Filtros : "ENDOCRINOLOGIA" "Reino Unido" "FM" Removidos: "IFSC333" "Indexado na Base de dados Science Citation Index" "Suécia" Limpar

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  • Fonte: Clinical Endocrinology. Unidades: FM, FMRP

    Assuntos: ENDOCRINOLOGIA, PUBERDADE PRECOCE

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    • ABNT

      ALMEIDA, Madson Queiroz et al. Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole. Clinical Endocrinology, v. 69, n. 1, p. 93-98, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2007.03160.x. Acesso em: 31 out. 2024.
    • APA

      Almeida, M. Q., Brito, V. N., Lins, T. S. S., Guerra-Júnior, G., Castro, M. de, Antonini, S. R., et al. (2008). Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole. Clinical Endocrinology, 69( 1), 93-98. doi:10.1111/j.1365-2265.2007.03160.x
    • NLM

      Almeida MQ, Brito VN, Lins TSS, Guerra-Júnior G, Castro M de, Antonini SR, Arnhold IJP, Mendonça BB, Latronico AC. Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole [Internet]. Clinical Endocrinology. 2008 ; 69( 1): 93-98.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.2007.03160.x
    • Vancouver

      Almeida MQ, Brito VN, Lins TSS, Guerra-Júnior G, Castro M de, Antonini SR, Arnhold IJP, Mendonça BB, Latronico AC. Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole [Internet]. Clinical Endocrinology. 2008 ; 69( 1): 93-98.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.2007.03160.x
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assuntos: ENDOCRINOLOGIA, HORMÔNIOS HIPOFISÁRIOS (DEFICIÊNCIA)

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    • ABNT

      ABRÃO, Milena Garcia et al. Combined pituitary hormone deficiency (CPHD) due to a complete PROP1 deletion. Clinical Endocrinology, v. 65, n. 3, p. 294-300, 2006Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2006.02592.x. Acesso em: 31 out. 2024.
    • APA

      Abrão, M. G., Leitet, M. V., Carvalho, L. R., Billerbeck, A. E. C., Nishi, M. Y., Barbosa, A. S., et al. (2006). Combined pituitary hormone deficiency (CPHD) due to a complete PROP1 deletion. Clinical Endocrinology, 65( 3), 294-300. doi:10.1111/j.1365-2265.2006.02592.x
    • NLM

      Abrão MG, Leitet MV, Carvalho LR, Billerbeck AEC, Nishi MY, Barbosa AS, Martin RM, Arnhold IJP, Mendonça BB. Combined pituitary hormone deficiency (CPHD) due to a complete PROP1 deletion [Internet]. Clinical Endocrinology. 2006 ; 65( 3): 294-300.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02592.x
    • Vancouver

      Abrão MG, Leitet MV, Carvalho LR, Billerbeck AEC, Nishi MY, Barbosa AS, Martin RM, Arnhold IJP, Mendonça BB. Combined pituitary hormone deficiency (CPHD) due to a complete PROP1 deletion [Internet]. Clinical Endocrinology. 2006 ; 65( 3): 294-300.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02592.x
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      JORGE, Alexander A. L. et al. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, v. 60, p. 36-40, 2004Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2004.01930.x. Acesso em: 31 out. 2024.
    • APA

      Jorge, A. A. L., Souza, S. C. A. L., Arnhold, I. J. P., & Mendonça, B. B. (2004). The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, 60, 36-40. doi:10.1111/j.1365-2265.2004.01930.x
    • NLM

      Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x
    • Vancouver

      Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      CARVALHO, Luciani R. et al. Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy. Clinical Endocrinology, v. 59, p. 788-792, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01899.x. Acesso em: 31 out. 2024.
    • APA

      Carvalho, L. R., Faria, M. E. J. de, Osorio, M. G. F., Estefan, V., Jorge, A. A. L., Arnhold, I. J. P., & Mendonça, B. B. (2003). Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy. Clinical Endocrinology, 59, 788-792. doi:10.1046/j.1365-2265.2003.01899.x
    • NLM

      Carvalho LR, Faria MEJ de, Osorio MGF, Estefan V, Jorge AAL, Arnhold IJP, Mendonça BB. Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy [Internet]. Clinical Endocrinology. 2003 ; 59 788-792.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01899.x
    • Vancouver

