Filtros : "FARMACOLOGIA" "2016" Removidos: "Guimarães, Francisco Silveira" "Simpósio Internacional de Iniciação Científica (SIICUSP)" Limpar

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  • Source: European Journal of Pharmacology. Unidade: FCFRP

    Subjects: SISTEMA RENINA-ANGIOTENSINA, ANGIOTENSINAS, ENZIMAS, RECEPTORES DE ANGIOTENSINA, FARMACOLOGIA

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      PERNOMIAN, Larissa et al. Pharmacological significance of the interplay between angiotensin receptors: MAS receptors as putative final mediators of the effects elicited by angiotensin AT1 receptors antagonists. European Journal of Pharmacology, v. 769, p. 143-146, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2015.11.009. Acesso em: 19 nov. 2024.
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      Pernomian, L., Pernomian, L., Gomes, M. S., & Silva, C. H. T. de P. da. (2016). Pharmacological significance of the interplay between angiotensin receptors: MAS receptors as putative final mediators of the effects elicited by angiotensin AT1 receptors antagonists. European Journal of Pharmacology, 769, 143-146. doi:10.1016/j.ejphar.2015.11.009
    • NLM

      Pernomian L, Pernomian L, Gomes MS, Silva CHT de P da. Pharmacological significance of the interplay between angiotensin receptors: MAS receptors as putative final mediators of the effects elicited by angiotensin AT1 receptors antagonists [Internet]. European Journal of Pharmacology. 2016 ; 769 143-146.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2015.11.009
    • Vancouver

      Pernomian L, Pernomian L, Gomes MS, Silva CHT de P da. Pharmacological significance of the interplay between angiotensin receptors: MAS receptors as putative final mediators of the effects elicited by angiotensin AT1 receptors antagonists [Internet]. European Journal of Pharmacology. 2016 ; 769 143-146.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2015.11.009
  • Source: Addiction Biology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      QUADIR, Sema G. et al. Homer2 regulates alcohol and stress cross-sensitization. Addiction Biology, v. 21, n. 3, p. 613-633, 2016Tradução . . Disponível em: https://doi.org/10.1111/adb.12252. Acesso em: 19 nov. 2024.
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      Quadir, S. G., Santos, J. R. B. dos, Campbell, R., Wroten, M. G., Singh, N., Holloway, J. J., et al. (2016). Homer2 regulates alcohol and stress cross-sensitization. Addiction Biology, 21( 3), 613-633. doi:10.1111/adb.12252
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      Quadir SG, Santos JRB dos, Campbell R, Wroten MG, Singh N, Holloway JJ, Bal SK, Camarini R, Szumlinski K. Homer2 regulates alcohol and stress cross-sensitization [Internet]. Addiction Biology. 2016 ; 21( 3): 613-633.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/adb.12252
    • Vancouver

      Quadir SG, Santos JRB dos, Campbell R, Wroten MG, Singh N, Holloway JJ, Bal SK, Camarini R, Szumlinski K. Homer2 regulates alcohol and stress cross-sensitization [Internet]. Addiction Biology. 2016 ; 21( 3): 613-633.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/adb.12252
  • Source: Brain Research Bulletin. Unidade: ICB

    Assunto: FARMACOLOGIA

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      CAMARINI, Rosana e PAUTASSI, Ricardo Marcos. Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression. Brain Research Bulletin, v. 125, p. 53-78, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2016.04.006. Acesso em: 19 nov. 2024.
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      Camarini, R., & Pautassi, R. M. (2016). Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression. Brain Research Bulletin, 125, 53-78. doi:10.1016/j.brainresbull.2016.04.006
    • NLM

      Camarini R, Pautassi RM. Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression [Internet]. Brain Research Bulletin. 2016 ; 125 53-78.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.brainresbull.2016.04.006
    • Vancouver

      Camarini R, Pautassi RM. Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression [Internet]. Brain Research Bulletin. 2016 ; 125 53-78.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.brainresbull.2016.04.006
  • Source: International Immunopharmacology. Unidade: ICB

