Filtros : "Indexado no Index Medicus" "Antunes-Rodrigues, José" "FMRP" Removidos: "Petersen, James B." "Indexado no : Science Citation Index Expanded" "Indexado no Biological Abstracts" "RPM" "Margatho, Lisandra Oliveira" "EVORA, PAULO ROBERTO BARBOSA" Limpar

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  • Source: American Journal of Hypertension. Unidade: FMRP

    Subjects: ÁLCOOL, HIPERTENSÃO, VASOPRESSINAS

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      RESSTEL, Leonardo B. M. et al. Increase circulating vasopressin may account for ethanol-induced hypertension in rats. American Journal of Hypertension, v. 21, n. 8, p. 930-935, 2008Tradução . . Acesso em: 14 out. 2024.
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      Resstel, L. B. M., Scopinho, A. A., Silva, A. L. da, Antunes-Rodrigues, J., & Corrêa, F. M. de A. (2008). Increase circulating vasopressin may account for ethanol-induced hypertension in rats. American Journal of Hypertension, 21( 8), 930-935.
    • NLM

      Resstel LBM, Scopinho AA, Silva AL da, Antunes-Rodrigues J, Corrêa FM de A. Increase circulating vasopressin may account for ethanol-induced hypertension in rats. American Journal of Hypertension. 2008 ; 21( 8): 930-935.[citado 2024 out. 14 ]
    • Vancouver

      Resstel LBM, Scopinho AA, Silva AL da, Antunes-Rodrigues J, Corrêa FM de A. Increase circulating vasopressin may account for ethanol-induced hypertension in rats. American Journal of Hypertension. 2008 ; 21( 8): 930-935.[citado 2024 out. 14 ]
  • Source: Experimental Neurology. Unidade: FMRP

    Subjects: NEUROENDOCRINOLOGIA (FISIOLOGIA), SÓDIO

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      BADAUÊ-PASSOS JÚNIOR, Daniel et al. Dorsal raphe nuclei integrate allostatic information evoked by depletion-induced sodium ingestion. Experimental Neurology, v. 206, n. 2, p. 86-94, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2007.04.008. Acesso em: 14 out. 2024.
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      Badauê-Passos Júnior, D., Godino, A., Johnson, A. K., Vivas, L., & Antunes-Rodrigues, J. (2007). Dorsal raphe nuclei integrate allostatic information evoked by depletion-induced sodium ingestion. Experimental Neurology, 206( 2), 86-94. doi:10.1016/j.expneurol.2007.04.008
    • NLM

      Badauê-Passos Júnior D, Godino A, Johnson AK, Vivas L, Antunes-Rodrigues J. Dorsal raphe nuclei integrate allostatic information evoked by depletion-induced sodium ingestion [Internet]. Experimental Neurology. 2007 ; 206( 2): 86-94.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.expneurol.2007.04.008
    • Vancouver

      Badauê-Passos Júnior D, Godino A, Johnson AK, Vivas L, Antunes-Rodrigues J. Dorsal raphe nuclei integrate allostatic information evoked by depletion-induced sodium ingestion [Internet]. Experimental Neurology. 2007 ; 206( 2): 86-94.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.expneurol.2007.04.008
  • Source: Autonomic Neuroscience: Basic and Clinical. Unidade: FMRP

    Assunto: HIPERTENSÃO

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      ZOCCAL, Daniel B. et al. Plasma corticosterone levels is elevated in rats submitted to chronic intermittent hypoxia. Autonomic Neuroscience: Basic and Clinical, v. 134, n. 1, p. 115-117, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2007.01.004. Acesso em: 14 out. 2024.
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      Zoccal, D. B., Bonagamba, L. G. H., Antunes-Rodrigues, J., & Machado, B. H. (2007). Plasma corticosterone levels is elevated in rats submitted to chronic intermittent hypoxia. Autonomic Neuroscience: Basic and Clinical, 134( 1), 115-117. doi:10.1016/j.autneu.2007.01.004
    • NLM

