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Artigo de periodico
Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation (2024)
Costa, Rafael Menezes da ; Dias, Mayara C.; Alves, Juliano Vilela ; Silva, João Lucas M.; Rodrigues, Daniel; Silva, Josiane Fernandes da ; Francescato, Heloisa Della Coletta; Ramalho, Leandra Naira Zambelli ; Coimbra, Terezila Machado ; Passaglia, Rita de Cassia Aleixo Tostes
Fonte: Biochemical Pharmacology. Unidade: FMRP
Assuntos: RIM, HIPERGLICEMIA, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
COSTA, Rafael Menezes da et al. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, v. 220, p. 1-10, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2023.115982. Acesso em: 13 jul. 2024.
APA
Costa, R. M. da, Dias, M. C., Alves, J. V., Silva, J. L. M., Rodrigues, D., Silva, J. F. da, et al. (2024). Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, 220, 1-10. doi:10.1016/j.bcp.2023.115982
NLM
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Vancouver
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Artigo de periodico
Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study (2024)
Pinto, Jairo Vinicius Merege de Mello Cruz; Crippa, José Alexandre de Souza ; Ceresér, Keila Maria; Vianna-Sulzbach, Miréia Fortes; Silveira Júnior, Érico de Moura; Rosa, Gabriel Santana da; Silva, Manoella Guatimuzim Testa da; Hizo, Gabriel Henrique; Medeiros, Leonardo Simão; Oliveira, Carlos Eduardo Santana de; Bristot, Giovana; Campos, Alline Cristina de ; Guimarães, Francisco Silveira ; Hallak, Jaime Eduardo Cecilio ; Zuardi, Antônio Waldo ; Yatham, Lakshmi N.; Kapczinski, Flávio; Kauer-Sant’anna, Márcia
Fonte: The Canadian Journal of Psychiatry. Unidades: FMRP, ESALQ
Assuntos: TRANSTORNO BIPOLAR, HUMOR, DEPRESSÃO, CANNABIS, CANABINOIDES, ENSAIO CLÍNICO CONTROLADO RANDOMIZADO
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ABNT
PINTO, Jairo Vinicius Merege de Mello Cruz et al. Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study. The Canadian Journal of Psychiatry, v. 69, n. 4, p. 242-251, 2024Tradução . . Disponível em: https://doi.org/10.1177/07067437231209650. Acesso em: 13 jul. 2024.
APA
Pinto, J. V. M. de M. C., Crippa, J. A. de S., Ceresér, K. M., Vianna-Sulzbach, M. F., Silveira Júnior, É. de M., Rosa, G. S. da, et al. (2024). Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study. The Canadian Journal of Psychiatry, 69( 4), 242-251. doi:10.1177/07067437231209650
NLM
Pinto JVM de MC, Crippa JA de S, Ceresér KM, Vianna-Sulzbach MF, Silveira Júnior É de M, Rosa GS da, Silva MGT da, Hizo GH, Medeiros LS, Oliveira CES de, Bristot G, Campos AC de, Guimarães FS, Hallak JEC, Zuardi AW, Yatham LN, Kapczinski F, Kauer-Sant’anna M. Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study [Internet]. The Canadian Journal of Psychiatry. 2024 ; 69( 4): 242-251.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1177/07067437231209650
Vancouver
Pinto JVM de MC, Crippa JA de S, Ceresér KM, Vianna-Sulzbach MF, Silveira Júnior É de M, Rosa GS da, Silva MGT da, Hizo GH, Medeiros LS, Oliveira CES de, Bristot G, Campos AC de, Guimarães FS, Hallak JEC, Zuardi AW, Yatham LN, Kapczinski F, Kauer-Sant’anna M. Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study [Internet]. The Canadian Journal of Psychiatry. 2024 ; 69( 4): 242-251.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1177/07067437231209650
Artigo de periodico
Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction (2024)
Morelo, David Fernando Colon ; Wanderley, Carlos Wagner de Souza ; Turato, Walter Miguel; Borges, Vanessa F.; Franchin, Marcelo; Castanheira, Fernanda Vargas e Silva; Nascimento, Daniele Carvalho Bernardo; Prado, Douglas da Silva; Lima, Mikhael Haruo Fernandes de ; Volpon, Leila Costa; Kavaguti, Silvia K.; Carlotti, Ana Paula de Carvalho Panzeri ; Carmona, Fabio ; Franklin, Bernardo S.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Fonte: British Journal of Pharmacology. Unidades: FCF, FMRP
Assuntos: MACRÓFAGOS, SEPSE, CRIANÇAS, IMUNOSSUPRESSÃO, SISTEMA IMUNE
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ABNT
MORELO, David Fernando Colon et al. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction. British Journal of Pharmacology, v. 181, n. 8, p. 1308-1323, 2024Tradução . . Disponível em: https://doi.org/10.1111/bph.16286. Acesso em: 13 jul. 2024.
