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Artigo de periodico
Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome (2020)
Silva, Josiane Fernandes da; Olivon, Vania Cláudia; Mestriner, Fabiola Leslie Antunes Cardoso; Zanotto, Camila Ziliotto; Ferreira, Raphael Gomes; Ferreira, Nathanne dos Santos; Silva, Carlos Alberto Aguiar da; Luiz, João Paulo Mesquita; Alves, Juliano Vilela; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, INFLAMAÇÃO, SEPSE, CITOCINAS, NEUTRÓFILOS
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ABNT
SILVA, Josiane Fernandes da et al. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome. Frontiers in Physiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.01614. Acesso em: 26 maio 2024.
APA
Silva, J. F. da, Olivon, V. C., Mestriner, F. L. A. C., Zanotto, C. Z., Ferreira, R. G., Ferreira, N. dos S., et al. (2020). Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome. Frontiers in Physiology, 10. doi:10.3389/fphys.2019.01614
NLM
Silva JF da, Olivon VC, Mestriner FLAC, Zanotto CZ, Ferreira RG, Ferreira N dos S, Silva CAA da, Luiz JPM, Alves JV, Fazan Júnior R, Cunha F de Q, Alves Filho JCF, Tostes R de CA. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 maio 26 ] Available from: https://doi.org/10.3389/fphys.2019.01614
Vancouver
Silva JF da, Olivon VC, Mestriner FLAC, Zanotto CZ, Ferreira RG, Ferreira N dos S, Silva CAA da, Luiz JPM, Alves JV, Fazan Júnior R, Cunha F de Q, Alves Filho JCF, Tostes R de CA. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 maio 26 ] Available from: https://doi.org/10.3389/fphys.2019.01614
Trabalho de evento-resumo periodico
Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis (2014)
Olivon, Vânia Cláudia; Ferreira, Raphael G.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENZIMAS, SEPSE, PRESSÃO SANGUÍNEA
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ABNT
OLIVON, Vânia Cláudia et al. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8. Acesso em: 26 maio 2024. , 2014
APA
Olivon, V. C., Ferreira, R. G., Mestriner, F. L. A. C., Alves Filho, J. C. F., Cunha, F. de Q., & Passaglia, R. de C. A. T. (2014). Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
NLM
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis [Internet]. Hypertension. 2014 ; 64[citado 2024 maio 26 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
Vancouver
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis [Internet]. Hypertension. 2014 ; 64[citado 2024 maio 26 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
Artigo de periodico
Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages (2014)
Saia, Rafael Simone; Mestriner, Fabiola Leslie Antunes Cardoso; Bertozi, Giuliana; Cunha, Fernando de Queiróz; Cárnio, Evelin Capellari
Source: Mediators of Inflammation. Unidades: FMRP, EERP
Subjects: HORMÔNIOS DO TRATO GASTROINTESTINAL, MACRÓFAGOS, ÓXIDO NÍTRICO
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ABNT
SAIA, Rafael Simone et al. Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages. Mediators of Inflammation, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/896029. Acesso em: 26 maio 2024.
APA
Saia, R. S., Mestriner, F. L. A. C., Bertozi, G., Cunha, F. de Q., & Cárnio, E. C. (2014). Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages. Mediators of Inflammation, 2014. doi:10.1155/2014/896029
NLM
Saia RS, Mestriner FLAC, Bertozi G, Cunha F de Q, Cárnio EC. Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 maio 26 ] Available from: https://doi.org/10.1155/2014/896029
Vancouver
Saia RS, Mestriner FLAC, Bertozi G, Cunha F de Q, Cárnio EC. Cholecystokinin inhibits inducible nitric oxide synthase expression by lipopolysaccharide-stimulated peritoneal macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 maio 26 ] Available from: https://doi.org/10.1155/2014/896029
Trabalho de evento-resumo
Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis (2014)
Olivon, Vânia Cláudia; Ferreira, Raphael G.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Passaglia, Rita de Cassia Aleixo Tostes
Source: Final Program. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENZIMAS, SEPSE, PRESSÃO SANGUÍNEA
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ABNT
OLIVON, Vânia Cláudia et al. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis. 2014, Anais.. San Francisco: AHA, 2014. Disponível em: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf. Acesso em: 26 maio 2024.
