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Artigo de periodico
Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 (2023)
Formiga, Rodrigo de Oliveira; Amaral, Flávia C.; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza ; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Cunha, Fernando de Queiroz
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: COVID-19, LIPOPOLISSACARÍDEOS, METALOPROTEINASES, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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FORMIGA, Rodrigo de Oliveira et al. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, v. 180, n. 11, p. 1460-1481, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16013. Acesso em: 18 nov. 2024.
APA
Formiga, R. de O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2023). Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, 180( 11), 1460-1481. doi:10.1111/bph.16013
NLM
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/bph.16013
Vancouver
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/bph.16013
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 18 nov. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
Hyper-inflammation and complement in COVID-19 (2023)
Silva, Bruno Garcia Peixoto Pires da; Calado, Rodrigo Tocantins
Source: American Journal of Hematology. Unidade: FMRP
Subjects: COVID-19, ATIVAÇÃO DE COMPLEMENTO, INFLAMAÇÃO, NEUTRÓFILOS
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SILVA, Bruno Garcia Peixoto Pires da e CALADO, Rodrigo Tocantins. Hyper-inflammation and complement in COVID-19. American Journal of Hematology, v. 98, p. S74-S81, 2023Tradução . . Disponível em: https://doi.org/10.1002/ajh.26746. Acesso em: 18 nov. 2024.
APA
Silva, B. G. P. P. da, & Calado, R. T. (2023). Hyper-inflammation and complement in COVID-19. American Journal of Hematology, 98, S74-S81. doi:10.1002/ajh.26746
NLM
Silva BGPP da, Calado RT. Hyper-inflammation and complement in COVID-19 [Internet]. American Journal of Hematology. 2023 ; 98 S74-S81.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/ajh.26746
Vancouver
Silva BGPP da, Calado RT. Hyper-inflammation and complement in COVID-19 [Internet]. American Journal of Hematology. 2023 ; 98 S74-S81.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/ajh.26746
Artigo de periodico
Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production (2023)
Córneo, Emily S.; Veras, Flávio Protásio ; Gomes, Giovanni F.; Schneider, Ayda Henriques ; Silva, Bruna Manuella Souza; Almeida, Cícero José Luíz dos Ramos; Silva, Camila M.; Martins, Ronaldo B.; Batah, Sabrina Setembre ; Simon, Carla S.; Prestes, Gabriele da S.; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Fabro, Alexandre Todorovic ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Dal-Pizzol, Felipe
Source: Clinical Immunology. Unidades: FMRP, ESALQ
Subjects: COVID-19, NEUTRÓFILOS, CORONAVIRUS
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ABNT
CÓRNEO, Emily S. et al. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, v. 257, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2023.109836. Acesso em: 18 nov. 2024.
APA
Córneo, E. S., Veras, F. P., Gomes, G. F., Schneider, A. H., Silva, B. M. S., Almeida, C. J. L. dos R., et al. (2023). Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, 257, 1-12. doi:10.1016/j.clim.2023.109836
NLM
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Vancouver
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Artigo de periodico
Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts (2022)
Charlie-Silva, Ives ; Feitosa, Natália M.; Pontes, Leticia Gomes de ; Fernandes, Bianca Helena Ventura ; Nóbrega, Rafael Henrique ; Gomes, Juliana Moreira Mendonça; Prata, Mariana N. L.; Ferraris, Fausto k; Freitas, Anderson Zanardi de ; Ribeiro, Martha Simões ; Cunha, Thiago Mattar
Source: Frontiers in Immunology. Unidades: FM, ICB, IPEN, FMRP
Subjects: PROTEÍNAS, CARRAGENINA, GLICOSAMINOGLICANAS, INFLAMAÇÃO, LEUCÓCITOS, NEUTRÓFILOS, PROTEÔMICA
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ABNT
CHARLIE-SILVA, Ives et al. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts. Frontiers in Immunology, v. 13, p. 1-21, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.1019201. Acesso em: 18 nov. 2024.
