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Artigo de periodico
mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function (2017)
Paschoal, Vivian Almeida; Amano, Mariane Tami; Oliveira,Thiago Belchior de; Magdalon, Juliana; Perandini, Patricia Chimin; Andrade, Maynara Lucca; Ortiz-Silva, Milene; Castro, Érique; Yamashita, Alex Shimura; Rosa Neto, José Cesar ; Câmara, Niels Olsen Saraiva ; Festuccia, William Tadeu Lara
Source: Immunobiology. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA, IMUNOLOGIA, MACRÓFAGOS, TECIDO ADIPOSO, INFLAMAÇÃO, FENÓTIPOS, CITOCINAS, OBESIDADE, CAMUNDONGOS
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PASCHOAL, Vivian Almeida et al. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, v. 222, n. 2, p. 261-271, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.09.014. Acesso em: 09 nov. 2024.
APA
Paschoal, V. A., Amano, M. T., Oliveira, T. B. de, Magdalon, J., Perandini, P. C., Andrade, M. L., et al. (2017). mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, 222( 2), 261-271. doi:10.1016/j.imbio.2016.09.014
NLM
Paschoal VA, Amano MT, Oliveira TB de, Magdalon J, Perandini PC, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Vancouver
Paschoal VA, Amano MT, Oliveira TB de, Magdalon J, Perandini PC, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Artigo de periodico
Mesenchymal stromal cell-derived microvesicles regulate an internal pro- inflammatory program in activated macrophages (2017)
Agudelo, Juan S. Henao; Braga, Tárcio Teodoro; Amano, Mariane Tami; Cenedeze, Marcos A.; Cavinato, Regiane A.; Peixoto-Santos, Amandda R.; Muscará, Marcelo Nicolás ; Teixeira, Simone A.; Cruz, Mario C.; Castoldi, Angela; Sinigaglia-Coimbra, Rita; Pacheco-Silva, Alvaro; Almeida, Danilo Cândido de; Câmara, Niels Olsen Saraiva
Source: Frontiers in Immunology. Unidade: ICB
Subjects: FARMACOLOGIA, IMUNOLOGIA, MACRÓFAGOS, INFLAMAÇÃO, ADJUVANTES IMUNOLÓGICOS, PERITONITE
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AGUDELO, Juan S. Henao et al. Mesenchymal stromal cell-derived microvesicles regulate an internal pro- inflammatory program in activated macrophages. Frontiers in Immunology, v. 8, n. 881, p. 1-15, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00881. Acesso em: 09 nov. 2024.
APA
Agudelo, J. S. H., Braga, T. T., Amano, M. T., Cenedeze, M. A., Cavinato, R. A., Peixoto-Santos, A. R., et al. (2017). Mesenchymal stromal cell-derived microvesicles regulate an internal pro- inflammatory program in activated macrophages. Frontiers in Immunology, 8( 881), 1-15. doi:10.3389/fimmu.2017.00881
NLM
Agudelo JSH, Braga TT, Amano MT, Cenedeze MA, Cavinato RA, Peixoto-Santos AR, Muscará MN, Teixeira SA, Cruz MC, Castoldi A, Sinigaglia-Coimbra R, Pacheco-Silva A, Almeida DC de, Câmara NOS. Mesenchymal stromal cell-derived microvesicles regulate an internal pro- inflammatory program in activated macrophages [Internet]. Frontiers in Immunology. 2017 ; 8( 881): 1-15.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00881
Vancouver
Agudelo JSH, Braga TT, Amano MT, Cenedeze MA, Cavinato RA, Peixoto-Santos AR, Muscará MN, Teixeira SA, Cruz MC, Castoldi A, Sinigaglia-Coimbra R, Pacheco-Silva A, Almeida DC de, Câmara NOS. Mesenchymal stromal cell-derived microvesicles regulate an internal pro- inflammatory program in activated macrophages [Internet]. Frontiers in Immunology. 2017 ; 8( 881): 1-15.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00881
Artigo de periodico
Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen (2017)
Araújo, Eliseu Frank de; Loures, Flavio Vieira ; Feriotti, Cláudia ; Costa, Tania; Vacca, Carmine; Puccetti, Paolo; Romani, Luigina; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, CÉLULAS DENDRÍTICAS, ATIVAÇÃO ENZIMÁTICA, ENZIMAS OXIRREDUTORAS
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ARAÚJO, Eliseu Frank de et al. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, v. 8, n. 1522, p. 1-14, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01522. Acesso em: 09 nov. 2024.