    Subjects: FARMACOLOGIA, ÓLEOS ESSENCIAIS, CANELA

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      MENDES, Saulo J. F. et al. Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms. International Immunopharmacology, v. 34, p. 60-70, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2016.02.012. Acesso em: 19 nov. 2024.
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      Mendes, S. J. F., Sousa, F. I. A. F., Pereira, D. M. S., Ferro, T. A. F., Pereira, I. C. P., Silva, B. L. R., et al. (2016). Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms. International Immunopharmacology, 34, 60-70. doi:10.1016/j.intimp.2016.02.012
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      Mendes SJF, Sousa FIAF, Pereira DMS, Ferro TAF, Pereira ICP, Silva BLR, Pinheiro AJMCR, Mouchrek AQS, Monteiro-Neto V, Costa SKP, Nascimento JLM, Grisottoa MAG, Costa R da, Fernandes ES. Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms [Internet]. International Immunopharmacology. 2016 ;34 60-70.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.intimp.2016.02.012
    • Vancouver

      Mendes SJF, Sousa FIAF, Pereira DMS, Ferro TAF, Pereira ICP, Silva BLR, Pinheiro AJMCR, Mouchrek AQS, Monteiro-Neto V, Costa SKP, Nascimento JLM, Grisottoa MAG, Costa R da, Fernandes ES. Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms [Internet]. International Immunopharmacology. 2016 ;34 60-70.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.intimp.2016.02.012
  • Source: Sexual Medicine. Unidade: ICB

    Subjects: FARMACOLOGIA, DISFUNÇÃO ERÉTIL

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      GAGLIANO-JUCÁ, Thiago et al. Hydrochlorothiazide Potentiates Contractile Activity of Mouse Cavernosal Smooth Muscle. Sexual Medicine, v. 4, n. 2, p. e113-e123, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.esxm.2016.02.003. Acesso em: 19 nov. 2024.
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      Gagliano-Jucá, T., Napolitano, M., Carvalho, F. D. G. F., Campos, R., Mónica, F. Z., Claudino, M. A., et al. (2016). Hydrochlorothiazide Potentiates Contractile Activity of Mouse Cavernosal Smooth Muscle. Sexual Medicine, 4( 2), e113-e123. doi:10.1016/j.esxm.2016.02.003
    • NLM

      Gagliano-Jucá T, Napolitano M, Carvalho FDGF, Campos R, Mónica FZ, Claudino MA, Antunes E, Lopes AG, De Nucci G. Hydrochlorothiazide Potentiates Contractile Activity of Mouse Cavernosal Smooth Muscle [Internet]. Sexual Medicine. 2016 ; 4( 2): e113-e123.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.esxm.2016.02.003
    • Vancouver

      Gagliano-Jucá T, Napolitano M, Carvalho FDGF, Campos R, Mónica FZ, Claudino MA, Antunes E, Lopes AG, De Nucci G. Hydrochlorothiazide Potentiates Contractile Activity of Mouse Cavernosal Smooth Muscle [Internet]. Sexual Medicine. 2016 ; 4( 2): e113-e123.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.esxm.2016.02.003
  • Source: Frontiers in Endocrinology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      ORELLANA, Ana Maria et al. Cardiotonic steroids as modulators of neuroinflammation. Frontiers in Endocrinology, v. 7, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.3389/fendo.2016.00010. Acesso em: 19 nov. 2024.
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      Orellana, A. M., Kinoshita, P. F., Leite, J. A., Kawamoto, E. M., & Scavone, C. (2016). Cardiotonic steroids as modulators of neuroinflammation. Frontiers in Endocrinology, 7, 1-10. doi:10.3389/fendo.2016.00010
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      Orellana AM, Kinoshita PF, Leite JA, Kawamoto EM, Scavone C. Cardiotonic steroids as modulators of neuroinflammation [Internet]. Frontiers in Endocrinology. 2016 ; 7 1-10.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fendo.2016.00010
    • Vancouver

      Orellana AM, Kinoshita PF, Leite JA, Kawamoto EM, Scavone C. Cardiotonic steroids as modulators of neuroinflammation [Internet]. Frontiers in Endocrinology. 2016 ; 7 1-10.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fendo.2016.00010
  • Source: Journal of the Brazilian Chemical Society. Unidade: ICB

