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  • Source: Microbes and Infection. Unidades: FMVZ, ICB

    Subjects: LEPTOSPIROSE, MODELOS ANIMAIS DE DOENÇAS, INFLAMAÇÃO, IMUNIDADE

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      VASSALAKIS, Julia Avian et al. Murine C3 of the complement system affects infection by Leptospira interrogans. Microbes and Infection, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2024.105413. Acesso em: 10 nov. 2024.
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      Vassalakis, J. A., Yamashita, D. H. S., Midon, L. M., Cogliati, B., Heinemann, M. B., Amamura, T. A., & Isaac, L. (2024). Murine C3 of the complement system affects infection by Leptospira interrogans. Microbes and Infection. doi:10.1016/j.micinf.2024.105413
    • NLM

      Vassalakis JA, Yamashita DHS, Midon LM, Cogliati B, Heinemann MB, Amamura TA, Isaac L. Murine C3 of the complement system affects infection by Leptospira interrogans [Internet]. Microbes and Infection. 2024 ;[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2024.105413
    • Vancouver

      Vassalakis JA, Yamashita DHS, Midon LM, Cogliati B, Heinemann MB, Amamura TA, Isaac L. Murine C3 of the complement system affects infection by Leptospira interrogans [Internet]. Microbes and Infection. 2024 ;[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2024.105413
  • Source: Microbes and Infection. Unidade: ICB

    Subjects: IMUNOLOGIA, MONÓCITOS, PARACOCCIDIOIDES, TABAGISMO, MICOSES, PENICILLIUM, FIBROSE PULMONAR, FUMO, INFLAMAÇÃO, CITOCINAS

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      AMORIM, Barbara Casella et al. Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia. Microbes and Infection. Issy les Moulineaux: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.micinf.2019.11.001. Acesso em: 10 nov. 2024. , 2020
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      Amorim, B. C., Latini, A. C. P., Golim, M. de A., Ruiz Júnior, R. L., Yoo, H. H. B., Arruda, M. S. P. de, et al. (2020). Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia. Microbes and Infection. Issy les Moulineaux: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.micinf.2019.11.001
    • NLM

      Amorim BC, Latini ACP, Golim M de A, Ruiz Júnior RL, Yoo HHB, Arruda MSP de, Tavares AHFP, Cavalcante R de S, Mendes RP, Pontillo A, Venturini J. Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia [Internet]. Microbes and Infection. 2020 ; 22( 3): 137-143.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2019.11.001
    • Vancouver

      Amorim BC, Latini ACP, Golim M de A, Ruiz Júnior RL, Yoo HHB, Arruda MSP de, Tavares AHFP, Cavalcante R de S, Mendes RP, Pontillo A, Venturini J. Enhanced expression of NLRP3 inflammasome components by monocytes of patients with pulmonary paracoccidioidomycosis is associated with smoking and intracellular hypoxemia [Internet]. Microbes and Infection. 2020 ; 22( 3): 137-143.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2019.11.001
  • Source: Microbes and Infection. Unidades: FMVZ, FM, ICB

    Subjects: LEPTOSPIROSE, FATORES IMUNOLÓGICOS, IMUNOGLOBULINAS, IMUNOLOGIA, IMUNOPROTEÍNAS, LECTINAS

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      SILVA, Priscilla Yuri Okochi Alves da et al. Contribution of complement system pathways to the killing of Leptospira spp. Microbes and Infection, v. 22, n. 10, p. 550-557, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2020.07.005. Acesso em: 10 nov. 2024.
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      Silva, P. Y. O. A. da, Midon, L. M., Heinemann, M. B., Vasconcelos, D. de M., Barbosa, A. S., & Isaac, L. (2020). Contribution of complement system pathways to the killing of Leptospira spp. Microbes and Infection, 22( 10), 550-557. doi:10.1016/j.micinf.2020.07.005
    • NLM

