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Artigo de periodico
Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients (2023)
Amaral, Natália Brasil do; Rodrigues, Tamara Silva; Giannini, Marcela Cavichioli; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Menezes, Pamella Indira da Silva Oliveira; Dib, Saulo Musse; Gigante, Samara Libich Gusmão; Benatti, Maíra Nilson; Rezek, Uebe C.; Emrich-Filho, Laerte L.; Sousa, Betania A. A.; Almeida, Sérgio Couto Luna de; Luppino-Assad, Rodrigo; Veras, Flávio Protásio ; Schneider, Ayda Henriques; Leiria, Luiz Osório Silveira ; Cunha, Larissa Dias da; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Martins, Maria Auxiliadora ; Borges, Marcos de Carvalho ; Fonseca, Benedito Antonio Lopes da ; Bollela, Valdes Roberto ; Del-Ben, Cristina Marta ; Cunha, Fernando de Queiroz ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Inflammation Research. Unidade: FMRP
Subjects: COVID-19, CITOCINAS, PROTEÍNAS, ALCALOIDES
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ABNT
AMARAL, Natália Brasil do et al. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, v. 72, n. 5, p. 895-899, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-023-01718-y. Acesso em: 13 jul. 2024.
APA
Amaral, N. B. do, Rodrigues, T. S., Giannini, M. C., Lopes, M. I. F., Bonjorno, L. P., Menezes, P. I. da S. O., et al. (2023). Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, 72( 5), 895-899. doi:10.1007/s00011-023-01718-y
NLM
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Vancouver
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Artigo de periodico
Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 (2023)
Formiga, Rodrigo de Oliveira; Amaral, Flávia C.; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza ; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Cunha, Fernando de Queiroz
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: COVID-19, LIPOPOLISSACARÍDEOS, METALOPROTEINASES, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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ABNT
FORMIGA, Rodrigo de Oliveira et al. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, v. 180, n. 11, p. 1460-1481, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16013. Acesso em: 13 jul. 2024.
APA
Formiga, R. de O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2023). Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, 180( 11), 1460-1481. doi:10.1111/bph.16013
NLM
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/bph.16013
Vancouver
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/bph.16013
Artigo de periodico
Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis (2023)
Souza, Flávio Falcão Lima de; Schneider, Ayda Henriques ; Machado, Caio Cavalcante ; Almeida, Sérgio Couto Luna de; Silva, Tarcília Aparecida da; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Clinical and Experimental Rheumatology. Unidade: FMRP
Subjects: BIOMARCADORES, ARTRITE REUMATOIDE, OSTEOARTRITE, ESTUDOS TRANSVERSAIS
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ABNT
SOUZA, Flávio Falcão Lima de et al. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, v. 41, n. 7, p. 1473-1479, 2023Tradução . . Disponível em: https://doi.org/10.55563/clinexprheumatol/flzpgb. Acesso em: 13 jul. 2024.
APA
Souza, F. F. L. de, Schneider, A. H., Machado, C. C., Almeida, S. C. L. de, Silva, T. A. da, Cunha, F. de Q., et al. (2023). Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, 41( 7), 1473-1479. doi:10.55563/clinexprheumatol/flzpgb
NLM
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 jul. 13 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Vancouver
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 jul. 13 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Artigo de periodico
Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function (2023)
Rocha, Eduardo Vieira da; Falchetti, Francisco; Pernomian, Laena; Mello, Marcela Maria Blascke de ; Parente, Juliana Montenegro ; Nogueira, Renato Corrêa; Gomes, Beatriz Quaresemin; Bertozi, Giuliana; Sanches-Lopes, Jessica M.; Tanus-Santos, José Eduardo ; Castro, Michele Mazzaron de
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidades: FMRP, FFCLRP
Subjects: PRESSÃO SANGUÍNEA, CARDIOMEGALIA, VASOS CORONÁRIOS, HIPERTENSÃO, FARMACOTERAPIA, NEFROPATIAS
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ABNT
ROCHA, Eduardo Vieira da et al. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 396, n. 5, p. 939-949, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00210-022-02349-6. Acesso em: 13 jul. 2024.
