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Artigo de periodico
Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice (2017)
Masi, Laureane Nunes; Martins, Amanda Roque; Crisma, Amanda Rabello; Amaral, Cátia Lira do; Davanso, Mariana Rodrigues; Serdan, Tamires Duarte Afonso; Sá, Roberta Dourado Cavalcante da Cunha de; Cruz, Maysa Mariana; Alonso-Vale, Maria Isabel Cardoso; Torres, Rosângela Pavan; Mancini-Filho, Jorge ; Pereira, Joice Naiara Bertaglia; Righetti, Marta Maria da Silva; Liberti, Edson Aparecido; Hirabara, Sandro Massao; Curi, Rui
Source: Scientific Reports. Unidades: ICB, FCF
Assunto: ANATOMIA
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ABNT
MASI, Laureane Nunes et al. Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice. Scientific Reports, v. 7, n. 1, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-04308-1. Acesso em: 07 nov. 2024.
APA
Masi, L. N., Martins, A. R., Crisma, A. R., Amaral, C. L. do, Davanso, M. R., Serdan, T. D. A., et al. (2017). Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice. Scientific Reports, 7( 1), 1-10. doi:10.1038/s41598-017-04308-1
NLM
Masi LN, Martins AR, Crisma AR, Amaral CL do, Davanso MR, Serdan TDA, Sá RDC da C de, Cruz MM, Alonso-Vale MIC, Torres RP, Mancini-Filho J, Pereira JNB, Righetti MM da S, Liberti EA, Hirabara SM, Curi R. Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice [Internet]. Scientific Reports. 2017 ; 7( 1): 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/s41598-017-04308-1
Vancouver
Masi LN, Martins AR, Crisma AR, Amaral CL do, Davanso MR, Serdan TDA, Sá RDC da C de, Cruz MM, Alonso-Vale MIC, Torres RP, Mancini-Filho J, Pereira JNB, Righetti MM da S, Liberti EA, Hirabara SM, Curi R. Combination of a high-fat diet with sweetened condensed milk exacerbates inflammation and insulin resistance induced by each separately in mice [Internet]. Scientific Reports. 2017 ; 7( 1): 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/s41598-017-04308-1
Artigo de periodico
Prophylactic supplementation of Bifidobacterium longum 51A protects mice from ovariectomy-induced exacerbated allergic airway inflammation and airway hyperrresponsiveness (2017)
Mendez, Eduardo; Accetturi, Beatriz Golegã; Thomas, Andrew Maltez; Martins, Flaviano dos Santos; Crisma, Amanda Rabello; Murata, Gilson Masahiro; Braga, Tárcio Teodoro; Câmara, Niels Olsen Saraiva ; Franco, Adriana Lino dos Santos; Setubal, João Carlos ; Ribeiro, Willian R.; Valduga, Claudete Justina; Curi, Rui; Dias Neto, Emmanuel; Lima, Wothan Tavares de ; Ferreira, Caroline Marcondes
Source: Frontiers in Microbiology. Unidades: ICB, IQ
Subjects: PROBIÓTICOS, OVARIECTOMIA, HIPERSENSIBILIDADE, ASMA, CAMUNDONGOS
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MENDEZ, Eduardo et al. Prophylactic supplementation of Bifidobacterium longum 51A protects mice from ovariectomy-induced exacerbated allergic airway inflammation and airway hyperrresponsiveness. Frontiers in Microbiology, v. 8, p. 1-14 art. 1732, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.01732. Acesso em: 07 nov. 2024.
