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Artigo de periodico
NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes (2021)
Costa, Frederico Ribeiro Campos ; Leite, Jefferson Antonio; Rassi, Diane Meyre ; Silva, Josiane Fernandes da ; Oliveira, Jefferson Elias ; Guimarães, Jhefferson Barbosa ; Freitas, Maria Cristina Foss de ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Carlos, Daniela
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, DIABETES MELLITUS, DOENÇAS AUTOIMUNES, PÂNCREAS, CAMUNDONGOS, POLIMORFISMO, SISTEMA IMUNE, INTERLEUCINAS
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COSTA, Frederico Ribeiro Campos et al. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, v. 35, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2021.109176. Acesso em: 12 nov. 2024.
APA
Costa, F. R. C., Leite, J. A., Rassi, D. M., Silva, J. F. da, Oliveira, J. E., Guimarães, J. B., et al. (2021). NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, 35, 1-16. doi:10.1016/j.celrep.2021.109176
NLM
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Vancouver
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
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SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 12 nov. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Artigo de periodico
NLRP3 inflammasome and mineralocorticoid receptors are associated with vascular dysfunction in type 2 diabetes mellitus (2019)
Ferreira, Nathanne Santos ; Bruder-Nascimento, Thiago; Pereira, Camila André; Zanotto, Camila Ziliotto; Prado, Douglas Silva ; Silva, Josiane Fernandes da ; Rassi, Diane Meyre ; Foss-Freitas, Maria Cristina ; Alves Filho, José Carlos Farias ; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: Cells. Unidade: FMRP
Subjects: DIABETES MELLITUS, ALDOSTERONA, ARTÉRIAS
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FERREIRA, Nathanne Santos et al. NLRP3 inflammasome and mineralocorticoid receptors are associated with vascular dysfunction in type 2 diabetes mellitus. Cells, v. 8, n. 12, p. [13] , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8121595. Acesso em: 12 nov. 2024.
APA
Ferreira, N. S., Bruder-Nascimento, T., Pereira, C. A., Zanotto, C. Z., Prado, D. S., Silva, J. F. da, et al. (2019). NLRP3 inflammasome and mineralocorticoid receptors are associated with vascular dysfunction in type 2 diabetes mellitus. Cells, 8( 12), [13] . doi:10.3390/cells8121595
NLM
Ferreira NS, Bruder-Nascimento T, Pereira CA, Zanotto CZ, Prado DS, Silva JF da, Rassi DM, Foss-Freitas MC, Alves Filho JCF, Carlos D, Tostes R de CA. NLRP3 inflammasome and mineralocorticoid receptors are associated with vascular dysfunction in type 2 diabetes mellitus [Internet]. Cells. 2019 ; 8( 12): [13] .[citado 2024 nov. 12 ] Available from: https://doi.org/10.3390/cells8121595
Vancouver
Ferreira NS, Bruder-Nascimento T, Pereira CA, Zanotto CZ, Prado DS, Silva JF da, Rassi DM, Foss-Freitas MC, Alves Filho JCF, Carlos D, Tostes R de CA. NLRP3 inflammasome and mineralocorticoid receptors are associated with vascular dysfunction in type 2 diabetes mellitus [Internet]. Cells. 2019 ; 8( 12): [13] .[citado 2024 nov. 12 ] Available from: https://doi.org/10.3390/cells8121595
Artigo de periodico
Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis (2018)
Pereira, Camila A.; Carneiro, Fernando Silva; Matsumoto, Takayuki; Passaglia, Rita de Cássia Aleixo Tostes
Source: Basic and Clinical Pharmacology and Toxicology. Unidade: FMRP
Subjects: DIABETES MELLITUS, FÁRMACOS, ENDOTÉLIO, ARTERIOSCLEROSE
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PEREIRA, Camila A. et al. Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis. Basic and Clinical Pharmacology and Toxicology, v. 123, n. 5, p. 523-538, 2018Tradução . . Disponível em: https://doi.org/10.1111/bcpt.13054. Acesso em: 12 nov. 2024.
