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Artigo de periodico
Endogenous galectin-3 is required for skeletal muscle repair (2021)
Cerri, Daniel Giuliano; Rodrigues, Lilian Cataldi ; Alves, Vani Maria; Machado, Juliano; Bastos, Victor Alexandre Félix; Kettelhut, Isis do Carmo ; Alberici, Luciane Carla; Costa, Maria Cristina R; Stowell, Sean R; Cummings, Richard D; Dias-Baruffi, Marcelo
Fonte: Glycobiology. Unidades: FCFRP, FMRP
Assuntos: MIOBLASTOS, BÁRIO, CLORETO, MÚSCULO ESQUELÉTICO
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ABNT
CERRI, Daniel Giuliano et al. Endogenous galectin-3 is required for skeletal muscle repair. Glycobiology, v. 31, n. 10, p. 1295-1307, 2021Tradução . . Disponível em: https://doi.org/10.1093/glycob/cwab071. Acesso em: 29 jun. 2024.
APA
Cerri, D. G., Rodrigues, L. C., Alves, V. M., Machado, J., Bastos, V. A. F., Kettelhut, I. do C., et al. (2021). Endogenous galectin-3 is required for skeletal muscle repair. Glycobiology, 31( 10), 1295-1307. doi:10.1093/glycob/cwab071
NLM
Cerri DG, Rodrigues LC, Alves VM, Machado J, Bastos VAF, Kettelhut I do C, Alberici LC, Costa MCR, Stowell SR, Cummings RD, Dias-Baruffi M. Endogenous galectin-3 is required for skeletal muscle repair [Internet]. Glycobiology. 2021 ; 31( 10): 1295-1307.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1093/glycob/cwab071
Vancouver
Cerri DG, Rodrigues LC, Alves VM, Machado J, Bastos VAF, Kettelhut I do C, Alberici LC, Costa MCR, Stowell SR, Cummings RD, Dias-Baruffi M. Endogenous galectin-3 is required for skeletal muscle repair [Internet]. Glycobiology. 2021 ; 31( 10): 1295-1307.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1093/glycob/cwab071
Artigo de periodico
Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S (2020)
Azevedo, Lara Ferreira ; Carneiro, Maria Fernanda Hornos ; Dechandt, Carlos Roberto Porto ; Cassoli, Juliana Silva ; Alberici, Luciane Carla ; Barbosa Junior, Fernando
Fonte: Environmental Research. Unidade: FCFRP
Assuntos: HOMEOSTASE, FÍGADO, PROTEÔMICA, FENÓIS
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ABNT
AZEVEDO, Lara Ferreira et al. Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S. Environmental Research, v. 182, p. 109080, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.envres.2019.109080. Acesso em: 29 jun. 2024.
APA
Azevedo, L. F., Carneiro, M. F. H., Dechandt, C. R. P., Cassoli, J. S., Alberici, L. C., & Barbosa Junior, F. (2020). Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S. Environmental Research, 182, 109080. doi:10.1016/j.envres.2019.109080
NLM
Azevedo LF, Carneiro MFH, Dechandt CRP, Cassoli JS, Alberici LC, Barbosa Junior F. Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S [Internet]. Environmental Research. 2020 ; 182 109080.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.envres.2019.109080
Vancouver
Azevedo LF, Carneiro MFH, Dechandt CRP, Cassoli JS, Alberici LC, Barbosa Junior F. Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S [Internet]. Environmental Research. 2020 ; 182 109080.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.envres.2019.109080
Artigo de periodico
Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles (2020)
Adeyemi, Joseph A.; Machado, Ana Rita Thomazela ; Ogunjimi, Abayomi T.; Alberici, Luciane Carla; Antunes, Lusânia Maria Greggi ; Barbosa Júnior, Fernando
Fonte: Ecotoxicology and Environmental Safety. Unidade: FCFRP
Assuntos: CARBONO, QUÍMICA, COBRE, POLUIÇÃO AMBIENTAL, MITOCÔNDRIAS, NANOPARTÍCULAS, ESTRESSE OXIDATIVO
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ADEYEMI, Joseph A. et al. Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles. Ecotoxicology and Environmental Safety, v. 189, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.ecoenv.2019.109982. Acesso em: 29 jun. 2024.