APA
Araújo, E. F. de, Loures, F. V., Feriotti, C., Costa, T., Vacca, C., Puccetti, P., et al. (2017). Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, 8( 1522), 1-14. doi:10.3389/fimmu.2017.01522
NLM
Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
Vancouver
Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
Artigo de periodico
SOCS1 favors the epithelial-mesenchymal transition in melanoma, promotes tumor progression and prevents antitumor immunity by PD-L1 expression (2017)
Berzaghi, Rodrigo; Maia, V. S. C.; Pereira, F. V.; Melo F. M.; Guedes, M. S.; Origassa, Clarice Silvia Taemi; Scutti, J. B.; Matsuo, A. L.; Câmara, Niels Olsen Saraiva ; Rodrigues, E. G.; Travassos, L. R.
Source: Scientific Reports. Unidade: ICB
Subjects: IMUNOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, CÉLULAS EPITELIAIS, MELANOMA, FIBROBLASTOS, IMUNIDADE, EXPRESSÃO GÊNICA
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BERZAGHI, Rodrigo et al. SOCS1 favors the epithelial-mesenchymal transition in melanoma, promotes tumor progression and prevents antitumor immunity by PD-L1 expression. Scientific Reports, v. 7, n. 40585, p. 1-16, 2017Tradução . . Disponível em: https://doi.org/10.1038/srep40585. Acesso em: 09 nov. 2024.
APA
Berzaghi, R., Maia, V. S. C., Pereira, F. V., Melo F. M.,, Guedes, M. S., Origassa, C. S. T., et al. (2017). SOCS1 favors the epithelial-mesenchymal transition in melanoma, promotes tumor progression and prevents antitumor immunity by PD-L1 expression. Scientific Reports, 7( 40585), 1-16. doi:10.1038/srep40585
NLM
Berzaghi R, Maia VSC, Pereira FV, Melo F. M., Guedes MS, Origassa CST, Scutti JB, Matsuo AL, Câmara NOS, Rodrigues EG, Travassos LR. SOCS1 favors the epithelial-mesenchymal transition in melanoma, promotes tumor progression and prevents antitumor immunity by PD-L1 expression [Internet]. Scientific Reports. 2017 ; 7( 40585): 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/srep40585
Vancouver
Berzaghi R, Maia VSC, Pereira FV, Melo F. M., Guedes MS, Origassa CST, Scutti JB, Matsuo AL, Câmara NOS, Rodrigues EG, Travassos LR. SOCS1 favors the epithelial-mesenchymal transition in melanoma, promotes tumor progression and prevents antitumor immunity by PD-L1 expression [Internet]. Scientific Reports. 2017 ; 7( 40585): 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/srep40585
Artigo de periodico
MATE-1 modulation by kinin B1 receptor enhances cisplant effux renal cells (2017)
Estrela, Gabriel R.; Wasinski, Frederick; Felizardo, Raphael J. F.; Souza, Laura L.; Câmara, Niels Olsen Saraiva ; Bader, Michael; Araujo, Ronaldo C.
Source: Molecular and Cellular Biochemistry. Unidade: ICB
Assunto: IMUNOLOGIA
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ESTRELA, Gabriel R. et al. MATE-1 modulation by kinin B1 receptor enhances cisplant effux renal cells. Molecular and Cellular Biochemistry, v. 428, n. 1-2, p. 101-108, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11010-016-2920-x. Acesso em: 09 nov. 2024.
APA
Estrela, G. R., Wasinski, F., Felizardo, R. J. F., Souza, L. L., Câmara, N. O. S., Bader, M., & Araujo, R. C. (2017). MATE-1 modulation by kinin B1 receptor enhances cisplant effux renal cells. Molecular and Cellular Biochemistry, 428( 1-2), 101-108. doi:10.1007/s11010-016-2920-x
NLM
Estrela GR, Wasinski F, Felizardo RJF, Souza LL, Câmara NOS, Bader M, Araujo RC. MATE-1 modulation by kinin B1 receptor enhances cisplant effux renal cells [Internet]. Molecular and Cellular Biochemistry. 2017 ; 428( 1-2): 101-108.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s11010-016-2920-x
Vancouver
Estrela GR, Wasinski F, Felizardo RJF, Souza LL, Câmara NOS, Bader M, Araujo RC. MATE-1 modulation by kinin B1 receptor enhances cisplant effux renal cells [Internet]. Molecular and Cellular Biochemistry. 2017 ; 428( 1-2): 101-108.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s11010-016-2920-x
Artigo de periodico
Respiratory gas exchange as a new aid to monitor acidosis in endotoxemic rats: relationship to metabolic fuel substrates and thermometabolic responses (2017)
Steiner, Alexandre Alarcon ; Flatow, Elizabeth Alessandra; Brito, Camila de Fátima Carvalho; Fonseca, Monique Thaís Costa; Komegae, Evilin N.
