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Artigo de periodico
The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease (2017)
Garcia, P. C.; Real, C. C.; Britto, Luiz Roberto Giorgetti de
Source: Journal of Molecular Neuroscience. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
GARCIA, P. C. e REAL, C. C. e BRITTO, Luiz Roberto Giorgetti de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, v. 61, n. 4, p. 542-552, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-017-0896-y. Acesso em: 17 jun. 2024.
APA
Garcia, P. C., Real, C. C., & Britto, L. R. G. de. (2017). The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, 61( 4), 542-552. doi:10.1007/s12031-017-0896-y
NLM
Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
Vancouver
Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
Artigo de periodico
Cardiovascular dysfunction associated with neurodegeneration in an experimental model of Parkinson's disease (2017)
Falquetto, Bárbara ; Tuppy, Marina; Potje, Simone R.; Moreira, Thiago dos Santos ; Antoniali, Cristina; Takakura, Ana C.
Source: Brain Research. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
FALQUETTO, Bárbara et al. Cardiovascular dysfunction associated with neurodegeneration in an experimental model of Parkinson's disease. Brain Research, v. 1627, p. 156-166, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2016.12.008. Acesso em: 17 jun. 2024.
APA
Falquetto, B., Tuppy, M., Potje, S. R., Moreira, T. dos S., Antoniali, C., & Takakura, A. C. (2017). Cardiovascular dysfunction associated with neurodegeneration in an experimental model of Parkinson's disease. Brain Research, 1627, 156-166. doi:10.1016/j.brainres.2016.12.008
NLM
Falquetto B, Tuppy M, Potje SR, Moreira T dos S, Antoniali C, Takakura AC. Cardiovascular dysfunction associated with neurodegeneration in an experimental model of Parkinson's disease [Internet]. Brain Research. 2017 ; 1627 156-166.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.brainres.2016.12.008
Vancouver
Falquetto B, Tuppy M, Potje SR, Moreira T dos S, Antoniali C, Takakura AC. Cardiovascular dysfunction associated with neurodegeneration in an experimental model of Parkinson's disease [Internet]. Brain Research. 2017 ; 1627 156-166.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.brainres.2016.12.008
Artigo de periodico
Regulation of adiposity by mTORC1 (2017)
Magdalon, Juliana; Festuccia, William Tadeu Lara
Source: Einstein. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MAGDALON, Juliana e FESTUCCIA, William Tadeu Lara. Regulation of adiposity by mTORC1. Einstein, v. 15, n. 4, p. 507-511, 2017Tradução . . Disponível em: https://doi.org/10.1590/S1679-45082017RB4106. Acesso em: 17 jun. 2024.
APA
Magdalon, J., & Festuccia, W. T. L. (2017). Regulation of adiposity by mTORC1. Einstein, 15( 4), 507-511. doi:10.1590/S1679-45082017RB4106
NLM
Magdalon J, Festuccia WTL. Regulation of adiposity by mTORC1 [Internet]. Einstein. 2017 ; 15( 4): 507-511.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/S1679-45082017RB4106
Vancouver
Magdalon J, Festuccia WTL. Regulation of adiposity by mTORC1 [Internet]. Einstein. 2017 ; 15( 4): 507-511.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/S1679-45082017RB4106
Artigo de periodico
PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction (2017)
Garcia-Galiano, David; Borges, Beatriz C.; Donato Junior, José ; Allen, Susan J.; Bellefontaine, Nicole; Wang, Mengjie; Zhao, Jean J.; Kozloff, Kenneth M.; Hill, Jennifer W.; Elias, Carol F.
Source: Journal of Clinical Investigation Insight - JCI insight. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
GARCIA-GALIANO, David et al. PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction. Journal of Clinical Investigation Insight - JCI insight, v. 2, n. 23, p. 1-19, 2017Tradução . . Disponível em: https://doi.org/10.1172/jci.insight.96728. Acesso em: 17 jun. 2024.