APA
Mendez, E., Accetturi, B. G., Thomas, A. M., Martins, F. dos S., Crisma, A. R., Murata, G. M., et al. (2017). Prophylactic supplementation of Bifidobacterium longum 51A protects mice from ovariectomy-induced exacerbated allergic airway inflammation and airway hyperrresponsiveness. Frontiers in Microbiology, 8, 1-14 art. 1732. doi:10.3389/fmicb.2017.01732
NLM
Mendez E, Accetturi BG, Thomas AM, Martins F dos S, Crisma AR, Murata GM, Braga TT, Câmara NOS, Franco AL dos S, Setubal JC, Ribeiro WR, Valduga CJ, Curi R, Dias Neto E, Lima WT de, Ferreira CM. Prophylactic supplementation of Bifidobacterium longum 51A protects mice from ovariectomy-induced exacerbated allergic airway inflammation and airway hyperrresponsiveness [Internet]. Frontiers in Microbiology. 2017 ; 8 1-14 art. 1732.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fmicb.2017.01732
Vancouver
Mendez E, Accetturi BG, Thomas AM, Martins F dos S, Crisma AR, Murata GM, Braga TT, Câmara NOS, Franco AL dos S, Setubal JC, Ribeiro WR, Valduga CJ, Curi R, Dias Neto E, Lima WT de, Ferreira CM. Prophylactic supplementation of Bifidobacterium longum 51A protects mice from ovariectomy-induced exacerbated allergic airway inflammation and airway hyperrresponsiveness [Internet]. Frontiers in Microbiology. 2017 ; 8 1-14 art. 1732.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fmicb.2017.01732
Artigo de periodico
Oral administration of antioxidants improves skin wound healing in diabetic mice (2016)
Pessoa , Ana Flávia Marça; Florim, Juliana Costa; Rodrigues, Hosana Gomes; Oliveira, Vinicius de Andrade; Teixeira, Simone Aparecida; Vitzel, Kaio Fernando; Curi, Rui; Câmara, Niels Olsen Saraiva ; Muscará, Marcelo Nicolás ; Lamers, Marcelo Lazzaron; Santos, Marinilce Fagundes dos
Source: Wound Repair and Regeneration. Unidade: ICB
Subjects: ANTIOXIDANTES, CICATRIZAÇÃO, RATOS, DIABETES MELLITUS
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ABNT
PESSOA , Ana Flávia Marça et al. Oral administration of antioxidants improves skin wound healing in diabetic mice. Wound Repair and Regeneration, v. 24, n. 6, p. 981-993, 2016Tradução . . Disponível em: https://doi.org/10.1111/wrr.12486. Acesso em: 07 nov. 2024.
APA
Pessoa , A. F. M., Florim, J. C., Rodrigues, H. G., Oliveira, V. de A., Teixeira, S. A., Vitzel, K. F., et al. (2016). Oral administration of antioxidants improves skin wound healing in diabetic mice. Wound Repair and Regeneration, 24( 6), 981-993. doi:10.1111/wrr.12486
NLM
Pessoa AFM, Florim JC, Rodrigues HG, Oliveira V de A, Teixeira SA, Vitzel KF, Curi R, Câmara NOS, Muscará MN, Lamers ML, Santos MF dos. Oral administration of antioxidants improves skin wound healing in diabetic mice [Internet]. Wound Repair and Regeneration. 2016 ; 24( 6): 981-993.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/wrr.12486
Vancouver
Pessoa AFM, Florim JC, Rodrigues HG, Oliveira V de A, Teixeira SA, Vitzel KF, Curi R, Câmara NOS, Muscará MN, Lamers ML, Santos MF dos. Oral administration of antioxidants improves skin wound healing in diabetic mice [Internet]. Wound Repair and Regeneration. 2016 ; 24( 6): 981-993.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/wrr.12486
Artigo de periodico
Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats (2016)
Rodrigues, Hosana Gomes; Vinolo, Marco Aurélio Ramirez; Sato, Fabio Takeo; Magdalon, Juliana; Kuhl, Carolina Meneguetti Cintra; Yamagata, Ana Sayuri; Pessoa, Ana Flávia Marçal; Moraes, Gabriélla Malheiros; Santos, Marinilce Fagundes dos ; Lima, Camila; Farsky, Sandra Helena Poliselli ; Câmara, Niels Olsen Saraiva ; Williner, Maria Rosa; Bernal, Claudio Adrián; Calder,Philip C; Curi, Rui
Source: PLOS ONE. Unidades: ICB, FCF
Subjects: ÁCIDO LINOLEICO, RATOS, CICATRIZAÇÃO
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ABNT
RODRIGUES, Hosana Gomes et al. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats. PLOS ONE, v. 11, n. 10, p. e0165115, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0165115. Acesso em: 07 nov. 2024.