    Assunto: FARMACOLOGIA

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      RIBEIRO, Paulo R. V. et al. New epimeric spirostanol and furostanol-type steroidal saponins from Cestrum laevigatum L. Journal of the Brazilian Chemical Society, v. 27, n. 12, p. 2170-2180, 2016Tradução . . Disponível em: https://doi.org/10.5935/0103-5053.20160109. Acesso em: 19 nov. 2024.
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      Ribeiro, P. R. V., Braz-Filho, R., Araújo, A. J., Costa-Lotufo, L. V., Souza, L. G. S., Nobre Junior, H. V., et al. (2016). New epimeric spirostanol and furostanol-type steroidal saponins from Cestrum laevigatum L. Journal of the Brazilian Chemical Society, 27( 12), 2170-2180. doi:10.5935/0103-5053.20160109
    • NLM

      Ribeiro PRV, Braz-Filho R, Araújo AJ, Costa-Lotufo LV, Souza LGS, Nobre Junior HV, Silva CR da, Andrade Neto JB de, Silveira ER, Lima MAS. New epimeric spirostanol and furostanol-type steroidal saponins from Cestrum laevigatum L. [Internet]. Journal of the Brazilian Chemical Society. 2016 ; 27( 12): 2170-2180.[citado 2024 nov. 19 ] Available from: https://doi.org/10.5935/0103-5053.20160109
    • Vancouver

      Ribeiro PRV, Braz-Filho R, Araújo AJ, Costa-Lotufo LV, Souza LGS, Nobre Junior HV, Silva CR da, Andrade Neto JB de, Silveira ER, Lima MAS. New epimeric spirostanol and furostanol-type steroidal saponins from Cestrum laevigatum L. [Internet]. Journal of the Brazilian Chemical Society. 2016 ; 27( 12): 2170-2180.[citado 2024 nov. 19 ] Available from: https://doi.org/10.5935/0103-5053.20160109
  • Source: European Journal of Medicinal Chemistry. Unidade: ESALQ

    Subjects: PRÓPOLIS, FARMACOLOGIA

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      FREIRES, Irlan de Almeida e ALENCAR, Severino Matias de e ROSALEN, Pedro Luiz. A pharmacological perspective on the use of Brazilian Red Propolis and its isolated compounds against human diseases. European Journal of Medicinal Chemistry, v. 110, p. 267–279, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejmech.2016.01.033. Acesso em: 19 nov. 2024.
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      Freires, I. de A., Alencar, S. M. de, & Rosalen, P. L. (2016). A pharmacological perspective on the use of Brazilian Red Propolis and its isolated compounds against human diseases. European Journal of Medicinal Chemistry, 110, 267–279. doi:10.1016/j.ejmech.2016.01.033
    • NLM

      Freires I de A, Alencar SM de, Rosalen PL. A pharmacological perspective on the use of Brazilian Red Propolis and its isolated compounds against human diseases [Internet]. European Journal of Medicinal Chemistry. 2016 ; 110 267–279.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejmech.2016.01.033
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      Freires I de A, Alencar SM de, Rosalen PL. A pharmacological perspective on the use of Brazilian Red Propolis and its isolated compounds against human diseases [Internet]. European Journal of Medicinal Chemistry. 2016 ; 110 267–279.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejmech.2016.01.033
  • Source: Biomedical Chromatography. Unidade: FCFRP

    Subjects: COMPOSITAE, FARMACOLOGIA, CROMATOGRAFIA, ESPECTROMETRIA DE MASSAS, COMPOSTOS ORGÂNICOS

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      SOUSA, João Paulo Barreto de et al. Validation of analytical procedures using HPLC-ELSD to determine six sesquiterpene lactones in Eremanthus species. Biomedical Chromatography, v. 30, n. 3, p. 484-493, 2016Tradução . . Disponível em: https://doi.org/10.1002/bmc.3576. Acesso em: 19 nov. 2024.
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      Sousa, J. P. B. de, Nogueira, E. F., Ferreira, L. S., Lopes, N. P., & Lopes, J. L. C. (2016). Validation of analytical procedures using HPLC-ELSD to determine six sesquiterpene lactones in Eremanthus species. Biomedical Chromatography, 30( 3), 484-493. doi:10.1002/bmc.3576
    • NLM

