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Artigo de periodico
Mass spectrometry-based characterization of protein aggregates in tissues and biofluid (2024)
Silva, Janaina Macedo da ; Fernandes, Livia Rosa ; Blanes, Claudia Angeli ; Palmisano, Giuseppe
Source: Advances in Experimental Medicine and Biology. Unidade: ICB
Subjects: PARASITOLOGIA, COMPLEXOS MULTIENZIMÁTICOS, MICRORNAS, PROTEÍNAS SANGUÍNEAS, ESPECTROMETRIA DE MASSAS, MICROSCOPIA ELETRÔNICA ANIMAL, PLASMA, PLASMA DE ANIMAL, WESTERN BLOTTING, DOENÇAS NEURODEGENERATIVAS, CAMUNDONGOS
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ABNT
SILVA, Janaina Macedo da et al. Mass spectrometry-based characterization of protein aggregates in tissues and biofluid. Advances in Experimental Medicine and Biology, v. 1443, p. 257-267, 2024Tradução . . Disponível em: https://doi.org/10.1007/978-3-031-50624-6_14. Acesso em: 10 nov. 2024.
APA
Silva, J. M. da, Fernandes, L. R., Blanes, C. A., & Palmisano, G. (2024). Mass spectrometry-based characterization of protein aggregates in tissues and biofluid. Advances in Experimental Medicine and Biology, 1443, 257-267. doi:10.1007/978-3-031-50624-6_14
NLM
Silva JM da, Fernandes LR, Blanes CA, Palmisano G. Mass spectrometry-based characterization of protein aggregates in tissues and biofluid [Internet]. Advances in Experimental Medicine and Biology. 2024 ; 1443 257-267.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/978-3-031-50624-6_14.
Vancouver
Silva JM da, Fernandes LR, Blanes CA, Palmisano G. Mass spectrometry-based characterization of protein aggregates in tissues and biofluid [Internet]. Advances in Experimental Medicine and Biology. 2024 ; 1443 257-267.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/978-3-031-50624-6_14.
Artigo de periodico
Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting (2024)
Rizzi, Liara; Grinberg, Lea Tenenholz
Source: Acta neuropathologica communications. Unidade: FM
Subjects: DOENÇAS NEURODEGENERATIVAS, DOENÇA DE ALZHEIMER, BIOMARCADORES
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RIZZI, Liara e GRINBERG, Lea Tenenholz. Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting. Acta neuropathologica communications, v. 12, n. 1, 2024Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/59005. Acesso em: 10 nov. 2024.
APA
Rizzi, L., & Grinberg, L. T. (2024). Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting. Acta neuropathologica communications, 12( 1). doi:10.1186/s40478-024-01744-9
NLM
Rizzi L, Grinberg LT. Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting [Internet]. Acta neuropathologica communications. 2024 ; 12( 1):[citado 2024 nov. 10 ] Available from: https://observatorio.fm.usp.br/handle/OPI/59005
Vancouver
Rizzi L, Grinberg LT. Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting [Internet]. Acta neuropathologica communications. 2024 ; 12( 1):[citado 2024 nov. 10 ] Available from: https://observatorio.fm.usp.br/handle/OPI/59005
Trabalho de evento-resumo
Formation of functional amyloids by the hormone neuropeptide Y in the context of cell secretion (2024)
Souza, Murilo de Oliveira; Mendes, Luís Felipe Santos
Source: Anais eletrônicos. Conference titles: Annual Meeting of the Brazilian Biophysical Society - SBBf. Unidade: IFSC
Subjects: SECREÇÃO, BIOFÍSICA, DOENÇAS NEURODEGENERATIVAS, NEUROPEPTÍDEOS
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SOUZA, Murilo de Oliveira e MENDES, Luís Felipe Santos. Formation of functional amyloids by the hormone neuropeptide Y in the context of cell secretion. 2024, Anais.. Campinas: Galoá, 2024. Disponível em: https://proceedings.science/sbff/sbbf-2024/papers/formation-of-functional-amyloids-by-the-hormone-neuropeptide-y-in-the-context-of?lang=en. Acesso em: 10 nov. 2024.
