Filtros : "Indexado na Base de Dados MEDLINE" "Journal of Immunology" Removido: "Congresso Brasileiro de Genética" Limpar

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  • Source: Journal of Immunology. Unidade: ICB

    Assunto: PARASITOLOGIA

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    • ABNT

      AMORIM, Kelly N. S. et al. Dendritic cells and their multiple roles during malaria infection. Journal of Immunology, v. 2016, p. 1-11, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/2926436. Acesso em: 09 nov. 2024.
    • APA

      Amorim, K. N. S., Chagas, D. C. G., Sulczewski, F. B., & Boscardin, S. B. (2016). Dendritic cells and their multiple roles during malaria infection. Journal of Immunology, 2016, 1-11. doi:10.1155/2016/2926436
    • NLM

      Amorim KNS, Chagas DCG, Sulczewski FB, Boscardin SB. Dendritic cells and their multiple roles during malaria infection [Internet]. Journal of Immunology. 2016 ; 2016 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2016/2926436
    • Vancouver

      Amorim KNS, Chagas DCG, Sulczewski FB, Boscardin SB. Dendritic cells and their multiple roles during malaria infection [Internet]. Journal of Immunology. 2016 ; 2016 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2016/2926436
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: PARASITOLOGIA

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      APOSTÓLICO, Juliana de Souza et al. Adjuvants: classification, Modus Operandi, and licensing. Journal of Immunology, v. 2016, p. 1-16, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/1459394. Acesso em: 09 nov. 2024.
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      Apostólico, J. de S., Lunardelli, V. A. S., Coirada, F. C., Boscardin, S. B., & Rosa, D. S. (2016). Adjuvants: classification, Modus Operandi, and licensing. Journal of Immunology, 2016, 1-16. doi:10.1155/2016/1459394
    • NLM

      Apostólico J de S, Lunardelli VAS, Coirada FC, Boscardin SB, Rosa DS. Adjuvants: classification, Modus Operandi, and licensing. [Internet]. Journal of Immunology. 2016 ; 2016 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2016/1459394
    • Vancouver

      Apostólico J de S, Lunardelli VAS, Coirada FC, Boscardin SB, Rosa DS. Adjuvants: classification, Modus Operandi, and licensing. [Internet]. Journal of Immunology. 2016 ; 2016 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2016/1459394
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      TONIOLO, Patricia Argenta. et al. Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation. Journal of Immunology, v. 194, n. 7, p. 3180-3190, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1401635. Acesso em: 09 nov. 2024.
    • APA

      Toniolo, P. A., Liu, S., Yeh, J. E., Moraes-Vieira,, Walker, S. R., Vafaizadeh, V., et al. (2015). Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation. Journal of Immunology, 194( 7), 3180-3190. doi:10.4049/jimmunol.1401635
    • NLM

      Toniolo PA, Liu S, Yeh JE, Moraes-Vieira, Walker SR, Vafaizadeh V, Barbuto JAM, Frank DA. Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation [Internet]. Journal of Immunology. 2015 ; 194( 7): 3180-3190.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1401635
    • Vancouver

      Toniolo PA, Liu S, Yeh JE, Moraes-Vieira, Walker SR, Vafaizadeh V, Barbuto JAM, Frank DA. Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation [Internet]. Journal of Immunology. 2015 ; 194( 7): 3180-3190.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1401635
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FLORSHEIM, Esther et al. Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin. Journal of Immunology, v. 194, n. 10, p. 4621-4630, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1402493. Acesso em: 09 nov. 2024.
    • APA

      Florsheim, E., Yu, S., Bargatto, I., Faustino, L., Gomes, E., Ramos, R. N., et al. (2015). Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin. Journal of Immunology, 194( 10), 4621-4630. doi:10.4049/jimmunol.1402493
    • NLM

      Florsheim E, Yu S, Bargatto I, Faustino L, Gomes E, Ramos RN, Barbuto JAM, Medzhitov R, Russo M. Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin [Internet]. Journal of Immunology. 2015 ; 194( 10): 4621-4630.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1402493
    • Vancouver

      Florsheim E, Yu S, Bargatto I, Faustino L, Gomes E, Ramos RN, Barbuto JAM, Medzhitov R, Russo M. Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin [Internet]. Journal of Immunology. 2015 ; 194( 10): 4621-4630.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1402493
  • Source: Journal of Immunology. Unidades: ICB, FMRP