APA
Garcia-Galiano, D., Borges, B. C., Donato Junior, J., Allen, S. J., Bellefontaine, N., Wang, M., et al. (2017). PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction. Journal of Clinical Investigation Insight - JCI insight, 2( 23), 1-19. doi:10.1172/jci.insight.96728
NLM
Garcia-Galiano D, Borges BC, Donato Junior J, Allen SJ, Bellefontaine N, Wang M, Zhao JJ, Kozloff KM, Hill JW, Elias CF. PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction [Internet]. Journal of Clinical Investigation Insight - JCI insight. 2017 ; 2( 23): 1-19.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1172/jci.insight.96728
Vancouver
Garcia-Galiano D, Borges BC, Donato Junior J, Allen SJ, Bellefontaine N, Wang M, Zhao JJ, Kozloff KM, Hill JW, Elias CF. PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction [Internet]. Journal of Clinical Investigation Insight - JCI insight. 2017 ; 2( 23): 1-19.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1172/jci.insight.96728
Artigo de periodico-carta/editorial
IUPS 38th world congress in the rhythms of life (2017)
Machado, Benedito Honório ; Antunes, Vagner Roberto
Source: Physiology. Unidades: FMRP, ICB
Subjects: FISIOLOGIA, RITMO CIRCADIANO, RITMOS BIOLÓGICOS, CRONOBIOLOGIA
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ABNT
MACHADO, Benedito Honório e ANTUNES, Vagner Roberto. IUPS 38th world congress in the rhythms of life. Physiology. Rockville: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1152/physiol.00001.2017. Acesso em: 17 jun. 2024. , 2017
APA
Machado, B. H., & Antunes, V. R. (2017). IUPS 38th world congress in the rhythms of life. Physiology. Rockville: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1152/physiol.00001.2017
NLM
Machado BH, Antunes VR. IUPS 38th world congress in the rhythms of life [Internet]. Physiology. 2017 ; 32( 2): 94-95.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/physiol.00001.2017
Vancouver
Machado BH, Antunes VR. IUPS 38th world congress in the rhythms of life [Internet]. Physiology. 2017 ; 32( 2): 94-95.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/physiol.00001.2017
Artigo de periodico
Iodide excess regulates its own efflux: a possible involvement of pendrin (2016)
Calil-Silveira, Jamile; Serrano-Nascimento, Caroline; Kopp, Peter Andreas; Nunes, Maria Tereza
Source: American Journal of Physiology. Cell Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CALIL-SILVEIRA, Jamile et al. Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, v. 310, n. 7, p. C576-C582, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00210.2015. Acesso em: 17 jun. 2024.
APA
Calil-Silveira, J., Serrano-Nascimento, C., Kopp, P. A., & Nunes, M. T. (2016). Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, 310( 7), C576-C582. doi:10.1152/ajpcell.00210.2015
NLM
Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
Vancouver
Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
Artigo de periodico
Renovascular remodeling and renal injury after extended angiotensin II infusion (2016)
Casare, Fernando Augusto Malavazzi; Thieme, Karina ; Costa-Pessoa, Juliana Martins; Rossoni, Luciana Venturini ; Couto, Gisele Kruger; Fernandes, Fernanda Barrinha; Casarini, Dulce Elena; Oliveira-Souza, Maria
Source: American Journal of Physiology. Renal Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CASARE, Fernando Augusto Malavazzi et al. Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, v. 310, n. 11, p. F1295-F1307, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00471.2015. Acesso em: 17 jun. 2024.