APA
Charlie-Silva, I., Feitosa, N. M., Pontes, L. G. de, Fernandes, B. H. V., Nóbrega, R. H., Gomes, J. M. M., et al. (2022). Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts. Frontiers in Immunology, 13, 1-21. doi:10.3389/fimmu.2022.1019201
NLM
Charlie-Silva I, Feitosa NM, Pontes LG de, Fernandes BHV, Nóbrega RH, Gomes JMM, Prata MNL, Ferraris F k, Freitas AZ de, Ribeiro MS, Cunha TM. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts [Internet]. Frontiers in Immunology. 2022 ; 13 1-21.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2022.1019201
Vancouver
Charlie-Silva I, Feitosa NM, Pontes LG de, Fernandes BHV, Nóbrega RH, Gomes JMM, Prata MNL, Ferraris F k, Freitas AZ de, Ribeiro MS, Cunha TM. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts [Internet]. Frontiers in Immunology. 2022 ; 13 1-21.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2022.1019201
Artigo de periodico
Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels (2022)
Freitas, Daniela Fernanda de; morelo, David Fernando Colon ; Silva, Rangel Leal; Santos, Eloá Mangabeira; Guimarães, Victor Hugo Dantas; Ribeiro, Guilherme Henrique Mendes; Paula, Alfredo Maurício Batista de; Guimarães, André Luiz Sena; Reis, Sidnei Tavares dos; Cunha, Fernando de Queiroz ; Antunes, Maísa Mota; Menezes, Gustavo Batista ; Santos, Sérgio Henrique Souza
Source: Molecular Biology Reports. Unidade: FMRP
Subjects: TECIDO ADIPOSO, INFLAMAÇÃO, NEUTRÓFILOS, OBESIDADE
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ABNT
FREITAS, Daniela Fernanda de et al. Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels. Molecular Biology Reports, v. 49, n. 4, p. 3225-3236, 2022Tradução . . Disponível em: https://doi.org/10.1007/s11033-022-07157-y. Acesso em: 18 nov. 2024.
APA
Freitas, D. F. de, morelo, D. F. C., Silva, R. L., Santos, E. M., Guimarães, V. H. D., Ribeiro, G. H. M., et al. (2022). Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels. Molecular Biology Reports, 49( 4), 3225-3236. doi:10.1007/s11033-022-07157-y
NLM
Freitas DF de, morelo DFC, Silva RL, Santos EM, Guimarães VHD, Ribeiro GHM, Paula AMB de, Guimarães ALS, Reis ST dos, Cunha F de Q, Antunes MM, Menezes GB, Santos SHS. Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels [Internet]. Molecular Biology Reports. 2022 ; 49( 4): 3225-3236.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s11033-022-07157-y
Vancouver
Freitas DF de, morelo DFC, Silva RL, Santos EM, Guimarães VHD, Ribeiro GHM, Paula AMB de, Guimarães ALS, Reis ST dos, Cunha F de Q, Antunes MM, Menezes GB, Santos SHS. Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels [Internet]. Molecular Biology Reports. 2022 ; 49( 4): 3225-3236.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s11033-022-07157-y
Artigo de periodico
The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes (2021)
Santos, Renato Elias Rodrigues de Souza ; Silva Júnior, Waldir P. da; Harrison, Simone; Skaar, Eric P.; Chazin, Walter J.; Silva Neto, José Freire da
Source: Infection and Immunity. Unidade: FMRP
Subjects: BIOFILMES, COMPLEXO ANTÍGENO-ANTICORPO, LEUCÓCITOS, NEUTRÓFILOS, VIRULÊNCIA, ZINCO
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ABNT
SANTOS, Renato Elias Rodrigues de Souza et al. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes. Infection and Immunity, v. 89, n. 11, 2021Tradução . . Disponível em: https://doi.org/10.1128/IAI.00311-21. Acesso em: 18 nov. 2024.
APA
Santos, R. E. R. de S., Silva Júnior, W. P. da, Harrison, S., Skaar, E. P., Chazin, W. J., & Silva Neto, J. F. da. (2021). The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes. Infection and Immunity, 89( 11). doi:10.1128/IAI.00311-21
NLM
Santos RER de S, Silva Júnior WP da, Harrison S, Skaar EP, Chazin WJ, Silva Neto JF da. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes [Internet]. Infection and Immunity. 2021 ; 89( 11):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1128/IAI.00311-21
Vancouver
Santos RER de S, Silva Júnior WP da, Harrison S, Skaar EP, Chazin WJ, Silva Neto JF da. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes [Internet]. Infection and Immunity. 2021 ; 89( 11):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1128/IAI.00311-21
Artigo de periodico
The influence of dehydroepiandrosterone on effector functions of neutrophils (2021)
Brauer, Verônica Soares; Zambuzi, Fabiana Albani; Espíndola, Milena Sobral; Cavalcanti Neto, Marinaldo Pacífico; Prado, Morgana Kelly Borges; Silva, Priscilla Mariane Cardoso; Soares, Luana Silva; Lima, Leonardo Judson Galvão de; Leopoldino, Andréia Machado ; Cardoso, Cristina Ribeiro de Barros; Frantz, Fabiani Gai
Source: Brazilian Journal of Pharmaceutical Sciences. Unidade: FCFRP
Subjects: FAGOCITOSE, NEUTRÓFILOS, HORMÔNIOS SEXUAIS, SALMONELLA TYPHIMURIUM
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ABNT
BRAUER, Verônica Soares et al. The influence of dehydroepiandrosterone on effector functions of neutrophils. Brazilian Journal of Pharmaceutical Sciences, v. 57, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1590/s2175-97902020000419139. Acesso em: 18 nov. 2024.