APA
Souza, M. de O., & Mendes, L. F. S. (2024). Formation of functional amyloids by the hormone neuropeptide Y in the context of cell secretion. In Anais eletrônicos. Campinas: Galoá. Recuperado de https://proceedings.science/sbff/sbbf-2024/papers/formation-of-functional-amyloids-by-the-hormone-neuropeptide-y-in-the-context-of?lang=en
NLM
Souza M de O, Mendes LFS. Formation of functional amyloids by the hormone neuropeptide Y in the context of cell secretion [Internet]. Anais eletrônicos. 2024 ;[citado 2024 nov. 10 ] Available from: https://proceedings.science/sbff/sbbf-2024/papers/formation-of-functional-amyloids-by-the-hormone-neuropeptide-y-in-the-context-of?lang=en
Vancouver
Souza M de O, Mendes LFS. Formation of functional amyloids by the hormone neuropeptide Y in the context of cell secretion [Internet]. Anais eletrônicos. 2024 ;[citado 2024 nov. 10 ] Available from: https://proceedings.science/sbff/sbbf-2024/papers/formation-of-functional-amyloids-by-the-hormone-neuropeptide-y-in-the-context-of?lang=en
Artigo de periodico
Opening avenues for treatment of neurodegenerative disease using post-biotics: breakthroughs and bottlenecks in clinical translation (2024)
Bashir, Bushra; Alam, Shahbaz; Khandale, Nikhil; Birla, Devendra; Vishwas, Sukriti ; Pandey, Narendra Kumar; Gupta, Gaurav; Paudel, Keshav Raj ; Dureja, Harish; Kumar, Puneet; Singh, Thakur Gurjeet; Kuppusamy, Gowthamarajan; Zacconi, Flavia C; Pinto, Terezinha de Jesus Andreoli ; Dhanasekaran, Muralikrishnan; Gulati, Monica; Dua, Kamal ; Singh, Sachin Kumar
Source: Ageing Research Reviews. Unidade: FCF
Subjects: DOENÇAS NEURODEGENERATIVAS, ENVELHECIMENTO
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BASHIR, Bushra et al. Opening avenues for treatment of neurodegenerative disease using post-biotics: breakthroughs and bottlenecks in clinical translation. Ageing Research Reviews, v. 95, p. 1-16 art. 102236, 2024Tradução . . Disponível em: https://dx.doi.org/10.1016/j.arr.2024.102236. Acesso em: 10 nov. 2024.
APA
Bashir, B., Alam, S., Khandale, N., Birla, D., Vishwas, S., Pandey, N. K., et al. (2024). Opening avenues for treatment of neurodegenerative disease using post-biotics: breakthroughs and bottlenecks in clinical translation. Ageing Research Reviews, 95, 1-16 art. 102236. doi:10.1016/j.arr.2024.102236
NLM
Bashir B, Alam S, Khandale N, Birla D, Vishwas S, Pandey NK, Gupta G, Paudel KR, Dureja H, Kumar P, Singh TG, Kuppusamy G, Zacconi FC, Pinto T de JA, Dhanasekaran M, Gulati M, Dua K, Singh SK. Opening avenues for treatment of neurodegenerative disease using post-biotics: breakthroughs and bottlenecks in clinical translation [Internet]. Ageing Research Reviews. 2024 ; 95 1-16 art. 102236.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1016/j.arr.2024.102236
Vancouver
Bashir B, Alam S, Khandale N, Birla D, Vishwas S, Pandey NK, Gupta G, Paudel KR, Dureja H, Kumar P, Singh TG, Kuppusamy G, Zacconi FC, Pinto T de JA, Dhanasekaran M, Gulati M, Dua K, Singh SK. Opening avenues for treatment of neurodegenerative disease using post-biotics: breakthroughs and bottlenecks in clinical translation [Internet]. Ageing Research Reviews. 2024 ; 95 1-16 art. 102236.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1016/j.arr.2024.102236
Artigo de periodico
GlyT1 Inhibition by NFPS promotes neuroprotection in amyloid-β-Induced Alzheimer’s disease animal model (2024)
Lima, Onésia Cristina Oliveira; Carvalho, Gustavo Almeida de ; Assunção, Leandro do Prado ; Bailão, Alexandre Melo; Ulrich, Henning ; Marques, Bruno Lemes; Oliveira, Antonio Carlos Pinheiro de; Gomez, Renato Santiago; Pinto, Mauro Cunha Xavier
Source: Neurochemical Research. Unidade: IQ
Subjects: DOENÇA DE ALZHEIMER, PROTEÔMICA, DOENÇAS NEURODEGENERATIVAS
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LIMA, Onésia Cristina Oliveira et al. GlyT1 Inhibition by NFPS promotes neuroprotection in amyloid-β-Induced Alzheimer’s disease animal model. Neurochemical Research, 2024Tradução . . Disponível em: https://dx.doi.org/10.1007/s11064-024-04190-0. Acesso em: 10 nov. 2024.
