Filtros : "Indexado na Base de Dados MEDLINE" "Kawamoto, Elisa Mitiko" Removido: "Burton Jr, G. A" Limpar

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  • Source: Ageing Research Reviews. Unidade: ICB

    Assunto: FARMACOLOGIA

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      CARARO-LOPES, Marina Minto et al. The relevance of α-KLOTHO to the central nervous system: some key questions. Ageing Research Reviews, v. 36, p. 137-148, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2017.03.003. Acesso em: 01 jun. 2024.
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      Cararo-Lopes, M. M., Mazucanti, C. H. Y., Scavone, C., Kawamoto, E. M., & Berwick, D. C. (2017). The relevance of α-KLOTHO to the central nervous system: some key questions. Ageing Research Reviews, 36, 137-148. doi:10.1016/j.arr.2017.03.003
    • NLM

      Cararo-Lopes MM, Mazucanti CHY, Scavone C, Kawamoto EM, Berwick DC. The relevance of α-KLOTHO to the central nervous system: some key questions [Internet]. Ageing Research Reviews. 2017 ; 36 137-148.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.arr.2017.03.003
    • Vancouver

      Cararo-Lopes MM, Mazucanti CHY, Scavone C, Kawamoto EM, Berwick DC. The relevance of α-KLOTHO to the central nervous system: some key questions [Internet]. Ageing Research Reviews. 2017 ; 36 137-148.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.arr.2017.03.003
  • Source: Scientific Reports. Unidade: ICB

    Assunto: FARMACOLOGIA

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      KINOSHITA, Paula F. et al. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells. Scientific Reports, v. 7, n. 1, p. 4894, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-05075-9. Acesso em: 01 jun. 2024.
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      Kinoshita, P. F., Yshii, L. M., Orellana, A. M. M., Paixão, A. G., Vasconcelos, A. R., Lima, L. de S., et al. (2017). Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells. Scientific Reports, 7( 1), 4894. doi:10.1038/s41598-017-05075-9
    • NLM

      Kinoshita PF, Yshii LM, Orellana AMM, Paixão AG, Vasconcelos AR, Lima L de S, Kawamoto EM, Scavone C. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells [Internet]. Scientific Reports. 2017 ; 7( 1): 4894.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1038/s41598-017-05075-9
    • Vancouver

      Kinoshita PF, Yshii LM, Orellana AMM, Paixão AG, Vasconcelos AR, Lima L de S, Kawamoto EM, Scavone C. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells [Internet]. Scientific Reports. 2017 ; 7( 1): 4894.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1038/s41598-017-05075-9
  • Source: Aging. Unidade: ICB

    Subjects: FARMACOLOGIA, RATOS, HIPOCAMPU DE ANIMAL

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      ORELLANA, Ana Maria Marques et al. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus. Aging, v. 7, n. 12, p. 1094-1111, 2015Tradução . . Disponível em: https://doi.org/10.18632/aging.100853. Acesso em: 01 jun. 2024.
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      Orellana, A. M. M., Vasconcelos, A. R., Leite, J. A., Lima, L. de S., Andreotti, D. Z., Munhoz, C. D., et al. (2015). Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus. Aging, 7( 12), 1094-1111. doi:10.18632/aging.100853
    • NLM

      Orellana AMM, Vasconcelos AR, Leite JA, Lima L de S, Andreotti DZ, Munhoz CD, Kawamoto EM, Scavone C. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus [Internet]. Aging. 2015 ; 7( 12): 1094-1111.[citado 2024 jun. 01 ] Available from: https://doi.org/10.18632/aging.100853
    • Vancouver

      Orellana AMM, Vasconcelos AR, Leite JA, Lima L de S, Andreotti DZ, Munhoz CD, Kawamoto EM, Scavone C. Age-related neuroinflammation and changes in AKT-GSK-3β and WNT/ β-CATENIN signaling in rat hippocampus [Internet]. Aging. 2015 ; 7( 12): 1094-1111.[citado 2024 jun. 01 ] Available from: https://doi.org/10.18632/aging.100853
  • Source: Current Topics in Medicinal Chemistry. Unidade: ICB