APA
Brauer, V. S., Zambuzi, F. A., Espíndola, M. S., Cavalcanti Neto, M. P., Prado, M. K. B., Silva, P. M. C., et al. (2021). The influence of dehydroepiandrosterone on effector functions of neutrophils. Brazilian Journal of Pharmaceutical Sciences, 57, 1-15. doi:10.1590/s2175-97902020000419139
NLM
Brauer VS, Zambuzi FA, Espíndola MS, Cavalcanti Neto MP, Prado MKB, Silva PMC, Soares LS, Lima LJG de, Leopoldino AM, Cardoso CR de B, Frantz FG. The influence of dehydroepiandrosterone on effector functions of neutrophils [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2021 ; 57 1-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1590/s2175-97902020000419139
Vancouver
Brauer VS, Zambuzi FA, Espíndola MS, Cavalcanti Neto MP, Prado MKB, Silva PMC, Soares LS, Lima LJG de, Leopoldino AM, Cardoso CR de B, Frantz FG. The influence of dehydroepiandrosterone on effector functions of neutrophils [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2021 ; 57 1-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1590/s2175-97902020000419139
Artigo de periodico
SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology (2020)
Veras, Flavio Protasio; Pontelli, Marjorie Cornejo ; Silva, Camila Meirelles de Souza; Toller-Kawahisa, Juliana E.; Lima, Mikhael de; Nascimento, Daniele Carvalho; Schneider, Ayda Henriques; Caetité, Diego; Tavares, Lucas Alves ; Paiva, Isadora M.; Rosales, Roberta Ribeiro Costa; morelo, David Fernando Colon ; Martins, Ronaldo Bragança ; Castro, Ítalo Araújo de ; Almeida, Glaucia Maria ; Lopes, Maria Isabel Fernandes; Benatti, Maíra Nilson; Bonjorno, Letícia Pastorelli; Giannini, Marcela Cavichioli; Luppino-Assad, Rodrigo; Almeida, Sérgio Luna; Vilar, Fernando; Santana, Rodrigo de Carvalho ; Bollela, Valdes Roberto ; Auxiliadora-Martins, Maria ; Borges, Marcos de Carvalho ; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Silva, Luis Lamberti Pinto da ; Cunha, Larissa Dias; Zamboni, Dario Simões ; Dal-Pizzol, Felipe; Leiria, Luiz Osório Silveira ; Siyuan, Li; Batah, Sabrina Setembre ; Fabro, Alexandre Todorovic ; Mauad, Thais ; Dolhnikoff, Marisa ; Duarte Neto, Amaro Nunes ; Saldiva, Paulo Hilário Nascimento ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti; Cunha, Fernando de Queiroz
Source: Journal of Experimental Medicine. Unidades: FMRP, FM
Subjects: COVID-19, CÉLULAS EPITELIAIS, PATOLOGIA, VIROLOGIA, PNEUMONIA, NEUTRÓFILOS, ANGIOTENSINA II
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ABNT
VERAS, Flavio Protasio et al. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology. Journal of Experimental Medicine, v. 217, n. 12, 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20201129. Acesso em: 18 nov. 2024.