APA
Lima, O. C. O., Carvalho, G. A. de, Assunção, L. do P., Bailão, A. M., Ulrich, H., Marques, B. L., et al. (2024). GlyT1 Inhibition by NFPS promotes neuroprotection in amyloid-β-Induced Alzheimer’s disease animal model. Neurochemical Research. doi:10.1007/s11064-024-04190-0
NLM
Lima OCO, Carvalho GA de, Assunção L do P, Bailão AM, Ulrich H, Marques BL, Oliveira ACP de, Gomez RS, Pinto MCX. GlyT1 Inhibition by NFPS promotes neuroprotection in amyloid-β-Induced Alzheimer’s disease animal model [Internet]. Neurochemical Research. 2024 ;[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1007/s11064-024-04190-0
Vancouver
Lima OCO, Carvalho GA de, Assunção L do P, Bailão AM, Ulrich H, Marques BL, Oliveira ACP de, Gomez RS, Pinto MCX. GlyT1 Inhibition by NFPS promotes neuroprotection in amyloid-β-Induced Alzheimer’s disease animal model [Internet]. Neurochemical Research. 2024 ;[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1007/s11064-024-04190-0
Artigo de periodico
Quantitative analysis of glutathione and carnosine Adducts with 4‑Hydroxy-2-nonenal in muscle in a hSOD1G93A ALS rat model (2024)
Reis, Pablo Victor Mendes dos; Vargas, Bianca Scigliano; Rebelo, Rafael Andrade; Massafera, Mariana Pereira ; Prado, Fernanda Manso ; Oreliana, Hector; Oliveira, Henrique V. de; Freitas, Florêncio Porto; Ronsein, Graziella Eliza ; Miyamoto, Sayuri ; Di Mascio, Paolo ; Medeiros, Marisa Helena Gennari de
Source: Chemical Research in Toxicology. Unidade: IQ
Subjects: ESCLEROSE AMIOTRÓFICA LATERAL, DOENÇAS NEURODEGENERATIVAS
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REIS, Pablo Victor Mendes dos et al. Quantitative analysis of glutathione and carnosine Adducts with 4‑Hydroxy-2-nonenal in muscle in a hSOD1G93A ALS rat model. Chemical Research in Toxicology, v. 37, n. 8, p. 1306–1314, 2024Tradução . . Disponível em: https://dx.doi.org/10.1021/acs.chemrestox.4c00052. Acesso em: 10 nov. 2024.
APA
Reis, P. V. M. dos, Vargas, B. S., Rebelo, R. A., Massafera, M. P., Prado, F. M., Oreliana, H., et al. (2024). Quantitative analysis of glutathione and carnosine Adducts with 4‑Hydroxy-2-nonenal in muscle in a hSOD1G93A ALS rat model. Chemical Research in Toxicology, 37( 8), 1306–1314. doi:10.1021/acs.chemrestox.4c00052
NLM
Reis PVM dos, Vargas BS, Rebelo RA, Massafera MP, Prado FM, Oreliana H, Oliveira HV de, Freitas FP, Ronsein GE, Miyamoto S, Di Mascio P, Medeiros MHG de. Quantitative analysis of glutathione and carnosine Adducts with 4‑Hydroxy-2-nonenal in muscle in a hSOD1G93A ALS rat model [Internet]. Chemical Research in Toxicology. 2024 ; 37( 8): 1306–1314.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1021/acs.chemrestox.4c00052
Vancouver
Reis PVM dos, Vargas BS, Rebelo RA, Massafera MP, Prado FM, Oreliana H, Oliveira HV de, Freitas FP, Ronsein GE, Miyamoto S, Di Mascio P, Medeiros MHG de. Quantitative analysis of glutathione and carnosine Adducts with 4‑Hydroxy-2-nonenal in muscle in a hSOD1G93A ALS rat model [Internet]. Chemical Research in Toxicology. 2024 ; 37( 8): 1306–1314.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1021/acs.chemrestox.4c00052
Artigo de periodico
Probable 4-repeat tauopathy criteria predict brain amyloid negativity, distinct clinical features, and FDG-PET/MRI neurodegeneneration patterns in corticobasal syndrome (2024)
Parmera, Jacy Bezerra; Carneiro, Camila de Godoi; Almeida, Isabel Junqueira de; Oliveira, Marcos Castello Barbosa de; Barbosa, Pedro Melo; Studart-Neto, Adalberto; Ono, Carla Rachel; Nitrini, Ricardo ; Buchpiguel, Carlos Alberto ; Barbosa, Egberto Reis
Source: Movement disorders clinical practice. Unidade: FM
Subjects: TRANSTORNOS NEUROPSICOLÓGICOS, DOENÇAS NEURODEGENERATIVAS, TOMOGRAFIA COMPUTADORIZADA POR EMISSÃO DE PÓSITRON, DIAGNÓSTICO POR IMAGEM
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ABNT
PARMERA, Jacy Bezerra et al. Probable 4-repeat tauopathy criteria predict brain amyloid negativity, distinct clinical features, and FDG-PET/MRI neurodegeneneration patterns in corticobasal syndrome. Movement disorders clinical practice, v. 11, n. 3, p. 238-247, 2024Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/58920. Acesso em: 10 nov. 2024.
