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  • Source: Journal of Molecular Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      GARCIA, P. C. e REAL, C. C. e BRITTO, Luiz Roberto Giorgetti de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, v. 61, n. 4, p. 542-552, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-017-0896-y. Acesso em: 01 nov. 2024.
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      Garcia, P. C., Real, C. C., & Britto, L. R. G. de. (2017). The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, 61( 4), 542-552. doi:10.1007/s12031-017-0896-y
    • NLM

      Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
    • Vancouver

      Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
  • Source: Cell Transplantation. Unidades: ICB, IQ

    Subjects: DOENÇA DE PARKINSON, ESTRESSE OXIDATIVO, RECEPTORES DOPAMINÉRGICOS

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      FERRAZOLI, Enéas Galdini et al. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease. Cell Transplantation, v. 26, n. 4, p. 669-677, 2017Tradução . . Disponível em: https://doi.org/10.3727/096368917X695227. Acesso em: 01 nov. 2024.
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      Ferrazoli, E. G., Souza, H. D. N. de, Nascimento, I. C. do, Giacomelli, Á. O., Schwindt, T. T., Britto, L. R. G. de, & Ulrich, H. (2017). Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease. Cell Transplantation, 26( 4), 669-677. doi:10.3727/096368917X695227
    • NLM

      Ferrazoli EG, Souza HDN de, Nascimento IC do, Giacomelli ÁO, Schwindt TT, Britto LRG de, Ulrich H. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease [Internet]. Cell Transplantation. 2017 ; 26( 4): 669-677.[citado 2024 nov. 01 ] Available from: https://doi.org/10.3727/096368917X695227
    • Vancouver

      Ferrazoli EG, Souza HDN de, Nascimento IC do, Giacomelli ÁO, Schwindt TT, Britto LRG de, Ulrich H. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease [Internet]. Cell Transplantation. 2017 ; 26( 4): 669-677.[citado 2024 nov. 01 ] Available from: https://doi.org/10.3727/096368917X695227
  • Source: Neurotoxicity Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RAVELLI, Katherine Garcia et al. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, v. 31, n. 3, p. 327-333, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12640-016-9684-7. Acesso em: 01 nov. 2024.
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      Ravelli, K. G., Rosário, B. dos A., Camarini, R., Hernandes, M. S., & Britto, L. R. G. de. (2017). Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, 31( 3), 327-333. doi:10.1007/s12640-016-9684-7
    • NLM

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
    • Vancouver

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
  • Source: Translational Psychiatry. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RESENDE, L. S. et al. Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX. Translational Psychiatry, v. 6, n. 10, 2016Tradução . . Disponível em: https://doi.org/10.1038/tp.2016.202. Acesso em: 01 nov. 2024.
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      Resende, L. S., Amaral, C. E., Soares, R. B. S., Alves, A. S., Alves-dos-Santos, L., Britto, L. R. G. de, & Chiavegatto, S. (2016). Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX. Translational Psychiatry, 6( 10). doi:10.1038/tp.2016.202
    • NLM

      Resende LS, Amaral CE, Soares RBS, Alves AS, Alves-dos-Santos L, Britto LRG de, Chiavegatto S. Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX [Internet]. Translational Psychiatry. 2016 ; 6( 10):[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/tp.2016.202
    • Vancouver

      Resende LS, Amaral CE, Soares RBS, Alves AS, Alves-dos-Santos L, Britto LRG de, Chiavegatto S. Social stress in adolescents induces depression and brain-region-specific modulation of the transcription factor MAX [Internet]. Translational Psychiatry. 2016 ; 6( 10):[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/tp.2016.202
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FISIOLOGIA

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      KINJO, Erika et al. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus. PLOS ONE, v. 9, n. 10, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0109149. Acesso em: 01 nov. 2024.
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      Kinjo, E., Higa, G. S. V., Morya, E., Valle, A. C., Kihara, A. H., & Britto, L. R. G. de. (2014). Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus. PLOS ONE, 9( 10), 1-12. doi:10.1371/journal.pone.0109149
    • NLM

      Kinjo E, Higa GSV, Morya E, Valle AC, Kihara AH, Britto LRG de. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus [Internet]. PLOS ONE. 2014 ; 9( 10): 1-12.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0109149
    • Vancouver

      Kinjo E, Higa GSV, Morya E, Valle AC, Kihara AH, Britto LRG de. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus [Internet]. PLOS ONE. 2014 ; 9( 10): 1-12.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0109149
  • Source: Journal of Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      DE PASQUALE, Roberto et al. LTP and LTD in the visual cortex require the activation of NOX2. Journal of Neuroscience, v. 34, n. 38, p. 12778-12787, 2014Tradução . . Disponível em: https://doi.org/10.1523/JNEUROSCI.1414-14.2014. Acesso em: 01 nov. 2024.
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      De Pasquale, R., Beckhauser, T. F., Hernandes, M. S., & Britto, L. R. G. de. (2014). LTP and LTD in the visual cortex require the activation of NOX2. Journal of Neuroscience, 34( 38), 12778-12787. doi:10.1523/JNEUROSCI.1414-14.2014
    • NLM