APA
Rodrigues, H. G., Vinolo, M. A. R., Sato, F. T., Magdalon, J., Kuhl, C. M. C., Yamagata, A. S., et al. (2016). Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats. PLOS ONE, 11( 10), e0165115. doi:10.1371/journal.pone.0165115
NLM
Rodrigues HG, Vinolo MAR, Sato FT, Magdalon J, Kuhl CMC, Yamagata AS, Pessoa AFM, Moraes GM, Santos MF dos, Lima C, Farsky SHP, Câmara NOS, Williner MR, Bernal CA, Calder PC, Curi R. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats [Internet]. PLOS ONE. 2016 ; 11( 10): e0165115.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0165115
Vancouver
Rodrigues HG, Vinolo MAR, Sato FT, Magdalon J, Kuhl CMC, Yamagata AS, Pessoa AFM, Moraes GM, Santos MF dos, Lima C, Farsky SHP, Câmara NOS, Williner MR, Bernal CA, Calder PC, Curi R. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats [Internet]. PLOS ONE. 2016 ; 11( 10): e0165115.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0165115
Artigo de periodico
Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats (2016)
Munhoz, Ana Cláudia; Riva, Patrícia; Simões, Daniel; Curi, Rui; Carpinelli, Angelo Rafael
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MUNHOZ, Ana Cláudia et al. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats. PLOS ONE, v. 11, n. 6, p. 1-17, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0158166. Acesso em: 07 nov. 2024.
APA
Munhoz, A. C., Riva, P., Simões, D., Curi, R., & Carpinelli, A. R. (2016). Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats. PLOS ONE, 11( 6), 1-17. doi:10.1371/journal.pone.0158166
NLM
Munhoz AC, Riva P, Simões D, Curi R, Carpinelli AR. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats [Internet]. PLOS ONE. 2016 ; 11( 6): 1-17.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0158166
Vancouver
Munhoz AC, Riva P, Simões D, Curi R, Carpinelli AR. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats [Internet]. PLOS ONE. 2016 ; 11( 6): 1-17.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0158166
Artigo de periodico
Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies (2015)
Lucena, Camila Ferraz; Roma, Leticia Prates; Graciano, Maria Fernanda Rodrigues; Veras, Katherine; Simões, Daniel; Curi, Rui; Carpinelli, Angelo Rafael
Source: Pancreas. Unidade: ICB
Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, SUPLEMENTOS ALIMENTARES PARA ANIMAIS, ÓLEOS DE PEIUXE
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ABNT
LUCENA, Camila Ferraz et al. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, v. 44, n. 2, p. 287-295, 2015Tradução . . Disponível em: https://doi.org/10.1097/MPA.0000000000000249. Acesso em: 07 nov. 2024.
APA
Lucena, C. F., Roma, L. P., Graciano, M. F. R., Veras, K., Simões, D., Curi, R., & Carpinelli, A. R. (2015). Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, 44( 2), 287-295. doi:10.1097/MPA.0000000000000249
NLM
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Vancouver
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Artigo de periodico
Glucose and fatty acid metabolism in infarcted heart from streptozotocin-induced diabetic rats after 2 weeks of tissue remodeling (2015)
Malfitano, Christiane; Souza Júnior, Alcione Lescano de; Carbonaro, Mariana; Bolsoni-Lopes, Andressa; Figueroa, Diego; Souza, Leandro Ezequiel de; Silva, Kleiton Augusto Santos; Consolim-Colombo, Fernanda; Curi, Rui; Irigoyen, Maria Claudia
Source: Cardiovascular Diabetology. Unidade: ICB
Subjects: FISIOLOGIA, ÁCIDOS GRAXOS, METABOLISMO ANIMAL, RATOS
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ABNT
MALFITANO, Christiane et al. Glucose and fatty acid metabolism in infarcted heart from streptozotocin-induced diabetic rats after 2 weeks of tissue remodeling. Cardiovascular Diabetology, v. 14, n. 149, p. 1-10, 2015Tradução . . Disponível em: https://doi.org/10.1186/s12933-015-0308-y. Acesso em: 07 nov. 2024.
