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  • Source: Journal of Chemical Neuroanatomy. Unidades: HU, ICB

    Subjects: COMPORTAMENTO PSICOSSEXUAL, HIPOTÁLAMO, ESTEROIDES, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, ESTRÓGENOS, RATOS, TRONCO DE ANIMAL, ESTRADIOL

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      PACHECO, Silvana da Silva et al. Neurons expressing estrogen receptor α differentially innervate the periaqueductal gray matter of female rats. Journal of Chemical Neuroanatomy, v. 97, p. 33-42, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.jchemneu.2019.01.004. Acesso em: 02 ago. 2024.
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      Pacheco, S. da S., Rondini, T. A., Bittencourt, J. C., & Elias, C. F. (2019). Neurons expressing estrogen receptor α differentially innervate the periaqueductal gray matter of female rats. Journal of Chemical Neuroanatomy, 97, 33-42. doi:10.1016/j.jchemneu.2019.01.004
    • NLM

      Pacheco S da S, Rondini TA, Bittencourt JC, Elias CF. Neurons expressing estrogen receptor α differentially innervate the periaqueductal gray matter of female rats [Internet]. Journal of Chemical Neuroanatomy. 2019 ; 97 33-42.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1016/j.jchemneu.2019.01.004
    • Vancouver

      Pacheco S da S, Rondini TA, Bittencourt JC, Elias CF. Neurons expressing estrogen receptor α differentially innervate the periaqueductal gray matter of female rats [Internet]. Journal of Chemical Neuroanatomy. 2019 ; 97 33-42.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1016/j.jchemneu.2019.01.004
  • Source: Autophagy. Unidades: EEFE, FCFRP, FMRP, ICB

    Subjects: CÉLULAS, BIOLOGIA CELULAR

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      KLIONSKY, D. J et al. Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy. Philadelphia: Escola de Educação Física e Esporte, Universidade de São Paulo. Disponível em: https://doi.org/10.1080/15548627.2015.1100356. Acesso em: 02 ago. 2024. , 2016
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      Klionsky, D. J., Brum, P. C., Goldman, G. H., Kettelhut, I. D. C., Navegantes, L. C. C., & Santelli, G. M. M. (2016). Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy. Philadelphia: Escola de Educação Física e Esporte, Universidade de São Paulo. doi:10.1080/15548627.2015.1100356
    • NLM

      Klionsky DJ, Brum PC, Goldman GH, Kettelhut IDC, Navegantes LCC, Santelli GMM. Guidelines for the use and interpretation of assays for monitoring autophagy [Internet]. Autophagy. 2016 ; 12( ja 2016): 1-222.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1080/15548627.2015.1100356
    • Vancouver

      Klionsky DJ, Brum PC, Goldman GH, Kettelhut IDC, Navegantes LCC, Santelli GMM. Guidelines for the use and interpretation of assays for monitoring autophagy [Internet]. Autophagy. 2016 ; 12( ja 2016): 1-222.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1080/15548627.2015.1100356
  • Source: Nucleic Acids Research. Unidade: ICB

    Subjects: MICROBIOLOGIA, DNA, RNA, GENES, RNA POLIMERASES

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      ANDRADE-LIMA, Leonardo Carmo de et al. DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes. Nucleic Acids Research, v. 43, n. 5, p. 2744-2756, 2015Tradução . . Disponível em: https://doi.org/10.1093/nar/gkv148. Acesso em: 02 ago. 2024.
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      Andrade-Lima, L. C. de, Veloso, A., Paulsen, M. T., Menck, C. F. M., & Ljungman, M. (2015). DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes. Nucleic Acids Research, 43( 5), 2744-2756. doi:10.1093/nar/gkv148
    • NLM

      Andrade-Lima LC de, Veloso A, Paulsen MT, Menck CFM, Ljungman M. DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes [Internet]. Nucleic Acids Research. 2015 ; 43( 5): 2744-2756.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1093/nar/gkv148
    • Vancouver

      Andrade-Lima LC de, Veloso A, Paulsen MT, Menck CFM, Ljungman M. DNA repair and recovery of RNA synthesis following exposure to ultraviolet light are delayed in long genes [Internet]. Nucleic Acids Research. 2015 ; 43( 5): 2744-2756.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1093/nar/gkv148
  • Source: American Journal of Tropical Medicine and Hygiene. Unidade: ICB