      Silva PYOA da, Midon LM, Heinemann MB, Vasconcelos D de M, Barbosa AS, Isaac L. Contribution of complement system pathways to the killing of Leptospira spp [Internet]. Microbes and Infection. 2020 ; 22( 10): 550-557.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2020.07.005
    • Vancouver

      Silva PYOA da, Midon LM, Heinemann MB, Vasconcelos D de M, Barbosa AS, Isaac L. Contribution of complement system pathways to the killing of Leptospira spp [Internet]. Microbes and Infection. 2020 ; 22( 10): 550-557.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2020.07.005
  • Source: Microbes and Infection. Unidade: ICB

    Subjects: IMUNOLOGIA, LEPTOSPIROSE, BACTÉRIAS GRAM-NEGATIVAS, PROTEÍNAS

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      TORRES, Azaf Moreno et al. Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins. Microbes and Infection, v. 21, n. 8–9, p. 377-385, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2019.03.001. Acesso em: 10 nov. 2024.
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      Torres, A. M., Jiménez, I. R. M., Moctezuma, A. de la P., Sánchez, L. O. C., Fraga, T. R., Barbosa, A. S., et al. (2019). Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins. Microbes and Infection, 21( 8–9), 377-385. doi:10.1016/j.micinf.2019.03.001
    • NLM

      Torres AM, Jiménez IRM, Moctezuma A de la P, Sánchez LOC, Fraga TR, Barbosa AS, Isaac L, Ruiz AS. Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins [Internet]. Microbes and Infection. 2019 ; 21( 8–9): 377-385.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2019.03.001
    • Vancouver

      Torres AM, Jiménez IRM, Moctezuma A de la P, Sánchez LOC, Fraga TR, Barbosa AS, Isaac L, Ruiz AS. Culture-attenuated pathogenic Leptospira lose the ability to survive to complement-mediated-killing due to lower expression of factor H binding proteins [Internet]. Microbes and Infection. 2019 ; 21( 8–9): 377-385.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2019.03.001
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      AMARAL, Eduardo P. e LASUNSKAIA, Elena B. e LIMA, Maria Regina D'Império. Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death. Microbes and Infection, v. 18, n. 1, p. 11-20, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2015.09.005. Acesso em: 10 nov. 2024.
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      Amaral, E. P., Lasunskaia, E. B., & Lima, M. R. D. 'I. (2016). Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death. Microbes and Infection, 18( 1), 11-20. doi:10.1016/j.micinf.2015.09.005
    • NLM

      Amaral EP, Lasunskaia EB, Lima MRD'I. Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death [Internet]. Microbes and Infection. 2016 ; 18( 1): 11-20.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2015.09.005
    • Vancouver

      Amaral EP, Lasunskaia EB, Lima MRD'I. Innate immunity in tuberculosis: how the sensing of mycobacteria and tissue damage modulates macrophage death [Internet]. Microbes and Infection. 2016 ; 18( 1): 11-20.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2015.09.005
  • Source: Microbes and Infection. Unidade: ICB

    Subjects: IMUNOLOGIA, LEISHMANIA BRASILIENSIS, MACRÓFAGOS, LEUCOTRIENOS

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      MORATO, Camila I. et al. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, v. 16, n. 11, p. 945-953, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.08.015. Acesso em: 10 nov. 2024.
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      Morato, C. I., Silva Jr, I. A. da, Borges, A. F., Dorta, M. L., Oliveira, M. A. P., Jancar, S., et al. (2014). Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, 16( 11), 945-953. doi:10.1016/j.micinf.2014.08.015
    • NLM

      Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2014.08.015
    • Vancouver

      Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2014.08.015
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SHIO, Marina Tiemi et al. Crucial cytokine interactions in nitric oxide production induced by Mycoplasma arthritidis superantigen. Microbes and Infection, v. 10, n. 14/15, p. 1543-1551, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2008.09.010. Acesso em: 10 nov. 2024.
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      Shio, M. T., Olivier, M., Jancar, S., & Ribeiro-Dias, F. (2008). Crucial cytokine interactions in nitric oxide production induced by Mycoplasma arthritidis superantigen. Microbes and Infection, 10( 14/15), 1543-1551. doi:10.1016/j.micinf.2008.09.010
    • NLM

