Filtros : "Indexado na Base de Dados Current Contents" "PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES" Removidos: "Faria, Dalva Lúcia Araújo de" "ca" "Financiado pela British Heart Foundation" Limpar

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  • Source: Vascular Pharmacology. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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    • ABNT

      COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 16 nov. 2024.
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      Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
    • NLM

      Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
    • Vancouver

      Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
  • Source: Journal of Diabetes. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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    • ABNT

      SARTORETTO, Simone Marcieli et al. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, v. 5, n. 2, p. 197-206, 2013Tradução . . Acesso em: 16 nov. 2024.
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      Sartoretto, S. M., Santos-Eichler, R., Tostes, R. de C. A., Carvalho, M. H. C. de, Akamine, E. H., & Fortes, Z. B. (2013). Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, 5( 2), 197-206.
    • NLM

      Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 nov. 16 ]
    • Vancouver

      Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 nov. 16 ]
  • Source: Brazilian Journal of Medical and Biological Research. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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    • ABNT

      LOBATO, Nubia de Souza et al. Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, v. 45, n. 5, p. 392-400, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500058. Acesso em: 16 nov. 2024.
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      Lobato, N. de S., Filgueira, F. P., Akamine, E. H., Passaglia, R. de C. A. T., Carvalho, M. H. C. de, & Fortes, Z. B. (2012). Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, 45( 5), 392-400. doi:10.1590/S0100-879X2012007500058
    • NLM

      Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
    • Vancouver

      Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
  • Source: Life Sciences. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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      FILGUEIRA, Fernando Paranaíba et al. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, v. 90, n. 17-18, p. 689-694, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.01.017. Acesso em: 16 nov. 2024.
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      Filgueira, F. P., Lobato, N. de S., Dos Santos, R. A., Oliveira, M. A., Akamine, E. H., Tostes-Passaglia, R. de C. A., et al. (2012). Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, 90( 17-18), 689-694. doi:10.1016/j.lfs.2012.01.017
    • NLM

      Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
    • Vancouver

      Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
  • Source: Cardiovascular Research. Unidade: ICB

    Subjects: FARMACOLOGIA, ANATOMIA

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      CARRILLO-SEPÚLVEDA, Maria Alicia et al. Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes. Cardiovascular Research, v. 85, n. 3, p. 560-570, 2010Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvp304. Acesso em: 16 nov. 2024.
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      Carrillo-Sepúlveda, M. A., Ceravolo, G. S., Fortes, Z. B., Carvalho, M. H. C., Passaglia, R. de C. A. T., Laurindo, F. R. M., et al. (2010). Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes. Cardiovascular Research, 85( 3), 560-570. doi:10.1093/cvr/cvp304
    • NLM

      Carrillo-Sepúlveda MA, Ceravolo GS, Fortes ZB, Carvalho MHC, Passaglia R de CAT, Laurindo FRM, Webb RC, Barreto-Chaves MLM. Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes [Internet]. Cardiovascular Research. 2010 ; 85( 3): 560-570.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1093/cvr/cvp304
    • Vancouver

      Carrillo-Sepúlveda MA, Ceravolo GS, Fortes ZB, Carvalho MHC, Passaglia R de CAT, Laurindo FRM, Webb RC, Barreto-Chaves MLM. Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes [Internet]. Cardiovascular Research. 2010 ; 85( 3): 560-570.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1093/cvr/cvp304
  • Source: Hypertension. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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      GIACHINI, Fernanda Regina et al. Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity. Hypertension, v. 55, n. 1, p. 172-179, 2010Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.109.140459. Acesso em: 16 nov. 2024.
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      Giachini, F. R., Suillivan, J. C., Lima, V. V., Carneiro, F. S., Fortes, Z. B., Pollock, D. M., et al. (2010). Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity. Hypertension, 55( 1), 172-179. doi:10.1161/HYPERTENSIONAHA.109.140459
    • NLM

      Giachini FR, Suillivan JC, Lima VV, Carneiro FS, Fortes ZB, Pollock DM, Carvalho MHC, Webb RC, Passaglia R de CAT. Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity [Internet]. Hypertension. 2010 ; 55( 1): 172-179.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.109.140459
    • Vancouver

      Giachini FR, Suillivan JC, Lima VV, Carneiro FS, Fortes ZB, Pollock DM, Carvalho MHC, Webb RC, Passaglia R de CAT. Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity [Internet]. Hypertension. 2010 ; 55( 1): 172-179.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.109.140459
  • Source: Clinical Science (1979). Unidade: ICB

    Assunto: FARMACOLOGIA

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      GIACHINI, Fernanda Regina Casagrande et al. Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats. Clinical Science (1979), v. 118, n. 7, p. 463-471, 2010Tradução . . Disponível em: https://doi.org/10.1042/cs20090392. Acesso em: 16 nov. 2024.
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      Giachini, F. R. C., Osmond, D. A., Zhang, S., Carneiro, F. S., Lima, V. V., Inscho, E. W., et al. (2010). Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats. Clinical Science (1979), 118( 7), 463-471. doi:10.1042/cs20090392
    • NLM

      Giachini FRC, Osmond DA, Zhang S, Carneiro FS, Lima VV, Inscho EW, Webb RC, Passaglia R de CAT. Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats [Internet]. Clinical Science (1979). 2010 ; 118( 7): 463-471.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/cs20090392
    • Vancouver

      Giachini FRC, Osmond DA, Zhang S, Carneiro FS, Lima VV, Inscho EW, Webb RC, Passaglia R de CAT. Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats [Internet]. Clinical Science (1979). 2010 ; 118( 7): 463-471.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/cs20090392
  • Source: Clinical Science (1979). Unidade: ICB

    Assunto: FARMACOLOGIA

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      GIACHINI, Fernanda Regina Casagrande e WEBB, R. Clinton e PASSAGLIA, Rita de Cássia Aleixo Tostes. STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction?. Clinical Science (1979), v. 118, n. 6, p. 391-396, 2010Tradução . . Disponível em: https://doi.org/10.1042/cs20090449. Acesso em: 16 nov. 2024.
    • APA

      Giachini, F. R. C., Webb, R. C., & Passaglia, R. de C. A. T. (2010). STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? Clinical Science (1979), 118( 6), 391-396. doi:10.1042/cs20090449
    • NLM

      Giachini FRC, Webb RC, Passaglia R de CAT. STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? [Internet]. Clinical Science (1979). 2010 ; 118( 6): 391-396.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/cs20090449
    • Vancouver

      Giachini FRC, Webb RC, Passaglia R de CAT. STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? [Internet]. Clinical Science (1979). 2010 ; 118( 6): 391-396.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/cs20090449

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