APA
Morelo, D. F. C., Wanderley, C. W. de S., Turato, W. M., Borges, V. F., Franchin, M., Castanheira, F. V. e S., et al. (2024). Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction. British Journal of Pharmacology, 181( 8), 1308-1323. doi:10.1111/bph.16286
NLM
Morelo DFC, Wanderley CW de S, Turato WM, Borges VF, Franchin M, Castanheira FV e S, Nascimento DCB, Prado D da S, Lima MHF de, Volpon LC, Kavaguti SK, Carlotti AP de CP, Carmona F, Franklin BS, Cunha TM, Alves Filho JCF, Cunha F de Q. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction [Internet]. British Journal of Pharmacology. 2024 ; 181( 8): 1308-1323.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/bph.16286
Vancouver
Morelo DFC, Wanderley CW de S, Turato WM, Borges VF, Franchin M, Castanheira FV e S, Nascimento DCB, Prado D da S, Lima MHF de, Volpon LC, Kavaguti SK, Carlotti AP de CP, Carmona F, Franklin BS, Cunha TM, Alves Filho JCF, Cunha F de Q. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction [Internet]. British Journal of Pharmacology. 2024 ; 181( 8): 1308-1323.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/bph.16286
Artigo de periodico
Clinical, immunological, and microbiological analysis of the association between periodontitis and COVID-19: a case–control study (2024)
Bemquerer, Larissa Marques ; Oliveira, Sicília Rezende ; Arruda, José Alcides Almeida de ; Costa, Fernanda Pereira Delgado ; Miguita, Lucyene ; Bemquerer, Ana Luísa Marques; Sena, Ana Carolina Velasco Pondé de ; Souza, Alessandra Figueiredo de ; Mendes, Daniel Fajardo ; Schneider, Ayda Henriques ; Azevedo, Michelle de Campos Soriani ; Travassos, Denise Vieira ; Garlet, Gustavo Pompermaier ; Cunha, Fernando de Queiroz ; Aguiar, Renato Santana de ; Souza, Renan Pedra de ; Gomez, Ricardo Santiago ; Spahr, Axel ; Obregon-Miano, Fabian ; Abreu, Lucas Guimarães ; Costa, Fernando Oliveira ; Silva, Tarcília Aparecida
Fonte: Odontology. Unidades: FOB, FMRP
Assuntos: COVID-19, DOENÇAS PERIODONTAIS, CITOCINAS, PERIODONTITE
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ABNT
BEMQUERER, Larissa Marques et al. Clinical, immunological, and microbiological analysis of the association between periodontitis and COVID-19: a case–control study. Odontology, v. 112, p. 208–220, 2024Tradução . . Disponível em: https://doi.org/10.1007/s10266-023-00811-2. Acesso em: 13 jul. 2024.