      Zoccal DB, Bonagamba LGH, Antunes-Rodrigues J, Machado BH. Plasma corticosterone levels is elevated in rats submitted to chronic intermittent hypoxia [Internet]. Autonomic Neuroscience: Basic and Clinical. 2007 ; 134( 1): 115-117.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.autneu.2007.01.004
    • Vancouver

      Zoccal DB, Bonagamba LGH, Antunes-Rodrigues J, Machado BH. Plasma corticosterone levels is elevated in rats submitted to chronic intermittent hypoxia [Internet]. Autonomic Neuroscience: Basic and Clinical. 2007 ; 134( 1): 115-117.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.autneu.2007.01.004
  • Source: Experimental Physiology. Unidade: FMRP

    Subjects: NEUROENDOCRINOLOGIA, VASOPRESSINAS, SEDE (ALTERAÇÃO)

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      MAGALHÃES-NUNES, Ana Paula de et al. Sertraline, a selective serotonin reuptake inhibitor, affects thirst, salt appetite and plasma levels of oxytocin and vasopressin in rats. Experimental Physiology, v. 92, n. 5, p. 913-922, 2007Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2007.037358. Acesso em: 14 out. 2024.
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      Magalhães-Nunes, A. P. de, Badauê-Passos Júnior, D., Ventura, R. R., Guedes Júnior, D. da S., Araújo, J. P., Granadeiro, P. C., et al. (2007). Sertraline, a selective serotonin reuptake inhibitor, affects thirst, salt appetite and plasma levels of oxytocin and vasopressin in rats. Experimental Physiology, 92( 5), 913-922. doi:10.1113/expphysiol.2007.037358
    • NLM

      Magalhães-Nunes AP de, Badauê-Passos Júnior D, Ventura RR, Guedes Júnior D da S, Araújo JP, Granadeiro PC, Milanez-Barbosa HK, Costa-e-Sousa RH da, Medeiros MA de, Antunes-Rodrigues J, Reis LC. Sertraline, a selective serotonin reuptake inhibitor, affects thirst, salt appetite and plasma levels of oxytocin and vasopressin in rats [Internet]. Experimental Physiology. 2007 ; 92( 5): 913-922.[citado 2024 out. 14 ] Available from: https://doi.org/10.1113/expphysiol.2007.037358
    • Vancouver

      Magalhães-Nunes AP de, Badauê-Passos Júnior D, Ventura RR, Guedes Júnior D da S, Araújo JP, Granadeiro PC, Milanez-Barbosa HK, Costa-e-Sousa RH da, Medeiros MA de, Antunes-Rodrigues J, Reis LC. Sertraline, a selective serotonin reuptake inhibitor, affects thirst, salt appetite and plasma levels of oxytocin and vasopressin in rats [Internet]. Experimental Physiology. 2007 ; 92( 5): 913-922.[citado 2024 out. 14 ] Available from: https://doi.org/10.1113/expphysiol.2007.037358
  • Source: Shock. Unidade: FMRP

    Subjects: FARMACOLOGIA, FISIOLOGIA CARDIOVASCULAR

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      GIUSTI-PAIVA, Alexandre et al. Vasopressin mediates the pressor effect of hypertonic saline solution in endotoxic shock. Shock, v. 27, n. 4, p. 416-421, 2007Tradução . . Acesso em: 14 out. 2024.
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      Giusti-Paiva, A., Martinez, M. R., Bispo-da-Silva, L. B., Salgado, M. C. de O., Elias, L. L. K., & Antunes-Rodrigues, J. (2007). Vasopressin mediates the pressor effect of hypertonic saline solution in endotoxic shock. Shock, 27( 4), 416-421.
    • NLM

      Giusti-Paiva A, Martinez MR, Bispo-da-Silva LB, Salgado MC de O, Elias LLK, Antunes-Rodrigues J. Vasopressin mediates the pressor effect of hypertonic saline solution in endotoxic shock. Shock. 2007 ; 27( 4): 416-421.[citado 2024 out. 14 ]
    • Vancouver