APA
Pereira, C. A., Carneiro, F. S., Matsumoto, T., & Passaglia, R. de C. A. T. (2018). Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis. Basic and Clinical Pharmacology and Toxicology, 123( 5), 523-538. doi:10.1111/bcpt.13054
NLM
Pereira CA, Carneiro FS, Matsumoto T, Passaglia R de CAT. Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis [Internet]. Basic and Clinical Pharmacology and Toxicology. 2018 ; 123( 5): 523-538.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/bcpt.13054
Vancouver
Pereira CA, Carneiro FS, Matsumoto T, Passaglia R de CAT. Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis [Internet]. Basic and Clinical Pharmacology and Toxicology. 2018 ; 123( 5): 523-538.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/bcpt.13054
Artigo de periodico
Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice (2018)
Neves, Karla Bianca; Montezano, Augusto Cesar; Alves-Lopes, Rheure; Bruder-Nascimento, Thiago; Costa, Rafael Menezes; Costa, Roberto Silva; Touyz, Rhian M.; Passaglia, Rita de Cássia Aleixo Tostes
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: RIM, DIABETES MELLITUS, ESTRESSE OXIDATIVO, RATOS
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NEVES, Karla Bianca et al. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice. International Journal of Molecular Sciences, v. 19, n. 8, 2018Tradução . . Disponível em: https://doi.org/10.3390/ijms19082454. Acesso em: 12 nov. 2024.
APA
Neves, K. B., Montezano, A. C., Alves-Lopes, R., Bruder-Nascimento, T., Costa, R. M., Costa, R. S., et al. (2018). Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice. International Journal of Molecular Sciences, 19( 8). doi:10.3390/ijms19082454
NLM
Neves KB, Montezano AC, Alves-Lopes R, Bruder-Nascimento T, Costa RM, Costa RS, Touyz RM, Passaglia R de CAT. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 8):[citado 2024 nov. 12 ] Available from: https://doi.org/10.3390/ijms19082454
Vancouver
Neves KB, Montezano AC, Alves-Lopes R, Bruder-Nascimento T, Costa RM, Costa RS, Touyz RM, Passaglia R de CAT. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 8):[citado 2024 nov. 12 ] Available from: https://doi.org/10.3390/ijms19082454
Artigo de periodico
Chemerin receptor blockade improves vascular function in diabetic obese mice via redox-sensitive and Akt-dependent pathways (2018)
Neves, Karla Bianca; Cat, Aurelie Nguyen Dinh; Lopes, Rheure Alves Moreira; Harvey, Katie Yates; Costa, Rafael Menezes da; Lobato, Núbia Souza; Montezano, Augusto Cesar Inforzato; Oliveira, Ana Maria de; Touyz, Rhian M.; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FCFRP, FMRP
Subjects: ENDOTÉLIO, CÉLULAS, INSULINA, DIABETES MELLITUS
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ABNT
NEVES, Karla Bianca et al. Chemerin receptor blockade improves vascular function in diabetic obese mice via redox-sensitive and Akt-dependent pathways. American Journal of Physiology-Heart and Circulatory Physiology, v. 315, n. 6, p. H1851-H1860, 2018Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00285.2018. Acesso em: 12 nov. 2024.
APA
Neves, K. B., Cat, A. N. D., Lopes, R. A. M., Harvey, K. Y., Costa, R. M. da, Lobato, N. S., et al. (2018). Chemerin receptor blockade improves vascular function in diabetic obese mice via redox-sensitive and Akt-dependent pathways. American Journal of Physiology-Heart and Circulatory Physiology, 315( 6), H1851-H1860. doi:10.1152/ajpheart.00285.2018
NLM
Neves KB, Cat AND, Lopes RAM, Harvey KY, Costa RM da, Lobato NS, Montezano ACI, Oliveira AM de, Touyz RM, Tostes R de CA. Chemerin receptor blockade improves vascular function in diabetic obese mice via redox-sensitive and Akt-dependent pathways [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2018 ; 315( 6): H1851-H1860.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpheart.00285.2018
Vancouver
Neves KB, Cat AND, Lopes RAM, Harvey KY, Costa RM da, Lobato NS, Montezano ACI, Oliveira AM de, Touyz RM, Tostes R de CA. Chemerin receptor blockade improves vascular function in diabetic obese mice via redox-sensitive and Akt-dependent pathways [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2018 ; 315( 6): H1851-H1860.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1152/ajpheart.00285.2018
Artigo de periodico
NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes (2017)
Carlos, Daniela; Costa, Frederico R. C.; Leite, Jefferson A.; André, Camila A.; Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Câmara, Niels Olsen Saraiva ; Silva, João Santana da
Source: Journal of Autoimmune Disorders. Unidade: FMRP
Subjects: DIABETES MELLITUS, INSULINA, IMUNOLOGIA
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ABNT
CARLOS, Daniela et al. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes. Journal of Autoimmune Disorders, v. 3, n. 2, 2017Tradução . . Disponível em: https://doi.org/10.21767/2471-8513.100033. Acesso em: 12 nov. 2024.