Source: Physiological Reports. Unidade: ICB
Subjects: IMUNOLOGIA, ACIDOSE, DESEQUILÍBRIO ÁCIDO-BÁSICO, TERMOQUÍMICA, HIPOTERMIA, OXIDAÇÃO, METABOLISMO RESPIRATÓRIO
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STEINER, Alexandre Alarcon et al. Respiratory gas exchange as a new aid to monitor acidosis in endotoxemic rats: relationship to metabolic fuel substrates and thermometabolic responses. Physiological Reports, v. 5, n. 1, p. 1-16, 2017Tradução . . Disponível em: https://doi.org/10.14814/phy2.13100. Acesso em: 09 nov. 2024.
APA
Steiner, A. A., Flatow, E. A., Brito, C. de F. C., Fonseca, M. T. C., & Komegae, E. N. (2017). Respiratory gas exchange as a new aid to monitor acidosis in endotoxemic rats: relationship to metabolic fuel substrates and thermometabolic responses. Physiological Reports, 5( 1), 1-16. doi:10.14814/phy2.13100
NLM
Steiner AA, Flatow EA, Brito C de FC, Fonseca MTC, Komegae EN. Respiratory gas exchange as a new aid to monitor acidosis in endotoxemic rats: relationship to metabolic fuel substrates and thermometabolic responses [Internet]. Physiological Reports. 2017 ; 5( 1): 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.14814/phy2.13100
Vancouver
Steiner AA, Flatow EA, Brito C de FC, Fonseca MTC, Komegae EN. Respiratory gas exchange as a new aid to monitor acidosis in endotoxemic rats: relationship to metabolic fuel substrates and thermometabolic responses [Internet]. Physiological Reports. 2017 ; 5( 1): 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.14814/phy2.13100
Artigo de periodico
Immunomodulatory and antitumor effects of type I interferons and their application in cancer therapy (2017)
Medrano, Ruan Felipe Vieira; Hunger, Aline; Mendonça, Samir Andrade; Barbuto, José Alexandre Marzagão ; Strauss, Bryan Eric
Source: Oncotarget. Unidade: ICB
Subjects: IMUNOLOGIA, CÉLULAS MORTAS, CÉLULAS SANGUÍNEAS, INTERFERON TIPO I, ADJUVANTES IMUNOLÓGICOS
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MEDRANO, Ruan Felipe Vieira et al. Immunomodulatory and antitumor effects of type I interferons and their application in cancer therapy. Oncotarget, v. 8, n. 41, p. 71249-71284, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.1953. Acesso em: 09 nov. 2024.
APA
Medrano, R. F. V., Hunger, A., Mendonça, S. A., Barbuto, J. A. M., & Strauss, B. E. (2017). Immunomodulatory and antitumor effects of type I interferons and their application in cancer therapy. Oncotarget, 8( 41), 71249-71284. doi:10.18632/oncotarget.1953
NLM
Medrano RFV, Hunger A, Mendonça SA, Barbuto JAM, Strauss BE. Immunomodulatory and antitumor effects of type I interferons and their application in cancer therapy [Internet]. Oncotarget. 2017 ; 8( 41): 71249-71284.[citado 2024 nov. 09 ] Available from: https://doi.org/10.18632/oncotarget.1953
Vancouver
Medrano RFV, Hunger A, Mendonça SA, Barbuto JAM, Strauss BE. Immunomodulatory and antitumor effects of type I interferons and their application in cancer therapy [Internet]. Oncotarget. 2017 ; 8( 41): 71249-71284.[citado 2024 nov. 09 ] Available from: https://doi.org/10.18632/oncotarget.1953
Artigo de periodico
The role of the P2X7 receptor in murine cutaneous leishmaniasis: aspects of Inflammation and parasite control (2017)
Figliuolo, Vanessa Ribeiro; Chaves, Suzana Passos; Savio, Luiz Eduardo Baggio; Thorstenberg, Maria Luiza Prates; Salles, Érika Machado; Takiya, Christina Maeda; Lima, Maria Regina D'Império ; Guedes, Herbert Leonel de Matos; Rossi-Bergmann, Bartira; Coutinho-Silva, Robson
Source: Purinergic Signalling. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FIGLIUOLO, Vanessa Ribeiro et al. The role of the P2X7 receptor in murine cutaneous leishmaniasis: aspects of Inflammation and parasite control. Purinergic Signalling, v. 13, n. 2, p. 143-152, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11302-016-9544-1. Acesso em: 09 nov. 2024.