APA
Rocha, E. V. da, Falchetti, F., Pernomian, L., Mello, M. M. B. de, Parente, J. M., Nogueira, R. C., et al. (2023). Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function. Naunyn-Schmiedeberg's Archives of Pharmacology, 396( 5), 939-949. doi:10.1007/s00210-022-02349-6
NLM
Rocha EV da, Falchetti F, Pernomian L, Mello MMB de, Parente JM, Nogueira RC, Gomes BQ, Bertozi G, Sanches-Lopes JM, Tanus-Santos JE, Castro MM de. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2023 ; 396( 5): 939-949.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00210-022-02349-6
Vancouver
Rocha EV da, Falchetti F, Pernomian L, Mello MMB de, Parente JM, Nogueira RC, Gomes BQ, Bertozi G, Sanches-Lopes JM, Tanus-Santos JE, Castro MM de. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2023 ; 396( 5): 939-949.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00210-022-02349-6
Artigo de periodico
Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections (2023)
Silva, Juliana Almeida da ; Almada, Rafael Carvalho ; Falconi-Sobrinho, Luiz Luciano ; Pigatto, Glauce Regina; Hernandes, Paloma Molina; Coimbra, Norberto Cysne
Source: Brain Research Bulletin. Unidade: FMRP
Subjects: ANTAGONISTAS, VIAS NEURAIS, MEDO, NEUROTRANSMISSORES
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ABNT
SILVA, Juliana Almeida da et al. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections. Brain Research Bulletin, v. 192, p. 128-141, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2022.11.014. Acesso em: 13 jul. 2024.
APA
Silva, J. A. da, Almada, R. C., Falconi-Sobrinho, L. L., Pigatto, G. R., Hernandes, P. M., & Coimbra, N. C. (2023). Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections. Brain Research Bulletin, 192, 128-141. doi:10.1016/j.brainresbull.2022.11.014
NLM
Silva JA da, Almada RC, Falconi-Sobrinho LL, Pigatto GR, Hernandes PM, Coimbra NC. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections [Internet]. Brain Research Bulletin. 2023 ; 192 128-141.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.11.014
Vancouver
Silva JA da, Almada RC, Falconi-Sobrinho LL, Pigatto GR, Hernandes PM, Coimbra NC. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections [Internet]. Brain Research Bulletin. 2023 ; 192 128-141.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.11.014
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 13 jul. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats (2023)
Cavichioli, Andreza M.; Silva, Thamyris Santos; Grace, Anthony A.; Guimarães, Francisco Silveira ; Gomes, Felipe Villela
Source: Schizophrenia Bulletin. Unidade: FMRP
Subjects: POTENCIAIS DE AÇÃO, NEUROTRANSMISSORES, ESQUIZOFRENIA, HIPOCAMPO
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ABNT
CAVICHIOLI, Andreza M. et al. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats. Schizophrenia Bulletin, v. 49, n. 1, p. 68-77, 2023Tradução . . Disponível em: https://doi.org/10.1093/schbul/sbac106. Acesso em: 13 jul. 2024.
APA
Cavichioli, A. M., Silva, T. S., Grace, A. A., Guimarães, F. S., & Gomes, F. V. (2023). Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats. Schizophrenia Bulletin, 49( 1), 68-77. doi:10.1093/schbul/sbac106
NLM
Cavichioli AM, Silva TS, Grace AA, Guimarães FS, Gomes FV. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats [Internet]. Schizophrenia Bulletin. 2023 ; 49( 1): 68-77.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/schbul/sbac106
Vancouver
Cavichioli AM, Silva TS, Grace AA, Guimarães FS, Gomes FV. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats [Internet]. Schizophrenia Bulletin. 2023 ; 49( 1): 68-77.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/schbul/sbac106
Artigo de periodico
Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 (2023)
Fraga-Silva, Thais Fernanda de Campos ; Rosa, Ualter Guilherme Cipriano; Fumagalli, Marcílio Jorge ; Corrêa, Giseli Furlan ; Fuzo, Carlos Alessandro ; Santos, Douglas dos; Mestriner, Fabíola Leslie Antunes Cardoso ; Ribeiro, Christiane Becari ; Teixeira-Carvalho, Andrea; Coelho-dos-Reis, Jordana; Menegueti, Mayra Gonçalves ; Figueiredo, Luiz Tadeu Moraes ; Cunha, Larissa Dias da; Martins-Filho, Olindo A.; Baruffi, Marcelo Dias ; Martins, Maria Auxiliadora ; Passaglia, Rita de Cassia Aleixo Tostes ; Bonato, Vânia Luiza Deperon
Source: iScience. Unidades: FMRP, EERP, FCFRP
Subjects: SISTEMA IMUNE, VIROLOGIA, MACRÓFAGOS
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ABNT
FRAGA-SILVA, Thais Fernanda de Campos et al. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, v. 26, n. 12, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108366. Acesso em: 13 jul. 2024.