APA
Casare, F. A. M., Thieme, K., Costa-Pessoa, J. M., Rossoni, L. V., Couto, G. K., Fernandes, F. B., et al. (2016). Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, 310( 11), F1295-F1307. doi:10.1152/ajprenal.00471.2015
NLM
Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
Vancouver
Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
Artigo de periodico-carta/editorial
Low carbohydrate high fat diets: when models do not match reality (2016)
Caminhotto, Rennan de Oliveira ; Lima, Fabio Bessa
Source: Archives of Endocrinology and Metabolism. Unidade: ICB
Subjects: FISIOLOGIA, DIETA ANIMAL, CARBOIDRATOS NA DIETA, RESISTÊNCIA À INSULINA, OBESIDADE, GLICOSE, MODELOS ANIMAIS, METABOLISMO ANIMAL, METABOLISMO DE GORDURA
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ABNT
CAMINHOTTO, Rennan de Oliveira e LIMA, Fabio Bessa. Low carbohydrate high fat diets: when models do not match reality. Archives of Endocrinology and Metabolism. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1590/2359-3997000000177. Acesso em: 17 jun. 2024. , 2016
APA
Caminhotto, R. de O., & Lima, F. B. (2016). Low carbohydrate high fat diets: when models do not match reality. Archives of Endocrinology and Metabolism. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1590/2359-3997000000177
NLM
Caminhotto R de O, Lima FB. Low carbohydrate high fat diets: when models do not match reality [Internet]. Archives of Endocrinology and Metabolism. 2016 ; 60( 4): 405-406.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/2359-3997000000177
Vancouver
Caminhotto R de O, Lima FB. Low carbohydrate high fat diets: when models do not match reality [Internet]. Archives of Endocrinology and Metabolism. 2016 ; 60( 4): 405-406.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/2359-3997000000177
Artigo de periodico
Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil (2016)
Café-Mendes, Cecília C.; Righi, Luana L.; Calil-Silveira, Jamile; Nunes, Maria Tereza ; Abdulkader, Fernando Rodrigues de Moraes
Source: Advances in Physiology Education. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CAFÉ-MENDES, Cecília C. et al. Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil. Advances in Physiology Education, v. 40, n. 4, p. 491-498, 2016Tradução . . Disponível em: https://doi.org/10.1152/advan.00031.2016. Acesso em: 17 jun. 2024.
APA
Café-Mendes, C. C., Righi, L. L., Calil-Silveira, J., Nunes, M. T., & Abdulkader, F. R. de M. (2016). Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil. Advances in Physiology Education, 40( 4), 491-498. doi:10.1152/advan.00031.2016
NLM
Café-Mendes CC, Righi LL, Calil-Silveira J, Nunes MT, Abdulkader FR de M. Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil [Internet]. Advances in Physiology Education. 2016 ; 40( 4): 491-498.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/advan.00031.2016
Vancouver
Café-Mendes CC, Righi LL, Calil-Silveira J, Nunes MT, Abdulkader FR de M. Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil [Internet]. Advances in Physiology Education. 2016 ; 40( 4): 491-498.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/advan.00031.2016
Artigo de periodico
Peroxisome proliferator-activated receptor γ activation favours selective subcutaneous lipid deposition by coordinately regulating lipoprotein lipase modulators, fatty acid transporters and lipogenic enzymes (2016)
Blanchard, P. G.; Turcotte, V.; Côté, M.; Gélinas, Y.; Nilsson, S.; Olivecrona, G.; Deshaies, Y.; Festuccia, William Tadeu Lara
Source: Acta Physiologica. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BLANCHARD, P. G. et al. Peroxisome proliferator-activated receptor γ activation favours selective subcutaneous lipid deposition by coordinately regulating lipoprotein lipase modulators, fatty acid transporters and lipogenic enzymes. Acta Physiologica, v. 217, n. 3, p. 227-239, 2016Tradução . . Disponível em: https://doi.org/10.1111/apha.12665. Acesso em: 17 jun. 2024.
APA
Blanchard, P. G., Turcotte, V., Côté, M., Gélinas, Y., Nilsson, S., Olivecrona, G., et al. (2016). Peroxisome proliferator-activated receptor γ activation favours selective subcutaneous lipid deposition by coordinately regulating lipoprotein lipase modulators, fatty acid transporters and lipogenic enzymes. Acta Physiologica, 217( 3), 227-239. doi:10.1111/apha.12665
NLM
Blanchard PG, Turcotte V, Côté M, Gélinas Y, Nilsson S, Olivecrona G, Deshaies Y, Festuccia WTL. Peroxisome proliferator-activated receptor γ activation favours selective subcutaneous lipid deposition by coordinately regulating lipoprotein lipase modulators, fatty acid transporters and lipogenic enzymes [Internet]. Acta Physiologica. 2016 ; 217( 3): 227-239.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/apha.12665
Vancouver
Blanchard PG, Turcotte V, Côté M, Gélinas Y, Nilsson S, Olivecrona G, Deshaies Y, Festuccia WTL. Peroxisome proliferator-activated receptor γ activation favours selective subcutaneous lipid deposition by coordinately regulating lipoprotein lipase modulators, fatty acid transporters and lipogenic enzymes [Internet]. Acta Physiologica. 2016 ; 217( 3): 227-239.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/apha.12665
Artigo de periodico
Interactions between prolactin and kisspeptin to control reproduction (2016)
Donato Junior, José ; Frazão, Renata
Source: Archives of Endocrinology and Metabolism. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
DONATO JUNIOR, José e FRAZÃO, Renata. Interactions between prolactin and kisspeptin to control reproduction. Archives of Endocrinology and Metabolism, v. 60, n. 6, p. 587-595, 2016Tradução . . Disponível em: https://doi.org/10.1590/2359-3997000000230. Acesso em: 17 jun. 2024.