      Giusti-Paiva A, Martinez MR, Bispo-da-Silva LB, Salgado MC de O, Elias LLK, Antunes-Rodrigues J. Vasopressin mediates the pressor effect of hypertonic saline solution in endotoxic shock. Shock. 2007 ; 27( 4): 416-421.[citado 2024 out. 14 ]
  • Source: Hormones and Behavior. Unidades: FMRP, ICB

    Subjects: HIPOTÁLAMO, ENDOTOXINAS

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      BORGES, B. C. et al. Expression of hypothalamic neuropeptides and the desensitization of pituitary-adrenal axis and hypophagia in the endotoxin tolerance. Hormones and Behavior, v. 52, n. 4, p. 508-519, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2007.07.006. Acesso em: 14 out. 2024.
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      Borges, B. C., Antunes-Rodrigues, J., Castro, M. de, Bittencourt, J. C., Elias, C. F., & Elias, L. L. K. (2007). Expression of hypothalamic neuropeptides and the desensitization of pituitary-adrenal axis and hypophagia in the endotoxin tolerance. Hormones and Behavior, 52( 4), 508-519. doi:10.1016/j.yhbeh.2007.07.006
    • NLM

      Borges BC, Antunes-Rodrigues J, Castro M de, Bittencourt JC, Elias CF, Elias LLK. Expression of hypothalamic neuropeptides and the desensitization of pituitary-adrenal axis and hypophagia in the endotoxin tolerance [Internet]. Hormones and Behavior. 2007 ; 52( 4): 508-519.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.yhbeh.2007.07.006
    • Vancouver

      Borges BC, Antunes-Rodrigues J, Castro M de, Bittencourt JC, Elias CF, Elias LLK. Expression of hypothalamic neuropeptides and the desensitization of pituitary-adrenal axis and hypophagia in the endotoxin tolerance [Internet]. Hormones and Behavior. 2007 ; 52( 4): 508-519.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.yhbeh.2007.07.006
  • Source: Stress. Unidade: FMRP

    Subjects: VASOPRESSINAS, ADRENALECTOMIA ANIMAL

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      LAGUNA-ABREU, M. T. C. et al. The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat. Stress, v. 10, n. 4, p. 332-341, 2007Tradução . . Disponível em: https://doi.org/10.1080/10253890701287614. Acesso em: 14 out. 2024.
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      Laguna-Abreu, M. T. C., Margatho, L., Germano, C. M. R., Antunes-Rodrigues, J., Elias, L. L. K., & Castro, M. de. (2007). The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat. Stress, 10( 4), 332-341. doi:10.1080/10253890701287614
    • NLM

      Laguna-Abreu MTC, Margatho L, Germano CMR, Antunes-Rodrigues J, Elias LLK, Castro M de. The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat [Internet]. Stress. 2007 ; 10( 4): 332-341.[citado 2024 out. 14 ] Available from: https://doi.org/10.1080/10253890701287614
    • Vancouver

      Laguna-Abreu MTC, Margatho L, Germano CMR, Antunes-Rodrigues J, Elias LLK, Castro M de. The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat [Internet]. Stress. 2007 ; 10( 4): 332-341.[citado 2024 out. 14 ] Available from: https://doi.org/10.1080/10253890701287614
  • Source: Experimental Neurology. Unidade: FMRP

    Assunto: NEUROENDOCRINOLOGIA (FISIOLOGIA)

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      RUGINSK, S. G. et al. Glucocorticoid modulation of neuronal activity and hormone secretion induced by blood volume expansion. Experimental Neurology, v. 206, n. 2, p. 192-200, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2007.04.012. Acesso em: 14 out. 2024.
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      Ruginsk, S. G., Oliveira, F. R. T., Margatho, L. O., Vivas, L., Elias, L. L. K., & Antunes-Rodrigues, J. (2007). Glucocorticoid modulation of neuronal activity and hormone secretion induced by blood volume expansion. Experimental Neurology, 206( 2), 192-200. doi:10.1016/j.expneurol.2007.04.012
    • NLM