      Carvalho LR, Faria MEJ de, Osorio MGF, Estefan V, Jorge AAL, Arnhold IJP, Mendonça BB. Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy [Internet]. Clinical Endocrinology. 2003 ; 59 788-792.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01899.x
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      ANTONINI, S. R. R. et al. Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours. Clinical Endocrinology, v. 57, n. 05, p. 657-662, 2002Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2002.01639.x. Acesso em: 31 out. 2024.
    • APA

      Antonini, S. R. R., Latronico, A. C., Elias, L. L. K., Cukier, A., Machado, H. R., Liberman, B., et al. (2002). Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours. Clinical Endocrinology, 57( 05), 657-662. doi:10.1046/j.1365-2265.2002.01639.x
    • NLM

      Antonini SRR, Latronico AC, Elias LLK, Cukier A, Machado HR, Liberman B, Mendonça BB, Moreira AC, Castro M. Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours [Internet]. Clinical Endocrinology. 2002 ; 57( 05): 657-662.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
    • Vancouver

      Antonini SRR, Latronico AC, Elias LLK, Cukier A, Machado HR, Liberman B, Mendonça BB, Moreira AC, Castro M. Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours [Internet]. Clinical Endocrinology. 2002 ; 57( 05): 657-662.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
  • Fonte: Lupus. Unidades: HU, FM

    Assuntos: AMENORREIA, ENDOCRINOLOGIA

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    • ABNT

      PASOTO, Sandra Gofinet e MENDONÇA, Berenice Bilharinho de e BONFÁ, Eloísa Silva Dutra de Oliveira. Menstrual disturbances in patients with systemic lupus erythematosus without alkylating therapy: clinical, hormonal and therapeutic associations. Lupus, v. 11, p. 175-180, 2002Tradução . . Acesso em: 31 out. 2024.
    • APA

      Pasoto, S. G., Mendonça, B. B. de, & Bonfá, E. S. D. de O. (2002). Menstrual disturbances in patients with systemic lupus erythematosus without alkylating therapy: clinical, hormonal and therapeutic associations. Lupus, 11, 175-180.
    • NLM

      Pasoto SG, Mendonça BB de, Bonfá ESD de O. Menstrual disturbances in patients with systemic lupus erythematosus without alkylating therapy: clinical, hormonal and therapeutic associations. Lupus. 2002 ; 11 175-180.[citado 2024 out. 31 ]
    • Vancouver

      Pasoto SG, Mendonça BB de, Bonfá ESD de O. Menstrual disturbances in patients with systemic lupus erythematosus without alkylating therapy: clinical, hormonal and therapeutic associations. Lupus. 2002 ; 11 175-180.[citado 2024 out. 31 ]
  • Fonte: Leukemia Research. Unidade: FM

    Assuntos: INTERFERONS, ENDOCRINOLOGIA

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    • ABNT

      OLIVEIRA, Maria Regina et al. Interferon-alpha therapy increases type I insulin-like growth factor receptors expression on lymphoid cells from patients with chronic myelogenous leukemia. Leukemia Research, n. 25, p. 711-717, 2001Tradução . . Disponível em: https://doi.org/10.1016/s0145-2126(01)00011-x. Acesso em: 31 out. 2024.
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      Oliveira, M. R., Ohnuma, L., Bendit, I., Dorlhiac-Lacer, P., & Gianella-Neto, D. (2001). Interferon-alpha therapy increases type I insulin-like growth factor receptors expression on lymphoid cells from patients with chronic myelogenous leukemia. Leukemia Research, ( 25), 711-717. doi:10.1016/s0145-2126(01)00011-x
    • NLM

      Oliveira MR, Ohnuma L, Bendit I, Dorlhiac-Lacer P, Gianella-Neto D. Interferon-alpha therapy increases type I insulin-like growth factor receptors expression on lymphoid cells from patients with chronic myelogenous leukemia [Internet]. Leukemia Research. 2001 ;( 25): 711-717.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/s0145-2126(01)00011-x
    • Vancouver

      Oliveira MR, Ohnuma L, Bendit I, Dorlhiac-Lacer P, Gianella-Neto D. Interferon-alpha therapy increases type I insulin-like growth factor receptors expression on lymphoid cells from patients with chronic myelogenous leukemia [Internet]. Leukemia Research. 2001 ;( 25): 711-717.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/s0145-2126(01)00011-x
  • Fonte: European Journal of Endocrinology. Unidade: FM