APA
Figliuolo, V. R., Chaves, S. P., Savio, L. E. B., Thorstenberg, M. L. P., Salles, É. M., Takiya, C. M., et al. (2017). The role of the P2X7 receptor in murine cutaneous leishmaniasis: aspects of Inflammation and parasite control. Purinergic Signalling, 13( 2), 143-152. doi:10.1007/s11302-016-9544-1
NLM
Figliuolo VR, Chaves SP, Savio LEB, Thorstenberg MLP, Salles ÉM, Takiya CM, Lima MRD'I, Guedes HL de M, Rossi-Bergmann B, Coutinho-Silva R. The role of the P2X7 receptor in murine cutaneous leishmaniasis: aspects of Inflammation and parasite control [Internet]. Purinergic Signalling. 2017 ; 13( 2): 143-152.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s11302-016-9544-1
Vancouver
Figliuolo VR, Chaves SP, Savio LEB, Thorstenberg MLP, Salles ÉM, Takiya CM, Lima MRD'I, Guedes HL de M, Rossi-Bergmann B, Coutinho-Silva R. The role of the P2X7 receptor in murine cutaneous leishmaniasis: aspects of Inflammation and parasite control [Internet]. Purinergic Signalling. 2017 ; 13( 2): 143-152.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s11302-016-9544-1
Artigo de periodico
Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor (2017)
Silva Junior, Ildefonso Alves da; Stone, Simone Cardozo; Rossetti, Renata Ariza Marques; Jancar, Sonia ; Lepique, Ana Paula
Source: Journal of Immunology Research. Unidade: ICB
Subjects: IMUNOLOGIA, MACRÓFAGOS, PLAQUETAS SANGUÍNEAS, CÉLULAS CULTIVADAS DE TUMOR, NEUTRÓFILOS, LINFÓCITOS
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SILVA JUNIOR, Ildefonso Alves da et al. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor. Journal of Immunology Research, v. 2017, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1155/2017/5482768. Acesso em: 09 nov. 2024.
APA
Silva Junior, I. A. da, Stone, S. C., Rossetti, R. A. M., Jancar, S., & Lepique, A. P. (2017). Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor. Journal of Immunology Research, 2017, 1-10. doi:10.1155/2017/5482768
NLM
Silva Junior IA da, Stone SC, Rossetti RAM, Jancar S, Lepique AP. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor [Internet]. Journal of Immunology Research. 2017 ; 2017 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2017/5482768
Vancouver
Silva Junior IA da, Stone SC, Rossetti RAM, Jancar S, Lepique AP. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor [Internet]. Journal of Immunology Research. 2017 ; 2017 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2017/5482768
Artigo de periodico
Editorial: Macrophages role in integrating tissue signals and biological processes in chronic inflammation and fibrosis (2017)
Braga, Tarcio Teodoro; Moura, Ivan Cruz; Lepique, Ana Paula ; Câmara, Niels Olsen Saraiva
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, MACRÓFAGOS, INFLAMAÇÃO, HOMEOSTASE
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ABNT
BRAGA, Tarcio Teodoro et al. Editorial: Macrophages role in integrating tissue signals and biological processes in chronic inflammation and fibrosis. Frontiers in Immunology, v. 8, n. 845, p. 1-3, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00845. Acesso em: 09 nov. 2024.
