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Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Subjects: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
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SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 04 nov. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice (2018)
Neves, Karla Bianca; Montezano, Augusto Cesar; Alves-Lopes, Rheure; Bruder-Nascimento, Thiago; Costa, Rafael Menezes; Costa, Roberto Silva; Touyz, Rhian M.; Passaglia, Rita de Cássia Aleixo Tostes
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: RIM, DIABETES MELLITUS, ESTRESSE OXIDATIVO, RATOS
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NEVES, Karla Bianca et al. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice. International Journal of Molecular Sciences, v. 19, n. 8, 2018Tradução . . Disponível em: https://doi.org/10.3390/ijms19082454. Acesso em: 04 nov. 2024.
APA
Neves, K. B., Montezano, A. C., Alves-Lopes, R., Bruder-Nascimento, T., Costa, R. M., Costa, R. S., et al. (2018). Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice. International Journal of Molecular Sciences, 19( 8). doi:10.3390/ijms19082454
NLM
Neves KB, Montezano AC, Alves-Lopes R, Bruder-Nascimento T, Costa RM, Costa RS, Touyz RM, Passaglia R de CAT. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 8):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/ijms19082454
Vancouver
Neves KB, Montezano AC, Alves-Lopes R, Bruder-Nascimento T, Costa RM, Costa RS, Touyz RM, Passaglia R de CAT. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 8):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/ijms19082454
Artigo de periodico
Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion (2018)
Omoto, Ana Carolina Mieko; Gava, Fábio Nelson; Silva, Carlos Alberto Aguiar da; Silva, Hadder Batista; Parente, Juliana Montenegro; Costa, Rafael Menezes; Castro, Michele Mazzaron de; Passaglia, Rita de Cássia Aleixo Tostes; Salgado, Hélio César; Fazan Júnior, Rubens
Source: PLOS ONE. Unidade: FMRP
Subjects: REPERFUSÃO, PROCEDIMENTOS CIRÚRGICOS CARDÍACOS, RATOS
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OMOTO, Ana Carolina Mieko et al. Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion. PLOS ONE, v. 13, n. 12, p. e0209190, 2018Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0209190. Acesso em: 04 nov. 2024.
APA
Omoto, A. C. M., Gava, F. N., Silva, C. A. A. da, Silva, H. B., Parente, J. M., Costa, R. M., et al. (2018). Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion. PLOS ONE, 13( 12), e0209190. doi:10.1371/journal.pone.0209190
NLM
Omoto ACM, Gava FN, Silva CAA da, Silva HB, Parente JM, Costa RM, Castro MM de, Passaglia R de CAT, Salgado HC, Fazan Júnior R. Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion [Internet]. PLOS ONE. 2018 ; 13( 12): e0209190.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1371/journal.pone.0209190
Vancouver
Omoto ACM, Gava FN, Silva CAA da, Silva HB, Parente JM, Costa RM, Castro MM de, Passaglia R de CAT, Salgado HC, Fazan Júnior R. Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion [Internet]. PLOS ONE. 2018 ; 13( 12): e0209190.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1371/journal.pone.0209190
Artigo de periodico
Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases (2016)
Gohar, Eman Y; Giachini, Fernanda R; Pollock, David M; Tostes, Rita de Cássia Aleixo
Source: Life Sciences. Unidade: FMRP
Subjects: DOENÇAS CARDIOVASCULARES, NEFROPATIAS, ENDOTELINAS, DIFERENÇAS SEXUAIS (PSICOGÊNESE), CROMOSSOMOS SEXUAIS
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GOHAR, Eman Y et al. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life Sciences, v. 159, p. 20-29, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.02.093. Acesso em: 04 nov. 2024.
