Filtros : "Indexado na Base de Dados PubMed" "ISAAC, LOURDES" Removidos: "Federal University of Sergipe, Department of Physiology, Sergipe, Brazil" "Teixeira, Marta Maria Geraldes" "IME-USP" Limpar

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  • Fonte: Frontiers in Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      MACEDO, Ana Catarina e ISAAC, Lourdes. Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway. Frontiers in Immunology, v. 7, p. 1-7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00055. Acesso em: 09 ago. 2024.
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      Macedo, A. C., & Isaac, L. (2016). Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway. Frontiers in Immunology, 7, 1-7. doi:10.3389/fimmu.2016.00055
    • NLM

      Macedo AC, Isaac L. Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway [Internet]. Frontiers in Immunology. 2016 ; 7 1-7.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00055
    • Vancouver

      Macedo AC, Isaac L. Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway [Internet]. Frontiers in Immunology. 2016 ; 7 1-7.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00055
  • Fonte: Immunobiology. Unidade: ICB

    Assunto: IMUNOBIOLOGIA

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      CASTIBLANCO-VALENCIA, Monica Marcela et al. Plasmin cleaves fibrinogen and the human complement proteins C3b and C5 in the presence of Leptospira interrogans proteins: A new role of LigA and LigB in invasion and complement immune evasion. Immunobiology, v. 221, n. 5, p. 679-689, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.01.001. Acesso em: 09 ago. 2024.
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      Castiblanco-Valencia, M. M., Fraga, T. R., Pagotto, A. H., Serrano, S. M. de T., Abreu, P. A. E., Barbosa, A. S., & Isaac, L. (2016). Plasmin cleaves fibrinogen and the human complement proteins C3b and C5 in the presence of Leptospira interrogans proteins: A new role of LigA and LigB in invasion and complement immune evasion. Immunobiology, 221( 5), 679-689. doi:10.1016/j.imbio.2016.01.001
    • NLM

      Castiblanco-Valencia MM, Fraga TR, Pagotto AH, Serrano SM de T, Abreu PAE, Barbosa AS, Isaac L. Plasmin cleaves fibrinogen and the human complement proteins C3b and C5 in the presence of Leptospira interrogans proteins: A new role of LigA and LigB in invasion and complement immune evasion [Internet]. Immunobiology. 2016 ; 221( 5): 679-689.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imbio.2016.01.001
    • Vancouver

      Castiblanco-Valencia MM, Fraga TR, Pagotto AH, Serrano SM de T, Abreu PAE, Barbosa AS, Isaac L. Plasmin cleaves fibrinogen and the human complement proteins C3b and C5 in the presence of Leptospira interrogans proteins: A new role of LigA and LigB in invasion and complement immune evasion [Internet]. Immunobiology. 2016 ; 221( 5): 679-689.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imbio.2016.01.001
  • Fonte: Immunology Letters. Unidades: FMVZ, ICB

    Assuntos: LEPTOSPIROSE, IMUNOGLOBULINAS, CITOMETRIA DE FLUXO, BACTÉRIAS

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      CASTIBLANCO-VALENCIA, Mónica Marcela et al. Acquisition of negative complement regulators by the saprophyte Leptospira biflexa expressing LigA or LigB confers enhanced survival in human serum. Immunology Letters, v. 173, p. 61-68, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.imlet.2016.03.005. Acesso em: 09 ago. 2024.
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      Castiblanco-Valencia, M. M., Fraga, T. R., Breda, L. C. D., Vasconcellos, S. A., Figueira, C. P., Picardeau, M., et al. (2016). Acquisition of negative complement regulators by the saprophyte Leptospira biflexa expressing LigA or LigB confers enhanced survival in human serum. Immunology Letters, 173, 61-68. doi:10.1016/j.imlet.2016.03.005
    • NLM

      Castiblanco-Valencia MM, Fraga TR, Breda LCD, Vasconcellos SA, Figueira CP, Picardeau M, Wunder E, Ko AI, Barbosa AS, Isaac L. Acquisition of negative complement regulators by the saprophyte Leptospira biflexa expressing LigA or LigB confers enhanced survival in human serum [Internet]. Immunology Letters. 2016 ; 173 61-68.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imlet.2016.03.005
    • Vancouver