    Subjects: PARASITOLOGIA, MALARIA

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      WILSON, Mark L. et al. Urban malaria: understanding its epidemiology, ecology, and transmission across seven diverse ICEMR network sites. American Journal of Tropical Medicine and Hygiene, v. 93, p. 110-123, 2015Tradução . . Disponível em: https://doi.org/10.4269/ajtmh.14-0834. Acesso em: 02 ago. 2024.
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      Wilson, M. L., Krogstad, D. J., Arinaitwe, E., Arevalo-Herrera, M., Chery, L., Ferreira, M. U., et al. (2015). Urban malaria: understanding its epidemiology, ecology, and transmission across seven diverse ICEMR network sites. American Journal of Tropical Medicine and Hygiene, 93, 110-123. doi:10.4269/ajtmh.14-0834
    • NLM

      Wilson ML, Krogstad DJ, Arinaitwe E, Arevalo-Herrera M, Chery L, Ferreira MU, Ndiaye D, Mathanga DP, Eapen A. Urban malaria: understanding its epidemiology, ecology, and transmission across seven diverse ICEMR network sites [Internet]. American Journal of Tropical Medicine and Hygiene. 2015 ; 93 110-123.[citado 2024 ago. 02 ] Available from: https://doi.org/10.4269/ajtmh.14-0834
    • Vancouver

      Wilson ML, Krogstad DJ, Arinaitwe E, Arevalo-Herrera M, Chery L, Ferreira MU, Ndiaye D, Mathanga DP, Eapen A. Urban malaria: understanding its epidemiology, ecology, and transmission across seven diverse ICEMR network sites [Internet]. American Journal of Tropical Medicine and Hygiene. 2015 ; 93 110-123.[citado 2024 ago. 02 ] Available from: https://doi.org/10.4269/ajtmh.14-0834
  • Source: American Journal of Physiology. Endocrinology and Metabolism. Unidade: ICB

    Subjects: ANATOMIA, ESTRADIOL, HORMONIOS SEXUAIS FEMININOS

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      FRAZÃO, Renata et al. Estradiol modulates Kiss1 neuronal response to ghrelin. American Journal of Physiology. Endocrinology and Metabolism, v. 306, n. 6, p. E606-E614, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00211.2013. Acesso em: 02 ago. 2024.
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      Frazão, R., Lemko, H. M. D., Silva, R. P. da, Ratra, D. V., Lee, C. E., Williams, K. W., et al. (2014). Estradiol modulates Kiss1 neuronal response to ghrelin. American Journal of Physiology. Endocrinology and Metabolism, 306( 6), E606-E614. doi:10.1152/ajpendo.00211.2013
    • NLM

      Frazão R, Lemko HMD, Silva RP da, Ratra DV, Lee CE, Williams KW, Zigman JM, Elias CF. Estradiol modulates Kiss1 neuronal response to ghrelin [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 306( 6): E606-E614.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1152/ajpendo.00211.2013
    • Vancouver

      Frazão R, Lemko HMD, Silva RP da, Ratra DV, Lee CE, Williams KW, Zigman JM, Elias CF. Estradiol modulates Kiss1 neuronal response to ghrelin [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 306( 6): E606-E614.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1152/ajpendo.00211.2013
  • Source: Molecular Metabolism. Unidades: FCF, ICB

    Subjects: DIABETES MELLITUS NÃO INSULINO-DEPENDENTE, LEPTINA, HIPOTÁLAMO

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      PEDROSO, João Alfredo Bolivar et al. Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª. Molecular Metabolism, v. 3, n. 6, p. 608-618, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2014.06.001. Acesso em: 02 ago. 2024.
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      Pedroso, J. A. B., Buonfiglio, D. do C., Cardinali, L. I., Furigo, I. C., Ramos-Lobo, A. M., Tirapegui, J., et al. (2014). Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª. Molecular Metabolism, 3( 6), 608-618. doi:10.1016/j.molmet.2014.06.001
    • NLM

