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  • Source: Neuroscience. Unidade: ICB

    Subjects: DOENÇA DE PARKINSON, RESPIRAÇÃO, NEURÔNIOS, SEROTONINA, RATOS

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      SANTOS, Luiz Marcelo Oliveira e MOREIRA, Thiago dos Santos e TAKAKURA, Ana Carolina. Raphe pallidus is not important to central chemoreception in a rat model of Parkinson’s disease. Neuroscience, v. 369, p. 350-362, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.11.038. Acesso em: 02 nov. 2024.
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      Santos, L. M. O., Moreira, T. dos S., & Takakura, A. C. (2018). Raphe pallidus is not important to central chemoreception in a rat model of Parkinson’s disease. Neuroscience, 369, 350-362. doi:10.1016/j.neuroscience.2017.11.038
    • NLM

      Santos LMO, Moreira T dos S, Takakura AC. Raphe pallidus is not important to central chemoreception in a rat model of Parkinson’s disease [Internet]. Neuroscience. 2018 ; 369 350-362.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.11.038
    • Vancouver

      Santos LMO, Moreira T dos S, Takakura AC. Raphe pallidus is not important to central chemoreception in a rat model of Parkinson’s disease [Internet]. Neuroscience. 2018 ; 369 350-362.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.11.038
  • Source: Neuroscience. Unidades: IQ, ICB

    Subjects: DOENÇA DE PARKINSON, NEURÔNIOS

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      DATI, L. M et al. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, v. 356, p. 176-181, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.05.013. Acesso em: 02 nov. 2024.
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      Dati, L. M., Ulrich, H., Real, C. C., Feng, Z. P., Sun, H. S., & Britto, L. R. G. de. (2017). Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, 356, 176-181. doi:10.1016/j.neuroscience.2017.05.013
    • NLM

      Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013
    • Vancouver

      Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013
  • Source: Neuroscience. Unidade: ICB

    Assunto: ANATOMIA

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      MÜLLER, Cláudia Janaina Torres et al. On the verge of a respiratory-type panic attack: Selective activations of rostrolateral and caudoventrolateral periaqueductal gray matter following short-lasting escape to a low dose of potassium cyanide. Neuroscience, v. 348, p. 228-240, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.02.022. Acesso em: 02 nov. 2024.
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      Müller, C. J. T., Quintino-dos-Santos, J. W., Schimitel, F. G., Tufik, S., Beijamini, V., Canteras, N. S., & Schenberg, L. C. (2017). On the verge of a respiratory-type panic attack: Selective activations of rostrolateral and caudoventrolateral periaqueductal gray matter following short-lasting escape to a low dose of potassium cyanide. Neuroscience, 348, 228-240. doi:10.1016/j.neuroscience.2017.02.022
    • NLM

      Müller CJT, Quintino-dos-Santos JW, Schimitel FG, Tufik S, Beijamini V, Canteras NS, Schenberg LC. On the verge of a respiratory-type panic attack: Selective activations of rostrolateral and caudoventrolateral periaqueductal gray matter following short-lasting escape to a low dose of potassium cyanide [Internet]. Neuroscience. 2017 ; 348 228-240.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.022
    • Vancouver

      Müller CJT, Quintino-dos-Santos JW, Schimitel FG, Tufik S, Beijamini V, Canteras NS, Schenberg LC. On the verge of a respiratory-type panic attack: Selective activations of rostrolateral and caudoventrolateral periaqueductal gray matter following short-lasting escape to a low dose of potassium cyanide [Internet]. Neuroscience. 2017 ; 348 228-240.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.022
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      MALHEIROS-LIMA, Milene R. e TAKAKURA, Ana Carolina e MOREIRA, Thiago dos Santos. Depletion of rostral ventrolateral medullary catecholaminergic neurons impairs the hypoxic ventilatory response in conscious rats. Neuroscience, v. 351, p. 1-14, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.03.031. Acesso em: 02 nov. 2024.
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      Malheiros-Lima, M. R., Takakura, A. C., & Moreira, T. dos S. (2017). Depletion of rostral ventrolateral medullary catecholaminergic neurons impairs the hypoxic ventilatory response in conscious rats. Neuroscience, 351, 1-14. doi:10.1016/j.neuroscience.2017.03.031
    • NLM