    Assuntos: CLÍNICA GERAL, ENDOCRINOLOGIA

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    • ABNT

      TREVINO, O. Gonzalez et al. Clinical and molecular analysis of three Mexican families with Pendred's syndrome. European Journal of Endocrinology. Reino Unido: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 31 out. 2024. , 2001
    • APA

      Trevino, O. G., Arseven Karamanoglu, O., Ceballos, C. J., Vives, V. I., Ramirez, R. C., Gomez, V. V., et al. (2001). Clinical and molecular analysis of three Mexican families with Pendred's syndrome. European Journal of Endocrinology. Reino Unido: Faculdade de Medicina, Universidade de São Paulo.
    • NLM

      Trevino OG, Arseven Karamanoglu O, Ceballos CJ, Vives VI, Ramirez RC, Gomez VV, Medeiros-Neto G, Kopp P. Clinical and molecular analysis of three Mexican families with Pendred's syndrome. European Journal of Endocrinology. 2001 ; 144( 6): 585-593.[citado 2024 out. 31 ]
    • Vancouver

      Trevino OG, Arseven Karamanoglu O, Ceballos CJ, Vives VI, Ramirez RC, Gomez VV, Medeiros-Neto G, Kopp P. Clinical and molecular analysis of three Mexican families with Pendred's syndrome. European Journal of Endocrinology. 2001 ; 144( 6): 585-593.[citado 2024 out. 31 ]
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      BARCA, Maria Fernanda et al. Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil. Clinical Endocrinology, v. 53, p. 21-31, 2000Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2000.01034.x. Acesso em: 31 out. 2024.
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      Barca, M. F., Knobel, M., Tomimori, E., Cardia, M. S., & Medeiros Neto, G. A. (2000). Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil. Clinical Endocrinology, 53, 21-31. doi:10.1046/j.1365-2265.2000.01034.x
    • NLM

      Barca MF, Knobel M, Tomimori E, Cardia MS, Medeiros Neto GA. Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil [Internet]. Clinical Endocrinology. 2000 ; 53 21-31.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.01034.x
    • Vancouver

      Barca MF, Knobel M, Tomimori E, Cardia MS, Medeiros Neto GA. Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil [Internet]. Clinical Endocrinology. 2000 ; 53 21-31.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.01034.x
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assuntos: ENDOCRINOLOGIA, HORMÔNIOS

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    • ABNT

      SANTOS, Cecilia L. S. et al. A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect. Clinical Endocrinology, v. 51, n. 2, p. 165-172, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00746.x. Acesso em: 31 out. 2024.
    • APA

      Santos, C. L. S., Bikker, H., Rego, K. G. M., Nascimento, A. C., Tambascia, M., Vijlder, J. J. M. de, & Medeiros Neto, G. A. (1999). A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect. Clinical Endocrinology, 51( 2), 165-172. doi:10.1046/j.1365-2265.1999.00746.x
    • NLM

      Santos CLS, Bikker H, Rego KGM, Nascimento AC, Tambascia M, Vijlder JJM de, Medeiros Neto GA. A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect [Internet]. Clinical Endocrinology. 1999 ; 51( 2): 165-172.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00746.x
    • Vancouver

      Santos CLS, Bikker H, Rego KGM, Nascimento AC, Tambascia M, Vijlder JJM de, Medeiros Neto GA. A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect [Internet]. Clinical Endocrinology. 1999 ; 51( 2): 165-172.[citado 2024 out. 31 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00746.x
  • Fonte: Journal of Medical Genetics. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      ENG, C et al. Mutations in the ret proto-oncogene and the von hippel-lindau disease tumour supressor gene in sporadic and syndromic phaeochromocytomas. Journal of Medical Genetics, v. 32, n. 12, p. 934-7, 1995Tradução . . Disponível em: https://doi.org/10.1136/jmg.32.12.934. Acesso em: 31 out. 2024.
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      Eng, C., Crossey, P. A., Mulligan, L. M., Healey, C. S., Houghton, C., Prowse, A., et al. (1995). Mutations in the ret proto-oncogene and the von hippel-lindau disease tumour supressor gene in sporadic and syndromic phaeochromocytomas. Journal of Medical Genetics, 32( 12), 934-7. doi:10.1136/jmg.32.12.934
    • NLM