      Pedroso JAB, Buonfiglio D do C, Cardinali LI, Furigo IC, Ramos-Lobo AM, Tirapegui J, Elias CF, Donato Junior J. Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª [Internet]. Molecular Metabolism. 2014 ; 3( 6): 608-618.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1016/j.molmet.2014.06.001
    • Vancouver

      Pedroso JAB, Buonfiglio D do C, Cardinali LI, Furigo IC, Ramos-Lobo AM, Tirapegui J, Elias CF, Donato Junior J. Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª [Internet]. Molecular Metabolism. 2014 ; 3( 6): 608-618.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1016/j.molmet.2014.06.001
  • Source: Journal of Immunology. Unidade: ICB

    Subjects: IMUNOLOGIA, LEUCOTRIENOS B, MACRÓFAGOS

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      WANG, Zhuo et al. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, v. 192, n. 5, p. 2349-2356, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1302982. Acesso em: 02 ago. 2024.
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      Wang, Z., Filgueiras, L. R., Wang, S., Serezani, A. P. M., Peters-Golden, M., Jancar, S., & Serezani, C. H. (2014). Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, 192( 5), 2349-2356. doi:10.4049/jimmunol.1302982
    • NLM

      Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2024 ago. 02 ] Available from: https://doi.org/10.4049/jimmunol.1302982
    • Vancouver

      Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2024 ago. 02 ] Available from: https://doi.org/10.4049/jimmunol.1302982
  • Source: PLOS ONE. Unidades: ICB, FSP

    Assunto: PARASITOLOGIA

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      CARDOSO, Marly Augusto et al. Underlying factors associated with anemia in Amazonian children: a population-based, cross-sectional study. PLOS ONE, v. 7, n. 5, p. 1-8, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0036341. Acesso em: 02 ago. 2024.
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      Cardoso, M. A., Scopel, K. K. G., Muniz, P. T., Villamor, E., & Ferreira, M. U. (2012). Underlying factors associated with anemia in Amazonian children: a population-based, cross-sectional study. PLOS ONE, 7( 5), 1-8. doi:10.1371/journal.pone.0036341
    • NLM

      Cardoso MA, Scopel KKG, Muniz PT, Villamor E, Ferreira MU. Underlying factors associated with anemia in Amazonian children: a population-based, cross-sectional study [Internet]. PLOS ONE. 2012 ; 7( 5): 1-8.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1371/journal.pone.0036341
    • Vancouver

      Cardoso MA, Scopel KKG, Muniz PT, Villamor E, Ferreira MU. Underlying factors associated with anemia in Amazonian children: a population-based, cross-sectional study [Internet]. PLOS ONE. 2012 ; 7( 5): 1-8.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1371/journal.pone.0036341
  • Source: Abstracts. Conference titles: World Congress on Inflammation. Unidades: FCF, ICB

    Subjects: DIABETES MELLITUS, INFLAMAÇÃO

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      RIBEIRO JUNIOR, Luciano Filgueiras et al. Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. 2011, Anais.. Paris: International Association of Inflammation Societies, 2011. . Acesso em: 02 ago. 2024.
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      Ribeiro Junior, L. F., Martins, J. de O., Serezani, C. H., Montes, M. B. A., & Jancar, S. (2011). Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. In Abstracts. Paris: International Association of Inflammation Societies.
    • NLM

      Ribeiro Junior LF, Martins J de O, Serezani CH, Montes MBA, Jancar S. Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. Abstracts. 2011 ;[citado 2024 ago. 02 ]
    • Vancouver

      Ribeiro Junior LF, Martins J de O, Serezani CH, Montes MBA, Jancar S. Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. Abstracts. 2011 ;[citado 2024 ago. 02 ]
  • Source: The Journal Of Biological Chemistry. Unidade: ICB

    Assunto: IMUNOLOGIA

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      MORATO-MARQUES, Mariana et al. Leukotrienes target F-actin/cofilin-1 to enhance alveolar macrophage anti-fungal activity. The Journal Of Biological Chemistry, v. 286, n. 33, p. 28902-28913, 2011Tradução . . Disponível em: https://doi.org/10.1074/jbc.M111.235309. Acesso em: 02 ago. 2024.
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      Morato-Marques, M., Campos, M. R., Kane, S., Rangel, A. P., Lewis, C., Ballinger, M. N., et al. (2011). Leukotrienes target F-actin/cofilin-1 to enhance alveolar macrophage anti-fungal activity. The Journal Of Biological Chemistry, 286( 33), 28902-28913. doi:10.1074/jbc.M111.235309
    • NLM

