Source: Clinical Autonomic Research. Conference titles: Joint Meeting of The International Society for Autonomic Neuroscience and The American Autonomic Society (ISAN/AAS). Unidade: FMRP
Subjects: FISIOLOGIA, RATOS, NEUROTRANSMISSORES
ABNT
ALMADO, C. E. e MACHADO, Benedito Honório e LEÃO, Ricardo Maurício Xavier. Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. Heidelberg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 04 out. 2024. , 2011APA
Almado, C. E., Machado, B. H., & Leão, R. M. X. (2011). Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. Heidelberg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.NLM
Almado CE, Machado BH, Leão RMX. Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. 2011 ; 21( 4): 262-263.[citado 2024 out. 04 ]Vancouver
Almado CE, Machado BH, Leão RMX. Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. 2011 ; 21( 4): 262-263.[citado 2024 out. 04 ]