      Ruginsk SG, Oliveira FRT, Margatho LO, Vivas L, Elias LLK, Antunes-Rodrigues J. Glucocorticoid modulation of neuronal activity and hormone secretion induced by blood volume expansion [Internet]. Experimental Neurology. 2007 ; 206( 2): 192-200.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.expneurol.2007.04.012
    • Vancouver

      Ruginsk SG, Oliveira FRT, Margatho LO, Vivas L, Elias LLK, Antunes-Rodrigues J. Glucocorticoid modulation of neuronal activity and hormone secretion induced by blood volume expansion [Internet]. Experimental Neurology. 2007 ; 206( 2): 192-200.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.expneurol.2007.04.012
  • Source: Molecular Psychiatry. Unidade: FMRP

    Subjects: RATOS, NEUROSSECREÇÃO, IMUNOFLUORESCÊNCIA, HIPOTÁLAMO

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      MARRONI, Simone Saldanha et al. Neuroanatomical and cellular substrates of hypergrooming induced by microinjection of oxytocin in central nucleus of amygdala: an experimental model of compulsive behavior. Molecular Psychiatry, v. 11, p. 1-15, 2007Tradução . . Disponível em: https://doi.org/10.1038/sj.mp.4002015. Acesso em: 14 out. 2024.
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      Marroni, S. S., Nakano, F. N., Gati, C. D. C., Oliveira, J. A. C., Antunes-Rodrigues, J., & Garcia-Cairasco, N. (2007). Neuroanatomical and cellular substrates of hypergrooming induced by microinjection of oxytocin in central nucleus of amygdala: an experimental model of compulsive behavior. Molecular Psychiatry, 11, 1-15. doi:10.1038/sj.mp.4002015
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      Marroni SS, Nakano FN, Gati CDC, Oliveira JAC, Antunes-Rodrigues J, Garcia-Cairasco N. Neuroanatomical and cellular substrates of hypergrooming induced by microinjection of oxytocin in central nucleus of amygdala: an experimental model of compulsive behavior [Internet]. Molecular Psychiatry. 2007 ; 11 1-15.[citado 2024 out. 14 ] Available from: https://doi.org/10.1038/sj.mp.4002015
    • Vancouver

      Marroni SS, Nakano FN, Gati CDC, Oliveira JAC, Antunes-Rodrigues J, Garcia-Cairasco N. Neuroanatomical and cellular substrates of hypergrooming induced by microinjection of oxytocin in central nucleus of amygdala: an experimental model of compulsive behavior [Internet]. Molecular Psychiatry. 2007 ; 11 1-15.[citado 2024 out. 14 ] Available from: https://doi.org/10.1038/sj.mp.4002015
  • Source: Regulatory Peptides. Unidades: FMRP, EERP

    Subjects: CHOQUE SÉPTICO, PRESSÃO SANGUÍNEA, VASOPRESSINAS

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      STABILE, Angelita Maria et al. Participation of the inducible nitric oxide synthase on atrial natriuretic peptide plasma concentration during endotoxemic shock. Regulatory Peptides, v. 140, n. 3, p. 136-141, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.regpep.2006.12.001. Acesso em: 14 out. 2024.
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      Stabile, A. M., Moreto, V., Antunes-Rodrigues, J., & Cárnio, E. C. (2007). Participation of the inducible nitric oxide synthase on atrial natriuretic peptide plasma concentration during endotoxemic shock. Regulatory Peptides, 140( 3), 136-141. doi:10.1016/j.regpep.2006.12.001
    • NLM