      Sousa JPB de, Nogueira EF, Ferreira LS, Lopes NP, Lopes JLC. Validation of analytical procedures using HPLC-ELSD to determine six sesquiterpene lactones in Eremanthus species [Internet]. Biomedical Chromatography. 2016 ; 30( 3): 484-493.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1002/bmc.3576
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      Sousa JPB de, Nogueira EF, Ferreira LS, Lopes NP, Lopes JLC. Validation of analytical procedures using HPLC-ELSD to determine six sesquiterpene lactones in Eremanthus species [Internet]. Biomedical Chromatography. 2016 ; 30( 3): 484-493.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1002/bmc.3576
  • Source: Vascular Pharmacology. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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      COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 19 nov. 2024.
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      Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
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      Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
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      Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
  • Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: ICB

    Subjects: FARMACOLOGIA, DOENÇAS CARDIOVASCULARES, ANTIOXIDANTES

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      TOSTES, Rita C. et al. Reactive oxygen species: players in the cardiovascular effects oftestosterone. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 310, n. 1, p. R1-R14, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00392.2014. Acesso em: 19 nov. 2024.
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      Tostes, R. C., Carvalho, M. H. C., Carneiro, F. S., & Reckelhoff, J. F. (2016). Reactive oxygen species: players in the cardiovascular effects oftestosterone. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 310( 1), R1-R14. doi:10.1152/ajpregu.00392.2014
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      Tostes RC, Carvalho MHC, Carneiro FS, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects oftestosterone [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
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      Tostes RC, Carvalho MHC, Carneiro FS, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects oftestosterone [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
  • Source: Mediators of Inflammation. Unidade: ICB

    Subjects: FARMACOLOGIA, IMUNOLOGIA

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      BELIZÁRIO, José E. et al. Thymic and postthymic regulation of naïve CD4(+) T-cell lineage fates in humans and mice models. Mediators of Inflammation, v. 2016, p. 1-16, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/9523628. Acesso em: 19 nov. 2024.
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      Belizário, J. E., Brandão, W., Rossato, C., & Peron, J. P. S. (2016). Thymic and postthymic regulation of naïve CD4(+) T-cell lineage fates in humans and mice models. Mediators of Inflammation, 2016, 1-16. doi:10.1155/2016/9523628
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      Belizário JE, Brandão W, Rossato C, Peron JPS. Thymic and postthymic regulation of naïve CD4(+) T-cell lineage fates in humans and mice models [Internet]. Mediators of Inflammation. 2016 ; 2016 1-16.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2016/9523628
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      Belizário JE, Brandão W, Rossato C, Peron JPS. Thymic and postthymic regulation of naïve CD4(+) T-cell lineage fates in humans and mice models [Internet]. Mediators of Inflammation. 2016 ; 2016 1-16.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2016/9523628
  • Source: International Immunopharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      CAMPOS, Daiana et al. Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation. International Immunopharmacology, v. 39, p. 57-62, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2016.07.009. Acesso em: 19 nov. 2024.
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      Campos, D., Ravagnani, F. G., Gurgueira, S. A., Vercesi, A. E., Teixeira, S. A., Costa, S. K. P., et al. (2016). Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation. International Immunopharmacology, 39, 57-62. doi:10.1016/j.intimp.2016.07.009
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      Campos D, Ravagnani FG, Gurgueira SA, Vercesi AE, Teixeira SA, Costa SKP, Muscará MN, Ferreira HHA. Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation [Internet]. International Immunopharmacology. 2016 ; 39 57-62.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.intimp.2016.07.009
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      Campos D, Ravagnani FG, Gurgueira SA, Vercesi AE, Teixeira SA, Costa SKP, Muscará MN, Ferreira HHA. Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation [Internet]. International Immunopharmacology. 2016 ; 39 57-62.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.intimp.2016.07.009
  • Source: BMC Gastroenterology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      GAGLIANO-JUCÁ, Thiago et al. Rebamipide does not protect against naproxen-induced gastric damage: a randomized double-blind controlled trial. BMC Gastroenterology, v. 16, n. 1, p. 1-7, 2016Tradução . . Disponível em: https://doi.org/10.1186/s12876-016-0472-x. Acesso em: 19 nov. 2024.
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      Gagliano-Jucá, T., Moreno, R. A., Zaminelli, T., Napolitano, M., Magalhães, A. F. N., Carvalhaes, A., et al. (2016). Rebamipide does not protect against naproxen-induced gastric damage: a randomized double-blind controlled trial. BMC Gastroenterology, 16( 1), 1-7. doi:10.1186/s12876-016-0472-x
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      Gagliano-Jucá T, Moreno RA, Zaminelli T, Napolitano M, Magalhães AFN, Carvalhaes A, Trevisan MS, Wallace JL, De Nucci G. Rebamipide does not protect against naproxen-induced gastric damage: a randomized double-blind controlled trial [Internet]. BMC Gastroenterology. 2016 ; 16( 1): 1-7.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/s12876-016-0472-x
    • Vancouver

