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Artigo de periodico
Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice (2018)
Santos, Jaqueline Rocha Borges dos ; Rae, Mariana ; Teixeira, Ana Maria Aristimunho; Teixeira, Simone Aparecida ; Munhoz, Carolina Demarchi ; Muscará, Marcelo Nicolás (Orientador) ; Marcourakis, Tania ; Szumlinski, Karen K. ; Camarini, Rosana
Source: Alcohol. Unidades: ICB, FCF
Subjects: ÓXIDO NÍTRICO, ETANOL, ESTRESSE, ADOLESCÊNCIA
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SANTOS, Jaqueline Rocha Borges dos et al. Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice. Alcohol, v. 68, p. 71-79, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.alcohol.2017.10.004. Acesso em: 17 jun. 2024.
APA
Santos, J. R. B. dos, Rae, M., Teixeira, A. M. A., Teixeira, S. A., Munhoz, C. D., Marcourakis, T., et al. (2018). Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice. Alcohol, 68, 71-79. doi:10.1016/j.alcohol.2017.10.004
NLM
Santos JRB dos, Rae M, Teixeira AMA, Teixeira SA, Munhoz CD, Marcourakis T, Szumlinski KK, Camarini R. Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice [Internet]. Alcohol. 2018 ; 68 71-79.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.alcohol.2017.10.004
Vancouver
Santos JRB dos, Rae M, Teixeira AMA, Teixeira SA, Munhoz CD, Marcourakis T, Szumlinski KK, Camarini R. Involvement of neuronal nitric oxide synthase in cross-sensitization between chronic unpredictable stress and ethanol in adolescent and adult mice [Internet]. Alcohol. 2018 ; 68 71-79.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.alcohol.2017.10.004
Artigo de periodico
Targeting the Plasmodium falciparum Plasmepsin V by ligand-based virtual screening (2018)
Meissner, Kamila Anna ; Kronenberger, Thales ; Maltarollo, Vinícius Gonçalves ; Trossini, Gustavo Henrique Goulart ; Wrenger, Carsten
Source: Chemical Biology and Drug Design. Unidades: FCF, ICB
Subjects: PLASMODIUM FALCIPARUM, PARASITOLOGIA, MALÁRIA, QUIMIOTERAPIA, ENZIMAS PROTEOLITICAS, ATIVAÇÃO ENZIMÁTICA
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MEISSNER, Kamila Anna et al. Targeting the Plasmodium falciparum Plasmepsin V by ligand-based virtual screening. Chemical Biology and Drug Design, v. 2018 p. 1-13, 2018Tradução . . Disponível em: https://doi.org/10.1111/cbdd.13416. Acesso em: 17 jun. 2024.
APA
Meissner, K. A., Kronenberger, T., Maltarollo, V. G., Trossini, G. H. G., & Wrenger, C. (2018). Targeting the Plasmodium falciparum Plasmepsin V by ligand-based virtual screening. Chemical Biology and Drug Design, 2018 p. 1-13. doi:10.1111/cbdd.13416
NLM
Meissner KA, Kronenberger T, Maltarollo VG, Trossini GHG, Wrenger C. Targeting the Plasmodium falciparum Plasmepsin V by ligand-based virtual screening [Internet]. Chemical Biology and Drug Design. 2018 ; 2018 p. 1-13[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/cbdd.13416
Vancouver
Meissner KA, Kronenberger T, Maltarollo VG, Trossini GHG, Wrenger C. Targeting the Plasmodium falciparum Plasmepsin V by ligand-based virtual screening [Internet]. Chemical Biology and Drug Design. 2018 ; 2018 p. 1-13[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/cbdd.13416
Artigo de periodico
Cagaita fruit (Eugenia dysenterica DC.) and obesity: role of polyphenols on already established obesity (2018)
Pestana, Carlos Mario Donado; Donado, Priscila Robertina dos Santos; Ramirez, Luis Daniel Daza; Oliveira, Thiago Belchior de; Festuccia, William Tadeu Lara ; Genovese, Maria Inês
Source: Food Research International. Unidades: ICB, FCF
Subjects: CERRADO, OBESIDADE, FRUTAS, COMPOSTOS FENÓLICOS, DISLIPIDEMIAS
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PESTANA, Carlos Mario Donado et al. Cagaita fruit (Eugenia dysenterica DC.) and obesity: role of polyphenols on already established obesity. Food Research International, v. 103, p. 40-47, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.foodres.2017.10.011. Acesso em: 17 jun. 2024.