APA
Malfitano, C., Souza Júnior, A. L. de, Carbonaro, M., Bolsoni-Lopes, A., Figueroa, D., Souza, L. E. de, et al. (2015). Glucose and fatty acid metabolism in infarcted heart from streptozotocin-induced diabetic rats after 2 weeks of tissue remodeling. Cardiovascular Diabetology, 14( 149), 1-10. doi:10.1186/s12933-015-0308-y
NLM
Malfitano C, Souza Júnior AL de, Carbonaro M, Bolsoni-Lopes A, Figueroa D, Souza LE de, Silva KAS, Consolim-Colombo F, Curi R, Irigoyen MC. Glucose and fatty acid metabolism in infarcted heart from streptozotocin-induced diabetic rats after 2 weeks of tissue remodeling [Internet]. Cardiovascular Diabetology. 2015 ; 14( 149): 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1186/s12933-015-0308-y
Vancouver
Malfitano C, Souza Júnior AL de, Carbonaro M, Bolsoni-Lopes A, Figueroa D, Souza LE de, Silva KAS, Consolim-Colombo F, Curi R, Irigoyen MC. Glucose and fatty acid metabolism in infarcted heart from streptozotocin-induced diabetic rats after 2 weeks of tissue remodeling [Internet]. Cardiovascular Diabetology. 2015 ; 14( 149): 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1186/s12933-015-0308-y
Artigo de periodico
NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells (2015)
Kuwabara, Wilson Mitsuo Tatagiba; Zhang, Liling; Schuiki, Irmgard; Curi, Rui; Volchuk, Allen; Loureiro, Tatiana Carolina Alba
Source: PLOS ONE. Unidade: ICB
Subjects: FISIOLOGIA, ATIVAÇÃO ENZIMÁTICA, NEUTRÓFILOS, LEUCEMIA, RETICULO ENDOPLASMÁTICO
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ABNT
KUWABARA, Wilson Mitsuo Tatagiba et al. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells. PLOS ONE, v. 10, n. 2, p. 1-15, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0116410. Acesso em: 07 nov. 2024.
APA
Kuwabara, W. M. T., Zhang, L., Schuiki, I., Curi, R., Volchuk, A., & Loureiro, T. C. A. (2015). NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells. PLOS ONE, 10( 2), 1-15. doi:10.1371/journal.pone.0116410
NLM
Kuwabara WMT, Zhang L, Schuiki I, Curi R, Volchuk A, Loureiro TCA. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells [Internet]. PLOS ONE. 2015 ; 10( 2): 1-15.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0116410
Vancouver
Kuwabara WMT, Zhang L, Schuiki I, Curi R, Volchuk A, Loureiro TCA. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells [Internet]. PLOS ONE. 2015 ; 10( 2): 1-15.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0116410
Artigo de periodico
Selection of reference genes for expression analyses in liver of rats with impaired glucose metabolism (2015)
Hernández, Alfonso H.; Curi, Rui; Salazar, Luis Antonio
Source: International Journal of Clinical and Experimental Pathology. Unidade: ICB
Subjects: FISIOLOGIA, EXPRESSÃO GÊNICA, GLICOSE, RATOS, METABOLISMO ANIMAL
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ABNT
HERNÁNDEZ, Alfonso H. e CURI, Rui e SALAZAR, Luis Antonio. Selection of reference genes for expression analyses in liver of rats with impaired glucose metabolism. International Journal of Clinical and Experimental Pathology, v. 8, n. 4, p. 3946-3954, 2015Tradução . . Disponível em: http://www.ijcep.com/files/ijcep0006758.pdf. Acesso em: 07 nov. 2024.