      De Pasquale R, Beckhauser TF, Hernandes MS, Britto LRG de. LTP and LTD in the visual cortex require the activation of NOX2 [Internet]. Journal of Neuroscience. 2014 ; 34( 38): 12778-12787.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1523/JNEUROSCI.1414-14.2014
    • Vancouver

      De Pasquale R, Beckhauser TF, Hernandes MS, Britto LRG de. LTP and LTD in the visual cortex require the activation of NOX2 [Internet]. Journal of Neuroscience. 2014 ; 34( 38): 12778-12787.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1523/JNEUROSCI.1414-14.2014
  • Source: Journal of Neurotrauma. Unidades: FO, ICB

    Subjects: LASER NÃO CIRÚRGICO, NERVO TRIGÊMEO

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      MARTINS, Daniel de Oliveira et al. Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins. Journal of Neurotrauma, v. 30, n. 6, p. 480-486, 2013Tradução . . Disponível em: https://doi.org/10.1089/neu.2012.2603. Acesso em: 01 nov. 2024.
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      Martins, D. de O., Santos, F. M. dos, Oliveira, M. E. de, Britto, L. R. G. de, Lemos, J. B. D., & Chacur, M. (2013). Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins. Journal of Neurotrauma, 30( 6), 480-486. doi:10.1089/neu.2012.2603
    • NLM

      Martins D de O, Santos FM dos, Oliveira ME de, Britto LRG de, Lemos JBD, Chacur M. Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins [Internet]. Journal of Neurotrauma. 2013 ; 30( 6): 480-486.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1089/neu.2012.2603
    • Vancouver

      Martins D de O, Santos FM dos, Oliveira ME de, Britto LRG de, Lemos JBD, Chacur M. Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins [Internet]. Journal of Neurotrauma. 2013 ; 30( 6): 480-486.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1089/neu.2012.2603
  • Source: Cellular and Molecular Neurobiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEONELLI, Mauro e MARTINS, Daniel O. e BRITTO, Luiz Roberto Giorgetti de. Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases. Cellular and Molecular Neurobiology, v. 33, n. 3, p. 379-392, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10571-012-9904-5. Acesso em: 01 nov. 2024.
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      Leonelli, M., Martins, D. O., & Britto, L. R. G. de. (2013). Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases. Cellular and Molecular Neurobiology, 33( 3), 379-392. doi:10.1007/s10571-012-9904-5
    • NLM

      Leonelli M, Martins DO, Britto LRG de. Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases [Internet]. Cellular and Molecular Neurobiology. 2013 ; 33( 3): 379-392.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s10571-012-9904-5
    • Vancouver

      Leonelli M, Martins DO, Britto LRG de. Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases [Internet]. Cellular and Molecular Neurobiology. 2013 ; 33( 3): 379-392.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s10571-012-9904-5
  • Source: PLOS ONE. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      HERNANDES, Marina S. et al. Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration. PLOS ONE, v. 8, n. 9, p. 1-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0075532. Acesso em: 01 nov. 2024.
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      Hernandes, M. S., Santos, G. D. R., Café-Mendes, C. C., Lima, L. de S., Scavone, C., Munhoz, C. D., & Britto, L. R. G. de. (2013). Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration. PLOS ONE, 8( 9), 1-13. doi:10.1371/journal.pone.0075532
    • NLM

      Hernandes MS, Santos GDR, Café-Mendes CC, Lima L de S, Scavone C, Munhoz CD, Britto LRG de. Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration [Internet]. PLOS ONE. 2013 ; 8( 9): 1-13.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0075532
    • Vancouver

      Hernandes MS, Santos GDR, Café-Mendes CC, Lima L de S, Scavone C, Munhoz CD, Britto LRG de. Microglial cells are involved in the susceptibility of NADPH oxidase knockout mice to 6-Hydroxy-Dopamine- induced neurodegeneration [Internet]. PLOS ONE. 2013 ; 8( 9): 1-13.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0075532
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FISIOLOGIA