      Morato-Marques M, Campos MR, Kane S, Rangel AP, Lewis C, Ballinger MN, Kim S-H, Peters-Golden M, Jancar S, Serezani CH. Leukotrienes target F-actin/cofilin-1 to enhance alveolar macrophage anti-fungal activity [Internet]. The Journal Of Biological Chemistry. 2011 ; 286( 33): 28902-28913.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1074/jbc.M111.235309
    • Vancouver

      Morato-Marques M, Campos MR, Kane S, Rangel AP, Lewis C, Ballinger MN, Kim S-H, Peters-Golden M, Jancar S, Serezani CH. Leukotrienes target F-actin/cofilin-1 to enhance alveolar macrophage anti-fungal activity [Internet]. The Journal Of Biological Chemistry. 2011 ; 286( 33): 28902-28913.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1074/jbc.M111.235309
  • Source: The Journal of Clinical Investigation. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SEREZANI, Carlos H. et al. Leukotriene B4 amplifies NF-κB activation in mouse macrophages by reducing SOCS1 inhibition of MyD88 expression. The Journal of Clinical Investigation, v. 121, n. 2, p. 671-682, 2011Tradução . . Disponível em: https://doi.org/10.1172/JCI43302. Acesso em: 02 ago. 2024.
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      Serezani, C. H., Lewis, C., Jancar, S., & Peters-Golden, M. (2011). Leukotriene B4 amplifies NF-κB activation in mouse macrophages by reducing SOCS1 inhibition of MyD88 expression. The Journal of Clinical Investigation, 121( 2), 671-682. doi:10.1172/JCI43302
    • NLM

      Serezani CH, Lewis C, Jancar S, Peters-Golden M. Leukotriene B4 amplifies NF-κB activation in mouse macrophages by reducing SOCS1 inhibition of MyD88 expression [Internet]. The Journal of Clinical Investigation. 2011 ; 121( 2): 671-682.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1172/JCI43302
    • Vancouver

      Serezani CH, Lewis C, Jancar S, Peters-Golden M. Leukotriene B4 amplifies NF-κB activation in mouse macrophages by reducing SOCS1 inhibition of MyD88 expression [Internet]. The Journal of Clinical Investigation. 2011 ; 121( 2): 671-682.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1172/JCI43302
  • Source: American Journal of Physiology. Lung Cellular and Molecular Physiology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      STUMM, Camila Leindecker et al. Airway remodeling in murine asthma correlates with a defect in PGE2 synthesis by lung fibroblastos. American Journal of Physiology. Lung Cellular and Molecular Physiology, v. 301, n. 5, p. L636-L644, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajplung.00158.2011. Acesso em: 02 ago. 2024.
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      Stumm, C. L., Wettlaufer, S. H., Jancar, S., & Peters-Golden, M. (2011). Airway remodeling in murine asthma correlates with a defect in PGE2 synthesis by lung fibroblastos. American Journal of Physiology. Lung Cellular and Molecular Physiology, 301( 5), L636-L644. doi:10.1152/ajplung.00158.2011
    • NLM

      Stumm CL, Wettlaufer SH, Jancar S, Peters-Golden M. Airway remodeling in murine asthma correlates with a defect in PGE2 synthesis by lung fibroblastos [Internet]. American Journal of Physiology. Lung Cellular and Molecular Physiology. 2011 ; 301( 5): L636-L644.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1152/ajplung.00158.2011
    • Vancouver

      Stumm CL, Wettlaufer SH, Jancar S, Peters-Golden M. Airway remodeling in murine asthma correlates with a defect in PGE2 synthesis by lung fibroblastos [Internet]. American Journal of Physiology. Lung Cellular and Molecular Physiology. 2011 ; 301( 5): L636-L644.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1152/ajplung.00158.2011
  • Source: Journal of Sexual Medicine. Unidade: ICB

