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  • Source: Inflammopharmacology. Unidades: RUSP, ICB

    Subjects: INFLAMAÇÃO, FENÓTIPOS, INTERLEUCINA, FIBROSE PULMONAR, MACROFAGOS, INTERFERON

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    • ABNT

      GRABARZ, Felip et al. Protective role of NKT cells and macrophage M2-drivenphenotype in bleomycin-induced pulmonary fibrosis. Inflammopharmacology, v. 26, p. 491-504, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10787-017-0383-7. Acesso em: 05 jul. 2024.
    • APA

      Grabarz, F., Aguiar, C. F., Correa-Costa, M., Hyane, M. I., Andrade-Oliveira, V., Landgraf, M. A., & Câmara, N. O. S. (2018). Protective role of NKT cells and macrophage M2-drivenphenotype in bleomycin-induced pulmonary fibrosis. Inflammopharmacology, 26, 491-504. doi:10.1007/s10787-017-0383-7
    • NLM

      Grabarz F, Aguiar CF, Correa-Costa M, Hyane MI, Andrade-Oliveira V, Landgraf MA, Câmara NOS. Protective role of NKT cells and macrophage M2-drivenphenotype in bleomycin-induced pulmonary fibrosis [Internet]. Inflammopharmacology. 2018 ; 26 491-504.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s10787-017-0383-7
    • Vancouver

      Grabarz F, Aguiar CF, Correa-Costa M, Hyane MI, Andrade-Oliveira V, Landgraf MA, Câmara NOS. Protective role of NKT cells and macrophage M2-drivenphenotype in bleomycin-induced pulmonary fibrosis [Internet]. Inflammopharmacology. 2018 ; 26 491-504.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s10787-017-0383-7
  • Source: Clinical and Experimental Allergy. Unidades: ICB, FMRP, FCFRP

    Assunto: IMUNOLOGIA

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      FONSECA, Denise Morais da et al. Recombinant DNA immunotherapy ameliorate established airway allergy in a IL-10 dependent pathway. Clinical and Experimental Allergy, v. 42, n. 1, p. 131-143, 2012Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2222.2011.03845.x. Acesso em: 05 jul. 2024.
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      Fonseca, D. M. da, Wowk, P. F., Paula, M. O., Campos, L. W., Gembre, A. F., Turato, W. M., et al. (2012). Recombinant DNA immunotherapy ameliorate established airway allergy in a IL-10 dependent pathway. Clinical and Experimental Allergy, 42( 1), 131-143. doi:10.1111/j.1365-2222.2011.03845.x
    • NLM

      Fonseca DM da, Wowk PF, Paula MO, Campos LW, Gembre AF, Turato WM, Ramos SG, Dias-Baruffi M, Barboza R, Gomes E, Silva CL, Russo M, Bonato VLD. Recombinant DNA immunotherapy ameliorate established airway allergy in a IL-10 dependent pathway [Internet]. Clinical and Experimental Allergy. 2012 ; 42( 1): 131-143.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1365-2222.2011.03845.x
    • Vancouver

      Fonseca DM da, Wowk PF, Paula MO, Campos LW, Gembre AF, Turato WM, Ramos SG, Dias-Baruffi M, Barboza R, Gomes E, Silva CL, Russo M, Bonato VLD. Recombinant DNA immunotherapy ameliorate established airway allergy in a IL-10 dependent pathway [Internet]. Clinical and Experimental Allergy. 2012 ; 42( 1): 131-143.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1365-2222.2011.03845.x
  • Source: Immunology and Cell Biology. Unidades: ICB, FMRP, FCFRP

    Assunto: IMUNOLOGIA

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      FONSECA, Denise Morais da et al. IFN-'gama'-mediated efficacy of allergen-free immunotherapy using mycobacterial antigens and CpG-ODN. Immunology and Cell Biology, v. 89, n. 7, p. 777-785, 2011Tradução . . Disponível em: https://doi.org/10.1038/icb.2011.9. Acesso em: 05 jul. 2024.
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      Fonseca, D. M. da, Paula, M. O. e, Wowk, P. F., Campos, L. W., Gembre, A. F., Turato, W. M., et al. (2011). IFN-'gama'-mediated efficacy of allergen-free immunotherapy using mycobacterial antigens and CpG-ODN. Immunology and Cell Biology, 89( 7), 777-785. doi:10.1038/icb.2011.9
    • NLM