      Malheiros-Lima MR, Takakura AC, Moreira T dos S. Depletion of rostral ventrolateral medullary catecholaminergic neurons impairs the hypoxic ventilatory response in conscious rats [Internet]. Neuroscience. 2017 ; 351 1-14.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.03.031
    • Vancouver

      Malheiros-Lima MR, Takakura AC, Moreira T dos S. Depletion of rostral ventrolateral medullary catecholaminergic neurons impairs the hypoxic ventilatory response in conscious rats [Internet]. Neuroscience. 2017 ; 351 1-14.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.03.031
  • Source: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      FERREIRA-NETO, H. C. et al. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation. Neuroscience, v. 349, p. 253-263, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.02.05. Acesso em: 02 nov. 2024.
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      Ferreira-Neto, H. C., Ribeiro, I. M. R., Moreira, T. dos S., Yao, S. T., & Antunes, V. R. (2017). Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation. Neuroscience, 349, 253-263. doi:10.1016/j.neuroscience.2017.02.05
    • NLM

      Ferreira-Neto HC, Ribeiro IMR, Moreira T dos S, Yao ST, Antunes VR. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation [Internet]. Neuroscience. 2017 ; 349 253-263.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.05
    • Vancouver

      Ferreira-Neto HC, Ribeiro IMR, Moreira T dos S, Yao ST, Antunes VR. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation [Internet]. Neuroscience. 2017 ; 349 253-263.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.05
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      TAXINI, Camila L. et al. Role of A5 noradrenergic neurons in the chemoreflex control of respiratory and sympathetic activities in unanesthetized conditions. Neuroscience, v. 354, p. 146-157, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.04.033. Acesso em: 02 nov. 2024.
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      Taxini, C. L., Moreira, T. dos S., Takakura, A. C., Bícego, K. C., Gargaglioni, L. H., & Zoccal, D. B. (2017). Role of A5 noradrenergic neurons in the chemoreflex control of respiratory and sympathetic activities in unanesthetized conditions. Neuroscience, 354, 146-157. doi:10.1016/j.neuroscience.2017.04.033
    • NLM

      Taxini CL, Moreira T dos S, Takakura AC, Bícego KC, Gargaglioni LH, Zoccal DB. Role of A5 noradrenergic neurons in the chemoreflex control of respiratory and sympathetic activities in unanesthetized conditions [Internet]. Neuroscience. 2017 ; 354 146-157.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.04.033
    • Vancouver

      Taxini CL, Moreira T dos S, Takakura AC, Bícego KC, Gargaglioni LH, Zoccal DB. Role of A5 noradrenergic neurons in the chemoreflex control of respiratory and sympathetic activities in unanesthetized conditions [Internet]. Neuroscience. 2017 ; 354 146-157.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.04.033
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RAVELLI, Katherine Garcia et al. NADPH oxidase contributes to streptozotocin-induced neurodegeneration. Neuroscience, v. 358, p. 227-237, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.06.050. Acesso em: 02 nov. 2024.
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      Ravelli, K. G., Rosário, B. dos A., Vasconcelos, A. R., Scavone, C., Camarini, R., Hernandes, M. S., & Britto, L. R. G. de. (2017). NADPH oxidase contributes to streptozotocin-induced neurodegeneration. Neuroscience, 358, 227-237. doi:10.1016/j.neuroscience.2017.06.050
    • NLM

