ReP USP - Resultado da busca

Artigo de periodico
Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers (2017)
Soares, Antonio Garcia; Carvalho, Maria Helena Catelli de; Akamine, Eliana Hiromi
Source: BioMed Research International. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOARES, Antonio Garcia e CARVALHO, Maria Helena Catelli de e AKAMINE, Eliana Hiromi. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers. BioMed Research International, v. 2017, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1155/2017/5038602. Acesso em: 09 nov. 2024.
APA
Soares, A. G., Carvalho, M. H. C. de, & Akamine, E. H. (2017). Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers. BioMed Research International, 2017, 1-10. doi:10.1155/2017/5038602
NLM
Soares AG, Carvalho MHC de, Akamine EH. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers [Internet]. BioMed Research International. 2017 ; 2017 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2017/5038602
Vancouver
Soares AG, Carvalho MHC de, Akamine EH. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers [Internet]. BioMed Research International. 2017 ; 2017 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2017/5038602
Artigo de periodico
Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? (2017)
Oliveira, V.; Silva Junior, S. D.; Carvalho, M. H. C. de; Akamine, Eliana Hiromi ; Michelini, Lisete Compagno ; Franco, M. C.
Source: Journal of Developmental Origins of Health and Disease. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, V. et al. Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations?. Journal of Developmental Origins of Health and Disease, v. 8, n. 2, p. 236-243, 2017Tradução . . Disponível em: https://doi.org/10.1017/S2040174416000714. Acesso em: 09 nov. 2024.
APA
Oliveira, V., Silva Junior, S. D., Carvalho, M. H. C. de, Akamine, E. H., Michelini, L. C., & Franco, M. C. (2017). Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? Journal of Developmental Origins of Health and Disease, 8( 2), 236-243. doi:10.1017/S2040174416000714
NLM
Oliveira V, Silva Junior SD, Carvalho MHC de, Akamine EH, Michelini LC, Franco MC. Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? [Internet]. Journal of Developmental Origins of Health and Disease. 2017 ; 8( 2): 236-243.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1017/S2040174416000714
Vancouver
Oliveira V, Silva Junior SD, Carvalho MHC de, Akamine EH, Michelini LC, Franco MC. Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? [Internet]. Journal of Developmental Origins of Health and Disease. 2017 ; 8( 2): 236-243.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1017/S2040174416000714
Artigo de periodico
Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats (2017)
Araujo, Priscila Xavier; Costa, Tiago Januário; Echem, Cinthya; Oliveira, Maria Aparecida de; Santos-Eichler, Rosangela Aparecida; Colli, Lucas Giglio; Jiménez-Altayó, Francesc; Vila, Elisabet; Akamine, Eliana Hiromi ; Dantas, Ana Paula; Ceravolo, Graziela Scalianti; Carvalho, Maria Helena Catelli de
Source: Journal of Pharmacology and Experimental Therapeutics. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAUJO, Priscila Xavier et al. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, v. 362, n. 1, p. 98-107, 2017Tradução . . Disponível em: https://doi.org/10.1124/jpet.117.240465. Acesso em: 09 nov. 2024.
APA
Araujo, P. X., Costa, T. J., Echem, C., Oliveira, M. A. de, Santos-Eichler, R. A., Colli, L. G., et al. (2017). Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, 362( 1), 98-107. doi:10.1124/jpet.117.240465
NLM
Araujo PX, Costa TJ, Echem C, Oliveira MA de, Santos-Eichler RA, Colli LG, Jiménez-Altayó F, Vila E, Akamine EH, Dantas AP, Ceravolo GS, Carvalho MHC de. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats [Internet]. Journal of Pharmacology and Experimental Therapeutics. 2017 ; 362( 1): 98-107.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1124/jpet.117.240465
Vancouver
Araujo PX, Costa TJ, Echem C, Oliveira MA de, Santos-Eichler RA, Colli LG, Jiménez-Altayó F, Vila E, Akamine EH, Dantas AP, Ceravolo GS, Carvalho MHC de. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats [Internet]. Journal of Pharmacology and Experimental Therapeutics. 2017 ; 362( 1): 98-107.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1124/jpet.117.240465
Artigo de periodico
Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products (2016)
Almeida, Paula Campi Locatelli de; Herrera, Bruno Schneider; Jesus, Flavia Neto de; Napolitano, Mauro; Teixeira, Simone Aparecida; Dantas, Aline Maia; Spolidorio, Luis Carlos; Akamine, Eliana Hiromi ; Mayer, Marcia Pinto Alves ; Carvalho, Maria Helena Catelli de; Costa, Soraia Katia Pereira ; Muscará, Marcelo Nicolás
Source: Archives of Oral Biology. Unidade: ICB
Subjects: PERIODONTITE, ENDOTÉLIO, AORTA, DOENÇAS CARDIOVASCULARES, ÓXIDO NÍTRICO, INFLAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Paula Campi Locatelli de et al. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products. Archives of Oral Biology, v. 63, p. 66-74, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.archoralbio.2015.11.022. Acesso em: 09 nov. 2024.