APA
Parmera, J. B., Carneiro, C. de G., Almeida, I. J. de, Oliveira, M. C. B. de, Barbosa, P. M., Studart-Neto, A., et al. (2024). Probable 4-repeat tauopathy criteria predict brain amyloid negativity, distinct clinical features, and FDG-PET/MRI neurodegeneneration patterns in corticobasal syndrome. Movement disorders clinical practice, 11( 3), 238-247. doi:10.1002/mdc3.13959
NLM
Parmera JB, Carneiro C de G, Almeida IJ de, Oliveira MCB de, Barbosa PM, Studart-Neto A, Ono CR, Nitrini R, Buchpiguel CA, Barbosa ER. Probable 4-repeat tauopathy criteria predict brain amyloid negativity, distinct clinical features, and FDG-PET/MRI neurodegeneneration patterns in corticobasal syndrome [Internet]. Movement disorders clinical practice. 2024 ; 11( 3): 238-247.[citado 2024 nov. 10 ] Available from: https://observatorio.fm.usp.br/handle/OPI/58920
Vancouver
Parmera JB, Carneiro C de G, Almeida IJ de, Oliveira MCB de, Barbosa PM, Studart-Neto A, Ono CR, Nitrini R, Buchpiguel CA, Barbosa ER. Probable 4-repeat tauopathy criteria predict brain amyloid negativity, distinct clinical features, and FDG-PET/MRI neurodegeneneration patterns in corticobasal syndrome [Internet]. Movement disorders clinical practice. 2024 ; 11( 3): 238-247.[citado 2024 nov. 10 ] Available from: https://observatorio.fm.usp.br/handle/OPI/58920
Trabalho de evento-resumo
Elucidating the molecular mechanisms of aggregation of functional amyloid formed by human corticotropin-releasing hormone (2024)
Oliveira, Gabriel Machado de; Mendes, Luís Felipe Santos
Source: Anais eletrônicos. Conference titles: Annual Meeting of the Brazilian Biophysical Society - SBBf. Unidade: IFSC
Subjects: PROTEÍNAS, DOENÇAS NEURODEGENERATIVAS, BIOLOGIA CELULAR
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ABNT
OLIVEIRA, Gabriel Machado de e MENDES, Luís Felipe Santos. Elucidating the molecular mechanisms of aggregation of functional amyloid formed by human corticotropin-releasing hormone. 2024, Anais.. Campinas: Galoá, 2024. Disponível em: https://proceedings.science/sbff/sbbf-2024/papers/elucidating-the-molecular-mechanisms-of-aggregation-of-functional-amyloid-formed?lang=en. Acesso em: 10 nov. 2024.
APA
Oliveira, G. M. de, & Mendes, L. F. S. (2024). Elucidating the molecular mechanisms of aggregation of functional amyloid formed by human corticotropin-releasing hormone. In Anais eletrônicos. Campinas: Galoá. Recuperado de https://proceedings.science/sbff/sbbf-2024/papers/elucidating-the-molecular-mechanisms-of-aggregation-of-functional-amyloid-formed?lang=en
NLM
Oliveira GM de, Mendes LFS. Elucidating the molecular mechanisms of aggregation of functional amyloid formed by human corticotropin-releasing hormone [Internet]. Anais eletrônicos. 2024 ;[citado 2024 nov. 10 ] Available from: https://proceedings.science/sbff/sbbf-2024/papers/elucidating-the-molecular-mechanisms-of-aggregation-of-functional-amyloid-formed?lang=en
Vancouver
Oliveira GM de, Mendes LFS. Elucidating the molecular mechanisms of aggregation of functional amyloid formed by human corticotropin-releasing hormone [Internet]. Anais eletrônicos. 2024 ;[citado 2024 nov. 10 ] Available from: https://proceedings.science/sbff/sbbf-2024/papers/elucidating-the-molecular-mechanisms-of-aggregation-of-functional-amyloid-formed?lang=en
Dissertação (Mestrado)
Avaliação dos efeitos do agonista canabinóide WIN55212-2 no modelo celular de deleção da klotho (2024)
Possebom, Isabela Ribeiro ; Iwashe, Elisa Mitiko Kawamoto (Orientador)
Unidade: ICB
Subjects: CANABINOIDES, DOENÇAS NEURODEGENERATIVAS, SISTEMA NERVOSO CENTRAL, EMBRIÃO DE ANIMAL, CAMUNDONGOS, TELENCÉFALO, HOMEOSTASE, FLUORESCÊNCIA
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ABNT
POSSEBOM, Isabela Ribeiro. Avaliação dos efeitos do agonista canabinóide WIN55212-2 no modelo celular de deleção da klotho. 2024. Dissertação (Mestrado) – Universidade de São Paulo, São Paulo, 2024. Disponível em: https://www.teses.usp.br/teses/disponiveis/42/42136/tde-24092024-155209/. Acesso em: 10 nov. 2024.