APA
Pestana, C. M. D., Donado, P. R. dos S., Ramirez, L. D. D., Oliveira, T. B. de, Festuccia, W. T. L., & Genovese, M. I. (2018). Cagaita fruit (Eugenia dysenterica DC.) and obesity: role of polyphenols on already established obesity. Food Research International, 103, 40-47. doi:10.1016/j.foodres.2017.10.011
NLM
Pestana CMD, Donado PR dos S, Ramirez LDD, Oliveira TB de, Festuccia WTL, Genovese MI. Cagaita fruit (Eugenia dysenterica DC.) and obesity: role of polyphenols on already established obesity [Internet]. Food Research International. 2018 ; 103 40-47.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.foodres.2017.10.011
Vancouver
Pestana CMD, Donado PR dos S, Ramirez LDD, Oliveira TB de, Festuccia WTL, Genovese MI. Cagaita fruit (Eugenia dysenterica DC.) and obesity: role of polyphenols on already established obesity [Internet]. Food Research International. 2018 ; 103 40-47.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.foodres.2017.10.011
Artigo de periodico
Alkaline pH promotes NADPH oxidase-independent neutrophil extracellular trap formation: a matter of mitochondrial reactive oxygen species generation and citrullination and cleavage of histone (2018)
Souza, Cristiane Naffah; Breda, Leandro Carvalho Dantas; Khan, Meraj A.; Sweezey, Neil; Palaniyar, Nades; Câmara, Niels Olsen Saraiva ; Almeida, Sandro Rogério de
Source: Frontiers in Immunology. Unidades: FCF, ICB
Subjects: CÁLCIO, PROTEÍNAS, CROMATINA, MITOCÔNDRIAS, NEUTRÓFILOS, WESTERN BLOTTING, HIDROGÊNIO
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SOUZA, Cristiane Naffah et al. Alkaline pH promotes NADPH oxidase-independent neutrophil extracellular trap formation: a matter of mitochondrial reactive oxygen species generation and citrullination and cleavage of histone. Frontiers in Immunology, v. 8, p. 1-15, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01849. Acesso em: 17 jun. 2024.
APA
Souza, C. N., Breda, L. C. D., Khan, M. A., Sweezey, N., Palaniyar, N., Câmara, N. O. S., & Almeida, S. R. de. (2018). Alkaline pH promotes NADPH oxidase-independent neutrophil extracellular trap formation: a matter of mitochondrial reactive oxygen species generation and citrullination and cleavage of histone. Frontiers in Immunology, 8, 1-15. doi:10.3389/fimmu.2017.01849
NLM
Souza CN, Breda LCD, Khan MA, Sweezey N, Palaniyar N, Câmara NOS, Almeida SR de. Alkaline pH promotes NADPH oxidase-independent neutrophil extracellular trap formation: a matter of mitochondrial reactive oxygen species generation and citrullination and cleavage of histone [Internet]. Frontiers in Immunology. 2018 ; 8 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.3389/fimmu.2017.01849
Vancouver
Souza CN, Breda LCD, Khan MA, Sweezey N, Palaniyar N, Câmara NOS, Almeida SR de. Alkaline pH promotes NADPH oxidase-independent neutrophil extracellular trap formation: a matter of mitochondrial reactive oxygen species generation and citrullination and cleavage of histone [Internet]. Frontiers in Immunology. 2018 ; 8 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.3389/fimmu.2017.01849
Artigo de periodico
Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer (2017)
Geraldo, Murilo Vieira; Nakaya, Helder Takashi Imoto ; Kimura, Edna Teruko
Source: Oncotarget. Unidades: FCF, ICB
Assunto: HISTOLOGIA
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GERALDO, Murilo Vieira e NAKAYA, Helder Takashi Imoto e KIMURA, Edna Teruko. Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer. Oncotarget, v. 8, n. 6, p. 9597-9607, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.14162. Acesso em: 17 jun. 2024.
APA
Geraldo, M. V., Nakaya, H. T. I., & Kimura, E. T. (2017). Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer. Oncotarget, 8( 6), 9597-9607. doi:10.18632/oncotarget.14162
NLM
Geraldo MV, Nakaya HTI, Kimura ET. Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer [Internet]. Oncotarget. 2017 ; 8( 6): 9597-9607.[citado 2024 jun. 17 ] Available from: https://doi.org/10.18632/oncotarget.14162
Vancouver
Geraldo MV, Nakaya HTI, Kimura ET. Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer [Internet]. Oncotarget. 2017 ; 8( 6): 9597-9607.[citado 2024 jun. 17 ] Available from: https://doi.org/10.18632/oncotarget.14162
Artigo de periodico
Co-encapsulation of paclitaxel and C6 ceramide in tributyrin-containing nanocarriers improve co-localization in the skin and potentiate cytotoxic effects in 2D and 3D models (2017)
Carvalho, Vanessa F. M; Migotto, Amanda; Giacone, Daniela V; Lemos, Débora P. de; Zanoni, Thalita B; Maria-Engler, Silvya Stuchi ; Lotufo, Leticia Veras Costa ; Lopes, Luciana Biagini
Source: European Journal of Pharmaceutical Sciences. Unidades: FCF, ICB
Subjects: PELE, MELANOMA
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ABNT
CARVALHO, Vanessa F. M et al. Co-encapsulation of paclitaxel and C6 ceramide in tributyrin-containing nanocarriers improve co-localization in the skin and potentiate cytotoxic effects in 2D and 3D models. European Journal of Pharmaceutical Sciences, v. 109, p. 131-143, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejps.2017.07.023. Acesso em: 17 jun. 2024.