    Subjects: DOENÇAS AUTOIMUNES, ARTRITE (IMUNOLOGIA), PERIODONTO, MICROBIOLOGIA ORAL

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      AQUINO, Sabrina G. de et al. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response. Journal of Immunology, v. 192, n. 9, p. 4103-4111, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1301970. Acesso em: 09 nov. 2024.
    • APA

      Aquino, S. G. de, Abdollahi-Roodsaz, S., Koenders, M. I., van de Loo, F. A. J., Pruijn, G. J. M., Marijnissen, R. J., et al. (2014). Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response. Journal of Immunology, 192( 9), 4103-4111. doi:10.4049/jimmunol.1301970
    • NLM

      Aquino SG de, Abdollahi-Roodsaz S, Koenders MI, van de Loo FAJ, Pruijn GJM, Marijnissen RJ, Walgreen B, Helsen MM, van den Bersselaar LA, Molon RS de, Avila-Campos MJ, Cunha F de Q, Cirelli JA, van den Berg WB. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response [Internet]. Journal of Immunology. 2014 ; 192( 9): 4103-4111.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1301970
    • Vancouver

      Aquino SG de, Abdollahi-Roodsaz S, Koenders MI, van de Loo FAJ, Pruijn GJM, Marijnissen RJ, Walgreen B, Helsen MM, van den Bersselaar LA, Molon RS de, Avila-Campos MJ, Cunha F de Q, Cirelli JA, van den Berg WB. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response [Internet]. Journal of Immunology. 2014 ; 192( 9): 4103-4111.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1301970
  • Source: Journal of Immunology. Unidade: ICB

    Subjects: IMUNOLOGIA, LEUCOTRIENOS B, MACRÓFAGOS

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      WANG, Zhuo et al. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, v. 192, n. 5, p. 2349-2356, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1302982. Acesso em: 09 nov. 2024.
    • APA

      Wang, Z., Filgueiras, L. R., Wang, S., Serezani, A. P. M., Peters-Golden, M., Jancar, S., & Serezani, C. H. (2014). Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, 192( 5), 2349-2356. doi:10.4049/jimmunol.1302982
    • NLM

      Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1302982
    • Vancouver

      Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1302982
  • Source: Journal of Immunology. Unidade: ICB

    Subjects: IMUNOLOGIA, PARASITOLOGIA

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      SILVA, Henrique Borges et al. IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria. Journal of Immunology, v. 191, n. 10, p. 5160-5169, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1300462. Acesso em: 09 nov. 2024.
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      Silva, H. B., Salles, É. M., Panatieri, R. H., Boscardin, S. B., Málaga, S. M. R., Alvarez, J. M., & Lima, M. R. D. 'I. (2013). IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria. Journal of Immunology, 191( 10), 5160-5169. doi:10.4049/jimmunol.1300462
    • NLM

      Silva HB, Salles ÉM, Panatieri RH, Boscardin SB, Málaga SMR, Alvarez JM, Lima MRD'I. IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria [Internet]. Journal of Immunology. 2013 ; 191( 10): 5160-5169.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1300462
    • Vancouver

      Silva HB, Salles ÉM, Panatieri RH, Boscardin SB, Málaga SMR, Alvarez JM, Lima MRD'I. IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria [Internet]. Journal of Immunology. 2013 ; 191( 10): 5160-5169.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1300462
  • Source: Journal of Immunology. Unidade: ICB

    Subjects: HISTOLOGIA, IMUNOLOGIA

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      ALBUQUERQUE, José Antonio Tavares de et al. Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient. Journal of Immunology, v. 189, n. 6, p. 3242-3248, 2012Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1201418. Acesso em: 09 nov. 2024.
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      Albuquerque, J. A. T. de, Lamers, M. L., Castiblanco-Valencia, M. M., Santos, M. F. dos, & Isaac, L. (2012). Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient. Journal of Immunology, 189( 6), 3242-3248. doi:10.4049/jimmunol.1201418
    • NLM

      Albuquerque JAT de, Lamers ML, Castiblanco-Valencia MM, Santos MF dos, Isaac L. Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient [Internet]. Journal of Immunology. 2012 ; 189( 6): 3242-3248.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1201418
    • Vancouver

      Albuquerque JAT de, Lamers ML, Castiblanco-Valencia MM, Santos MF dos, Isaac L. Chemical chaperones curcumin and 4-phenylbutyric acid improve secretion of mutant factor H R127H by fibroblasts from a factor H-deficient patient [Internet]. Journal of Immunology. 2012 ; 189( 6): 3242-3248.[citado 2024 nov. 09 ] Available from: https://doi.org/10.4049/jimmunol.1201418

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