APA
Bemquerer, L. M., Oliveira, S. R., Arruda, J. A. A. de, Costa, F. P. D., Miguita, L., Bemquerer, A. L. M., et al. (2024). Clinical, immunological, and microbiological analysis of the association between periodontitis and COVID-19: a case–control study. Odontology, 112, 208–220. doi:10.1007/s10266-023-00811-2
NLM
Bemquerer LM, Oliveira SR, Arruda JAA de, Costa FPD, Miguita L, Bemquerer ALM, Sena ACVP de, Souza AF de, Mendes DF, Schneider AH, Azevedo M de CS, Travassos DV, Garlet GP, Cunha F de Q, Aguiar RS de, Souza RP de, Gomez RS, Spahr A, Obregon-Miano F, Abreu LG, Costa FO, Silva TA. Clinical, immunological, and microbiological analysis of the association between periodontitis and COVID-19: a case–control study [Internet]. Odontology. 2024 ; 112 208–220.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s10266-023-00811-2
Vancouver
Bemquerer LM, Oliveira SR, Arruda JAA de, Costa FPD, Miguita L, Bemquerer ALM, Sena ACVP de, Souza AF de, Mendes DF, Schneider AH, Azevedo M de CS, Travassos DV, Garlet GP, Cunha F de Q, Aguiar RS de, Souza RP de, Gomez RS, Spahr A, Obregon-Miano F, Abreu LG, Costa FO, Silva TA. Clinical, immunological, and microbiological analysis of the association between periodontitis and COVID-19: a case–control study [Internet]. Odontology. 2024 ; 112 208–220.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s10266-023-00811-2
Artigo de periodico
A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice (2024)
Lessa, Tássia Liz Araújo dos Santos; Correia, Thiago Macêdo Lopes ; Santos, Talita Costa dos; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da ; Cavallini, Maria Cláudia Magalhães; Rocha, Leonardo Silva; Peixoto, Álbert Souza; Cugnasca, Beatriz dos Santos ; Cervi, Gustavo ; Correra, Thiago Carita ; Gonçalves, Augusto Cesar; Festuccia, William Tadeu Lara ; Cunha, Thiago Mattar ; Yatsuda, Regiane ; Magalhães, Amélia Cristina Mendes de ; Santos, Alcindo Aparecido dos ; Meotti, Flavia Carla ; Queiroz, Raphael Ferreira
Fonte: Free Radical Research. Unidades: IQ, ICB, FMRP
Assuntos: NEUTRÓFILOS, INFLAMAÇÃO
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ABNT
LESSA, Tássia Liz Araújo dos Santos et al. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research, 2024Tradução . . Disponível em: https://dx.doi.org/10.1080/10715762.2024.2336566. Acesso em: 13 jul. 2024.
APA
Lessa, T. L. A. dos S., Correia, T. M. L., Santos, T. C. dos, Silva, R. P. da, Silva, B. P. da, Cavallini, M. C. M., et al. (2024). A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research. doi:10.1080/10715762.2024.2336566
NLM
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto ÁS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Festuccia WTL, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ;[citado 2024 jul. 13 ] Available from: https://dx.doi.org/10.1080/10715762.2024.2336566
Vancouver
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto ÁS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Festuccia WTL, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ;[citado 2024 jul. 13 ] Available from: https://dx.doi.org/10.1080/10715762.2024.2336566
Artigo de periodico
Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients (2023)
Amaral, Natália Brasil do; Rodrigues, Tamara Silva; Giannini, Marcela Cavichioli; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Menezes, Pamella Indira da Silva Oliveira; Dib, Saulo Musse; Gigante, Samara Libich Gusmão; Benatti, Maíra Nilson; Rezek, Uebe C.; Emrich-Filho, Laerte L.; Sousa, Betania A. A.; Almeida, Sérgio Couto Luna de; Luppino-Assad, Rodrigo; Veras, Flávio Protásio ; Schneider, Ayda Henriques; Leiria, Luiz Osório Silveira ; Cunha, Larissa Dias da; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Martins, Maria Auxiliadora ; Borges, Marcos de Carvalho ; Fonseca, Benedito Antonio Lopes da ; Bollela, Valdes Roberto ; Del-Ben, Cristina Marta ; Cunha, Fernando de Queiroz ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Fonte: Inflammation Research. Unidade: FMRP
Assuntos: COVID-19, CITOCINAS, PROTEÍNAS, ALCALOIDES
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ABNT
AMARAL, Natália Brasil do et al. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, v. 72, n. 5, p. 895-899, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-023-01718-y. Acesso em: 13 jul. 2024.
APA
Amaral, N. B. do, Rodrigues, T. S., Giannini, M. C., Lopes, M. I. F., Bonjorno, L. P., Menezes, P. I. da S. O., et al. (2023). Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, 72( 5), 895-899. doi:10.1007/s00011-023-01718-y
NLM
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Vancouver
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Artigo de periodico
Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 (2023)
Formiga, Rodrigo de Oliveira; Amaral, Flávia C.; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza ; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Cunha, Fernando de Queiroz
Fonte: British Journal of Pharmacology. Unidade: FMRP
Assuntos: COVID-19, LIPOPOLISSACARÍDEOS, METALOPROTEINASES, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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ABNT
FORMIGA, Rodrigo de Oliveira et al. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, v. 180, n. 11, p. 1460-1481, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16013. Acesso em: 13 jul. 2024.