    Subjects: FARMACOLOGIA, ENVELHECIMENTO, INSULINA, DEGENERAÇÃO NEURAL

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      MAZUCANTI, Caio Henrique et al. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration. Current Topics in Medicinal Chemistry, v. 15, n. 21, p. 2116-2138, 2015Tradução . . Disponível em: https://doi.org/10.2174/1568026615666150610125715. Acesso em: 01 jun. 2024.
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      Mazucanti, C. H., Cabral-Costa, J. V., Vasconcelos, A. R., Andreotti, D. Z., Scavone, C., & Kawamoto, E. M. (2015). Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration. Current Topics in Medicinal Chemistry, 15( 21), 2116-2138. doi:10.2174/1568026615666150610125715
    • NLM

      Mazucanti CH, Cabral-Costa JV, Vasconcelos AR, Andreotti DZ, Scavone C, Kawamoto EM. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration [Internet]. Current Topics in Medicinal Chemistry. 2015 ; 15( 21): 2116-2138.[citado 2024 jun. 01 ] Available from: https://doi.org/10.2174/1568026615666150610125715
    • Vancouver

      Mazucanti CH, Cabral-Costa JV, Vasconcelos AR, Andreotti DZ, Scavone C, Kawamoto EM. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration [Internet]. Current Topics in Medicinal Chemistry. 2015 ; 15( 21): 2116-2138.[citado 2024 jun. 01 ] Available from: https://doi.org/10.2174/1568026615666150610125715
  • Source: Neurobiology of Aging. Unidades: FCF, ICB

    Subjects: FARMACOLOGIA, LIPOLISSACARIDEOS, HIPOCAMPU DE ANIMAL, DOENÇAS DEGENERATIVAS

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      VASCONCELOS, Andrea Rodrigues et al. Effects of intermittent fasting on age-related changes on Na,K-ATPase activity and oxidative status induced by lipopolysaccharide in rat hippocampus. Neurobiology of Aging, v. 36, n. 5, p. 1914-1923, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.neurobiolaging.2015.02.020. Acesso em: 01 jun. 2024.
    • APA

      Vasconcelos, A. R., Kinoshita, P. F., Yshii, L. M., Orellana, A. M. M., Böhmer, A. E., Lima, L. de S., et al. (2015). Effects of intermittent fasting on age-related changes on Na,K-ATPase activity and oxidative status induced by lipopolysaccharide in rat hippocampus. Neurobiology of Aging, 36( 5), 1914-1923. doi:10.1016/j.neurobiolaging.2015.02.020
    • NLM

      Vasconcelos AR, Kinoshita PF, Yshii LM, Orellana AMM, Böhmer AE, Lima L de S, Alves R, Andreotti DZ, Marcourakis T, Scavone C, Kawamoto EM. Effects of intermittent fasting on age-related changes on Na,K-ATPase activity and oxidative status induced by lipopolysaccharide in rat hippocampus [Internet]. Neurobiology of Aging. 2015 ; 36( 5): 1914-1923.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.neurobiolaging.2015.02.020
    • Vancouver

      Vasconcelos AR, Kinoshita PF, Yshii LM, Orellana AMM, Böhmer AE, Lima L de S, Alves R, Andreotti DZ, Marcourakis T, Scavone C, Kawamoto EM. Effects of intermittent fasting on age-related changes on Na,K-ATPase activity and oxidative status induced by lipopolysaccharide in rat hippocampus [Internet]. Neurobiology of Aging. 2015 ; 36( 5): 1914-1923.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.neurobiolaging.2015.02.020
  • Source: Journal of Neuroinflammation. Unidades: ICB, EACH

    Assunto: FARMACOLOGIA

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      VASCONCELOS, Andrea Rodrigues et al. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, v. 11, n. 85, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-85. Acesso em: 01 jun. 2024.
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      Vasconcelos, A. R., Yshii, L. M., Viel, T. A., Buck, H. S., Mattson, M. P., Scavone, C., & Kawamoto, E. M. (2014). Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, 11( 85), 1-14. doi:10.1186/1742-2094-11-85
    • NLM

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1186/1742-2094-11-85
    • Vancouver

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1186/1742-2094-11-85
  • Source: International Journal of Immunopathology and Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      TZANNO-MARTINS, C. et al. Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology, v. 27, n. 1, p. 25-35, 2014Tradução . . Acesso em: 01 jun. 2024.
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      Tzanno-Martins, C., Biavo, B. M. M., Ferreira-Filho, O., Ribeiro-Junior, E., João-Luiz, M. V. de S., Degaspari, S., et al. (2014). Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology, 27( 1), 25-35.
    • NLM