APA
Braga, T. T., Moura, I. C., Lepique, A. P., & Câmara, N. O. S. (2017). Editorial: Macrophages role in integrating tissue signals and biological processes in chronic inflammation and fibrosis. Frontiers in Immunology, 8( 845), 1-3. doi:10.3389/fimmu.2017.00845
NLM
Braga TT, Moura IC, Lepique AP, Câmara NOS. Editorial: Macrophages role in integrating tissue signals and biological processes in chronic inflammation and fibrosis [Internet]. Frontiers in Immunology. 2017 ; 8( 845): 1-3.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00845
Vancouver
Braga TT, Moura IC, Lepique AP, Câmara NOS. Editorial: Macrophages role in integrating tissue signals and biological processes in chronic inflammation and fibrosis [Internet]. Frontiers in Immunology. 2017 ; 8( 845): 1-3.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00845
Artigo de periodico
Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ (2017)
Cabral-Marques, Otavio; Ramos, Rodrigo Nalio; Schimke, Lena Friederick; Khan, Taj Ali; Amaral, Eduardo Pinheiro; Bomfim, Caio César Barbosa; Reis Junior, Osvaldo; França, Tabata Takahashi; Kubo, Christina Arslanian; Lima, Joanna Darck Carola Correia; Weber, Cristina Worm; Ferreira, Janaíra Fernandes; Tavares, Fabiola Scancetti; Sun, Jing; Lima, Maria Regina D'Império ; Seelaender, Marília; Calich, Vera Lucia Garcia; Barbuto, José Alexandre Marzagão ; Costa-Carvalho, Beatriz Tavares; Riemekasten, Gabriela; Seminario, Gisela; Bezrodnik, Liliana; Notarangelo, Luigi; Torgerson, Troy R.; Ochs, Hans D.; Condino-Neto, Antonio
Source: Journal of Allergy and Clinical Immunology. Unidade: ICB
Subjects: HISTOLOGIA, IMUNOLOGIA, MACRÓFAGOS, ADJUVANTES IMUNOLÓGICOS, CITOCINAS
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ABNT
CABRAL-MARQUES, Otavio et al. Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ. Journal of Allergy and Clinical Immunology, v. 139, n. 3, p. 900-912, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jaci.2016.07.018. Acesso em: 09 nov. 2024.
APA
Cabral-Marques, O., Ramos, R. N., Schimke, L. F., Khan, T. A., Amaral, E. P., Bomfim, C. C. B., et al. (2017). Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ. Journal of Allergy and Clinical Immunology, 139( 3), 900-912. doi:10.1016/j.jaci.2016.07.018
NLM
Cabral-Marques O, Ramos RN, Schimke LF, Khan TA, Amaral EP, Bomfim CCB, Reis Junior O, França TT, Kubo CA, Lima JDCC, Weber CW, Ferreira JF, Tavares FS, Sun J, Lima MRD'I, Seelaender M, Calich VLG, Barbuto JAM, Costa-Carvalho BT, Riemekasten G, Seminario G, Bezrodnik L, Notarangelo L, Torgerson TR, Ochs HD, Condino-Neto A. Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ [Internet]. Journal of Allergy and Clinical Immunology. 2017 ; 139( 3): 900-912.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.jaci.2016.07.018
Vancouver
Cabral-Marques O, Ramos RN, Schimke LF, Khan TA, Amaral EP, Bomfim CCB, Reis Junior O, França TT, Kubo CA, Lima JDCC, Weber CW, Ferreira JF, Tavares FS, Sun J, Lima MRD'I, Seelaender M, Calich VLG, Barbuto JAM, Costa-Carvalho BT, Riemekasten G, Seminario G, Bezrodnik L, Notarangelo L, Torgerson TR, Ochs HD, Condino-Neto A. Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ [Internet]. Journal of Allergy and Clinical Immunology. 2017 ; 139( 3): 900-912.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.jaci.2016.07.018
Artigo de periodico
Nasal polyp-derived mesenchymal stromal cells exhibit lack of imune-associated molecules and high levels of stem/progenitor cells markers (2017)
Oliveira, Pedro Wey Barbosa de; Pezato, Rogério; Agudelo, Juan Sebastian Henao; Perez-Novo, Claudina Angela; Berghe, Wim Vanden; Câmara, Niels Olsen Saraiva ; Almeida, Danilo Candido de; Gregorio, Luís Carlos
Source: Frontiers in Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
OLIVEIRA, Pedro Wey Barbosa de et al. Nasal polyp-derived mesenchymal stromal cells exhibit lack of imune-associated molecules and high levels of stem/progenitor cells markers. Frontiers in Immunology, v. 8, n. 39, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00039. Acesso em: 09 nov. 2024.