APA
Carvalho, V. F. M., Migotto, A., Giacone, D. V., Lemos, D. P. de, Zanoni, T. B., Maria-Engler, S. S., et al. (2017). Co-encapsulation of paclitaxel and C6 ceramide in tributyrin-containing nanocarriers improve co-localization in the skin and potentiate cytotoxic effects in 2D and 3D models. European Journal of Pharmaceutical Sciences, 109, 131-143. doi:10.1016/j.ejps.2017.07.023
NLM
Carvalho VFM, Migotto A, Giacone DV, Lemos DP de, Zanoni TB, Maria-Engler SS, Lotufo LVC, Lopes LB. Co-encapsulation of paclitaxel and C6 ceramide in tributyrin-containing nanocarriers improve co-localization in the skin and potentiate cytotoxic effects in 2D and 3D models [Internet]. European Journal of Pharmaceutical Sciences. 2017 ; 109 131-143.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.ejps.2017.07.023
Vancouver
Carvalho VFM, Migotto A, Giacone DV, Lemos DP de, Zanoni TB, Maria-Engler SS, Lotufo LVC, Lopes LB. Co-encapsulation of paclitaxel and C6 ceramide in tributyrin-containing nanocarriers improve co-localization in the skin and potentiate cytotoxic effects in 2D and 3D models [Internet]. European Journal of Pharmaceutical Sciences. 2017 ; 109 131-143.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.ejps.2017.07.023
Artigo de periodico
Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis (2017)
Nascimento, Ivan Pereira; Rodriguez, Dúnia; Santos, Carina Carvalho dos; Amaral, Eduardo Pinheiro; Rofatto, Henrique Krambeck; Junqueira-Kipnis, Ana Paula; Gonçalves, Eduardo Dantas Casillo; Lima, Maria Regina D'Império ; Hirata, Mario Hiroyuki ; Silva, Célio Lopes; Winter, Nathalie; Gicquel, Brigitte; Mills, Kingston H G; Pizza, Mariagrazia; Rappuoli, Rino; Leite, Luciana C. C
Source: Scientific Reports. Unidades: ICB, FCF, FMRP
Subjects: IMUNOLOGIA, TUBERCULOSE, MYCOBACTERIUM TUBERCULOSIS, ADJUVANTES IMUNOLÓGICOS, PROTEÍNAS RECOMBINANTES, VACINAS, CITOCININAS, CAMUNDONGOS
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NASCIMENTO, Ivan Pereira et al. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis. Scientific Reports, v. 7, n. 1, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-02003-9. Acesso em: 17 jun. 2024.
APA
Nascimento, I. P., Rodriguez, D., Santos, C. C. dos, Amaral, E. P., Rofatto, H. K., Junqueira-Kipnis, A. P., et al. (2017). Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis. Scientific Reports, 7( 1), 1-11. doi:10.1038/s41598-017-02003-9
NLM
Nascimento IP, Rodriguez D, Santos CC dos, Amaral EP, Rofatto HK, Junqueira-Kipnis AP, Gonçalves EDC, Lima MRD'I, Hirata MH, Silva CL, Winter N, Gicquel B, Mills KHG, Pizza M, Rappuoli R, Leite LCC. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/s41598-017-02003-9
Vancouver
Nascimento IP, Rodriguez D, Santos CC dos, Amaral EP, Rofatto HK, Junqueira-Kipnis AP, Gonçalves EDC, Lima MRD'I, Hirata MH, Silva CL, Winter N, Gicquel B, Mills KHG, Pizza M, Rappuoli R, Leite LCC. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/s41598-017-02003-9
Artigo de periodico
Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88 (2017)
Castoldi, Angela; Oliveira, Vinícius Alves de; Aguiar, Cristhiane Fávero; Amano, Mariane Tami; Lee, Jennifer; Miyagi, Marcelli Terumi; Latância, Marcela Teatin; Braga, Tárcio Teodoro; Silva, Marina Burgos da; Ignacio, Aline; Lima, Joanna Darck Carola Correia; Loures, Flavio Vieira; Albuquerque, José Antônio Tavares de; Macêdo, Marina Barguil; Almeida, Rafael Ribeiro; Gaiarsa, Jonas W.; Martinez, Luis Alberto Luevano; Belchior, Thiago; Hiyane, Meire Ioshie; Brown, Gordon D; Mori, Marcelo A.; Hoffmann, Christian ; Seelaender, Marília Cerqueira Leite; Festuccia, William Tadeu Lara ; Vieira, Pedro Manoel Moraes; Câmara, Niels Olsen Saraiva
Source: Cell Reports. Unidades: ICB, FCF
Subjects: OBESIDADE, INSULINA
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CASTOLDI, Angela et al. Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88. Cell Reports, v. 19, p. 2272-2288, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2017.05.059. Acesso em: 17 jun. 2024.