      Castiblanco-Valencia MM, Fraga TR, Breda LCD, Vasconcellos SA, Figueira CP, Picardeau M, Wunder E, Ko AI, Barbosa AS, Isaac L. Acquisition of negative complement regulators by the saprophyte Leptospira biflexa expressing LigA or LigB confers enhanced survival in human serum [Internet]. Immunology Letters. 2016 ; 173 61-68.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imlet.2016.03.005
  • Fonte: Immunobiology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SILVA, Karina Ribeiro da et al. Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency. Immunobiology, v. 221, n. 5, p. 641-649, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.01.005. Acesso em: 09 ago. 2024.
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      Silva, K. R. da, Fraga, T. R., Lucatelli, J. F., Grumach, A. S., & Isaac, L. (2016). Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency. Immunobiology, 221( 5), 641-649. doi:10.1016/j.imbio.2016.01.005
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      Silva KR da, Fraga TR, Lucatelli JF, Grumach AS, Isaac L. Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency [Internet]. Immunobiology. 2016 ; 221( 5): 641-649.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imbio.2016.01.005
    • Vancouver

      Silva KR da, Fraga TR, Lucatelli JF, Grumach AS, Isaac L. Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency [Internet]. Immunobiology. 2016 ; 221( 5): 641-649.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imbio.2016.01.005
  • Fonte: Frontiers in Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FRAGA, Tatiana Rodrigues e ISAAC, Lourdes e BARBOSA, Angela Silva. Complement evasion by pathogenic Leptospira. Frontiers in Immunology, v. 7, n. 623, p. 1-7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00623. Acesso em: 09 ago. 2024.
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      Fraga, T. R., Isaac, L., & Barbosa, A. S. (2016). Complement evasion by pathogenic Leptospira. Frontiers in Immunology, 7( 623), 1-7. doi:10.3389/fimmu.2016.00623
    • NLM

      Fraga TR, Isaac L, Barbosa AS. Complement evasion by pathogenic Leptospira [Internet]. Frontiers in Immunology. 2016 ; 7( 623): 1-7.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00623
    • Vancouver

      Fraga TR, Isaac L, Barbosa AS. Complement evasion by pathogenic Leptospira [Internet]. Frontiers in Immunology. 2016 ; 7( 623): 1-7.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00623
  • Fonte: Infection and Immunity. Unidade: ICB

    Assuntos: IMUNOLOGIA, IMUNOPROTEINAS, POLIMERIZAÇÃO, IMUNIDADE, ANTICORPO

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      SILVA, Ludmila Bezerra da et al. Pathogenic Leptospira species acquire factor H and vitronectin via the surface protein LcpA. Infection and Immunity, v. 83, n. 3, p. 888-897, 2015Tradução . . Disponível em: https://doi.org/10.1128/IAI.02844-14. Acesso em: 09 ago. 2024.
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      Silva, L. B. da, Miragaia, L. dos S., Breda, L. C. D., Abe, C. M., Schmidt, M. C. B., Moro, A. M., et al. (2015). Pathogenic Leptospira species acquire factor H and vitronectin via the surface protein LcpA. Infection and Immunity, 83( 3), 888-897. doi:10.1128/IAI.02844-14
    • NLM

      Silva LB da, Miragaia L dos S, Breda LCD, Abe CM, Schmidt MCB, Moro AM, Monaris D, Conde JN, Józsi M, Isaac L, Abreu PAE, Barbosa AS. Pathogenic Leptospira species acquire factor H and vitronectin via the surface protein LcpA [Internet]. Infection and Immunity. 2015 ; 83( 3): 888-897.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1128/IAI.02844-14
    • Vancouver

      Silva LB da, Miragaia L dos S, Breda LCD, Abe CM, Schmidt MCB, Moro AM, Monaris D, Conde JN, Józsi M, Isaac L, Abreu PAE, Barbosa AS. Pathogenic Leptospira species acquire factor H and vitronectin via the surface protein LcpA [Internet]. Infection and Immunity. 2015 ; 83( 3): 888-897.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1128/IAI.02844-14
  • Fonte: Immunobiology. Unidade: ICB