APA
Oliveira, P. W. B. de, Pezato, R., Agudelo, J. S. H., Perez-Novo, C. A., Berghe, W. V., Câmara, N. O. S., et al. (2017). Nasal polyp-derived mesenchymal stromal cells exhibit lack of imune-associated molecules and high levels of stem/progenitor cells markers. Frontiers in Immunology, 8( 39), 1-11. doi:10.3389/fimmu.2017.00039
NLM
Oliveira PWB de, Pezato R, Agudelo JSH, Perez-Novo CA, Berghe WV, Câmara NOS, Almeida DC de, Gregorio LC. Nasal polyp-derived mesenchymal stromal cells exhibit lack of imune-associated molecules and high levels of stem/progenitor cells markers [Internet]. Frontiers in Immunology. 2017 ; 8( 39): 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00039
Vancouver
Oliveira PWB de, Pezato R, Agudelo JSH, Perez-Novo CA, Berghe WV, Câmara NOS, Almeida DC de, Gregorio LC. Nasal polyp-derived mesenchymal stromal cells exhibit lack of imune-associated molecules and high levels of stem/progenitor cells markers [Internet]. Frontiers in Immunology. 2017 ; 8( 39): 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00039
Artigo de periodico
Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis (2017)
Rossato, Cristiano; Brandão, Wesley Nogueira; Castro, Sandra Bertelli Ribeiro de; Almeida, Danilo Cândido de; Maranduba, Carlos Magno da Costa; Câmara, Niels Olsen Saraiva ; Peron, Jean Pierre Schatzmann ; Silva, Fernando S.
Source: Biologicals. Unidade: ICB
Subjects: IMUNOLOGIA, CÉLULAS-TRONCO, ENCEFALOMIELITE ANIMAL, ESCLEROSE MÚLTIPLA, ADJUVANTES IMUNOLÓGICOS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSATO, Cristiano et al. Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis. Biologicals, v. 49, p. 62-68, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.biologicals.2017.06.007. Acesso em: 09 nov. 2024.
APA
Rossato, C., Brandão, W. N., Castro, S. B. R. de, Almeida, D. C. de, Maranduba, C. M. da C., Câmara, N. O. S., et al. (2017). Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis. Biologicals, 49, 62-68. doi:10.1016/j.biologicals.2017.06.007
NLM
Rossato C, Brandão WN, Castro SBR de, Almeida DC de, Maranduba CM da C, Câmara NOS, Peron JPS, Silva FS. Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis [Internet]. Biologicals. 2017 ; 49 62-68.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.biologicals.2017.06.007
Vancouver
Rossato C, Brandão WN, Castro SBR de, Almeida DC de, Maranduba CM da C, Câmara NOS, Peron JPS, Silva FS. Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis [Internet]. Biologicals. 2017 ; 49 62-68.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.biologicals.2017.06.007
Artigo de periodico
CpG-ODN shapes alum adjuvant activity signaling via MyD88 and IL-10 (2017)
Mirotti, Luciana; Custódio, Ricardo Wesley Alberca; Gomes, Eliane; Rammauro, Florencia; Araujo, Eliseu Frank de; Calich, Vera Lucia Garcia; Russo, Momtchilo
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, ADJUVANTES IMUNOLÓGICOS, INFLAMAÇÃO, PULMÃO
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ABNT
MIROTTI, Luciana et al. CpG-ODN shapes alum adjuvant activity signaling via MyD88 and IL-10. Frontiers in Immunology, v. 8, p. 1-13, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00047. Acesso em: 09 nov. 2024.
APA
Mirotti, L., Custódio, R. W. A., Gomes, E., Rammauro, F., Araujo, E. F. de, Calich, V. L. G., & Russo, M. (2017). CpG-ODN shapes alum adjuvant activity signaling via MyD88 and IL-10. Frontiers in Immunology, 8, 1-13. doi:10.3389/fimmu.2017.00047
NLM
Mirotti L, Custódio RWA, Gomes E, Rammauro F, Araujo EF de, Calich VLG, Russo M. CpG-ODN shapes alum adjuvant activity signaling via MyD88 and IL-10 [Internet]. Frontiers in Immunology. 2017 ; 8 1-13.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00047
Vancouver
Mirotti L, Custódio RWA, Gomes E, Rammauro F, Araujo EF de, Calich VLG, Russo M. CpG-ODN shapes alum adjuvant activity signaling via MyD88 and IL-10 [Internet]. Frontiers in Immunology. 2017 ; 8 1-13.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00047
Artigo de periodico
P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis (2017)
Bomfim, Caio César Barbosa; Amaral, Eduardo Pinheiro; Cassado, Alexandra dos Anjos; Salles, Érika Machado; Nascimento, Rogério Silva do; Lasunskaia, Elena; Hirata, Mario Hiroyuki ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Source: Frontiers in Immunology. Unidades: ICB, FCF
Assunto: IMUNOLOGIA
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ABNT
BOMFIM, Caio César Barbosa et al. P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis. Frontiers in Immunology, v. 8, n. 435, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00435. Acesso em: 09 nov. 2024.
