ReP USP - Resultado da busca

Artigo de periodico
Synephrine and caffeine combination promotes cytotoxicity, DNA damage and transcriptional modulation of apoptosis-related genes in human HepG2 cells (2021)
Leão, Tainá Keiller ; Ribeiro, Diego Luis ; Machado, Ana Rita Thomazela ; Costa, Tássia Rafaella; Sampaio, Suely Vilela ; Antunes, Lusânia Maria Greggi
Source: Mutation Research - Genetic Toxicology and Environmental Mutagenesis. Unidade: FCFRP
Subjects: APOPTOSE, CAFEÍNA, CICLO CELULAR, SOBREVIVÊNCIA CELULAR, DANO AO DNA, NEOPLASIAS HEPÁTICAS, TRANSCRIÇÃO GÊNICA
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LEÃO, Tainá Keiller et al. Synephrine and caffeine combination promotes cytotoxicity, DNA damage and transcriptional modulation of apoptosis-related genes in human HepG2 cells. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, v. 868/869, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.mrgentox.2021.503375. Acesso em: 17 nov. 2024.
APA
Leão, T. K., Ribeiro, D. L., Machado, A. R. T., Costa, T. R., Sampaio, S. V., & Antunes, L. M. G. (2021). Synephrine and caffeine combination promotes cytotoxicity, DNA damage and transcriptional modulation of apoptosis-related genes in human HepG2 cells. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, 868/869. doi:10.1016/j.mrgentox.2021.503375
NLM
Leão TK, Ribeiro DL, Machado ART, Costa TR, Sampaio SV, Antunes LMG. Synephrine and caffeine combination promotes cytotoxicity, DNA damage and transcriptional modulation of apoptosis-related genes in human HepG2 cells [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2021 ; 868/869[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.mrgentox.2021.503375
Vancouver
Leão TK, Ribeiro DL, Machado ART, Costa TR, Sampaio SV, Antunes LMG. Synephrine and caffeine combination promotes cytotoxicity, DNA damage and transcriptional modulation of apoptosis-related genes in human HepG2 cells [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2021 ; 868/869[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.mrgentox.2021.503375
Artigo de periodico
Gold(III) complexes with thiosemicarbazonate ligands as potential anticancer agents: cytotoxicity and interactions with biomolecular targets (2021)
Possato, Bruna; Dalmolin, Luciana Facco; Pereira, Luiz Miguel; Alves, Jacqueline Querino; Silva, Raphael Tristão Cruvinel; Gelamo, Rogerio Valentim; Yatsuda, Ana Patrícia ; Lopez, Renata Fonseca Vianna ; Albuquerque, Sérgio de ; Leite, Natália Bueno; Maia, Pedro Ivo da Silva
Source: European Journal of Pharmaceutical Sciences. Unidades: FCFRP, PUSP-RP
Subjects: ANTINEOPLÁSICOS, APOPTOSE, LINHAGEM CELULAR, LIGANTES, NEOPLASIAS OVARIANAS
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POSSATO, Bruna et al. Gold(III) complexes with thiosemicarbazonate ligands as potential anticancer agents: cytotoxicity and interactions with biomolecular targets. European Journal of Pharmaceutical Sciences, v. 162, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.ejps.2021.105834. Acesso em: 17 nov. 2024.
