ReP USP - Resultado da busca

Artigo de periodico
Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation (2024)
Costa, Rafael Menezes da ; Dias, Mayara C.; Alves, Juliano Vilela ; Silva, João Lucas M.; Rodrigues, Daniel; Silva, Josiane Fernandes da ; Francescato, Heloisa Della Coletta; Ramalho, Leandra Naira Zambelli ; Coimbra, Terezila Machado ; Passaglia, Rita de Cassia Aleixo Tostes
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: RIM, HIPERGLICEMIA, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael Menezes da et al. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, v. 220, p. 1-10, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2023.115982. Acesso em: 16 nov. 2024.
APA
Costa, R. M. da, Dias, M. C., Alves, J. V., Silva, J. L. M., Rodrigues, D., Silva, J. F. da, et al. (2024). Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, 220, 1-10. doi:10.1016/j.bcp.2023.115982
NLM
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Vancouver
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Artigo de periodico
Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 (2023)
Simplicio, Janaina Aparecida ; Dourado, Thales de Mileto Henrique; Awata, Wanessa Mayumi Carvalho; Vale, Gabriel Tavares do ; Dias, Vinícius Ribeiro; Barros, Paula Rodrigues de ; Martinis, Bruno Spinosa de ; Tostes, Rita de Cássia Aleixo ; Tirapelli, Carlos Renato
Source: Life Sciences. Unidades: FFCLRP, FMRP, EERP
Subjects: ETANOL, INTERLEUCINA 6, TECIDO ADIPOSO, ESTRESSE OXIDATIVO, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SIMPLICIO, Janaina Aparecida et al. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, v. 319, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121526. Acesso em: 16 nov. 2024.
APA
Simplicio, J. A., Dourado, T. de M. H., Awata, W. M. C., Vale, G. T. do, Dias, V. R., Barros, P. R. de, et al. (2023). Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, 319, 1-10. doi:10.1016/j.lfs.2023.121526
NLM
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Vancouver
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Artigo de periodico
Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 (2023)
Fraga-Silva, Thais Fernanda de Campos ; Rosa, Ualter Guilherme Cipriano; Fumagalli, Marcílio Jorge ; Corrêa, Giseli Furlan ; Fuzo, Carlos Alessandro ; Santos, Douglas dos; Mestriner, Fabíola Leslie Antunes Cardoso ; Ribeiro, Christiane Becari ; Teixeira-Carvalho, Andrea; Coelho-dos-Reis, Jordana; Menegueti, Mayra Gonçalves ; Figueiredo, Luiz Tadeu Moraes ; Cunha, Larissa Dias da; Martins-Filho, Olindo A.; Baruffi, Marcelo Dias ; Martins, Maria Auxiliadora ; Passaglia, Rita de Cassia Aleixo Tostes ; Bonato, Vânia Luiza Deperon
Source: iScience. Unidades: FMRP, EERP, FCFRP
Subjects: SISTEMA IMUNE, VIROLOGIA, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRAGA-SILVA, Thais Fernanda de Campos et al. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, v. 26, n. 12, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108366. Acesso em: 16 nov. 2024.
APA
Fraga-Silva, T. F. de C., Rosa, U. G. C., Fumagalli, M. J., Corrêa, G. F., Fuzo, C. A., Santos, D. dos, et al. (2023). Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, 26( 12), 1-19. doi:10.1016/j.isci.2023.108366
NLM
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Vancouver
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2023)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Source: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Subjects: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 16 nov. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2023). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
NLRP3 activation contributes to endothelin-1-induced erectile dysfunction (2023)
Fais, Rafael Sobrano; Costa, Rafael Menezes da ; Mendes, Allan Carvalho; Mestriner, Fabiola Leslie Antunes Cardoso; Comerma-Steffensen, Simón Gabriel; Tostes, Rita de Cássia Aleixo ; Simonsen, Ulf; Carneiro, Fernando Silva
Source: Journal of Cellular and Molecular Medicine. Unidade: FMRP
Subjects: ANTAGONISTAS, ENDOTELINAS, DISFUNÇÃO ERÉTIL, ESPÉCIES REATIVAS DE OXIGÊNIO, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAIS, Rafael Sobrano et al. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, v. 27, n. 1, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1111/jcmm.17463. Acesso em: 16 nov. 2024.