      Fonseca DM da, Paula MO e, Wowk PF, Campos LW, Gembre AF, Turato WM, Ramos SG, Dias-Baruffi M, Barboza R, Gomes E, Horn C, Marchal G, Arruda LK de P, Russo M, Bonato VLD. IFN-'gama'-mediated efficacy of allergen-free immunotherapy using mycobacterial antigens and CpG-ODN [Internet]. Immunology and Cell Biology. 2011 ; 89( 7): 777-785.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1038/icb.2011.9
    • Vancouver

      Fonseca DM da, Paula MO e, Wowk PF, Campos LW, Gembre AF, Turato WM, Ramos SG, Dias-Baruffi M, Barboza R, Gomes E, Horn C, Marchal G, Arruda LK de P, Russo M, Bonato VLD. IFN-'gama'-mediated efficacy of allergen-free immunotherapy using mycobacterial antigens and CpG-ODN [Internet]. Immunology and Cell Biology. 2011 ; 89( 7): 777-785.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1038/icb.2011.9
  • Source: FEMS Immunology and Medical Microbiology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      NISHIKAKU, Angela Satie et al. Immunolocalization of IFN-gamma in the lesions of resistant and susceptible mice to Paracoccidioides brasiliensis infection. FEMS Immunology and Medical Microbiology, v. 63, n. 2, p. 281-288, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1574-695X.2011.00851.x. Acesso em: 05 jul. 2024.
    • APA

      Nishikaku, A. S., Molina, R. F. S., Albe, B. P., Cunha, C. da S., Scavone, R., Pizzo, C. R. P., et al. (2011). Immunolocalization of IFN-gamma in the lesions of resistant and susceptible mice to Paracoccidioides brasiliensis infection. FEMS Immunology and Medical Microbiology, 63( 2), 281-288. doi:10.1111/j.1574-695X.2011.00851.x
    • NLM

      Nishikaku AS, Molina RFS, Albe BP, Cunha C da S, Scavone R, Pizzo CRP, Camargo ZP de, Burger E. Immunolocalization of IFN-gamma in the lesions of resistant and susceptible mice to Paracoccidioides brasiliensis infection [Internet]. FEMS Immunology and Medical Microbiology. 2011 ; 63( 2): 281-288.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1574-695X.2011.00851.x
    • Vancouver

      Nishikaku AS, Molina RFS, Albe BP, Cunha C da S, Scavone R, Pizzo CRP, Camargo ZP de, Burger E. Immunolocalization of IFN-gamma in the lesions of resistant and susceptible mice to Paracoccidioides brasiliensis infection [Internet]. FEMS Immunology and Medical Microbiology. 2011 ; 63( 2): 281-288.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1574-695X.2011.00851.x
  • Source: Cell and Tissue Research. Unidade: ICB

    Subjects: ANATOMIA, IMUNOLOGIA

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    • ABNT

      MATTOS, Gabriele Ebling et al. Comparative effect of FGF2, synthetic peptides 1-28 N-POMC and ACTH on proliferation in rat adrenal cell primary cultures. Cell and Tissue Research, v. 345, n. 3, p. 343-356, 2011Tradução . . Disponível em: https://doi.org/10.1007/s00441-011-1220-8. Acesso em: 05 jul. 2024.
    • APA

      Mattos, G. E., Jacysyn, J. de F., Amarante-Mendes, J. G. P., & Lotfi, C. F. P. (2011). Comparative effect of FGF2, synthetic peptides 1-28 N-POMC and ACTH on proliferation in rat adrenal cell primary cultures. Cell and Tissue Research, 345( 3), 343-356. doi:10.1007/s00441-011-1220-8
    • NLM

      Mattos GE, Jacysyn J de F, Amarante-Mendes JGP, Lotfi CFP. Comparative effect of FGF2, synthetic peptides 1-28 N-POMC and ACTH on proliferation in rat adrenal cell primary cultures [Internet]. Cell and Tissue Research. 2011 ; 345( 3): 343-356.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00441-011-1220-8
    • Vancouver

      Mattos GE, Jacysyn J de F, Amarante-Mendes JGP, Lotfi CFP. Comparative effect of FGF2, synthetic peptides 1-28 N-POMC and ACTH on proliferation in rat adrenal cell primary cultures [Internet]. Cell and Tissue Research. 2011 ; 345( 3): 343-356.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00441-011-1220-8
  • Source: Oncogene. Unidades: FMRP, ICB, FCFRP