APA
Possebom, I. R. (2024). Avaliação dos efeitos do agonista canabinóide WIN55212-2 no modelo celular de deleção da klotho (Dissertação (Mestrado). Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/42/42136/tde-24092024-155209/
NLM
Possebom IR. Avaliação dos efeitos do agonista canabinóide WIN55212-2 no modelo celular de deleção da klotho [Internet]. 2024 ;[citado 2024 nov. 10 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42136/tde-24092024-155209/
Vancouver
Possebom IR. Avaliação dos efeitos do agonista canabinóide WIN55212-2 no modelo celular de deleção da klotho [Internet]. 2024 ;[citado 2024 nov. 10 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42136/tde-24092024-155209/
Artigo de periodico
The connection between gut microbiota and its metabolites with neurodegenerative diseases in humans (2024)
Fabi, João Paulo
Source: Metabolic Brain Disease. Unidade: FCF
Subjects: BIOTA INTESTINAL, DOENÇAS NEURODEGENERATIVAS
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ABNT
FABI, João Paulo. The connection between gut microbiota and its metabolites with neurodegenerative diseases in humans. Metabolic Brain Disease, v. 39, p. 967-984, 2024Tradução . . Disponível em: https://dx.doi.org/10.1007/s11011-024-01369-w. Acesso em: 10 nov. 2024.
APA
Fabi, J. P. (2024). The connection between gut microbiota and its metabolites with neurodegenerative diseases in humans. Metabolic Brain Disease, 39, 967-984. doi:10.1007/s11011-024-01369-w
NLM
Fabi JP. The connection between gut microbiota and its metabolites with neurodegenerative diseases in humans [Internet]. Metabolic Brain Disease. 2024 ; 39 967-984.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1007/s11011-024-01369-w
Vancouver
Fabi JP. The connection between gut microbiota and its metabolites with neurodegenerative diseases in humans [Internet]. Metabolic Brain Disease. 2024 ; 39 967-984.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1007/s11011-024-01369-w
Trabalho de evento-resumo
Molecular docking to predict the interaction of piperine with the enzyme acetylcholinesterase for Alzheimer's disease treatment (2024)
Beretta, Gabriela Zutin; Godoi, Ian Raffaelo Gallo; Dias, Aniele Beatriz; Döring, Thiago Henrique ; Andricopulo, Adriano Defini ; Ferreira, Leonardo Luiz Gomes ; Ribeiro, Tatiana Santana
Source: Anais. Conference titles: Reunião Anual da Sociedade Brasileira de Química - RASBQ. Unidade: IFSC
Subjects: DOENÇAS NEURODEGENERATIVAS, PIMENTA-DO-REINO, ENZIMAS, DOENÇA DE ALZHEIMER
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ABNT
BERETTA, Gabriela Zutin et al. Molecular docking to predict the interaction of piperine with the enzyme acetylcholinesterase for Alzheimer's disease treatment. 2024, Anais.. São Paulo: Sociedade Brasileira de Química - SBQ, 2024. Disponível em: https://www.eventweb.com.br/rasbq2024/specific-files/manuscripts/rasbq2024/139_1706811489.pdf. Acesso em: 10 nov. 2024.