APA
Olivon, V. C., Ferreira, R. G., Mestriner, F. L. A. C., Alves Filho, J. C. F., Cunha, F. de Q., & Passaglia, R. de C. A. T. (2014). Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis. In Final Program. San Francisco: AHA. Recuperado de http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
NLM
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis [Internet]. Final Program. 2014 ;[citado 2024 maio 26 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Vancouver
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis [Internet]. Final Program. 2014 ;[citado 2024 maio 26 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Artigo de periodico
Cardiovascular and inflammatory response to cholecystokinin during endotoxemic shock (2013)
Saia, Rafael Simone; Bertozi, Giuliana; Mestriner, Fabiola Leslie Antunes Cardoso; Antunes-Rodrigues, José; Cunha, Fernando de Queiróz; Cárnio, Evelin Capellari
Source: Shock. Unidades: FMRP, EERP
Subjects: NECROSE, ÓXIDO NÍTRICO, HIPOTENSÃO, MACRÓFAGOS
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ABNT
SAIA, Rafael Simone et al. Cardiovascular and inflammatory response to cholecystokinin during endotoxemic shock. Shock, v. 39, n. 1, p. 104-112, 2013Tradução . . Acesso em: 26 maio 2024.
APA
Saia, R. S., Bertozi, G., Mestriner, F. L. A. C., Antunes-Rodrigues, J., Cunha, F. de Q., & Cárnio, E. C. (2013). Cardiovascular and inflammatory response to cholecystokinin during endotoxemic shock. Shock, 39( 1), 104-112.
NLM
Saia RS, Bertozi G, Mestriner FLAC, Antunes-Rodrigues J, Cunha F de Q, Cárnio EC. Cardiovascular and inflammatory response to cholecystokinin during endotoxemic shock. Shock. 2013 ; 39( 1): 104-112.[citado 2024 maio 26 ]
Vancouver
Saia RS, Bertozi G, Mestriner FLAC, Antunes-Rodrigues J, Cunha F de Q, Cárnio EC. Cardiovascular and inflammatory response to cholecystokinin during endotoxemic shock. Shock. 2013 ; 39( 1): 104-112.[citado 2024 maio 26 ]
Artigo de periodico
Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice (2011)
Spiller, Fernando; Costa, Carlotta; Souto, Fabrício Oliveira; Vinchi, Francesca; Mestriner, Fabiola Leslie Antunes Cardoso; Laure, Hélen Julie; Alves Filho, José C.; Freitas, Andressa; Rosa, José César; Ferreira, Sérgio Henrique; Altruda, Fiorella; Hirsch, Emilio; Greene, Lewis Joel; Tolosano, Emanuela; Cunha, Fernando de Queiroz
Source: American Journal of Respiratory and Critical Care Medicine. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
SPILLER, Fernando et al. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, v. 183, n. 7, p. 922-931, 2011Tradução . . Acesso em: 26 maio 2024.
APA
Spiller, F., Costa, C., Souto, F. O., Vinchi, F., Mestriner, F. L. A. C., Laure, H. J., et al. (2011). Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, 183( 7), 922-931.
NLM
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 maio 26 ]
Vancouver
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 maio 26 ]
Trabalho de evento-resumo
Role of cholecystokinin on inducible nitric oxide synthase expression in LPS-stimulated peritoneal macrophage culture (2010)
Saia, R. S.; Mestriner, Fabiola Leslie Antunes Cardoso; Vieira, S. M.; Cunha, Fernando de Queiroz; Cárnio, Evelin Capellari
Source: Resumos. Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE). Unidades: FMRP, EERP
Subjects: ÓXIDO NÍTRICO, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SAIA, R. S. et al. Role of cholecystokinin on inducible nitric oxide synthase expression in LPS-stimulated peritoneal macrophage culture. 2010, Anais.. São Paulo: FeSBE, 2010. . Acesso em: 26 maio 2024.