APA
Gohar, E. Y., Giachini, F. R., Pollock, D. M., & Tostes, R. de C. A. (2016). Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life Sciences, 159, 20-29. doi:10.1016/j.lfs.2016.02.093
NLM
Gohar EY, Giachini FR, Pollock DM, Tostes R de CA. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases [Internet]. Life Sciences. 2016 ; 159 20-29.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.093
Vancouver
Gohar EY, Giachini FR, Pollock DM, Tostes R de CA. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases [Internet]. Life Sciences. 2016 ; 159 20-29.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.093
Artigo de periodico
Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery (2016)
Rodrigues, Fernanda Luciano; Lopes, Rhéure Alves M.; Fais, Rafael Sobrano; Oliveira, Lucas de; Prado, Cibele M.; Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva
Source: Life Sciences. Unidade: FMRP
Subjects: DISFUNÇÃO ERÉTIL, PÊNIS, INSUFICIÊNCIA CARDÍACA, ARTÉRIAS, TRANSDUÇÃO DE SINAL CELULAR
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RODRIGUES, Fernanda Luciano et al. Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery. Life Sciences, v. 145, p. 9-18, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.12.005. Acesso em: 04 nov. 2024.
APA
Rodrigues, F. L., Lopes, R. A. M., Fais, R. S., Oliveira, L. de, Prado, C. M., Tostes, R. de C. A., & Carneiro, F. S. (2016). Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery. Life Sciences, 145, 9-18. doi:10.1016/j.lfs.2015.12.005
NLM
Rodrigues FL, Lopes RAM, Fais RS, Oliveira L de, Prado CM, Tostes R de CA, Carneiro FS. Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery [Internet]. Life Sciences. 2016 ; 145 9-18.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.005
Vancouver
Rodrigues FL, Lopes RAM, Fais RS, Oliveira L de, Prado CM, Tostes R de CA, Carneiro FS. Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery [Internet]. Life Sciences. 2016 ; 145 9-18.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.005
Artigo de periodico
Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice (2016)
Lima, Victor V; Zemse, Saiprasad M; Chiao, Chin-Wei; Bomfim, Gisele F; Tostes, Rita de Cássia Aleixo; Webb, R. Clinton; Giachini, Fernanda R
Source: Life Sciences. Unidade: FMRP
Subjects: HIPERTENSÃO, INFLAMAÇÃO, INTERLEUCINA-10, ANGIOTENSINA II, AORTA
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ABNT
LIMA, Victor V et al. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice. Life Sciences, v. 145, p. 137-143, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.12.009. Acesso em: 04 nov. 2024.
APA
Lima, V. V., Zemse, S. M., Chiao, C. -W., Bomfim, G. F., Tostes, R. de C. A., Webb, R. C., & Giachini, F. R. (2016). Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice. Life Sciences, 145, 137-143. doi:10.1016/j.lfs.2015.12.009
NLM
Lima VV, Zemse SM, Chiao C-W, Bomfim GF, Tostes R de CA, Webb RC, Giachini FR. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice [Internet]. Life Sciences. 2016 ; 145 137-143.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.009
Vancouver
Lima VV, Zemse SM, Chiao C-W, Bomfim GF, Tostes R de CA, Webb RC, Giachini FR. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice [Internet]. Life Sciences. 2016 ; 145 137-143.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2015.12.009
Artigo de periodico
Reactive oxygen species: players in the cardiovascular effects of testosterone (2016)
Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Carvalho, Maria Helena Catelli de; Reckelhoff, Jane F.
Source: American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. Unidades: FMRP, ICB
Subjects: DOENÇAS CARDIOVASCULARES, SISTEMA CARDIOVASCULAR, TERAPIA DA REPOSIÇÃO HORMONAL, ESTRESSE OXIDATIVO, TESTOSTERONA
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ABNT
TOSTES, Rita de Cássia Aleixo et al. Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, v. 310, n. 1, p. R1-R14, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00392.2014. Acesso em: 04 nov. 2024.
APA
Tostes, R. de C. A., Carneiro, F. S., Carvalho, M. H. C. de, & Reckelhoff, J. F. (2016). Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, 310( 1), R1-R14. doi:10.1152/ajpregu.00392.2014
NLM
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Vancouver
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Artigo de periodico
Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways (2015)
Bruder-Nascimento, Thiago; Callera, Glaucia E; Montezano, Augusto C; He, Ying; Antunes, Tayze T; Cat, Aurelie Nguyen Dinh; Tostes, Rita de Cássia Aleixo; Touyz, Rhian M
Source: Clinical Science. Unidade: FMRP
Subjects: DIABETES MELLITUS (FISIOPATOLOGIA), LEPTINA, VASODILATAÇÃO (FARMACOLOGIA), PROTEÍNAS QUINASES, ARTÉRIAS (EFEITOS DE DROGAS)
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BRUDER-NASCIMENTO, Thiago et al. Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways. Clinical Science, v. 128, n. 7, p. 411-423, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140456. Acesso em: 04 nov. 2024.
