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Artigo de periodico
H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
Costa, Rafael M.; Filgueira, Fernando P.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena C.; Akamine, Eliana Hiromi ; Lobato, Nubia S.
Fonte: Vascular Pharmacology. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 17 nov. 2024.
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Fonte: Plos One. Unidades: ICB, FMRP
Assuntos: ANATOMIA, FARMACOLOGIA
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ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. Plos One, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 17 nov. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. Plos One, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. Plos One. 2014 ; 9( 11):[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. Plos One. 2014 ; 9( 11):[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats (2013)
Ceravolo, Graziela Scalianti; Filgueira, Fernando Paranaiba; Costa, Tiago Januário da; Lobato, Núbia de Souza; Chignalia, Andréia Zago; Araujo, Priscila Xavier de; Tostes, Rita de Cassia; Dantas, Ana P.; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli de
Fonte: Steroids. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
CERAVOLO, Graziela Scalianti et al. Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats. Steroids, v. 78, n. 3, p. 341-346, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.steroids.2012.11.018. Acesso em: 17 nov. 2024.
APA
Ceravolo, G. S., Filgueira, F. P., Costa, T. J. da, Lobato, N. de S., Chignalia, A. Z., Araujo, P. X. de, et al. (2013). Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats. Steroids, 78( 3), 341-346. doi:10.1016/j.steroids.2012.11.018
NLM
Ceravolo GS, Filgueira FP, Costa TJ da, Lobato N de S, Chignalia AZ, Araujo PX de, Tostes R de C, Dantas AP, Fortes ZB, Carvalho MHC de. Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats [Internet]. Steroids. 2013 ; 78( 3): 341-346.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.steroids.2012.11.018
Vancouver
Ceravolo GS, Filgueira FP, Costa TJ da, Lobato N de S, Chignalia AZ, Araujo PX de, Tostes R de C, Dantas AP, Fortes ZB, Carvalho MHC de. Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats [Internet]. Steroids. 2013 ; 78( 3): 341-346.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.steroids.2012.11.018
Artigo de periodico
Mechanisms of endothelial dysfunction in obesity-associated hypertension (2012)
Lobato, Nubia de Souza; Filgueira, Fernando Paranaiba; Akamine, Eliana Hiromi ; Passaglia, Rita de Cassia Aleixo Tostes; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Fonte: Brazilian Journal of Medical and Biological Research. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
LOBATO, Nubia de Souza et al. Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, v. 45, n. 5, p. 392-400, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500058. Acesso em: 17 nov. 2024.
APA
Lobato, N. de S., Filgueira, F. P., Akamine, E. H., Passaglia, R. de C. A. T., Carvalho, M. H. C. de, & Fortes, Z. B. (2012). Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, 45( 5), 392-400. doi:10.1590/S0100-879X2012007500058
NLM
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Vancouver
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Artigo de periodico
Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways (2012)
Chignalia, Andreia Zago; Schuldt, Elke Z.; Camargo, Lívia de Lucca; Montezano, Augusto C.; Callera, Gláucia E.; Laurindo, Francisco R.; Lopes, Lucia Rossetti ; Avellar, Maria Cristina W.; Carvalho, Maria Helena Catelli; Fortes, Zuleica Bruno; Touyz, Rhian M.; Passaglia, Rita de Cássia Aleixo Tostes
Fonte: Hypertension. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
CHIGNALIA, Andreia Zago et al. Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways. Hypertension, v. 59, n. 6, p. 1263-1271, 2012Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.111.180620. Acesso em: 17 nov. 2024.
APA
Chignalia, A. Z., Schuldt, E. Z., Camargo, L. de L., Montezano, A. C., Callera, G. E., Laurindo, F. R., et al. (2012). Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways. Hypertension, 59( 6), 1263-1271. doi:10.1161/HYPERTENSIONAHA.111.180620
NLM
Chignalia AZ, Schuldt EZ, Camargo L de L, Montezano AC, Callera GE, Laurindo FR, Lopes LR, Avellar MCW, Carvalho MHC, Fortes ZB, Touyz RM, Passaglia R de CAT. Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways [Internet]. Hypertension. 2012 ; 59( 6): 1263-1271.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.180620
Vancouver
Chignalia AZ, Schuldt EZ, Camargo L de L, Montezano AC, Callera GE, Laurindo FR, Lopes LR, Avellar MCW, Carvalho MHC, Fortes ZB, Touyz RM, Passaglia R de CAT. Testosterone induces vascular smooth muscle cell migration by NADPH oxidase and c-Src–dependent pathways [Internet]. Hypertension. 2012 ; 59( 6): 1263-1271.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.111.180620
Artigo de periodico
Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats (2012)
Filgueira, Fernando Paranaíba; Lobato, Núbia de Souza; Dos Santos, R. A.; Oliveira, M. A.; Akamine, Eliana Hiromi ; Tostes-Passaglia, Rita de Cássia Aleixo; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli de
Fonte: Life Sciences. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
FILGUEIRA, Fernando Paranaíba et al. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, v. 90, n. 17-18, p. 689-694, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.01.017. Acesso em: 17 nov. 2024.