APA
Carlos, D., Costa, F. R. C., Leite, J. A., André, C. A., Tostes, R. de C. A., Carneiro, F. S., et al. (2017). NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes. Journal of Autoimmune Disorders, 3( 2). doi:10.21767/2471-8513.100033
NLM
Carlos D, Costa FRC, Leite JA, André CA, Tostes R de CA, Carneiro FS, Câmara NOS, Silva JS da. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes [Internet]. Journal of Autoimmune Disorders. 2017 ; 3( 2):[citado 2024 nov. 12 ] Available from: https://doi.org/10.21767/2471-8513.100033
Vancouver
Carlos D, Costa FRC, Leite JA, André CA, Tostes R de CA, Carneiro FS, Câmara NOS, Silva JS da. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes [Internet]. Journal of Autoimmune Disorders. 2017 ; 3( 2):[citado 2024 nov. 12 ] Available from: https://doi.org/10.21767/2471-8513.100033
Artigo de periodico
Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model (2017)
Carlos, Daniela ; Costa, Frederico Ribeiro Campos ; Pereira, Camila André; Rocha, Fernanda Agostini; Yaochite, Juliana Navarro Ueda; Oliveira, Gabriela G.; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Câmara, Niels Olsen Saraiva ; Ryffel, Bernhard; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB
Subjects: CITOCINAS, DIABETES MELLITUS, DNA MITOCONDRIAL
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ABNT
CARLOS, Daniela et al. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00164. Acesso em: 12 nov. 2024.
APA
Carlos, D., Costa, F. R. C., Pereira, C. A., Rocha, F. A., Yaochite, J. N. U., Oliveira, G. G., et al. (2017). Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00164
NLM
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Vancouver
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Trabalho de evento-resumo
NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes (2016)
Ferreira, Nathanne S.; Bruder-Nascimento, Thiago; Pereira, Camila A.; Zanotto, Camila Z.; Prado, Douglas S.; Alves-Filho, José Carlos; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: Final Program. Conference titles: Council on Hypertension 2016. Unidade: FMRP
Subjects: INFLAMAÇÃO, ALDOSTERONA, DIABETES MELLITUS
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ABNT
FERREIRA, Nathanne S. et al. NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. 2016, Anais.. Orlando: AHA, 2016. . Acesso em: 12 nov. 2024.
APA
Ferreira, N. S., Bruder-Nascimento, T., Pereira, C. A., Zanotto, C. Z., Prado, D. S., Alves-Filho, J. C., et al. (2016). NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. In Final Program. Orlando: AHA.
NLM
Ferreira NS, Bruder-Nascimento T, Pereira CA, Zanotto CZ, Prado DS, Alves-Filho JC, Carlos D, Tostes R de CA. NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. Final Program. 2016 ;[citado 2024 nov. 12 ]
Vancouver
Ferreira NS, Bruder-Nascimento T, Pereira CA, Zanotto CZ, Prado DS, Alves-Filho JC, Carlos D, Tostes R de CA. NLRP3/inflammasome activation contributes to aldosterone-induced vascular dysfunction in type 2 diabetes. Final Program. 2016 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice (2016)
Pereira, Camila A.; Ferreira, Nathanne S.; Zanotto, Camila Z.; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: Final Program. Conference titles: Council on Hypertension 2016. Unidade: FMRP
Subjects: INFLAMAÇÃO, DIABETES MELLITUS, DOENÇAS CARDIOVASCULARES
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ABNT
PEREIRA, Camila A. et al. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. 2016, Anais.. Orlando: AHA, 2016. . Acesso em: 12 nov. 2024.
APA
Pereira, C. A., Ferreira, N. S., Zanotto, C. Z., Carlos, D., & Tostes, R. de C. A. (2016). NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. In Final Program. Orlando: AHA.