APA
Veras, F. P., Pontelli, M. C., Silva, C. M. de S., Toller-Kawahisa, J. E., Lima, M. de, Nascimento, D. C., et al. (2020). SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology. Journal of Experimental Medicine, 217( 12). doi:10.1084/jem.20201129
NLM
Veras FP, Pontelli MC, Silva CM de S, Toller-Kawahisa JE, Lima M de, Nascimento DC, Schneider AH, Caetité D, Tavares LA, Paiva IM, Rosales RRC, morelo DFC, Martins RB, Castro ÍA de, Almeida GM, Lopes MIF, Benatti MN, Bonjorno LP, Giannini MC, Luppino-Assad R, Almeida SL, Vilar F, Santana R de C, Bollela VR, Auxiliadora-Martins M, Borges M de C, Miranda CH, Pazin Filho A, Silva LLP da, Cunha LD, Zamboni DS, Dal-Pizzol F, Leiria LOS, Siyuan L, Batah SS, Fabro AT, Mauad T, Dolhnikoff M, Duarte Neto AN, Saldiva PHN, Cunha TM, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RD, Cunha F de Q. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology [Internet]. Journal of Experimental Medicine. 2020 ; 217( 12):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1084/jem.20201129
Vancouver
Veras FP, Pontelli MC, Silva CM de S, Toller-Kawahisa JE, Lima M de, Nascimento DC, Schneider AH, Caetité D, Tavares LA, Paiva IM, Rosales RRC, morelo DFC, Martins RB, Castro ÍA de, Almeida GM, Lopes MIF, Benatti MN, Bonjorno LP, Giannini MC, Luppino-Assad R, Almeida SL, Vilar F, Santana R de C, Bollela VR, Auxiliadora-Martins M, Borges M de C, Miranda CH, Pazin Filho A, Silva LLP da, Cunha LD, Zamboni DS, Dal-Pizzol F, Leiria LOS, Siyuan L, Batah SS, Fabro AT, Mauad T, Dolhnikoff M, Duarte Neto AN, Saldiva PHN, Cunha TM, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RD, Cunha F de Q. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology [Internet]. Journal of Experimental Medicine. 2020 ; 217( 12):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1084/jem.20201129
Artigo de periodico
Neutrophils and COVID-19:: what is going on? (2020)
Andrade, Micássio Fernandes de ; Marzocchi-Machado, Cleni Mara
Source: Critical Reviews in Immunology. Unidade: FCFRP
Subjects: COVID-19, METABOLISMO, IMUNOLOGIA, NEUTRÓFILOS, FISIOLOGIA
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ABNT
ANDRADE, Micássio Fernandes de e MARZOCCHI-MACHADO, Cleni Mara. Neutrophils and COVID-19:: what is going on?. Critical Reviews in Immunology, v. 40, n. 6, p. 527-536, 2020Tradução . . Disponível em: https://doi.org/10.1615/CritRevImmunol.2020035900. Acesso em: 18 nov. 2024.
APA
Andrade, M. F. de, & Marzocchi-Machado, C. M. (2020). Neutrophils and COVID-19:: what is going on? Critical Reviews in Immunology, 40( 6), 527-536. doi:10.1615/CritRevImmunol.2020035900
NLM
Andrade MF de, Marzocchi-Machado CM. Neutrophils and COVID-19:: what is going on? [Internet]. Critical Reviews in Immunology. 2020 ; 40( 6): 527-536.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1615/CritRevImmunol.2020035900
Vancouver
Andrade MF de, Marzocchi-Machado CM. Neutrophils and COVID-19:: what is going on? [Internet]. Critical Reviews in Immunology. 2020 ; 40( 6): 527-536.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1615/CritRevImmunol.2020035900
Artigo de periodico
Neutrophil Extracellular Traps Effectively Control Acute Chikungunya Virus Infection (2020)
Hiroki, Carlos Hiroji; Toller-kawahisa, Juliana Escher; Fumagalli, Marcilio Jorge ; morelo, David Fernando Colon ; Figueiredo, Luiz Tadeu Moraes ; Fonseca, Benedito Antonio Lopes da ; Franca, Rafael F. O.; Cunha, Fernando de Queiroz
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, VÍRUS
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ABNT
HIROKI, Carlos Hiroji et al. Neutrophil Extracellular Traps Effectively Control Acute Chikungunya Virus Infection. Frontiers in Immunology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.03108. Acesso em: 18 nov. 2024.