    Assuntos: IMUNOLOGIA, LECTINAS, IMUNOPROTEINAS

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    • ABNT

      SAHAGÚN-RUIZ, Alfredo et al. Studies of the binding of ficolin-2 and ficolin-3 from the complement lectin pathway to Leptospira biflexa, Pasteurella pneumotropica and Diarrheagenic Escherichia coli. Immunobiology, v. 220, n. 10, p. 1177-1185, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2015.06.001. Acesso em: 09 ago. 2024.
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      Sahagún-Ruiz, A., Breda, L. C. D., Valencia, M. M. C., Elias, W. P., Munthe-Fog, L., Garred, P., et al. (2015). Studies of the binding of ficolin-2 and ficolin-3 from the complement lectin pathway to Leptospira biflexa, Pasteurella pneumotropica and Diarrheagenic Escherichia coli. Immunobiology, 220( 10), 1177-1185. doi:10.1016/j.imbio.2015.06.001
    • NLM

      Sahagún-Ruiz A, Breda LCD, Valencia MMC, Elias WP, Munthe-Fog L, Garred P, Barbosa AS, Isaac L. Studies of the binding of ficolin-2 and ficolin-3 from the complement lectin pathway to Leptospira biflexa, Pasteurella pneumotropica and Diarrheagenic Escherichia coli [Internet]. Immunobiology. 2015 ; 220( 10): 1177-1185.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imbio.2015.06.001
    • Vancouver

      Sahagún-Ruiz A, Breda LCD, Valencia MMC, Elias WP, Munthe-Fog L, Garred P, Barbosa AS, Isaac L. Studies of the binding of ficolin-2 and ficolin-3 from the complement lectin pathway to Leptospira biflexa, Pasteurella pneumotropica and Diarrheagenic Escherichia coli [Internet]. Immunobiology. 2015 ; 220( 10): 1177-1185.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imbio.2015.06.001
  • Fonte: European Journal of Immunology. Unidade: ICB

    Assuntos: IMUNOLOGIA, IMUNOPROTEINAS, IMUNOGLOBULINA

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      PEKKARINEN, Pirkka T. et al. Dysregulation of adaptive immune responses in complement C3-deficient patients. European Journal of Immunology, v. 45, p. 915-921, 2015Tradução . . Disponível em: https://doi.org/10.1002/eji.201444948. Acesso em: 09 ago. 2024.
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      Pekkarinen, P. T., Heikkilä, N., Kisand, K., Peterson, P., Botto, M., Daha, M. R., et al. (2015). Dysregulation of adaptive immune responses in complement C3-deficient patients. European Journal of Immunology, 45, 915-921. doi:10.1002/eji.201444948
    • NLM

      Pekkarinen PT, Heikkilä N, Kisand K, Peterson P, Botto M, Daha MR, Drouet C, Isaac L, Helminen M, Haahtela T, Meri S, Jarva H, Arstila P. Dysregulation of adaptive immune responses in complement C3-deficient patients [Internet]. European Journal of Immunology. 2015 ; 45 915-921.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1002/eji.201444948
    • Vancouver

      Pekkarinen PT, Heikkilä N, Kisand K, Peterson P, Botto M, Daha MR, Drouet C, Isaac L, Helminen M, Haahtela T, Meri S, Jarva H, Arstila P. Dysregulation of adaptive immune responses in complement C3-deficient patients [Internet]. European Journal of Immunology. 2015 ; 45 915-921.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1002/eji.201444948
  • Fonte: Journal of Infectious Diseases. Unidade: ICB

    Assuntos: ESCHEICHIA COLI, IMUNOPROTEINAS

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      ABREU, Afonso G. et al. The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins. Journal of Infectious Diseases, v. 212, n. 1, p. 106-115, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiv013. Acesso em: 09 ago. 2024.
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      Abreu, A. G., Fraga, T. R., Martínez, A. P. G., Kondo, M. Y., Juliano, M. A., Juliano, L., et al. (2015). The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins. Journal of Infectious Diseases, 212( 1), 106-115. doi:10.1093/infdis/jiv013
    • NLM

      Abreu AG, Fraga TR, Martínez APG, Kondo MY, Juliano MA, Juliano L, Navarro-Garcia F, Isaac L, Barbosa AS, Elias WP. The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins [Internet]. Journal of Infectious Diseases. 2015 ; 212( 1): 106-115.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1093/infdis/jiv013
    • Vancouver

      Abreu AG, Fraga TR, Martínez APG, Kondo MY, Juliano MA, Juliano L, Navarro-Garcia F, Isaac L, Barbosa AS, Elias WP. The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins [Internet]. Journal of Infectious Diseases. 2015 ; 212( 1): 106-115.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1093/infdis/jiv013
  • Fonte: Immunology Letters. Unidade: ICB