APA
Fais, R. S., Costa, R. M. da, Mendes, A. C., Mestriner, F. L. A. C., Comerma-Steffensen, S. G., Tostes, R. de C. A., et al. (2023). NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, 27( 1), 1-14. doi:10.1111/jcmm.17463
NLM
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/jcmm.17463
Vancouver
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/jcmm.17463
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Subjects: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 16 nov. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 16 nov. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction (2022)
Silva, Josiane Fernandes da; Bolsoni, Juliana A.; Costa, Rafael Menezes da ; Alves, Juliano Vilela ; Bressan, Alecsander Fabrício Moreira ; Silva, Luiz Eduardo Virgilio da ; Costa, Tiago Januário da ; Oliveira, Antonio E. R.; Manzato, Carla Pavan; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Nakaya, Helder Takashi Imoto ; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo
Source: British Journal of Pharmacology. Unidades: FMRP, FCF
Subjects: GORDURAS NA DIETA, CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, OBESIDADE, HIDROCARBONETOS, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Josiane Fernandes da et al. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, v. 179, n. 12, p. 2938-2952, 2022Tradução . . Disponível em: https://doi.org/10.1111/bph.15789. Acesso em: 16 nov. 2024.
APA
Silva, J. F. da, Bolsoni, J. A., Costa, R. M. da, Alves, J. V., Bressan, A. F. M., Silva, L. E. V. da, et al. (2022). Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, 179( 12), 2938-2952. doi:10.1111/bph.15789
NLM
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/bph.15789
Vancouver
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/bph.15789
Artigo de periodico
Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood (2022)
Vidigal, Camila B.; Moura, Kawane F.; Costa, Tiago Januário da ; Borges, Lorena I.; Figaro, Pedro M. M.; Pinto, Ingrid C.; Andrade, Fábio G. de; Gerardin, Daniela C. C.; Casagrande, Rúbia ; Tostes, Rita de Cássia Aleixo ; Franco, Maria do Carmo ; Ceravolo, Graziela S.
Source: Life Sciences. Unidade: FMRP
Subjects: ANTICONVULSIVANTES, AORTA, REGULAÇÃO GÊNICA, PROSTAGLANDINA SINTASE, FATORES SEXUAIS, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VIDIGAL, Camila B. et al. Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood. Life Sciences, v. 288, p. 1-7, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.120189. Acesso em: 16 nov. 2024.
APA
Vidigal, C. B., Moura, K. F., Costa, T. J. da, Borges, L. I., Figaro, P. M. M., Pinto, I. C., et al. (2022). Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood. Life Sciences, 288, 1-7. doi:10.1016/j.lfs.2021.120189
NLM
Vidigal CB, Moura KF, Costa TJ da, Borges LI, Figaro PMM, Pinto IC, Andrade FG de, Gerardin DCC, Casagrande R, Tostes R de CA, Franco M do C, Ceravolo GS. Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood [Internet]. Life Sciences. 2022 ; 288 1-7.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.lfs.2021.120189
Vancouver
Vidigal CB, Moura KF, Costa TJ da, Borges LI, Figaro PMM, Pinto IC, Andrade FG de, Gerardin DCC, Casagrande R, Tostes R de CA, Franco M do C, Ceravolo GS. Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood [Internet]. Life Sciences. 2022 ; 288 1-7.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.lfs.2021.120189
Artigo de periodico
O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system (2022)
Passos Junior, Rinaldo Rodrigues dos; Bomfim, Gisele Facholi ; Giachini, Fernanda R.; Tostes, Rita de Cássia Aleixo ; Lima, Victor Vitorino
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: HIPERTENSÃO, SISTEMA IMUNE, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PASSOS JUNIOR, Rinaldo Rodrigues dos et al. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system. Frontiers in Immunology, v. 13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.852115. Acesso em: 16 nov. 2024.