NLM
Pereira CA, Ferreira NS, Zanotto CZ, Carlos D, Tostes R de CA. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. Final Program. 2016 ;[citado 2024 nov. 12 ]
Vancouver
Pereira CA, Ferreira NS, Zanotto CZ, Carlos D, Tostes R de CA. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. Final Program. 2016 ;[citado 2024 nov. 12 ]
Artigo de periodico
Renoprotective effects of atorvastatin in diabetic mice: downregulation of RhoA and upregulation of Akt/GSK3 (2016)
Bruder-Nascimento, Thiago; Callera, Glaucia; Montezano, Augusto Cesar; Antunes, Tayze T.; He, Ying; Cat, Aurelie Nguyen Dinh; Ferreira, Nathanne S.; Barreto, Pedro A.; Olivon, Vânia C.; Tostes, Rita de Cássia Aleixo; Touyz, Rhian M.
Source: PLOS One. Unidade: FMRP
Subjects: NEFROPATIAS EM ANIMAL, DIABETES MELLITUS, DOENÇA CRÔNICA
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ABNT
BRUDER-NASCIMENTO, Thiago et al. Renoprotective effects of atorvastatin in diabetic mice: downregulation of RhoA and upregulation of Akt/GSK3. PLOS One, v. 11, n. 9, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0162731. Acesso em: 12 nov. 2024.
APA
Bruder-Nascimento, T., Callera, G., Montezano, A. C., Antunes, T. T., He, Y., Cat, A. N. D., et al. (2016). Renoprotective effects of atorvastatin in diabetic mice: downregulation of RhoA and upregulation of Akt/GSK3. PLOS One, 11( 9). doi:10.1371/journal.pone.0162731
NLM
Bruder-Nascimento T, Callera G, Montezano AC, Antunes TT, He Y, Cat AND, Ferreira NS, Barreto PA, Olivon VC, Tostes R de CA, Touyz RM. Renoprotective effects of atorvastatin in diabetic mice: downregulation of RhoA and upregulation of Akt/GSK3 [Internet]. PLOS One. 2016 ; 11( 9):[citado 2024 nov. 12 ] Available from: https://doi.org/10.1371/journal.pone.0162731
Vancouver
Bruder-Nascimento T, Callera G, Montezano AC, Antunes TT, He Y, Cat AND, Ferreira NS, Barreto PA, Olivon VC, Tostes R de CA, Touyz RM. Renoprotective effects of atorvastatin in diabetic mice: downregulation of RhoA and upregulation of Akt/GSK3 [Internet]. PLOS One. 2016 ; 11( 9):[citado 2024 nov. 12 ] Available from: https://doi.org/10.1371/journal.pone.0162731
Artigo de periodico
Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms (2016)
Lopes, Rhéure Alves Moreira; Neves, Karla B.; Montezano, Augusto C.; Harvey, Adam; Carneiro, Fernando Silva; Touyz, Rhian M.; Tostes, Rita de Cássia Aleixo
Source: Hypertension. Unidade: FMRP
Subjects: DIABETES MELLITUS, PROTEÍNAS QUINASES, DISFUNÇÃO ERÉTIL, ESTRESSE OXIDATIVO
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ABNT
LOPES, Rhéure Alves Moreira et al. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, v. 68, n. 4, p. 1056-1064, 2016Tradução . . Disponível em: https://doi.org/10.1161/hypertensionaha.116.07518. Acesso em: 12 nov. 2024.
APA
Lopes, R. A. M., Neves, K. B., Montezano, A. C., Harvey, A., Carneiro, F. S., Touyz, R. M., & Tostes, R. de C. A. (2016). Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, 68( 4), 1056-1064. doi:10.1161/hypertensionaha.116.07518
NLM
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518
Vancouver
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518
Artigo de periodico
Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes (2015)
Matsumoto, Takayuki; Goulopoulou, Styliani; Taguchi, Kumiko; Tostes, Rita de Cássia Aleixo; Kobayashi, Tsuneo
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: ENDOTÉLIO VASCULAR, ADENOSINA, HIPERTENSÃO, DIABETES MELLITUS
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ABNT
MATSUMOTO, Takayuki et al. Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes. British Journal of Pharmacology, v. 172, n. 16, p. 3980-4001, 2015Tradução . . Disponível em: https://doi.org/10.1111/bph.13205. Acesso em: 12 nov. 2024.
