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Artigo de periodico
Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia (2022)
Lima, Keli ; Pereira-Martins Diego A. ; Miranda, Lívia Bassani Lins de ; Coelho-Silva, Juan L. ; Leandro, Giovana da Silva ; Isabel Weinhäuser ; Cavaglieri, Rita de Cássia ; Silva, Wellington F.; Leal, Aline Medeiros; Lange, Ana Paula Alencar de Lima; Velloso, Elvira Deolinda Rodrigues Pereira ; Griessinger, Emmanuel ; Hilberink, Jacobien R. ; Huls, Gerwin A. ; Schuringa, Jan Jacob ; Rego, Eduardo M. ; Machado Neto, João Agostinho
Source: Blood. Unidades: FM, ICB, FMRP
Subjects: FARMACOLOGIA, LEUCEMIA MIELOIDE AGUDA, NEOPLASIAS POR TIPO HISTOLÓGICO, AMINOÁCIDOS, PROTEÍNAS QUINASES, DANO AO DNA, MITOCÔNDRIAS, HOMEOSTASE, LISOSSOMOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Keli et al. Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia. Blood, v. 140, p. 5965–5966, 2022Tradução . . Disponível em: https://doi.org/10.1182/blood-2022-165172. Acesso em: 18 nov. 2024.
APA
Lima, K., Pereira-Martins Diego A.,, Miranda, L. B. L. de, Coelho-Silva, J. L., Leandro, G. da S., Isabel Weinhäuser,, et al. (2022). Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia. Blood, 140, 5965–5966. doi:10.1182/blood-2022-165172
NLM
Lima K, Pereira-Martins Diego A., Miranda LBL de, Coelho-Silva JL, Leandro G da S, Isabel Weinhäuser, Cavaglieri R de C, Silva WF, Leal AM, Lange APA de L, Velloso EDRP, Griessinger E, Hilberink JR, Huls GA, Schuringa JJ, Rego EM, Machado Neto JA. Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia [Internet]. Blood. 2022 ; 140 5965–5966.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1182/blood-2022-165172
Vancouver
Lima K, Pereira-Martins Diego A., Miranda LBL de, Coelho-Silva JL, Leandro G da S, Isabel Weinhäuser, Cavaglieri R de C, Silva WF, Leal AM, Lange APA de L, Velloso EDRP, Griessinger E, Hilberink JR, Huls GA, Schuringa JJ, Rego EM, Machado Neto JA. Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia [Internet]. Blood. 2022 ; 140 5965–5966.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1182/blood-2022-165172
Artigo de periodico
The Angiotensin II Type 1 receptor-associated protein attenuates Angiotensin II-mediated inhibition of the renal outer medullary potassium channel in collecting duct cells (2021)
Polidoro, Juliano Zequini ; Rebouças, Nancy Amaral ; Girardi, Adriana Castello Costa
Source: Frontiers in Physiology. Unidades: ICB, FM
Subjects: FISIOLOGIA, ANGIOTENSINA II, HOMEOSTASE, RECEPTORES DE ANGIOTENSINA, FLUORESCÊNCIA, PROTEÍNAS QUINASES
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ABNT
POLIDORO, Juliano Zequini e REBOUÇAS, Nancy Amaral e GIRARDI, Adriana Castello Costa. The Angiotensin II Type 1 receptor-associated protein attenuates Angiotensin II-mediated inhibition of the renal outer medullary potassium channel in collecting duct cells. Frontiers in Physiology, v. 12, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphys.2021.642409. Acesso em: 18 nov. 2024.
