Filtros : "Indexado no Current Abstracts" "Passaglia, Rita de Cassia Aleixo Tostes" Limpar

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  • Source: Journal of the American Society of Hypertension. Unidade: FMRP

    Subjects: HIPERTENSÃO, ALDEÍDOS

    Acesso à fonteDOIHow to cite
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    • ABNT

      CHOI, Hyehun e PASSAGLIA, Rita de Cassia Aleixo Tostes e WEBB, R. Clinton. Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice. Journal of the American Society of Hypertension, v. 5, n. 3, p. 154-160, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.jash.2011.02.005. Acesso em: 05 out. 2024.
    • APA

      Choi, H., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice. Journal of the American Society of Hypertension, 5( 3), 154-160. doi:10.1016/j.jash.2011.02.005
    • NLM

      Choi H, Passaglia R de CAT, Webb RC. Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice [Internet]. Journal of the American Society of Hypertension. 2011 ; 5( 3): 154-160.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.jash.2011.02.005
    • Vancouver

      Choi H, Passaglia R de CAT, Webb RC. Mitochondrial aldehyde dehydrogenase prevents ROS-induced vascular contraction in angiotensin-II hypertensive mice [Internet]. Journal of the American Society of Hypertension. 2011 ; 5( 3): 154-160.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.jash.2011.02.005
  • Source: Cardiovascular Research. Unidade: FMRP

    Subjects: ALDOSTERONA, CÉLULAS MUSCULARES

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    • ABNT

      CALLERA, Gláucia Elena et al. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research, v. 91, n. 4, p. 720-731, 2011Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvr131. Acesso em: 05 out. 2024.
    • APA

      Callera, G. E., Yogi, Á., Briones, A. M., Montezano, A. C. I., He, Y., Passaglia, R. de C. A. T., et al. (2011). Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research, 91( 4), 720-731. doi:10.1093/cvr/cvr131
    • NLM

      Callera GE, Yogi Á, Briones AM, Montezano ACI, He Y, Passaglia R de CAT, Schiffrin EL, Touyz RM. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR [Internet]. Cardiovascular Research. 2011 ; 91( 4): 720-731.[citado 2024 out. 05 ] Available from: https://doi.org/10.1093/cvr/cvr131
    • Vancouver

      Callera GE, Yogi Á, Briones AM, Montezano ACI, He Y, Passaglia R de CAT, Schiffrin EL, Touyz RM. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR [Internet]. Cardiovascular Research. 2011 ; 91( 4): 720-731.[citado 2024 out. 05 ] Available from: https://doi.org/10.1093/cvr/cvr131
  • Source: Hypertension. Unidade: FMRP

    Subjects: ANGIOTENSINA II, HIPERTENSÃO

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    • ABNT

      GIACHINI, Fernanda R. et al. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, v. 57, n. 3, p. 655-663, 2011Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327. Acesso em: 05 out. 2024.
    • APA

      Giachini, F. R., Lima, V. V., Carneiro, F. S., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1. Hypertension, 57( 3), 655-663. doi:10.1161/HYPERTENSIONAHA.110.164327
    • NLM

      Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 out. 05 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
    • Vancouver

      Giachini FR, Lima VV, Carneiro FS, Passaglia R de CAT, Webb RC. Decreased cGMP level contributes to increased contraction in arteries from hypertensive rats: role of phosphodiesterase 1 [Internet]. Hypertension. 2011 ; 57( 3): 655-663.[citado 2024 out. 05 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.110.164327
  • Source: American Journal of Physiology: Heart and Circulatory Physiology. Unidade: FMRP

    Subjects: HIPERTENSÃO, ENDOTÉLIO, ANTAGONISTAS

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    • ABNT

      MATSUMOTO, Takayuri e PASSAGLIA, Rita de Cassia Aleixo Tostes e WEBB, R. Clinton. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology, v. 301, n. 2, p. H409-H417, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00084.2011. Acesso em: 05 out. 2024.
    • APA

      Matsumoto, T., Passaglia, R. de C. A. T., & Webb, R. C. (2011). Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats. American Journal of Physiology: Heart and Circulatory Physiology, 301( 2), H409-H417. doi:10.1152/ajpheart.00084.2011
    • NLM

      Matsumoto T, Passaglia R de CAT, Webb RC. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2011 ; 301( 2): H409-H417.[citado 2024 out. 05 ] Available from: https://doi.org/10.1152/ajpheart.00084.2011
    • Vancouver

      Matsumoto T, Passaglia R de CAT, Webb RC. Uridine adenosine tetraphosphate-induced contraction is increases in renal but not pulmonary arteries from DOCA-salt hypertensive rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2011 ; 301( 2): H409-H417.[citado 2024 out. 05 ] Available from: https://doi.org/10.1152/ajpheart.00084.2011

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