    Assuntos: IMUNOLOGIA, CAMUNDONGOS, IMUNOPROTEINAS

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      BAVIA, Lorena et al. Basal physiological parameters of two congenic mice strains: C5 deficient C57BL/6 and C5 sufficient A/J. Immunology Letters, v. 159, n. 1-2, p. 47-54, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.imlet.2014.02.010. Acesso em: 09 ago. 2024.
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      Bavia, L., Castro, Í. A. de, Massironi, S. M. G., & Isaac, L. (2014). Basal physiological parameters of two congenic mice strains: C5 deficient C57BL/6 and C5 sufficient A/J. Immunology Letters, 159( 1-2), 47-54. doi:10.1016/j.imlet.2014.02.010
    • NLM

      Bavia L, Castro ÍA de, Massironi SMG, Isaac L. Basal physiological parameters of two congenic mice strains: C5 deficient C57BL/6 and C5 sufficient A/J [Internet]. Immunology Letters. 2014 ; 159( 1-2): 47-54.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imlet.2014.02.010
    • Vancouver

      Bavia L, Castro ÍA de, Massironi SMG, Isaac L. Basal physiological parameters of two congenic mice strains: C5 deficient C57BL/6 and C5 sufficient A/J [Internet]. Immunology Letters. 2014 ; 159( 1-2): 47-54.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.imlet.2014.02.010
  • Fonte: PLOS ONE. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SAHAGÚN-RUIZ, Alfredo et al. Pasteurella pneumotropica Evades the human complement system by acquisition of the complement regulators factor H and C4BP. PLOS ONE, v. 9, n. 10, p. 1-8, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111194. Acesso em: 09 ago. 2024.
    • APA

      Sahagún-Ruiz, A., Martinez, A. P. G., Breda, L. C. D., Fraga, T. R., Valencia, M. M. C., Barbosa, A. S., & Isaac, L. (2014). Pasteurella pneumotropica Evades the human complement system by acquisition of the complement regulators factor H and C4BP. PLOS ONE, 9( 10), 1-8. doi:10.1371/journal.pone.0111194
    • NLM

      Sahagún-Ruiz A, Martinez APG, Breda LCD, Fraga TR, Valencia MMC, Barbosa AS, Isaac L. Pasteurella pneumotropica Evades the human complement system by acquisition of the complement regulators factor H and C4BP [Internet]. PLOS ONE. 2014 ; 9( 10): 1-8.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0111194
    • Vancouver

      Sahagún-Ruiz A, Martinez APG, Breda LCD, Fraga TR, Valencia MMC, Barbosa AS, Isaac L. Pasteurella pneumotropica Evades the human complement system by acquisition of the complement regulators factor H and C4BP [Internet]. PLOS ONE. 2014 ; 9( 10): 1-8.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0111194
  • Fonte: PLoS Neglected Tropical Diseases. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FARIAS, Leonardo P. et al. On the three-finger protein domain fold and CD59-like proteins in Schistosoma mansoni. PLoS Neglected Tropical Diseases, v. 7, n. 10, p. 1-15, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0002482. Acesso em: 09 ago. 2024.
    • APA

      Farias, L. P., Krautz-Peterson, G., Tararam, C. A., Araújo-Montoya, B. O., Fraga, T. R., Rofatto, H. K., et al. (2013). On the three-finger protein domain fold and CD59-like proteins in Schistosoma mansoni. PLoS Neglected Tropical Diseases, 7( 10), 1-15. doi:10.1371/journal.pntd.0002482
    • NLM

      Farias LP, Krautz-Peterson G, Tararam CA, Araújo-Montoya BO, Fraga TR, Rofatto HK, Silva-Jr FP, Isaac L, Da'dara AA, Wilson RA, Shoemaker CB, Leite LCC. On the three-finger protein domain fold and CD59-like proteins in Schistosoma mansoni [Internet]. PLoS Neglected Tropical Diseases. 2013 ; 7( 10): 1-15.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pntd.0002482
    • Vancouver