APA
Fraga-Silva, T. F. de C., Rosa, U. G. C., Fumagalli, M. J., Corrêa, G. F., Fuzo, C. A., Santos, D. dos, et al. (2023). Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, 26( 12), 1-19. doi:10.1016/j.isci.2023.108366
NLM
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Vancouver
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Artigo de periodico
Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 (2023)
Dourado, Thales de Mileto Henrique; Assis, Victor Oliveira; Awata, Wanessa Mayumi Carvalho; Mello, Marcela Maria Blascke de ; Cárnio, Evelin Capellari ; Castro, Michele Mazzaron de ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: EERP, FMRP
Subjects: ETANOL, ESPÉCIES REATIVAS DE OXIGÊNIO, TROMBOXANOS, HIPERTENSÃO, ENDOTÉLIO VASCULAR
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ABNT
DOURADO, Thales de Mileto Henrique et al. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2. European Journal of Pharmacology, v. 949, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2023.175723. Acesso em: 13 jul. 2024.
APA
Dourado, T. de M. H., Assis, V. O., Awata, W. M. C., Mello, M. M. B. de, Cárnio, E. C., Castro, M. M. de, & Tirapelli, C. R. (2023). Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2. European Journal of Pharmacology, 949, 1-10. doi:10.1016/j.ejphar.2023.175723
NLM
Dourado T de MH, Assis VO, Awata WMC, Mello MMB de, Cárnio EC, Castro MM de, Tirapelli CR. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 [Internet]. European Journal of Pharmacology. 2023 ; 949 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2023.175723
Vancouver
Dourado T de MH, Assis VO, Awata WMC, Mello MMB de, Cárnio EC, Castro MM de, Tirapelli CR. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 [Internet]. European Journal of Pharmacology. 2023 ; 949 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2023.175723
Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2023)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Source: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Subjects: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
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ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 13 jul. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2023). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:https://doi.org/10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections (2023)
Santiago-Carvalho, Igor ; Almeida-Santos, Gislane ; Macêdo, Bruna de Gois ; Barbosa-Bomfim, Caio César ; Almeida, Fabricio Moreira ; Cione, Marcos Vinícios Pinheiro ; Vardam-Kaur, Trupti ; Masuda, Mia ; Dijk, Sarah Van ; Melo, Bruno Marcel Silva de ; Nascimento, Rogério Silva do; Souza, Rebeka da Conceição ; Peixoto-Rangel, Alba Lucínia ; Coutinho-Silva, Robson ; Lassounskaia, Elena ; Silva, Henrique Borges da ; Hirata, Mario Hiroyuki ; Alves Filho, José Carlos ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Source: Cell Reports. Unidades: FCF, FMRP, ICB
Subjects: IMUNOLOGIA, PNEUMOPATIAS, TUBERCULOSE PULMONAR, INFLUENZA, MODELOS ANIMAIS DE DOENÇAS, RATOS, QUIMIOCINAS, PULMÃO
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ABNT
SANTIAGO-CARVALHO, Igor et al. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Cell Reports, v. 42, n. 11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2023.113448. Acesso em: 13 jul. 2024.
APA
Santiago-Carvalho, I., Almeida-Santos, G., Macêdo, B. de G., Barbosa-Bomfim, C. C., Almeida, F. M., Cione, M. V. P., et al. (2023). T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Cell Reports, 42( 11). doi:10.1016/j.celrep.2023.113448
NLM
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Vardam-Kaur T, Masuda M, Dijk SV, Melo BMS de, Nascimento RS do, Souza R da C, Peixoto-Rangel AL, Coutinho-Silva R, Lassounskaia E, Silva HB da, Hirata MH, Alves Filho JC, Alvarez JM, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Cell Reports. 2023 ; 42( 11):[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.celrep.2023.113448
Vancouver
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Vardam-Kaur T, Masuda M, Dijk SV, Melo BMS de, Nascimento RS do, Souza R da C, Peixoto-Rangel AL, Coutinho-Silva R, Lassounskaia E, Silva HB da, Hirata MH, Alves Filho JC, Alvarez JM, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Cell Reports. 2023 ; 42( 11):[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.celrep.2023.113448
Artigo de periodico
Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor (2023)
Rodrigues, Daniel ; Machado, Mirele Resende ; Alves, Juliano Vilela ; Fraga-Silva, Thais Fernanda de Campos ; Martins, Ronaldo Bragança ; Campos, Lígia Cristina Borges ; Francisco, Daniely Franco ; Couto, Ariel Emiliano Souza do ; Bonato, Vânia Luiza Deperon ; Arruda, Eurico ; Becari, Christiane ; Auxiliadora-Martins, Maria ; Louzada Júnior, Paulo ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FCFRP, FMRP, FOB
Subjects: CÉLULAS ENDOTELIAIS, CITOCINAS, COVID-19, ANORMALIDADES CARDIOVASCULARES
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ABNT
RODRIGUES, Daniel et al. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor. American Journal of Physiology. Heart and Circulatory Physiology, v. 325, n. 2, p. H252–H263 Aug. 2023, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00096.2023. Acesso em: 13 jul. 2024.
