Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome (2020)
- Authors:
- Silva, Josiane Fernandes da
- Olivon, Vania Cláudia
- Mestriner, Fabiola Leslie Antunes Cardoso
- Zanotto, Camila Ziliotto
- Ferreira, Raphael Gomes
- Ferreira, Nathanne dos Santos
- Silva, Carlos Alberto Aguiar da
- Luiz, João Paulo Mesquita
- Alves, Juliano Vilela
- Fazan Júnior, Rubens
- Cunha, Fernando de Queiroz
- Alves Filho, José Carlos Farias
- Tostes, Rita de Cássia Aleixo
- USP affiliated authors: SILVA, CARLOS ALBERTO AGUIAR DA - FMRP ; FAZAN JÚNIOR, RUBENS - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP ; SILVA, JOSIANE FERNANDES DA - FMRP ; MESTRINER, FABIOLA LESLIE ANTUNES CARDOSO - FMRP ; LUIZ, JOÃO PAULO MESQUITA - FMRP ; ALVES, JULIANO VILELA - FMRP
- Unidade: FMRP
- DOI: 10.3389/fphys.2019.01614
- Subjects: PRESSÃO SANGUÍNEA; INFLAMAÇÃO; SEPSE; CITOCINAS; NEUTRÓFILOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Physiology
- ISSN: 1664-042X
- Volume/Número/Paginação/Ano: v. 10, art. 1614, 2020
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
SILVA, Josiane Fernandes da et al. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome. Frontiers in Physiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.01614. Acesso em: 01 nov. 2024. -
APA
Silva, J. F. da, Olivon, V. C., Mestriner, F. L. A. C., Zanotto, C. Z., Ferreira, R. G., Ferreira, N. dos S., et al. (2020). Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome. Frontiers in Physiology, 10. doi:10.3389/fphys.2019.01614 -
NLM
Silva JF da, Olivon VC, Mestriner FLAC, Zanotto CZ, Ferreira RG, Ferreira N dos S, Silva CAA da, Luiz JPM, Alves JV, Fazan Júnior R, Cunha F de Q, Alves Filho JCF, Tostes R de CA. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 01 ] Available from: https://doi.org/10.3389/fphys.2019.01614 -
Vancouver
Silva JF da, Olivon VC, Mestriner FLAC, Zanotto CZ, Ferreira RG, Ferreira N dos S, Silva CAA da, Luiz JPM, Alves JV, Fazan Júnior R, Cunha F de Q, Alves Filho JCF, Tostes R de CA. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 01 ] Available from: https://doi.org/10.3389/fphys.2019.01614 - Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis
- Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis
- Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis
- Vascular stress signaling in hypertension
- NLRP3 inflammasome mediates aldosterone-induced vascular damage
- Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human
- Toll-like receptor 9 (TLR9) plays a key role in the autonomic cardiac and baroreflex control of arterial pressure
- Involvement of serum factor(s) expressed in severe sepsis on failure of neutrophil migration toward inflammatory focus
- Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome
- Analysis of heart rate variability by multiscale entropy in spontaneuously hypertensive rats
Informações sobre o DOI: 10.3389/fphys.2019.01614 (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
 | 003036732.pdf | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