APA
Matsumoto, T., Goulopoulou, S., Taguchi, K., Tostes, R. de C. A., & Kobayashi, T. (2015). Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes. British Journal of Pharmacology, 172( 16), 3980-4001. doi:10.1111/bph.13205
NLM
Matsumoto T, Goulopoulou S, Taguchi K, Tostes R de CA, Kobayashi T. Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes [Internet]. British Journal of Pharmacology. 2015 ; 172( 16): 3980-4001.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/bph.13205
Vancouver
Matsumoto T, Goulopoulou S, Taguchi K, Tostes R de CA, Kobayashi T. Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes [Internet]. British Journal of Pharmacology. 2015 ; 172( 16): 3980-4001.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/bph.13205
Artigo de periodico
Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation (2015)
Ferreira, Nathanne S.; Cau, Stefany B. A; Silva, Marcondes A. B.; Manzato, Carla Pavan; Mestriner, Fabiola Leslie Antunes Cardoso; Matsumoto, Takayuki; Carneiro, Fernando Silva; Passaglia, Rita de Cassia Aleixo Tostes
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: ALDOSTERONA, CÉLULAS ENDOTELIAIS, DIABETES MELLITUS, RECEPTORES HORMONAIS, MODELOS ANIMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Nathanne S. et al. Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation. Frontiers in Pharmacology, v. 6, 2015Tradução . . Disponível em: https://doi.org/10.3389/fphar.2015.00034. Acesso em: 12 nov. 2024.
APA
Ferreira, N. S., Cau, S. B. A., Silva, M. A. B., Manzato, C. P., Mestriner, F. L. A. C., Matsumoto, T., et al. (2015). Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation. Frontiers in Pharmacology, 6. doi:10.3389/fphar.2015.00034
NLM
Ferreira NS, Cau SBA, Silva MAB, Manzato CP, Mestriner FLAC, Matsumoto T, Carneiro FS, Passaglia R de CAT. Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation [Internet]. Frontiers in Pharmacology. 2015 ; 6[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fphar.2015.00034
Vancouver
Ferreira NS, Cau SBA, Silva MAB, Manzato CP, Mestriner FLAC, Matsumoto T, Carneiro FS, Passaglia R de CAT. Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation [Internet]. Frontiers in Pharmacology. 2015 ; 6[citado 2024 nov. 12 ] Available from: https://doi.org/10.3389/fphar.2015.00034
Artigo de periodico
The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes (2014)
Bruder-Nascimento, Thiago; Silva, Marcondes Alves Barbosa da; Tostes, Rita de Cássia Aleixo
Source: Diabetology & Metabolic Syndrome. Unidade: FMRP
Subjects: ALDOSTERONA, DIABETES MELLITUS, INSULINA, OBESIDADE, RESISTÊNCIA VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRUDER-NASCIMENTO, Thiago e SILVA, Marcondes Alves Barbosa da e TOSTES, Rita de Cássia Aleixo. The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes. Diabetology & Metabolic Syndrome, v. 6, 2014Tradução . . Disponível em: https://doi.org/10.1186/1758-5996-6-90. Acesso em: 12 nov. 2024.
APA
Bruder-Nascimento, T., Silva, M. A. B. da, & Tostes, R. de C. A. (2014). The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes. Diabetology & Metabolic Syndrome, 6. doi:10.1186/1758-5996-6-90
NLM
Bruder-Nascimento T, Silva MAB da, Tostes R de CA. The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes [Internet]. Diabetology & Metabolic Syndrome. 2014 ; 6[citado 2024 nov. 12 ] Available from: https://doi.org/10.1186/1758-5996-6-90
Vancouver
Bruder-Nascimento T, Silva MAB da, Tostes R de CA. The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes [Internet]. Diabetology & Metabolic Syndrome. 2014 ; 6[citado 2024 nov. 12 ] Available from: https://doi.org/10.1186/1758-5996-6-90
Artigo de periodico
Linking the beneficial effects of current therapeutic approaches in diabetes to the vascular endothelin system (2014)
Matsumoto, Takayuki; Lopes, Rheure A. M.; Taguchi, Kumiko; Kobayashi, Tsuneo; Tostes, Rita de Cassia Aleixo
Source: Life Science. Unidade: FMRP
Subjects: DIABETES MELLITUS, ENDOTÉLIO VASCULAR, MÚSCULO LISO VASCULAR, ENDOTELINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MATSUMOTO, Takayuki et al. Linking the beneficial effects of current therapeutic approaches in diabetes to the vascular endothelin system. Life Science, v. 118, n. 2, p. 129-135, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.12.216. Acesso em: 12 nov. 2024.