    Assunto: IMUNOLOGIA

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      CARVALHO, Daniel Diniz de et al. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, v. 30, n. 2, p. 223-233, 2011Tradução . . Disponível em: https://doi.org/10.1038/onc.2010.409. Acesso em: 05 jul. 2024.
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      Carvalho, D. D. de, Binato, R., Pereira, W. de O., Leroy, J. M. G., Colassanti, M. D., Proto-Siqueira, R., et al. (2011). BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, 30( 2), 223-233. doi:10.1038/onc.2010.409
    • NLM

      Carvalho DD de, Binato R, Pereira W de O, Leroy JMG, Colassanti MD, Proto-Siqueira R, Silva AEBB da, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1038/onc.2010.409
    • Vancouver

      Carvalho DD de, Binato R, Pereira W de O, Leroy JMG, Colassanti MD, Proto-Siqueira R, Silva AEBB da, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1038/onc.2010.409
  • Source: Circulation. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      KETELHUTH, Daniel F. J. et al. Identification of a danger-associated peptide from apolipoprotein B100 (ApoBDS-1) that triggers innate proatherogenic responses. Circulation, v. 124, n. 22, p. 2433-2443 supl. 1-7, 2011Tradução . . Acesso em: 05 jul. 2024.
    • APA

      Ketelhuth, D. F. J., Rios, F. J. O., Wang, Y., Liu, H., Johansson, M. E., Fredrikson, G. N., et al. (2011). Identification of a danger-associated peptide from apolipoprotein B100 (ApoBDS-1) that triggers innate proatherogenic responses. Circulation, 124( 22), 2433-2443 supl. 1-7.
    • NLM

      Ketelhuth DFJ, Rios FJO, Wang Y, Liu H, Johansson ME, Fredrikson GN, Hedin U, Gidlund MA, Nilsson J, Hansson GK, Yan Z-qun. Identification of a danger-associated peptide from apolipoprotein B100 (ApoBDS-1) that triggers innate proatherogenic responses. Circulation. 2011 ; 124( 22): 2433-2443 supl. 1-7.[citado 2024 jul. 05 ]
    • Vancouver

      Ketelhuth DFJ, Rios FJO, Wang Y, Liu H, Johansson ME, Fredrikson GN, Hedin U, Gidlund MA, Nilsson J, Hansson GK, Yan Z-qun. Identification of a danger-associated peptide from apolipoprotein B100 (ApoBDS-1) that triggers innate proatherogenic responses. Circulation. 2011 ; 124( 22): 2433-2443 supl. 1-7.[citado 2024 jul. 05 ]
  • Source: Arteriosclerosis, Thrombosis Vascular Biology. Unidades: ICB, FM, FCF

    Assunto: IMUNOLOGIA

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      RUDNICKI, Martina et al. Hypoxia inducible factor–dependent regulation of angiogenesis by nitro–fatty acids. Arteriosclerosis, Thrombosis Vascular Biology, v. 31, n. 6, p. 1360-1367, 2011Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.111.224626. Acesso em: 05 jul. 2024.
    • APA

      Rudnicki, M., Faine, L. A., Dehne, N., Namgaladze, D., Ferderbar, S., Weinlich, R., et al. (2011). Hypoxia inducible factor–dependent regulation of angiogenesis by nitro–fatty acids. Arteriosclerosis, Thrombosis Vascular Biology, 31( 6), 1360-1367. doi:10.1161/ATVBAHA.111.224626
    • NLM

      Rudnicki M, Faine LA, Dehne N, Namgaladze D, Ferderbar S, Weinlich R, Amarante-Mendes JGP, Yan CYI, Krieger JE, Brüne B, Abdalla DSP. Hypoxia inducible factor–dependent regulation of angiogenesis by nitro–fatty acids [Internet]. Arteriosclerosis, Thrombosis Vascular Biology. 2011 ; 31( 6): 1360-1367.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1161/ATVBAHA.111.224626
    • Vancouver