APA
Beretta, G. Z., Godoi, I. R. G., Dias, A. B., Döring, T. H., Andricopulo, A. D., Ferreira, L. L. G., & Ribeiro, T. S. (2024). Molecular docking to predict the interaction of piperine with the enzyme acetylcholinesterase for Alzheimer's disease treatment. In Anais. São Paulo: Sociedade Brasileira de Química - SBQ. Recuperado de https://www.eventweb.com.br/rasbq2024/specific-files/manuscripts/rasbq2024/139_1706811489.pdf
NLM
Beretta GZ, Godoi IRG, Dias AB, Döring TH, Andricopulo AD, Ferreira LLG, Ribeiro TS. Molecular docking to predict the interaction of piperine with the enzyme acetylcholinesterase for Alzheimer's disease treatment [Internet]. Anais. 2024 ;[citado 2024 nov. 10 ] Available from: https://www.eventweb.com.br/rasbq2024/specific-files/manuscripts/rasbq2024/139_1706811489.pdf
Vancouver
Beretta GZ, Godoi IRG, Dias AB, Döring TH, Andricopulo AD, Ferreira LLG, Ribeiro TS. Molecular docking to predict the interaction of piperine with the enzyme acetylcholinesterase for Alzheimer's disease treatment [Internet]. Anais. 2024 ;[citado 2024 nov. 10 ] Available from: https://www.eventweb.com.br/rasbq2024/specific-files/manuscripts/rasbq2024/139_1706811489.pdf
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Experimental Neurology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 10 nov. 2024.
APA
Ferreira, A. F. F., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function (2024)
Cardinali, Camila Aparecida Errerias Fernandes ; Martins, Yandara Akamine ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Alencar, Mayke Bezerra ; Silber, Ariel Mariano ; Torrão, Andréa da Silva
Source: ACS Chemical Neuroscience ISSN:1948-7193. Unidades: ICB, FM
Subjects: FISIOLOGIA, PARASITOLOGIA, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, HIPOCAMPU DE ANIMAL, COGNIÇÃO, INSULINA, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARDINALI, Camila Aparecida Errerias Fernandes et al. Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function. ACS Chemical Neuroscience ISSN:1948-7193, v. 15, n. 16, p. 2982-2994, 2024Tradução . . Disponível em: https://doi.org/10.1021/acschemneuro.4c00113. Acesso em: 10 nov. 2024.
APA
Cardinali, C. A. E. F., Martins, Y. A., Moraes, R. C. M. de, Costa, A. P., Alencar, M. B., Silber, A. M., & Torrão, A. da S. (2024). Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function. ACS Chemical Neuroscience ISSN:1948-7193, 15( 16), 2982-2994. doi:10.1021/acschemneuro.4c00113
NLM
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Alencar MB, Silber AM, Torrão A da S. Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function [Internet]. ACS Chemical Neuroscience ISSN:1948-7193. 2024 ; 15( 16): 2982-2994.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1021/acschemneuro.4c00113
Vancouver
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Alencar MB, Silber AM, Torrão A da S. Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function [Internet]. ACS Chemical Neuroscience ISSN:1948-7193. 2024 ; 15( 16): 2982-2994.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1021/acschemneuro.4c00113
Artigo de periodico
Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models (2024)
Ribeiro, Raul Izidoro; Carvalho, Gustavo Almeida de ; Chiareli, Raphaela Almeida; Lima, Isabel Vieira de Assis; Bellozi, Paula Maria Quaglio; Lima, Onésia Cristina Oliveira; Giacomelli, Ágatha Oliveira ; Birbrair, Alexander; Gomez, Renato Santiago; Oliveira, Antonio Carlos Pinheiro de; Ulrich, Henning ; Pinto, Mauro Cunha Xavier
Source: Neuroscience Letters. Unidade: IQ
Subjects: DOENÇA DE PARKINSON, PROCESSOS COGNITIVOS, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIBEIRO, Raul Izidoro et al. Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models. Neuroscience Letters, v. 826, n. 15, p. 1-7 art. 137715, 2024Tradução . . Disponível em: https://dx.doi.org/10.1016/j.neulet.2024.137715. Acesso em: 10 nov. 2024.