      Shio MT, Olivier M, Jancar S, Ribeiro-Dias F. Crucial cytokine interactions in nitric oxide production induced by Mycoplasma arthritidis superantigen [Internet]. Microbes and Infection. 2008 ; 10( 14/15): 1543-1551.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2008.09.010
    • Vancouver

      Shio MT, Olivier M, Jancar S, Ribeiro-Dias F. Crucial cytokine interactions in nitric oxide production induced by Mycoplasma arthritidis superantigen [Internet]. Microbes and Infection. 2008 ; 10( 14/15): 1543-1551.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2008.09.010
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      CHIARELLA, Andressa P. et al. The relative importance of 'CD4 POT. +' and 'CD8 POT. +'T cells in immunity to pulmonary paracoccidioidomycosis. Microbes and Infection, v. 9, n. 9, p. 1078-1088, 2007Tradução . . Acesso em: 10 nov. 2024.
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      Chiarella, A. P., Arruda, C., Pina, A., Costa, T. A., Ferreira, R. de C. V., & Calich, V. L. G. (2007). The relative importance of 'CD4 POT. +' and 'CD8 POT. +'T cells in immunity to pulmonary paracoccidioidomycosis. Microbes and Infection, 9( 9), 1078-1088.
    • NLM

      Chiarella AP, Arruda C, Pina A, Costa TA, Ferreira R de CV, Calich VLG. The relative importance of 'CD4 POT. +' and 'CD8 POT. +'T cells in immunity to pulmonary paracoccidioidomycosis. Microbes and Infection. 2007 ; 9( 9): 1078-1088.[citado 2024 nov. 10 ]
    • Vancouver

      Chiarella AP, Arruda C, Pina A, Costa TA, Ferreira R de CV, Calich VLG. The relative importance of 'CD4 POT. +' and 'CD8 POT. +'T cells in immunity to pulmonary paracoccidioidomycosis. Microbes and Infection. 2007 ; 9( 9): 1078-1088.[citado 2024 nov. 10 ]
  • Source: Microbes and Infection. Unidades: ICB, FM

    Assunto: IMUNOLOGIA

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      BILATE, Angelina Morand Bianchi et al. TNF blockade aggravates experimental chronic Chagas disease cardiomyopathy. Microbes and Infection, v. 9, n. 9, p. 1104-1113, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2007.05.014. Acesso em: 10 nov. 2024.
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      Bilate, A. M. B., Salemi, V. M., Ramires, F. J., Brito, T. de, Russo, M., Fonseca, S. G., et al. (2007). TNF blockade aggravates experimental chronic Chagas disease cardiomyopathy. Microbes and Infection, 9( 9), 1104-1113. doi:10.1016/j.micinf.2007.05.014
    • NLM

      Bilate AMB, Salemi VM, Ramires FJ, Brito T de, Russo M, Fonseca SG, Faé KC, Martins DG, Silva AM, Mady C, Kalil Filho JE, Cunha-Neto E. TNF blockade aggravates experimental chronic Chagas disease cardiomyopathy [Internet]. Microbes and Infection. 2007 ; 9( 9): 1104-1113.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2007.05.014
    • Vancouver

      Bilate AMB, Salemi VM, Ramires FJ, Brito T de, Russo M, Fonseca SG, Faé KC, Martins DG, Silva AM, Mady C, Kalil Filho JE, Cunha-Neto E. TNF blockade aggravates experimental chronic Chagas disease cardiomyopathy [Internet]. Microbes and Infection. 2007 ; 9( 9): 1104-1113.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2007.05.014
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      ARRUDA, Celina et al. A primary subcutaneous infection with Paracoccidioides brasiliensis leads to immunoprotection or exacerbated disease depending on the route of challenge. Microbes and Infection, v. 9, n. 3, p. 308-316, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2006.12.005. Acesso em: 10 nov. 2024.
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      Arruda, C., Kashino, S. S., Fazioli, R. A., & Calich, V. L. G. (2007). A primary subcutaneous infection with Paracoccidioides brasiliensis leads to immunoprotection or exacerbated disease depending on the route of challenge. Microbes and Infection, 9( 3), 308-316. doi:10.1016/j.micinf.2006.12.005
    • NLM