APA
Passos Junior, R. R. dos, Bomfim, G. F., Giachini, F. R., Tostes, R. de C. A., & Lima, V. V. (2022). O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system. Frontiers in Immunology, 13. doi:10.3389/fimmu.2022.852115
NLM
Passos Junior RR dos, Bomfim GF, Giachini FR, Tostes R de CA, Lima VV. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system [Internet]. Frontiers in Immunology. 2022 ; 13[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fimmu.2022.852115
Vancouver
Passos Junior RR dos, Bomfim GF, Giachini FR, Tostes R de CA, Lima VV. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system [Internet]. Frontiers in Immunology. 2022 ; 13[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fimmu.2022.852115
Artigo de periodico
Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats (2022)
Brazão, Vânia; Colato, Rafaela Pravato ; Santello, Fabricia Helena; Duarte, Andressa ; Goulart, Amanda; Oliveira, Pedro Alexandre Sampaio ; Silva, Carla Brigagão Pacheco da ; Tirapelli, Carlos Renato ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo ; Prado Júnior, José Clóvis do
Source: Experimental Gerontology. Unidades: EERP, FMRP, FCFRP
Subjects: ANTIOXIDANTES, DOENÇA DE CHAGAS, PERÓXIDO DE HIDROGÊNIO, MELATONINA, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRAZÃO, Vânia et al. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, v. 167, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2022.111895. Acesso em: 16 nov. 2024.
APA
Brazão, V., Colato, R. P., Santello, F. H., Duarte, A., Goulart, A., Oliveira, P. A. S., et al. (2022). Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, 167, 1-9. doi:10.1016/j.exger.2022.111895
NLM
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Vancouver
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Source: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Subjects: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 16 nov. 2024.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Artigo de periodico
Programming of vascular dysfunction by maternal stress: immune system implications (2022)
Costa, Tiago Januário da ; Oliveira, Julio Cezar de; Giachini, Fernanda Regina; Lima, Victor Vitorino; Tostes, Rita de Cássia Aleixo ; Bomfim, Gisele Facholi
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: SISTEMA IMUNE, ESPÉCIES REATIVAS DE OXIGÊNIO, ESTRESSE, SISTEMA RENINA-ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Programming of vascular dysfunction by maternal stress: immune system implications. Frontiers in Physiology, v. 13, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.787617. Acesso em: 16 nov. 2024.
APA
Costa, T. J. da, Oliveira, J. C. de, Giachini, F. R., Lima, V. V., Tostes, R. de C. A., & Bomfim, G. F. (2022). Programming of vascular dysfunction by maternal stress: immune system implications. Frontiers in Physiology, 13, 1-17. doi:10.3389/fphys.2022.787617
NLM
Costa TJ da, Oliveira JC de, Giachini FR, Lima VV, Tostes R de CA, Bomfim GF. Programming of vascular dysfunction by maternal stress: immune system implications [Internet]. Frontiers in Physiology. 2022 ; 13 1-17.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fphys.2022.787617
Vancouver
Costa TJ da, Oliveira JC de, Giachini FR, Lima VV, Tostes R de CA, Bomfim GF. Programming of vascular dysfunction by maternal stress: immune system implications [Internet]. Frontiers in Physiology. 2022 ; 13 1-17.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fphys.2022.787617
Artigo de periodico
The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters (2021)
Domingos-Souza, Gean; Santos-Almeida, Fernanda Machado; Meschiari, César Arruda; Ferreira, Nathanne dos Santos; Pereira, Camila André; Oliveira, Nayara Pestana de; Costa, Thaís Caroline Prates; Tostes, Rita de Cássia Aleixo ; White, Carl; Fazan Júnior, Rubens
Source: Hypertension Research. Unidade: FMRP
Subjects: PRESSORECEPTORES, HIPERTENSÃO, ESTIMULAÇÃO ELÉTRICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DOMINGOS-SOUZA, Gean et al. The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters. Hypertension Research, v. 44, n. 8, p. 932–940, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41440-021-00639-9. Acesso em: 16 nov. 2024.
