Filtros : "Indexado na Base de Dados MEDLINE" "Kawamoto, Elisa Mitiko" "ICB" Removidos: "Fisisologia Humana" "Indexado na Base de dados Scopus" "Harvard University (HU)" "Ferro, Emer Suavinho" "bb" "Áustria" "2023" "FCF" Limpar

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  • Fonte: Ageing Research Reviews. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      CARARO-LOPES, Marina Minto et al. The relevance of α-KLOTHO to the central nervous system: some key questions. Ageing Research Reviews, v. 36, p. 137-148, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2017.03.003. Acesso em: 14 out. 2024.
    • APA

      Cararo-Lopes, M. M., Mazucanti, C. H. Y., Scavone, C., Kawamoto, E. M., & Berwick, D. C. (2017). The relevance of α-KLOTHO to the central nervous system: some key questions. Ageing Research Reviews, 36, 137-148. doi:10.1016/j.arr.2017.03.003
    • NLM

      Cararo-Lopes MM, Mazucanti CHY, Scavone C, Kawamoto EM, Berwick DC. The relevance of α-KLOTHO to the central nervous system: some key questions [Internet]. Ageing Research Reviews. 2017 ; 36 137-148.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.arr.2017.03.003
    • Vancouver

      Cararo-Lopes MM, Mazucanti CHY, Scavone C, Kawamoto EM, Berwick DC. The relevance of α-KLOTHO to the central nervous system: some key questions [Internet]. Ageing Research Reviews. 2017 ; 36 137-148.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.arr.2017.03.003
  • Fonte: Scientific Reports. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      KINOSHITA, Paula F. et al. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells. Scientific Reports, v. 7, n. 1, p. 4894, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-05075-9. Acesso em: 14 out. 2024.
    • APA

      Kinoshita, P. F., Yshii, L. M., Orellana, A. M. M., Paixão, A. G., Vasconcelos, A. R., Lima, L. de S., et al. (2017). Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells. Scientific Reports, 7( 1), 4894. doi:10.1038/s41598-017-05075-9
    • NLM

      Kinoshita PF, Yshii LM, Orellana AMM, Paixão AG, Vasconcelos AR, Lima L de S, Kawamoto EM, Scavone C. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells [Internet]. Scientific Reports. 2017 ; 7( 1): 4894.[citado 2024 out. 14 ] Available from: https://doi.org/10.1038/s41598-017-05075-9
    • Vancouver

      Kinoshita PF, Yshii LM, Orellana AMM, Paixão AG, Vasconcelos AR, Lima L de S, Kawamoto EM, Scavone C. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells [Internet]. Scientific Reports. 2017 ; 7( 1): 4894.[citado 2024 out. 14 ] Available from: https://doi.org/10.1038/s41598-017-05075-9
  • Fonte: Aging. Unidade: ICB

    Assuntos: FARMACOLOGIA, RATOS, HIPOCAMPU DE ANIMAL

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    • ABNT

      ORELLANA, Ana Maria Marques et al. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus. Aging, v. 7, n. 12, p. 1094-1111, 2015Tradução . . Disponível em: https://doi.org/10.18632/aging.100853. Acesso em: 14 out. 2024.
    • APA

      Orellana, A. M. M., Vasconcelos, A. R., Leite, J. A., Lima, L. de S., Andreotti, D. Z., Munhoz, C. D., et al. (2015). Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus. Aging, 7( 12), 1094-1111. doi:10.18632/aging.100853
    • NLM

      Orellana AMM, Vasconcelos AR, Leite JA, Lima L de S, Andreotti DZ, Munhoz CD, Kawamoto EM, Scavone C. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus [Internet]. Aging. 2015 ; 7( 12): 1094-1111.[citado 2024 out. 14 ] Available from: https://doi.org/10.18632/aging.100853
    • Vancouver

      Orellana AMM, Vasconcelos AR, Leite JA, Lima L de S, Andreotti DZ, Munhoz CD, Kawamoto EM, Scavone C. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus [Internet]. Aging. 2015 ; 7( 12): 1094-1111.[citado 2024 out. 14 ] Available from: https://doi.org/10.18632/aging.100853
  • Fonte: Current Topics in Medicinal Chemistry. Unidade: ICB

    Assuntos: FARMACOLOGIA, ENVELHECIMENTO, INSULINA, DEGENERAÇÃO NEURAL

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    • ABNT

      MAZUCANTI, Caio Henrique et al. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration. Current Topics in Medicinal Chemistry, v. 15, n. 21, p. 2116-2138, 2015Tradução . . Disponível em: https://doi.org/10.2174/1568026615666150610125715. Acesso em: 14 out. 2024.
    • APA