APA
Formiga, R. de O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2023). Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, 180( 11), 1460-1481. doi:10.1111/bph.16013
NLM
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/bph.16013
Vancouver
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/bph.16013
Artigo de periodico
Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis (2023)
Souza, Flávio Falcão Lima de; Schneider, Ayda Henriques ; Machado, Caio Cavalcante ; Almeida, Sérgio Couto Luna de; Silva, Tarcília Aparecida da; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Fonte: Clinical and Experimental Rheumatology. Unidade: FMRP
Assuntos: BIOMARCADORES, ARTRITE REUMATOIDE, OSTEOARTRITE, ESTUDOS TRANSVERSAIS
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ABNT
SOUZA, Flávio Falcão Lima de et al. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, v. 41, n. 7, p. 1473-1479, 2023Tradução . . Disponível em: https://doi.org/10.55563/clinexprheumatol/flzpgb. Acesso em: 13 jul. 2024.
APA
Souza, F. F. L. de, Schneider, A. H., Machado, C. C., Almeida, S. C. L. de, Silva, T. A. da, Cunha, F. de Q., et al. (2023). Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, 41( 7), 1473-1479. doi:10.55563/clinexprheumatol/flzpgb
NLM
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 jul. 13 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Vancouver
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 jul. 13 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Artigo de periodico
Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function (2023)
Rocha, Eduardo Vieira da; Falchetti, Francisco; Pernomian, Laena; Mello, Marcela Maria Blascke de ; Parente, Juliana Montenegro ; Nogueira, Renato Corrêa; Gomes, Beatriz Quaresemin; Bertozi, Giuliana; Sanches-Lopes, Jessica M.; Tanus-Santos, José Eduardo ; Castro, Michele Mazzaron de
Fonte: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidades: FMRP, FFCLRP
Assuntos: PRESSÃO SANGUÍNEA, CARDIOMEGALIA, VASOS CORONÁRIOS, HIPERTENSÃO, FARMACOTERAPIA, NEFROPATIAS
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ABNT
ROCHA, Eduardo Vieira da et al. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 396, n. 5, p. 939-949, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00210-022-02349-6. Acesso em: 13 jul. 2024.
APA
Rocha, E. V. da, Falchetti, F., Pernomian, L., Mello, M. M. B. de, Parente, J. M., Nogueira, R. C., et al. (2023). Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function. Naunyn-Schmiedeberg's Archives of Pharmacology, 396( 5), 939-949. doi:10.1007/s00210-022-02349-6
NLM
Rocha EV da, Falchetti F, Pernomian L, Mello MMB de, Parente JM, Nogueira RC, Gomes BQ, Bertozi G, Sanches-Lopes JM, Tanus-Santos JE, Castro MM de. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2023 ; 396( 5): 939-949.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00210-022-02349-6
Vancouver
Rocha EV da, Falchetti F, Pernomian L, Mello MMB de, Parente JM, Nogueira RC, Gomes BQ, Bertozi G, Sanches-Lopes JM, Tanus-Santos JE, Castro MM de. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2023 ; 396( 5): 939-949.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00210-022-02349-6
Artigo de periodico
Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections (2023)
Silva, Juliana Almeida da ; Almada, Rafael Carvalho ; Falconi-Sobrinho, Luiz Luciano ; Pigatto, Glauce Regina; Hernandes, Paloma Molina; Coimbra, Norberto Cysne
Fonte: Brain Research Bulletin. Unidade: FMRP
Assuntos: ANTAGONISTAS, VIAS NEURAIS, MEDO, NEUROTRANSMISSORES
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ABNT
SILVA, Juliana Almeida da et al. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections. Brain Research Bulletin, v. 192, p. 128-141, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2022.11.014. Acesso em: 13 jul. 2024.