      Rudnicki M, Faine LA, Dehne N, Namgaladze D, Ferderbar S, Weinlich R, Amarante-Mendes JGP, Yan CYI, Krieger JE, Brüne B, Abdalla DSP. Hypoxia inducible factor–dependent regulation of angiogenesis by nitro–fatty acids [Internet]. Arteriosclerosis, Thrombosis Vascular Biology. 2011 ; 31( 6): 1360-1367.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1161/ATVBAHA.111.224626
  • Source: American Journal of Reproductive Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      OLIVEIRA, Leandro de et al. Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood. American Journal of Reproductive Immunology, v. 65, n. 1, p. 11-12, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1600-0897.2010.00915.x. Acesso em: 05 jul. 2024.
    • APA

      Oliveira, L. de, Larocca, R. A., Sass, N., & Câmara, N. O. S. (2011). Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood. American Journal of Reproductive Immunology, 65( 1), 11-12. doi:10.1111/j.1600-0897.2010.00915.x
    • NLM

      Oliveira L de, Larocca RA, Sass N, Câmara NOS. Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood [Internet]. American Journal of Reproductive Immunology. 2011 ; 65( 1): 11-12.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1600-0897.2010.00915.x
    • Vancouver

      Oliveira L de, Larocca RA, Sass N, Câmara NOS. Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood [Internet]. American Journal of Reproductive Immunology. 2011 ; 65( 1): 11-12.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1600-0897.2010.00915.x
  • Source: Journal of Immunology. Unidades: ICB, FMRP

    Assunto: IMUNOLOGIA

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      CARREGARO, Vanessa et al. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions. Journal of Immunology, v. 187, n. 8, p. 4347-4359, 2011Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1003404. Acesso em: 05 jul. 2024.
    • APA

      Carregaro, V., Nunes, A. de S., Cunha, T. M., Grespan, R., Oliveira, C. J. F., Lima-Junior, D. S., et al. (2011). Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions. Journal of Immunology, 187( 8), 4347-4359. doi:10.4049/jimmunol.1003404
    • NLM

      Carregaro V, Nunes A de S, Cunha TM, Grespan R, Oliveira CJF, Lima-Junior DS, Costa DL, Verri Jr WA, Milanezi CM, Pham VM, Brand DD, Venezuela JG, Silva JS da, Ribeiro JMC, Cunha FQ. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions [Internet]. Journal of Immunology. 2011 ; 187( 8): 4347-4359.[citado 2024 jul. 05 ] Available from: https://doi.org/10.4049/jimmunol.1003404
    • Vancouver

      Carregaro V, Nunes A de S, Cunha TM, Grespan R, Oliveira CJF, Lima-Junior DS, Costa DL, Verri Jr WA, Milanezi CM, Pham VM, Brand DD, Venezuela JG, Silva JS da, Ribeiro JMC, Cunha FQ. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions [Internet]. Journal of Immunology. 2011 ; 187( 8): 4347-4359.[citado 2024 jul. 05 ] Available from: https://doi.org/10.4049/jimmunol.1003404
  • Source: Scandinavian Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      MACCHIAVERNI, Patrícia et al. Mother to child transfer of IgG and IgA antibodies against Dermatophagoides pteronyssinus. Scandinavian Journal of Immunology, v. 74, n. 6, p. 619-627, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1365-3083.2011.02615.x. Acesso em: 05 jul. 2024.
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      Macchiaverni, P., Arslanian, C., Frazão, J. B., Palmeira, P., Russo, M., Verhasselt, V., & Condino-Neto, A. (2011). Mother to child transfer of IgG and IgA antibodies against Dermatophagoides pteronyssinus. Scandinavian Journal of Immunology, 74( 6), 619-627. doi:10.1111/j.1365-3083.2011.02615.x
    • NLM

      Macchiaverni P, Arslanian C, Frazão JB, Palmeira P, Russo M, Verhasselt V, Condino-Neto A. Mother to child transfer of IgG and IgA antibodies against Dermatophagoides pteronyssinus [Internet]. Scandinavian Journal of Immunology. 2011 ; 74( 6): 619-627.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1365-3083.2011.02615.x
    • Vancouver

      Macchiaverni P, Arslanian C, Frazão JB, Palmeira P, Russo M, Verhasselt V, Condino-Neto A. Mother to child transfer of IgG and IgA antibodies against Dermatophagoides pteronyssinus [Internet]. Scandinavian Journal of Immunology. 2011 ; 74( 6): 619-627.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1111/j.1365-3083.2011.02615.x
  • Source: Cellular and Molecular Life Sciences. Unidade: ICB