APA
Possato, B., Dalmolin, L. F., Pereira, L. M., Alves, J. Q., Silva, R. T. C., Gelamo, R. V., et al. (2021). Gold(III) complexes with thiosemicarbazonate ligands as potential anticancer agents: cytotoxicity and interactions with biomolecular targets. European Journal of Pharmaceutical Sciences, 162. doi:10.1016/j.ejps.2021.105834
NLM
Possato B, Dalmolin LF, Pereira LM, Alves JQ, Silva RTC, Gelamo RV, Yatsuda AP, Lopez RFV, Albuquerque S de, Leite NB, Maia PI da S. Gold(III) complexes with thiosemicarbazonate ligands as potential anticancer agents: cytotoxicity and interactions with biomolecular targets [Internet]. European Journal of Pharmaceutical Sciences. 2021 ; 162[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ejps.2021.105834
Vancouver
Possato B, Dalmolin LF, Pereira LM, Alves JQ, Silva RTC, Gelamo RV, Yatsuda AP, Lopez RFV, Albuquerque S de, Leite NB, Maia PI da S. Gold(III) complexes with thiosemicarbazonate ligands as potential anticancer agents: cytotoxicity and interactions with biomolecular targets [Internet]. European Journal of Pharmaceutical Sciences. 2021 ; 162[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ejps.2021.105834
Artigo de periodico
Intravitreal ketamine promotes neuroprotection in rat eyes after experimental ischemia (2021)
Dourado, Lays Fernanda Nunes; Oliveira, Lucas Gomes; Silva, Carolina Nunes da; Toledo, Cibele Rodrigues; Fialho, Silvia Ligório; Jorge, Rodrigo ; Silva-Cunha, Armando
Source: Biomedicine & Pharmacotherapy. Unidade: FMRP
Subjects: ANIMAIS, APOPTOSE, ANTAGONISTAS, ISQUEMIA, RATOS WISTAR
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DOURADO, Lays Fernanda Nunes et al. Intravitreal ketamine promotes neuroprotection in rat eyes after experimental ischemia. Biomedicine & Pharmacotherapy, v. 133, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2020.110948. Acesso em: 17 nov. 2024.
APA
Dourado, L. F. N., Oliveira, L. G., Silva, C. N. da, Toledo, C. R., Fialho, S. L., Jorge, R., & Silva-Cunha, A. (2021). Intravitreal ketamine promotes neuroprotection in rat eyes after experimental ischemia. Biomedicine & Pharmacotherapy, 133. doi:10.1016/j.biopha.2020.110948
NLM
Dourado LFN, Oliveira LG, Silva CN da, Toledo CR, Fialho SL, Jorge R, Silva-Cunha A. Intravitreal ketamine promotes neuroprotection in rat eyes after experimental ischemia [Internet]. Biomedicine & Pharmacotherapy. 2021 ; 133[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.biopha.2020.110948
Vancouver
Dourado LFN, Oliveira LG, Silva CN da, Toledo CR, Fialho SL, Jorge R, Silva-Cunha A. Intravitreal ketamine promotes neuroprotection in rat eyes after experimental ischemia [Internet]. Biomedicine & Pharmacotherapy. 2021 ; 133[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.biopha.2020.110948
Artigo de periodico
Ruthenium (II) complex cis-[RuII(ŋ2-O2CC7H7O2)(dppm)2]PF6-hmxbato induces ROS-mediated apoptosis in lung tumor cells producing selective cytotoxicity (2020)
Costa, Mônica Soares; Gonçalves, Yasmim Garcia; Borges, Bruna Cristina; Silva, Marcelo José Barbosa; Amstalden, Martin Krähenbühl; Costa, Tássia Rafaella; Antunes, Lusânia Maria Greggi ; Rodrigues, Renata Santos; Rodrigues, Veridiana de Melo; Franca, Eduardo de Faria; Zóia, Mariana Alves Pereira; Araújo, Thaise Gonçalves de; Goulart, Luiz Ricardo; Von Poelhsitz, Gustavo; Yoneyama, Kelly Aparecida Geraldo
Source: Scientific Reports. Unidade: FCFRP
Subjects: ANTINEOPLÁSICOS, FARMACOLOGIA, APOPTOSE, LINHAGEM CELULAR, PROLIFERAÇÃO CELULAR, DANO AO DNA, LEISHMANIA, METABOLISMO, NEOPLASIAS PULMONARES, QUIMIOTERAPIA, ESPÉCIES REATIVAS DE OXIGÊNIO, RUTÊNIO
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COSTA, Mônica Soares et al. Ruthenium (II) complex cis-[RuII(ŋ2-O2CC7H7O2)(dppm)2]PF6-hmxbato induces ROS-mediated apoptosis in lung tumor cells producing selective cytotoxicity. Scientific Reports, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-72420-w. Acesso em: 17 nov. 2024.