APA
Silva, J. A. da, Almada, R. C., Falconi-Sobrinho, L. L., Pigatto, G. R., Hernandes, P. M., & Coimbra, N. C. (2023). Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections. Brain Research Bulletin, 192, 128-141. doi:10.1016/j.brainresbull.2022.11.014
NLM
Silva JA da, Almada RC, Falconi-Sobrinho LL, Pigatto GR, Hernandes PM, Coimbra NC. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections [Internet]. Brain Research Bulletin. 2023 ; 192 128-141.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.11.014
Vancouver
Silva JA da, Almada RC, Falconi-Sobrinho LL, Pigatto GR, Hernandes PM, Coimbra NC. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections [Internet]. Brain Research Bulletin. 2023 ; 192 128-141.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.11.014
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Fonte: Inflammation Research. Unidade: FMRP
Assuntos: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 13 jul. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats (2023)
Cavichioli, Andreza M.; Silva, Thamyris Santos; Grace, Anthony A.; Guimarães, Francisco Silveira ; Gomes, Felipe Villela
Fonte: Schizophrenia Bulletin. Unidade: FMRP
Assuntos: POTENCIAIS DE AÇÃO, NEUROTRANSMISSORES, ESQUIZOFRENIA, HIPOCAMPO
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ABNT
CAVICHIOLI, Andreza M. et al. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats. Schizophrenia Bulletin, v. 49, n. 1, p. 68-77, 2023Tradução . . Disponível em: https://doi.org/10.1093/schbul/sbac106. Acesso em: 13 jul. 2024.
APA
Cavichioli, A. M., Silva, T. S., Grace, A. A., Guimarães, F. S., & Gomes, F. V. (2023). Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats. Schizophrenia Bulletin, 49( 1), 68-77. doi:10.1093/schbul/sbac106
NLM
Cavichioli AM, Silva TS, Grace AA, Guimarães FS, Gomes FV. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats [Internet]. Schizophrenia Bulletin. 2023 ; 49( 1): 68-77.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/schbul/sbac106
Vancouver
Cavichioli AM, Silva TS, Grace AA, Guimarães FS, Gomes FV. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats [Internet]. Schizophrenia Bulletin. 2023 ; 49( 1): 68-77.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/schbul/sbac106
Artigo de periodico
Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 (2023)
Dourado, Thales de Mileto Henrique; Assis, Victor Oliveira; Awata, Wanessa Mayumi Carvalho; Mello, Marcela Maria Blascke de ; Cárnio, Evelin Capellari ; Castro, Michele Mazzaron de ; Tirapelli, Carlos Renato
Fonte: European Journal of Pharmacology. Unidades: EERP, FMRP
Assuntos: ETANOL, ESPÉCIES REATIVAS DE OXIGÊNIO, TROMBOXANOS, HIPERTENSÃO, ENDOTÉLIO VASCULAR
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ABNT
DOURADO, Thales de Mileto Henrique et al. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2. European Journal of Pharmacology, v. 949, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2023.175723. Acesso em: 13 jul. 2024.
APA
Dourado, T. de M. H., Assis, V. O., Awata, W. M. C., Mello, M. M. B. de, Cárnio, E. C., Castro, M. M. de, & Tirapelli, C. R. (2023). Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2. European Journal of Pharmacology, 949, 1-10. doi:10.1016/j.ejphar.2023.175723
NLM
Dourado T de MH, Assis VO, Awata WMC, Mello MMB de, Cárnio EC, Castro MM de, Tirapelli CR. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 [Internet]. European Journal of Pharmacology. 2023 ; 949 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2023.175723
Vancouver
Dourado T de MH, Assis VO, Awata WMC, Mello MMB de, Cárnio EC, Castro MM de, Tirapelli CR. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 [Internet]. European Journal of Pharmacology. 2023 ; 949 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2023.175723
Artigo de periodico
The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism (2023)
Britto Júnior, José ; Prado, Gustavo L. Pereira do ; Cunha, Fernando ; Moraes, Manoel Odorico ; Moraes, Maria Elisabete A. ; Monica, Fabiola Z. ; Antunes, Edson ; Chiavegatto, Silvana ; De Nucci, Gilberto
Fonte: Nitric Oxide: Biology and Chemistry. Unidades: ICB, FMRP
Assuntos: FARMACOLOGIA, ÁTRIOS DO CORAÇÃO, VENTRÍCULO CARDÍACO DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, ÓXIDO NÍTRICO, ESPECTROMETRIA DE MASSAS
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ABNT
BRITTO JÚNIOR, José et al. The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism. Nitric Oxide: Biology and Chemistry, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2023.06.001. Acesso em: 13 jul. 2024.