APA
Castoldi, A., Oliveira, V. A. de, Aguiar, C. F., Amano, M. T., Lee, J., Miyagi, M. T., et al. (2017). Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88. Cell Reports, 19, 2272-2288. doi:10.1016/j.celrep.2017.05.059
NLM
Castoldi A, Oliveira VA de, Aguiar CF, Amano MT, Lee J, Miyagi MT, Latância MT, Braga TT, Silva MB da, Ignacio A, Lima JDCC, Loures FV, Albuquerque JAT de, Macêdo MB, Almeida RR, Gaiarsa JW, Martinez LAL, Belchior T, Hiyane MI, Brown GD, Mori MA, Hoffmann C, Seelaender MCL, Festuccia WTL, Vieira PMM, Câmara NOS. Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88 [Internet]. Cell Reports. 2017 ; 19 2272-2288.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.celrep.2017.05.059
Vancouver
Castoldi A, Oliveira VA de, Aguiar CF, Amano MT, Lee J, Miyagi MT, Latância MT, Braga TT, Silva MB da, Ignacio A, Lima JDCC, Loures FV, Albuquerque JAT de, Macêdo MB, Almeida RR, Gaiarsa JW, Martinez LAL, Belchior T, Hiyane MI, Brown GD, Mori MA, Hoffmann C, Seelaender MCL, Festuccia WTL, Vieira PMM, Câmara NOS. Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88 [Internet]. Cell Reports. 2017 ; 19 2272-2288.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.celrep.2017.05.059
Artigo de periodico
4-Nerolidylcatechol induces autophagy in human glioblastoma cells (2017)
Massaro, Renato Ramos; Brohem, Carla Abdo; Almeida, Rebeca Leite de; Rivelli, Diogo Pineda; Miyake, Juliano Andreoli; Colquhoun, Alison ; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Unidades: ICB, FCF
Assunto: HISTOLOGIA
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ABNT
MASSARO, Renato Ramos et al. 4-Nerolidylcatechol induces autophagy in human glioblastoma cells. Brazilian Journal of Pharmaceutical Sciences, v. 53, n. 3, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1590/s2175-97902017000300169. Acesso em: 17 jun. 2024.
APA
Massaro, R. R., Brohem, C. A., Almeida, R. L. de, Rivelli, D. P., Miyake, J. A., Colquhoun, A., et al. (2017). 4-Nerolidylcatechol induces autophagy in human glioblastoma cells. Brazilian Journal of Pharmaceutical Sciences, 53( 3), 1-10. doi:10.1590/s2175-97902017000300169
NLM
Massaro RR, Brohem CA, Almeida RL de, Rivelli DP, Miyake JA, Colquhoun A, Barros SB de M, Maria-Engler SS. 4-Nerolidylcatechol induces autophagy in human glioblastoma cells [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2017 ; 53( 3): 1-10.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/s2175-97902017000300169
Vancouver
Massaro RR, Brohem CA, Almeida RL de, Rivelli DP, Miyake JA, Colquhoun A, Barros SB de M, Maria-Engler SS. 4-Nerolidylcatechol induces autophagy in human glioblastoma cells [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2017 ; 53( 3): 1-10.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/s2175-97902017000300169
Artigo de periodico
Influence of CYP3A4 and CYP3A5 polymorphisms on tacrolimus and sirolimus exposure in stable kidney transplant recipients (2017)
Tamashiro, Erika Yumi; Felipe, Claudia R; Genvigir, Fabiana Dalla Vecchia ; Rodrigues, Alice Cristina ; Campos, Antony B.; Hirata, Rosario Dominguez Crespo ; Silva Junior, Helio Tedesco; Pestana, José O. Medina
Source: Drug Metabolism Personalized Theraphy. Unidade: FCF
Subjects: POLIMORFISMO, TRANSPLANTE DE RIM
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TAMASHIRO, Erika Yumi et al. Influence of CYP3A4 and CYP3A5 polymorphisms on tacrolimus and sirolimus exposure in stable kidney transplant recipients. Drug Metabolism Personalized Theraphy, v. 32, n. 2, p. 89-95, 2017Tradução . . Disponível em: https://doi.org/10.1515/dmpt-2016-0036. Acesso em: 17 jun. 2024.
APA
Tamashiro, E. Y., Felipe, C. R., Genvigir, F. D. V., Rodrigues, A. C., Campos, A. B., Hirata, R. D. C., et al. (2017). Influence of CYP3A4 and CYP3A5 polymorphisms on tacrolimus and sirolimus exposure in stable kidney transplant recipients. Drug Metabolism Personalized Theraphy, 32( 2), 89-95. doi:10.1515/dmpt-2016-0036
NLM
Tamashiro EY, Felipe CR, Genvigir FDV, Rodrigues AC, Campos AB, Hirata RDC, Silva Junior HT, Pestana JOM. Influence of CYP3A4 and CYP3A5 polymorphisms on tacrolimus and sirolimus exposure in stable kidney transplant recipients [Internet]. Drug Metabolism Personalized Theraphy. 2017 ; 32( 2): 89-95.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1515/dmpt-2016-0036
Vancouver
Tamashiro EY, Felipe CR, Genvigir FDV, Rodrigues AC, Campos AB, Hirata RDC, Silva Junior HT, Pestana JOM. Influence of CYP3A4 and CYP3A5 polymorphisms on tacrolimus and sirolimus exposure in stable kidney transplant recipients [Internet]. Drug Metabolism Personalized Theraphy. 2017 ; 32( 2): 89-95.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1515/dmpt-2016-0036
Artigo de periodico
The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation (2017)
Aquino, Sabrina G. de ; Talbot, Jhimmy; Sônego, Fabiane; Turato, Walter Miguel; Grespan, Renata; Campos, Mario Julio Avila; Cunha, Fernando de Queiroz; Cirelli, Joni Augusto
Source: Journal of Clinical Periodontology. Unidades: ICB, FCF, FMRP
Subjects: MICROBIOLOGIA, PERIODONTITE AGRESSIVA, PORPHYROMONAS GINGIVALIS, ARTRITE REUMATOIDE, DOENÇAS PERIODONTAIS, RATOS, NEUTRÓFILOS
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ABNT
AQUINO, Sabrina G. de et al. The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation. Journal of Clinical Periodontology, v. 44, n. 9, p. 881-891, 2017Tradução . . Disponível em: https://doi.org/10.1111/jcpe.1274. Acesso em: 17 jun. 2024.