APA
Bomfim, C. C. B., Amaral, E. P., Cassado, A. dos A., Salles, É. M., Nascimento, R. S. do, Lasunskaia, E., et al. (2017). P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis. Frontiers in Immunology, 8( 435), 1-10. doi:10.3389/fimmu.2017.00435
NLM
Bomfim CCB, Amaral EP, Cassado A dos A, Salles ÉM, Nascimento RS do, Lasunskaia E, Hirata MH, Alvarez JM, Lima MRD'I. P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis [Internet]. Frontiers in Immunology. 2017 ; 8( 435): 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00435
Vancouver
Bomfim CCB, Amaral EP, Cassado A dos A, Salles ÉM, Nascimento RS do, Lasunskaia E, Hirata MH, Alvarez JM, Lima MRD'I. P2X7 receptor in bone marrow-derived cells aggravates tuberculosis caused by hypervirulent Mycobacterium bovis [Internet]. Frontiers in Immunology. 2017 ; 8( 435): 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2017.00435
Artigo de periodico
Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice (2017)
Filgueiras, Luciano Ribeiro; Brandt, Stephanie L.; Ramalho, Theresa Raquel de Oliveira; Jancar, Sonia ; Serezani, C. Henrique
Source: Journal of Diabetes and its Complications. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FILGUEIRAS, Luciano Ribeiro et al. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, v. 31, n. 2, p. 334-339, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2016.08.001. Acesso em: 09 nov. 2024.
APA
Filgueiras, L. R., Brandt, S. L., Ramalho, T. R. de O., Jancar, S., & Serezani, C. H. (2017). Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, 31( 2), 334-339. doi:10.1016/j.jdiacomp.2016.08.001
NLM
Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001
Vancouver
Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001
Artigo de periodico
Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway (2016)
Macedo, Ana Catarina; Isaac, Lourdes
Source: Frontiers in Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACEDO, Ana Catarina e ISAAC, Lourdes. Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway. Frontiers in Immunology, v. 7, p. 1-7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00055. Acesso em: 09 nov. 2024.
APA
Macedo, A. C., & Isaac, L. (2016). Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway. Frontiers in Immunology, 7, 1-7. doi:10.3389/fimmu.2016.00055
NLM
Macedo AC, Isaac L. Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway [Internet]. Frontiers in Immunology. 2016 ; 7 1-7.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00055
Vancouver
Macedo AC, Isaac L. Systemic lupus erythematosus and deficiencies of early components of the complement classical pathway [Internet]. Frontiers in Immunology. 2016 ; 7 1-7.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00055
Artigo de periodico
Innate sensing of the gut microbiota: modulation of inflammatory and autoimmune diseases (2016)
Ignacio, Aline; Morales, Camila Ideli; Câmara, Niels Olsen Saraiva ; Almeida, Rafael Ribeiro
Source: Frontiers in Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
IGNACIO, Aline et al. Innate sensing of the gut microbiota: modulation of inflammatory and autoimmune diseases. Frontiers in Immunology, v. 7, p. 1-11, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00054. Acesso em: 09 nov. 2024.