      Gagliano-Jucá T, Moreno RA, Zaminelli T, Napolitano M, Magalhães AFN, Carvalhaes A, Trevisan MS, Wallace JL, De Nucci G. Rebamipide does not protect against naproxen-induced gastric damage: a randomized double-blind controlled trial [Internet]. BMC Gastroenterology. 2016 ; 16( 1): 1-7.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/s12876-016-0472-x
  • Source: Autonomic Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      ALVES, Thales B. et al. GABA mechanisms of the nucleus of the solitary tract regulates the cardiovascular and sympathetic effects of moxonidine. Autonomic Neuroscience, v. 194, p. 1-7, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2015.11.001. Acesso em: 19 nov. 2024.
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      Alves, T. B., Totola, L. T., Takakura, A. C., Colombari, E., & Moreira, T. dos S. (2016). GABA mechanisms of the nucleus of the solitary tract regulates the cardiovascular and sympathetic effects of moxonidine. Autonomic Neuroscience, 194, 1-7. doi:10.1016/j.autneu.2015.11.001
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      Alves TB, Totola LT, Takakura AC, Colombari E, Moreira T dos S. GABA mechanisms of the nucleus of the solitary tract regulates the cardiovascular and sympathetic effects of moxonidine [Internet]. Autonomic Neuroscience. 2016 ; 194 1-7.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.autneu.2015.11.001
    • Vancouver

      Alves TB, Totola LT, Takakura AC, Colombari E, Moreira T dos S. GABA mechanisms of the nucleus of the solitary tract regulates the cardiovascular and sympathetic effects of moxonidine [Internet]. Autonomic Neuroscience. 2016 ; 194 1-7.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.autneu.2015.11.001
  • Source: Respiratory Physiology & Neurobiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      SILVA, Josiane N. et al. Neuroanatomical and physiological evidence that the retrotrapezoid nucleus/parafacial region regulates expiration in adult rats. Respiratory Physiology & Neurobiology, v. 227, p. 9-22, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.resp.2016.02.005. Acesso em: 19 nov. 2024.
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      Silva, J. N., Tanabe, F. M., Moreira, T. dos S., & Takakura, A. C. (2016). Neuroanatomical and physiological evidence that the retrotrapezoid nucleus/parafacial region regulates expiration in adult rats. Respiratory Physiology & Neurobiology, 227, 9-22. doi:10.1016/j.resp.2016.02.005
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      Silva JN, Tanabe FM, Moreira T dos S, Takakura AC. Neuroanatomical and physiological evidence that the retrotrapezoid nucleus/parafacial region regulates expiration in adult rats [Internet]. Respiratory Physiology & Neurobiology. 2016 ; 227 9-22.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.resp.2016.02.005
    • Vancouver

      Silva JN, Tanabe FM, Moreira T dos S, Takakura AC. Neuroanatomical and physiological evidence that the retrotrapezoid nucleus/parafacial region regulates expiration in adult rats [Internet]. Respiratory Physiology & Neurobiology. 2016 ; 227 9-22.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.resp.2016.02.005
  • Source: Journal of Neurophysiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      MOREIRA, Thiago dos Santos et al. Respiratory and autonomic dysfunction in congenital central hypoventilation syndrome. Journal of Neurophysiology, v. 116, n. 2, p. 742-752, 2016Tradução . . Disponível em: https://doi.org/10.1152/jn.00026.2016. Acesso em: 19 nov. 2024.
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      Moreira, T. dos S., Takakura, A. C., Czeisler, C., & Otero, J. J. (2016). Respiratory and autonomic dysfunction in congenital central hypoventilation syndrome. Journal of Neurophysiology, 116( 2), 742-752. doi:10.1152/jn.00026.2016
    • NLM