APA
Aquino, S. G. de, Talbot, J., Sônego, F., Turato, W. M., Grespan, R., Campos, M. J. A., et al. (2017). The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation. Journal of Clinical Periodontology, 44( 9), 881-891. doi:10.1111/jcpe.1274
NLM
Aquino SG de, Talbot J, Sônego F, Turato WM, Grespan R, Campos MJA, Cunha F de Q, Cirelli JA. The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation [Internet]. Journal of Clinical Periodontology. 2017 ; 44( 9): 881-891.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/jcpe.1274
Vancouver
Aquino SG de, Talbot J, Sônego F, Turato WM, Grespan R, Campos MJA, Cunha F de Q, Cirelli JA. The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation [Internet]. Journal of Clinical Periodontology. 2017 ; 44( 9): 881-891.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/jcpe.1274
Artigo de periodico
TLR4-mediated placental pathology and pregnancy outcome in experimental malaria (2017)
Barboza, Renato ; Lima, Flávia Afonso ; Reis, Aramys Silva dos ; Murillo, Oscar Javier ; Peixoto, Erika Paula Machado ; Bandeira, Carla Letícia dos Santos ; Fotoran, Wesley Luzetti; Sardinha, Luiz Roberto ; Wunderlich, Gerhard ; Bevilacqua, Estela Maris Andrade Forell ; Lima, Maria Regina D'Império ; Alvarez, José Maria; Costa, Fabio Trindade Maranhão ; Gonçalves, Ligia Antunes ; Epiphanio, Sabrina ; Marinho, Claudio Romero Farias
Source: Scientific Reports. Unidades: ICB, FCF
Subjects: GRAVIDEZ, MALÁRIA, PLACENTA
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ABNT
BARBOZA, Renato et al. TLR4-mediated placental pathology and pregnancy outcome in experimental malaria. Scientific Reports, v. 7, p. 1-12 art. 8623, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-08299-x. Acesso em: 17 jun. 2024.
APA
Barboza, R., Lima, F. A., Reis, A. S. dos, Murillo, O. J., Peixoto, E. P. M., Bandeira, C. L. dos S., et al. (2017). TLR4-mediated placental pathology and pregnancy outcome in experimental malaria. Scientific Reports, 7, 1-12 art. 8623. doi:10.1038/s41598-017-08299-x
NLM
Barboza R, Lima FA, Reis AS dos, Murillo OJ, Peixoto EPM, Bandeira CL dos S, Fotoran WL, Sardinha LR, Wunderlich G, Bevilacqua EMAF, Lima MRD'I, Alvarez JM, Costa FTM, Gonçalves LA, Epiphanio S, Marinho CRF. TLR4-mediated placental pathology and pregnancy outcome in experimental malaria [Internet]. Scientific Reports. 2017 ; 7 1-12 art. 8623.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/s41598-017-08299-x
Vancouver
Barboza R, Lima FA, Reis AS dos, Murillo OJ, Peixoto EPM, Bandeira CL dos S, Fotoran WL, Sardinha LR, Wunderlich G, Bevilacqua EMAF, Lima MRD'I, Alvarez JM, Costa FTM, Gonçalves LA, Epiphanio S, Marinho CRF. TLR4-mediated placental pathology and pregnancy outcome in experimental malaria [Internet]. Scientific Reports. 2017 ; 7 1-12 art. 8623.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/s41598-017-08299-x
Artigo de periodico
Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice (2017)
Masi, Laureane Nunes; Martins, Amanda Roque; Crisma, Amanda Rabello; Amaral, Cátia Lira do; Davanso, Mariana Rodrigues; Serdan, Tamires Duarte Afonso; Sá, Roberta Dourado Cavalcante da Cunha de; Cruz, Maysa Mariana; Alonso-Vale, Maria Isabel Cardoso; Torres, Rosângela Pavan; Mancini-Filho, Jorge ; Pereira, Joice Naiara Bertaglia; Righetti, Marta Maria da Silva; Liberti, Edson Aparecido; Hirabara, Sandro Massao; Curi, Rui
Source: Scientific Reports. Unidades: ICB, FCF
Assunto: ANATOMIA
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ABNT
MASI, Laureane Nunes et al. Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice. Scientific Reports, v. 7, n. 1, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-04308-1. Acesso em: 17 jun. 2024.