      Eng C, Crossey PA, Mulligan LM, Healey CS, Houghton C, Prowse A, Chew SL, Dahia PLM, Oriordan JLH, Toledo SPA, Smith DP, Maher ER. Mutations in the ret proto-oncogene and the von hippel-lindau disease tumour supressor gene in sporadic and syndromic phaeochromocytomas [Internet]. Journal of Medical Genetics. 1995 ;32( 12): 934-7.[citado 2024 out. 31 ] Available from: https://doi.org/10.1136/jmg.32.12.934
    • Vancouver

      Eng C, Crossey PA, Mulligan LM, Healey CS, Houghton C, Prowse A, Chew SL, Dahia PLM, Oriordan JLH, Toledo SPA, Smith DP, Maher ER. Mutations in the ret proto-oncogene and the von hippel-lindau disease tumour supressor gene in sporadic and syndromic phaeochromocytomas [Internet]. Journal of Medical Genetics. 1995 ;32( 12): 934-7.[citado 2024 out. 31 ] Available from: https://doi.org/10.1136/jmg.32.12.934
  • Fonte: Journal of Endocrinology. Nome do evento: Joint Meeting of the British Endocrine Societies and European Federation of Endocrine Societies. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      DAHIA, P L M et al. Lack of evidence of p53 involvement in phaeochromocytoma oncogenesis. Journal of Endocrinology. London: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 31 out. 2024. , 1995
    • APA

      Dahia, P. L. M., Tsanaclis, A. M. C., Ezabella, M. C. L., Hayashida, C. Y., Abelin, N. M. A., & Toledo, S. P. A. (1995). Lack of evidence of p53 involvement in phaeochromocytoma oncogenesis. Journal of Endocrinology. London: Faculdade de Medicina, Universidade de São Paulo.
    • NLM

      Dahia PLM, Tsanaclis AMC, Ezabella MCL, Hayashida CY, Abelin NMA, Toledo SPA. Lack of evidence of p53 involvement in phaeochromocytoma oncogenesis. Journal of Endocrinology. 1995 ;144( suppl.):[citado 2024 out. 31 ]
    • Vancouver

      Dahia PLM, Tsanaclis AMC, Ezabella MCL, Hayashida CY, Abelin NMA, Toledo SPA. Lack of evidence of p53 involvement in phaeochromocytoma oncogenesis. Journal of Endocrinology. 1995 ;144( suppl.):[citado 2024 out. 31 ]
  • Fonte: European Journal of Endocrinology. Nome do evento: European Congress of Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      BORGES, M F et al. Calcitonin (ct) deficiency in chronic hashimoto's thyroiditis. European Journal of Endocrinology. Oxford: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 31 out. 2024. , 1994
    • APA

      Borges, M. F., Toledo, S. P. A., Abelin, N. M. A., Cunha, D. F., & Barros, M. A. E. (1994). Calcitonin (ct) deficiency in chronic hashimoto's thyroiditis. European Journal of Endocrinology. Oxford: Faculdade de Medicina, Universidade de São Paulo.
    • NLM

      Borges MF, Toledo SPA, Abelin NMA, Cunha DF, Barros MAE. Calcitonin (ct) deficiency in chronic hashimoto's thyroiditis. European Journal of Endocrinology. 1994 ;130( ju 1994): 140.[citado 2024 out. 31 ]
    • Vancouver

      Borges MF, Toledo SPA, Abelin NMA, Cunha DF, Barros MAE. Calcitonin (ct) deficiency in chronic hashimoto's thyroiditis. European Journal of Endocrinology. 1994 ;130( ju 1994): 140.[citado 2024 out. 31 ]
  • Fonte: Endocrine Pathology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      MATOS, P. S. e BISI, Hélio e MEDEIROS NETO, Geraldo Antônio. Dyshormonogenetic goiter: a morphological and immunohistochemical study. Endocrine Pathology, v. 5, n. 1, p. 49-58, 1994Tradução . . Disponível em: https://doi.org/10.1007/bf02921370. Acesso em: 31 out. 2024.
    • APA