APA
Hiroki, C. H., Toller-kawahisa, J. E., Fumagalli, M. J., morelo, D. F. C., Figueiredo, L. T. M., Fonseca, B. A. L. da, et al. (2020). Neutrophil Extracellular Traps Effectively Control Acute Chikungunya Virus Infection. Frontiers in Immunology, 10. doi:10.3389/fimmu.2019.03108
NLM
Hiroki CH, Toller-kawahisa JE, Fumagalli MJ, morelo DFC, Figueiredo LTM, Fonseca BAL da, Franca RFO, Cunha F de Q. Neutrophil Extracellular Traps Effectively Control Acute Chikungunya Virus Infection [Internet]. Frontiers in Immunology. 2020 ; 10[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2019.03108
Vancouver
Hiroki CH, Toller-kawahisa JE, Fumagalli MJ, morelo DFC, Figueiredo LTM, Fonseca BAL da, Franca RFO, Cunha F de Q. Neutrophil Extracellular Traps Effectively Control Acute Chikungunya Virus Infection [Internet]. Frontiers in Immunology. 2020 ; 10[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2019.03108
Artigo de periodico
Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection (2020)
Malacco, Nathalia Luisa Sousa de Oliveira; Souza, Jessica Amanda Marques; Martins, Flavia Rayssa Braga; Rachid, Milene Alvarenga; Simplicio, Janaina Aparecida; Tirapelli, Carlos Renato ; Sabino, Adriano de Paula; Queiroz-Junior, Celso Martins; Goes, Grazielle Ribeiro; Vieira, Leda Quercia; Souza, Danielle Glória; Pinho, Vanessa; Teixeira, Mauro Martins; Soriani, Frederico Marianetti
Source: eLife. Unidade: EERP
Subjects: ASPERGILLUS, ETANOL, CONSUMO, IMUNOLOGIA, DOENÇAS INFECCIOSAS, INFLAMAÇÃO, MICROBIOLOGIA, NEUTRÓFILOS
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ABNT
MALACCO, Nathalia Luisa Sousa de Oliveira et al. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection. eLife, v. 9, 2020Tradução . . Disponível em: https://doi.org/10.7554/eLife.58855. Acesso em: 18 nov. 2024.
APA
Malacco, N. L. S. de O., Souza, J. A. M., Martins, F. R. B., Rachid, M. A., Simplicio, J. A., Tirapelli, C. R., et al. (2020). Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection. eLife, 9. doi:10.7554/eLife.58855
NLM
Malacco NLS de O, Souza JAM, Martins FRB, Rachid MA, Simplicio JA, Tirapelli CR, Sabino A de P, Queiroz-Junior CM, Goes GR, Vieira LQ, Souza DG, Pinho V, Teixeira MM, Soriani FM. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection [Internet]. eLife. 2020 ; 9[citado 2024 nov. 18 ] Available from: https://doi.org/10.7554/eLife.58855
Vancouver
Malacco NLS de O, Souza JAM, Martins FRB, Rachid MA, Simplicio JA, Tirapelli CR, Sabino A de P, Queiroz-Junior CM, Goes GR, Vieira LQ, Souza DG, Pinho V, Teixeira MM, Soriani FM. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection [Internet]. eLife. 2020 ; 9[citado 2024 nov. 18 ] Available from: https://doi.org/10.7554/eLife.58855
Artigo de periodico
Incorporation of Baccharis dracunculifolia DC (Asteraceae) leaf extract into phosphatidylcholine-cholesterol liposomes improves its anti-inflammatory effect in vivo (2019)
Figueiredo-Rinhel, Andréa Silva Garcia de; Andrade, Micássio Fernandes de ; Landi-Librandi, Ana Paula; Azzolini, Ana Elisa Caleiro Seixas; Kabeya, Luciana Mariko ; Bastos, Jairo Kenupp ; Valim, Yara Maria Lucisano
Source: Natural Product Research. Unidades: FCFRP, FMRP
Subjects: ANTI-INFLAMATÓRIOS, ARTRITE, NEUTRÓFILOS, COMPOSITAE, PLANTAS MEDICINAIS
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ABNT
FIGUEIREDO-RINHEL, Andréa Silva Garcia de et al. Incorporation of Baccharis dracunculifolia DC (Asteraceae) leaf extract into phosphatidylcholine-cholesterol liposomes improves its anti-inflammatory effect in vivo. Natural Product Research, v. 33, n. 17, p. 2521-2525, 2019Tradução . . Disponível em: https://doi.org/10.1080/14786419.2018.1448809. Acesso em: 18 nov. 2024.