APA
Filgueira, F. P., Lobato, N. de S., Dos Santos, R. A., Oliveira, M. A., Akamine, E. H., Tostes-Passaglia, R. de C. A., et al. (2012). Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, 90( 17-18), 689-694. doi:10.1016/j.lfs.2012.01.017
NLM
Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
Vancouver
Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
Artigo de periodico
O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1 (2010)
Lima, Victor Vitorino; Giachini, Fernanda Regina Casa Grande; Carneiro, Fernando S.; Carneiro, Zidonia N.; Saleh, Mohamed A.; Polock, David M.; Ergul, Adviye; Webb, R. Clinton; Passaglia, Rita de Cassia Aleixo Tostes; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli
Fonte: Hypertension. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
LIMA, Victor Vitorino et al. O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension, v. 55, n. 1, p. 180-188, 2010Tradução . . Acesso em: 17 nov. 2024.
APA
Lima, V. V., Giachini, F. R. C. G., Carneiro, F. S., Carneiro, Z. N., Saleh, M. A., Polock, D. M., et al. (2010). O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension, 55( 1), 180-188.
NLM
Lima VV, Giachini FRCG, Carneiro FS, Carneiro ZN, Saleh MA, Polock DM, Ergul A, Webb RC, Passaglia R de CAT, Fortes ZB, Carvalho MHC. O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension. 2010 ; 55( 1): 180-188.[citado 2024 nov. 17 ]
Vancouver
Lima VV, Giachini FRCG, Carneiro FS, Carneiro ZN, Saleh MA, Polock DM, Ergul A, Webb RC, Passaglia R de CAT, Fortes ZB, Carvalho MHC. O-GlcNAcylation contributes to augmentend vascular reactivity induced by endothelin 1. Hypertension. 2010 ; 55( 1): 180-188.[citado 2024 nov. 17 ]
Artigo de periodico
Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes (2010)
Carrillo-Sepúlveda, Maria Alicia; Ceravolo, Graziela Scalianti; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli; Passaglia, Rita de Cássia Aleixo Tostes; Laurindo, Francisco Rafael Martins; Webb, R. Clinton; Barreto-Chaves, Maria Luiza Morais
Fonte: Cardiovascular Research. Unidade: ICB
Assuntos: FARMACOLOGIA, ANATOMIA
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ABNT
CARRILLO-SEPÚLVEDA, Maria Alicia et al. Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes. Cardiovascular Research, v. 85, n. 3, p. 560-570, 2010Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvp304. Acesso em: 17 nov. 2024.
APA
Carrillo-Sepúlveda, M. A., Ceravolo, G. S., Fortes, Z. B., Carvalho, M. H. C., Passaglia, R. de C. A. T., Laurindo, F. R. M., et al. (2010). Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes. Cardiovascular Research, 85( 3), 560-570. doi:10.1093/cvr/cvp304
NLM
Carrillo-Sepúlveda MA, Ceravolo GS, Fortes ZB, Carvalho MHC, Passaglia R de CAT, Laurindo FRM, Webb RC, Barreto-Chaves MLM. Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes [Internet]. Cardiovascular Research. 2010 ; 85( 3): 560-570.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1093/cvr/cvp304
Vancouver
Carrillo-Sepúlveda MA, Ceravolo GS, Fortes ZB, Carvalho MHC, Passaglia R de CAT, Laurindo FRM, Webb RC, Barreto-Chaves MLM. Thyroid hormone stimulate NO production via activation of the P13K/Akt pathway in vascular myocytes [Internet]. Cardiovascular Research. 2010 ; 85( 3): 560-570.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1093/cvr/cvp304
Artigo de periodico
Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity (2010)
Giachini, Fernanda Regina; Suillivan, Jennifer C.; Lima, Victor Vitorino; Carneiro, Fernando Silva; Fortes, Zuleica Bruno; Pollock, David M.; Carvalho, Maria Helena Catelli; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes
Fonte: Hypertension. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
GIACHINI, Fernanda Regina et al. Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity. Hypertension, v. 55, n. 1, p. 172-179, 2010Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.109.140459. Acesso em: 17 nov. 2024.