      Matos, P. S., Bisi, H., & Medeiros Neto, G. A. (1994). Dyshormonogenetic goiter: a morphological and immunohistochemical study. Endocrine Pathology, 5( 1), 49-58. doi:10.1007/bf02921370
    • NLM

      Matos PS, Bisi H, Medeiros Neto GA. Dyshormonogenetic goiter: a morphological and immunohistochemical study [Internet]. Endocrine Pathology. 1994 ; 5( 1): 49-58.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/bf02921370
    • Vancouver

      Matos PS, Bisi H, Medeiros Neto GA. Dyshormonogenetic goiter: a morphological and immunohistochemical study [Internet]. Endocrine Pathology. 1994 ; 5( 1): 49-58.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/bf02921370
  • Fonte: European Journal of Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      DAHIA, P L M et al. Low cor blood levels of catecholamine from a newborn of a pheochromocytoma patient. European Journal of Endocrinology, v. 130, n. 3 , p. 217-9, 1994Tradução . . Disponível em: https://doi.org/10.1530/eje.0.1300217. Acesso em: 31 out. 2024.
    • APA

      Dahia, P. L. M., Hayashida, C. Y., Strunz, C. M. C., Abelin, N. M. A., & Toledo, S. P. A. (1994). Low cor blood levels of catecholamine from a newborn of a pheochromocytoma patient. European Journal of Endocrinology, 130( 3 ), 217-9. doi:10.1530/eje.0.1300217
    • NLM

      Dahia PLM, Hayashida CY, Strunz CMC, Abelin NMA, Toledo SPA. Low cor blood levels of catecholamine from a newborn of a pheochromocytoma patient [Internet]. European Journal of Endocrinology. 1994 ;130( 3 ): 217-9.[citado 2024 out. 31 ] Available from: https://doi.org/10.1530/eje.0.1300217
    • Vancouver

      Dahia PLM, Hayashida CY, Strunz CMC, Abelin NMA, Toledo SPA. Low cor blood levels of catecholamine from a newborn of a pheochromocytoma patient [Internet]. European Journal of Endocrinology. 1994 ;130( 3 ): 217-9.[citado 2024 out. 31 ] Available from: https://doi.org/10.1530/eje.0.1300217
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      MEDEIROS NETO, Geraldo Antônio. Letters to the editors. Clinical Endocrinology, v. 40, n. 3 , p. 435, 1994Tradução . . Acesso em: 31 out. 2024.
    • APA

      Medeiros Neto, G. A. (1994). Letters to the editors. Clinical Endocrinology, 40( 3 ), 435.
    • NLM

      Medeiros Neto GA. Letters to the editors. Clinical Endocrinology. 1994 ;40( 3 ): 435.[citado 2024 out. 31 ]
    • Vancouver

      Medeiros Neto GA. Letters to the editors. Clinical Endocrinology. 1994 ;40( 3 ): 435.[citado 2024 out. 31 ]
  • Fonte: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      TARGOVNIK, H M et al. Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger. Journal of Clinical Endocrinology and Metabolism, v. 77, n. 1 , p. 210-5, 1993Tradução . . Disponível em: https://doi.org/10.1210/jcem.77.1.8325944. Acesso em: 31 out. 2024.
    • APA

      Targovnik, H. M., Medeiros Neto, G. A., Varela, V., Cochaux, P., Wajchenberg, B. L., & Vassart, G. (1993). Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger. Journal of Clinical Endocrinology and Metabolism, 77( 1 ), 210-5. doi:10.1210/jcem.77.1.8325944
    • NLM

      Targovnik HM, Medeiros Neto GA, Varela V, Cochaux P, Wajchenberg BL, Vassart G. Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger [Internet]. Journal of Clinical Endocrinology and Metabolism. 1993 ;77( 1 ): 210-5.[citado 2024 out. 31 ] Available from: https://doi.org/10.1210/jcem.77.1.8325944
    • Vancouver

      Targovnik HM, Medeiros Neto GA, Varela V, Cochaux P, Wajchenberg BL, Vassart G. Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger [Internet]. Journal of Clinical Endocrinology and Metabolism. 1993 ;77( 1 ): 210-5.[citado 2024 out. 31 ] Available from: https://doi.org/10.1210/jcem.77.1.8325944
  • Fonte: Biochemical Journal. Unidade: FM