      Ravelli KG, Rosário B dos A, Vasconcelos AR, Scavone C, Camarini R, Hernandes MS, Britto LRG de. NADPH oxidase contributes to streptozotocin-induced neurodegeneration [Internet]. Neuroscience. 2017 ; 358 227-237.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.06.050
    • Vancouver

      Ravelli KG, Rosário B dos A, Vasconcelos AR, Scavone C, Camarini R, Hernandes MS, Britto LRG de. NADPH oxidase contributes to streptozotocin-induced neurodegeneration [Internet]. Neuroscience. 2017 ; 358 227-237.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.06.050
  • Source: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      RAMOS-LOBO, Angela M. et al. SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation. Neuroscience, v. 365, p. 114-124, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.09.048. Acesso em: 02 nov. 2024.
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      Ramos-Lobo, A. M., Teixeira, P. D. S., Furigo, I. C., & Donato Junior, J. (2017). SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation. Neuroscience, 365, 114-124. doi:10.1016/j.neuroscience.2017.09.048
    • NLM

      Ramos-Lobo AM, Teixeira PDS, Furigo IC, Donato Junior J. SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation [Internet]. Neuroscience. 2017 ; 365 114-124.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.09.048
    • Vancouver

      Ramos-Lobo AM, Teixeira PDS, Furigo IC, Donato Junior J. SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation [Internet]. Neuroscience. 2017 ; 365 114-124.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.09.048
  • Source: Neuroscience. Unidade: ICB

    Subjects: RESPIRAÇÃO, NEURÔNIOS

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      SILVA, Josiane do Nascimento et al. Inhibition of the pontine Kölliker-Fuse nucleus reduces genioglossal activity elicited by stimulation of the retrotrapezoid chemoreceptor neurons. Neuroscience, v. 328, p. 9-21, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2016.04.028. Acesso em: 02 nov. 2024.
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      Silva, J. do N., Lucena, E. V. de, Silva, T. de M. e, Damasceno, R. dos S., Takakura, A. C., & Moreira, T. dos S. (2016). Inhibition of the pontine Kölliker-Fuse nucleus reduces genioglossal activity elicited by stimulation of the retrotrapezoid chemoreceptor neurons. Neuroscience, 328, 9-21. doi:10.1016/j.neuroscience.2016.04.028
    • NLM

      Silva J do N, Lucena EV de, Silva T de M e, Damasceno R dos S, Takakura AC, Moreira T dos S. Inhibition of the pontine Kölliker-Fuse nucleus reduces genioglossal activity elicited by stimulation of the retrotrapezoid chemoreceptor neurons [Internet]. Neuroscience. 2016 ; 328 9-21.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2016.04.028
    • Vancouver

      Silva J do N, Lucena EV de, Silva T de M e, Damasceno R dos S, Takakura AC, Moreira T dos S. Inhibition of the pontine Kölliker-Fuse nucleus reduces genioglossal activity elicited by stimulation of the retrotrapezoid chemoreceptor neurons [Internet]. Neuroscience. 2016 ; 328 9-21.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2016.04.028
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇA DE PARKINSON, RESPIRAÇÃO (DEFICIÊNCIA), RATOS, MODELOS ANIMAIS

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      TUPPY, Marina et al. Respiratory deficits in a rat model of Parkinson's disease. Neuroscience, v. 297, p. 194-204, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2015.03.048. Acesso em: 02 nov. 2024.
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      Tuppy, M., Falquetto, B., Santos, L. A. dos, Britto, L. R. G. de, Chiavegatto, S., Moreira, T. dos S., & Takakura, A. C. (2015). Respiratory deficits in a rat model of Parkinson's disease. Neuroscience, 297, 194-204. doi:10.1016/j.neuroscience.2015.03.048
    • NLM

      Tuppy M, Falquetto B, Santos LA dos, Britto LRG de, Chiavegatto S, Moreira T dos S, Takakura AC. Respiratory deficits in a rat model of Parkinson's disease [Internet]. Neuroscience. 2015 ; 297 194-204.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2015.03.048
    • Vancouver