APA
Ribeiro, R. I., Carvalho, G. A. de, Chiareli, R. A., Lima, I. V. de A., Bellozi, P. M. Q., Lima, O. C. O., et al. (2024). Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models. Neuroscience Letters, 826( 15), 1-7 art. 137715. doi:10.1016/j.neulet.2024.137715
NLM
Ribeiro RI, Carvalho GA de, Chiareli RA, Lima IV de A, Bellozi PMQ, Lima OCO, Giacomelli ÁO, Birbrair A, Gomez RS, Oliveira ACP de, Ulrich H, Pinto MCX. Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models [Internet]. Neuroscience Letters. 2024 ; 826( 15): 1-7 art. 137715.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1016/j.neulet.2024.137715
Vancouver
Ribeiro RI, Carvalho GA de, Chiareli RA, Lima IV de A, Bellozi PMQ, Lima OCO, Giacomelli ÁO, Birbrair A, Gomez RS, Oliveira ACP de, Ulrich H, Pinto MCX. Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models [Internet]. Neuroscience Letters. 2024 ; 826( 15): 1-7 art. 137715.[citado 2024 nov. 10 ] Available from: https://dx.doi.org/10.1016/j.neulet.2024.137715
Artigo de periodico
Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease (2024)
Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Figueroa, Paulina Belén Sepulveda ; Cuyul-Vásquez, Iván; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de
Source: Ageing Research Reviews. Unidades: ICB, FM
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇA DE PARKINSON, LÍTIO, DOENÇAS NEURODEGENERATIVAS, REVISÃO SISTEMÁTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SINGULANI, Monique Patricio et al. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, v. 95, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2024.102231. Acesso em: 10 nov. 2024.
APA
Singulani, M. P., Ferreira, A. F. F., Figueroa, P. B. S., Cuyul-Vásquez, I., Talib, L. L., Forlenza, O. V., & Britto, L. R. G. de. (2024). Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, 95, 20 . doi:10.1016/j.arr.2024.102231
NLM
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Vancouver
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Trabalho de evento-resumo
Efeitos neuroprotetores do α-tocoferol na prevenção e no controle de doenças neurodegenerativas (2023)
Nogueira, Laisa Viviany Macedo; Fideles, Simone Ortiz Moura ; Ortiz, Adriana de Cássia ; Buchaim, Daniela Vieira ; Soares, T. P.; Buchaim, Rogério Leone
Source: Anais. Conference titles: COB - Congresso Odontológico de Bauru "Prof. Dr. Estevam Augusto Bonfante". Unidades: FOB, FORP
Subjects: ANTIOXIDANTES, VITAMINA E, USOS TERAPÊUTICOS, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NOGUEIRA, Laisa Viviany Macedo et al. Efeitos neuroprotetores do α-tocoferol na prevenção e no controle de doenças neurodegenerativas. 2023, Anais.. Bauru: Faculdade de Odontologia de Bauru, Universidade de São Paulo, 2023. . Acesso em: 10 nov. 2024.
APA
Nogueira, L. V. M., Fideles, S. O. M., Ortiz, A. de C., Buchaim, D. V., Soares, T. P., & Buchaim, R. L. (2023). Efeitos neuroprotetores do α-tocoferol na prevenção e no controle de doenças neurodegenerativas. In Anais. Bauru: Faculdade de Odontologia de Bauru, Universidade de São Paulo.
NLM
Nogueira LVM, Fideles SOM, Ortiz A de C, Buchaim DV, Soares TP, Buchaim RL. Efeitos neuroprotetores do α-tocoferol na prevenção e no controle de doenças neurodegenerativas. Anais. 2023 ;[citado 2024 nov. 10 ]
Vancouver
Nogueira LVM, Fideles SOM, Ortiz A de C, Buchaim DV, Soares TP, Buchaim RL. Efeitos neuroprotetores do α-tocoferol na prevenção e no controle de doenças neurodegenerativas. Anais. 2023 ;[citado 2024 nov. 10 ]
Tese (Doutorado)
Investigando o papel da ativação dos inflamassomas NLRP1 e CARD8 em modelos de doenças neurodegenerativas (2023)
Fragas, Matheus Garcia ; Camara, Niels Olsen Saraiva (Orientador)
Unidade: ICB
Subjects: IMUNOLOGIA, DOENÇA DE ALZHEIMER, DOENÇAS NEURODEGENERATIVAS, MACRÓFAGOS, FAGOCITOSE, MITOCÔNDRIAS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRAGAS, Matheus Garcia. Investigando o papel da ativação dos inflamassomas NLRP1 e CARD8 em modelos de doenças neurodegenerativas. 2023. Tese (Doutorado) – Universidade de São Paulo, São Paulo, 2023. Disponível em: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-27092023-145510/. Acesso em: 10 nov. 2024.