APA
Costa, M. S., Gonçalves, Y. G., Borges, B. C., Silva, M. J. B., Amstalden, M. K., Costa, T. R., et al. (2020). Ruthenium (II) complex cis-[RuII(ŋ2-O2CC7H7O2)(dppm)2]PF6-hmxbato induces ROS-mediated apoptosis in lung tumor cells producing selective cytotoxicity. Scientific Reports, 10. doi:10.1038/s41598-020-72420-w
NLM
Costa MS, Gonçalves YG, Borges BC, Silva MJB, Amstalden MK, Costa TR, Antunes LMG, Rodrigues RS, Rodrigues V de M, Franca E de F, Zóia MAP, Araújo TG de, Goulart LR, Von Poelhsitz G, Yoneyama KAG. Ruthenium (II) complex cis-[RuII(ŋ2-O2CC7H7O2)(dppm)2]PF6-hmxbato induces ROS-mediated apoptosis in lung tumor cells producing selective cytotoxicity [Internet]. Scientific Reports. 2020 ; 10[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41598-020-72420-w
Vancouver
Costa MS, Gonçalves YG, Borges BC, Silva MJB, Amstalden MK, Costa TR, Antunes LMG, Rodrigues RS, Rodrigues V de M, Franca E de F, Zóia MAP, Araújo TG de, Goulart LR, Von Poelhsitz G, Yoneyama KAG. Ruthenium (II) complex cis-[RuII(ŋ2-O2CC7H7O2)(dppm)2]PF6-hmxbato induces ROS-mediated apoptosis in lung tumor cells producing selective cytotoxicity [Internet]. Scientific Reports. 2020 ; 10[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41598-020-72420-w
Artigo de periodico
Transcriptome and DNA methylation changes modulated by sulforaphane induce cell cycle arrest, apoptosis, DNA damage, and suppression of proliferation in human liver cancer cells (2020)
Santos, Patrick Wellington da Silva ; Machado, Ana Rita Thomazela ; De Grandis, Rone Aparecido; Ribeiro, Diego Luis ; Tuttis, Katiuska ; Morselli, Marco; Aissa, Alexandre Ferro ; Pellegrini, Matteo; Antunes, Lusânia Maria Greggi
Source: Food and Chemical Toxicology. Unidades: FCFRP, FMRP
Subjects: APOPTOSE, PROLIFERAÇÃO CELULAR, DANO AO DNA, METILAÇÃO DE DNA, EXPRESSÃO GÊNICA, TRANSDUÇÃO DE SINAL CELULAR
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SANTOS, Patrick Wellington da Silva et al. Transcriptome and DNA methylation changes modulated by sulforaphane induce cell cycle arrest, apoptosis, DNA damage, and suppression of proliferation in human liver cancer cells. Food and Chemical Toxicology, v. 136, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.fct.2019.111047. Acesso em: 17 nov. 2024.