APA
Almeida, P. C. L. de, Herrera, B. S., Jesus, F. N. de, Napolitano, M., Teixeira, S. A., Dantas, A. M., et al. (2016). Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products. Archives of Oral Biology, 63, 66-74. doi:10.1016/j.archoralbio.2015.11.022
NLM
Almeida PCL de, Herrera BS, Jesus FN de, Napolitano M, Teixeira SA, Dantas AM, Spolidorio LC, Akamine EH, Mayer MPA, Carvalho MHC de, Costa SKP, Muscará MN. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products [Internet]. Archives of Oral Biology. 2016 ; 63 66-74.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.archoralbio.2015.11.022
Vancouver
Almeida PCL de, Herrera BS, Jesus FN de, Napolitano M, Teixeira SA, Dantas AM, Spolidorio LC, Akamine EH, Mayer MPA, Carvalho MHC de, Costa SKP, Muscará MN. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products [Internet]. Archives of Oral Biology. 2016 ; 63 66-74.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.archoralbio.2015.11.022
Artigo de periodico
H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
Costa, Rafael M.; Filgueira, Fernando P.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena C.; Akamine, Eliana Hiromi ; Lobato, Nubia S.
Source: Vascular Pharmacology. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 09 nov. 2024.
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Artigo de periodico
Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a (2015)
Queiroz, Eveline A. I. F. de; Akamine, Eliana Hiromi ; Carvalho, Maria Helena Catelli de; Sampaio, Sandra Concuzzo; Fortes, Zuleica Bruno
Source: Life Sciences. Unidade: ICB
Subjects: FARMACOLOGIA, TUMOR DE WALKER ANIMAL, RATOS, OBESIDADE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUEIROZ, Eveline A. I. F. de et al. Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a. Life Sciences, v. 121, p. 78-87, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.11.028. Acesso em: 09 nov. 2024.
APA
Queiroz, E. A. I. F. de, Akamine, E. H., Carvalho, M. H. C. de, Sampaio, S. C., & Fortes, Z. B. (2015). Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a. Life Sciences, 121, 78-87. doi:10.1016/j.lfs.2014.11.028
NLM
Queiroz EAIF de, Akamine EH, Carvalho MHC de, Sampaio SC, Fortes ZB. Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a [Internet]. Life Sciences. 2015 ; 121 78-87.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.lfs.2014.11.028
Vancouver
Queiroz EAIF de, Akamine EH, Carvalho MHC de, Sampaio SC, Fortes ZB. Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a [Internet]. Life Sciences. 2015 ; 121 78-87.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.lfs.2014.11.028
Artigo de periodico
Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats (2015)
Bomfim, G. F.; Echem, Cinthya; Martins, C. B.; Costa, T. J.; Sartoretto, Simone Marcieli; Santos, R. A. dos; Oliveira, M. A.; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo; Webb, R. C.; Carvalho, Maria Helena Catelli de
Source: Life Sciences. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, INFLAMAÇÃO, HIPERTENSÃO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOMFIM, G. F. et al. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, v. 122, p. 1-7, 2015Tradução . . Disponível em: http://lattes.cnpq.br/5055165961832291. Acesso em: 09 nov. 2024.
APA
Bomfim, G. F., Echem, C., Martins, C. B., Costa, T. J., Sartoretto, S. M., Santos, R. A. dos, et al. (2015). Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, 122, 1-7. doi:10.1016/j.lfs.2014.12.001
NLM
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 nov. 09 ] Available from: http://lattes.cnpq.br/5055165961832291
Vancouver
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 nov. 09 ] Available from: http://lattes.cnpq.br/5055165961832291
Artigo de periodico
Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells (2015)
Costa, Tiago Januário da; Ceravolo, Graziela Scalianti; Santos, Rosangela Aparecida dos; Oliveira, Maria Aparecida de; Araújo, Priscila Xavier de; Giaquinto, Luciana Rigueiral; Tostes, Rita de Cássia Aleixo; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli de
Source: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, TESTOSTERONA, ESTROGENOS, AORTA DE ANIMAL, CÉLULAS ENDOTELIAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, v. 308, n. 7, p. H723-H732, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00681.2014. Acesso em: 09 nov. 2024.