APA
Hernández, A. H., Curi, R., & Salazar, L. A. (2015). Selection of reference genes for expression analyses in liver of rats with impaired glucose metabolism. International Journal of Clinical and Experimental Pathology, 8( 4), 3946-3954. Recuperado de http://www.ijcep.com/files/ijcep0006758.pdf
NLM
Hernández AH, Curi R, Salazar LA. Selection of reference genes for expression analyses in liver of rats with impaired glucose metabolism [Internet]. International Journal of Clinical and Experimental Pathology. 2015 ; 8( 4): 3946-3954.[citado 2024 nov. 07 ] Available from: http://www.ijcep.com/files/ijcep0006758.pdf
Vancouver
Hernández AH, Curi R, Salazar LA. Selection of reference genes for expression analyses in liver of rats with impaired glucose metabolism [Internet]. International Journal of Clinical and Experimental Pathology. 2015 ; 8( 4): 3946-3954.[citado 2024 nov. 07 ] Available from: http://www.ijcep.com/files/ijcep0006758.pdf
Artigo de periodico
The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management (2015)
Krause, Mauricio; Heck, Thiago Gomes; Bittencourt, Aline; Scomazzon, Sofia Pizzato; Newsholme, Philip; Curi, Rui; Bittencourt Junior, Paulo Ivo Homem de
Source: Mediators of Inflammation. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, EQUILIBRIO MUSCULOSQUELÉTICO, INFLAMAÇÃO, PROTEINAS DO CHOQUE TÉRMICO
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ABNT
KRAUSE, Mauricio et al. The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management. Mediators of Inflammation, v. 2015, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/249205. Acesso em: 07 nov. 2024.
APA
Krause, M., Heck, T. G., Bittencourt, A., Scomazzon, S. P., Newsholme, P., Curi, R., & Bittencourt Junior, P. I. H. de. (2015). The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management. Mediators of Inflammation, 2015. doi:10.1155/2015/249205
NLM
Krause M, Heck TG, Bittencourt A, Scomazzon SP, Newsholme P, Curi R, Bittencourt Junior PIH de. The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management [Internet]. Mediators of Inflammation. 2015 ; 2015[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2015/249205
Vancouver
Krause M, Heck TG, Bittencourt A, Scomazzon SP, Newsholme P, Curi R, Bittencourt Junior PIH de. The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management [Internet]. Mediators of Inflammation. 2015 ; 2015[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2015/249205
Artigo de periodico
Overload-induced skeletal muscle hypertrophy is not impaired in STZ-diabetic rats (2015)
Fortes, Marco Aurélio Salomão; Pinheiro, Carlos Hermano Justa; Ferreira, Lucas Guimarães; Vitzel, Kaio Fernando; Vasconcelos, Diogo Antonio Alves de; Curi, Rui
Source: Physiological Reports. Unidade: ICB
Subjects: FISIOLOGIA, RATOS, SISTEMA MÚSCULO ESQUELÉTICO
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ABNT
FORTES, Marco Aurélio Salomão et al. Overload-induced skeletal muscle hypertrophy is not impaired in STZ-diabetic rats. Physiological Reports, v. 3, n. 7, p. 1-16, 2015Tradução . . Disponível em: https://doi.org/10.14814/phy2.12457. Acesso em: 07 nov. 2024.