APA
Figueiredo-Rinhel, A. S. G. de, Andrade, M. F. de, Landi-Librandi, A. P., Azzolini, A. E. C. S., Kabeya, L. M., Bastos, J. K., & Valim, Y. M. L. (2019). Incorporation of Baccharis dracunculifolia DC (Asteraceae) leaf extract into phosphatidylcholine-cholesterol liposomes improves its anti-inflammatory effect in vivo. Natural Product Research, 33( 17), 2521-2525. doi:10.1080/14786419.2018.1448809
NLM
Figueiredo-Rinhel ASG de, Andrade MF de, Landi-Librandi AP, Azzolini AECS, Kabeya LM, Bastos JK, Valim YML. Incorporation of Baccharis dracunculifolia DC (Asteraceae) leaf extract into phosphatidylcholine-cholesterol liposomes improves its anti-inflammatory effect in vivo [Internet]. Natural Product Research. 2019 ; 33( 17): 2521-2525.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/14786419.2018.1448809
Vancouver
Figueiredo-Rinhel ASG de, Andrade MF de, Landi-Librandi AP, Azzolini AECS, Kabeya LM, Bastos JK, Valim YML. Incorporation of Baccharis dracunculifolia DC (Asteraceae) leaf extract into phosphatidylcholine-cholesterol liposomes improves its anti-inflammatory effect in vivo [Internet]. Natural Product Research. 2019 ; 33( 17): 2521-2525.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/14786419.2018.1448809
Artigo de periodico
Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus (2018)
Ferreira, Raphael Gomes; Rodrigues, Lilian Cataldi ; Nascimento, Daniele Carvalho Bernardo; Kanashiro, Alexandre ; Melo, Paulo Henrique de; Borges, Vanessa de Fátima; Gozzi, Aline ; Prado, Douglas da Silva ; Borges, Marcos de Carvalho; Ramalho, Fernando Silva ; Stowell, Sean R.; Cummings, Richard D.; Dias-Baruffi, Marcelo ; Cunha, Fernando de Queiroz ; Alves-Filho, José Carlos Farias
Source: Journal of Infection. Unidades: FCFRP, FMRP
Subjects: SEPSE, NEUTRÓFILOS, INFLAMAÇÃO
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ABNT
FERREIRA, Raphael Gomes et al. Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus. Journal of Infection, v. 77, n. 5, p. 391-397, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jinf.2018.06.010. Acesso em: 18 nov. 2024.
APA
Ferreira, R. G., Rodrigues, L. C., Nascimento, D. C. B., Kanashiro, A., Melo, P. H. de, Borges, V. de F., et al. (2018). Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus. Journal of Infection, 77( 5), 391-397. doi:10.1016/j.jinf.2018.06.010
NLM
Ferreira RG, Rodrigues LC, Nascimento DCB, Kanashiro A, Melo PH de, Borges V de F, Gozzi A, Prado D da S, Borges M de C, Ramalho FS, Stowell SR, Cummings RD, Dias-Baruffi M, Cunha F de Q, Alves-Filho JCF. Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus [Internet]. Journal of Infection. 2018 ; 77( 5): 391-397.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jinf.2018.06.010
Vancouver
Ferreira RG, Rodrigues LC, Nascimento DCB, Kanashiro A, Melo PH de, Borges V de F, Gozzi A, Prado D da S, Borges M de C, Ramalho FS, Stowell SR, Cummings RD, Dias-Baruffi M, Cunha F de Q, Alves-Filho JCF. Galectin-3 aggravates experimental polymicrobial sepsis by impairing neutrophil recruitment to the infectious focus [Internet]. Journal of Infection. 2018 ; 77( 5): 391-397.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jinf.2018.06.010
Artigo de periodico
The 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3′,4′-methylenedioxyphenyl]-coumarin downmodulates the FcγR- and CR-mediated oxidative metabolism and elastase release in human neutrophils: possible mechanisms underlying inhibition of the formation and release of neutrophil extracellular traps (2018)
Andrade, Micássio F.; Kabeya, Luciana M.; Bortot, Leandro O.; Santos, Gabriela B. dos; Santos, Everton O. L.; Albiero, Lucinéia R.; Figueiredo-Rinhel, Andréa S. G.; Carvalho, Camila A.; Azzolini, Ana Elisa Caleiro Seixas; Caliri, Antônio; Pupo, Mônica Tallarico; Emery, Flavio da Silva; Lucisano-Valim, Yara Maria
Source: Free Radical Biology and Medicine. Unidade: FCFRP
Subjects: ANTI-INFLAMATÓRIOS, CÉLULAS CULTIVADAS, NEUTRÓFILOS, ESTRESSE OXIDATIVO, PEROXIDASE, FAGOCITOSE, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDRADE, Micássio F. et al. The 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3′,4′-methylenedioxyphenyl]-coumarin downmodulates the FcγR- and CR-mediated oxidative metabolism and elastase release in human neutrophils: possible mechanisms underlying inhibition of the formation and release of neutrophil extracellular traps. Free Radical Biology and Medicine, v. 115, p. 421-435, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2017.12.012. Acesso em: 18 nov. 2024.