    Assunto: FARMACOLOGIA

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      PASSAGLIA, Rita de Cássia Aleixo Tostes et al. Cigarette smoking and erectile dysfunction: focus on NO bioavailability and ROS generation. Journal of Sexual Medicine, v. 5, n. 6, p. 1284-1295, 2008Tradução . . Acesso em: 02 ago. 2024.
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      Passaglia, R. de C. A. T., Carneiro, F. S., Lee, A. J., Giachini, F. R. C., Leite, R., Osawa, Y., & Webb, R. C. (2008). Cigarette smoking and erectile dysfunction: focus on NO bioavailability and ROS generation. Journal of Sexual Medicine, 5( 6), 1284-1295.
    • NLM

      Passaglia R de CAT, Carneiro FS, Lee AJ, Giachini FRC, Leite R, Osawa Y, Webb RC. Cigarette smoking and erectile dysfunction: focus on NO bioavailability and ROS generation. Journal of Sexual Medicine. 2008 ; 5( 6): 1284-1295.[citado 2024 ago. 02 ]
    • Vancouver

      Passaglia R de CAT, Carneiro FS, Lee AJ, Giachini FRC, Leite R, Osawa Y, Webb RC. Cigarette smoking and erectile dysfunction: focus on NO bioavailability and ROS generation. Journal of Sexual Medicine. 2008 ; 5( 6): 1284-1295.[citado 2024 ago. 02 ]
  • Source: Cell Death and Differentiation. Unidade: ICB

    Assunto: IMUNOLOGIA

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      WEINLICH, Ricardo et al. TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells. Cell Death and Differentiation, v. 15, n. 12, p. 1901-1909, 2008Tradução . . Disponível em: https://doi.org/10.1038/cdd.2008.128. Acesso em: 02 ago. 2024.
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      Weinlich, R., Bortoluci, K. R., Chehab, C. F., Serezani, C. H., Ulbrich, A. G., Peters-Golden, M., et al. (2008). TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells. Cell Death and Differentiation, 15( 12), 1901-1909. doi:10.1038/cdd.2008.128
    • NLM

      Weinlich R, Bortoluci KR, Chehab CF, Serezani CH, Ulbrich AG, Peters-Golden M, Russo M, Amarante-Mendes JGP. TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells [Internet]. Cell Death and Differentiation. 2008 ; 15( 12): 1901-1909.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1038/cdd.2008.128
    • Vancouver

      Weinlich R, Bortoluci KR, Chehab CF, Serezani CH, Ulbrich AG, Peters-Golden M, Russo M, Amarante-Mendes JGP. TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells [Internet]. Cell Death and Differentiation. 2008 ; 15( 12): 1901-1909.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1038/cdd.2008.128
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SEREZANI, Carlos H. et al. Leukotrienes are essential for the control of Leishmania amazonensis infection and contribute to strain variation in susceptibility 1. Journal of Immunology, v. 177, p. 1126-1132, 2006Tradução . . Acesso em: 02 ago. 2024.
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      Serezani, C. H., Perrela, J. H., Russo, M., Peters-Golden, M., & Jancar, S. (2006). Leukotrienes are essential for the control of Leishmania amazonensis infection and contribute to strain variation in susceptibility 1. Journal of Immunology, 177, 1126-1132.
    • NLM

      Serezani CH, Perrela JH, Russo M, Peters-Golden M, Jancar S. Leukotrienes are essential for the control of Leishmania amazonensis infection and contribute to strain variation in susceptibility 1. Journal of Immunology. 2006 ; 177 1126-1132.[citado 2024 ago. 02 ]
    • Vancouver

      Serezani CH, Perrela JH, Russo M, Peters-Golden M, Jancar S. Leukotrienes are essential for the control of Leishmania amazonensis infection and contribute to strain variation in susceptibility 1. Journal of Immunology. 2006 ; 177 1126-1132.[citado 2024 ago. 02 ]
  • Source: Journal of Leukocyte Biology. Unidade: ICB