APA
Masi, L. N., Martins, A. R., Crisma, A. R., Amaral, C. L. do, Davanso, M. R., Serdan, T. D. A., et al. (2017). Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice. Scientific Reports, 7( 1), 1-10. doi:10.1038/s41598-017-04308-1
NLM
Masi LN, Martins AR, Crisma AR, Amaral CL do, Davanso MR, Serdan TDA, Sá RDC da C de, Cruz MM, Alonso-Vale MIC, Torres RP, Mancini-Filho J, Pereira JNB, Righetti MM da S, Liberti EA, Hirabara SM, Curi R. Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice [Internet]. Scientific Reports. 2017 ; 7( 1): 1-10.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/s41598-017-04308-1
Vancouver
Masi LN, Martins AR, Crisma AR, Amaral CL do, Davanso MR, Serdan TDA, Sá RDC da C de, Cruz MM, Alonso-Vale MIC, Torres RP, Mancini-Filho J, Pereira JNB, Righetti MM da S, Liberti EA, Hirabara SM, Curi R. Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice [Internet]. Scientific Reports. 2017 ; 7( 1): 1-10.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/s41598-017-04308-1
Artigo de periodico
Beneficial effects of vitamin C treatment on pregnant rats exposed to formaldehyde: Reversal of immunosuppression in the offspring (2016)
Ibrahim, Beatriz Silva; Barioni, Eric Diego; Heluany, Cíntia Scucuglia; Braga, Tárcio Teodoro; Drewes, Carine Cristiane; Costa, Silvia Goes; Câmara, Niels Olsen Saraiva ; Farsky, Sandra Helena Poliselli ; Franco, Adriana Lino dos Santos
Source: Toxicology and Applied Pharmacology. Unidades: ICB, FCF
Subjects: ESTRESSE OXIDATIVO, INFLAMAÇÃO, IMUNOSSUPRESSÃO, IMUNOLOGIA
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ABNT
IBRAHIM, Beatriz Silva et al. Beneficial effects of vitamin C treatment on pregnant rats exposed to formaldehyde: Reversal of immunosuppression in the offspring. Toxicology and Applied Pharmacology, v. 300, p. 77-81, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2016.03.010. Acesso em: 17 jun. 2024.
APA
Ibrahim, B. S., Barioni, E. D., Heluany, C. S., Braga, T. T., Drewes, C. C., Costa, S. G., et al. (2016). Beneficial effects of vitamin C treatment on pregnant rats exposed to formaldehyde: Reversal of immunosuppression in the offspring. Toxicology and Applied Pharmacology, 300, 77-81. doi:10.1016/j.taap.2016.03.010
NLM
Ibrahim BS, Barioni ED, Heluany CS, Braga TT, Drewes CC, Costa SG, Câmara NOS, Farsky SHP, Franco AL dos S. Beneficial effects of vitamin C treatment on pregnant rats exposed to formaldehyde: Reversal of immunosuppression in the offspring [Internet]. Toxicology and Applied Pharmacology. 2016 ; 300 77-81.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.taap.2016.03.010
Vancouver
Ibrahim BS, Barioni ED, Heluany CS, Braga TT, Drewes CC, Costa SG, Câmara NOS, Farsky SHP, Franco AL dos S. Beneficial effects of vitamin C treatment on pregnant rats exposed to formaldehyde: Reversal of immunosuppression in the offspring [Internet]. Toxicology and Applied Pharmacology. 2016 ; 300 77-81.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.taap.2016.03.010
Artigo de periodico
The presence of T cell epitopes is important for induction of antibody responses against antigens directed to DEC205+ dendritic cells (2016)
Amorim, Kelly N. S.; Rampazo, Eline V.; Antonialli, Renan; Yamamoto, Marcio Massao; Rodrigues, Mauricio M.; Soares, Irene da Silva ; Boscardin, Silvia Beatriz
Source: Scientific Reports. Unidades: ICB, FCF
Subjects: PARASITOLOGIA, CÉLULAS DENDRÍTICAS, PLASMODIUM, EPITOPOS, ANTICORPOS MONOCLONAIS
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ABNT
AMORIM, Kelly N. S. et al. The presence of T cell epitopes is important for induction of antibody responses against antigens directed to DEC205+ dendritic cells. Scientific Reports, v. 6, p. 1-15, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep39250. Acesso em: 17 jun. 2024.
APA
Amorim, K. N. S., Rampazo, E. V., Antonialli, R., Yamamoto, M. M., Rodrigues, M. M., Soares, I. da S., & Boscardin, S. B. (2016). The presence of T cell epitopes is important for induction of antibody responses against antigens directed to DEC205+ dendritic cells. Scientific Reports, 6, 1-15. doi:10.1038/srep39250
NLM
Amorim KNS, Rampazo EV, Antonialli R, Yamamoto MM, Rodrigues MM, Soares I da S, Boscardin SB. The presence of T cell epitopes is important for induction of antibody responses against antigens directed to DEC205+ dendritic cells [Internet]. Scientific Reports. 2016 ; 6 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/srep39250
Vancouver
Amorim KNS, Rampazo EV, Antonialli R, Yamamoto MM, Rodrigues MM, Soares I da S, Boscardin SB. The presence of T cell epitopes is important for induction of antibody responses against antigens directed to DEC205+ dendritic cells [Internet]. Scientific Reports. 2016 ; 6 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1038/srep39250
Artigo de periodico
Atorvastatin attenuation of ABCB1 expression is mediated by microRNA miR-491-3p in Caco-2 cells (2016)
Rodrigues, Alice Cristina ; Neri, Elida Adalgisa; Veríssimo Filho, Sidney; Rebouças, Nancy Amaral; Hirata, Rosario Dominguez Crespo ; Yu, Ai Ming
Source: European Journal of Pharmaceutical Sciences. Unidades: ICB, FCF
Subjects: HIPERCOLESTEROLEMIA, EXPRESSÃO GÊNICA, RNA MENSAGEIRO, FISIOLOGIA
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ABNT
RODRIGUES, Alice Cristina et al. Atorvastatin attenuation of ABCB1 expression is mediated by microRNA miR-491-3p in Caco-2 cells. European Journal of Pharmaceutical Sciences, v. 93, p. 431-436, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejps.2016.08.044. Acesso em: 17 jun. 2024.