APA
Bruder-Nascimento, T., Callera, G. E., Montezano, A. C., He, Y., Antunes, T. T., Cat, A. N. D., et al. (2015). Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways. Clinical Science, 128( 7), 411-423. doi:10.1042/CS20140456
NLM
Bruder-Nascimento T, Callera GE, Montezano AC, He Y, Antunes TT, Cat AND, Tostes R de CA, Touyz RM. Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways [Internet]. Clinical Science. 2015 ; 128( 7): 411-423.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1042/CS20140456
Vancouver
Bruder-Nascimento T, Callera GE, Montezano AC, He Y, Antunes TT, Cat AND, Tostes R de CA, Touyz RM. Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways [Internet]. Clinical Science. 2015 ; 128( 7): 411-423.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1042/CS20140456
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidades: FMRP, FM, ICB
Subjects: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
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CHIGNALIA, Andreia Z et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, v. 129, n. 1, p. 39-48, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140548. Acesso em: 04 nov. 2024.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1042/CS20140548
Artigo de periodico
Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice (2011)
Choi, Hyehun; Passaglia, Rita de Cassia Aleixo Tostes; Webb, R. Clinton
Source: Journal of the American Society of Hypertension. Unidade: FMRP
Subjects: HIPERTENSÃO, ALDEÍDOS
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ABNT
CHOI, Hyehun e PASSAGLIA, Rita de Cassia Aleixo Tostes e WEBB, R. Clinton. Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice. Journal of the American Society of Hypertension, v. 5, n. 3, p. 154-160, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.jash.2011.02.005. Acesso em: 04 nov. 2024.
APA
Choi, H., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice. Journal of the American Society of Hypertension, 5( 3), 154-160. doi:10.1016/j.jash.2011.02.005
NLM
Choi H, Passaglia R de CAT, Webb RC. Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice [Internet]. Journal of the American Society of Hypertension. 2011 ; 5( 3): 154-160.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jash.2011.02.005
Vancouver
Choi H, Passaglia R de CAT, Webb RC. Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice [Internet]. Journal of the American Society of Hypertension. 2011 ; 5( 3): 154-160.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jash.2011.02.005
Artigo de periodico
Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR (2011)
Callera, Gláucia Elena; Yogi, Álvaro; Briones, Ana Maria; Montezano, Augusto Cesar Inforzato; He, Ying; Passaglia, Rita de Cassia Aleixo Tostes; Schiffrin, Ernesto L; Touyz, Rhian M.
Source: Cardiovascular Research. Unidade: FMRP
Subjects: ALDOSTERONA, CÉLULAS MUSCULARES
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ABNT
CALLERA, Gláucia Elena et al. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research, v. 91, n. 4, p. 720-731, 2011Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvr131. Acesso em: 04 nov. 2024.
APA
Callera, G. E., Yogi, Á., Briones, A. M., Montezano, A. C. I., He, Y., Passaglia, R. de C. A. T., et al. (2011). Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research, 91( 4), 720-731. doi:10.1093/cvr/cvr131
NLM
Callera GE, Yogi Á, Briones AM, Montezano ACI, He Y, Passaglia R de CAT, Schiffrin EL, Touyz RM. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR [Internet]. Cardiovascular Research. 2011 ; 91( 4): 720-731.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/cvr/cvr131
Vancouver
Callera GE, Yogi Á, Briones AM, Montezano ACI, He Y, Passaglia R de CAT, Schiffrin EL, Touyz RM. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR [Internet]. Cardiovascular Research. 2011 ; 91( 4): 720-731.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/cvr/cvr131
Artigo de periodico
Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 (2011)
Giachini, Fernanda R.; Lima, Victor V.; Carneiro, Fernando S.; Passaglia, Rita de Cassia Aleixo Tostes; Webb, R. Clinton
Source: Hypertension. Unidade: FMRP
Subjects: ANGIOTENSINA II, HIPERTENSÃO
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ABNT
GIACHINI, Fernanda R. et al. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, v. 57, n. 3, p. 655-663, 2011Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327. Acesso em: 04 nov. 2024.