APA
Adeyemi, J. A., Machado, A. R. T., Ogunjimi, A. T., Alberici, L. C., Antunes, L. M. G., & Barbosa Júnior, F. (2020). Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles. Ecotoxicology and Environmental Safety, 189. doi:10.1016/j.ecoenv.2019.109982
NLM
Adeyemi JA, Machado ART, Ogunjimi AT, Alberici LC, Antunes LMG, Barbosa Júnior F. Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles [Internet]. Ecotoxicology and Environmental Safety. 2020 ; 189[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.ecoenv.2019.109982
Vancouver
Adeyemi JA, Machado ART, Ogunjimi AT, Alberici LC, Antunes LMG, Barbosa Júnior F. Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles [Internet]. Ecotoxicology and Environmental Safety. 2020 ; 189[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.ecoenv.2019.109982
Artigo de periodico
Long-term exposure to bisphenol A or S promotes glucose intolerance and changes hepatic mitochondrial metabolism in male Wistar rats (2019)
Azevedo, Lara Ferreira; Dechandt, Carlos Roberto Porto; Rocha, Cecília Cristina de Souza; Carneiro, Maria Fernanda Hornos; Alberici, Luciane Carla ; Barbosa Junior, Fernando
Fonte: Food and Chemical Toxicology. Unidade: FCFRP
Assuntos: MITOCÔNDRIAS, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
AZEVEDO, Lara Ferreira et al. Long-term exposure to bisphenol A or S promotes glucose intolerance and changes hepatic mitochondrial metabolism in male Wistar rats. Food and Chemical Toxicology, v. 132, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.fct.2019.110694. Acesso em: 29 jun. 2024.
APA
Azevedo, L. F., Dechandt, C. R. P., Rocha, C. C. de S., Carneiro, M. F. H., Alberici, L. C., & Barbosa Junior, F. (2019). Long-term exposure to bisphenol A or S promotes glucose intolerance and changes hepatic mitochondrial metabolism in male Wistar rats. Food and Chemical Toxicology, 132. doi:10.1016/j.fct.2019.110694
NLM
Azevedo LF, Dechandt CRP, Rocha CC de S, Carneiro MFH, Alberici LC, Barbosa Junior F. Long-term exposure to bisphenol A or S promotes glucose intolerance and changes hepatic mitochondrial metabolism in male Wistar rats [Internet]. Food and Chemical Toxicology. 2019 ; 132[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.fct.2019.110694
Vancouver
Azevedo LF, Dechandt CRP, Rocha CC de S, Carneiro MFH, Alberici LC, Barbosa Junior F. Long-term exposure to bisphenol A or S promotes glucose intolerance and changes hepatic mitochondrial metabolism in male Wistar rats [Internet]. Food and Chemical Toxicology. 2019 ; 132[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.fct.2019.110694
Artigo de periodico
The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms (2017)
Prida, Javier Marín; Andreu, Gilberto L. Pardo; Rossignoli, Camila Pederiva; Durruthy, Michael González; Rodríguez, Estael Ochoa; Reyes, Yamila Verdecia; Acosta, Roberto Fernández; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Fonte: Toxicology in Vitro. Unidade: FCFRP
Assuntos: NEOPLASIAS, CITOTOXICIDADE IMUNOLÓGICA, MITOCÔNDRIAS, FÁRMACOS
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ABNT
PRIDA, Javier Marín et al. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, v. 42, p. 21-30, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2017.03.011. Acesso em: 29 jun. 2024.
APA
Prida, J. M., Andreu, G. L. P., Rossignoli, C. P., Durruthy, M. G., Rodríguez, E. O., Reyes, Y. V., et al. (2017). The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, 42, 21-30. doi:10.1016/j.tiv.2017.03.011
NLM
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Vancouver
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Artigo de periodico
SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line (2017)
Sobral, Lays M.; Coletta, Ricardo D.; Alberici, Luciane Carla; Curti, Carlos; Leopoldino, Andréia Machado
Fonte: The FEBS Journal. Unidade: FCFRP
Assuntos: TOXICOLOGIA, CÉLULAS, METABOLISMO, MITOCÔNDRIAS, RESPIRAÇÃO
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ABNT
SOBRAL, Lays M. et al. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, v. 284, n. 17, p. 2774-2785, 2017Tradução . . Disponível em: https://doi.org/10.1111/febs.14148. Acesso em: 29 jun. 2024.