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      CHAVES-KIRSTEN, Gabriela Pena et al. Temporal changes of CB1 cannabinoid receptor in the basal ganglia as a possible structure-specific plasticity process in 6-OHDA lesioned rats. PLOS ONE, v. 8, n. 10, p. 1-15, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076874. Acesso em: 01 nov. 2024.
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      Chaves-Kirsten, G. P., Mazucanti, C. H. Y., Real, C. C., Souza, B. M., Britto, L. R. G. de, & Torrão, A. da S. (2013). Temporal changes of CB1 cannabinoid receptor in the basal ganglia as a possible structure-specific plasticity process in 6-OHDA lesioned rats. PLOS ONE, 8( 10), 1-15. doi:10.1371/journal.pone.0076874
    • NLM

      Chaves-Kirsten GP, Mazucanti CHY, Real CC, Souza BM, Britto LRG de, Torrão A da S. Temporal changes of CB1 cannabinoid receptor in the basal ganglia as a possible structure-specific plasticity process in 6-OHDA lesioned rats [Internet]. PLOS ONE. 2013 ; 8( 10): 1-15.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0076874
    • Vancouver

      Chaves-Kirsten GP, Mazucanti CHY, Real CC, Souza BM, Britto LRG de, Torrão A da S. Temporal changes of CB1 cannabinoid receptor in the basal ganglia as a possible structure-specific plasticity process in 6-OHDA lesioned rats [Internet]. PLOS ONE. 2013 ; 8( 10): 1-15.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0076874
  • Source: Brain Research Bulletin. Unidade: ICB

    Assunto: FISIOLOGIA

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      HERNANDES, Marina S. e BRITTO, Luiz Roberto Giorgetti de. Inflammatory responses in the rat superior colliculus after eye enucleation. Brain Research Bulletin, v. 101, p. 1-6, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2013.12.001. Acesso em: 01 nov. 2024.
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      Hernandes, M. S., & Britto, L. R. G. de. (2013). Inflammatory responses in the rat superior colliculus after eye enucleation. Brain Research Bulletin, 101, 1-6. doi:10.1016/j.brainresbull.2013.12.001
    • NLM

      Hernandes MS, Britto LRG de. Inflammatory responses in the rat superior colliculus after eye enucleation [Internet]. Brain Research Bulletin. 2013 ; 101 1-6.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.brainresbull.2013.12.001
    • Vancouver

      Hernandes MS, Britto LRG de. Inflammatory responses in the rat superior colliculus after eye enucleation [Internet]. Brain Research Bulletin. 2013 ; 101 1-6.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.brainresbull.2013.12.001
  • Source: Experimental Neurology. Unidade: ICB

    Assunto: FISIOLOGIA

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      KINJO, Erika Reime et al. A possible new mechanism for the control of miRNA expression in neurons. Experimental Neurology, v. 248, p. 546-558, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2013.07.022. Acesso em: 01 nov. 2024.
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      Kinjo, E. R., Higa, G. S. V., Sousa, E. de, Casado, O. A. N., Damico, M. V., Britto, L. R. G. de, & Kihara, A. H. (2013). A possible new mechanism for the control of miRNA expression in neurons. Experimental Neurology, 248, 546-558. doi:10.1016/j.expneurol.2013.07.022
    • NLM

      Kinjo ER, Higa GSV, Sousa E de, Casado OAN, Damico MV, Britto LRG de, Kihara AH. A possible new mechanism for the control of miRNA expression in neurons [Internet]. Experimental Neurology. 2013 ; 248 546-558.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.expneurol.2013.07.022
    • Vancouver

      Kinjo ER, Higa GSV, Sousa E de, Casado OAN, Damico MV, Britto LRG de, Kihara AH. A possible new mechanism for the control of miRNA expression in neurons [Internet]. Experimental Neurology. 2013 ; 248 546-558.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.expneurol.2013.07.022
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FISIOLOGIA

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      PASCHON, Vera et al. Blocking of connexin-mediated communication promotes neuroprotection during acute degeneration induced by mechanical trauma. PLOS ONE, v. 7, n. 9, p. 1-12, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0045449. Acesso em: 01 nov. 2024.
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      Paschon, V., Higa, G. S. V., Resende, R. R., Britto, L. R. G. de, & Kihara, A. H. (2012). Blocking of connexin-mediated communication promotes neuroprotection during acute degeneration induced by mechanical trauma. PLOS ONE, 7( 9), 1-12. doi:10.1371/journal.pone.0045449
    • NLM

      Paschon V, Higa GSV, Resende RR, Britto LRG de, Kihara AH. Blocking of connexin-mediated communication promotes neuroprotection during acute degeneration induced by mechanical trauma [Internet]. PLOS ONE. 2012 ; 7( 9): 1-12.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0045449
    • Vancouver

      Paschon V, Higa GSV, Resende RR, Britto LRG de, Kihara AH. Blocking of connexin-mediated communication promotes neuroprotection during acute degeneration induced by mechanical trauma [Internet]. PLOS ONE. 2012 ; 7( 9): 1-12.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0045449

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