APA
Giachini, F. R., Lima, V. V., Carneiro, F. S., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, 57( 3), 655-663. doi:10.1161/HYPERTENSIONAHA.110.164327
NLM
Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
Vancouver
Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
Artigo de periodico
Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats (2011)
Matsumoto, Takayuri; Passaglia, Rita de Cassia Aleixo Tostes; Webb, R. Clinton
Source: American Journal of Physiology: Heart and Circulatory Physiology. Unidade: FMRP
Subjects: HIPERTENSÃO, ENDOTÉLIO, ANTAGONISTAS
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ABNT
MATSUMOTO, Takayuri e PASSAGLIA, Rita de Cassia Aleixo Tostes e WEBB, R. Clinton. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology, v. 301, n. 2, p. H409-H417, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00084.2011. Acesso em: 04 nov. 2024.
APA
Matsumoto, T., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology, 301( 2), H409-H417. doi:10.1152/ajpheart.00084.2011
NLM
Matsumoto T, Passaglia R de CAT, Webb RC. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2011 ; 301( 2): H409-H417.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpheart.00084.2011
Vancouver
Matsumoto T, Passaglia R de CAT, Webb RC. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2011 ; 301( 2): H409-H417.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpheart.00084.2011
Artigo de periodico
Increased endothelin-1 reactivity and endothelial dysfunction in carotid arteries from rats with hyperhomocysteinemia (2009)
Andrade, Cláudia Roberta de; Leite, P. F.; Montezano, Augusto C.; Casolari, Débora Arcieri; Yogi, Alvaro; Passaglia, Rita de Cássia Aleixo Tostes; Haddad, Renato; Eberlin, Marcos Nogueira; Laurindo, Francisco Rafael Martins; Souza, Heraldo Possolo de; Corrêa, Fernando Morgan de Aguiar; Oliveira, Ana Maria de
Source: British Journal of Pharmacology. Unidades: FM, FMRP, FCFRP, ICB
Subjects: ARTÉRIAS CARÓTIDAS, ENDOTÉLIO, FARMACOLOGIA
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ABNT
ANDRADE, Cláudia Roberta de et al. Increased endothelin-1 reactivity and endothelial dysfunction in carotid arteries from rats with hyperhomocysteinemia. British Journal of Pharmacology, v. 157, n. 4, p. 568-580, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1476-5381.2009.00165.x. Acesso em: 04 nov. 2024.
APA
Andrade, C. R. de, Leite, P. F., Montezano, A. C., Casolari, D. A., Yogi, A., Passaglia, R. de C. A. T., et al. (2009). Increased endothelin-1 reactivity and endothelial dysfunction in carotid arteries from rats with hyperhomocysteinemia. British Journal of Pharmacology, 157( 4), 568-580. doi:10.1111/j.1476-5381.2009.00165.x
NLM
Andrade CR de, Leite PF, Montezano AC, Casolari DA, Yogi A, Passaglia R de CAT, Haddad R, Eberlin MN, Laurindo FRM, Souza HP de, Corrêa FM de A, Oliveira AM de. Increased endothelin-1 reactivity and endothelial dysfunction in carotid arteries from rats with hyperhomocysteinemia [Internet]. British Journal of Pharmacology. 2009 ; 157( 4): 568-580.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1111/j.1476-5381.2009.00165.x
Vancouver
Andrade CR de, Leite PF, Montezano AC, Casolari DA, Yogi A, Passaglia R de CAT, Haddad R, Eberlin MN, Laurindo FRM, Souza HP de, Corrêa FM de A, Oliveira AM de. Increased endothelin-1 reactivity and endothelial dysfunction in carotid arteries from rats with hyperhomocysteinemia [Internet]. British Journal of Pharmacology. 2009 ; 157( 4): 568-580.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1111/j.1476-5381.2009.00165.x

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