APA
Sobral, L. M., Coletta, R. D., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2017). SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, 284( 17), 2774-2785. doi:10.1111/febs.14148
NLM
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1111/febs.14148
Vancouver
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1111/febs.14148
Artigo de periodico
The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice (2016)
Baraldi, Flávia G.; Vicentini, Tatiane M.; Teodoro, Bruno G.; Dalalio, Felipe M.; Dechandt, Carlos R. P.; Prado, Ieda M. R.; Curti, Carlos; Cardoso, Fernanda C.; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Fonte: The Journal of Nutritional Biochemistry. Unidade: FCFRP
Assuntos: METABOLISMO, OBESIDADE, AZEITE, ÁCIDO LINOLEICO, ÁCIDOS GRAXOS OMEGA 6, MITOCÔNDRIAS
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ABNT
BARALDI, Flávia G. et al. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice. The Journal of Nutritional Biochemistry, v. 28, p. 147-154, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2015.10.008. Acesso em: 29 jun. 2024.
APA
Baraldi, F. G., Vicentini, T. M., Teodoro, B. G., Dalalio, F. M., Dechandt, C. R. P., Prado, I. M. R., et al. (2016). The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice. The Journal of Nutritional Biochemistry, 28, 147-154. doi:10.1016/j.jnutbio.2015.10.008
NLM
Baraldi FG, Vicentini TM, Teodoro BG, Dalalio FM, Dechandt CRP, Prado IMR, Curti C, Cardoso FC, Uyemura SA, Alberici LC. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice [Internet]. The Journal of Nutritional Biochemistry. 2016 ; 28 147-154.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.jnutbio.2015.10.008
Vancouver
Baraldi FG, Vicentini TM, Teodoro BG, Dalalio FM, Dechandt CRP, Prado IMR, Curti C, Cardoso FC, Uyemura SA, Alberici LC. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice [Internet]. The Journal of Nutritional Biochemistry. 2016 ; 28 147-154.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.jnutbio.2015.10.008
Artigo de periodico
The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling (2015)
Andreu, Gilberto L. Pardo; Reis, Felippe H. Z; Dalalio, Felipe M; Figueredo, Yanier Nuñez; Rubio, Osmany Cuesta; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Fonte: Chemico-Biological Interactions. Unidade: FCFRP
Assuntos: PRÓPOLIS, MITOCÔNDRIAS, TESTES DE TOXICIDADE, TOXICOLOGIA
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ABNT
ANDREU, Gilberto L. Pardo et al. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, v. 228, p. 28-34, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.01.010. Acesso em: 29 jun. 2024.
APA
Andreu, G. L. P., Reis, F. H. Z., Dalalio, F. M., Figueredo, Y. N., Rubio, O. C., Uyemura, S. A., et al. (2015). The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, 228, 28-34. doi:10.1016/j.cbi.2015.01.010
NLM
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Vancouver
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Artigo de periodico
Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle (2014)
Teodoro, Bruno G.; Baraldi, Flavia G; Sampaio, Igor H.; Bomfim, Lucas H. M.; Queiroz, André L.; Passos, Madla A.; Carneiro, Everardo M.; Alberici, Luciane Carla; Gomis, Ramon; Amaral, Fernanda G.; Cipolla-Neto, Jose; Araújo, Michel B.; Lima, Tanes; Uyemura, Sérgio Akira; Silveira, Leonardo dos Reis; Vieira, Elaine
Fonte: Journal of Pineal Research. Unidades: ICB, FCFRP, EEFERP
Assunto: FISIOLOGIA
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ABNT
TEODORO, Bruno G. et al. Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle. Journal of Pineal Research, v. 57, n. 2, p. 155-167, 2014Tradução . . Disponível em: https://doi.org/10.1111/jpi.12157. Acesso em: 29 jun. 2024.