APA
Rodrigues, D., Machado, M. R., Alves, J. V., Fraga-Silva, T. F. de C., Martins, R. B., Campos, L. C. B., et al. (2023). Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor. American Journal of Physiology. Heart and Circulatory Physiology, 325( 2), H252–H263 Aug. 2023. doi:10.1152/ajpheart.00096.2023
NLM
Rodrigues D, Machado MR, Alves JV, Fraga-Silva TF de C, Martins RB, Campos LCB, Francisco DF, Couto AES do, Bonato VLD, Arruda E, Becari C, Auxiliadora-Martins M, Louzada Júnior P, Costa RM da, Tostes R de CA. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 325( 2): H252–H263 Aug. 2023.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00096.2023
Vancouver
Rodrigues D, Machado MR, Alves JV, Fraga-Silva TF de C, Martins RB, Campos LCB, Francisco DF, Couto AES do, Bonato VLD, Arruda E, Becari C, Auxiliadora-Martins M, Louzada Júnior P, Costa RM da, Tostes R de CA. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 325( 2): H252–H263 Aug. 2023.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00096.2023
Artigo de periodico
Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection (2023)
Moreira, Thaiane Pinto; Sousa, Carla Daiane Ferreira de; Costa, Victor Rodrigues de Melo; Queiroz Junior, Celso Martins; Santos, Franciele Martins; Bonilha, Caio Santos ; Ésper, Lísia Maria; Nogueira, Mauricio Lacerda ; Cunha, Thiago Mattar ; Teixeira, Mauro Martins ; Costa, Vivian Vasconcelos; Souza, Daniele da Glória de
Source: Immunology. Unidade: FMRP
Subjects: ARTRALGIA, VÍRUS CHIKUNGUNYA, NECROSE, REPLICAÇÃO VIRAL
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ABNT
MOREIRA, Thaiane Pinto et al. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, v. 168, n. 3, p. 444-458, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13583. Acesso em: 13 jul. 2024.
APA
Moreira, T. P., Sousa, C. D. F. de, Costa, V. R. de M., Queiroz Junior, C. M., Santos, F. M., Bonilha, C. S., et al. (2023). Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, 168( 3), 444-458. doi:10.1111/imm.13583
NLM
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/imm.13583
Vancouver
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/imm.13583
Artigo de periodico
Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures (2022)
Crespo, Luiz Gustavo Soares Carvalho; Leite Júnior, Joaquim Barbosa; Bastos, João Marcos de Mello; Samuels, Richard Ian; Coimbra, Norberto Cysne ; Carey, Robert J.; Carrera, Marinete Pinheiro
Source: Pharmacology Biochemistry and Behavior. Unidade: FMRP
Subjects: CÉREBRO, CONDICIONAMENTO OPERANTE, MORFINA, ESTIMULAÇÃO CEREBRAL
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ABNT
CRESPO, Luiz Gustavo Soares Carvalho et al. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures. Pharmacology Biochemistry and Behavior, v. 214, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2022.173356. Acesso em: 13 jul. 2024.
APA
Crespo, L. G. S. C., Leite Júnior, J. B., Bastos, J. M. de M., Samuels, R. I., Coimbra, N. C., Carey, R. J., & Carrera, M. P. (2022). Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures. Pharmacology Biochemistry and Behavior, 214, 1-10. doi:10.1016/j.pbb.2022.173356
NLM
Crespo LGSC, Leite Júnior JB, Bastos JM de M, Samuels RI, Coimbra NC, Carey RJ, Carrera MP. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures [Internet]. Pharmacology Biochemistry and Behavior. 2022 ; 214 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.pbb.2022.173356
Vancouver
Crespo LGSC, Leite Júnior JB, Bastos JM de M, Samuels RI, Coimbra NC, Carey RJ, Carrera MP. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures [Internet]. Pharmacology Biochemistry and Behavior. 2022 ; 214 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.pbb.2022.173356
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 13 jul. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
Panic‐like responses of female Wistar rats confronted by Bothrops alternatus pit vipers, or exposure to acute hypoxia: effect of oestrous cycle (2022)
Sgobbi, Renata Ferreira ; Figueiredo, Rebeca Machado de ; Frias, Alana Tercino ; Matthiesen, Melina; Batistela, Matheus Fitipaldi; Falconi‐Sobrinho, Luiz Luciano ; Costa, Heloísa Helena Vilela; Sá, Susana Isabel; Lovick, Thelma Anderson; Zangrossi Junior, Hélio ; Coimbra, Norberto Cysne
Source: European Journal of Neuroscience. Unidades: FMRP, FFCLRP
Subjects: ANÓXIA, CICLO ESTRAL ANIMAL, PÂNICO, SERPENTES
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ABNT
SGOBBI, Renata Ferreira et al. Panic‐like responses of female Wistar rats confronted by Bothrops alternatus pit vipers, or exposure to acute hypoxia: effect of oestrous cycle. European Journal of Neuroscience, v. 55, n. 1, p. 32-48, 2022Tradução . . Disponível em: https://doi.org/10.1111/ejn.15548. Acesso em: 13 jul. 2024.