      Mazucanti, C. H., Cabral-Costa, J. V., Vasconcelos, A. R., Andreotti, D. Z., Scavone, C., & Kawamoto, E. M. (2015). Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration. Current Topics in Medicinal Chemistry, 15( 21), 2116-2138. doi:10.2174/1568026615666150610125715
    • NLM

      Mazucanti CH, Cabral-Costa JV, Vasconcelos AR, Andreotti DZ, Scavone C, Kawamoto EM. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration [Internet]. Current Topics in Medicinal Chemistry. 2015 ; 15( 21): 2116-2138.[citado 2024 out. 14 ] Available from: https://doi.org/10.2174/1568026615666150610125715
    • Vancouver

      Mazucanti CH, Cabral-Costa JV, Vasconcelos AR, Andreotti DZ, Scavone C, Kawamoto EM. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration [Internet]. Current Topics in Medicinal Chemistry. 2015 ; 15( 21): 2116-2138.[citado 2024 out. 14 ] Available from: https://doi.org/10.2174/1568026615666150610125715
  • Fonte: Journal of Neuroinflammation. Unidades: ICB, EACH

    Assunto: FARMACOLOGIA

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    • ABNT

      VASCONCELOS, Andrea Rodrigues et al. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, v. 11, n. 85, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-85. Acesso em: 14 out. 2024.
    • APA

      Vasconcelos, A. R., Yshii, L. M., Viel, T. A., Buck, H. S., Mattson, M. P., Scavone, C., & Kawamoto, E. M. (2014). Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, 11( 85), 1-14. doi:10.1186/1742-2094-11-85
    • NLM

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 out. 14 ] Available from: https://doi.org/10.1186/1742-2094-11-85
    • Vancouver

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 out. 14 ] Available from: https://doi.org/10.1186/1742-2094-11-85
  • Fonte: International Journal of Immunopathology and Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      TZANNO-MARTINS, C. et al. Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology, v. 27, n. 1, p. 25-35, 2014Tradução . . Acesso em: 14 out. 2024.
    • APA

      Tzanno-Martins, C., Biavo, B. M. M., Ferreira-Filho, O., Ribeiro-Junior, E., João-Luiz, M. V. de S., Degaspari, S., et al. (2014). Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology, 27( 1), 25-35.
    • NLM

      Tzanno-Martins C, Biavo BMM, Ferreira-Filho O, Ribeiro-Junior E, João-Luiz MV de S, Degaspari S, Scavone C, Kawamoto EM. Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology. 2014 ; 27( 1): 25-35.[citado 2024 out. 14 ]
    • Vancouver

      Tzanno-Martins C, Biavo BMM, Ferreira-Filho O, Ribeiro-Junior E, João-Luiz MV de S, Degaspari S, Scavone C, Kawamoto EM. Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology. 2014 ; 27( 1): 25-35.[citado 2024 out. 14 ]
  • Fonte: Journal of Neuroinflammation. Unidade: ICB

    Assuntos: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO

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    • ABNT

      KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 14 out. 2024.
    • APA

      Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
    • NLM

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 out. 14 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
    • Vancouver

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 out. 14 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
  • Fonte: Neuropharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 14 out. 2024.
    • APA

      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 out. 14 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
  • Fonte: Neurosignals. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      KAWAMOTO, Elisa Mitiko et al. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neurosignals, v. 21, n. 1-2, p. 75-88, 2013Tradução . . Disponível em: https://doi.org/10.1159/000336074. Acesso em: 14 out. 2024.
    • APA

      Kawamoto, E. M., Scavone, C., Mattson, M. P., & Camandola, S. (2013). Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neurosignals, 21( 1-2), 75-88. doi:10.1159/000336074
    • NLM

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neurosignals. 2013 ; 21( 1-2): 75-88.[citado 2024 out. 14 ] Available from: https://doi.org/10.1159/000336074
    • Vancouver

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neurosignals. 2013 ; 21( 1-2): 75-88.[citado 2024 out. 14 ] Available from: https://doi.org/10.1159/000336074
  • Fonte: Neuro-Signals. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      KAWAMOTO, Elisa Mitiko et al. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neuro-Signals, v. 21, n. 1-2, p. 75-88, 2012Tradução . . Disponível em: https://doi.org/10.1159/000336074. Acesso em: 14 out. 2024.
    • APA

      Kawamoto, E. M., Scavone, C., Mattson, M. P., & Camandola, S. (2012). Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neuro-Signals, 21( 1-2), 75-88. doi:10.1159/000336074
    • NLM

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neuro-Signals. 2012 ; 21( 1-2): 75-88.[citado 2024 out. 14 ] Available from: https://doi.org/10.1159/000336074
    • Vancouver

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neuro-Signals. 2012 ; 21( 1-2): 75-88.[citado 2024 out. 14 ] Available from: https://doi.org/10.1159/000336074

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