      Arruda C, Kashino SS, Fazioli RA, Calich VLG. A primary subcutaneous infection with Paracoccidioides brasiliensis leads to immunoprotection or exacerbated disease depending on the route of challenge [Internet]. Microbes and Infection. 2007 ; 9( 3): 308-316.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2006.12.005
    • Vancouver

      Arruda C, Kashino SS, Fazioli RA, Calich VLG. A primary subcutaneous infection with Paracoccidioides brasiliensis leads to immunoprotection or exacerbated disease depending on the route of challenge [Internet]. Microbes and Infection. 2007 ; 9( 3): 308-316.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2006.12.005
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      ARRUDA, Celina e VAZ, Celidéia Aparecida Coppi e CALICH, Vera Lúcia Garcia. Aseptic cure of pulmonary paracoccidioidomycosis can be achieved after a previous subcutaneous immunization of susceptible but not resistant mice. Microbes and Infection, v. 9, n. 6, p. 704-713, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2007.02.014. Acesso em: 10 nov. 2024.
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      Arruda, C., Vaz, C. A. C., & Calich, V. L. G. (2007). Aseptic cure of pulmonary paracoccidioidomycosis can be achieved after a previous subcutaneous immunization of susceptible but not resistant mice. Microbes and Infection, 9( 6), 704-713. doi:10.1016/j.micinf.2007.02.014
    • NLM

      Arruda C, Vaz CAC, Calich VLG. Aseptic cure of pulmonary paracoccidioidomycosis can be achieved after a previous subcutaneous immunization of susceptible but not resistant mice [Internet]. Microbes and Infection. 2007 ; 9( 6): 704-713.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2007.02.014
    • Vancouver

      Arruda C, Vaz CAC, Calich VLG. Aseptic cure of pulmonary paracoccidioidomycosis can be achieved after a previous subcutaneous immunization of susceptible but not resistant mice [Internet]. Microbes and Infection. 2007 ; 9( 6): 704-713.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2007.02.014
  • Source: Microbes and Infection. Unidades: FCF, ICB

    Subjects: IMUNOLOGIA, MACRÓFAGOS, DERMATOMICOSES

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      CAMPOS, Marina Reis de Moura et al. Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum. Microbes and Infection, v. 8, n. 2, p. 372-379, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2005.07.028. Acesso em: 10 nov. 2024.
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      Campos, M. R. de M., Russo, M., Gomes, E., & Almeida, S. R. de. (2006). Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum. Microbes and Infection, 8( 2), 372-379. doi:10.1016/j.micinf.2005.07.028
    • NLM

      Campos MR de M, Russo M, Gomes E, Almeida SR de. Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum [Internet]. Microbes and Infection. 2006 ; 8( 2): 372-379.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2005.07.028
    • Vancouver

      Campos MR de M, Russo M, Gomes E, Almeida SR de. Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum [Internet]. Microbes and Infection. 2006 ; 8( 2): 372-379.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2005.07.028
  • Source: Microbes and Infection. Unidades: ICB, FCF

    Assunto: IMUNOLOGIA

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      CAMPOS, M. R. M. e RUSSO, Momtchilo e ALMEIDA, Sandro Rogério de. Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum. Microbes and Infection, v. 8, n. 2, p. 372-379, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2005.07.028. Acesso em: 10 nov. 2024.
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      Campos, M. R. M., Russo, M., & Almeida, S. R. de. (2005). Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum. Microbes and Infection, 8( 2), 372-379. doi:10.1016/j.micinf.2005.07.028
    • NLM