APA
Sgobbi, R. F., Figueiredo, R. M. de, Frias, A. T., Matthiesen, M., Batistela, M. F., Falconi‐Sobrinho, L. L., et al. (2022). Panic‐like responses of female Wistar rats confronted by Bothrops alternatus pit vipers, or exposure to acute hypoxia: effect of oestrous cycle. European Journal of Neuroscience, 55( 1), 32-48. doi:10.1111/ejn.15548
NLM
Sgobbi RF, Figueiredo RM de, Frias AT, Matthiesen M, Batistela MF, Falconi‐Sobrinho LL, Costa HHV, Sá SI, Lovick TA, Zangrossi Junior H, Coimbra NC. Panic‐like responses of female Wistar rats confronted by Bothrops alternatus pit vipers, or exposure to acute hypoxia: effect of oestrous cycle [Internet]. European Journal of Neuroscience. 2022 ; 55( 1): 32-48.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/ejn.15548
Vancouver
Sgobbi RF, Figueiredo RM de, Frias AT, Matthiesen M, Batistela MF, Falconi‐Sobrinho LL, Costa HHV, Sá SI, Lovick TA, Zangrossi Junior H, Coimbra NC. Panic‐like responses of female Wistar rats confronted by Bothrops alternatus pit vipers, or exposure to acute hypoxia: effect of oestrous cycle [Internet]. European Journal of Neuroscience. 2022 ; 55( 1): 32-48.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/ejn.15548
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
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ABNT
COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 13 jul. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 jul. 13 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 jul. 13 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex (2022)
Katayama, Pedro L.; Leirão, Isabela P.; Kanashiro, Alexandre; Luiz, João Paulo Mesquita ; Cunha, Fernando de Queiroz ; Navegantes, Luiz Carlos Carvalho ; Menani, Jose Vanderlei; Zoccal, Daniel Breseghello; Colombari, Debora Simoes de Almeida ; Colombari, Eduardo
Source: Brain, Behavior, and Immunity. Unidade: FMRP
Subjects: SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, ANTI-INFLAMATÓRIOS
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ABNT
KATAYAMA, Pedro L. et al. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, v. 102, p. 370-386, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2022.03.014. Acesso em: 13 jul. 2024.
APA
Katayama, P. L., Leirão, I. P., Kanashiro, A., Luiz, J. P. M., Cunha, F. de Q., Navegantes, L. C. C., et al. (2022). The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, 102, 370-386. doi:10.1016/j.bbi.2022.03.014
NLM
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Vancouver
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Subjects: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 13 jul. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation (2022)
Borghi, Sergio M.; Bussulo, Sylvia K. D.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Carvalho, Thacyana T.; Rasquel-Oliveira, Fernanda S.; Zaninelli, Tiago H.; Ferraz, Camila R.; Casella, Antônio M. B.; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar ; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: EXERCÍCIOS DE RESISTÊNCIA MUSCULAR, MEDULA ESPINHAL, INFLAMAÇÃO, DOR
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ABNT
BORGHI, Sergio M. et al. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, v. 12, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphar.2021.734091. Acesso em: 13 jul. 2024.
APA
Borghi, S. M., Bussulo, S. K. D., Pinho-Ribeiro, F. A., Fattori, V., Carvalho, T. T., Rasquel-Oliveira, F. S., et al. (2022). Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, 12, 1-17. doi:10.3389/fphar.2021.734091
NLM
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 jul. 13 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Vancouver
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 jul. 13 ] Available from: https://doi.org/10.3389/fphar.2021.734091

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