APA
Costa, T. J. da, Ceravolo, G. S., Santos, R. A. dos, Oliveira, M. A. de, Araújo, P. X. de, Giaquinto, L. R., et al. (2015). Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, 308( 7), H723-H732. doi:10.1152/ajpheart.00681.2014
NLM
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Vancouver
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Artigo de periodico
Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) (2015)
Echem, Cinthya; Bomfim, Gisele Facholi; Ceravolo, Graziela Scalianti; Oliveira, Maria Aparecida; Eichler, Rosangela Aparecida Santos; Bechara, Luiz Roberto; Veras, Mariana Matera; Saldiva, Paulo Hilário Nascimento ; Ferreira, Julio Cesar Batista ; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli
Source: International Journal of Cardiology. Unidades: ICB, FM
Subjects: ANATOMIA, FARMACOLOGIA, HIPERTENSÃO, RATOS, PRESSÃO SANGUÍNEA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ECHEM, Cinthya et al. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR). International Journal of Cardiology, v. 187, p. 243-245, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2015.03.190. Acesso em: 09 nov. 2024.
APA
Echem, C., Bomfim, G. F., Ceravolo, G. S., Oliveira, M. A., Eichler, R. A. S., Bechara, L. R., et al. (2015). Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR). International Journal of Cardiology, 187, 243-245. doi:10.1016/j.ijcard.2015.03.190
NLM
Echem C, Bomfim GF, Ceravolo GS, Oliveira MA, Eichler RAS, Bechara LR, Veras MM, Saldiva PHN, Ferreira JCB, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) [Internet]. International Journal of Cardiology. 2015 ; 187 243-245.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ijcard.2015.03.190
Vancouver
Echem C, Bomfim GF, Ceravolo GS, Oliveira MA, Eichler RAS, Bechara LR, Veras MM, Saldiva PHN, Ferreira JCB, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) [Internet]. International Journal of Cardiology. 2015 ; 187 243-245.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ijcard.2015.03.190
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Source: Plos One. Unidades: ICB, FMRP
Subjects: ANATOMIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. Plos One, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 09 nov. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. Plos One, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. Plos One. 2014 ; 9( 11):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. Plos One. 2014 ; 9( 11):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats (2014)
Hagihara, Graziela Neves; Lobato, Núbia Souza; Filgueira, Fernando P.; Akamine, Eliana Hiromi ; Aragão, Danielle S.; Casarini, Dulce Elena; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Source: PLOS ONE. Unidade: ICB
Subjects: FARMACOLOGIA, ANGIOTENSINA II, OBESIDADE, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HAGIHARA, Graziela Neves et al. Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats. PLOS ONE, v. 9, n. 8, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone. Acesso em: 09 nov. 2024.
APA
Hagihara, G. N., Lobato, N. S., Filgueira, F. P., Akamine, E. H., Aragão, D. S., Casarini, D. E., et al. (2014). Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats. PLOS ONE, 9( 8), 1-10. doi:10.1371/journal.pone
NLM
Hagihara GN, Lobato NS, Filgueira FP, Akamine EH, Aragão DS, Casarini DE, Carvalho MHC de, Fortes ZB. Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats [Internet]. PLOS ONE. 2014 ; 9( 8): 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone
Vancouver
Hagihara GN, Lobato NS, Filgueira FP, Akamine EH, Aragão DS, Casarini DE, Carvalho MHC de, Fortes ZB. Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats [Internet]. PLOS ONE. 2014 ; 9( 8): 1-10.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone
Artigo de periodico
Influence of aerobic training on the reduced vasoconstriction to angiotensin II in rats exposed to intrauterine growth restriction: possible role of oxidative stress and AT2 receptor of angiotensin II (2014)
Oliveira, Vanessa; Akamine, Eliana Hiromi ; Carvalho, Maria Helena Catelli de; Michelini, Lisete Compagno ; Fortes, Zuleica Bruno; Cunha, Tatiana Sousa; Franco, Maria do Carmo
Source: PLOS ONE. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Vanessa et al. Influence of aerobic training on the reduced vasoconstriction to angiotensin II in rats exposed to intrauterine growth restriction: possible role of oxidative stress and AT2 receptor of angiotensin II. PLOS ONE, v. 9, n. 11, p. 1-11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0113035. Acesso em: 09 nov. 2024.