    Assunto: IMUNOLOGIA

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      AMARANTE-MENDES, João Gustavo Pessini. Cell death and the well of the organismo. Cellular and Molecular Life Sciences, v. 67, n. 10, p. 1565-1566, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00018-010-0334-6. Acesso em: 05 jul. 2024.
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      Amarante-Mendes, J. G. P. (2010). Cell death and the well of the organismo. Cellular and Molecular Life Sciences, 67( 10), 1565-1566. doi:10.1007/s00018-010-0334-6
    • NLM

      Amarante-Mendes JGP. Cell death and the well of the organismo [Internet]. Cellular and Molecular Life Sciences. 2010 ; 67( 10): 1565-1566.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00018-010-0334-6
    • Vancouver

      Amarante-Mendes JGP. Cell death and the well of the organismo [Internet]. Cellular and Molecular Life Sciences. 2010 ; 67( 10): 1565-1566.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00018-010-0334-6
  • Source: Shock. Unidade: ICB

    Subjects: FARMACOLOGIA, IMUNOLOGIA

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      MARTINS, Joilson de Oliveira et al. Signaling pathways and mediators in lps-induced lung inflammation in diabetic rats: role of insulin. Shock, v. 33, n. 1, p. 76-82, 2010Tradução . . Acesso em: 05 jul. 2024.
    • APA

      Martins, J. de O., Ferracini, M., Anger, D. B. C., Martins, D. O., Ribeiro Jr., L. F., Sannomiya, P., & Jancar, S. (2010). Signaling pathways and mediators in lps-induced lung inflammation in diabetic rats: role of insulin. Shock, 33( 1), 76-82.
    • NLM

      Martins J de O, Ferracini M, Anger DBC, Martins DO, Ribeiro Jr. LF, Sannomiya P, Jancar S. Signaling pathways and mediators in lps-induced lung inflammation in diabetic rats: role of insulin. Shock. 2010 ; 33( 1): 76-82.[citado 2024 jul. 05 ]
    • Vancouver

      Martins J de O, Ferracini M, Anger DBC, Martins DO, Ribeiro Jr. LF, Sannomiya P, Jancar S. Signaling pathways and mediators in lps-induced lung inflammation in diabetic rats: role of insulin. Shock. 2010 ; 33( 1): 76-82.[citado 2024 jul. 05 ]
  • Source: European Journal of Pharmacology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      CAMPANHOLE, Gabriela et al. Bradykinin inducible receptor is essential to lipopolysaccharide-induced acute lung injury in mice. European Journal of Pharmacology, v. 634, n. 1/3, p. 132-7, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2010.02.002. Acesso em: 05 jul. 2024.
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      Campanhole, G., Landgraf, R. G., Borducchi, E. N., Semedo, P., Wang, P. H. M., Amano, M. T., et al. (2010). Bradykinin inducible receptor is essential to lipopolysaccharide-induced acute lung injury in mice. European Journal of Pharmacology, 634( 1/3), 132-7. doi:10.1016/j.ejphar.2010.02.002
    • NLM

      Campanhole G, Landgraf RG, Borducchi EN, Semedo P, Wang PHM, Amano MT, Russo M, Pacheco-Silva A, Jancar S, Câmara NOS. Bradykinin inducible receptor is essential to lipopolysaccharide-induced acute lung injury in mice [Internet]. European Journal of Pharmacology. 2010 ; 634( 1/3): 132-7.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.ejphar.2010.02.002
    • Vancouver

      Campanhole G, Landgraf RG, Borducchi EN, Semedo P, Wang PHM, Amano MT, Russo M, Pacheco-Silva A, Jancar S, Câmara NOS. Bradykinin inducible receptor is essential to lipopolysaccharide-induced acute lung injury in mice [Internet]. European Journal of Pharmacology. 2010 ; 634( 1/3): 132-7.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.ejphar.2010.02.002
  • Source: European Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SINGH, Satya P. et al. Changes in histone acetylation and methylation that are important for persistent but not transient expression. of CCR4 in human CD4+ T cells. European Journal of Immunology, v. 40, n. 11, p. 3183-3197, 2010Tradução . . Disponível em: https://doi.org/10.1002/eji.201040443. Acesso em: 05 jul. 2024.
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      Singh, S. P., Camargo, M. M. de, Zhang, H. H., Foley, J. F., Hedrick, M. N., & Farber, J. M. (2010). Changes in histone acetylation and methylation that are important for persistent but not transient expression. of CCR4 in human CD4+ T cells. European Journal of Immunology, 40( 11), 3183-3197. doi:10.1002/eji.201040443
    • NLM