      Campos MRM, Russo M, Almeida SR de. Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum [Internet]. Microbes and Infection. 2005 ; 8( 2): 372-379.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2005.07.028
    • Vancouver

      Campos MRM, Russo M, Almeida SR de. Stimulation, inhibition and death of macrophages infected with Trichophyton rubrum [Internet]. Microbes and Infection. 2005 ; 8( 2): 372-379.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2005.07.028
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      OLIVEIRA, Milton A. P. et al. Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania. Microbes and Infection, v. 7, n. 2, p. 213-223, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2004.10.013. Acesso em: 10 nov. 2024.
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      Oliveira, M. A. P., Tadokoro, C. E., Lima, G. M. C. A., Mosca, T., Vieira, L. Q., Leenen, P. J. M., & Abrahamsohn, I. A. (2005). Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania. Microbes and Infection, 7( 2), 213-223. doi:10.1016/j.micinf.2004.10.013
    • NLM

      Oliveira MAP, Tadokoro CE, Lima GMCA, Mosca T, Vieira LQ, Leenen PJM, Abrahamsohn IA. Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania [Internet]. Microbes and Infection. 2005 ; 7( 2): 213-223.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2004.10.013
    • Vancouver

      Oliveira MAP, Tadokoro CE, Lima GMCA, Mosca T, Vieira LQ, Leenen PJM, Abrahamsohn IA. Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania [Internet]. Microbes and Infection. 2005 ; 7( 2): 213-223.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2004.10.013
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      BASTOS, Karina R. B. et al. What kind of message does IL-12/IL-23 bring to macrophages and dentritic cells?. Microbes and Infection, v. 6, p. 630-6, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2004.02.012. Acesso em: 10 nov. 2024.
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      Bastos, K. R. B., Marinho, C. R. F., Barboza, R., Russo, M., Alvarez, J. M., & Lima, M. R. D. 'I. (2004). What kind of message does IL-12/IL-23 bring to macrophages and dentritic cells? Microbes and Infection, 6, 630-6. doi:10.1016/j.micinf.2004.02.012
    • NLM

      Bastos KRB, Marinho CRF, Barboza R, Russo M, Alvarez JM, Lima MRD'I. What kind of message does IL-12/IL-23 bring to macrophages and dentritic cells? [Internet]. Microbes and Infection. 2004 ; 6 630-6.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2004.02.012
    • Vancouver

      Bastos KRB, Marinho CRF, Barboza R, Russo M, Alvarez JM, Lima MRD'I. What kind of message does IL-12/IL-23 bring to macrophages and dentritic cells? [Internet]. Microbes and Infection. 2004 ; 6 630-6.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2004.02.012
  • Source: Microbes and Infection. Unidades: ICB, FMRP

    Assunto: IMUNOLOGIA

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      MARTINS, Gislâine Aparecida et al. CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection, v. 6, p. 1133-1144, 2004Tradução . . Acesso em: 10 nov. 2024.
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      Martins, G. A., Campanelli, A. P., Silva, R. B., Takodoro, C. E., Russo, M., Cunha, F. de Q., et al. (2004). CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection, 6, 1133-1144.
    • NLM

      Martins GA, Campanelli AP, Silva RB, Takodoro CE, Russo M, Cunha F de Q, Rizzo LV, Silva JS da. CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection. 2004 ; 6 1133-1144.[citado 2024 nov. 10 ]
    • Vancouver

      Martins GA, Campanelli AP, Silva RB, Takodoro CE, Russo M, Cunha F de Q, Rizzo LV, Silva JS da. CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection. 2004 ; 6 1133-1144.[citado 2024 nov. 10 ]
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      ANDRÉ, Denise C. et al. Binding of laminin to Paracoccidioides brasiliensis induces a less severe pulmonary paracoccidioidomycosis caused by virulent and low-virulence isolates. Microbes and Infection, v. 6, n. 6, p. 549-58, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2004.02.010. Acesso em: 10 nov. 2024.
    • APA