APA
Donato Junior, J., & Frazão, R. (2016). Interactions between prolactin and kisspeptin to control reproduction. Archives of Endocrinology and Metabolism, 60( 6), 587-595. doi:10.1590/2359-3997000000230
NLM
Donato Junior J, Frazão R. Interactions between prolactin and kisspeptin to control reproduction [Internet]. Archives of Endocrinology and Metabolism. 2016 ; 60( 6): 587-595.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/2359-3997000000230
Vancouver
Donato Junior J, Frazão R. Interactions between prolactin and kisspeptin to control reproduction [Internet]. Archives of Endocrinology and Metabolism. 2016 ; 60( 6): 587-595.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1590/2359-3997000000230
Artigo de periodico
High-fat diet-induced hypertension and autonomic imbalance are associated with an upregulation of CART in the dorsomedial hypothalamus of mice (2016)
Chaar, Laiali J.; Coelho, Aline; Silva, Natalia M.; Festuccia, William Tadeu Lara ; Antunes, Vagner Roberto
Source: Physiological Reports. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CHAAR, Laiali J. et al. High-fat diet-induced hypertension and autonomic imbalance are associated with an upregulation of CART in the dorsomedial hypothalamus of mice. Physiological Reports, v. 4, n. 11, p. 1-15, 2016Tradução . . Disponível em: https://doi.org/10.14814/phy2.12811. Acesso em: 17 jun. 2024.
APA
Chaar, L. J., Coelho, A., Silva, N. M., Festuccia, W. T. L., & Antunes, V. R. (2016). High-fat diet-induced hypertension and autonomic imbalance are associated with an upregulation of CART in the dorsomedial hypothalamus of mice. Physiological Reports, 4( 11), 1-15. doi:10.14814/phy2.12811
NLM
Chaar LJ, Coelho A, Silva NM, Festuccia WTL, Antunes VR. High-fat diet-induced hypertension and autonomic imbalance are associated with an upregulation of CART in the dorsomedial hypothalamus of mice [Internet]. Physiological Reports. 2016 ; 4( 11): 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.14814/phy2.12811
Vancouver
Chaar LJ, Coelho A, Silva NM, Festuccia WTL, Antunes VR. High-fat diet-induced hypertension and autonomic imbalance are associated with an upregulation of CART in the dorsomedial hypothalamus of mice [Internet]. Physiological Reports. 2016 ; 4( 11): 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.14814/phy2.12811
Artigo de periodico
The retrotrapezoid nucleus as a central brainstem area for central and peripheral chemoreceptor interactions (2016)
Takakura, Ana C.; Moreira, Thiago dos Santos
Source: Experimental Physiology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
TAKAKURA, Ana C. e MOREIRA, Thiago dos Santos. The retrotrapezoid nucleus as a central brainstem area for central and peripheral chemoreceptor interactions. Experimental Physiology, v. 101, n. 4, p. 455-456, 2016Tradução . . Disponível em: https://doi.org/10.1113/EP085615. Acesso em: 17 jun. 2024.
