Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª (2014)
- Authors:
- USP affiliated authors: TOLEDO, JULIO ORLANDO TIRAPEGUI - FCF ; DONATO JÚNIOR, JOSÉ - ICB
- Unidades: FCF; ICB
- DOI: 10.1016/j.molmet.2014.06.001
- Subjects: DIABETES MELLITUS NÃO INSULINO-DEPENDENTE; LEPTINA; HIPOTÁLAMO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Molecular Metabolism
- ISSN: 2212-8778
- Volume/Número/Paginação/Ano: v. 3, n. 6, p. 608-618, 2014
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc-nd
-
ABNT
PEDROSO, João Alfredo Bolivar et al. Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª. Molecular Metabolism, v. 3, n. 6, p. 608-618, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2014.06.001. Acesso em: 18 abr. 2024. -
APA
Pedroso, J. A. B., Buonfiglio, D. do C., Cardinali, L. I., Furigo, I. C., Ramos-Lobo, A. M., Tirapegui, J., et al. (2014). Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª. Molecular Metabolism, 3( 6), 608-618. doi:10.1016/j.molmet.2014.06.001 -
NLM
Pedroso JAB, Buonfiglio D do C, Cardinali LI, Furigo IC, Ramos-Lobo AM, Tirapegui J, Elias CF, Donato Junior J. Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª [Internet]. Molecular Metabolism. 2014 ; 3( 6): 608-618.[citado 2024 abr. 18 ] Available from: https://doi.org/10.1016/j.molmet.2014.06.001 -
Vancouver
Pedroso JAB, Buonfiglio D do C, Cardinali LI, Furigo IC, Ramos-Lobo AM, Tirapegui J, Elias CF, Donato Junior J. Inactivation of SOCS3 in leptin receptor-expressing cells protects mice from diet-induced insulin resistance but does not prevent obesityª [Internet]. Molecular Metabolism. 2014 ; 3( 6): 608-618.[citado 2024 abr. 18 ] Available from: https://doi.org/10.1016/j.molmet.2014.06.001 - Oral supplementations with free and dipeptide forms of L-glutamine in endotoxemic mice: effects on muscle glutamine-glutathione axis and heat shock proteins
- Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus
- Long-term leucine supplementation reduces fat mass gain without changing body protein status of aging rats
- Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus
- Leucine improves protein nutritional status and regulates hepatic lipid metabolism in calorie restricted rats
- Leucine is essential for attenuating fetal growth restriction caused by a protein-restricted diet in rats
- Mecanismos da regulação do peso corporal e a influência da atividade física
- Nutrição, exercício e envelhecimento
- Leucine supplementation increases serum insulin-like growth factor 1 concentration and liver protein/RNA ratio in rats after a period of nutritional recovery
- Obesidade
Informações sobre o DOI: 10.1016/j.molmet.2014.06.001 (Fonte: oaDOI API)
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