APA
Andrade, M. F., Kabeya, L. M., Bortot, L. O., Santos, G. B. dos, Santos, E. O. L., Albiero, L. R., et al. (2018). The 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3′,4′-methylenedioxyphenyl]-coumarin downmodulates the FcγR- and CR-mediated oxidative metabolism and elastase release in human neutrophils: possible mechanisms underlying inhibition of the formation and release of neutrophil extracellular traps. Free Radical Biology and Medicine, 115, 421-435. doi:10.1016/j.freeradbiomed.2017.12.012
NLM
Andrade MF, Kabeya LM, Bortot LO, Santos GB dos, Santos EOL, Albiero LR, Figueiredo-Rinhel ASG, Carvalho CA, Azzolini AECS, Caliri A, Pupo MT, Emery F da S, Lucisano-Valim YM. The 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3′,4′-methylenedioxyphenyl]-coumarin downmodulates the FcγR- and CR-mediated oxidative metabolism and elastase release in human neutrophils: possible mechanisms underlying inhibition of the formation and release of neutrophil extracellular traps [Internet]. Free Radical Biology and Medicine. 2018 ; 115 421-435.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2017.12.012
Vancouver
Andrade MF, Kabeya LM, Bortot LO, Santos GB dos, Santos EOL, Albiero LR, Figueiredo-Rinhel ASG, Carvalho CA, Azzolini AECS, Caliri A, Pupo MT, Emery F da S, Lucisano-Valim YM. The 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3′,4′-methylenedioxyphenyl]-coumarin downmodulates the FcγR- and CR-mediated oxidative metabolism and elastase release in human neutrophils: possible mechanisms underlying inhibition of the formation and release of neutrophil extracellular traps [Internet]. Free Radical Biology and Medicine. 2018 ; 115 421-435.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2017.12.012
Artigo de periodico
Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation (2018)
Kanashiro, Alexandre; Franchin, Marcelo; Bassi, Gabriel Shimizu; Santana, Dênis Augusto Reis; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Ulloa, Luis; Rodrigues, Gerson Jonathan
Source: Fundamental and Clinical Pharmacology. Unidade: FMRP
Subjects: ARTRITE, RECEPTORES ANDROGÊNICOS, SISTEMA NERVOSO PARASSIMPÁTICO, NEUTRÓFILOS
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ABNT
KANASHIRO, Alexandre et al. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, v. 32, n. 2, p. 155-162, 2018Tradução . . Disponível em: https://doi.org/10.1111/fcp.12338. Acesso em: 18 nov. 2024.
APA
Kanashiro, A., Franchin, M., Bassi, G. S., Santana, D. A. R., Cunha, T. M., Cunha, F. de Q., et al. (2018). Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, 32( 2), 155-162. doi:10.1111/fcp.12338
NLM
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/fcp.12338
Vancouver
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/fcp.12338
Artigo de periodico
Effect of ArtinM on human blood cells during infection with paracoccidioides brasiliensis (2018)
Ruas, Luciana P.; Genaro, Livia M.; Justo-Junior, Amauri S.; Coser, Lilian O.; Castro, Lívia F. de; Trabasso, Plinio; Mamoni, Ronei L.; Roque-Barreira, Maria Cristina; Blotta, Maria-Heloisa S. L.
Source: Frontiers in Microbiology. Unidade: FMRP
Subjects: PARACOCCIDIOIDES, MICOSES, LECTINAS, NEUTRÓFILOS, PARACOCCIDIOIDOMICOSE
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ABNT
RUAS, Luciana P. et al. Effect of ArtinM on human blood cells during infection with paracoccidioides brasiliensis. Frontiers in Microbiology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2018.00867. Acesso em: 18 nov. 2024.
APA
Ruas, L. P., Genaro, L. M., Justo-Junior, A. S., Coser, L. O., Castro, L. F. de, Trabasso, P., et al. (2018). Effect of ArtinM on human blood cells during infection with paracoccidioides brasiliensis. Frontiers in Microbiology, 9. doi:10.3389/fmicb.2018.00867
NLM
Ruas LP, Genaro LM, Justo-Junior AS, Coser LO, Castro LF de, Trabasso P, Mamoni RL, Roque-Barreira MC, Blotta M-HSL. Effect of ArtinM on human blood cells during infection with paracoccidioides brasiliensis [Internet]. Frontiers in Microbiology. 2018 ; 9[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fmicb.2018.00867
Vancouver
Ruas LP, Genaro LM, Justo-Junior AS, Coser LO, Castro LF de, Trabasso P, Mamoni RL, Roque-Barreira MC, Blotta M-HSL. Effect of ArtinM on human blood cells during infection with paracoccidioides brasiliensis [Internet]. Frontiers in Microbiology. 2018 ; 9[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fmicb.2018.00867
Artigo de periodico
Activation status of peripheral blood neutrophils and the complement system in adult rheumatoid arthritis patients undergoing combined therapy with infliximab and methotrexate (2018)
Marchi, Larissa F. ; Paschoalato, Adriana Balbina Paoliello ; Oliveira, Renê Donizete Ribeiro de; Azzolini, Ana Elisa Caleiro Seixas ; Kabeya, Luciana Mariko ; Donadi, Eduardo Antônio ; Lucisano-Valim, Yara Maria
Source: Rheumatology International. Unidades: FCFRP, FMRP
Subjects: METOTREXATO, NEUTRÓFILOS, ARTRITE REUMATOIDE, ANTICORPOS MONOCLONAIS
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ABNT
MARCHI, Larissa F. et al. Activation status of peripheral blood neutrophils and the complement system in adult rheumatoid arthritis patients undergoing combined therapy with infliximab and methotrexate. Rheumatology International, v. 38, n. 6, p. 1043-1052, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00296-018-3997-1. Acesso em: 18 nov. 2024.