APA
Takakura, A. C., & Moreira, T. dos S. (2016). The retrotrapezoid nucleus as a central brainstem area for central and peripheral chemoreceptor interactions. Experimental Physiology, 101( 4), 455-456. doi:10.1113/EP085615
NLM
Takakura AC, Moreira T dos S. The retrotrapezoid nucleus as a central brainstem area for central and peripheral chemoreceptor interactions [Internet]. Experimental Physiology. 2016 ; 101( 4): 455-456.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1113/EP085615
Vancouver
Takakura AC, Moreira T dos S. The retrotrapezoid nucleus as a central brainstem area for central and peripheral chemoreceptor interactions [Internet]. Experimental Physiology. 2016 ; 101( 4): 455-456.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1113/EP085615
Artigo de periodico
Purinergic receptor blockade in the retrotrapezoid nucleus attenuates the respiratory chemoreflexes in awake rats (2016)
Falquetto, Bárbara ; Takakura, A. C.; Mulkey, D. K.; Moreira, Thiago dos Santos
Source: Acta Physiologica. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FALQUETTO, Bárbara et al. Purinergic receptor blockade in the retrotrapezoid nucleus attenuates the respiratory chemoreflexes in awake rats. Acta Physiologica, v. 217, n. 1, p. 80-93, 2016Tradução . . Disponível em: https://doi.org/10.1111/apha.12637. Acesso em: 17 jun. 2024.
APA
Falquetto, B., Takakura, A. C., Mulkey, D. K., & Moreira, T. dos S. (2016). Purinergic receptor blockade in the retrotrapezoid nucleus attenuates the respiratory chemoreflexes in awake rats. Acta Physiologica, 217( 1), 80-93. doi:10.1111/apha.12637
NLM
Falquetto B, Takakura AC, Mulkey DK, Moreira T dos S. Purinergic receptor blockade in the retrotrapezoid nucleus attenuates the respiratory chemoreflexes in awake rats [Internet]. Acta Physiologica. 2016 ; 217( 1): 80-93.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/apha.12637
Vancouver
Falquetto B, Takakura AC, Mulkey DK, Moreira T dos S. Purinergic receptor blockade in the retrotrapezoid nucleus attenuates the respiratory chemoreflexes in awake rats [Internet]. Acta Physiologica. 2016 ; 217( 1): 80-93.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/apha.12637
Artigo de periodico
Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies (2015)
Lucena, Camila Ferraz; Roma, Leticia Prates; Graciano, Maria Fernanda Rodrigues; Veras, Katherine; Simões, Daniel; Curi, Rui; Carpinelli, Angelo Rafael
Source: Pancreas. Unidade: ICB
Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, SUPLEMENTOS ALIMENTARES PARA ANIMAIS, ÓLEOS DE PEIUXE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUCENA, Camila Ferraz et al. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, v. 44, n. 2, p. 287-295, 2015Tradução . . Disponível em: https://doi.org/10.1097/MPA.0000000000000249. Acesso em: 17 jun. 2024.
APA
Lucena, C. F., Roma, L. P., Graciano, M. F. R., Veras, K., Simões, D., Curi, R., & Carpinelli, A. R. (2015). Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, 44( 2), 287-295. doi:10.1097/MPA.0000000000000249
NLM
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Vancouver
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Artigo de periodico
Sex-dependent differences in renal angiotensinogen as an early marker of diabetic nephropathy (2015)
Costa, D. de Alencar Franco; Todiras, M.; Campos, L. A.; Cipolla Neto, José; Bader, Marcos; Baltatu, Ovidiu C.
Source: Acta Physiologica. Unidade: ICB
Subjects: FISIOLOGIA, SISTEMA RENINA-ANGIOTENSINA, NEFROPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, D. de Alencar Franco et al. Sex-dependent differences in renal angiotensinogen as an early marker of diabetic nephropathy. Acta Physiologica, v. 213, n. 3, p. 740-746, 2015Tradução . . Disponível em: https://doi.org/10.1111/apha.12441. Acesso em: 17 jun. 2024.