    Subjects: IMUNOLOGIA, LEUCOTRIENOS B, NEUTRÓFILOS, ÁCIDOS GRAXOS

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      SEREZANI, Carlos Henrique Cardoso et al. Leukotriene B4 mediates p47phox phosphorylation and membrane translocation in polyunsaturated fatty acidstimulated neutrophils. Journal of Leukocyte Biology, v. 78, p. 976-984, 2005Tradução . . Disponível em: https://doi.org/10.1189/jlb.1004587. Acesso em: 02 ago. 2024.
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      Serezani, C. H. C., Aronoff, D. M., Jancar, S., & Peters-Golden, M. (2005). Leukotriene B4 mediates p47phox phosphorylation and membrane translocation in polyunsaturated fatty acidstimulated neutrophils. Journal of Leukocyte Biology, 78, 976-984. doi:10.1189/jlb.1004587
    • NLM

      Serezani CHC, Aronoff DM, Jancar S, Peters-Golden M. Leukotriene B4 mediates p47phox phosphorylation and membrane translocation in polyunsaturated fatty acidstimulated neutrophils [Internet]. Journal of Leukocyte Biology. 2005 ; 78 976-984.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1189/jlb.1004587
    • Vancouver

      Serezani CHC, Aronoff DM, Jancar S, Peters-Golden M. Leukotriene B4 mediates p47phox phosphorylation and membrane translocation in polyunsaturated fatty acidstimulated neutrophils [Internet]. Journal of Leukocyte Biology. 2005 ; 78 976-984.[citado 2024 ago. 02 ] Available from: https://doi.org/10.1189/jlb.1004587
  • Source: Inflammation. Unidade: ICB

    Assunto: FARMACOLOGIA

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      PICCOLO, Maria Thereza S. et al. Role of chemotactic factors in neutrophil activation after thermal injury in rats. Inflammation, v. 23, n. 4, p. 371-385, 1999Tradução . . Acesso em: 02 ago. 2024.
    • APA

      Piccolo, M. T. S., Wang, Y., Verbrugge, S., Warner, R. L., Sannomiya, P., Piccolo, N. S., et al. (1999). Role of chemotactic factors in neutrophil activation after thermal injury in rats. Inflammation, 23( 4), 371-385.
    • NLM

      Piccolo MTS, Wang Y, Verbrugge S, Warner RL, Sannomiya P, Piccolo NS, Piccolo MS, Hugli TE, Ward PA, Till GO. Role of chemotactic factors in neutrophil activation after thermal injury in rats. Inflammation. 1999 ; 23( 4): 371-385.[citado 2024 ago. 02 ]
    • Vancouver

      Piccolo MTS, Wang Y, Verbrugge S, Warner RL, Sannomiya P, Piccolo NS, Piccolo MS, Hugli TE, Ward PA, Till GO. Role of chemotactic factors in neutrophil activation after thermal injury in rats. Inflammation. 1999 ; 23( 4): 371-385.[citado 2024 ago. 02 ]
  • Source: Experimental and Molecular Pathology. Unidade: ICB

    Subjects: FARMACOLOGIA, FARMACOCINÉTICA

    How to cite
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    • ABNT

      PICCOLO, Maria-Thereza S. et al. Chemotatic mediator requirements in lung injury following skin burns in rats. Experimental and Molecular Pathology, v. 66, p. 220-226, 1999Tradução . . Acesso em: 02 ago. 2024.
    • APA

      Piccolo, M. -T. S., Wang, Y., Piccolo, N. S., Piccolo, M. S., Hugli, T. E., Ward, P. A., et al. (1999). Chemotatic mediator requirements in lung injury following skin burns in rats. Experimental and Molecular Pathology, 66, 220-226.
    • NLM

      Piccolo M-TS, Wang Y, Piccolo NS, Piccolo MS, Hugli TE, Ward PA, Till GO, Sannomiya P. Chemotatic mediator requirements in lung injury following skin burns in rats. Experimental and Molecular Pathology. 1999 ; 66 220-226.[citado 2024 ago. 02 ]
    • Vancouver

      Piccolo M-TS, Wang Y, Piccolo NS, Piccolo MS, Hugli TE, Ward PA, Till GO, Sannomiya P. Chemotatic mediator requirements in lung injury following skin burns in rats. Experimental and Molecular Pathology. 1999 ; 66 220-226.[citado 2024 ago. 02 ]

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