APA
Teodoro, B. G., Baraldi, F. G., Sampaio, I. H., Bomfim, L. H. M., Queiroz, A. L., Passos, M. A., et al. (2014). Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle. Journal of Pineal Research, 57( 2), 155-167. doi:10.1111/jpi.12157
NLM
Teodoro BG, Baraldi FG, Sampaio IH, Bomfim LHM, Queiroz AL, Passos MA, Carneiro EM, Alberici LC, Gomis R, Amaral FG, Cipolla-Neto J, Araújo MB, Lima T, Uyemura SA, Silveira L dos R, Vieira E. Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle [Internet]. Journal of Pineal Research. 2014 ; 57( 2): 155-167.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1111/jpi.12157
Vancouver
Teodoro BG, Baraldi FG, Sampaio IH, Bomfim LHM, Queiroz AL, Passos MA, Carneiro EM, Alberici LC, Gomis R, Amaral FG, Cipolla-Neto J, Araújo MB, Lima T, Uyemura SA, Silveira L dos R, Vieira E. Melatonin prevents mitochondrial dysfunction and insulin resistance in rat skeletal muscle [Internet]. Journal of Pineal Research. 2014 ; 57( 2): 155-167.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1111/jpi.12157
Artigo de periodico
Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death (2014)
Reis, Felippe H. Z.; Pardo-Andreu, Gilberto L.; Nuñez-Figueredo, Yanier; Cuesta-Rubio, Osmany; Marín-Prida, Javier; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Fonte: Chemico-Biological Interactions. Unidade: FCFRP
Assuntos: ADENOSINA (METABOLISMO), ANTINEOPLÁSICOS (FARMACOLOGIA), TRANSPORTE BIOLÓGICO (EFEITOS DE DROGAS), MITOCÔNDRIAS, PRODUTOS NATURAIS
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ABNT
REIS, Felippe H. Z. et al. Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death. Chemico-Biological Interactions, v. 212, p. 20\201329, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2014.01.015. Acesso em: 29 jun. 2024.
APA
Reis, F. H. Z., Pardo-Andreu, G. L., Nuñez-Figueredo, Y., Cuesta-Rubio, O., Marín-Prida, J., Uyemura, S. A., et al. (2014). Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death. Chemico-Biological Interactions, 212, 20\201329. doi:10.1016/j.cbi.2014.01.015
NLM
Reis FHZ, Pardo-Andreu GL, Nuñez-Figueredo Y, Cuesta-Rubio O, Marín-Prida J, Uyemura SA, Curti C, Alberici LC. Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death [Internet]. Chemico-Biological Interactions. 2014 ; 212 20\201329.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.cbi.2014.01.015
Vancouver
Reis FHZ, Pardo-Andreu GL, Nuñez-Figueredo Y, Cuesta-Rubio O, Marín-Prida J, Uyemura SA, Curti C, Alberici LC. Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death [Internet]. Chemico-Biological Interactions. 2014 ; 212 20\201329.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.cbi.2014.01.015
Trabalho de evento-resumo
C-Phycocyanin: a new promising therapeutic candidate against ischemic stroke (2013)
Marin-Prida, J.; Pavón-Fuentes, N.; Rodrigues, F. P.; Alberici, Luciane Carla; Stringhetta, Karina; Leopoldino, Andréia Machado; Llópiz-Arzuaga, A.; Liberato, José Luiz; Santos, Wagner Ferreira dos; Uyemura, Sérgio Akira; Pentón-Arias, E.; Curti, Carlos; Pardo-Andreu, Gilberto L.; Pentón-Rol, G.
Fonte: Abstracts. Nome do evento: Annual Meeting of SBBq. Unidades: FCFRP, FFCLRP
Assuntos: ISQUEMIA CEREBRAL, BIOQUÍMICA CELULAR, NEURÔNIOS
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ABNT
MARIN-PRIDA, J. et al. C-Phycocyanin: a new promising therapeutic candidate against ischemic stroke. 2013, Anais.. Foz do Iguaçu: SBBq, 2013. . Acesso em: 29 jun. 2024.
APA
Marin-Prida, J., Pavón-Fuentes, N., Rodrigues, F. P., Alberici, L. C., Stringhetta, K., Leopoldino, A. M., et al. (2013). C-Phycocyanin: a new promising therapeutic candidate against ischemic stroke. In Abstracts. Foz do Iguaçu: SBBq.
