Parasite load during the acute phase of murine T cruzi infection determines the activation pattern pf the immune system in late chronic phase (1997)
- Authors:
- USP affiliated authors: LIMA, MARIA REGINA D'IMPERIO - ICB ; MOSIG, JOSE MARIA ALVAREZ - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Immunology Letters
- Volume/Número/Paginação/Ano: v. 56, n. 1-3, p. 42, res. P.4.08.05, 1997
- Conference titles: European Immunology Meeting
-
ABNT
MARINHO, Claudio Romero Farias et al. Parasite load during the acute phase of murine T cruzi infection determines the activation pattern pf the immune system in late chronic phase. Immunology Letters. Amsterdam: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 11 fev. 2026. , 1997 -
APA
Marinho, C. R. F., Lima, M. R. D. 'I., Grisotto, M. G., & Alvarez, J. M. (1997). Parasite load during the acute phase of murine T cruzi infection determines the activation pattern pf the immune system in late chronic phase. Immunology Letters. Amsterdam: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Marinho CRF, Lima MRD'I, Grisotto MG, Alvarez JM. Parasite load during the acute phase of murine T cruzi infection determines the activation pattern pf the immune system in late chronic phase. Immunology Letters. 1997 ; 56( 1-3): 42.[citado 2026 fev. 11 ] -
Vancouver
Marinho CRF, Lima MRD'I, Grisotto MG, Alvarez JM. Parasite load during the acute phase of murine T cruzi infection determines the activation pattern pf the immune system in late chronic phase. Immunology Letters. 1997 ; 56( 1-3): 42.[citado 2026 fev. 11 ] - Role of CD1d- and I-Ab-restricted T cells during the polyclonal lymphocyte activation induced by blood-stage Plasmodium chabaudi AS malaria
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- Influence of the polyclonal activation induced by plasmodium chabaudi on ongoing OVA-specific B- and T-cell responses
- Role of CD28 in polyclonal and specific T and B cell responses required for protection against blood stage malaria
- Characterization of memory CD4+ T and B cells in the spleen of mice infected with blood stages of Plasmodium chabaundi AS
- IL-2 secretion and CD4+CD25 T cells in polyclonal lymphocyte activation (PLA) induced by Plasmodium chabaudi infection
- IFN-'gama', but not nitric oxide or specific IgG, is essential for the in vivo control of low-virulence Sylvio X10/4 Trypanosoma cruzi parasites
- CD1d-restricted 'CD4 POT. +' 'NK1.1 POT. +' T cells are a major source of IFN-'gama' during acute Plasmodium chabaudi AS malaria
- Camundongos C57BL/6 com 200 dias pós-infecção por Plasmodium chabaudi AS são capazes de controlar a reinvasão pela cepa AS, mas não pela CEPA AJ
- Production of IFN-'gama' by 'CD45RB POT. HIGH' and 'CD45RB POT. LOW' CD4+ T cells subsets along the acute and chronic phases of experimental Chagas disease
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