APA
Giachini, F. R., Suillivan, J. C., Lima, V. V., Carneiro, F. S., Fortes, Z. B., Pollock, D. M., et al. (2010). Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity. Hypertension, 55( 1), 172-179. doi:10.1161/HYPERTENSIONAHA.109.140459
NLM
Giachini FR, Suillivan JC, Lima VV, Carneiro FS, Fortes ZB, Pollock DM, Carvalho MHC, Webb RC, Passaglia R de CAT. Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity [Internet]. Hypertension. 2010 ; 55( 1): 172-179.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.109.140459
Vancouver
Giachini FR, Suillivan JC, Lima VV, Carneiro FS, Fortes ZB, Pollock DM, Carvalho MHC, Webb RC, Passaglia R de CAT. Extracellular Signal–Regulated Kinase 1/2 Activation, via Downregulation of Mitogen-Activated Protein Kinase Phosphatase 1, Mediates Sex Differences in Desoxycorticosterone Acetate-Salt Hypertension Vascular Reactivity [Internet]. Hypertension. 2010 ; 55( 1): 172-179.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.109.140459
Artigo de periodico
Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats (2010)
Giachini, Fernanda Regina Casagrande; Osmond, David A.; Zhang, Shali; Carneiro, Fernando Silva; Lima, Victor V.; Inscho, Edward W.; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes
Fonte: Clinical Science (1979). Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GIACHINI, Fernanda Regina Casagrande et al. Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats. Clinical Science (1979), v. 118, n. 7, p. 463-471, 2010Tradução . . Disponível em: https://doi.org/10.1042/cs20090392. Acesso em: 17 nov. 2024.
APA
Giachini, F. R. C., Osmond, D. A., Zhang, S., Carneiro, F. S., Lima, V. V., Inscho, E. W., et al. (2010). Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats. Clinical Science (1979), 118( 7), 463-471. doi:10.1042/cs20090392
NLM
Giachini FRC, Osmond DA, Zhang S, Carneiro FS, Lima VV, Inscho EW, Webb RC, Passaglia R de CAT. Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats [Internet]. Clinical Science (1979). 2010 ; 118( 7): 463-471.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1042/cs20090392
Vancouver
Giachini FRC, Osmond DA, Zhang S, Carneiro FS, Lima VV, Inscho EW, Webb RC, Passaglia R de CAT. Clopidogrel, independent of the vascular P2Y12 receptor, improves arterial function in small mesenteric arteries from AngII-hypertensive rats [Internet]. Clinical Science (1979). 2010 ; 118( 7): 463-471.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1042/cs20090392
Artigo de periodico
STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? (2010)
Giachini, Fernanda Regina Casagrande; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes
Fonte: Clinical Science (1979). Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GIACHINI, Fernanda Regina Casagrande e WEBB, R. Clinton e PASSAGLIA, Rita de Cássia Aleixo Tostes. STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction?. Clinical Science (1979), v. 118, n. 6, p. 391-396, 2010Tradução . . Disponível em: https://doi.org/10.1042/cs20090449. Acesso em: 17 nov. 2024.
APA
Giachini, F. R. C., Webb, R. C., & Passaglia, R. de C. A. T. (2010). STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? Clinical Science (1979), 118( 6), 391-396. doi:10.1042/cs20090449
NLM
Giachini FRC, Webb RC, Passaglia R de CAT. STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? [Internet]. Clinical Science (1979). 2010 ; 118( 6): 391-396.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1042/cs20090449
Vancouver
Giachini FRC, Webb RC, Passaglia R de CAT. STIM and Orai proteins: players in sexual differences in hypertension-associated vascular dysfunction? [Internet]. Clinical Science (1979). 2010 ; 118( 6): 391-396.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1042/cs20090449

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