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Artigo de periodico
Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation (2016)
Meyerovich, Kira; Ortis, Fernanda ; Allagnat, Florent; Cardozo, Alessandra K.
Source: Journal of Molecular Endocrinology. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MEYEROVICH, Kira et al. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of Molecular Endocrinology, v. 57, n. 1, p. R1-R17, 2016Tradução . . Disponível em: https://doi.org/10.1530/JME-15-0306. Acesso em: 08 nov. 2024.
APA
Meyerovich, K., Ortis, F., Allagnat, F., & Cardozo, A. K. (2016). Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of Molecular Endocrinology, 57( 1), R1-R17. doi:10.1530/JME-15-0306
NLM
Meyerovich K, Ortis F, Allagnat F, Cardozo AK. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation [Internet]. Journal of Molecular Endocrinology. 2016 ; 57( 1): R1-R17.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1530/JME-15-0306
Vancouver
Meyerovich K, Ortis F, Allagnat F, Cardozo AK. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation [Internet]. Journal of Molecular Endocrinology. 2016 ; 57( 1): R1-R17.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1530/JME-15-0306
Artigo de periodico
The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro (2016)
Meyerovich, Kira; Fukaya, Makiko; Terra, Leticia F.; Ortis, Fernanda ; Eiziriki, Decio L.; Cardozo, Alessandra K.
Source: Diabetologia. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MEYEROVICH, Kira et al. The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro. Diabetologia, v. 59, n. 3, p. 512-521, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00125-015-3817-z. Acesso em: 08 nov. 2024.
APA
Meyerovich, K., Fukaya, M., Terra, L. F., Ortis, F., Eiziriki, D. L., & Cardozo, A. K. (2016). The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro. Diabetologia, 59( 3), 512-521. doi:10.1007/s00125-015-3817-z
NLM
Meyerovich K, Fukaya M, Terra LF, Ortis F, Eiziriki DL, Cardozo AK. The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro [Internet]. Diabetologia. 2016 ; 59( 3): 512-521.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1007/s00125-015-3817-z
Vancouver
Meyerovich K, Fukaya M, Terra LF, Ortis F, Eiziriki DL, Cardozo AK. The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro [Internet]. Diabetologia. 2016 ; 59( 3): 512-521.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1007/s00125-015-3817-z
Artigo de periodico
Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling (2014)
Protzek, André O. P.; Costa-Júnior, José M.; Rezende, Luiz F.; Santos, Gustavo J.; Araújo, Tiago Gomes; Vettorazzi, Jean F.; Ortis, Fernanda ; Carneiro, Everardo M.; Rafacho, Alex; Boschero, Antonio C.
Source: International Journal of Endocrinology. Unidade: ICB
Subjects: HISTOLOGIA, INSULINA (RESISTÊNCIA), CAMUNDONGOS, RATOS WISTAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PROTZEK, André O. P. et al. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling. International Journal of Endocrinology, v. 2014, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/983453. Acesso em: 08 nov. 2024.
APA
Protzek, A. O. P., Costa-Júnior, J. M., Rezende, L. F., Santos, G. J., Araújo, T. G., Vettorazzi, J. F., et al. (2014). Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling. International Journal of Endocrinology, 2014, 1-14. doi:10.1155/2014/983453
NLM
Protzek AOP, Costa-Júnior JM, Rezende LF, Santos GJ, Araújo TG, Vettorazzi JF, Ortis F, Carneiro EM, Rafacho A, Boschero AC. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-14.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1155/2014/983453
Vancouver
Protzek AOP, Costa-Júnior JM, Rezende LF, Santos GJ, Araújo TG, Vettorazzi JF, Ortis F, Carneiro EM, Rafacho A, Boschero AC. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-14.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1155/2014/983453
Artigo de periodico
Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII (2014)
Santos, Gustavo Jorge dos; Ferreira, Sandra Mara; Ortis, Fernanda ; Rezende, Luiz Fernando; Li, Chengyang; Naji, Ali; Carneiro, Everardo Magalhães; Kaestner, Klaus H.; Boschero, Antonio Carlos
Source: Molecular Metabolism. Unidade: ICB
Subjects: HISTOLOGIA, METABOLISMO, SECREÇÃO, INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Gustavo Jorge dos et al. Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII. Molecular Metabolism, v. 3, n. 4, p. 484-489, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2014.03.011. Acesso em: 08 nov. 2024.
APA
Santos, G. J. dos, Ferreira, S. M., Ortis, F., Rezende, L. F., Li, C., Naji, A., et al. (2014). Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII. Molecular Metabolism, 3( 4), 484-489. doi:10.1016/j.molmet.2014.03.011
NLM
Santos GJ dos, Ferreira SM, Ortis F, Rezende LF, Li C, Naji A, Carneiro EM, Kaestner KH, Boschero AC. Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII [Internet]. Molecular Metabolism. 2014 ; 3( 4): 484-489.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.molmet.2014.03.011
Vancouver
Santos GJ dos, Ferreira SM, Ortis F, Rezende LF, Li C, Naji A, Carneiro EM, Kaestner KH, Boschero AC. Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII [Internet]. Molecular Metabolism. 2014 ; 3( 4): 484-489.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.molmet.2014.03.011

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