APA
Marchi, L. F., Paschoalato, A. B. P., Oliveira, R. D. R. de, Azzolini, A. E. C. S., Kabeya, L. M., Donadi, E. A., & Lucisano-Valim, Y. M. (2018). Activation status of peripheral blood neutrophils and the complement system in adult rheumatoid arthritis patients undergoing combined therapy with infliximab and methotrexate. Rheumatology International, 38( 6), 1043-1052. doi:10.1007/s00296-018-3997-1
NLM
Marchi LF, Paschoalato ABP, Oliveira RDR de, Azzolini AECS, Kabeya LM, Donadi EA, Lucisano-Valim YM. Activation status of peripheral blood neutrophils and the complement system in adult rheumatoid arthritis patients undergoing combined therapy with infliximab and methotrexate [Internet]. Rheumatology International. 2018 ; 38( 6): 1043-1052.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00296-018-3997-1
Vancouver
Marchi LF, Paschoalato ABP, Oliveira RDR de, Azzolini AECS, Kabeya LM, Donadi EA, Lucisano-Valim YM. Activation status of peripheral blood neutrophils and the complement system in adult rheumatoid arthritis patients undergoing combined therapy with infliximab and methotrexate [Internet]. Rheumatology International. 2018 ; 38( 6): 1043-1052.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00296-018-3997-1
Artigo de periodico
The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 (2018)
Sônego, Fabiane; Castanheira, Fernanda V. S.; Horta, Catarina V.; Kanashiro, Alexandre; Czaikoski, Paula G.; Zamboni, Dario Simões; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: PNEUMONIA, NEUTRÓFILOS, QUIMIOCINAS, BACTÉRIAS GRAM-NEGATIVAS
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ABNT
SÔNEGO, Fabiane et al. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88. Inflammation Research, v. 67, n. 5, p. 435-443, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-018-1135-x. Acesso em: 18 nov. 2024.
APA
Sônego, F., Castanheira, F. V. S., Horta, C. V., Kanashiro, A., Czaikoski, P. G., Zamboni, D. S., et al. (2018). The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88. Inflammation Research, 67( 5), 435-443. doi:10.1007/s00011-018-1135-x
NLM
Sônego F, Castanheira FVS, Horta CV, Kanashiro A, Czaikoski PG, Zamboni DS, Alves Filho JCF, Cunha F de Q. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 [Internet]. Inflammation Research. 2018 ; 67( 5): 435-443.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00011-018-1135-x
Vancouver
Sônego F, Castanheira FVS, Horta CV, Kanashiro A, Czaikoski PG, Zamboni DS, Alves Filho JCF, Cunha F de Q. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 [Internet]. Inflammation Research. 2018 ; 67( 5): 435-443.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00011-018-1135-x
Artigo de periodico
Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection (2017)
Sacramento, Laís A.; Costa, Jéssica L. da; Lima, Mikhael H. F. de; Sampaio, Pedro A.; Almeida, Roque P.; Cunha, Fernando Queiróz; Silva, João Santana da; Carregaro, Vanessa
Source: Frontiers in Microbiology. Unidade: FMRP
Subjects: LEISHMANIA INFANTUM, CÉLULAS DENDRÍTICAS, LEISHMANIOSE VISCERAL, NEUTRÓFILOS
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ABNT
SACRAMENTO, Laís A. et al. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.00262. Acesso em: 18 nov. 2024.
APA
Sacramento, L. A., Costa, J. L. da, Lima, M. H. F. de, Sampaio, P. A., Almeida, R. P., Cunha, F. Q., et al. (2017). Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, 8. doi:10.3389/fmicb.2017.00262
NLM
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Vancouver
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fmicb.2017.00262

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