriamycin nephropathy (2011)
Pereira, R. L.; Buscariollo, B.; Correa-Costa, M.; Semedo, P.; Oliveira, C.; Reis, V.; Maquigussa, E.; Cenedeze, M.; Araujo, R.; Braga, T.; Soares, M.; Moura, I.; Malheiros, D.; Castoldi, A.; Burgos, M.; Vieira, P.; Pacheco-Silva, A.; Keller, A.; Câmara, Niels Olsen Saraiva
Source: Inflammation Research. Conference titles: The world congress on inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, R. L. et al. Bradykinin receptors role on inflammatory reaction induced during adriamycin nephropathy. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 08 nov. 2024. , 2011
APA
Pereira, R. L., Buscariollo, B., Correa-Costa, M., Semedo, P., Oliveira, C., Reis, V., et al. (2011). Bradykinin receptors role on inflammatory reaction induced during adriamycin nephropathy. Inflammation Research. Paris: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Pereira RL, Buscariollo B, Correa-Costa M, Semedo P, Oliveira C, Reis V, Maquigussa E, Cenedeze M, Araujo R, Braga T, Soares M, Moura I, Malheiros D, Castoldi A, Burgos M, Vieira P, Pacheco-Silva A, Keller A, Câmara NOS. Bradykinin receptors role on inflammatory reaction induced during adriamycin nephropathy. Inflammation Research. 2011 ; 60 S251 res. P-506.[citado 2024 nov. 08 ]
Vancouver
Pereira RL, Buscariollo B, Correa-Costa M, Semedo P, Oliveira C, Reis V, Maquigussa E, Cenedeze M, Araujo R, Braga T, Soares M, Moura I, Malheiros D, Castoldi A, Burgos M, Vieira P, Pacheco-Silva A, Keller A, Câmara NOS. Bradykinin receptors role on inflammatory reaction induced during adriamycin nephropathy. Inflammation Research. 2011 ; 60 S251 res. P-506.[citado 2024 nov. 08 ]

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