APA
Britto Júnior, J., Prado, G. L. P. do, Cunha, F., Moraes, M. O., Moraes, M. E. A., Monica, F. Z., et al. (2023). The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism. Nitric Oxide: Biology and Chemistry. doi:10.1016/j.niox.2023.06.001
NLM
Britto Júnior J, Prado GLP do, Cunha F, Moraes MO, Moraes MEA, Monica FZ, Antunes E, Chiavegatto S, De Nucci G. The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism [Internet]. Nitric Oxide: Biology and Chemistry. 2023 ;[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.niox.2023.06.001
Vancouver
Britto Júnior J, Prado GLP do, Cunha F, Moraes MO, Moraes MEA, Monica FZ, Antunes E, Chiavegatto S, De Nucci G. The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism [Internet]. Nitric Oxide: Biology and Chemistry. 2023 ;[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.niox.2023.06.001
Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2023)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Assuntos: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
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ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 13 jul. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2023). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:https://doi.org/10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections (2023)
Santiago-Carvalho, Igor ; Almeida-Santos, Gislane ; Macêdo, Bruna de Gois ; Barbosa-Bomfim, Caio César ; Almeida, Fabricio Moreira ; Cione, Marcos Vinícios Pinheiro ; Vardam-Kaur, Trupti ; Masuda, Mia ; Dijk, Sarah Van ; Melo, Bruno Marcel Silva de ; Nascimento, Rogério Silva do; Souza, Rebeka da Conceição ; Peixoto-Rangel, Alba Lucínia ; Coutinho-Silva, Robson ; Lassounskaia, Elena ; Silva, Henrique Borges da ; Hirata, Mario Hiroyuki ; Alves Filho, José Carlos ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Fonte: Cell Reports. Unidades: FCF, FMRP, ICB
Assuntos: IMUNOLOGIA, PNEUMOPATIAS, TUBERCULOSE PULMONAR, INFLUENZA, MODELOS ANIMAIS DE DOENÇAS, RATOS, QUIMIOCINAS, PULMÃO
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ABNT
SANTIAGO-CARVALHO, Igor et al. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Cell Reports, v. 42, n. 11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2023.113448. Acesso em: 13 jul. 2024.
APA
Santiago-Carvalho, I., Almeida-Santos, G., Macêdo, B. de G., Barbosa-Bomfim, C. C., Almeida, F. M., Cione, M. V. P., et al. (2023). T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Cell Reports, 42( 11). doi:10.1016/j.celrep.2023.113448
NLM
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Vardam-Kaur T, Masuda M, Dijk SV, Melo BMS de, Nascimento RS do, Souza R da C, Peixoto-Rangel AL, Coutinho-Silva R, Lassounskaia E, Silva HB da, Hirata MH, Alves Filho JC, Alvarez JM, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Cell Reports. 2023 ; 42( 11):[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.celrep.2023.113448
Vancouver
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Vardam-Kaur T, Masuda M, Dijk SV, Melo BMS de, Nascimento RS do, Souza R da C, Peixoto-Rangel AL, Coutinho-Silva R, Lassounskaia E, Silva HB da, Hirata MH, Alves Filho JC, Alvarez JM, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Cell Reports. 2023 ; 42( 11):[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.celrep.2023.113448
Artigo de periodico
Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection (2023)
Moreira, Thaiane Pinto; Sousa, Carla Daiane Ferreira de; Costa, Victor Rodrigues de Melo; Queiroz Junior, Celso Martins; Santos, Franciele Martins; Bonilha, Caio Santos ; Ésper, Lísia Maria; Nogueira, Mauricio Lacerda ; Cunha, Thiago Mattar ; Teixeira, Mauro Martins ; Costa, Vivian Vasconcelos; Souza, Daniele da Glória de
Fonte: Immunology. Unidade: FMRP
Assuntos: ARTRALGIA, VÍRUS CHIKUNGUNYA, NECROSE, REPLICAÇÃO VIRAL
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ABNT
MOREIRA, Thaiane Pinto et al. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, v. 168, n. 3, p. 444-458, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13583. Acesso em: 13 jul. 2024.