APA
Fortes, M. A. S., Pinheiro, C. H. J., Ferreira, L. G., Vitzel, K. F., Vasconcelos, D. A. A. de, & Curi, R. (2015). Overload-induced skeletal muscle hypertrophy is not impaired in STZ-diabetic rats. Physiological Reports, 3( 7), 1-16. doi:10.14814/phy2.12457
NLM
Fortes MAS, Pinheiro CHJ, Ferreira LG, Vitzel KF, Vasconcelos DAA de, Curi R. Overload-induced skeletal muscle hypertrophy is not impaired in STZ-diabetic rats [Internet]. Physiological Reports. 2015 ; 3( 7): 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.14814/phy2.12457
Vancouver
Fortes MAS, Pinheiro CHJ, Ferreira LG, Vitzel KF, Vasconcelos DAA de, Curi R. Overload-induced skeletal muscle hypertrophy is not impaired in STZ-diabetic rats [Internet]. Physiological Reports. 2015 ; 3( 7): 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.14814/phy2.12457
Artigo de periodico
Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids (2015)
Fiamoncini, Jarlei ; Lima, Thais Martins de; Hirabara, Sandro Massao; Ecker, Josef; Gorjão, Renata; Romanatto, Talita; Elolimy, Ahmed; Worsch, Stefanie; Laumen, Helmut; Bader, Bernhard; Daniel, Hannelore; Curi, Rui
Source: Molecular Nutrition & Food Research. Unidade: ICB
Subjects: FISIOLOGIA, OLEO DE PEIXE, CAMUNDONGOS, PEROXISSOMOS, OXIDAÇÃO, METABOLISMO ANIMAL
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ABNT
FIAMONCINI, Jarlei et al. Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids. Molecular Nutrition & Food Research, v. 59, n. 8, p. 1573-1583, 2015Tradução . . Disponível em: https://doi.org/10.1002/mnfr.201400743. Acesso em: 07 nov. 2024.
APA
Fiamoncini, J., Lima, T. M. de, Hirabara, S. M., Ecker, J., Gorjão, R., Romanatto, T., et al. (2015). Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids. Molecular Nutrition & Food Research, 59( 8), 1573-1583. doi:10.1002/mnfr.201400743
NLM
Fiamoncini J, Lima TM de, Hirabara SM, Ecker J, Gorjão R, Romanatto T, Elolimy A, Worsch S, Laumen H, Bader B, Daniel H, Curi R. Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids [Internet]. Molecular Nutrition & Food Research. 2015 ; 59( 8): 1573-1583.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1002/mnfr.201400743
Vancouver
Fiamoncini J, Lima TM de, Hirabara SM, Ecker J, Gorjão R, Romanatto T, Elolimy A, Worsch S, Laumen H, Bader B, Daniel H, Curi R. Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids [Internet]. Molecular Nutrition & Food Research. 2015 ; 59( 8): 1573-1583.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1002/mnfr.201400743
Artigo de periodico
Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells (2014)
Martín-Rufián, Mercedes; Nascimento-Gomes, Renata; Higuero, Ana; Crisma, Amanda R.; Campos-Sandoval, José A.; Gómez-Garcia, María C.; Cardona, Carolina; Cheng, Tzuling; Lobo, Carolina; Segura, Juan A.; Alonso, Francisco J.; Szeliga, Monika; Albrecht, Jan; Curi, Rui; Márquez, Javier; Colquhoun, Alison ; DeBerardinis, Ralph J.; Matés, José M.
Source: Journal of Molecular Medicine. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA
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ABNT
MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 07 nov. 2024.
APA
Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2
NLM
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Vancouver
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Artigo de periodico
An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages (2014)
Moreira, Vanessa; Lomonte, Bruno; Vinolo, Marco Aurélio Ramirez; Curi, Rui; Gutiérrez, José María; Teixeira, Catarina
Source: Mediators of Inflammation. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOREIRA, Vanessa et al. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/105879. Acesso em: 07 nov. 2024.
APA
Moreira, V., Lomonte, B., Vinolo, M. A. R., Curi, R., Gutiérrez, J. M., & Teixeira, C. (2014). An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, 2014, 1-10. doi:10.1155/2014/105879
NLM
Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2014/105879
Vancouver
Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2014/105879
Artigo de periodico
DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice (2014)
Amaral, Catia Lira do; Crisma, Amanda Rabello; Masi, Laureane Nunes; Martins, Amanda Roque; Hirabara, Sandro Massao; Curi, Rui
Source: Journal of Nutrigenetics and Nutrigenomics. Unidade: ICB
Subjects: CAMUNDONGOS, MÚSCULO ESQUELÉTICO, ÓLEOS DE PEIXE, TRANSCRIÇÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMARAL, Catia Lira do et al. DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice. Journal of Nutrigenetics and Nutrigenomics, v. 7, n. 4-6, p. 314-326, 2014Tradução . . Disponível em: https://doi.org/10.1159/000381777. Acesso em: 07 nov. 2024.