APA
Ignacio, A., Morales, C. I., Câmara, N. O. S., & Almeida, R. R. (2016). Innate sensing of the gut microbiota: modulation of inflammatory and autoimmune diseases. Frontiers in Immunology, 7, 1-11. doi:10.3389/fimmu.2016.00054
NLM
Ignacio A, Morales CI, Câmara NOS, Almeida RR. Innate sensing of the gut microbiota: modulation of inflammatory and autoimmune diseases [Internet]. Frontiers in Immunology. 2016 ; 7 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00054
Vancouver
Ignacio A, Morales CI, Câmara NOS, Almeida RR. Innate sensing of the gut microbiota: modulation of inflammatory and autoimmune diseases [Internet]. Frontiers in Immunology. 2016 ; 7 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00054
Artigo de periodico
Interferon-gamma reduces the proliferation of M. tuberculosis within macrophages from a patient with a novel hypomorphic NEMO mutation (2016)
Khan, Taj Ali; Ishfaq, Muhammad; Bonﬁm, Caio César Barbosa; Rahman, Hazir; Iqbal, Asif; Lima, Maria Regina D'Império ; Carvalho, Beatriz Tavares Costa ; Cabral-Marques, Otavio; Condino Neto, Antonio
Source: Pediatric Bood & Cancer. Unidade: ICB
Subjects: DISPLASIA ECTODÉRMICA, IMUNOLOGIA, INTERFERONS, MACRÓFAGOS, MYCOBACTERIUM TUBERCULOSIS, MUTAÇÃO GENÉTICA, LINFÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KHAN, Taj Ali et al. Interferon-gamma reduces the proliferation of M. tuberculosis within macrophages from a patient with a novel hypomorphic NEMO mutation. Pediatric Bood & Cancer, v. 63, p. 1863-1866, 2016Tradução . . Disponível em: https://doi.org/10.1002/pbc.26098. Acesso em: 09 nov. 2024.
APA
Khan, T. A., Ishfaq, M., Bonﬁm, C. C. B., Rahman, H., Iqbal, A., Lima, M. R. D. 'I., et al. (2016). Interferon-gamma reduces the proliferation of M. tuberculosis within macrophages from a patient with a novel hypomorphic NEMO mutation. Pediatric Bood & Cancer, 63, 1863-1866. doi:10.1002/pbc.26098
NLM
Khan TA, Ishfaq M, Bonﬁm CCB, Rahman H, Iqbal A, Lima MRD'I, Carvalho BTC, Cabral-Marques O, Condino Neto A. Interferon-gamma reduces the proliferation of M. tuberculosis within macrophages from a patient with a novel hypomorphic NEMO mutation [Internet]. Pediatric Bood & Cancer. 2016 ; 63 1863-1866.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/pbc.26098
Vancouver
Khan TA, Ishfaq M, Bonﬁm CCB, Rahman H, Iqbal A, Lima MRD'I, Carvalho BTC, Cabral-Marques O, Condino Neto A. Interferon-gamma reduces the proliferation of M. tuberculosis within macrophages from a patient with a novel hypomorphic NEMO mutation [Internet]. Pediatric Bood & Cancer. 2016 ; 63 1863-1866.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/pbc.26098
Artigo de periodico
Tolerogenic IDO(+) dendritic cells are induced by PD-1-expressing mast cells (2016)
Rodrigues, Cecilia Pessoa; Ferreira, Ana Carolina Franco; Pinho, Mariana Pereira; Moraes, Cristiano Jacob de; Bergami-Santos, Patricia Cruz; Barbuto, José Alexandre Marzagão
Source: Frontiers in Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Cecilia Pessoa et al. Tolerogenic IDO(+) dendritic cells are induced by PD-1-expressing mast cells. Frontiers in Immunology, v. 7, n. 9, p. 1-17, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00009. Acesso em: 09 nov. 2024.
APA
Rodrigues, C. P., Ferreira, A. C. F., Pinho, M. P., Moraes, C. J. de, Bergami-Santos, P. C., & Barbuto, J. A. M. (2016). Tolerogenic IDO(+) dendritic cells are induced by PD-1-expressing mast cells. Frontiers in Immunology, 7( 9), 1-17. doi:10.3389/fimmu.2016.00009
NLM
Rodrigues CP, Ferreira ACF, Pinho MP, Moraes CJ de, Bergami-Santos PC, Barbuto JAM. Tolerogenic IDO(+) dendritic cells are induced by PD-1-expressing mast cells [Internet]. Frontiers in Immunology. 2016 ; 7( 9): 1-17.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00009
Vancouver
Rodrigues CP, Ferreira ACF, Pinho MP, Moraes CJ de, Bergami-Santos PC, Barbuto JAM. Tolerogenic IDO(+) dendritic cells are induced by PD-1-expressing mast cells [Internet]. Frontiers in Immunology. 2016 ; 7( 9): 1-17.[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fimmu.2016.00009

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