APA
Moreira, T. P., Sousa, C. D. F. de, Costa, V. R. de M., Queiroz Junior, C. M., Santos, F. M., Bonilha, C. S., et al. (2023). Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, 168( 3), 444-458. doi:10.1111/imm.13583
NLM
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/imm.13583
Vancouver
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1111/imm.13583
Artigo de periodico
The rs2682826 polymorphism of the NOS1 gene is associated with the degree of disability of erectile dysfunction (2023)
Ferezin, Letícia Perticarrara; Cotta Filho, Cezar Kayzuka ; Pereira, Vitória Carolina Rondon; Andrade, Murilo Ferreira de; Molina, Carlos Augusto Fernandes ; Tucci Júnior, Silvio ; Tanus-Santos, José Eduardo ; Lacchini, Riccardo
Fonte: Life. Unidades: FMRP, EERP
Assuntos: DISFUNÇÃO ERÉTIL, POLIMORFISMO, ÓXIDO NÍTRICO
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ABNT
FEREZIN, Letícia Perticarrara et al. The rs2682826 polymorphism of the NOS1 gene is associated with the degree of disability of erectile dysfunction. Life, v. 13, n. 5, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.3390/life13051082. Acesso em: 13 jul. 2024.
APA
Ferezin, L. P., Cotta Filho, C. K., Pereira, V. C. R., Andrade, M. F. de, Molina, C. A. F., Tucci Júnior, S., et al. (2023). The rs2682826 polymorphism of the NOS1 gene is associated with the degree of disability of erectile dysfunction. Life, 13( 5), 1-13. doi:10.3390/life13051082
NLM
Ferezin LP, Cotta Filho CK, Pereira VCR, Andrade MF de, Molina CAF, Tucci Júnior S, Tanus-Santos JE, Lacchini R. The rs2682826 polymorphism of the NOS1 gene is associated with the degree of disability of erectile dysfunction [Internet]. Life. 2023 ; 13( 5): 1-13.[citado 2024 jul. 13 ] Available from: https://doi.org/10.3390/life13051082
Vancouver
Ferezin LP, Cotta Filho CK, Pereira VCR, Andrade MF de, Molina CAF, Tucci Júnior S, Tanus-Santos JE, Lacchini R. The rs2682826 polymorphism of the NOS1 gene is associated with the degree of disability of erectile dysfunction [Internet]. Life. 2023 ; 13( 5): 1-13.[citado 2024 jul. 13 ] Available from: https://doi.org/10.3390/life13051082
Artigo de periodico
Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures (2022)
Crespo, Luiz Gustavo Soares Carvalho; Leite Júnior, Joaquim Barbosa; Bastos, João Marcos de Mello; Samuels, Richard Ian; Coimbra, Norberto Cysne ; Carey, Robert J.; Carrera, Marinete Pinheiro
Fonte: Pharmacology Biochemistry and Behavior. Unidade: FMRP
Assuntos: CÉREBRO, CONDICIONAMENTO OPERANTE, MORFINA, ESTIMULAÇÃO CEREBRAL
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ABNT
CRESPO, Luiz Gustavo Soares Carvalho et al. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures. Pharmacology Biochemistry and Behavior, v. 214, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2022.173356. Acesso em: 13 jul. 2024.
APA
Crespo, L. G. S. C., Leite Júnior, J. B., Bastos, J. M. de M., Samuels, R. I., Coimbra, N. C., Carey, R. J., & Carrera, M. P. (2022). Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures. Pharmacology Biochemistry and Behavior, 214, 1-10. doi:10.1016/j.pbb.2022.173356
NLM
Crespo LGSC, Leite Júnior JB, Bastos JM de M, Samuels RI, Coimbra NC, Carey RJ, Carrera MP. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures [Internet]. Pharmacology Biochemistry and Behavior. 2022 ; 214 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.pbb.2022.173356
Vancouver
Crespo LGSC, Leite Júnior JB, Bastos JM de M, Samuels RI, Coimbra NC, Carey RJ, Carrera MP. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures [Internet]. Pharmacology Biochemistry and Behavior. 2022 ; 214 1-10.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.pbb.2022.173356
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Fonte: The Journal of Infectious Diseases. Unidade: FMRP
Assuntos: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 13 jul. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1093/infdis/jiab405

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