APA
Costa, D. de A. F., Todiras, M., Campos, L. A., Cipolla Neto, J., Bader, M., & Baltatu, O. C. (2015). Sex-dependent differences in renal angiotensinogen as an early marker of diabetic nephropathy. Acta Physiologica, 213( 3), 740-746. doi:10.1111/apha.12441
NLM
Costa D de AF, Todiras M, Campos LA, Cipolla Neto J, Bader M, Baltatu OC. Sex-dependent differences in renal angiotensinogen as an early marker of diabetic nephropathy [Internet]. Acta Physiologica. 2015 ; 213( 3): 740-746.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/apha.12441
Vancouver
Costa D de AF, Todiras M, Campos LA, Cipolla Neto J, Bader M, Baltatu OC. Sex-dependent differences in renal angiotensinogen as an early marker of diabetic nephropathy [Internet]. Acta Physiologica. 2015 ; 213( 3): 740-746.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1111/apha.12441
Artigo de periodico
Molecular underpinnings of ventral surface chemoreceptor function: focus on KCNQ channels (2015)
Mulkey, Daniel K.; Hawkins, Virginia E.; Hawryluk, Joanna M.; Takakura, Ana Carolina Thomaz; Moreira, Thiago dos Santos ; Tzingounis, Anastasios V.
Source: Journal of Physiology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, FISIOLOGIA RESPIRATÓRIA E CIRCULATÓRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MULKEY, Daniel K. et al. Molecular underpinnings of ventral surface chemoreceptor function: focus on KCNQ channels. Journal of Physiology, v. 593, n. 5, p. 1075-1081, 2015Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2014.286500. Acesso em: 17 jun. 2024.
APA
Mulkey, D. K., Hawkins, V. E., Hawryluk, J. M., Takakura, A. C. T., Moreira, T. dos S., & Tzingounis, A. V. (2015). Molecular underpinnings of ventral surface chemoreceptor function: focus on KCNQ channels. Journal of Physiology, 593( 5), 1075-1081. doi:10.1113/jphysiol.2014.286500
NLM
Mulkey DK, Hawkins VE, Hawryluk JM, Takakura ACT, Moreira T dos S, Tzingounis AV. Molecular underpinnings of ventral surface chemoreceptor function: focus on KCNQ channels [Internet]. Journal of Physiology. 2015 ; 593( 5): 1075-1081.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1113/jphysiol.2014.286500
Vancouver
Mulkey DK, Hawkins VE, Hawryluk JM, Takakura ACT, Moreira T dos S, Tzingounis AV. Molecular underpinnings of ventral surface chemoreceptor function: focus on KCNQ channels [Internet]. Journal of Physiology. 2015 ; 593( 5): 1075-1081.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1113/jphysiol.2014.286500
Artigo de periodico
Parathyroid hormone inhibition of Na(+)/H(+) exchanger 3 transcription: intracellular signaling pathways and transcription factor expression (2015)
Neri, Elida Adalgisa; Bezerra, Camila Nogueira Alves; Leite, Gabriella Duarte Queiroz; Polidoro, Juliano Zequini; Rebouças, Nancy Amaral
Source: Biochemical and Biophysical Research Communications. Unidade: ICB
Subjects: FISIOLOGIA, TUBULOS RENAIS PROXIMAIS, HORMÔNIOS PARATIREOIDEOS, TRANSPORTEDE SÓDIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NERI, Elida Adalgisa et al. Parathyroid hormone inhibition of Na(+)/H(+) exchanger 3 transcription: intracellular signaling pathways and transcription factor expression. Biochemical and Biophysical Research Communications, v. 461, n. 4, p. 582-588, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2015.04.049. Acesso em: 17 jun. 2024.