      Stabile AM, Moreto V, Antunes-Rodrigues J, Cárnio EC. Participation of the inducible nitric oxide synthase on atrial natriuretic peptide plasma concentration during endotoxemic shock [Internet]. Regulatory Peptides. 2007 ; 140( 3): 136-141.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.regpep.2006.12.001
    • Vancouver

      Stabile AM, Moreto V, Antunes-Rodrigues J, Cárnio EC. Participation of the inducible nitric oxide synthase on atrial natriuretic peptide plasma concentration during endotoxemic shock [Internet]. Regulatory Peptides. 2007 ; 140( 3): 136-141.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.regpep.2006.12.001
  • Source: Experimental Physiology. Unidade: FMRP

    Assunto: FISIOLOGIA

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      ZOCCAL, Daniel B. et al. Increased sympathetic activity in rats submitted to chronic intermittent hypoxia. Experimental Physiology, v. 92, n. 1, p. 79-85, 2006Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2006.035501. Acesso em: 14 out. 2024.
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      Zoccal, D. B., Bonagamba, L. G. H., Oliveira, F. R. T., Antunes-Rodrigues, J., & Machado, B. H. (2006). Increased sympathetic activity in rats submitted to chronic intermittent hypoxia. Experimental Physiology, 92( 1), 79-85. doi:10.1113/expphysiol.2006.035501
    • NLM

      Zoccal DB, Bonagamba LGH, Oliveira FRT, Antunes-Rodrigues J, Machado BH. Increased sympathetic activity in rats submitted to chronic intermittent hypoxia [Internet]. Experimental Physiology. 2006 ; 92( 1): 79-85.[citado 2024 out. 14 ] Available from: https://doi.org/10.1113/expphysiol.2006.035501
    • Vancouver

      Zoccal DB, Bonagamba LGH, Oliveira FRT, Antunes-Rodrigues J, Machado BH. Increased sympathetic activity in rats submitted to chronic intermittent hypoxia [Internet]. Experimental Physiology. 2006 ; 92( 1): 79-85.[citado 2024 out. 14 ] Available from: https://doi.org/10.1113/expphysiol.2006.035501
  • Source: Shock. Unidades: FMRP, EERP

    Subjects: CHOQUE SÉPTICO, VASOPRESSINAS

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      MORETO, Viviana et al. Role of heme-oxygenase pathway on vasopressin deficiency during endotoxemic shock-like conditions. Shock, v. 26, n. 5, p. 472-476, 2006Tradução . . Acesso em: 14 out. 2024.
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      Moreto, V., Stabile, A. M., Antunes-Rodrigues, J., & Cárnio, E. C. (2006). Role of heme-oxygenase pathway on vasopressin deficiency during endotoxemic shock-like conditions. Shock, 26( 5), 472-476.
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      Moreto V, Stabile AM, Antunes-Rodrigues J, Cárnio EC. Role of heme-oxygenase pathway on vasopressin deficiency during endotoxemic shock-like conditions. Shock. 2006 ; 26( 5): 472-476.[citado 2024 out. 14 ]
    • Vancouver

      Moreto V, Stabile AM, Antunes-Rodrigues J, Cárnio EC. Role of heme-oxygenase pathway on vasopressin deficiency during endotoxemic shock-like conditions. Shock. 2006 ; 26( 5): 472-476.[citado 2024 out. 14 ]
  • Source: Endocrine, Metabolic & Immune Disorders-Drug Targets. Unidades: EERP, FMRP

    Subjects: CHOQUE SÉPTICO, CITOCINAS, ÓXIDO NÍTRICO

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      CÁRNIO, Evelin Capellari et al. Neuro-immune-endocrine mechanisms during septic shock: role for nitric oxide in vasopressin and oxytocin release. Endocrine, Metabolic & Immune Disorders-Drug Targets, v. 6, n. 2, p. 7-16, 2006Tradução . . Acesso em: 14 out. 2024.
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      Cárnio, E. C., Moreto, V., Giusti-Paiva, A., & Antunes-Rodrigues, J. (2006). Neuro-immune-endocrine mechanisms during septic shock: role for nitric oxide in vasopressin and oxytocin release. Endocrine, Metabolic & Immune Disorders-Drug Targets, 6( 2), 7-16.
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      Cárnio EC, Moreto V, Giusti-Paiva A, Antunes-Rodrigues J. Neuro-immune-endocrine mechanisms during septic shock: role for nitric oxide in vasopressin and oxytocin release. Endocrine, Metabolic & Immune Disorders-Drug Targets. 2006 ; 6( 2): 7-16.[citado 2024 out. 14 ]
    • Vancouver