APA
Saia, R. S., Mestriner, F. L. A. C., Vieira, S. M., Cunha, F. de Q., & Cárnio, E. C. (2010). Role of cholecystokinin on inducible nitric oxide synthase expression in LPS-stimulated peritoneal macrophage culture. In Resumos. São Paulo: FeSBE.
NLM
Saia RS, Mestriner FLAC, Vieira SM, Cunha F de Q, Cárnio EC. Role of cholecystokinin on inducible nitric oxide synthase expression in LPS-stimulated peritoneal macrophage culture. Resumos. 2010 ;[citado 2024 maio 26 ]
Vancouver
Saia RS, Mestriner FLAC, Vieira SM, Cunha F de Q, Cárnio EC. Role of cholecystokinin on inducible nitric oxide synthase expression in LPS-stimulated peritoneal macrophage culture. Resumos. 2010 ;[citado 2024 maio 26 ]
Artigo de periodico
Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway (2008)
Napimoga, Marcelo H.; Vieira, Silvio M.; Dal-Secco, Daniela; Freitas, Andressa; Souto, Fabrício O.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves-Filho, José C.; Grespan, Renata; Kawai, Toshihisa; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO
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ABNT
NAPIMOGA, Marcelo H. et al. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, v. 180, n. 1, p. 609-617, 2008Tradução . . Acesso em: 26 maio 2024.
APA
Napimoga, M. H., Vieira, S. M., Dal-Secco, D., Freitas, A., Souto, F. O., Mestriner, F. L. A. C., et al. (2008). Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, 180( 1), 609-617.
NLM
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 maio 26 ]
Vancouver
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 maio 26 ]
Artigo de periodico
Acute-phase protein 'alfa'-1-acid glycoprotein mediates neutrophil migration failure in sepsis by a nitric oxide-dependent mechanism (2007)
Mestriner, F. L. A. C.; Spiller, F.; Laure, H. J.; Souto, F. O.; Tavares-Murta, B. M.; Rosa, José César; Basile Filho, Aníbal; Ferreira, S. H.; Greene, Lewis Joel; Cunha, Fernando de Queiroz
Source: Proceedings of the National Academy of Sciences of the United States of America (PNAS). Unidade: FMRP
Subjects: FARMACOLOGIA MÉDICA, MICROSCOPIA, TÉCNICAS INVESTIGATIVAS
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ABNT
MESTRINER, F. L. A. C. et al. Acute-phase protein 'alfa'-1-acid glycoprotein mediates neutrophil migration failure in sepsis by a nitric oxide-dependent mechanism. Proceedings of the National Academy of Sciences of the United States of America (PNAS), v. 104, n. 49, p. 19595-19600, 2007Tradução . . Disponível em: https://doi.org/10.1073/pnas.0709681104. Acesso em: 26 maio 2024.