APA
Domingos-Souza, G., Santos-Almeida, F. M., Meschiari, C. A., Ferreira, N. dos S., Pereira, C. A., Oliveira, N. P. de, et al. (2021). The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters. Hypertension Research, 44( 8), 932–940. doi:10.1038/s41440-021-00639-9
NLM
Domingos-Souza G, Santos-Almeida FM, Meschiari CA, Ferreira N dos S, Pereira CA, Oliveira NP de, Costa TCP, Tostes R de CA, White C, Fazan Júnior R. The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters [Internet]. Hypertension Research. 2021 ; 44( 8): 932–940.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41440-021-00639-9
Vancouver
Domingos-Souza G, Santos-Almeida FM, Meschiari CA, Ferreira N dos S, Pereira CA, Oliveira NP de, Costa TCP, Tostes R de CA, White C, Fazan Júnior R. The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters [Internet]. Hypertension Research. 2021 ; 44( 8): 932–940.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41440-021-00639-9
Artigo de periodico
NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes (2021)
Costa, Frederico Ribeiro Campos ; Leite, Jefferson Antonio; Rassi, Diane Meyre ; Silva, Josiane Fernandes da ; Oliveira, Jefferson Elias ; Guimarães, Jhefferson Barbosa ; Freitas, Maria Cristina Foss de ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Carlos, Daniela
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, DIABETES MELLITUS, DOENÇAS AUTOIMUNES, PÂNCREAS, CAMUNDONGOS, POLIMORFISMO, SISTEMA IMUNE, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Frederico Ribeiro Campos et al. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, v. 35, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2021.109176. Acesso em: 16 nov. 2024.
APA
Costa, F. R. C., Leite, J. A., Rassi, D. M., Silva, J. F. da, Oliveira, J. E., Guimarães, J. B., et al. (2021). NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, 35, 1-16. doi:10.1016/j.celrep.2021.109176
NLM
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Vancouver
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Artigo de periodico
Melatonin reverses the loss of the anticontractile effect of perivascular adipose tissue in obese rats (2021)
Gonzaga, Natália de Almeida; Awata, Wanessa Mayumi Carvalho; Ficher, Sabrina Paixão; Assis, Victor Oliveira; Alves, Juliano Vilela; Tostes, Rita de Cássia Aleixo ; Tirapelli, Carlos Renato
Source: Journal of Pineal Research. Unidades: FMRP, EERP
Subjects: AORTA, MELATONINA, ÓXIDO NÍTRICO, OBESIDADE, ESPÉCIES REATIVAS DE OXIGÊNIO, TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZAGA, Natália de Almeida et al. Melatonin reverses the loss of the anticontractile effect of perivascular adipose tissue in obese rats. Journal of Pineal Research, v. 70, n. 2, 2021Tradução . . Disponível em: https://doi.org/10.1111/jpi.12710. Acesso em: 16 nov. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Ficher, S. P., Assis, V. O., Alves, J. V., Tostes, R. de C. A., & Tirapelli, C. R. (2021). Melatonin reverses the loss of the anticontractile effect of perivascular adipose tissue in obese rats. Journal of Pineal Research, 70( 2). doi:10.1111/jpi.12710
NLM
Gonzaga N de A, Awata WMC, Ficher SP, Assis VO, Alves JV, Tostes R de CA, Tirapelli CR. Melatonin reverses the loss of the anticontractile effect of perivascular adipose tissue in obese rats [Internet]. Journal of Pineal Research. 2021 ; 70( 2):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/jpi.12710
Vancouver
Gonzaga N de A, Awata WMC, Ficher SP, Assis VO, Alves JV, Tostes R de CA, Tirapelli CR. Melatonin reverses the loss of the anticontractile effect of perivascular adipose tissue in obese rats [Internet]. Journal of Pineal Research. 2021 ; 70( 2):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/jpi.12710
Artigo de periodico
Disease activity index is associated with subclinical atherosclerosis in childhood-onset systemic lupus erythematosus (2021)