      Singh SP, Camargo MM de, Zhang HH, Foley JF, Hedrick MN, Farber JM. Changes in histone acetylation and methylation that are important for persistent but not transient expression. of CCR4 in human CD4+ T cells [Internet]. European Journal of Immunology. 2010 ; 40( 11): 3183-3197.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/eji.201040443
    • Vancouver

      Singh SP, Camargo MM de, Zhang HH, Foley JF, Hedrick MN, Farber JM. Changes in histone acetylation and methylation that are important for persistent but not transient expression. of CCR4 in human CD4+ T cells [Internet]. European Journal of Immunology. 2010 ; 40( 11): 3183-3197.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/eji.201040443
  • Source: Cellular and Molecular Life Sciences. Unidade: ICB

    Assunto: IMUNOLOGIA

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      WEINLICH, Ricardo e BRUNNER, T. e AMARANTE-MENDES, João Gustavo Pessini. Control of death receptor ligand activity by posttranslational modifications. Cellular and Molecular Life Sciences, v. 67, n. 10, p. 1631-1642, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00018-010-0289-7. Acesso em: 05 jul. 2024.
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      Weinlich, R., Brunner, T., & Amarante-Mendes, J. G. P. (2010). Control of death receptor ligand activity by posttranslational modifications. Cellular and Molecular Life Sciences, 67( 10), 1631-1642. doi:10.1007/s00018-010-0289-7
    • NLM

      Weinlich R, Brunner T, Amarante-Mendes JGP. Control of death receptor ligand activity by posttranslational modifications [Internet]. Cellular and Molecular Life Sciences. 2010 ; 67( 10): 1631-1642.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00018-010-0289-7
    • Vancouver

      Weinlich R, Brunner T, Amarante-Mendes JGP. Control of death receptor ligand activity by posttranslational modifications [Internet]. Cellular and Molecular Life Sciences. 2010 ; 67( 10): 1631-1642.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00018-010-0289-7
  • Source: Inflammation Research. Unidade: ICB

    Assunto: IMUNOLOGIA

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      AMANO, Mariane Tami et al. A new model of outbred genetically selected mice which present a strong acute inflammatory response in the absence of complement component C5. Inflammation Research, v. 58, n. 4, p. 204-209, 2009Tradução . . Disponível em: https://doi.org/10.1007/s00011-008-8132-4. Acesso em: 05 jul. 2024.
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      Amano, M. T., Carneiro, A. S., Ribeiro, O. G., Cabrera, W. K., De Franco, M., Ibañez, O. M., et al. (2009). A new model of outbred genetically selected mice which present a strong acute inflammatory response in the absence of complement component C5. Inflammation Research, 58( 4), 204-209. doi:10.1007/s00011-008-8132-4
    • NLM

      Amano MT, Carneiro AS, Ribeiro OG, Cabrera WK, De Franco M, Ibañez OM, Isaac L, Starobinas N. A new model of outbred genetically selected mice which present a strong acute inflammatory response in the absence of complement component C5 [Internet]. Inflammation Research. 2009 ; 58( 4): 204-209.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00011-008-8132-4
    • Vancouver

      Amano MT, Carneiro AS, Ribeiro OG, Cabrera WK, De Franco M, Ibañez OM, Isaac L, Starobinas N. A new model of outbred genetically selected mice which present a strong acute inflammatory response in the absence of complement component C5 [Internet]. Inflammation Research. 2009 ; 58( 4): 204-209.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00011-008-8132-4
  • Source: Arquivos Brasileiros de Endocrinologia e Metabologia. Unidade: ICB

    Assunto: IMUNOLOGIA

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      MOULIN, Cristiane Martins et al. Impact of adiposity on immunological parameters. Arquivos Brasileiros de Endocrinologia e Metabologia, v. 53, n. 2, p. 183-189, 2009Tradução . . Disponível em: https://doi.org/10.1590/s0004-27302009000200010. Acesso em: 05 jul. 2024.
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      Moulin, C. M., Marguti, I., Peron, J. P. S., Rizzo, L. V., & Halpern, A. (2009). Impact of adiposity on immunological parameters. Arquivos Brasileiros de Endocrinologia e Metabologia, 53( 2), 183-189. doi:10.1590/s0004-27302009000200010
    • NLM