    Assunto: ENDOCRINOLOGIA

    Acesso à fonteDOIComo citar
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    • ABNT

      REDGRAVE, T et al. Clearence from plasma of triacylglycerol and cholesteryl ester after intravenous injection of chylomicron-like lipid emulsions in rats and man. Biochemical Journal, v. 290, p. 843-7, 1993Tradução . . Disponível em: https://doi.org/10.1042/bj2900843. Acesso em: 31 out. 2024.
    • APA

      Redgrave, T., Ly, H. L., Quintão, E. C. da R., Ramberg, C. F., & Boston, R. C. (1993). Clearence from plasma of triacylglycerol and cholesteryl ester after intravenous injection of chylomicron-like lipid emulsions in rats and man. Biochemical Journal, 290, 843-7. doi:10.1042/bj2900843
    • NLM

      Redgrave T, Ly HL, Quintão EC da R, Ramberg CF, Boston RC. Clearence from plasma of triacylglycerol and cholesteryl ester after intravenous injection of chylomicron-like lipid emulsions in rats and man [Internet]. Biochemical Journal. 1993 ;290 843-7.[citado 2024 out. 31 ] Available from: https://doi.org/10.1042/bj2900843
    • Vancouver

      Redgrave T, Ly HL, Quintão EC da R, Ramberg CF, Boston RC. Clearence from plasma of triacylglycerol and cholesteryl ester after intravenous injection of chylomicron-like lipid emulsions in rats and man [Internet]. Biochemical Journal. 1993 ;290 843-7.[citado 2024 out. 31 ] Available from: https://doi.org/10.1042/bj2900843
  • Fonte: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      MALERBI, D A et al. Desmopressin stimulation test in the differential diagnosis of cushing's syndrome. Clinical Endocrinology, v. 38, n. 5 , p. 463-72, 1993Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.1993.tb00341.x. Acesso em: 31 out. 2024.
    • APA

      Malerbi, D. A., Mendonça, B. B., Liberman, B., Toledo, S. P. A., Corradini, M. C. M., Cunha Neto, M. B. C., et al. (1993). Desmopressin stimulation test in the differential diagnosis of cushing's syndrome. Clinical Endocrinology, 38( 5 ), 463-72. doi:10.1111/j.1365-2265.1993.tb00341.x
    • NLM

      Malerbi DA, Mendonça BB, Liberman B, Toledo SPA, Corradini MCM, Cunha Neto MBC, Fragoso MCBV, Wajchenberg BL. Desmopressin stimulation test in the differential diagnosis of cushing's syndrome [Internet]. Clinical Endocrinology. 1993 ;38( 5 ): 463-72.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.1993.tb00341.x
    • Vancouver

      Malerbi DA, Mendonça BB, Liberman B, Toledo SPA, Corradini MCM, Cunha Neto MBC, Fragoso MCBV, Wajchenberg BL. Desmopressin stimulation test in the differential diagnosis of cushing's syndrome [Internet]. Clinical Endocrinology. 1993 ;38( 5 ): 463-72.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2265.1993.tb00341.x
  • Fonte: Progress in Endocrinology. Nome do evento: International Congress of the Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      MEDEIROS NETO, Geraldo Antônio e BILLERBECK, A E C e WAJCHENBERG, B L. Genetic and molecular biological studies in defective iodide organification causing hereditary goiter and hypothyroidism. 1993, Anais.. Carnforth: Parthenon, 1993. . Acesso em: 31 out. 2024.
    • APA

      Medeiros Neto, G. A., Billerbeck, A. E. C., & Wajchenberg, B. L. (1993). Genetic and molecular biological studies in defective iodide organification causing hereditary goiter and hypothyroidism. In Progress in Endocrinology. Carnforth: Parthenon.
    • NLM

      Medeiros Neto GA, Billerbeck AEC, Wajchenberg BL. Genetic and molecular biological studies in defective iodide organification causing hereditary goiter and hypothyroidism. Progress in Endocrinology. 1993 ;[citado 2024 out. 31 ]
    • Vancouver

      Medeiros Neto GA, Billerbeck AEC, Wajchenberg BL. Genetic and molecular biological studies in defective iodide organification causing hereditary goiter and hypothyroidism. Progress in Endocrinology. 1993 ;[citado 2024 out. 31 ]

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