APA
Santos, P. W. da S., Machado, A. R. T., De Grandis, R. A., Ribeiro, D. L., Tuttis, K., Morselli, M., et al. (2020). Transcriptome and DNA methylation changes modulated by sulforaphane induce cell cycle arrest, apoptosis, DNA damage, and suppression of proliferation in human liver cancer cells. Food and Chemical Toxicology, 136. doi:10.1016/j.fct.2019.111047
NLM
Santos PW da S, Machado ART, De Grandis RA, Ribeiro DL, Tuttis K, Morselli M, Aissa AF, Pellegrini M, Antunes LMG. Transcriptome and DNA methylation changes modulated by sulforaphane induce cell cycle arrest, apoptosis, DNA damage, and suppression of proliferation in human liver cancer cells [Internet]. Food and Chemical Toxicology. 2020 ; 136[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.fct.2019.111047
Vancouver
Santos PW da S, Machado ART, De Grandis RA, Ribeiro DL, Tuttis K, Morselli M, Aissa AF, Pellegrini M, Antunes LMG. Transcriptome and DNA methylation changes modulated by sulforaphane induce cell cycle arrest, apoptosis, DNA damage, and suppression of proliferation in human liver cancer cells [Internet]. Food and Chemical Toxicology. 2020 ; 136[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.fct.2019.111047
Artigo de periodico
Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer (2010)
Nagai, Maria Aparecida; Gerhard, Renê; Salaorni, Sibeli; Fregnani, José Humberto Tavares; Nonogaki, Suely; Mourão Netto, Mário; Soares, Fernando Augusto
Source: International journal of oncology. Unidades: FM, FO
Subjects: GENES SUPRESSORES, APOPTOSE, NEOPLASIAS MAMÁRIAS (GENÉTICA), PROGNÓSTICO, IMUNOHISTOQUÍMICA, BIOMARCADORES
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NAGAI, Maria Aparecida et al. Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer. International journal of oncology, v. 37, n. 1, p. 41-49, 2010Tradução . . Disponível em: https://doi.org/10.3892/ijo_00000651. Acesso em: 17 nov. 2024.
APA
Nagai, M. A., Gerhard, R., Salaorni, S., Fregnani, J. H. T., Nonogaki, S., Mourão Netto, M., & Soares, F. A. (2010). Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer. International journal of oncology, 37( 1), 41-49. doi:10.3892/ijo_00000651
NLM
Nagai MA, Gerhard R, Salaorni S, Fregnani JHT, Nonogaki S, Mourão Netto M, Soares FA. Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer [Internet]. International journal of oncology. 2010 ; 37( 1): 41-49.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3892/ijo_00000651
Vancouver
Nagai MA, Gerhard R, Salaorni S, Fregnani JHT, Nonogaki S, Mourão Netto M, Soares FA. Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer [Internet]. International journal of oncology. 2010 ; 37( 1): 41-49.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3892/ijo_00000651
Artigo de periodico
Galectin-1 induces reversible phosphatidylserine exposure at the plasma membrane (2009)
Stowell, Sean R.; Karmakar, Sougata; Arthur, C M.onnie; Ju, Tongzhong; Rodrigues, Lilian Cataldi; Riul, Thalita Bachelli; Baruffi, Marcelo Dias; Miner, Jonathan; McEver, Rodger P.; Cummings, Richard D.
Source: Molecular Biology of the Cell. Unidade: FCFRP
Subjects: MEMBRANA PLASMÁTICA, APOPTOSE, FAGÓCITOS
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ABNT
STOWELL, Sean R. et al. Galectin-1 induces reversible phosphatidylserine exposure at the plasma membrane. Molecular Biology of the Cell, v. 20, n. 5, p. 1408-1418, 2009Tradução . . Acesso em: 17 nov. 2024.
APA
Stowell, S. R., Karmakar, S., Arthur, C. Monnie, Ju, T., Rodrigues, L. C., Riul, T. B., et al. (2009). Galectin-1 induces reversible phosphatidylserine exposure at the plasma membrane. Molecular Biology of the Cell, 20( 5), 1408-1418.
NLM
Stowell SR, Karmakar S, Arthur CMonnie, Ju T, Rodrigues LC, Riul TB, Baruffi MD, Miner J, McEver RP, Cummings RD. Galectin-1 induces reversible phosphatidylserine exposure at the plasma membrane. Molecular Biology of the Cell. 2009 ; 20( 5): 1408-1418.[citado 2024 nov. 17 ]
Vancouver
Stowell SR, Karmakar S, Arthur CMonnie, Ju T, Rodrigues LC, Riul TB, Baruffi MD, Miner J, McEver RP, Cummings RD. Galectin-1 induces reversible phosphatidylserine exposure at the plasma membrane. Molecular Biology of the Cell. 2009 ; 20( 5): 1408-1418.[citado 2024 nov. 17 ]

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