APA
Oliveira, V., Akamine, E. H., Carvalho, M. H. C. de, Michelini, L. C., Fortes, Z. B., Cunha, T. S., & Franco, M. do C. (2014). Influence of aerobic training on the reduced vasoconstriction to angiotensin II in rats exposed to intrauterine growth restriction: possible role of oxidative stress and AT2 receptor of angiotensin II. PLOS ONE, 9( 11), 1-11. doi:10.1371/journal.pone.0113035
NLM
Oliveira V, Akamine EH, Carvalho MHC de, Michelini LC, Fortes ZB, Cunha TS, Franco M do C. Influence of aerobic training on the reduced vasoconstriction to angiotensin II in rats exposed to intrauterine growth restriction: possible role of oxidative stress and AT2 receptor of angiotensin II [Internet]. PLOS ONE. 2014 ; 9( 11): 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0113035
Vancouver
Oliveira V, Akamine EH, Carvalho MHC de, Michelini LC, Fortes ZB, Cunha TS, Franco M do C. Influence of aerobic training on the reduced vasoconstriction to angiotensin II in rats exposed to intrauterine growth restriction: possible role of oxidative stress and AT2 receptor of angiotensin II [Internet]. PLOS ONE. 2014 ; 9( 11): 1-11.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0113035
Artigo de periodico
Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats (2013)
Sartoretto, Simone Marcieli; Santos-Eichler, Rosângela; Tostes, Rita de Cássia Aleixo; Carvalho, Maria Helena Catelli de; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno
Source: Journal of Diabetes. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORETTO, Simone Marcieli et al. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, v. 5, n. 2, p. 197-206, 2013Tradução . . Acesso em: 09 nov. 2024.
APA
Sartoretto, S. M., Santos-Eichler, R., Tostes, R. de C. A., Carvalho, M. H. C. de, Akamine, E. H., & Fortes, Z. B. (2013). Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, 5( 2), 197-206.
NLM
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 nov. 09 ]
Vancouver
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 nov. 09 ]
Artigo de periodico
Mechanisms of endothelial dysfunction in obesity-associated hypertension (2012)
Lobato, Nubia de Souza; Filgueira, Fernando Paranaiba; Akamine, Eliana Hiromi ; Passaglia, Rita de Cassia Aleixo Tostes; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Source: Brazilian Journal of Medical and Biological Research. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOBATO, Nubia de Souza et al. Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, v. 45, n. 5, p. 392-400, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500058. Acesso em: 09 nov. 2024.
APA
Lobato, N. de S., Filgueira, F. P., Akamine, E. H., Passaglia, R. de C. A. T., Carvalho, M. H. C. de, & Fortes, Z. B. (2012). Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, 45( 5), 392-400. doi:10.1590/S0100-879X2012007500058
NLM
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Vancouver
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Artigo de periodico
Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats (2012)
Bomfim, Gisele Facholi; Santos, Rosangela A. dos; Oliveira, Maria Aparecida; Giachini, Fernanda R.; Akamine, Eliana Hiromi ; Passaglia, Rita de Cássia Aleixo Tostes; Fortes, Zuleica Bruno; Webb, R. Clinton; Carvalho, Maria Helena Catelli de
Source: Clinical Science. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOMFIM, Gisele Facholi et al. Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats. Clinical Science, v. 122, n. 11, p. 535-543, 2012Tradução . . Disponível em: https://doi.org/10.1042/CS20110523. Acesso em: 09 nov. 2024.
APA
Bomfim, G. F., Santos, R. A. dos, Oliveira, M. A., Giachini, F. R., Akamine, E. H., Passaglia, R. de C. A. T., et al. (2012). Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats. Clinical Science, 122( 11), 535-543. doi:10.1042/CS20110523
NLM
Bomfim GF, Santos RA dos, Oliveira MA, Giachini FR, Akamine EH, Passaglia R de CAT, Fortes ZB, Webb RC, Carvalho MHC de. Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats [Internet]. Clinical Science. 2012 ; 122( 11): 535-543.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1042/CS20110523
Vancouver
Bomfim GF, Santos RA dos, Oliveira MA, Giachini FR, Akamine EH, Passaglia R de CAT, Fortes ZB, Webb RC, Carvalho MHC de. Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats [Internet]. Clinical Science. 2012 ; 122( 11): 535-543.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1042/CS20110523
Artigo de periodico
Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats (2012)
Lobato, Núbia de Souza; Filgueira, Fernando Paranaiba; Hagihara, Graziela Neves; Akamine, Eliana Hiromi ; Pariz, J. R.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Source: Life Sciences. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOBATO, Núbia de Souza et al. Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats. Life Sciences, v. 90, n. 5-6, p. 228-235, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2011.11.005. Acesso em: 09 nov. 2024.
APA
Lobato, N. de S., Filgueira, F. P., Hagihara, G. N., Akamine, E. H., Pariz, J. R., Passaglia, R. de C. A. T., et al. (2012). Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats. Life Sciences, 90( 5-6), 228-235. doi:10.1016/j.lfs.2011.11.005
NLM
Lobato N de S, Filgueira FP, Hagihara GN, Akamine EH, Pariz JR, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats [Internet]. Life Sciences. 2012 ; 90( 5-6): 228-235.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.lfs.2011.11.005
Vancouver
Lobato N de S, Filgueira FP, Hagihara GN, Akamine EH, Pariz JR, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats [Internet]. Life Sciences. 2012 ; 90( 5-6): 228-235.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.lfs.2011.11.005

USP Schools

ReP

Filtros

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Indexado em

Non normalized affiliation