APA
Fragas, M. G. (2023). Investigando o papel da ativação dos inflamassomas NLRP1 e CARD8 em modelos de doenças neurodegenerativas (Tese (Doutorado). Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/42/42133/tde-27092023-145510/
NLM
Fragas MG. Investigando o papel da ativação dos inflamassomas NLRP1 e CARD8 em modelos de doenças neurodegenerativas [Internet]. 2023 ;[citado 2024 nov. 10 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-27092023-145510/
Vancouver
Fragas MG. Investigando o papel da ativação dos inflamassomas NLRP1 e CARD8 em modelos de doenças neurodegenerativas [Internet]. 2023 ;[citado 2024 nov. 10 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-27092023-145510/
Artigo de periodico
The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? (2023)
Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Neural Regeneration Research. Unidade: ICB
Subjects: FISIOLOGIA, CANAIS IÔNICOS, ESTRESSE OXIDATIVO, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, DOENÇAS NEURODEGENERATIVAS, DOENÇAS NEUROMUSCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Ana Flávia Fernandes e BRITTO, Luiz Roberto Giorgetti de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease?. Neural Regeneration Research, v. 18, n. 8, p. 1652-1656, 2023Tradução . . Disponível em: https://doi.org/10.4103/1673-5374.360343. Acesso em: 10 nov. 2024.
APA
Ferreira, A. F. F., & Britto, L. R. G. de. (2023). The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? Neural Regeneration Research, 18( 8), 1652-1656. doi:10.4103/1673-5374.360343
NLM
Ferreira AFF, Britto LRG de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? [Internet]. Neural Regeneration Research. 2023 ; 18( 8): 1652-1656.[citado 2024 nov. 10 ] Available from: https://doi.org/10.4103/1673-5374.360343
Vancouver
Ferreira AFF, Britto LRG de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? [Internet]. Neural Regeneration Research. 2023 ; 18( 8): 1652-1656.[citado 2024 nov. 10 ] Available from: https://doi.org/10.4103/1673-5374.360343
Artigo de periodico
Purinergic receptors in cognitive disturbances (2023)
Illes, Peter ; Ulrich, Henning ; Chen, Jiang Fan ; Tang, Yong
Source: Neurobiology of Disease. Unidade: IQ
Subjects: TRANSTORNOS MENTAIS, DOENÇAS NEURODEGENERATIVAS, RECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ILLES, Peter et al. Purinergic receptors in cognitive disturbances. Neurobiology of Disease, v. 185, p. 1-16, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.nbd.2023.106229. Acesso em: 10 nov. 2024.
APA
Illes, P., Ulrich, H., Chen, J. F., & Tang, Y. (2023). Purinergic receptors in cognitive disturbances. Neurobiology of Disease, 185, 1-16. doi:10.1016/j.nbd.2023.106229
NLM
Illes P, Ulrich H, Chen JF, Tang Y. Purinergic receptors in cognitive disturbances [Internet]. Neurobiology of Disease. 2023 ; 185 1-16.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.nbd.2023.106229
Vancouver
Illes P, Ulrich H, Chen JF, Tang Y. Purinergic receptors in cognitive disturbances [Internet]. Neurobiology of Disease. 2023 ; 185 1-16.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.nbd.2023.106229
Artigo de periodico
Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients (2023)
Ravagnania, Felipe G; Valerio, Hellen Paula ; Maués, Jersey H. S ; Oliveira, Arthur N. de; Puga, Renato David ; Oliveira, Karina Griesi ; Picosse, Fabíola Rosa ; Ferraz, Henrique Ballalai ; Catharino, Rodrigo Ramos ; Ronsein, Graziella Eliza ; Aguiar, Patricia Maria de Carvalho
Source: Parkinsonism and Related Disorders. Unidade: IQ
Subjects: ASTRÓCITOS, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAVAGNANIA, Felipe G et al. Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients. Parkinsonism and Related Disorders, v. 116, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.parkreldis.2023.105847. Acesso em: 10 nov. 2024.
APA
Ravagnania, F. G., Valerio, H. P., Maués, J. H. S., Oliveira, A. N. de, Puga, R. D., Oliveira, K. G., et al. (2023). Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients. Parkinsonism and Related Disorders, 116, 1-12. doi:10.1016/j.parkreldis.2023.105847
NLM
Ravagnania FG, Valerio HP, Maués JHS, Oliveira AN de, Puga RD, Oliveira KG, Picosse FR, Ferraz HB, Catharino RR, Ronsein GE, Aguiar PM de C. Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients [Internet]. Parkinsonism and Related Disorders. 2023 ; 116 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.parkreldis.2023.105847
Vancouver
Ravagnania FG, Valerio HP, Maués JHS, Oliveira AN de, Puga RD, Oliveira KG, Picosse FR, Ferraz HB, Catharino RR, Ronsein GE, Aguiar PM de C. Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients [Internet]. Parkinsonism and Related Disorders. 2023 ; 116 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.parkreldis.2023.105847

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