APA
Rodrigues, A. C., Neri, E. A., Veríssimo Filho, S., Rebouças, N. A., Hirata, R. D. C., & Yu, A. M. (2016). Atorvastatin attenuation of ABCB1 expression is mediated by microRNA miR-491-3p in Caco-2 cells. European Journal of Pharmaceutical Sciences, 93, 431-436. doi:10.1016/j.ejps.2016.08.044
NLM
Rodrigues AC, Neri EA, Veríssimo Filho S, Rebouças NA, Hirata RDC, Yu AM. Atorvastatin attenuation of ABCB1 expression is mediated by microRNA miR-491-3p in Caco-2 cells [Internet]. European Journal of Pharmaceutical Sciences. 2016 ; 93 431-436.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.ejps.2016.08.044
Vancouver
Rodrigues AC, Neri EA, Veríssimo Filho S, Rebouças NA, Hirata RDC, Yu AM. Atorvastatin attenuation of ABCB1 expression is mediated by microRNA miR-491-3p in Caco-2 cells [Internet]. European Journal of Pharmaceutical Sciences. 2016 ; 93 431-436.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.ejps.2016.08.044
Artigo de periodico
Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients (2016)
Bastos, Alliny de Souza; Graves, Dana T; Loureiro, Ana Paula de Melo ; Rossa Junior, Carlos; Corbi, Sâmia Cruz Tfaile; Frizzera, Fausto; Caminaga, Raquel Mantuaneli Scarel; Câmara, Niels Olsen Saraiva ; Andriankaja, Oelisoa M; Hiyane, Meire Ioshie; Orrico, Silvana Regina Perez
Source: Journal of Diabetes and Its Complications. Unidades: FCF, ICB
Subjects: PEROXIDASE, DIABETES MELLITUS NÃO INSULINO-DEPENDENTE, CITOCINAS, DISLIPIDEMIAS, INFLAMAÇÃO
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ABNT
BASTOS, Alliny de Souza et al. Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients. Journal of Diabetes and Its Complications, v. 30, n. 8, p. 1593–1599, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2016.07.011. Acesso em: 17 jun. 2024.
APA
Bastos, A. de S., Graves, D. T., Loureiro, A. P. de M., Rossa Junior, C., Corbi, S. C. T., Frizzera, F., et al. (2016). Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients. Journal of Diabetes and Its Complications, 30( 8), 1593–1599. doi:10.1016/j.jdiacomp.2016.07.011
NLM
Bastos A de S, Graves DT, Loureiro AP de M, Rossa Junior C, Corbi SCT, Frizzera F, Caminaga RMS, Câmara NOS, Andriankaja OM, Hiyane MI, Orrico SRP. Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients [Internet]. Journal of Diabetes and Its Complications. 2016 ; 30( 8): 1593–1599.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.07.011
Vancouver
Bastos A de S, Graves DT, Loureiro AP de M, Rossa Junior C, Corbi SCT, Frizzera F, Caminaga RMS, Câmara NOS, Andriankaja OM, Hiyane MI, Orrico SRP. Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients [Internet]. Journal of Diabetes and Its Complications. 2016 ; 30( 8): 1593–1599.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.07.011
Artigo de periodico
Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats (2016)
Rodrigues, Hosana Gomes; Vinolo, Marco Aurélio Ramirez; Sato, Fabio Takeo; Magdalon, Juliana; Kuhl, Carolina Meneguetti Cintra; Yamagata, Ana Sayuri; Pessoa, Ana Flávia Marçal; Moraes, Gabriélla Malheiros; Santos, Marinilce Fagundes dos ; Lima, Camila; Farsky, Sandra Helena Poliselli ; Câmara, Niels Olsen Saraiva ; Williner, Maria Rosa; Bernal, Claudio Adrián; Calder,Philip C; Curi, Rui
Source: PLOS ONE. Unidades: ICB, FCF
Subjects: ÁCIDO LINOLEICO, RATOS, CICATRIZAÇÃO
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ABNT
RODRIGUES, Hosana Gomes et al. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats. PLOS ONE, v. 11, n. 10, p. e0165115, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0165115. Acesso em: 17 jun. 2024.