APA
Neri, E. A., Bezerra, C. N. A., Leite, G. D. Q., Polidoro, J. Z., & Rebouças, N. A. (2015). Parathyroid hormone inhibition of Na(+)/H(+) exchanger 3 transcription: intracellular signaling pathways and transcription factor expression. Biochemical and Biophysical Research Communications, 461( 4), 582-588. doi:10.1016/j.bbrc.2015.04.049
NLM
Neri EA, Bezerra CNA, Leite GDQ, Polidoro JZ, Rebouças NA. Parathyroid hormone inhibition of Na(+)/H(+) exchanger 3 transcription: intracellular signaling pathways and transcription factor expression [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 461( 4): 582-588.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.bbrc.2015.04.049
Vancouver
Neri EA, Bezerra CNA, Leite GDQ, Polidoro JZ, Rebouças NA. Parathyroid hormone inhibition of Na(+)/H(+) exchanger 3 transcription: intracellular signaling pathways and transcription factor expression [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 461( 4): 582-588.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1016/j.bbrc.2015.04.049
Artigo de periodico
NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells (2015)
Kuwabara, Wilson Mitsuo Tatagiba; Zhang, Liling; Schuiki, Irmgard; Curi, Rui; Volchuk, Allen; Loureiro, Tatiana Carolina Alba
Source: PLOS ONE. Unidade: ICB
Subjects: FISIOLOGIA, ATIVAÇÃO ENZIMÁTICA, NEUTRÓFILOS, LEUCEMIA, RETICULO ENDOPLASMÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KUWABARA, Wilson Mitsuo Tatagiba et al. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells. PLOS ONE, v. 10, n. 2, p. 1-15, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0116410. Acesso em: 17 jun. 2024.
APA
Kuwabara, W. M. T., Zhang, L., Schuiki, I., Curi, R., Volchuk, A., & Loureiro, T. C. A. (2015). NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells. PLOS ONE, 10( 2), 1-15. doi:10.1371/journal.pone.0116410
NLM
Kuwabara WMT, Zhang L, Schuiki I, Curi R, Volchuk A, Loureiro TCA. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells [Internet]. PLOS ONE. 2015 ; 10( 2): 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1371/journal.pone.0116410
Vancouver
Kuwabara WMT, Zhang L, Schuiki I, Curi R, Volchuk A, Loureiro TCA. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells [Internet]. PLOS ONE. 2015 ; 10( 2): 1-15.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1371/journal.pone.0116410
Artigo de periodico
Proximal tubule NHE3 activity is inhibited by beta-arrestin-biased angiotensin II type 1 receptor signaling (2015)
Morais, Carla P. Carneiro de; Polidoro, Juliano Zequini; Ralph, Donna L.; Pessoa, Thaissa Dantas; Oliveira-Souza, Maria; Barauna, Valério Garrone; Rebouças, Nancy Amaral; Malnic, Gerhard; McDonough, Alicia A.; Girardi, Adriana Castello Costa
Source: American Journal of Physiology. Unidade: ICB
Subjects: FISIOLOGIA, TUBULOS RENAIS PROXIMAIS, ANGIOTENSINA II
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAIS, Carla P. Carneiro de et al. Proximal tubule NHE3 activity is inhibited by beta-arrestin-biased angiotensin II type 1 receptor signaling. American Journal of Physiology, v. 309, n. 8, p. C541-C550, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00072.2015. Acesso em: 17 jun. 2024.
APA
Morais, C. P. C. de, Polidoro, J. Z., Ralph, D. L., Pessoa, T. D., Oliveira-Souza, M., Barauna, V. G., et al. (2015). Proximal tubule NHE3 activity is inhibited by beta-arrestin-biased angiotensin II type 1 receptor signaling. American Journal of Physiology, 309( 8), C541-C550. doi:10.1152/ajpcell.00072.2015
NLM
Morais CPC de, Polidoro JZ, Ralph DL, Pessoa TD, Oliveira-Souza M, Barauna VG, Rebouças NA, Malnic G, McDonough AA, Girardi ACC. Proximal tubule NHE3 activity is inhibited by beta-arrestin-biased angiotensin II type 1 receptor signaling [Internet]. American Journal of Physiology. 2015 ; 309( 8): C541-C550.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/ajpcell.00072.2015
Vancouver
Morais CPC de, Polidoro JZ, Ralph DL, Pessoa TD, Oliveira-Souza M, Barauna VG, Rebouças NA, Malnic G, McDonough AA, Girardi ACC. Proximal tubule NHE3 activity is inhibited by beta-arrestin-biased angiotensin II type 1 receptor signaling [Internet]. American Journal of Physiology. 2015 ; 309( 8): C541-C550.[citado 2024 jun. 17 ] Available from: https://doi.org/10.1152/ajpcell.00072.2015

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