      André, D. C., Lopes, J. D., Franco, M. F., Vaz, C. A. C., & Calich, V. L. G. (2004). Binding of laminin to Paracoccidioides brasiliensis induces a less severe pulmonary paracoccidioidomycosis caused by virulent and low-virulence isolates. Microbes and Infection, 6( 6), 549-58. doi:10.1016/j.micinf.2004.02.010
    • NLM

      André DC, Lopes JD, Franco MF, Vaz CAC, Calich VLG. Binding of laminin to Paracoccidioides brasiliensis induces a less severe pulmonary paracoccidioidomycosis caused by virulent and low-virulence isolates [Internet]. Microbes and Infection. 2004 ; 6( 6): 549-58.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2004.02.010
    • Vancouver

      André DC, Lopes JD, Franco MF, Vaz CAC, Calich VLG. Binding of laminin to Paracoccidioides brasiliensis induces a less severe pulmonary paracoccidioidomycosis caused by virulent and low-virulence isolates [Internet]. Microbes and Infection. 2004 ; 6( 6): 549-58.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.micinf.2004.02.010
  • Source: Microbes and Infection. Unidade: ICB

    Subjects: FARMACOLOGIA, IMUNOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      BALESTIERI, Filomena M P. et al. Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages. Microbes and Infection, v. 4, p. 23-29, 2002Tradução . . Disponível em: https://doi.org/10.1016/s1286-4579(01)01505-2. Acesso em: 10 nov. 2024.
    • APA

      Balestieri, F. M. P., Queiroz, A. R. P., Scavone, C., Costa, V. M. A., Barral-Netto, M., & Abrahamsohn, I. de A. (2002). Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages. Microbes and Infection, 4, 23-29. doi:10.1016/s1286-4579(01)01505-2
    • NLM

      Balestieri FMP, Queiroz ARP, Scavone C, Costa VMA, Barral-Netto M, Abrahamsohn I de A. Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages [Internet]. Microbes and Infection. 2002 ;4 23-29.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/s1286-4579(01)01505-2
    • Vancouver

      Balestieri FMP, Queiroz ARP, Scavone C, Costa VMA, Barral-Netto M, Abrahamsohn I de A. Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages [Internet]. Microbes and Infection. 2002 ;4 23-29.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/s1286-4579(01)01505-2
  • Source: Microbes and Infection. Unidades: FCF, ICB

    Assunto: IMUNOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      MARCONDES, Maria C G et al. Acute "Trypanosoma cruzi" infection is associated with anemia, trombocytopenia, leukopenia, and bone marrow hypoplasia: reversal by nifurtimox treatment. Microbes and Infection, v. 2, p. 347-352, 2000Tradução . . Disponível em: https://doi.org/10.1016/s1286-4579(00)00333-6. Acesso em: 10 nov. 2024.
    • APA

      Marcondes, M. C. G., Borelli, P., Yoshida, N., & Russo, M. (2000). Acute "Trypanosoma cruzi" infection is associated with anemia, trombocytopenia, leukopenia, and bone marrow hypoplasia: reversal by nifurtimox treatment. Microbes and Infection, 2, 347-352. doi:10.1016/s1286-4579(00)00333-6
    • NLM

      Marcondes MCG, Borelli P, Yoshida N, Russo M. Acute "Trypanosoma cruzi" infection is associated with anemia, trombocytopenia, leukopenia, and bone marrow hypoplasia: reversal by nifurtimox treatment [Internet]. Microbes and Infection. 2000 ; 2 347-352.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/s1286-4579(00)00333-6
    • Vancouver

      Marcondes MCG, Borelli P, Yoshida N, Russo M. Acute "Trypanosoma cruzi" infection is associated with anemia, trombocytopenia, leukopenia, and bone marrow hypoplasia: reversal by nifurtimox treatment [Internet]. Microbes and Infection. 2000 ; 2 347-352.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/s1286-4579(00)00333-6

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