Medeiros, Priscila Beatriz de Souza; Salomão, Roberta Garcia ; Teixeira, Sara Reis; Rassi, Diane Meyre; Rodrigues, Luciana; Aragon, Davi Casale ; Fassini, Priscila Giacomo; Ued, Fábio da Veiga ; Tostes, Rita de Cássia Aleixo ; Monteiro, Jacqueline Pontes; Ferriani, Virgínia Paes Leme ; Carvalho, Luciana Martins de
Source: Pediatric Rheumatology. Unidade: FMRP
Subjects: ARTERIOSCLEROSE, ESTUDOS TRANSVERSAIS, LÚPUS ERITEMATOSO SISTÊMICO, FATORES DE RISCO, ESTUDOS PROSPECTIVOS, AVALIAÇÃO NUTRICIONAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MEDEIROS, Priscila Beatriz de Souza et al. Disease activity index is associated with subclinical atherosclerosis in childhood-onset systemic lupus erythematosus. Pediatric Rheumatology, v. 19, n. 1, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.1186/s12969-021-00513-5. Acesso em: 16 nov. 2024.
APA
Medeiros, P. B. de S., Salomão, R. G., Teixeira, S. R., Rassi, D. M., Rodrigues, L., Aragon, D. C., et al. (2021). Disease activity index is associated with subclinical atherosclerosis in childhood-onset systemic lupus erythematosus. Pediatric Rheumatology, 19( 1), 1-12. doi:10.1186/s12969-021-00513-5
NLM
Medeiros PB de S, Salomão RG, Teixeira SR, Rassi DM, Rodrigues L, Aragon DC, Fassini PG, Ued F da V, Tostes R de CA, Monteiro JP, Ferriani VPL, Carvalho LM de. Disease activity index is associated with subclinical atherosclerosis in childhood-onset systemic lupus erythematosus [Internet]. Pediatric Rheumatology. 2021 ; 19( 1): 1-12.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1186/s12969-021-00513-5
Vancouver
Medeiros PB de S, Salomão RG, Teixeira SR, Rassi DM, Rodrigues L, Aragon DC, Fassini PG, Ued F da V, Tostes R de CA, Monteiro JP, Ferriani VPL, Carvalho LM de. Disease activity index is associated with subclinical atherosclerosis in childhood-onset systemic lupus erythematosus [Internet]. Pediatric Rheumatology. 2021 ; 19( 1): 1-12.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1186/s12969-021-00513-5
Artigo de periodico
Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats (2021)
Troiano, Jéssica A.; Potje, Simone Regina ; Graton, Murilo E.; Gonçalves, Emily T.; Tostes, Rita de Cássia Aleixo ; Antoniali, Cristina
Source: Frontiers in Physiology. Unidades: FMRP, FCFRP
Subjects: AORTA TORÁCICA, ÓXIDO NÍTRICO, GRAVIDEZ, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TROIANO, Jéssica A. et al. Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats. Frontiers in Physiology, v. 12, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphys.2021.760237. Acesso em: 16 nov. 2024.
APA
Troiano, J. A., Potje, S. R., Graton, M. E., Gonçalves, E. T., Tostes, R. de C. A., & Antoniali, C. (2021). Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats. Frontiers in Physiology, 12, 1-13. doi:10.3389/fphys.2021.760237
NLM
Troiano JA, Potje SR, Graton ME, Gonçalves ET, Tostes R de CA, Antoniali C. Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats [Internet]. Frontiers in Physiology. 2021 ; 12 1-13.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fphys.2021.760237
Vancouver
Troiano JA, Potje SR, Graton ME, Gonçalves ET, Tostes R de CA, Antoniali C. Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats [Internet]. Frontiers in Physiology. 2021 ; 12 1-13.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fphys.2021.760237
Artigo de periodico
Ethanol: striking the cardiovascular system by harming the gut microbiota (2021)
Silva, Carla Brigagão Pacheco da ; Oliveira, Jefferson Elias ; McCarthy, Cameron; Wenceslau, Camilla F.; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology : Heart and Circulatory Physiology. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, ÁLCOOL, DOENÇAS CARDIOVASCULARES, PREBIÓTICOS, PROBIÓTICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Carla Brigagão Pacheco da et al. Ethanol: striking the cardiovascular system by harming the gut microbiota. American Journal of Physiology : Heart and Circulatory Physiology, v. 321, n. 2, p. H275-H291, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00225.2021. Acesso em: 16 nov. 2024.