APA
Rodrigues, H. G., Vinolo, M. A. R., Sato, F. T., Magdalon, J., Kuhl, C. M. C., Yamagata, A. S., et al. (2016). Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats. PLOS ONE, 11( 10), e0165115. doi:10.1371/journal.pone.0165115
NLM
Rodrigues HG, Vinolo MAR, Sato FT, Magdalon J, Kuhl CMC, Yamagata AS, Pessoa AFM, Moraes GM, Santos MF dos, Lima C, Farsky SHP, Câmara NOS, Williner MR, Bernal CA, Calder PC, Curi R. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats [Internet]. PLOS ONE. 2016 ; 11( 10): e0165115.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1371/journal.pone.0165115
Vancouver
Rodrigues HG, Vinolo MAR, Sato FT, Magdalon J, Kuhl CMC, Yamagata AS, Pessoa AFM, Moraes GM, Santos MF dos, Lima C, Farsky SHP, Câmara NOS, Williner MR, Bernal CA, Calder PC, Curi R. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats [Internet]. PLOS ONE. 2016 ; 11( 10): e0165115.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1371/journal.pone.0165115
Artigo de periodico
BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity (2016)
Ferreira, Adilson Kleber; Pasqualoto, Kerly Fernanda Mesquita; Kruyt, Frank A. E; Berl, Fanny Palace; Azevedo, Ricardo Alexandre de; Turra, Kely Medeiros; Rodrigues, Cecília Pessoa; Ferreira, Ana Carolina Franco; Salomon, Maria Alejandra Clavijo; Sá Júnior, Paulo Luiz de; Farias, Camyla Fernandes; Figueiredo, Carlos Rogério; Tavares, Leoberto Costa ; Barbuto, José Alexandre Marzagão ; Jorge, Salomão Dória
Source: Toxicology and Applied Pharmacology. Unidades: FCF, ICB
Subjects: MELANOMA, METÁSTASE NEOPLÁSICA, APOPTOSE
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ABNT
FERREIRA, Adilson Kleber et al. BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity. Toxicology and Applied Pharmacology, v. 295, p. 56-67, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2016.02.008. Acesso em: 17 jun. 2024.
APA
Ferreira, A. K., Pasqualoto, K. F. M., Kruyt, F. A. E., Berl, F. P., Azevedo, R. A. de, Turra, K. M., et al. (2016). BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity. Toxicology and Applied Pharmacology, 295, 56-67. doi:10.1016/j.taap.2016.02.008
NLM
Ferreira AK, Pasqualoto KFM, Kruyt FAE, Berl FP, Azevedo RA de, Turra KM, Rodrigues CP, Ferreira ACF, Salomon MAC, Sá Júnior PL de, Farias CF, Figueiredo CR, Tavares LC, Barbuto JAM, Jorge SD. BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity [Internet]. Toxicology and Applied Pharmacology. 2016 ; 295 56-67.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.taap.2016.02.008
Vancouver
Ferreira AK, Pasqualoto KFM, Kruyt FAE, Berl FP, Azevedo RA de, Turra KM, Rodrigues CP, Ferreira ACF, Salomon MAC, Sá Júnior PL de, Farias CF, Figueiredo CR, Tavares LC, Barbuto JAM, Jorge SD. BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity [Internet]. Toxicology and Applied Pharmacology. 2016 ; 295 56-67.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.taap.2016.02.008
Artigo de periodico
Photobiomodulation therapy decreases oxidative stress in the lung tissue after formaldehyde exposure: role of oxidant/antioxidant enzymes (2016)
Macedo, Rodrigo Silva; Leal, Mayara Peres; Braga, Tárcio Teodoro; Barioni, Éric Diego; Duro, Stephanie de Oliveira ; Horliana, Anna Carolina Ratto Tempestini; Câmara, Niels Olsen Saraiva ; Marcourakis, Tania ; Farsky, Sandra Helena Poliselli ; Franco, Adriana Lino dos Santos
Source: Mediators of Inflammation. Unidades: FCF, ICB
Subjects: ESTRESSE OXIDATIVO, ANTIOXIDANTES, ENZIMAS
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ABNT
MACEDO, Rodrigo Silva et al. Photobiomodulation therapy decreases oxidative stress in the lung tissue after formaldehyde exposure: role of oxidant/antioxidant enzymes. Mediators of Inflammation, v. 2016, p. 1-9 art. 9303126, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/9303126. Acesso em: 17 jun. 2024.
APA
Macedo, R. S., Leal, M. P., Braga, T. T., Barioni, É. D., Duro, S. de O., Horliana, A. C. R. T., et al. (2016). Photobiomodulation therapy decreases oxidative stress in the lung tissue after formaldehyde exposure: role of oxidant/antioxidant enzymes. Mediators of Inflammation, 2016, 1-9 art. 9303126. doi:10.1155/2016/9303126
NLM
Macedo RS, Leal MP, Braga TT, Barioni ÉD, Duro S de O, Horliana ACRT, Câmara NOS, Marcourakis T, Farsky SHP, Franco AL dos S. Photobiomodulation therapy decreases oxidative stress in the lung tissue after formaldehyde exposure: role of oxidant/antioxidant enzymes [Internet]. Mediators of Inflammation. 2016 ; 2016 1-9 art. 9303126.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1155/2016/9303126
Vancouver
Macedo RS, Leal MP, Braga TT, Barioni ÉD, Duro S de O, Horliana ACRT, Câmara NOS, Marcourakis T, Farsky SHP, Franco AL dos S. Photobiomodulation therapy decreases oxidative stress in the lung tissue after formaldehyde exposure: role of oxidant/antioxidant enzymes [Internet]. Mediators of Inflammation. 2016 ; 2016 1-9 art. 9303126.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1155/2016/9303126

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