      Moreira T dos S, Takakura AC, Czeisler C, Otero JJ. Respiratory and autonomic dysfunction in congenital central hypoventilation syndrome [Internet]. Journal of Neurophysiology. 2016 ; 116( 2): 742-752.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/jn.00026.2016
    • Vancouver

      Moreira T dos S, Takakura AC, Czeisler C, Otero JJ. Respiratory and autonomic dysfunction in congenital central hypoventilation syndrome [Internet]. Journal of Neurophysiology. 2016 ; 116( 2): 742-752.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/jn.00026.2016
  • Source: Journal of Neurophysiology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      KUO, Fu-Shan et al. In vitro characterization of noradrenergic modulation of chemosensitive neurons in the retrotrapezoid nucleus. Journal of Neurophysiology, v. 116, n. 3, p. 1024-1035, 2016Tradução . . Disponível em: https://doi.org/10.1152/jn.00022.2016. Acesso em: 19 nov. 2024.
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      Kuo, F. -S., Falquetto, B., Chen, D., Oliveira, L. M., Takakura, A. C., & Mulkey, D. K. (2016). In vitro characterization of noradrenergic modulation of chemosensitive neurons in the retrotrapezoid nucleus. Journal of Neurophysiology, 116( 3), 1024-1035. doi:10.1152/jn.00022.2016
    • NLM

      Kuo F-S, Falquetto B, Chen D, Oliveira LM, Takakura AC, Mulkey DK. In vitro characterization of noradrenergic modulation of chemosensitive neurons in the retrotrapezoid nucleus [Internet]. Journal of Neurophysiology. 2016 ; 116( 3): 1024-1035.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/jn.00022.2016
    • Vancouver

      Kuo F-S, Falquetto B, Chen D, Oliveira LM, Takakura AC, Mulkey DK. In vitro characterization of noradrenergic modulation of chemosensitive neurons in the retrotrapezoid nucleus [Internet]. Journal of Neurophysiology. 2016 ; 116( 3): 1024-1035.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/jn.00022.2016
  • Source: Translational Psychiatry. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RESENDE, L. S. et al. Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX. Translational Psychiatry, v. 6, n. 10, 2016Tradução . . Disponível em: https://doi.org/10.1038/tp.2016.202. Acesso em: 19 nov. 2024.
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      Resende, L. S., Amaral, C. E., Soares, R. B. S., Alves, A. S., Alves-dos-Santos, L., Britto, L. R. G. de, & Chiavegatto, S. (2016). Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX. Translational Psychiatry, 6( 10). doi:10.1038/tp.2016.202
    • NLM

      Resende LS, Amaral CE, Soares RBS, Alves AS, Alves-dos-Santos L, Britto LRG de, Chiavegatto S. Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX [Internet]. Translational Psychiatry. 2016 ; 6( 10):[citado 2024 nov. 19 ] Available from: https://doi.org/10.1038/tp.2016.202
    • Vancouver

      Resende LS, Amaral CE, Soares RBS, Alves AS, Alves-dos-Santos L, Britto LRG de, Chiavegatto S. Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX [Internet]. Translational Psychiatry. 2016 ; 6( 10):[citado 2024 nov. 19 ] Available from: https://doi.org/10.1038/tp.2016.202
  • Source: Clinical Science. Unidade: ICB

    Assunto: FARMACOLOGIA

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      GIMENEZ, Marcela et al. Nox1 in cardiovascular diseases: regulation and pathophysiology. Clinical Science, v. 130, n. 3, p. 151-165, 2016Tradução . . Disponível em: https://doi.org/10.1042/CS20150404. Acesso em: 19 nov. 2024.
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      Gimenez, M., Schickling, B. M., Lopes, L. R., & Miller, F. J. (2016). Nox1 in cardiovascular diseases: regulation and pathophysiology. Clinical Science, 130( 3), 151-165. doi:10.1042/CS20150404
    • NLM

      Gimenez M, Schickling BM, Lopes LR, Miller FJ. Nox1 in cardiovascular diseases: regulation and pathophysiology [Internet]. Clinical Science. 2016 ; 130( 3): 151-165.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1042/CS20150404
    • Vancouver

      Gimenez M, Schickling BM, Lopes LR, Miller FJ. Nox1 in cardiovascular diseases: regulation and pathophysiology [Internet]. Clinical Science. 2016 ; 130( 3): 151-165.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1042/CS20150404

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