NLM
Marin-Prida J, Pavón-Fuentes N, Rodrigues FP, Alberici LC, Stringhetta K, Leopoldino AM, Llópiz-Arzuaga A, Liberato JL, Santos WF dos, Uyemura SA, Pentón-Arias E, Curti C, Pardo-Andreu GL, Pentón-Rol G. C-Phycocyanin: a new promising therapeutic candidate against ischemic stroke. Abstracts. 2013 ;[citado 2024 jun. 29 ]
Vancouver
Marin-Prida J, Pavón-Fuentes N, Rodrigues FP, Alberici LC, Stringhetta K, Leopoldino AM, Llópiz-Arzuaga A, Liberato JL, Santos WF dos, Uyemura SA, Pentón-Arias E, Curti C, Pardo-Andreu GL, Pentón-Rol G. C-Phycocyanin: a new promising therapeutic candidate against ischemic stroke. Abstracts. 2013 ;[citado 2024 jun. 29 ]
Artigo de periodico
Administration of a murine diet supplemented with conjugated linoleic acid increases the expression and activity of hepatic uncoupling proteins (2012)
Pereira, Amanda F.; Sá, Larissa L.; Reis, Felippe H. Z.; Cardoso, Fernanda C.; Alberici, Rosana M.; Prado, Ieda Maria Razaboni; Eberlin, Marcos N.; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Fonte: Journal of Bioenergetics and Biomembranes. Unidade: FCFRP
Assuntos: OBESIDADE, METABOLISMO ENERGÉTICO, SUPLEMENTOS DIETÉTICOS, FÍGADO, MITOCÔNDRIAS
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ABNT
PEREIRA, Amanda F. et al. Administration of a murine diet supplemented with conjugated linoleic acid increases the expression and activity of hepatic uncoupling proteins. Journal of Bioenergetics and Biomembranes, v. 44, n. 55, p. 587-596, 2012Tradução . . Disponível em: https://doi.org/10.1007/s10863-012-9463-y. Acesso em: 29 jun. 2024.
APA
Pereira, A. F., Sá, L. L., Reis, F. H. Z., Cardoso, F. C., Alberici, R. M., Prado, I. M. R., et al. (2012). Administration of a murine diet supplemented with conjugated linoleic acid increases the expression and activity of hepatic uncoupling proteins. Journal of Bioenergetics and Biomembranes, 44( 55), 587-596. doi:10.1007/s10863-012-9463-y
NLM
Pereira AF, Sá LL, Reis FHZ, Cardoso FC, Alberici RM, Prado IMR, Eberlin MN, Uyemura SA, Curti C, Alberici LC. Administration of a murine diet supplemented with conjugated linoleic acid increases the expression and activity of hepatic uncoupling proteins [Internet]. Journal of Bioenergetics and Biomembranes. 2012 ; 44( 55): 587-596.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s10863-012-9463-y
Vancouver
Pereira AF, Sá LL, Reis FHZ, Cardoso FC, Alberici RM, Prado IMR, Eberlin MN, Uyemura SA, Curti C, Alberici LC. Administration of a murine diet supplemented with conjugated linoleic acid increases the expression and activity of hepatic uncoupling proteins [Internet]. Journal of Bioenergetics and Biomembranes. 2012 ; 44( 55): 587-596.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s10863-012-9463-y
Artigo de periodico
Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria (2012)
Rodrigues, Fernando P.; Pestana, Cezar R.; Polizello, Ana Cristina Morseli; Pardo-Andreu, Gilberto L.; Uyemura, Sérgio Akira; Santos, Antônio Cardozo dos; Alberici, Luciane Carla; Silva, Roberto Santana da; Curti, Carlos
Fonte: Nitric Oxide. Unidade: FCFRP
Assuntos: MITOCÔNDRIAS, RUTÊNIO, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Fernando P. et al. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria. Nitric Oxide, v. 26, n. 3, p. 174-181, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2012.02.001. Acesso em: 29 jun. 2024.
