Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis (2011)
- Authors:
- USP affiliated authors: HIYANE, MEIRE IOSHIE - ICB ; CÂMARA, NIELS OLSEN SARAIVA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Inflammation Research
- ISSN: 1521-6616
- Volume/Número/Paginação/Ano: v. 60, Supplement 1, p. S63 res. OC-083, 2011
- Conference titles: The world congress on inflammation
-
ABNT
BRAGA, T. T.; CORREA-COSTA, M.; CASTOLDI, A.; et al. Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research[S.l: s.n.], 2011. -
APA
Braga, T. T., Correa-Costa, M., Castoldi, A., Oliveira, C. D. O., Hiyane, M. I., Silva-Cunha, C., et al. (2011). Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. Paris. -
NLM
Braga TT, Correa-Costa M, Castoldi A, Oliveira CDO, Hiyane MI, Silva-Cunha C, Albe P, Reis MA, Faleiros ACG, Gonçalves GM, Camara NOS. Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. 2011 ; 60 S63 res. OC-083. -
Vancouver
Braga TT, Correa-Costa M, Castoldi A, Oliveira CDO, Hiyane MI, Silva-Cunha C, Albe P, Reis MA, Faleiros ACG, Gonçalves GM, Camara NOS. Uric acid signalling via adaptor molecule MYD88 and M1/M2 macrophage balance in the development of renal fibrosis. Inflammation Research. 2011 ; 60 S63 res. OC-083. - Reduction of cytokinases/chemokines expression attenuates the allergic lung inflammation in leptin-deficient mice
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- Acute renal injury modulates phagocytic and microbicide activities macrophage model of experimental allergic lung inflammation in rats
- Acetylcholine and renal fibrosis
- TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis
- Adipose tissue-derived stem cells reduce acute and chronic kidney damage in mice
- Exogenous leptin modulates acute lung inflammation induced by LPS in mice
- Role of toll like receptors 2 and 4, and MYD88 molecule in an experimental model of tubulointerstitial nephritis
- NADPH-OXIDASE inhibition though apocynin modulates inflammatory process in renal ischemia and reperfusion injury
- Balance between the two kinin receptors in the progression of experimental focal and segmental glomerulosclerosis in mice
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