APA
Amaral, C. L. do, Crisma, A. R., Masi, L. N., Martins, A. R., Hirabara, S. M., & Curi, R. (2014). DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice. Journal of Nutrigenetics and Nutrigenomics, 7( 4-6), 314-326. doi:10.1159/000381777
NLM
Amaral CL do, Crisma AR, Masi LN, Martins AR, Hirabara SM, Curi R. DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice [Internet]. Journal of Nutrigenetics and Nutrigenomics. 2014 ; 7( 4-6): 314-326.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1159/000381777
Vancouver
Amaral CL do, Crisma AR, Masi LN, Martins AR, Hirabara SM, Curi R. DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice [Internet]. Journal of Nutrigenetics and Nutrigenomics. 2014 ; 7( 4-6): 314-326.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1159/000381777
Artigo de periodico
The central role of the gut microbiota in chronic inflammatory diseases (2014)
Ferreira, Caroline Marcantonio; Vieira, Angélica Thomaz; Vinolo, Marco Aurelio Ramirez ; Oliveira, Fernando A. ; Martins, Flaviano dos Santos ; Curi, Rui
Source: Journal of Immunology Research. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA CELULAR, SISTEMA IMUNE, CAMUNDONGOS, DOENÇAS IMUNOLÓGICAS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Caroline Marcantonio et al. The central role of the gut microbiota in chronic inflammatory diseases. Journal of Immunology Research, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/689492. Acesso em: 07 nov. 2024.
APA
Ferreira, C. M., Vieira, A. T., Vinolo, M. A. R., Oliveira, F. A., Martins, F. dos S., & Curi, R. (2014). The central role of the gut microbiota in chronic inflammatory diseases. Journal of Immunology Research, 2014. doi:10.1155/2014/689492
NLM
Ferreira CM, Vieira AT, Vinolo MAR, Oliveira FA, Martins F dos S, Curi R. The central role of the gut microbiota in chronic inflammatory diseases [Internet]. Journal of Immunology Research. 2014 ; 2014[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2014/689492
Vancouver
Ferreira CM, Vieira AT, Vinolo MAR, Oliveira FA, Martins F dos S, Curi R. The central role of the gut microbiota in chronic inflammatory diseases [Internet]. Journal of Immunology Research. 2014 ; 2014[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2014/689492
Artigo de periodico
DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil (2014)
Amaral, Cátia Lira do; Milagro, Fermín I.; Curi, Rui; Martínez, J. Alfredo
Source: BioMed Research International. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMARAL, Cátia Lira do et al. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil. BioMed Research International, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/675021. Acesso em: 07 nov. 2024.
APA
Amaral, C. L. do, Milagro, F. I., Curi, R., & Martínez, J. A. (2014). DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil. BioMed Research International, 2014, 1-10. doi:10.1155/2014/675021
NLM
Amaral CL do, Milagro FI, Curi R, Martínez JA. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil [Internet]. BioMed Research International. 2014 ; 2014 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2014/675021
Vancouver
Amaral CL do, Milagro FI, Curi R, Martínez JA. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil [Internet]. BioMed Research International. 2014 ; 2014 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2014/675021
Artigo de periodico
Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice (2014)
Romanatto, Talita; Fiamoncini, Jarlei ; Wang, Bin; Curi, Rui; Kang, Jing X.
Source: Biochimica et Biophysica Acta: Molecular Basis Diseases. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROMANATTO, Talita et al. Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice. Biochimica et Biophysica Acta: Molecular Basis Diseases, v. 1842, n. 2, p. 186-191, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2013.10.017. Acesso em: 07 nov. 2024.