      Cárnio EC, Moreto V, Giusti-Paiva A, Antunes-Rodrigues J. Neuro-immune-endocrine mechanisms during septic shock: role for nitric oxide in vasopressin and oxytocin release. Endocrine, Metabolic & Immune Disorders-Drug Targets. 2006 ; 6( 2): 7-16.[citado 2024 out. 14 ]
  • Source: Regulatory Peptides. Unidades: FM, FMRP

    Subjects: VASOPRESSINAS, RECEPTORES DE ANGIOTENSINA

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      SAAD, Wilson Abrão et al. Interaction between arginine vasopressin and angiotensin II receptors in the central regulation of sodium balance. Regulatory Peptides, v. 132, p. 53-58, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.regpep.2005.09.003. Acesso em: 14 out. 2024.
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      Saad, W. A., Camargo, L. A. de A., Antunes-Rodrigues, J., Saad, W. A., Guarda, I. F. M. S., & Guarda, R. S. (2005). Interaction between arginine vasopressin and angiotensin II receptors in the central regulation of sodium balance. Regulatory Peptides, 132, 53-58. doi:10.1016/j.regpep.2005.09.003
    • NLM

      Saad WA, Camargo LA de A, Antunes-Rodrigues J, Saad WA, Guarda IFMS, Guarda RS. Interaction between arginine vasopressin and angiotensin II receptors in the central regulation of sodium balance [Internet]. Regulatory Peptides. 2005 ; 132 53-58.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.regpep.2005.09.003
    • Vancouver

      Saad WA, Camargo LA de A, Antunes-Rodrigues J, Saad WA, Guarda IFMS, Guarda RS. Interaction between arginine vasopressin and angiotensin II receptors in the central regulation of sodium balance [Internet]. Regulatory Peptides. 2005 ; 132 53-58.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.regpep.2005.09.003
  • Source: Journal of Endocrinology Investigation. Unidade: FMRP

    Subjects: ENZIMAS, DOENÇAS GENÉTICAS, PEPTÍDEO NATRIURÉTICO ATRIAL

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      GERMANO, C. M. R. et al. The interaction of plasma renin activity and plasma atrial natriuretic peptide in 21-hydroxylase deficiency patients. Journal of Endocrinology Investigation, v. 20, p. 300-304, 2005Tradução . . Disponível em: https://doi.org/10.1007/bf03347194. Acesso em: 14 out. 2024.
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      Germano, C. M. R., Castro, M. de, Crescêncio, J. C., Gallo Júnior, L., Antunes-Rodrigues, J., Moreira, A. C., & Elias, L. L. K. (2005). The interaction of plasma renin activity and plasma atrial natriuretic peptide in 21-hydroxylase deficiency patients. Journal of Endocrinology Investigation, 20, 300-304. doi:10.1007/bf03347194
    • NLM

      Germano CMR, Castro M de, Crescêncio JC, Gallo Júnior L, Antunes-Rodrigues J, Moreira AC, Elias LLK. The interaction of plasma renin activity and plasma atrial natriuretic peptide in 21-hydroxylase deficiency patients [Internet]. Journal of Endocrinology Investigation. 2005 ; 20 300-304.[citado 2024 out. 14 ] Available from: https://doi.org/10.1007/bf03347194
    • Vancouver