APA
Rodrigues, F. P., Pestana, C. R., Polizello, A. C. M., Pardo-Andreu, G. L., Uyemura, S. A., Santos, A. C. dos, et al. (2012). Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria. Nitric Oxide, 26( 3), 174-181. doi:10.1016/j.niox.2012.02.001
NLM
Rodrigues FP, Pestana CR, Polizello ACM, Pardo-Andreu GL, Uyemura SA, Santos AC dos, Alberici LC, Silva RS da, Curti C. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria [Internet]. Nitric Oxide. 2012 ; 26( 3): 174-181.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.niox.2012.02.001
Vancouver
Rodrigues FP, Pestana CR, Polizello ACM, Pardo-Andreu GL, Uyemura SA, Santos AC dos, Alberici LC, Silva RS da, Curti C. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria [Internet]. Nitric Oxide. 2012 ; 26( 3): 174-181.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.niox.2012.02.001
Artigo de periodico
C-Phycocyanin protects SH-SY5Y cells from oxidative injury, rat retina from transient ischemia and rat brain mitochondria from 'Ca POT.2+'/phospatase-induced impairment (2012)
Marín-Prida, Javier; Pentól-Rol, Giselle; Rodrigues, Fernando Postalli; Alberici, Luciane Carla; Stringhetta, Karina; Leopoldino, Andréia Machado; Naal, Zeki; Polizello, Ana Cristina Morseli; Llópiz-Arzuaga, Alexey; Rosa, Marcela Nunes; Liberato, José Luiz; Santos, Wagner Ferreira dos; Uyemura, Sérgio Akira; Pentón-Arias, Eduardo; Curti, Carlos; Pardo-Andreu, Gilberto L.
Fonte: Brain Research Bulletin. Unidades: FCFRP, FFCLRP
Assuntos: PROTEÍNAS, BIOQUÍMICA CELULAR, ISQUEMIA CEREBRAL, ANTIOXIDANTES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARÍN-PRIDA, Javier et al. C-Phycocyanin protects SH-SY5Y cells from oxidative injury, rat retina from transient ischemia and rat brain mitochondria from 'Ca POT.2+'/phospatase-induced impairment. Brain Research Bulletin, v. 89, n. 5-6, p. 159-167, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2012.08.011. Acesso em: 29 jun. 2024.
APA
Marín-Prida, J., Pentól-Rol, G., Rodrigues, F. P., Alberici, L. C., Stringhetta, K., Leopoldino, A. M., et al. (2012). C-Phycocyanin protects SH-SY5Y cells from oxidative injury, rat retina from transient ischemia and rat brain mitochondria from 'Ca POT.2+'/phospatase-induced impairment. Brain Research Bulletin, 89( 5-6), 159-167. doi:10.1016/j.brainresbull.2012.08.011
NLM
Marín-Prida J, Pentól-Rol G, Rodrigues FP, Alberici LC, Stringhetta K, Leopoldino AM, Naal Z, Polizello ACM, Llópiz-Arzuaga A, Rosa MN, Liberato JL, Santos WF dos, Uyemura SA, Pentón-Arias E, Curti C, Pardo-Andreu GL. C-Phycocyanin protects SH-SY5Y cells from oxidative injury, rat retina from transient ischemia and rat brain mitochondria from 'Ca POT.2+'/phospatase-induced impairment [Internet]. Brain Research Bulletin. 2012 ; 89( 5-6): 159-167.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.brainresbull.2012.08.011
Vancouver
Marín-Prida J, Pentól-Rol G, Rodrigues FP, Alberici LC, Stringhetta K, Leopoldino AM, Naal Z, Polizello ACM, Llópiz-Arzuaga A, Rosa MN, Liberato JL, Santos WF dos, Uyemura SA, Pentón-Arias E, Curti C, Pardo-Andreu GL. C-Phycocyanin protects SH-SY5Y cells from oxidative injury, rat retina from transient ischemia and rat brain mitochondria from 'Ca POT.2+'/phospatase-induced impairment [Internet]. Brain Research Bulletin. 2012 ; 89( 5-6): 159-167.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.brainresbull.2012.08.011
Artigo de periodico
The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler (2011)
Andreu, Gilberto Lázaro Pardo; Figueredo, Yanier Nuñez; Tudella, Valéria Gomes; Rubio, Osmany Cuesta; Rodrigues, Fernando P.; Pestana, Cezar Rangel; Uyemura, Sérgio Akira; Leopoldino, Andréia Machado; Alberici, Luciane Carla; Curti, Carlos
Fonte: Mitochondrion. Unidade: FCFRP
Assuntos: NEOPLASIAS, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREU, Gilberto Lázaro Pardo et al. The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler. Mitochondrion, v. 11, n. 2, p. 255-263, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mito.2010.10.008. Acesso em: 29 jun. 2024.