      Tuppy M, Falquetto B, Santos LA dos, Britto LRG de, Chiavegatto S, Moreira T dos S, Takakura AC. Respiratory deficits in a rat model of Parkinson's disease [Internet]. Neuroscience. 2015 ; 297 194-204.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2015.03.048
  • Source: Neuroscience. Unidade: ICB

    Subjects: ANATOMIA, MELANINAS, HORMÔNIO CONCENTRADOR DE MELANINA, GABA, COLINÉRGICOS, CÉREBRO, RATOS

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      HAEMMERLE, Carlos Alexandre dos Santos e CAMPOS, Ana Maria Peraçoli e BITTENCOURT, Jackson Cioni. Melanin-concentrating hormone inputs to the nucleus accumbens originate from distinct hypothalamic sources and are apposed to GABAergic and cholinergic cells in the Long-Evans rat brain. Neuroscience, v. 289, p. 392-405, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2015.01.014. Acesso em: 02 nov. 2024.
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      Haemmerle, C. A. dos S., Campos, A. M. P., & Bittencourt, J. C. (2015). Melanin-concentrating hormone inputs to the nucleus accumbens originate from distinct hypothalamic sources and are apposed to GABAergic and cholinergic cells in the Long-Evans rat brain. Neuroscience, 289, 392-405. doi:10.1016/j.neuroscience.2015.01.014
    • NLM

      Haemmerle CA dos S, Campos AMP, Bittencourt JC. Melanin-concentrating hormone inputs to the nucleus accumbens originate from distinct hypothalamic sources and are apposed to GABAergic and cholinergic cells in the Long-Evans rat brain [Internet]. Neuroscience. 2015 ; 289 392-405.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2015.01.014
    • Vancouver

      Haemmerle CA dos S, Campos AMP, Bittencourt JC. Melanin-concentrating hormone inputs to the nucleus accumbens originate from distinct hypothalamic sources and are apposed to GABAergic and cholinergic cells in the Long-Evans rat brain [Internet]. Neuroscience. 2015 ; 289 392-405.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2015.01.014
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA, TRONCO CEREBRAL, APNÉIA, RATOS

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      FERREIRA, C. B. et al. Brainstem areas activated by intermittent apnea in awake unrestrained rats. Neuroscience, v. 297, p. 262-271, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2015.04.007. Acesso em: 02 nov. 2024.
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      Ferreira, C. B., Schoorlemmer, G. H., Rossi, M. V., Takakura, A. C., Falquetto, B., Moreira, T. dos S., & Cravo, S. L. D. (2015). Brainstem areas activated by intermittent apnea in awake unrestrained rats. Neuroscience, 297, 262-271. doi:10.1016/j.neuroscience.2015.04.007
    • NLM

      Ferreira CB, Schoorlemmer GH, Rossi MV, Takakura AC, Falquetto B, Moreira T dos S, Cravo SLD. Brainstem areas activated by intermittent apnea in awake unrestrained rats [Internet]. Neuroscience. 2015 ; 297 262-271.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2015.04.007
    • Vancouver

      Ferreira CB, Schoorlemmer GH, Rossi MV, Takakura AC, Falquetto B, Moreira T dos S, Cravo SLD. Brainstem areas activated by intermittent apnea in awake unrestrained rats [Internet]. Neuroscience. 2015 ; 297 262-271.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2015.04.007
  • Source: Neuroscience. Unidades: ICB, FMRP

    Subjects: ANATOMIA, FISIOLOGIA

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      DONATO JUNIOR, José et al. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition. Neuroscience, v. 241, p. 67-79, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.03.013. Acesso em: 02 nov. 2024.
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      Donato Junior, J., Lee. C,, Ratra, D. V., Franci, C. R., Canteras, N. S., & Elias, C. F. (2013). Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition. Neuroscience, 241, 67-79. doi:10.1016/j.neuroscience.2013.03.013
    • NLM