APA
Silva, C. B. P. da, Oliveira, J. E., McCarthy, C., Wenceslau, C. F., Carlos, D., & Tostes, R. de C. A. (2021). Ethanol: striking the cardiovascular system by harming the gut microbiota. American Journal of Physiology : Heart and Circulatory Physiology, 321( 2), H275-H291. doi:10.1152/ajpheart.00225.2021
NLM
Silva CBP da, Oliveira JE, McCarthy C, Wenceslau CF, Carlos D, Tostes R de CA. Ethanol: striking the cardiovascular system by harming the gut microbiota [Internet]. American Journal of Physiology : Heart and Circulatory Physiology. 2021 ; 321( 2): H275-H291.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1152/ajpheart.00225.2021
Vancouver
Silva CBP da, Oliveira JE, McCarthy C, Wenceslau CF, Carlos D, Tostes R de CA. Ethanol: striking the cardiovascular system by harming the gut microbiota [Internet]. American Journal of Physiology : Heart and Circulatory Physiology. 2021 ; 321( 2): H275-H291.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1152/ajpheart.00225.2021
Artigo de periodico
Angiotensin-II activates vascular inflammasome and induces vascular damage (2021)
Cáu, Stêfany Bruno de Assis; Bruder-Nascimento, Ariane; Silva, Marcondes Alves Barbosa da; Ramalho, Fernanda Naira Zambelli; Mestriner, Fabiola Leslie Antunes Cardoso; Lopes, Rheure Alves Moreira; Ferreira, Nathanne dos Santos; Tostes, Rita de Cássia Aleixo ; Nascimento, Thiago Bruder do
Source: Vascular Pharmacology. Unidade: FMRP
Subjects: ANGIOTENSINA II, CÉLULAS CULTIVADAS, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CÁU, Stêfany Bruno de Assis et al. Angiotensin-II activates vascular inflammasome and induces vascular damage. Vascular Pharmacology, v. 139, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106881. Acesso em: 16 nov. 2024.
APA
Cáu, S. B. de A., Bruder-Nascimento, A., Silva, M. A. B. da, Ramalho, F. N. Z., Mestriner, F. L. A. C., Lopes, R. A. M., et al. (2021). Angiotensin-II activates vascular inflammasome and induces vascular damage. Vascular Pharmacology, 139, 1-10. doi:10.1016/j.vph.2021.106881
NLM
Cáu SB de A, Bruder-Nascimento A, Silva MAB da, Ramalho FNZ, Mestriner FLAC, Lopes RAM, Ferreira N dos S, Tostes R de CA, Nascimento TB do. Angiotensin-II activates vascular inflammasome and induces vascular damage [Internet]. Vascular Pharmacology. 2021 ; 139 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.vph.2021.106881
Vancouver
Cáu SB de A, Bruder-Nascimento A, Silva MAB da, Ramalho FNZ, Mestriner FLAC, Lopes RAM, Ferreira N dos S, Tostes R de CA, Nascimento TB do. Angiotensin-II activates vascular inflammasome and induces vascular damage [Internet]. Vascular Pharmacology. 2021 ; 139 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.vph.2021.106881

USP Schools

ReP

Filtros

USP Schools

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Countries/Territories

Funding Agencies

Indexado em

Non normalized affiliation

Countries of institutions of collaboration