APA
Polidoro, J. Z., Rebouças, N. A., & Girardi, A. C. C. (2021). The Angiotensin II Type 1 receptor-associated protein attenuates Angiotensin II-mediated inhibition of the renal outer medullary potassium channel in collecting duct cells. Frontiers in Physiology, 12, 1-10. doi:10.3389/fphys.2021.642409
NLM
Polidoro JZ, Rebouças NA, Girardi ACC. The Angiotensin II Type 1 receptor-associated protein attenuates Angiotensin II-mediated inhibition of the renal outer medullary potassium channel in collecting duct cells [Internet]. Frontiers in Physiology. 2021 ; 12 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fphys.2021.642409
Vancouver
Polidoro JZ, Rebouças NA, Girardi ACC. The Angiotensin II Type 1 receptor-associated protein attenuates Angiotensin II-mediated inhibition of the renal outer medullary potassium channel in collecting duct cells [Internet]. Frontiers in Physiology. 2021 ; 12 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fphys.2021.642409
Artigo de periodico
Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise (2019)
Batatinha, Helena Angelica Pereira ; Diniz, Tiego Aparecido ; Teixeira, Alexandre Abilio de Souza ; Krüger, Karsten; Rosa Neto, José Cesar
Source: Journal of Cellular Physiology. Unidade: ICB
Subjects: BIOLOGIA CELULAR, DOENÇAS NEURODEGENERATIVAS, EXERCÍCIO FÍSICO, NEOPLASIAS, ENVELHECIMENTO, FATORES DE RISCO, INFLAMAÇÃO, HOMEOSTASE, PROTEÍNAS QUINASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BATATINHA, Helena Angelica Pereira et al. Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise. Journal of Cellular Physiology, v. 234, n. 9, p. 14883-14895, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.28318. Acesso em: 18 nov. 2024.
APA
Batatinha, H. A. P., Diniz, T. A., Teixeira, A. A. de S., Krüger, K., & Rosa Neto, J. C. (2019). Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise. Journal of Cellular Physiology, 234( 9), 14883-14895. doi:10.1002/jcp.28318
NLM
Batatinha HAP, Diniz TA, Teixeira AA de S, Krüger K, Rosa Neto JC. Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise [Internet]. Journal of Cellular Physiology. 2019 ; 234( 9): 14883-14895.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/jcp.28318
Vancouver
Batatinha HAP, Diniz TA, Teixeira AA de S, Krüger K, Rosa Neto JC. Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise [Internet]. Journal of Cellular Physiology. 2019 ; 234( 9): 14883-14895.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/jcp.28318
Artigo de periodico
The Aspergillus fumigatus pkcAG579R mutant is defective in the activation of the cell wall integrity pathway but is dispensable for virulence in a neutropenic mouse infection model (2015)
Rocha, Marina Campos; Godoy, Krissia Franco de; Castro, Patrícia Alves de; Hori, Juliana Issa; Bom, Vinicius Leite Pedro; Brown, Neil Andrew; Cunha, Anderson Ferreira da; Goldman, Gustavo Henrique; Malavazi, Iran
Source: PLOS One. Unidade: FCFRP
Subjects: ASPERGILLUS, HOMEOSTASE, DOENÇA PULMONAR (ESPECIALIDADE), FUNGOS, PROTEÍNAS QUINASES, ANTIFÚNGICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROCHA, Marina Campos et al. The Aspergillus fumigatus pkcAG579R mutant is defective in the activation of the cell wall integrity pathway but is dispensable for virulence in a neutropenic mouse infection model. PLOS One, v. 10, n. 8, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0135195. Acesso em: 18 nov. 2024.
APA
Rocha, M. C., Godoy, K. F. de, Castro, P. A. de, Hori, J. I., Bom, V. L. P., Brown, N. A., et al. (2015). The Aspergillus fumigatus pkcAG579R mutant is defective in the activation of the cell wall integrity pathway but is dispensable for virulence in a neutropenic mouse infection model. PLOS One, 10( 8). doi:10.1371/journal.pone.0135195
NLM
Rocha MC, Godoy KF de, Castro PA de, Hori JI, Bom VLP, Brown NA, Cunha AF da, Goldman GH, Malavazi I. The Aspergillus fumigatus pkcAG579R mutant is defective in the activation of the cell wall integrity pathway but is dispensable for virulence in a neutropenic mouse infection model [Internet]. PLOS One. 2015 ; 10( 8):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0135195
Vancouver
Rocha MC, Godoy KF de, Castro PA de, Hori JI, Bom VLP, Brown NA, Cunha AF da, Goldman GH, Malavazi I. The Aspergillus fumigatus pkcAG579R mutant is defective in the activation of the cell wall integrity pathway but is dispensable for virulence in a neutropenic mouse infection model [Internet]. PLOS One. 2015 ; 10( 8):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0135195

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