APA
Romanatto, T., Fiamoncini, J., Wang, B., Curi, R., & Kang, J. X. (2014). Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice. Biochimica et Biophysica Acta: Molecular Basis Diseases, 1842( 2), 186-191. doi:10.1016/j.bbadis.2013.10.017
NLM
Romanatto T, Fiamoncini J, Wang B, Curi R, Kang JX. Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice [Internet]. Biochimica et Biophysica Acta: Molecular Basis Diseases. 2014 ; 1842( 2): 186-191.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.bbadis.2013.10.017
Vancouver
Romanatto T, Fiamoncini J, Wang B, Curi R, Kang JX. Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice [Internet]. Biochimica et Biophysica Acta: Molecular Basis Diseases. 2014 ; 1842( 2): 186-191.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.bbadis.2013.10.017
Artigo de periodico
Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects (2013)
Felipe, E. T.; Maestri, J. S.; Kanunfre, C. C.; Curi, Rui; Newsholme, Philip; Carpinelli, Angelo Rafael ; Oliveira-Emilio, H. R.
Source: Natural Product Research. Unidade: ICB
Subjects: FISIOLOGIA, PRODUTOS NATURAIS, INSULINA, FLAVONÓIDES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FELIPE, E. T. et al. Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects. Natural Product Research, v. 27, n. 10, p. 925-928, 2013Tradução . . Disponível em: https://doi.org/10.1080/14786419.2012.671315. Acesso em: 07 nov. 2024.
APA
Felipe, E. T., Maestri, J. S., Kanunfre, C. C., Curi, R., Newsholme, P., Carpinelli, A. R., & Oliveira-Emilio, H. R. (2013). Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects. Natural Product Research, 27( 10), 925-928. doi:10.1080/14786419.2012.671315
NLM
Felipe ET, Maestri JS, Kanunfre CC, Curi R, Newsholme P, Carpinelli AR, Oliveira-Emilio HR. Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects [Internet]. Natural Product Research. 2013 ; 27( 10): 925-928.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1080/14786419.2012.671315
Vancouver
Felipe ET, Maestri JS, Kanunfre CC, Curi R, Newsholme P, Carpinelli AR, Oliveira-Emilio HR. Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects [Internet]. Natural Product Research. 2013 ; 27( 10): 925-928.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1080/14786419.2012.671315
Artigo de periodico
Effects of melatonin on DNA damage induced by cyclophosphamide in rats (2013)
Ferreira, S. G.; Peliciari-Garcia, R. A.; Takahashi-Hyodo, S. A.; Rodrigues, Alice Cristina ; Amaral, F. G.; Berra, Carolina Maria; Bordin, Silvana ; Curi, Rui; Cipolla-Neto, Jose
Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, S. G. et al. Effects of melatonin on DNA damage induced by cyclophosphamide in rats. Brazilian Journal of Medical and Biological Research, v. 46, n. 3, p. 278-286, 2013Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20122230. Acesso em: 07 nov. 2024.
APA
Ferreira, S. G., Peliciari-Garcia, R. A., Takahashi-Hyodo, S. A., Rodrigues, A. C., Amaral, F. G., Berra, C. M., et al. (2013). Effects of melatonin on DNA damage induced by cyclophosphamide in rats. Brazilian Journal of Medical and Biological Research, 46( 3), 278-286. doi:10.1590/1414-431X20122230
NLM
Ferreira SG, Peliciari-Garcia RA, Takahashi-Hyodo SA, Rodrigues AC, Amaral FG, Berra CM, Bordin S, Curi R, Cipolla-Neto J. Effects of melatonin on DNA damage induced by cyclophosphamide in rats [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 3): 278-286.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1590/1414-431X20122230
Vancouver
Ferreira SG, Peliciari-Garcia RA, Takahashi-Hyodo SA, Rodrigues AC, Amaral FG, Berra CM, Bordin S, Curi R, Cipolla-Neto J. Effects of melatonin on DNA damage induced by cyclophosphamide in rats [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 3): 278-286.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1590/1414-431X20122230

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