      Germano CMR, Castro M de, Crescêncio JC, Gallo Júnior L, Antunes-Rodrigues J, Moreira AC, Elias LLK. The interaction of plasma renin activity and plasma atrial natriuretic peptide in 21-hydroxylase deficiency patients [Internet]. Journal of Endocrinology Investigation. 2005 ; 20 300-304.[citado 2024 out. 14 ] Available from: https://doi.org/10.1007/bf03347194
  • Source: SHOCK. Unidades: FORP, EERP, FMRP

    Subjects: SEPSE, PRESSÃO SANGUÍNEA, LIPOPOLISSACARÍDEOS, ÓXIDO NÍTRICO (SÍNTESE)

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      GIUSTI-PAIVA, Alexandre et al. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK, v. 21, n. 3, p. 271-275, 2004Tradução . . Acesso em: 14 out. 2024.
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      Giusti-Paiva, A., Martinez, M. R., Felix, J. V. C., Rocha, M. J. A. da, Cárnio, E. C., Elias, L. L. K., & Antunes-Rodrigues, J. (2004). Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK, 21( 3), 271-275.
    • NLM

      Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Cárnio EC, Elias LLK, Antunes-Rodrigues J. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK. 2004 ; 21( 3): 271-275.[citado 2024 out. 14 ]
    • Vancouver

      Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Cárnio EC, Elias LLK, Antunes-Rodrigues J. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK. 2004 ; 21( 3): 271-275.[citado 2024 out. 14 ]
  • Source: Critical Care Medicine. Unidades: FMRP, EERP

    Subjects: CHOQUE SÉPTICO (FISIOLOGIA), ÓXIDO NÍTRICO

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      GIUSTI-PAIVA, Alexandre et al. Inducible nitric oxide synthase pathway in the central nervous system and vasopressin release during experimental septic shock. Critical Care Medicine, v. 30, n. 6, p. 1306-1310, 2002Tradução . . Disponível em: https://doi.org/10.1097/00003246-200206000-00025. Acesso em: 14 out. 2024.
    • APA

      Giusti-Paiva, A., Castro, M. de, Antunes-Rodrigues, J., & Cárnio, E. C. (2002). Inducible nitric oxide synthase pathway in the central nervous system and vasopressin release during experimental septic shock. Critical Care Medicine, 30( 6), 1306-1310. doi:10.1097/00003246-200206000-00025
    • NLM

      Giusti-Paiva A, Castro M de, Antunes-Rodrigues J, Cárnio EC. Inducible nitric oxide synthase pathway in the central nervous system and vasopressin release during experimental septic shock [Internet]. Critical Care Medicine. 2002 ; 30( 6): 1306-1310.[citado 2024 out. 14 ] Available from: https://doi.org/10.1097/00003246-200206000-00025
    • Vancouver

      Giusti-Paiva A, Castro M de, Antunes-Rodrigues J, Cárnio EC. Inducible nitric oxide synthase pathway in the central nervous system and vasopressin release during experimental septic shock [Internet]. Critical Care Medicine. 2002 ; 30( 6): 1306-1310.[citado 2024 out. 14 ] Available from: https://doi.org/10.1097/00003246-200206000-00025
  • Source: Brazilian Journal of Medical and Biological Research. Unidades: FMRP, FORP

    Subjects: FARMACOLOGIA, FISIOLOGIA

    Acesso à fonteAcesso à fonteDOIHow to cite
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    • ABNT

      MCCANN, S. M. et al. Role of the hypothalamic pituitary adrenal axis in the control of the response to stress and infection. Brazilian Journal of Medical and Biological Research, v. 33, n. 10, p. 1121-1131, 2000Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2000001000001. Acesso em: 14 out. 2024.
    • APA

      McCann, S. M., Antunes-Rodrigues, J., Franci, C. R., Anselmo-Franci, J. A., Karanth, S., & Rettori, V. (2000). Role of the hypothalamic pituitary adrenal axis in the control of the response to stress and infection. Brazilian Journal of Medical and Biological Research, 33( 10), 1121-1131. doi:10.1590/s0100-879x2000001000001
    • NLM