APA
Andreu, G. L. P., Figueredo, Y. N., Tudella, V. G., Rubio, O. C., Rodrigues, F. P., Pestana, C. R., et al. (2011). The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler. Mitochondrion, 11( 2), 255-263. doi:10.1016/j.mito.2010.10.008
NLM
Andreu GLP, Figueredo YN, Tudella VG, Rubio OC, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler [Internet]. Mitochondrion. 2011 ; 11( 2): 255-263.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mito.2010.10.008
Vancouver
Andreu GLP, Figueredo YN, Tudella VG, Rubio OC, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler [Internet]. Mitochondrion. 2011 ; 11( 2): 255-263.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mito.2010.10.008
Artigo de periodico
The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications (2011)
Pardo-Andreu, Gilberto L.; Nuñez-Figueredo, Yanier; Tudelli, Valeria G.; Cuesta-Rubio, Osmany; Rodrigues, Fernando P.; Pestana, Cezar R.; Uyemura, Sérgio Akira; Leopoldino, Andréia Machado; Alberici, Luciane Carla; Curti, Carlos
Fonte: Toxicology and Applied Pharmacology. Unidade: FCFRP
Assuntos: MITOCÔNDRIAS, ESTRESSE OXIDATIVO, TOXICOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PARDO-ANDREU, Gilberto L. et al. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications. Toxicology and Applied Pharmacology, v. 253, n. 3, p. 282-289, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2011.04.011. Acesso em: 29 jun. 2024.
APA
Pardo-Andreu, G. L., Nuñez-Figueredo, Y., Tudelli, V. G., Cuesta-Rubio, O., Rodrigues, F. P., Pestana, C. R., et al. (2011). The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications. Toxicology and Applied Pharmacology, 253( 3), 282-289. doi:10.1016/j.taap.2011.04.011
NLM
Pardo-Andreu GL, Nuñez-Figueredo Y, Tudelli VG, Cuesta-Rubio O, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications [Internet]. Toxicology and Applied Pharmacology. 2011 ; 253( 3): 282-289.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.taap.2011.04.011
Vancouver
Pardo-Andreu GL, Nuñez-Figueredo Y, Tudelli VG, Cuesta-Rubio O, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications [Internet]. Toxicology and Applied Pharmacology. 2011 ; 253( 3): 282-289.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.taap.2011.04.011
Artigo de periodico
Characterization of the stimulus for reactive oxygen species generation in calcium-overloaded mitochondria (2011)
Rodrigues, Fernando P.; Pestana, Cezar Rangel; Santos, Guilherme A. dos; Pardo-Andreu, Gilberto L.; Santos, Antônio Cardozo dos; Uyemura, Sérgio Akira; Alberici, Luciane Carla; Curti, Carlos
Fonte: Redox Report. Unidade: FCFRP
Assuntos: MITOCÕNDRIAS, CÁLCIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Fernando P. et al. Characterization of the stimulus for reactive oxygen species generation in calcium-overloaded mitochondria. Redox Report, v. 16, n. 3, p. 108-113, 2011Tradução . . Disponível em: https://doi.org/10.1179/1351000211y.0000000001. Acesso em: 29 jun. 2024.
APA
Rodrigues, F. P., Pestana, C. R., Santos, G. A. dos, Pardo-Andreu, G. L., Santos, A. C. dos, Uyemura, S. A., et al. (2011). Characterization of the stimulus for reactive oxygen species generation in calcium-overloaded mitochondria. Redox Report, 16( 3), 108-113. doi:10.1179/1351000211y.0000000001
NLM
Rodrigues FP, Pestana CR, Santos GA dos, Pardo-Andreu GL, Santos AC dos, Uyemura SA, Alberici LC, Curti C. Characterization of the stimulus for reactive oxygen species generation in calcium-overloaded mitochondria [Internet]. Redox Report. 2011 ; 16( 3): 108-113.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1179/1351000211y.0000000001
Vancouver
Rodrigues FP, Pestana CR, Santos GA dos, Pardo-Andreu GL, Santos AC dos, Uyemura SA, Alberici LC, Curti C. Characterization of the stimulus for reactive oxygen species generation in calcium-overloaded mitochondria [Internet]. Redox Report. 2011 ; 16( 3): 108-113.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1179/1351000211y.0000000001

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