IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria (2013)
- Authors:
- USP affiliated authors: BOSCARDIN, SILVIA BEATRIZ - ICB ; MOSIG, JOSE MARIA ALVAREZ - ICB ; LIMA, MARIA REGINA D'IMPERIO - ICB
- Unidade: ICB
- DOI: 10.4049/jimmunol.1300462
- Subjects: IMUNOLOGIA; PARASITOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Immunology
- ISSN: 1550-6606
- Volume/Número/Paginação/Ano: v. 191, n. 10, p. 5160-5169, 2013
- Status:
- Artigo possui acesso gratuito no site do editor (Bronze Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
SILVA, Henrique Borges et al. IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria. Journal of Immunology, v. 191, n. 10, p. 5160-5169, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1300462. Acesso em: 17 mar. 2026. -
APA
Silva, H. B., Salles, É. M., Panatieri, R. H., Boscardin, S. B., Málaga, S. M. R., Alvarez, J. M., & Lima, M. R. D. 'I. (2013). IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria. Journal of Immunology, 191( 10), 5160-5169. doi:10.4049/jimmunol.1300462 -
NLM
Silva HB, Salles ÉM, Panatieri RH, Boscardin SB, Málaga SMR, Alvarez JM, Lima MRD'I. IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria [Internet]. Journal of Immunology. 2013 ; 191( 10): 5160-5169.[citado 2026 mar. 17 ] Available from: https://doi.org/10.4049/jimmunol.1300462 -
Vancouver
Silva HB, Salles ÉM, Panatieri RH, Boscardin SB, Málaga SMR, Alvarez JM, Lima MRD'I. IFN-y-induced priming maintains long-term strain-transcending immunity against blood-stage plasmodium chabaudi malaria [Internet]. Journal of Immunology. 2013 ; 191( 10): 5160-5169.[citado 2026 mar. 17 ] Available from: https://doi.org/10.4049/jimmunol.1300462 - Role of CD1d- and I-Ab-restricted T cells during the polyclonal lymphocyte activation induced by blood-stage Plasmodium chabaudi AS malaria
- Characterization of a 250kd molecule of plasmodium chabaudi chabaudi schizont forms present in the serum of acutely infected mice
- Influence of the polyclonal activation induced by plasmodium chabaudi on ongoing OVA-specific B- and T-cell responses
- Role of CD28 in polyclonal and specific T and B cell responses required for protection against blood stage malaria
- Characterization of memory CD4+ T and B cells in the spleen of mice infected with blood stages of Plasmodium chabaundi AS
- IL-2 secretion and CD4+CD25 T cells in polyclonal lymphocyte activation (PLA) induced by Plasmodium chabaudi infection
- IFN-'gama', but not nitric oxide or specific IgG, is essential for the in vivo control of low-virulence Sylvio X10/4 Trypanosoma cruzi parasites
- CD1d-restricted 'CD4 POT. +' 'NK1.1 POT. +' T cells are a major source of IFN-'gama' during acute Plasmodium chabaudi AS malaria
- Camundongos C57BL/6 com 200 dias pós-infecção por Plasmodium chabaudi AS são capazes de controlar a reinvasão pela cepa AS, mas não pela CEPA AJ
- Parasite load during the acute phase of murine T cruzi infection determines the activation pattern pf the immune system in late chronic phase
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