      McCann SM, Antunes-Rodrigues J, Franci CR, Anselmo-Franci JA, Karanth S, Rettori V. Role of the hypothalamic pituitary adrenal axis in the control of the response to stress and infection [Internet]. Brazilian Journal of Medical and Biological Research. 2000 ; 33( 10): 1121-1131.[citado 2024 out. 14 ] Available from: https://doi.org/10.1590/s0100-879x2000001000001
    • Vancouver

      McCann SM, Antunes-Rodrigues J, Franci CR, Anselmo-Franci JA, Karanth S, Rettori V. Role of the hypothalamic pituitary adrenal axis in the control of the response to stress and infection [Internet]. Brazilian Journal of Medical and Biological Research. 2000 ; 33( 10): 1121-1131.[citado 2024 out. 14 ] Available from: https://doi.org/10.1590/s0100-879x2000001000001
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP

    Assunto: FARMACOLOGIA

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    • ABNT

      PICANÇO-DINIZ, D. L. W. et al. Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro. Brazilian Journal of Medical and Biological Research, v. 32, n. 9, p. 1167-1173, 1999Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x1999000900017. Acesso em: 14 out. 2024.
    • APA

      Picanço-Diniz, D. L. W., Valença, M. M., Favaretto, A. L. V., McCann, S. M., & Antunes-Rodrigues, J. (1999). Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro. Brazilian Journal of Medical and Biological Research, 32( 9), 1167-1173. doi:10.1590/s0100-879x1999000900017
    • NLM

      Picanço-Diniz DLW, Valença MM, Favaretto ALV, McCann SM, Antunes-Rodrigues J. Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro [Internet]. Brazilian Journal of Medical and Biological Research. 1999 ; 32( 9): 1167-1173.[citado 2024 out. 14 ] Available from: https://doi.org/10.1590/s0100-879x1999000900017
    • Vancouver

      Picanço-Diniz DLW, Valença MM, Favaretto ALV, McCann SM, Antunes-Rodrigues J. Possible involvement of A[IND.1] receptors in the inhibition of gonadotropin secretion indeced by adenosine in rat hemipituitaries in vitro [Internet]. Brazilian Journal of Medical and Biological Research. 1999 ; 32( 9): 1167-1173.[citado 2024 out. 14 ] Available from: https://doi.org/10.1590/s0100-879x1999000900017
  • Source: Bollettino Chimico Farmaceutico. Conference titles: Congress of Pharmaceutical Sciences. Unidades: EERP, FMRP, FORP

    Assunto: SISTEMA CARDIOVASCULAR (FARMACOLOGIA)

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    • ABNT

      STEINER, Alexandre Alarcon et al. The central nitric oxide pathway plays a major role in systemic vasopressin-induced hypothermia. Bollettino Chimico Farmaceutico. Milano: Societá Editoriale Farmaceutica. . Acesso em: 14 out. 2024. , 1999
    • APA

      Steiner, A. A., Cárnio, E. C., Antunes-Rodrigues, J., & Branco, L. G. de S. (1999). The central nitric oxide pathway plays a major role in systemic vasopressin-induced hypothermia. Bollettino Chimico Farmaceutico. Milano: Societá Editoriale Farmaceutica.
    • NLM

      Steiner AA, Cárnio EC, Antunes-Rodrigues J, Branco LG de S. The central nitric oxide pathway plays a major role in systemic vasopressin-induced hypothermia. Bollettino Chimico Farmaceutico. 1999 ; 132( 2): CLXXXVII.[citado 2024 out. 14 ]
    • Vancouver

      Steiner AA, Cárnio EC, Antunes-Rodrigues J, Branco LG de S. The central nitric oxide pathway plays a major role in systemic vasopressin-induced hypothermia. Bollettino Chimico Farmaceutico. 1999 ; 132( 2): CLXXXVII.[citado 2024 out. 14 ]

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