Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats (2011)
- Authors:
- Autor USP: PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP
- Unidade: FMRP
- DOI: 10.1152/ajpheart.00084.2011
- Subjects: HIPERTENSÃO; ENDOTÉLIO; ANTAGONISTAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: American Journal of Physiology: Heart and Circulatory Physiology
- ISSN: 0363-6135
- Volume/Número/Paginação/Ano: v. 301, n. 2, p. H409-H417, 2011
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
MATSUMOTO, Takayuri; PASSAGLIA, Rita de Cassia Aleixo Tostes; WEBB, R. Clinton. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology, Bethesda, v. 301, n. 2, p. H409-H417, 2011. DOI: 10.1152/ajpheart.00084.2011. -
APA
Matsumoto, T., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology, 301( 2), H409-H417. doi:10.1152/ajpheart.00084.2011 -
NLM
Matsumoto T, Passaglia R de CAT, Webb RC. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology. 2011 ; 301( 2): H409-H417. -
Vancouver
Matsumoto T, Passaglia R de CAT, Webb RC. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology. 2011 ; 301( 2): H409-H417. - Novos mecanismos moleculares da hipertensão, sistema endotelia na hipertensão arterial
- Contribution of endothelin-I to renal activator protein-I activation and macrophage infiltration in aldosterone-induced hypertension
- ET-1-induced oxidative stress in doca-salt hypertension involves NAD(P)H oxidase-independent mechanisms
- Aldosterone activates vascular p38MAP kinase and NADPH oxidase Via c-Src
- Nox 5 expression is regulated by endothelin-1, angiotensin II, and aldosterone in human microvascular endothelial cells and may be upregulated in vascular smooth muscle cells from hypertensive patients
- A key role for the Na/K ATPase pump in the endothelium-dependent oscillatory activity of mouse resistance mesenteric arteries
- c-Src regulates vascular inflammatory and fibrogenic but not growth signaling pathways in aldosterone-induced hypertension
- Vascular smooth muscle contraction and relaxation
- Nongenomic vascular signaling by aldosterone involves the novel Mg2+ transporter transient receptor potential melastatin 7 cation channel
- Differential regulation of calcium-sensitive NOX 5 by vasoactive agents in human endothelial and vascular smooth muscle cells: upregulation in hypertension
Informações sobre o DOI: 10.1152/ajpheart.00084.2011 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