      Tzanno-Martins C, Biavo BMM, Ferreira-Filho O, Ribeiro-Junior E, João-Luiz MV de S, Degaspari S, Scavone C, Kawamoto EM. Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology. 2014 ; 27( 1): 25-35.[citado 2024 jun. 01 ]
    • Vancouver

      Tzanno-Martins C, Biavo BMM, Ferreira-Filho O, Ribeiro-Junior E, João-Luiz MV de S, Degaspari S, Scavone C, Kawamoto EM. Clinical efficacy, safety and anti-inflammatory activity of two sevelamer tablet forms in patients on low-flux hemodialysis. International Journal of Immunopathology and Pharmacology. 2014 ; 27( 1): 25-35.[citado 2024 jun. 01 ]
  • Source: Journal of Neuroinflammation. Unidade: ICB

    Subjects: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO

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      KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 01 jun. 2024.
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      Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
    • NLM

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
    • Vancouver

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
  • Source: Neuropharmacology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 01 jun. 2024.
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      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
  • Source: Neurosignals. Unidade: ICB

    Assunto: FARMACOLOGIA

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      KAWAMOTO, Elisa Mitiko et al. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neurosignals, v. 21, n. 1-2, p. 75-88, 2013Tradução . . Disponível em: https://doi.org/10.1159/000336074. Acesso em: 01 jun. 2024.
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      Kawamoto, E. M., Scavone, C., Mattson, M. P., & Camandola, S. (2013). Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neurosignals, 21( 1-2), 75-88. doi:10.1159/000336074
    • NLM

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neurosignals. 2013 ; 21( 1-2): 75-88.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1159/000336074
    • Vancouver

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neurosignals. 2013 ; 21( 1-2): 75-88.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1159/000336074
  • Source: Age. Unidades: ICB, FCF

    Subjects: ÓXIDO NÍTRICO, ENVELHECIMENTO

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      KAWAMOTO, Elisa Mitiko et al. Age-related changes in nitric oxide activity, cyclic GMP, and TBARS levels in platelets and erythrocytes reflect the oxidative status in central nervous system. Age, v. 35, p. 331-342, 2013Tradução . . Disponível em: https://doi.org/10.1007/s11357-011-9365-7. Acesso em: 01 jun. 2024.
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      Kawamoto, E. M., Vasconcelos, A. R., Degaspari, S., Böhmer, A. E., Scavone, C., & Marcourakis, T. (2013). Age-related changes in nitric oxide activity, cyclic GMP, and TBARS levels in platelets and erythrocytes reflect the oxidative status in central nervous system. Age, 35, 331-342. doi:10.1007/s11357-011-9365-7
    • NLM

      Kawamoto EM, Vasconcelos AR, Degaspari S, Böhmer AE, Scavone C, Marcourakis T. Age-related changes in nitric oxide activity, cyclic GMP, and TBARS levels in platelets and erythrocytes reflect the oxidative status in central nervous system [Internet]. Age. 2013 ; 35 331-342.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1007/s11357-011-9365-7
    • Vancouver

      Kawamoto EM, Vasconcelos AR, Degaspari S, Böhmer AE, Scavone C, Marcourakis T. Age-related changes in nitric oxide activity, cyclic GMP, and TBARS levels in platelets and erythrocytes reflect the oxidative status in central nervous system [Internet]. Age. 2013 ; 35 331-342.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1007/s11357-011-9365-7
  • Source: Neuro-Signals. Unidade: ICB

    Assunto: FARMACOLOGIA

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      KAWAMOTO, Elisa Mitiko et al. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neuro-Signals, v. 21, n. 1-2, p. 75-88, 2012Tradução . . Disponível em: https://doi.org/10.1159/000336074. Acesso em: 01 jun. 2024.
    • APA

      Kawamoto, E. M., Scavone, C., Mattson, M. P., & Camandola, S. (2012). Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neuro-Signals, 21( 1-2), 75-88. doi:10.1159/000336074
    • NLM

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neuro-Signals. 2012 ; 21( 1-2): 75-88.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1159/000336074
    • Vancouver

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neuro-Signals. 2012 ; 21( 1-2): 75-88.[citado 2024 jun. 01 ] Available from: https://doi.org/10.1159/000336074

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