      Donato Junior J, Lee. C, Ratra DV, Franci CR, Canteras NS, Elias CF. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition [Internet]. Neuroscience. 2013 ; 241 67-79.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.03.013
    • Vancouver

      Donato Junior J, Lee. C, Ratra DV, Franci CR, Canteras NS, Elias CF. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition [Internet]. Neuroscience. 2013 ; 241 67-79.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.03.013
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      FALQUETTO, Bárbara e TAKAKURA, Ana Carolina e MOREIRA, Thiago dos Santos. Pontomedullary and hypothalamic distribution of fos-like immunoreactive neurons after acute exercise in rats. Neuroscience, v. 212, p. 120-130, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2012.03.039. Acesso em: 02 nov. 2024.
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      Falquetto, B., Takakura, A. C., & Moreira, T. dos S. (2012). Pontomedullary and hypothalamic distribution of fos-like immunoreactive neurons after acute exercise in rats. Neuroscience, 212, 120-130. doi:10.1016/j.neuroscience.2012.03.039
    • NLM

      Falquetto B, Takakura AC, Moreira T dos S. Pontomedullary and hypothalamic distribution of fos-like immunoreactive neurons after acute exercise in rats [Internet]. Neuroscience. 2012 ; 212 120-130.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2012.03.039
    • Vancouver

      Falquetto B, Takakura AC, Moreira T dos S. Pontomedullary and hypothalamic distribution of fos-like immunoreactive neurons after acute exercise in rats [Internet]. Neuroscience. 2012 ; 212 120-130.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2012.03.039
  • Source: Neuroscience. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      MARTINEZ, R. C. et al. Amygdalar roles during exposure to a live predator and to a predator-associated context. Neuroscience, v. 172, n. 13, p. 314-328, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2010.10.033. Acesso em: 02 nov. 2024.
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      Martinez, R. C., Carvalho-Netto, E. F., Ribeiro-Barbosa, É. R., Baldo, M. V. C., & Canteras, N. S. (2011). Amygdalar roles during exposure to a live predator and to a predator-associated context. Neuroscience, 172( 13), 314-328. doi:10.1016/j.neuroscience.2010.10.033
    • NLM

      Martinez RC, Carvalho-Netto EF, Ribeiro-Barbosa ÉR, Baldo MVC, Canteras NS. Amygdalar roles during exposure to a live predator and to a predator-associated context [Internet]. Neuroscience. 2011 ; 172( 13): 314-328.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2010.10.033
    • Vancouver

      Martinez RC, Carvalho-Netto EF, Ribeiro-Barbosa ÉR, Baldo MVC, Canteras NS. Amygdalar roles during exposure to a live predator and to a predator-associated context [Internet]. Neuroscience. 2011 ; 172( 13): 314-328.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2010.10.033
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      TAKAKURA, Ana Carolina et al. Inhibition of the caudal pressor area reduces cardiorespiratory chemoreflex responses. Neuroscience, v. 177, n. Único, p. 84-92, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2011.01.002. Acesso em: 02 nov. 2024.
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      Takakura, A. C., Moreira, T. dos S., Menani, J. V., & Colombari, E. (2011). Inhibition of the caudal pressor area reduces cardiorespiratory chemoreflex responses. Neuroscience, 177( Único), 84-92. doi:10.1016/j.neuroscience.2011.01.002
    • NLM

      Takakura AC, Moreira T dos S, Menani JV, Colombari E. Inhibition of the caudal pressor area reduces cardiorespiratory chemoreflex responses [Internet]. Neuroscience. 2011 ; 177( Único): 84-92.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.01.002
    • Vancouver

      Takakura AC, Moreira T dos S, Menani JV, Colombari E. Inhibition of the caudal pressor area reduces cardiorespiratory chemoreflex responses [Internet]. Neuroscience. 2011 ; 177( Único): 84-92.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.01.002

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