      Farias LP, Krautz-Peterson G, Tararam CA, Araújo-Montoya BO, Fraga TR, Rofatto HK, Silva-Jr FP, Isaac L, Da'dara AA, Wilson RA, Shoemaker CB, Leite LCC. On the three-finger protein domain fold and CD59-like proteins in Schistosoma mansoni [Internet]. PLoS Neglected Tropical Diseases. 2013 ; 7( 10): 1-15.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pntd.0002482
  • Fonte: Journal of Immunology. Unidade: ICB

    Assuntos: HISTOLOGIA, IMUNOLOGIA

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      ALBUQUERQUE, José Antonio Tavares de et al. Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient. Journal of Immunology, v. 189, n. 6, p. 3242-3248, 2012Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1201418. Acesso em: 09 ago. 2024.
    • APA

      Albuquerque, J. A. T. de, Lamers, M. L., Castiblanco-Valencia, M. M., Santos, M. F. dos, & Isaac, L. (2012). Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient. Journal of Immunology, 189( 6), 3242-3248. doi:10.4049/jimmunol.1201418
    • NLM

      Albuquerque JAT de, Lamers ML, Castiblanco-Valencia MM, Santos MF dos, Isaac L. Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient [Internet]. Journal of Immunology. 2012 ; 189( 6): 3242-3248.[citado 2024 ago. 09 ] Available from: https://doi.org/10.4049/jimmunol.1201418
    • Vancouver

      Albuquerque JAT de, Lamers ML, Castiblanco-Valencia MM, Santos MF dos, Isaac L. Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient [Internet]. Journal of Immunology. 2012 ; 189( 6): 3242-3248.[citado 2024 ago. 09 ] Available from: https://doi.org/10.4049/jimmunol.1201418
  • Fonte: Journal of Infectious Diseases. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      CASTIBLANCO-VALENCIA, Mónica Marcela et al. Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP. Journal of Infectious Diseases, v. 205, n. 6, p. 995-1004, 2012Tradução . . Disponível em: https://doi.org/10.1093/infdis/jir875. Acesso em: 09 ago. 2024.
    • APA

      Castiblanco-Valencia, M. M., Fraga, T. R., Silva, L. B. da, Monaris, D., Abreu, P. A. E., Strobel, S., et al. (2012). Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP. Journal of Infectious Diseases, 205( 6), 995-1004. doi:10.1093/infdis/jir875
    • NLM

      Castiblanco-Valencia MM, Fraga TR, Silva LB da, Monaris D, Abreu PAE, Strobel S, Józsi M, Isaac L, Barbosa AS. Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP [Internet]. Journal of Infectious Diseases. 2012 ; 205( 6): 995-1004.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1093/infdis/jir875
    • Vancouver

      Castiblanco-Valencia MM, Fraga TR, Silva LB da, Monaris D, Abreu PAE, Strobel S, Józsi M, Isaac L, Barbosa AS. Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP [Internet]. Journal of Infectious Diseases. 2012 ; 205( 6): 995-1004.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1093/infdis/jir875
  • Fonte: Molecular Vision. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      SILVA, Aldacilene Souza da et al. Plasma levels of complement proteins from the alternative pathway in patients with age-related macular degeneration are independent of Complement Factor H Tyr402His polymorphism. Molecular Vision, v. 18, p. 2288-2299, 2012Tradução . . Acesso em: 09 ago. 2024.
    • APA

      Silva, A. S. da, Teixeira, A. G., Bavia, L., Lin, F., Velletri, R., Belfort Jr, R., & Isaac, L. (2012). Plasma levels of complement proteins from the alternative pathway in patients with age-related macular degeneration are independent of Complement Factor H Tyr402His polymorphism. Molecular Vision, 18, 2288-2299.
    • NLM

      Silva AS da, Teixeira AG, Bavia L, Lin F, Velletri R, Belfort Jr R, Isaac L. Plasma levels of complement proteins from the alternative pathway in patients with age-related macular degeneration are independent of Complement Factor H Tyr402His polymorphism. Molecular Vision. 2012 ; 18 2288-2299.[citado 2024 ago. 09 ]
    • Vancouver

      Silva AS da, Teixeira AG, Bavia L, Lin F, Velletri R, Belfort Jr R, Isaac L. Plasma levels of complement proteins from the alternative pathway in patients with age-related macular degeneration are independent of Complement Factor H Tyr402His polymorphism. Molecular Vision. 2012 ; 18 2288-2299.[citado 2024 ago. 09 ]

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