APA
Mestriner, F. L. A. C., Spiller, F., Laure, H. J., Souto, F. O., Tavares-Murta, B. M., Rosa, J. C., et al. (2007). Acute-phase protein 'alfa'-1-acid glycoprotein mediates neutrophil migration failure in sepsis by a nitric oxide-dependent mechanism. Proceedings of the National Academy of Sciences of the United States of America (PNAS), 104( 49), 19595-19600. doi:10.1073/pnas.0709681104
NLM
Mestriner FLAC, Spiller F, Laure HJ, Souto FO, Tavares-Murta BM, Rosa JC, Basile Filho A, Ferreira SH, Greene LJ, Cunha F de Q. Acute-phase protein 'alfa'-1-acid glycoprotein mediates neutrophil migration failure in sepsis by a nitric oxide-dependent mechanism [Internet]. Proceedings of the National Academy of Sciences of the United States of America (PNAS). 2007 ; 104( 49): 19595-19600.[citado 2024 maio 26 ] Available from: https://doi.org/10.1073/pnas.0709681104
Vancouver
Mestriner FLAC, Spiller F, Laure HJ, Souto FO, Tavares-Murta BM, Rosa JC, Basile Filho A, Ferreira SH, Greene LJ, Cunha F de Q. Acute-phase protein 'alfa'-1-acid glycoprotein mediates neutrophil migration failure in sepsis by a nitric oxide-dependent mechanism [Internet]. Proceedings of the National Academy of Sciences of the United States of America (PNAS). 2007 ; 104( 49): 19595-19600.[citado 2024 maio 26 ] Available from: https://doi.org/10.1073/pnas.0709681104
Artigo de periodico
Trypanosoma cruzi-infected cardiomycytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity (2000)
Machado, Fabiana Simão; Martins, Gislâine Aparecida; Aliberti, Júlio César Soares; Mestriner, Fabiola Leslie Antunes Cardoso; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: Circulation. Unidade: FMRP
Subjects: DOENÇAS CARDIOVASCULARES (EPIDEMIOLOGIA), IMUNOLOGIA
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ABNT
MACHADO, Fabiana Simão et al. Trypanosoma cruzi-infected cardiomycytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity. Circulation, v. 102, p. 3003-3008, 2000Tradução . . Acesso em: 26 maio 2024.
APA
Machado, F. S., Martins, G. A., Aliberti, J. C. S., Mestriner, F. L. A. C., Cunha, F. de Q., & Silva, J. S. da. (2000). Trypanosoma cruzi-infected cardiomycytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity. Circulation, 102, 3003-3008.
NLM
Machado FS, Martins GA, Aliberti JCS, Mestriner FLAC, Cunha F de Q, Silva JS da. Trypanosoma cruzi-infected cardiomycytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity. Circulation. 2000 ; 102 3003-3008.[citado 2024 maio 26 ]
Vancouver
Machado FS, Martins GA, Aliberti JCS, Mestriner FLAC, Cunha F de Q, Silva JS da. Trypanosoma cruzi-infected cardiomycytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity. Circulation. 2000 ; 102 3003-3008.[citado 2024 maio 26 ]
Artigo de periodico
Trypanosoma cruzi-intected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity (2000)
Machado, Fabiana Simão; Martins, Gislâine Aparecida; Aliberti, Júlio César Soares; Mestriner, Fabiola Leslie Antunes Cardoso; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: Circulation. Unidade: FMRP
Subjects: TRYPANOSOMA CRUZI, MIOCARDITE, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACHADO, Fabiana Simão et al. Trypanosoma cruzi-intected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity. Circulation, v. 102, p. 3003-3008, 2000Tradução . . Disponível em: http://www.circulationaha.org. Acesso em: 26 maio 2024.
APA
Machado, F. S., Martins, G. A., Aliberti, J. C. S., Mestriner, F. L. A. C., Cunha, F. de Q., & Silva, J. S. da. (2000). Trypanosoma cruzi-intected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity. Circulation, 102, 3003-3008. Recuperado de http://www.circulationaha.org
NLM
Machado FS, Martins GA, Aliberti JCS, Mestriner FLAC, Cunha F de Q, Silva JS da. Trypanosoma cruzi-intected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity [Internet]. Circulation. 2000 ; 102 3003-3008.[citado 2024 maio 26 ] Available from: http://www.circulationaha.org
Vancouver
Machado FS, Martins GA, Aliberti JCS, Mestriner FLAC, Cunha F de Q, Silva JS da. Trypanosoma cruzi-intected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity [Internet]. Circulation. 2000 ; 102 3003-3008.[citado 2024 maio 26 ] Available from: http://www.circulationaha.org

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