      Moulin CM, Marguti I, Peron JPS, Rizzo LV, Halpern A. Impact of adiposity on immunological parameters [Internet]. Arquivos Brasileiros de Endocrinologia e Metabologia. 2009 ; 53( 2): 183-189.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1590/s0004-27302009000200010
    • Vancouver

      Moulin CM, Marguti I, Peron JPS, Rizzo LV, Halpern A. Impact of adiposity on immunological parameters [Internet]. Arquivos Brasileiros de Endocrinologia e Metabologia. 2009 ; 53( 2): 183-189.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1590/s0004-27302009000200010
  • Source: Experimental and Toxicologic Pathology. Unidades: ICB, FMVZ

    Assunto: IMUNOLOGIA

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      NAGAMINE, Marcia Kazumi et al. Cytotoxic effects of butanolic extract from Pfaffia paniculata (Brazilian Ginseng) on cultured human breast cancer cell line MCF-7. Experimental and Toxicologic Pathology, v. 61, n. 1, p. 75-82, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.etp.2008.01.017. Acesso em: 05 jul. 2024.
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      Nagamine, M. K., Silva, T. C. da, Matsuzaki, P., Pinello, K. C., Cogliati, B., Pizzo, C. R., et al. (2009). Cytotoxic effects of butanolic extract from Pfaffia paniculata (Brazilian Ginseng) on cultured human breast cancer cell line MCF-7. Experimental and Toxicologic Pathology, 61( 1), 75-82. doi:10.1016/j.etp.2008.01.017
    • NLM

      Nagamine MK, Silva TC da, Matsuzaki P, Pinello KC, Cogliati B, Pizzo CR, Akisue G, Haraguchi M, Górniak SL, Sinhorini IL, Rao KVK, Barbuto JAM, Dagli MLZ. Cytotoxic effects of butanolic extract from Pfaffia paniculata (Brazilian Ginseng) on cultured human breast cancer cell line MCF-7 [Internet]. Experimental and Toxicologic Pathology. 2009 ; 61( 1): 75-82.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.etp.2008.01.017
    • Vancouver

      Nagamine MK, Silva TC da, Matsuzaki P, Pinello KC, Cogliati B, Pizzo CR, Akisue G, Haraguchi M, Górniak SL, Sinhorini IL, Rao KVK, Barbuto JAM, Dagli MLZ. Cytotoxic effects of butanolic extract from Pfaffia paniculata (Brazilian Ginseng) on cultured human breast cancer cell line MCF-7 [Internet]. Experimental and Toxicologic Pathology. 2009 ; 61( 1): 75-82.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.etp.2008.01.017
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      NUNES, Anderson de Sá et al. The immunomodulatory action of sialostatin L on dendritic cells reveals its potential to interfere with autoimmunity. Journal of Immunology, v. 182, n. 12, p. 7422-9, 2009Tradução . . Acesso em: 05 jul. 2024.
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      Nunes, A. de S., Bafica, A., Antonelli, L. R., Choi, E. Y., Francischetti, I. M. B., Andersen, J. F., et al. (2009). The immunomodulatory action of sialostatin L on dendritic cells reveals its potential to interfere with autoimmunity. Journal of Immunology, 182( 12), 7422-9.
    • NLM

      Nunes A de S, Bafica A, Antonelli LR, Choi EY, Francischetti IMB, Andersen JF, Shi G-P, Chavakis T, Ribeiro JM, Kotsyfakis M. The immunomodulatory action of sialostatin L on dendritic cells reveals its potential to interfere with autoimmunity. Journal of Immunology. 2009 ; 182( 12): 7422-9.[citado 2024 jul. 05 ]
    • Vancouver

      Nunes A de S, Bafica A, Antonelli LR, Choi EY, Francischetti IMB, Andersen JF, Shi G-P, Chavakis T, Ribeiro JM, Kotsyfakis M. The immunomodulatory action of sialostatin L on dendritic cells reveals its potential to interfere with autoimmunity. Journal of Immunology. 2009 ; 182( 12): 7422-9.[citado 2024 jul. 05 ]

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