APA
Matsumoto, T., Lopes, R. A. M., Taguchi, K., Kobayashi, T., & Tostes, R. de C. A. (2014). Linking the beneficial effects of current therapeutic approaches in diabetes to the vascular endothelin system. Life Science, 118( 2), 129-135. doi:10.1016/j.lfs.2013.12.216
NLM
Matsumoto T, Lopes RAM, Taguchi K, Kobayashi T, Tostes R de CA. Linking the beneficial effects of current therapeutic approaches in diabetes to the vascular endothelin system [Internet]. Life Science. 2014 ; 118( 2): 129-135.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.lfs.2013.12.216
Vancouver
Matsumoto T, Lopes RAM, Taguchi K, Kobayashi T, Tostes R de CA. Linking the beneficial effects of current therapeutic approaches in diabetes to the vascular endothelin system [Internet]. Life Science. 2014 ; 118( 2): 129-135.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.lfs.2013.12.216
Trabalho de evento-resumo periodico
G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction (2014)
Ferreira, Nathanne dos Santos; Cau, Stefany B.; Manzato, Carla P.; Silva, Marcondes A.; Carneiro, Fernando Silva; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENDOTÉLIO VASCULAR, DIABETES MELLITUS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Nathanne dos Santos et al. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366. Acesso em: 12 nov. 2024. , 2014
APA
Ferreira, N. dos S., Cau, S. B., Manzato, C. P., Silva, M. A., Carneiro, F. S., & Passaglia, R. de C. A. T. (2014). G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366
NLM
Ferreira N dos S, Cau SB, Manzato CP, Silva MA, Carneiro FS, Passaglia R de CAT. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction [Internet]. Hypertension. 2014 ; 64[citado 2024 nov. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366
Vancouver
Ferreira N dos S, Cau SB, Manzato CP, Silva MA, Carneiro FS, Passaglia R de CAT. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction [Internet]. Hypertension. 2014 ; 64[citado 2024 nov. 12 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A337.abstract?sid=e3c869a1-749e-4bcb-bf9a-4c8b5536e366
Trabalho de evento-resumo
G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction (2014)
Ferreira, Nathanne; Cau, Stêfany B.; Manzato, Carla Pavan; Silva, Marcondes A.; Carneiro, Fernando Silva; Tostes, Rita de Cassia Aleixo
Source: Final Program. Conference titles: High Blood Pressure Research - Scientific Sessions. Unidade: FMRP
Subjects: ENDOTÉLIO VASCULAR, DIABETES MELLITUS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Nathanne et al. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. 2014, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2014. . Acesso em: 12 nov. 2024.
APA
Ferreira, N., Cau, S. B., Manzato, C. P., Silva, M. A., Carneiro, F. S., & Tostes, R. de C. A. (2014). G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. In Final Program. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Ferreira N, Cau SB, Manzato CP, Silva MA, Carneiro FS, Tostes R de CA. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Final Program. 2014 ;[citado 2024 nov. 12 ]
Vancouver
Ferreira N, Cau SB, Manzato CP, Silva MA, Carneiro FS, Tostes R de CA. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Final Program. 2014 ;[citado 2024 nov. 12 ]
Artigo de periodico
O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? (2012)
Lima, Victor V.; Spitler, Kathryn; Choi, Hyehun; Webb, R. Clinton; Tostes, Rita de Cassia Aleixo
Source: Clinical Science. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR (METABOLISMO), DIABETES MELLITUS, ESTRESSE OXIDATIVO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Victor V. et al. O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter?. Clinical Science, v. 12, n. 8, p. 473-486, 2012Tradução . . Disponível em: https://doi.org/10.1042/CS20110638. Acesso em: 12 nov. 2024.
APA
Lima, V. V., Spitler, K., Choi, H., Webb, R. C., & Tostes, R. de C. A. (2012). O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? Clinical Science, 12( 8), 473-486. doi:10.1042/CS20110638
NLM
Lima VV, Spitler K, Choi H, Webb RC, Tostes R de CA. O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? [Internet]. Clinical Science. 2012 ; 12( 8): 473-486.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1042/CS20110638
Vancouver
Lima VV, Spitler K, Choi H, Webb RC, Tostes R de CA. O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? [Internet]. Clinical Science. 2012 ; 12( 8): 473-486.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1042/CS20110638

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